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Article ; Online: Correction to: Public Reason in a Pandemic: John Rawls on Truth in the Age of COVID-19.

Warner, Calvin H

2022 Volume 50, Issue 3, Page(s) 1515

Abstract: This corrects the article DOI: 10.1007/s11406-021-00459-8.]. ...

Abstract	[This corrects the article DOI: 10.1007/s11406-021-00459-8.].
Language	English
Publishing date	2022-03-02
Publishing country	Netherlands
Document type	Published Erratum
ZDB-ID	2244570-5
ISSN	1574-9274 ; 0048-3893
ISSN (online)	1574-9274
ISSN	0048-3893
DOI	10.1007/s11406-022-00491-2
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Article ; Online: Public Reason in a Pandemic: John Rawls on Truth in the Age of COVID-19.

Warner, Calvin H

Philosophia (Ramat-Gan, Israel)

2022 Volume 50, Issue 3, Page(s) 1503–1513

Abstract: In "Justice as Fairness: Political not Metaphysical," John Rawls suggests an approach to a public conception of justice that eschews any dependence on metaphysical conceptions of justice in favor of a political conception of justice. This means that if ... ...

Abstract	In "Justice as Fairness: Political not Metaphysical," John Rawls suggests an approach to a public conception of justice that eschews any dependence on metaphysical conceptions of justice in favor of a political conception of justice. This means that if there is a metaphysical conception of justice that actually obtains, then Rawls' theory would not (and could not) be sensitive to it. Rawls himself admitted in Political Liberalism that "the political conception does without the truth." Similarly, in Law of Peoples, Rawls endorses a political conception of justice to govern the society of peoples that is not concerned with truth, but instead concerned with being sufficiently neutral so as to avoid conflict with any reasonable comprehensive doctrines. The odd result is that this neutrality excludes any conception of truth at all. Therefore, in times of crisis that demand incisive decision making based on scientific, economic or moral considerations, public reason will stall because it can contain no coherent conception of truth.
Language	English
Publishing date	2022-01-31
Publishing country	Netherlands
Document type	Journal Article
ZDB-ID	2244570-5
ISSN	1574-9274 ; 0048-3893
ISSN (online)	1574-9274
ISSN	0048-3893
DOI	10.1007/s11406-021-00459-8
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Article ; Online: Summary for Policymakers

Arneth, A. / Barbosa, H. / Benton, T. / Calvin, K. / Calvo, E. / Connors, S. / Cowie, A. / Davin, E. / Denton, F. / van Diemen, R. / Driouech, F. / Elbehri, A. / Evans, J. / Ferrat, M. / Harold, J. / Haughey, E. / Herrero, M. / House, J. / Howden, M. /

Hurlbert, M. / Jia, G. / Johansen, T.G. / Krishnaswamy, J. / Kurz, W. / Lennard, C. / Myeong, S. / Mahmoud, N. / Masson-Delmotte, V. / Mbow, C. / McElwee, P. / Mirzabaev, A. / Morelli, A. / Moufouma, W. / Nedjraoui, D. / Neogi, S. / Nkem, J. / De Noblet-Ducoudre, N. / Olsson, L. / Pathak, M. / Petzold, J. / Pichs-Madruga, R. / Poloczanska, E. / Popp, A. / Pörtner, H-O. / Pereira, J.P. / Pradhan, P. / Reisinger, A. / Roberts, D.C / Rosenzweig, C. / Rounsevell, M. / Shevliakova, E. / Shukla, P.R. / Skea, J. / Slade, R. / Smith, P. / Sokona, Y. / Sonwa, D.J. / Soussana, J-F. / Tuniello, F. / Verchot, L.V. / Warner, K. / Weyer, N.M. / Wu, J. / Yassaa, N. / Zhai, P. / Zommers, Z.

2021

Keywords	climate change ; land management ; degradation
Language	English
Publishing date	2021-03-08T08:47:50Z
Publishing country	fr
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: Technical Summary

Shukla, P.R. / Skea, J. / Slade, R. / van Diemen, R. / Haughey, E. / Malley, J. / Pathak, M. / Pereira, J.P. / Agus, F. / Arneth, A. / Artaxo Netto, P.E. / Barbosa, H. / Barioni, L.G. / Benton, T. / Bhadwal, S. / Calvin, K. / Calvo, E. / Campbell, D. / Cherubini, F. /

