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Article ; Online: Fractures and other chest wall abnormalities after thoracotomy for esophageal cancer: A retrospective cohort study.

Van Wijck, Suzanne F M / Barza, Athiná / Vermeulen, Jefrey / Eyck, Ben M / Van der Wilk, Berend J / Van der Harst, Erwin / Verhofstad, Michael H J / Lagarde, Sjoerd M / Van Lieshout, Esther M M / Wijffels, Mathieu M E

World journal of surgery

2024 Volume 48, Issue 3, Page(s) 662–672

Abstract: Background: Chest pain following a thoracotomy for esophageal cancer is frequently reported but poorly understood. This study aimed to (1) determine the prevalence of thoracotomy-related thoracic fractures on postoperative imaging and (2) compare ... ...

Abstract	Background: Chest pain following a thoracotomy for esophageal cancer is frequently reported but poorly understood. This study aimed to (1) determine the prevalence of thoracotomy-related thoracic fractures on postoperative imaging and (2) compare complications, long-term pain, and quality of life in patients with versus without these fractures. Methods: This retrospective cohort study enrolled patients with esophageal cancer who underwent a thoracotomy between 2010 and 2020 with pre- and postoperative CTs (<1 and/or >6 months). Disease-free patients were invited for questionnaires on pain and quality of life. Results: Of a total of 366 patients, thoracotomy-related rib fractures were seen in 144 (39%) and thoracic transverse process fractures in 4 (2%) patients. Patients with thoracic fractures more often developed complications (89% vs. 74%, p = 0.002), especially pneumonia (51% vs. 39%, p = 0.032). Questionnaires were completed by 77 after a median of 41 (P Conclusions: Thoracic fractures are prevalent in patients following a thoracotomy for esophageal cancer. These thoracic fractures were associated with an increased risk of postoperative complications, especially pneumonia, but an association with long-term pain or reduced quality of life was not confirmed.
MeSH term(s)	Humans ; Thoracic Wall ; Thoracotomy/adverse effects ; Retrospective Studies ; Quality of Life ; Rib Fractures/surgery ; Pneumonia/etiology ; Chest Pain/surgery ; Esophageal Neoplasms/complications
Language	English
Publishing date	2024-02-02
Publishing country	United States
Document type	Journal Article
ZDB-ID	224043-9
ISSN	1432-2323 ; 0364-2313
ISSN (online)	1432-2323
ISSN	0364-2313
DOI	10.1002/wjs.12083
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Article ; Online: Evaluation of pericoronary adipose tissue attenuation on CT.

Ma, Runlei / Fari, Roberto / van der Harst, Pim / N De Cecco, Carlo / E Stillman, Arthur / Vliegenthart, Rozemarijn / van Assen, Marly

The British journal of radiology

2023 Volume 96, Issue 1145, Page(s) 20220885

Abstract: Pericoronary adipose tissue (PCAT) is the fat deposit surrounding coronary arteries. Although PCAT is part of the larger epicardial adipose tissue (EAT) depot, it has different pathophysiological features and roles in the atherosclerosis process. While ... ...

Abstract	Pericoronary adipose tissue (PCAT) is the fat deposit surrounding coronary arteries. Although PCAT is part of the larger epicardial adipose tissue (EAT) depot, it has different pathophysiological features and roles in the atherosclerosis process. While EAT evaluation has been studied for years, PCAT evaluation is a relatively new concept. PCAT, especially the mean attenuation derived from CT images may be used to evaluate the inflammatory status of coronary arteries non-invasively. The most commonly used measure, PCAT
MeSH term(s)	Humans ; Coronary Angiography/methods ; Coronary Artery Disease ; Plaque, Atherosclerotic/pathology ; Adipose Tissue/diagnostic imaging ; Tomography, X-Ray Computed/methods ; Computed Tomography Angiography/methods ; Coronary Vessels
Language	English
Publishing date	2023-01-23
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	2982-8
ISSN	1748-880X ; 0007-1285
ISSN (online)	1748-880X
ISSN	0007-1285
DOI	10.1259/bjr.20220885
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Article ; Online: Incidence and predictors of heart failure with reduced and preserved ejection fraction after ST-elevation myocardial infarction in the contemporary era of early percutaneous coronary intervention.

Lenselink, Chris / Ricken, Kim W L M / Groot, Hilde E / de Bruijne, Tijs J / Hendriks, Tom / van der Harst, Pim / Voors, Adriaan A / Lipsic, Erik

European journal of heart failure

2024

Abstract: Aims: The development and incidence of de-novo heart failure after ST-elevation myocardial infarction (STEMI) in the contemporary era of rapid reperfusion are largely unknown. We aimed to establish the incidence of post-STEMI heart failure, stratified ... ...

