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Article: Right cranial lung lobe torsion after a diaphragmatic rupture repair in a Jack Russell terrier.

Terzo, E / Pink, J / Puggioni, A / Shiel, R / Andreoni, V / McAllister, H

2008 Volume 61, Issue 3, Page(s) 170–174

Abstract: A seven-year-old male Jack Russell terrier was presented with a history of coughing, generalised ...

Abstract	A seven-year-old male Jack Russell terrier was presented with a history of coughing, generalised weakness and lethargy 10 days after an abdominal coeliotomy to repair a large diaphragmatic rupture. Thoracic radiographs demonstrated a soft tissue mass in the midcaudal right thoracic cavity. Ultrasonographic studies, bronchoscopy and subsequent exploratory thoracotomy confirmed a diagnosis of a right cranial lung lobe torsion (LLT), with an anomalous caudodorsal displacement of the affected lobe. LLT should be considered as a differential diagnosis for respiratory tract disease following diaphragmatic rupture repair.
Language	English
Publishing date	2008-03-01
Publishing country	Ireland
Document type	Journal Article
ZDB-ID	2571182-9
ISSN	2046-0481 ; 0368-0762
ISSN (online)	2046-0481
ISSN	0368-0762
DOI	10.1186/2046-0481-61-3-170
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Article ; Online: Right cranial lung lobe torsion after a diaphragmatic rupture repair in a Jack Russell terrier

Terzo E / Pink J / Puggioni A / Shiel R / Andreoni V / McAllister H

Irish Veterinary Journal, Vol 61, Iss 3, Pp 170-

2008 Volume 174

Abstract: Abstract A seven-year-old male Jack Russell terrier was presented with a history of coughing ...

Abstract	Abstract A seven-year-old male Jack Russell terrier was presented with a history of coughing, generalised weakness and lethargy 10 days after an abdominal coeliotomy to repair a large diaphragmatic rupture. Thoracic radiographs demonstrated a soft tissue mass in the midcaudal right thoracic cavity. Ultrasonographic studies, bronchoscopy and subsequent exploratory thoracotomy confirmed a diagnosis of a right cranial lung lobe torsion (LLT), with an anomalous caudodorsal displacement of the affected lobe. LLT should be considered as a differential diagnosis for respiratory tract disease following diaphragmatic rupture repair.
Keywords	diaphragmatic rupture ; dog ; lung lobe torsion ; Veterinary medicine ; SF600-1100
Language	English
Publishing date	2008-03-01T00:00:00Z
Publisher	BMC
Document type	Article ; Online
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Article: A Life Less than Ordinary: The Schooner

Pink, Jack / Whitewright, Julian

Historical archaeology

2022 Volume 56, Issue 1, Page(s) 3–15

Abstract: The East Winner Bank Shipwreck takes its name from the southern sandbank on Hayling Island near Portsmouth, UK. Examination of the wreck indicates a 19th-century carvel-built vessel. The sandbank is an active environment, meaning the wreck is rarely ... ...

Abstract	The East Winner Bank Shipwreck takes its name from the southern sandbank on Hayling Island near Portsmouth, UK. Examination of the wreck indicates a 19th-century carvel-built vessel. The sandbank is an active environment, meaning the wreck is rarely exposed to its full extent. Discussed here is work completed on the site before and during the social-distancing restrictions imposed by COVID-19. Documentary sources and previous detailed surveys suggest a possible identification for the wreck. The site appears to be an example of an everyday 19th-century coastal trading vessel, rarely explored archaeologically in the UK, with potential to contribute to discussions of the maritime technologies and maritime cultural landscape of regular folk. The investigation represents an excellent example of combining historical and archaeological data sets to further the interpretation of both sources, revealing details about the ship and its lasting impact on this stretch of coastline.
Language	English
Publishing date	2022-01-03
Publishing country	United States
Document type	Journal Article
ZDB-ID	2050471-8
ISSN	2328-1103 ; 0440-9213
ISSN (online)	2328-1103
ISSN	0440-9213
DOI	10.1007/s41636-021-00322-3
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Article ; Online: Interactions Between Neuropsychiatric Symptoms and Alzheimer's Disease Neuroimaging Biomarkers in Predicting Longitudinal Cognitive Decline.