Connors, S. / Cowie, A. / Davin, E. / Delusca, K. / Denton, F. / Elbehri, A. / Erb, K. / Evans, J. / Flores-Renteria, D. / Garcia-Oliva, F. / Grassi, G. / Hermans, K. / Herrero, M. / Houghton, R. / House, J. / Howden, M. / Hurlbert, M. / Hussein, I.A.G. / Iqbal, M.M. / Jia, G. / Jobbagy, E. / Johnson, F.X. / Kimutai, J. / Kitajima, K. / Knowles, T. / Korotkov, V. / Krishnapillai, M.V. / Krishnaswamy, J. / Kurz, W. / Le Hoang, A. / Lennard, C. / Li, D. / Liwenga, E. / Lwasa, S. / Mahmoud, N. / Masson-Delmotte, V. / Mbow, C. / McElwee, P. / Mena, C.F. / Meza, F. / Mirzabaev, A. / Morton, J. / Moufouma, W. / Myeong, S. / Nedjraoui, D. / Nkem, J. / Nkonya, E. / De Noblet-Ducoudre, N. / Olsson, L. / Osman-Elasha, B. / Petzold, J. / Pichs-Madruga, R. / Poloczanska, E. / Popp, A. / Pörtner, H-O. / Pradhan, P. / Rahimi, M. / Reisinger, A. / Rivera-Ferre, M.G. / Roberts, D.C. / Rosenzweig, C. / Rounsevell, M. / Saigusa, N. / Sapkota, T. / Shevliakova, E. / Sirin, A. / Smith, P. / Sokona, Y. / Sonwa, D.J. / Sousasana, J-F. / Spence, A. / Stringer, L. / Sukumar, R. / Taboada, M.A. / Tena, F. / Tubiello, F. / Türkes, M. / Valentini, R. / Vazquez Montenegro, R.J. / Verchot, L.V. / Viner, D. / Warner, K. / Weltz, M. / Weyer, N.M. / Wreford, A. / Wu, J. / Yassa, N. / Zakieldeen, S. / Zhai, P. / Zommers, Z.

2021

Keywords	climate change ; land management ; degradation
Language	English
Publishing date	2021-03-08T09:02:30Z
Publishing country	fr
Document type	Article ; Online
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Article ; Online: Chronic + binge alcohol exposure promotes inflammation and alters airway mechanics in the lung.

Poole, Lauren G / Beier, Juliane I / Torres-Gonzales, Edilson / Schlueter, Connie F / Hudson, Shanice V / Artis, Amanda / Warner, Nikole L / Nguyen-Ho, Calvin T / Dolin, Christine E / Ritzenthaler, Jeffrey D / Hoyle, Gary W / Roman, Jesse / Arteel, Gavin E

Alcohol (Fayetteville, N.Y.)

2018 Volume 80, Page(s) 53–63

Abstract: ... in the bronchoalveolar lavage fluid 24 h post-binge. This inflammatory cell recruitment was associated with airway hyper ...

Abstract	Introduction: Alcohol use disorders are major risk factors for the development of and susceptibility to acute respiratory distress syndrome. Although these risks of alcohol consumption on the lung are well described, mechanisms by which alcohol abuse promotes acute lung injury are poorly understood. These gaps in our understanding are due, at least in part, to limitations of animal models to recapitulate human alcohol consumption. Recently, a new model of chronic plus binge alcohol exposure was developed that is hypothesized to better model drinking patterns of individuals with alcohol use disorders. Specifically, this paradigm models chronic consumption coupled with periodic bouts of heavy drinking. The impacts of this alcohol-exposure regimen on the lung are uncharacterized. Therefore, the goal of this study was to examine lung injury and inflammation in a well-characterized experimental model of chronic + binge alcohol exposure. Methods: 10-week-old male C57Bl6/J mice were administered ethanol-containing (or isocaloric control) liquid diet for 10 days, followed by a single ethanol gavage (5 g/kg). Lung inflammation and pulmonary function were assessed. Results: Ten days of ethanol-containing liquid diet alone (chronic) did not detectably affect any variables measured. However, ethanol diet plus gavage (chronic + binge) caused neutrophils to accumulate in the lung tissue and in the bronchoalveolar lavage fluid 24 h post-binge. This inflammatory cell recruitment was associated with airway hyper-responsiveness to inhaled methacholine, as indicated by elevated resistance, Newtonian resistance, and respiratory resistance. Conclusions: Taken together, the novel findings reveal that ethanol alone, absent of any secondary inflammatory insult, is sufficient to produce inflammation in the lung. Although these changes were relatively mild, they were associated with functional changes in the central airways. This animal model may be useful in the future for identifying mechanisms by which alcohol abuse sensitizes at-risk individuals to lung injury.
MeSH term(s)	Alcoholism/complications ; Alcoholism/pathology ; Alcoholism/physiopathology ; Animals ; Binge Drinking/complications ; Binge Drinking/pathology ; Binge Drinking/physiopathology ; Disease Models, Animal ; Lung/drug effects ; Lung/pathology ; Lung/physiopathology ; Male ; Mice ; Mice, Inbred C57BL ; Pneumonia/chemically induced ; Pneumonia/pathology ; Pneumonia/physiopathology ; Reverse Transcriptase Polymerase Chain Reaction
Language	English
Publishing date	2018-11-14
Publishing country	United States
Document type	Journal Article
ZDB-ID	605912-0
ISSN	1873-6823 ; 0741-8329
ISSN (online)	1873-6823
ISSN	0741-8329
DOI	10.1016/j.alcohol.2018.10.008
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Article: Effects of metalloporphyrin catalytic antioxidants in experimental brain ischemia.