Abstract	Aims: The development and incidence of de-novo heart failure after ST-elevation myocardial infarction (STEMI) in the contemporary era of rapid reperfusion are largely unknown. We aimed to establish the incidence of post-STEMI heart failure, stratified by left ventricular ejection fraction (LVEF) and to find predictors for its occurrence. Furthermore, we investigated the course of left ventricular systolic and diastolic function after STEMI. Methods and results: A total of 1172 all-comer STEMI patients from the CardioLines Biobank were included. Patients were predominantly male (74.5%) and 64 ± 12 years of age. During a median follow-up of 3.7 years (2.0, 5.5) we found a total incidence of post-STEMI heart failure of 10.9%, of which 52.1% heart failure with reduced ejection fraction (HFrEF), 29.4% heart failure with mildly reduced ejection fraction and 18.5% heart failure with preserved ejection fraction (HFpEF). Independent predictors for the development of HFrEF were male sex (β = 0.97, p = 0.009), lung crepitations (β = 1.09, p = 0.001), potassium level (mmol/L, β = 0.43, p = 0.012), neutrophil count (10 Conclusion: In the current era of early STEMI reperfusion, still one in 10 patients develops heart failure, with approximately half of the patients with a reduced and half with a mildly reduced or normal LVEF. Predictors for the development of HFrEF were different from HFpEF.
Language	English
Publishing date	2024-04-04
Publishing country	England
Document type	Journal Article
ZDB-ID	1483672-5
ISSN	1879-0844 ; 1388-9842
ISSN (online)	1879-0844
ISSN	1388-9842
DOI	10.1002/ejhf.3225
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Zs.A 5299: Show issues

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Article ; Online: Predictors of adverse diastolic remodeling in non-diabetic patients presenting with ST-elevation myocardial infarction.

Al Ali, Lawien / Groot, Hilde E / Assa, Solmaz / Lipsic, Erik / Hummel, Yoran M / van Veldhuisen, Dirk J / Voors, Adriaan A / van der Horst, Iwan C C / Lam, Carolyn S / van der Harst, Pim

BMC cardiovascular disorders

2023 Volume 23, Issue 1, Page(s) 44

Abstract: ... III trial. As parameters of diastolic remodeling we used (1.) the E/e' at 4 months adjusted for the E ... e' at hospitalization and (2.) the change in E/e' between hospitalization and 4 months. Multivariable ... predictors of higher E/e' at 4 months in a multivariable model (R: Conclusions: Our data supports ...

Abstract	Background: Adverse systolic remodeling after ST-elevation myocardial infarction (STEMI) is associated with poor clinical outcomes. However, little is known about diastolic remodeling. The purpose of this study was to identify the factors leading to diastolic remodeling. Methods: Echocardiography was performed during hospitalization and at 4 months follow-up in 267 non-diabetic STEMI patients from the GIPS-III trial. As parameters of diastolic remodeling we used (1.) the E/e' at 4 months adjusted for the E/e' at hospitalization and (2.) the change in E/e' between hospitalization and 4 months. Multivariable regression models correcting for age and sex were constructed to identify possible association of clinical and angiographic variables as well as biomarkers with diastolic remodeling. Results: Older age, female gender, hypertension, multi vessel disease, higher glucose and higher peak CK were independent predictors of higher E/e' at 4 months in a multivariable model (R Conclusions: Our data supports the hypothesis that female gender, multivessel coronary artery disease, and microvascular damage are important predictors of adverse diastolic remodeling after STEMI. In addition, our data suggests that older age and hypertension prior to STEMI may have contributed to worse pre-existing diastolic function. Trial registration: NIH, NCT01217307. Prospectively registered on October 8th 2010, https://clinicaltrials.gov/ct2/show/NCT01217307 .
MeSH term(s)	Humans ; Female ; ST Elevation Myocardial Infarction ; Echocardiography ; Myocardium ; Hypertension ; Glucose ; Percutaneous Coronary Intervention
Chemical Substances	Glucose (IY9XDZ35W2)
Language	English
Publishing date	2023-01-23
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2059859-2
ISSN	1471-2261 ; 1471-2261
ISSN (online)	1471-2261
ISSN	1471-2261
DOI	10.1186/s12872-023-03064-7
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Article: ANOCA patients with and without coronary vasomotor dysfunction present with limited electrocardiographic remodeling.