Pink, Anna / Krell-Roesch, Janina / Syrjanen, Jeremy A / Christenson, Luke R / Lowe, Val J / Vemuri, Prashanthi / Fields, Julie A / Stokin, Gorazd B / Kremers, Walter K / Scharf, Eugene L / Jack, Clifford R / Knopman, David S / Petersen, Ronald C / Vassilaki, Maria / Geda, Yonas E

Psychiatric research and clinical practice

2023 Volume 5, Issue 1, Page(s) 4–15

Abstract: Objective: To examine interactions between Neuropsychiatric symptoms (NPS) with Pittsburgh Compound B (PiB) and fluorodeoxyglucose positron emission tomography (FDG-PET) in predicting cognitive trajectories.: Methods: We conducted a longitudinal ... ...

Abstract	Objective: To examine interactions between Neuropsychiatric symptoms (NPS) with Pittsburgh Compound B (PiB) and fluorodeoxyglucose positron emission tomography (FDG-PET) in predicting cognitive trajectories. Methods: We conducted a longitudinal study in the setting of the population-based Mayo Clinic Study of Aging in Olmsted County, MN, involving 1581 cognitively unimpaired (CU) persons aged ≥50 years (median age 71.83 years, 54.0% males, 27.5% APOE ɛ4 carriers). NPS at baseline were assessed using the Neuropsychiatric Inventory Questionnaire (NPI-Q). Brain glucose hypometabolism was defined as a SUVR ≤ 1.47 (measured by FDG-PET) in regions typically affected in Alzheimer's disease. Abnormal cortical amyloid deposition was measured using PiB-PET (SUVR ≥ 1.48). Neuropsychological testing was done approximately every 15 months, and we calculated global and domain-specific (memory, language, attention, and visuospatial skills) cognitive z-scores. We ran linear mixed-effect models to examine the associations and interactions between NPS at baseline and z-scored PiB- and FDG-PET SUVRs in predicting cognitive z-scores adjusted for age, sex, education, and previous cognitive testing. Results: Individuals at the average PiB and without NPS at baseline declined over time on cognitive z-scores. Those with increased PiB at baseline declined faster (two-way interaction), and those with increased PiB and NPS declined even faster (three-way interaction). We observed interactions between time, increased PiB and anxiety or irritability indicating accelerated decline on global z-scores, and between time, increased PiB and several NPS (e.g., agitation) showing faster domain-specific decline, especially on the attention domain. Conclusions: NPS and increased brain amyloid deposition synergistically interact in accelerating global and domain-specific cognitive decline among CU persons at baseline.
Language	English
Publishing date	2023-01-20
Publishing country	United States
Document type	Journal Article
ISSN	2575-5609
ISSN (online)	2575-5609
DOI	10.1176/appi.prcp.20220036
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Article ; Online: Novel colchicine derivative CR42-24 demonstrates potent anti-tumor activity in urothelial carcinoma.

Bell, Clayton J / Potts, Kyle G / Hitt, Mary M / Pink, Desmond / Tuszynski, Jack A / Lewis, John D

Cancer letters

2021 Volume 526, Page(s) 168–179

Abstract: Bladder cancers, and specifically urothelial carcinoma, have few effective treatment options, and tumors typically develop resistance against standard of care chemotherapies leading to significant mortality. The development of alternative therapies with ... ...