Sheng, Huaxin / Enghild, Jan J / Bowler, Russell / Patel, Manisha / Batinić-Haberle, Ines / Calvi, Carla L / Day, Brian J / Pearlstein, Robert D / Crapo, James D / Warner, David S

Free radical biology & medicine

2002 Volume 33, Issue 7, Page(s) 947–961

Abstract: ... 10150, given 6 h after 90 min MCAO, reduced total infarct size by 43% without temperature effects. Brain ... AEOL 10150 elimination t(1/2) was 10 h. In the mouse, intravenous AEOL 10150 infusion post-MCAO reduced ... changes caused by ischemia. In primary mixed neuronal/glial cultures exposed to 2 h of O(2)/glucose ...

Abstract	Reactive oxygen species play a role in the response of brain to ischemia. The effects of metalloporphyrin catalytic antioxidants (AEOL 10113 and AEOL 10150) were examined after murine middle cerebral artery occlusion (MCAO). Ninety minutes after reperfusion from 90 min MCAO in the rat, AEOL 10113, AEOL 10150, or vehicle were given intracerebroventricularly. AEOL 10113 and AEOL 10150 similarly reduced infarct size (35%) and neurologic deficit. AEOL 10113 caused behavioral side effects at twice the neuroprotective dose while AEOL 10150 required a 15-fold increase from the neuroprotective dose to cause behavioral changes. AEOL 10150, given 6 h after 90 min MCAO, reduced total infarct size by 43% without temperature effects. Brain AEOL 10150 elimination t(1/2) was 10 h. In the mouse, intravenous AEOL 10150 infusion post-MCAO reduced both infarct size (25%) and neurologic deficit. Brain AEOL 10150 uptake, greater in the ischemic hemisphere, was dose- and time-dependent. AEOL 10150 had direct effects on proteomic events and ameliorated changes caused by ischemia. In primary mixed neuronal/glial cultures exposed to 2 h of O(2)/glucose deprivation, AEOL 10150 reduced lactate dehydrogenase release dose-dependently and selectively preserved aconitase activity in concentrations consistent with neuroprotection in vivo. AEOL 10150 is an effective neuroprotective compound offering a wide therapeutic window with a large margin of safety against adverse behavioral side effects.
MeSH term(s)	Animals ; Antioxidants/pharmacology ; Brain/drug effects ; Brain/pathology ; Brain Ischemia/drug therapy ; Brain Ischemia/physiopathology ; Brain Ischemia/prevention & control ; Carotid Artery, Internal ; Cerebral Infarction/pathology ; Cerebral Infarction/prevention & control ; Disease Models, Animal ; Hemodynamics/drug effects ; Injections, Intraventricular ; Manganese/pharmacology ; Metalloporphyrins/administration & dosage ; Metalloporphyrins/pharmacology ; Rats
Chemical Substances	Antioxidants ; Metalloporphyrins ; manganese (III) meso-tetrakis(N-ethylpyridinium-2-yl)porphyrin ; Manganese (42Z2K6ZL8P)
Language	English
Publishing date	2002-05-20
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, P.H.S.
ZDB-ID	807032-5
ISSN	1873-4596 ; 0891-5849
ISSN (online)	1873-4596
ISSN	0891-5849
DOI	10.1016/s0891-5849(02)00979-6
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Article ; Online: Genome sequencing analysis identifies new loci associated with Lewy body dementia and provides insights into its genetic architecture.