Schipaanboord, Diantha J M / Jansen, Tijn P J / Crooijmans, Caïa / Onland-Moret, N Charlotte / Elias-Smale, Suzette E / Dimitriu-Leen, Aukelien C / van der Harst, Pim / van de Hoef, Tim P / van Es, René / Damman, Peter / den Ruijter, Hester M

International journal of cardiology. Heart & vasculature

2024 Volume 50, Page(s) 101347

Abstract: Background: Coronary vasomotor dysfunction (CVDys) comprises coronary vasospasm (CVS) and/or coronary microvascular dysfunction (CMD) and is highly prevalent in patients with angina and non-obstructive coronary artery disease (ANOCA). Invasive coronary ... ...

Abstract	Background: Coronary vasomotor dysfunction (CVDys) comprises coronary vasospasm (CVS) and/or coronary microvascular dysfunction (CMD) and is highly prevalent in patients with angina and non-obstructive coronary artery disease (ANOCA). Invasive coronary function testing (CFT) to diagnose CVDys is becoming more common, enabling pathophysiologic research of CVDys. This study aims to explore the electrophysiological characteristics of ANOCA patients with CVDys. Methods: We collected pre-procedural 12-lead electrocardiograms of ANOCA patients with CVS (n = 35), CMD (n = 24), CVS/CMD (n = 26) and patients without CVDys (CFT-, n = 23) who participated in the NL-CFT registry and underwent CFT. Heart axis and conduction times were compared between patients with CVS, CMD or CVS/CMD and patients without CVDys. Results: Heart axis, heart rate, PQ interval and QRS duration were comparable between the groups. A small prolongation of the QT-interval corrected with Bazett (QTcB) and Fridericia (QTcF) was observed in patients with CVDys compared to patients without CVDys (CVS vs CFT-: QTcB = 422 ± 18 vs 414 ± 18 ms (p = 0.14), QTcF = 410 ± 14 vs 406 ± 12 ms (p = 0.21); CMD vs CFT-: QTcB = 426 ± 17 vs 414 ± 18 ms (p = 0.03), QTcF = 413 ± 11 vs 406 ± 12 ms (p = 0.04); CVS/CMD vs CFT-: QTcB = 424 ± 17 vs 414 ± 18 ms (p = 0.05), QTcF = 414 ± 14 vs 406 ± 12 ms (p = 0.04)). Conclusions: Pre-procedural 12-lead electrocardiograms were comparable between patients with and without CVDys undergoing CFT except for a slightly longer QTc interval in patients with CVDys compared to patients without CVDys, suggesting limited cardiac remodeling in patients with CVDys.
Language	English
Publishing date	2024-01-31
Publishing country	Ireland
Document type	Journal Article
ZDB-ID	2818464-6
ISSN	2352-9067
ISSN	2352-9067
DOI	10.1016/j.ijcha.2024.101347
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Article ; Online: Atrial fibrillation and left atrial size and function: a Mendelian randomization study.

van de Vegte, Yordi J / Siland, Joylene E / Rienstra, Michiel / van der Harst, Pim

Scientific reports

2021 Volume 11, Issue 1, Page(s) 8431

Abstract: Atrial fibrillation (AF) patients have enlarged left atria (LA), but prior studies suggested enlarged atria as both cause and consequence of AF. The aim of this study is to investigate the causal association between AF and LA size and function. In the UK ...

Abstract	Atrial fibrillation (AF) patients have enlarged left atria (LA), but prior studies suggested enlarged atria as both cause and consequence of AF. The aim of this study is to investigate the causal association between AF and LA size and function. In the UK Biobank, all individuals with contoured cardiovascular magnetic resonance data were selected. LA maximal volume (LA max), LA minimal volume (LA min), LA stroke volume and LA ejection fraction were measured and indexed to body surface area (BSA). Two-sample Mendelian randomization analyses were performed using 84 of the known genetic variants associated with AF to assess the association with all LA size and function in individuals without prevalent AF. A total of 4274 individuals (mean age 62.0 ± 7.5 years, 53.2% women) were included. Mendelian randomization analyses estimated a causal effect between genetically determined AF and BSA-indexed LA max, LA min, and LA ejection fraction, but not between AF and LA stroke volume. Leave-one-out analyses showed that the causal associations were attenuated after exclusion of rs67249485, located near PITX2 gene. Our results suggest that AF causally increases LA size and decreases LA ejection fraction. The AF risk allele of rs67249485, located near the PITX2 gene, contributes strongly to these associations.
MeSH term(s)	Aged ; Alleles ; Atrial Fibrillation/genetics ; Atrial Fibrillation/physiopathology ; Atrial Function, Left ; Female ; Heart Atria/anatomy & histology ; Heart Atria/physiopathology ; Homeodomain Proteins/genetics ; Humans ; Male ; Mendelian Randomization Analysis/methods ; Middle Aged ; Risk Factors ; Transcription Factors/genetics ; Homeobox Protein PITX2
Chemical Substances	Homeodomain Proteins ; Transcription Factors
Language	English
Publishing date	2021-04-19
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2615211-3
ISSN	2045-2322 ; 2045-2322
ISSN (online)	2045-2322
ISSN	2045-2322
DOI	10.1038/s41598-021-87859-8
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Article ; Online: Translational insights from single-cell technologies across the cardiovascular disease continuum.