Abstract	Bladder cancers, and specifically urothelial carcinoma, have few effective treatment options, and tumors typically develop resistance against standard of care chemotherapies leading to significant mortality. The development of alternative therapies with increased selectivity and improved tolerability would significantly impact this patient population. Here, we investigate a novel colchicine derivative, CR42-24, with increased selectivity for the βIII tubulin subtype as a treatment for urothelial carcinoma. βIII tubulin is a promising target due to its low expression in healthy tissues and its clinical association with poor prognosis. This study demonstrated that CR42-24 is selectively cytotoxic to several cancer cell lines at low nanomolar IC
MeSH term(s)	Animals ; Carcinoma, Transitional Cell/drug therapy ; Cell Line, Tumor ; Colchicine/pharmacology ; Disease Models, Animal ; HeLa Cells ; Humans ; Male ; Mice ; Mice, Inbred NOD ; Mice, SCID ; Tubulin Modulators/pharmacology ; Urinary Bladder Neoplasms/drug therapy ; Xenograft Model Antitumor Assays
Chemical Substances	Tubulin Modulators ; Colchicine (SML2Y3J35T)
Language	English
Publishing date	2021-11-25
Publishing country	Ireland
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	195674-7
ISSN	1872-7980 ; 0304-3835
ISSN (online)	1872-7980
ISSN	0304-3835
DOI	10.1016/j.canlet.2021.11.028
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Zs.A 1177: Show issues

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Article ; Online: A longitudinal investigation of Aβ, anxiety, depression, and mild cognitive impairment.

Pink, Anna / Krell-Roesch, Janina / Syrjanen, Jeremy A / Vassilaki, Maria / Lowe, Val J / Vemuri, Prashanthi / Stokin, Gorazd B / Christianson, Teresa J / Kremers, Walter K / Jack, Clifford R / Knopman, David S / Petersen, Ronald C / Geda, Yonas E

Alzheimer's & dementia : the journal of the Alzheimer's Association

2021 Volume 18, Issue 10, Page(s) 1824–1831

Abstract: Introduction: We investigated the longitudinal relationship between cortical amyloid deposition, anxiety, and depression and the risk of incident mild cognitive impairment (MCI).: Methods: We followed 1440 community-dwelling, cognitively unimpaired ... ...

Abstract	Introduction: We investigated the longitudinal relationship between cortical amyloid deposition, anxiety, and depression and the risk of incident mild cognitive impairment (MCI). Methods: We followed 1440 community-dwelling, cognitively unimpaired individuals aged ≥ 50 years for a median of 5.5 years. Clinical anxiety and depression were assessed using Beck Anxiety and Depression Inventories (BAI, BDI-II). Cortical amyloid beta (Aβ) was measured by Pittsburgh compound B positron emission tomography (PiB-PET) and elevated deposition (PiB+) was defined as standardized uptake value ratio ≥ 1.48. We calculated Cox proportional hazards models with age as the time scale, adjusted for sex, education, and medical comorbidity. Results: Cortical Aβ deposition (PiB+) independent of anxiety (BAI ≥ 10) or depression (BDI-II ≥ 13) increased the risk of MCI. There was a significant additive interaction between PiB+ and anxiety (joint effect hazard ratio 6.77; 95% confidence interval 3.58-12.79; P = .031) that is, being PiB+ and having anxiety further amplified the risk of MCI. Discussion: Anxiety modified the association between PiB+ and incident MCI.
MeSH term(s)	Humans ; Alzheimer Disease/psychology ; Amyloid beta-Peptides/metabolism ; Aniline Compounds ; Anxiety/epidemiology ; Anxiety/psychology ; Brain/metabolism ; Cognitive Dysfunction/diagnostic imaging ; Cognitive Dysfunction/epidemiology ; Cognitive Dysfunction/psychology ; Depression/epidemiology ; Depression/psychology ; Neuropsychological Tests ; Positron-Emission Tomography/methods
Chemical Substances	Amyloid beta-Peptides ; Aniline Compounds
Language	English
Publishing date	2021-12-08
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	2211627-8
ISSN	1552-5279 ; 1552-5260
ISSN (online)	1552-5279
ISSN	1552-5260
DOI	10.1002/alz.12504
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Zs.A 6316: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular ab Jg. 2022: Lesesaal (EG)
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Article ; Online: A lightweight magnetically shielded room with active shielding.