Chia, Ruth / Sabir, Marya S / Bandres-Ciga, Sara / Saez-Atienzar, Sara / Reynolds, Regina H / Gustavsson, Emil / Walton, Ronald L / Ahmed, Sarah / Viollet, Coralie / Ding, Jinhui / Makarious, Mary B / Diez-Fairen, Monica / Portley, Makayla K / Shah, Zalak / Abramzon, Yevgeniya / Hernandez, Dena G / Blauwendraat, Cornelis / Stone, David J / Eicher, John /

Parkkinen, Laura / Ansorge, Olaf / Clark, Lorraine / Honig, Lawrence S / Marder, Karen / Lemstra, Afina / St George-Hyslop, Peter / Londos, Elisabet / Morgan, Kevin / Lashley, Tammaryn / Warner, Thomas T / Jaunmuktane, Zane / Galasko, Douglas / Santana, Isabel / Tienari, Pentti J / Myllykangas, Liisa / Oinas, Minna / Cairns, Nigel J / Morris, John C / Halliday, Glenda M / Van Deerlin, Vivianna M / Trojanowski, John Q / Grassano, Maurizio / Calvo, Andrea / Mora, Gabriele / Canosa, Antonio / Floris, Gianluca / Bohannan, Ryan C / Brett, Francesca / Gan-Or, Ziv / Geiger, Joshua T / Moore, Anni / May, Patrick / Krüger, Rejko / Goldstein, David S / Lopez, Grisel / Tayebi, Nahid / Sidransky, Ellen / Norcliffe-Kaufmann, Lucy / Palma, Jose-Alberto / Kaufmann, Horacio / Shakkottai, Vikram G / Perkins, Matthew / Newell, Kathy L / Gasser, Thomas / Schulte, Claudia / Landi, Francesco / Salvi, Erika / Cusi, Daniele / Masliah, Eliezer / Kim, Ronald C / Caraway, Chad A / Monuki, Edwin S / Brunetti, Maura / Dawson, Ted M / Rosenthal, Liana S / Albert, Marilyn S / Pletnikova, Olga / Troncoso, Juan C / Flanagan, Margaret E / Mao, Qinwen / Bigio, Eileen H / Rodríguez-Rodríguez, Eloy / Infante, Jon / Lage, Carmen / González-Aramburu, Isabel / Sanchez-Juan, Pascual / Ghetti, Bernardino / Keith, Julia / Black, Sandra E / Masellis, Mario / Rogaeva, Ekaterina / Duyckaerts, Charles / Brice, Alexis / Lesage, Suzanne / Xiromerisiou, Georgia / Barrett, Matthew J / Tilley, Bension S / Gentleman, Steve / Logroscino, Giancarlo / Serrano, Geidy E / Beach, Thomas G / McKeith, Ian G / Thomas, Alan J / Attems, Johannes / Morris, Christopher M / Palmer, Laura / Love, Seth / Troakes, Claire / Al-Sarraj, Safa / Hodges, Angela K / Aarsland, Dag / Klein, Gregory / Kaiser, Scott M / Woltjer, Randy / Pastor, Pau / Bekris, Lynn M / Leverenz, James B / Besser, Lilah M / Kuzma, Amanda / Renton, Alan E / Goate, Alison / Bennett, David A / Scherzer, Clemens R / Morris, Huw R / Ferrari, Raffaele / Albani, Diego / Pickering-Brown, Stuart / Faber, Kelley / Kukull, Walter A / Morenas-Rodriguez, Estrella / Lleó, Alberto / Fortea, Juan / Alcolea, Daniel / Clarimon, Jordi / Nalls, Mike A / Ferrucci, Luigi / Resnick, Susan M / Tanaka, Toshiko / Foroud, Tatiana M / Graff-Radford, Neill R / Wszolek, Zbigniew K / Ferman, Tanis / Boeve, Bradley F / Hardy, John A / Topol, Eric J / Torkamani, Ali / Singleton, Andrew B / Ryten, Mina / Dickson, Dennis W / Chiò, Adriano / Ross, Owen A / Gibbs, J Raphael / Dalgard, Clifton L / Traynor, Bryan J / Scholz, Sonja W

Nature genetics

2021 Volume 53, Issue 3, Page(s) 294–303

Abstract: The genetic basis of Lewy body dementia (LBD) is not well understood. Here, we performed whole-genome sequencing in large cohorts of LBD cases and neurologically healthy controls to study the genetic architecture of this understudied form of dementia, ... ...