van Blokland, Irene V / Groot, Hilde E / Franke, Lude H / van der Wijst, Monique G P / van der Harst, Pim

Trends in cardiovascular medicine

2021 Volume 32, Issue 3, Page(s) 127–135

Abstract: Cardiovascular disease is the leading cause of death worldwide. The societal health burden it represents can be reduced by taking preventive measures and developing more effective therapies. Reaching these goals, however, requires a better understanding ... ...

Abstract	Cardiovascular disease is the leading cause of death worldwide. The societal health burden it represents can be reduced by taking preventive measures and developing more effective therapies. Reaching these goals, however, requires a better understanding of the pathophysiological processes leading to and occurring in the diseased heart. In the last 5 years, several biological advances applying single-cell technologies have enabled researchers to study cardiovascular diseases with unprecedented resolution. This has produced many new insights into how specific cell types change their gene expression level, activation status and potential cellular interactions with the development of cardiovascular disease, but a comprehensive overview of the clinical implications of these findings is lacking. In this review, we summarize and discuss these recent advances and the promise of single-cell technologies from a translational perspective across the cardiovascular disease continuum, covering both animal and human studies, and explore the future directions of the field.
MeSH term(s)	Animals ; Cardiovascular Diseases/diagnosis ; Cardiovascular Diseases/genetics ; Cardiovascular Diseases/therapy ; Heart ; Humans ; Protein Processing, Post-Translational ; Sequence Analysis, RNA ; Single-Cell Analysis
Language	English
Publishing date	2021-03-02
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	1097434-9
ISSN	1873-2615 ; 1050-1738
ISSN (online)	1873-2615
ISSN	1050-1738
DOI	10.1016/j.tcm.2021.02.009
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Zs.A 3419: Show issues

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Article: Research within international non-governmental organisation programmes in low and middle-income countries: challenges amid opportunities.

Ishaku, Salisu Mohammed / van der Harst, Maria / Warren, Charlotte E / Franx, Arie / Kayode, Gbenga Ayodele / Grobbee, Diederick / Browne, Joyce L

BMJ global health

2021 Volume 6, Issue 8

MeSH term(s)	Developing Countries ; Humans ; Income ; Poverty
Language	English
Publishing date	2021-08-11
Publishing country	England
Document type	Journal Article
ISSN	2059-7908
ISSN	2059-7908
DOI	10.1136/bmjgh-2021-006210
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Article ; Online: PROMISE: effect of protein supplementation on fat-free mass preservation after bariatric surgery, a randomized double-blind placebo-controlled trial.

Taselaar, A E / Boes, A J / de Bruin, R W F / Kuijper, T M / Van Lancker, K / van der Harst, E / Klaassen, R A

Trials

2023 Volume 24, Issue 1, Page(s) 717

Abstract: Introduction: Protein malnutrition after bariatric surgery is a severe complication and leads to significant morbidity. Previous studies have shown that protein intake and physical activity are the most important factors in the preservation of fat-free ... ...