Abstract	The genetic basis of Lewy body dementia (LBD) is not well understood. Here, we performed whole-genome sequencing in large cohorts of LBD cases and neurologically healthy controls to study the genetic architecture of this understudied form of dementia, and to generate a resource for the scientific community. Genome-wide association analysis identified five independent risk loci, whereas genome-wide gene-aggregation tests implicated mutations in the gene GBA. Genetic risk scores demonstrate that LBD shares risk profiles and pathways with Alzheimer's disease and Parkinson's disease, providing a deeper molecular understanding of the complex genetic architecture of this age-related neurodegenerative condition.
MeSH term(s)	Adaptor Proteins, Signal Transducing/genetics ; Alzheimer Disease/genetics ; Case-Control Studies ; Gene Expression Profiling ; Genetic Predisposition to Disease ; Genome, Human ; Genome-Wide Association Study ; Glucosylceramidase/genetics ; Humans ; Lewy Body Disease/genetics ; Nuclear Proteins/genetics ; Parkinson Disease/genetics ; Polymorphism, Single Nucleotide ; Tumor Suppressor Proteins/genetics ; alpha-Synuclein/genetics
Chemical Substances	Adaptor Proteins, Signal Transducing ; BIN1 protein, human ; Nuclear Proteins ; SNCA protein, human ; Tumor Suppressor Proteins ; alpha-Synuclein ; GBA protein, human (EC 3.2.1.45) ; Glucosylceramidase (EC 3.2.1.45)
Language	English
Publishing date	2021-02-15
Publishing country	United States
Document type	Journal Article ; Multicenter Study ; Research Support, N.I.H., Intramural
ZDB-ID	1108734-1
ISSN	1546-1718 ; 1061-4036
ISSN (online)	1546-1718
ISSN	1061-4036
DOI	10.1038/s41588-021-00785-3
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Article ; Online: Neuroprotection from delayed postischemic administration of a metalloporphyrin catalytic antioxidant.

Mackensen, G B / Patel, M / Sheng, H / Calvi, C L / Batinic-Haberle, I / Day, B J / Liang, L P / Fridovich, I / Crapo, J D / Pearlstein, R D / Warner, D S

The Journal of neuroscience : the official journal of the Society for Neuroscience

2001 Volume 21, Issue 13, Page(s) 4582–4592

Abstract: Reactive oxygen species contribute to ischemic brain injury. This study examined whether the porphyrin catalytic antioxidant manganese (III) meso-tetrakis (N-ethylpyridinium-2-yl)porphyrin (MnTE-2-PyP(5+)) reduces oxidative stress and improves outcome ... ...

Abstract	Reactive oxygen species contribute to ischemic brain injury. This study examined whether the porphyrin catalytic antioxidant manganese (III) meso-tetrakis (N-ethylpyridinium-2-yl)porphyrin (MnTE-2-PyP(5+)) reduces oxidative stress and improves outcome from experimental cerebral ischemia. Rats that were subjected to 90 min focal ischemia and 7 d recovery were given MnTE-2-PyP(5+) (or vehicle) intracerebroventricularly 60 min before ischemia, or 5 or 90 min or 6 or 12 hr after reperfusion. Biomarkers of brain oxidative stress were measured at 4 hr after postischemic treatment (5 min or 6 hr). MnTE-2-PyP(5+), given 60 min before ischemia, improved neurologic scores and reduced total infarct size by 70%. MnTE-2-PyP(5+), given 5 or 90 min after reperfusion, reduced infarct size by 70-77% and had no effect on temperature. MnTE-2-PyP(5+) treatment 6 hr after ischemia reduced total infarct volume by 54% (vehicle, 131 +/- 60 mm(3); MnTE-2-PyP(5+), 300 ng, 60 +/- 68 mm(3)). Protection was observed in both cortex and caudoputamen, and neurologic scores were improved. No MnTE-2-PyP(5+) effect was observed if it was given 12 hr after ischemia. MnTE-2-PyP(5+) prevented mitochondrial aconitase inactivation and reduced 8-hydroxy-2'-deoxyguanosine formation when it was given 5 min or 6 hr after ischemia. In mice, MnTE-2-PyP(5+) reduced infarct size and improved neurologic scores when it was given intravenously 5 min after ischemia. There was no effect of 150 or 300 ng of MnTE-2-PyP(5+) pretreatment on selective neuronal necrosis resulting from 10 min forebrain ischemia and 5 d recovery in rats. Administration of a metalloporphyrin catalytic antioxidant had marked neuroprotective effects against focal ischemic insults when it was given up to 6 hr after ischemia. This was associated with decreased postischemic superoxide-mediated oxidative stress.
MeSH term(s)	Aconitate Hydratase/metabolism ; Animals ; Antioxidants/administration & dosage ; Antioxidants/chemistry ; Brain/blood supply ; Brain/drug effects ; Brain/enzymology ; Brain/pathology ; Brain Ischemia/drug therapy ; Brain Ischemia/etiology ; Catalysis ; Cerebral Infarction/etiology ; Cerebral Infarction/prevention & control ; DNA/metabolism ; Disease Models, Animal ; Dose-Response Relationship, Drug ; Drug Administration Schedule ; Fumarate Hydratase/metabolism ; Infarction, Middle Cerebral Artery/complications ; Injections, Intravenous ; Injections, Intraventricular ; Male ; Metalloporphyrins/administration & dosage ; Metalloporphyrins/chemistry ; Mice ; Mice, Inbred C57BL ; Mitochondria/enzymology ; Necrosis ; Neuroprotective Agents/administration & dosage ; Neuroprotective Agents/chemistry ; Oxidative Stress/drug effects ; Rats ; Rats, Wistar ; Treatment Outcome
Chemical Substances	Antioxidants ; Metalloporphyrins ; Neuroprotective Agents ; manganese (III) meso-tetrakis(N-ethylpyridinium-2-yl)porphyrin ; DNA (9007-49-2) ; Fumarate Hydratase (EC 4.2.1.2) ; Aconitate Hydratase (EC 4.2.1.3)
Language	English
Publishing date	2001-07-01
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, P.H.S.
ZDB-ID	604637-x
ISSN	1529-2401 ; 0270-6474
ISSN (online)	1529-2401
ISSN	0270-6474
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Article: Mechanism of suppression in Drosophila. H. Enzymatic discrimination of wild-type and suppressor tyrosine transfer RNA.

Jacobson, K B / Calviño, J F / Murphy, J B / Warner, C K

Journal of molecular biology

1975 Volume 93, Issue 1, Page(s) 89–97

MeSH term(s)	Amino Acyl-tRNA Synthetases/metabolism ; Animals ; Chromatography ; Chromatography, Gel ; Drosophila melanogaster/enzymology ; Mutation ; RNA, Transfer/analysis ; RNA, Transfer/metabolism ; Structure-Activity Relationship ; Suppression, Genetic ; Tyrosine ; Tyrosine-tRNA Ligase/analysis ; Tyrosine-tRNA Ligase/metabolism
Chemical Substances	Tyrosine (42HK56048U) ; RNA, Transfer (9014-25-9) ; Amino Acyl-tRNA Synthetases (EC 6.1.1.-) ; Tyrosine-tRNA Ligase (EC 6.1.1.1)
Language	English
Publishing date	1975-03-25
Publishing country	England
Document type	Journal Article
ZDB-ID	80229-3
ISSN	1089-8638 ; 0022-2836
ISSN (online)	1089-8638
ISSN	0022-2836
DOI	10.1016/0022-2836(75)90362-9
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Article: Scintillation light detection in the 6-m drift-length ProtoDUNE Dual Phase liquid argon TPC.

Abud, A Abed / Abi, B / Acciarri, R / Acero, M A / Adames, M R / Adamov, G / Adamowski, M / Adams, D / Adinolfi, M / Aduszkiewicz, A / Aguilar, J / Ahmad, Z / Ahmed, J / Aimard, B / Ali-Mohammadzadeh, B / Alion, T / Allison, K / Monsalve, S Alonso / AlRashed, M /

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The European physical journal. C, Particles and fields

2022 Volume 82, Issue 7, Page(s) 618

Abstract: DUNE is a dual-site experiment for long-baseline neutrino oscillation studies, neutrino astrophysics and nucleon decay searches. ProtoDUNE Dual Phase (DP) is a ... ...

Abstract	DUNE is a dual-site experiment for long-baseline neutrino oscillation studies, neutrino astrophysics and nucleon decay searches. ProtoDUNE Dual Phase (DP) is a 6
Language	English
Publishing date	2022-07-16
Publishing country	France
Document type	Journal Article
ZDB-ID	1459069-4
ISSN	1434-6052 ; 1434-6044
ISSN (online)	1434-6052
ISSN	1434-6044
DOI	10.1140/epjc/s10052-022-10549-w
Database	MEDical Literature Analysis and Retrieval System OnLINE

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