Abstract	Introduction: Protein malnutrition after bariatric surgery is a severe complication and leads to significant morbidity. Previous studies have shown that protein intake and physical activity are the most important factors in the preservation of fat-free mass during weight loss. Low protein intake is very common in patients undergoing bariatric surgery despite dietary counseling. Protein powder supplements might help patients to achieve the protein intake recommendations after bariatric surgery and could therefore contribute to preserve fat-free mass. This double-blind randomized placebo-controlled intervention study aims to assess the effect of a daily consumed clear protein powder shake during the first 6 months after bariatric surgery on fat-free mass loss in the first 12 months after laparoscopic Roux-en-Y gastric bypass (LRYGB). Methods and analysis: Inclusion will take place at the outpatient clinic of the bariatric expertise center for obesity of the Maasstad Hospital. Patients will be randomly assigned to either the intervention or control group before surgery. The intervention group will receive a clear protein powder shake of 200 ml containing 20 g of whey protein dissolved in water which should be taken daily during the first 6 months after LRYGB on top of their normal postoperative diet. The control group will receive an isocaloric, clear, placebo shake containing maltodextrine. Postoperative rehabilitation and physiotherapeutical guidance will be standardized and similar in both groups. Also, both groups will receive the same dietary advice from specialized dieticians. The main study parameter is the percentage of fat-free mass loss 6 months after surgery, assessed by multi-frequency bioelectrical impedance analysis (MF-BIA). Ethics and dissemination: The protocol, version 2 (February 20, 2022) has been approved by the Medical Research Ethics Committees United (MEC-U) (NL 80414.100.22). The results of this study will be submitted to peer-reviewed journals. Trial registration: ClinicalTrials.gov NCT05570474. Registered on October 5, 2022.
MeSH term(s)	Humans ; Obesity, Morbid/surgery ; Powders ; Bariatric Surgery/adverse effects ; Gastric Bypass/adverse effects ; Gastric Bypass/methods ; Dietary Supplements
Chemical Substances	Powders
Language	English
Publishing date	2023-11-09
Publishing country	England
Document type	Randomized Controlled Trial ; Journal Article
ZDB-ID	2040523-6
ISSN	1745-6215 ; 1468-6694 ; 1745-6215
ISSN (online)	1745-6215
ISSN	1468-6694 ; 1745-6215
DOI	10.1186/s13063-023-07654-w
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Article ; Online: Fetuin-A and its genetic association with cardiometabolic disease.

Al Ali, Lawien / van de Vegte, Yordi J / Said, M Abdullah / Groot, Hilde E / Hendriks, Tom / Yeung, Ming Wai / Lipsic, Erik / van der Harst, Pim

Scientific reports

2023 Volume 13, Issue 1, Page(s) 21469

Abstract: Fetuin-A acts as both an inhibitor of calcification and insulin signaling. Previous studies reported conflicting results on the association between fetuin-A and cardiometabolic diseases. We aim to provide further insights into the association between ... ...

Abstract	Fetuin-A acts as both an inhibitor of calcification and insulin signaling. Previous studies reported conflicting results on the association between fetuin-A and cardiometabolic diseases. We aim to provide further insights into the association between genetically predicted levels of fetuin-A and cardiometabolic diseases using a Mendelian randomization strategy. Genetic variants associated with fetuin-A and their effect sizes were obtained from previous genetic studies. A series of two-sample Mendelian randomization analyses in 412,444 unrelated individuals from the UK Biobank did not show evidence for an association of genetically predicted fetuin-A with any stroke, ischemic stroke, or myocardial infarction. We do find that increased levels of genetically predicted fetuin-A are associated with increased risk of type 2 diabetes (OR = 1.21, 95%CI 1.13-1.30, P = < 0.01). Furthermore, genetically predicted fetuin-A increases the risk of coronary artery disease in individuals with type 2 diabetes, but we did not find evidence for an association between genetically predicted fetuin-A and coronary artery disease in those without type 2 diabetes (P for interaction = 0.03). One SD increase in genetically predicted fetuin-A decreases risk of myocardial infarction in women, but we do not find evidence for an association between genetically predicted fetuin-A and myocardial infarction in men (P for interaction = < 0.01). Genetically predicted fetuin-A is associated with type 2 diabetes. Furthermore, type 2 diabetes status modifies the association of genetically predicted fetuin-A with coronary artery disease, indicating that fetuin-A increases risk in individuals with type 2 diabetes. Finally, higher genetically predicted fetuin-A reduces the risk of myocardial infarction in women, but we do not find evidence for an association between genetically predicted fetuin-A and myocardial infarction in men.
MeSH term(s)	Female ; Humans ; Male ; alpha-2-HS-Glycoprotein/genetics ; alpha-Fetoproteins/genetics ; Coronary Artery Disease/genetics ; Diabetes Mellitus, Type 2/genetics ; Genome-Wide Association Study ; Mendelian Randomization Analysis ; Myocardial Infarction/genetics ; Polymorphism, Single Nucleotide ; Stroke/genetics
Chemical Substances	alpha-2-HS-Glycoprotein ; alpha-Fetoproteins ; AHSG protein, human
Language	English
Publishing date	2023-12-06
Publishing country	England
Document type	Journal Article
ZDB-ID	2615211-3
ISSN	2045-2322 ; 2045-2322
ISSN (online)	2045-2322
ISSN	2045-2322
DOI	10.1038/s41598-023-48600-9
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