Holmes, Niall / Rea, Molly / Chalmers, James / Leggett, James / Edwards, Lucy J / Nell, Paul / Pink, Stephen / Patel, Prashant / Wood, Jack / Murby, Nick / Woolger, David / Dawson, Eliot / Mariani, Christopher / Tierney, Tim M / Mellor, Stephanie / O'Neill, George C / Boto, Elena / Hill, Ryan M / Shah, Vishal /

Osborne, James / Pardington, Rosemarie / Fierlinger, Peter / Barnes, Gareth R / Glover, Paul / Brookes, Matthew J / Bowtell, Richard

Scientific reports

2022 Volume 12, Issue 1, Page(s) 13561

Abstract: Magnetically shielded rooms (MSRs) use multiple layers of materials such as MuMetal to screen external magnetic fields that would otherwise interfere with high precision magnetic field measurements such as magnetoencephalography (MEG). Optically pumped ... ...

Abstract	Magnetically shielded rooms (MSRs) use multiple layers of materials such as MuMetal to screen external magnetic fields that would otherwise interfere with high precision magnetic field measurements such as magnetoencephalography (MEG). Optically pumped magnetometers (OPMs) have enabled the development of wearable MEG systems which have the potential to provide a motion tolerant functional brain imaging system with high spatiotemporal resolution. Despite significant promise, OPMs impose stringent magnetic shielding requirements, operating around a zero magnetic field resonance within a dynamic range of ± 5 nT. MSRs developed for OPM-MEG must therefore effectively shield external sources and provide a low remnant magnetic field inside the enclosure. Existing MSRs optimised for OPM-MEG are expensive, heavy, and difficult to site. Electromagnetic coils are used to further cancel the remnant field inside the MSR enabling participant movements during OPM-MEG, but present coil systems are challenging to engineer and occupy space in the MSR limiting participant movements and negatively impacting patient experience. Here we present a lightweight MSR design (30% reduction in weight and 40-60% reduction in external dimensions compared to a standard OPM-optimised MSR) which takes significant steps towards addressing these barriers. We also designed a 'window coil' active shielding system, featuring a series of simple rectangular coils placed directly onto the walls of the MSR. By mapping the remnant magnetic field inside the MSR, and the magnetic field produced by the coils, we can identify optimal coil currents and cancel the remnant magnetic field over the central cubic metre to just \|B\|= 670 ± 160 pT. These advances reduce the cost, installation time and siting restrictions of MSRs which will be essential for the widespread deployment of OPM-MEG.
MeSH term(s)	Brain ; Functional Neuroimaging ; Humans ; Magnetic Fields ; Magnetic Resonance Imaging ; Magnetic Resonance Spectroscopy ; Magnetoencephalography/methods
Language	English
Publishing date	2022-08-09
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2615211-3
ISSN	2045-2322 ; 2045-2322
ISSN (online)	2045-2322
ISSN	2045-2322
DOI	10.1038/s41598-022-17346-1
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Article ; Online: Endothelial dysfunction contributes to severe COVID-19 in combination with dysregulated lymphocyte responses and cytokine networks.

Ruhl, Louisa / Pink, Isabell / Kühne, Jenny F / Beushausen, Kerstin / Keil, Jana / Christoph, Stella / Sauer, Andrea / Boblitz, Lennart / Schmidt, Julius / David, Sascha / Jäck, Hans-Martin / Roth, Edith / Cornberg, Markus / Schulz, Thomas F / Welte, Tobias / Höper, Marius M / Falk, Christine S

Signal transduction and targeted therapy

2021 Volume 6, Issue 1, Page(s) 418

Abstract: The systemic processes involved in the manifestation of life-threatening COVID-19 and in disease recovery are still incompletely understood, despite investigations focusing on the dysregulation of immune responses after SARS-CoV-2 infection. To define ... ...

Abstract	The systemic processes involved in the manifestation of life-threatening COVID-19 and in disease recovery are still incompletely understood, despite investigations focusing on the dysregulation of immune responses after SARS-CoV-2 infection. To define hallmarks of severe COVID-19 in acute disease (n = 58) and in disease recovery in convalescent patients (n = 28) from Hannover Medical School, we used flow cytometry and proteomics data with unsupervised clustering analyses. In our observational study, we combined analyses of immune cells and cytokine/chemokine networks with endothelial activation and injury. ICU patients displayed an altered immune signature with prolonged lymphopenia but the expansion of granulocytes and plasmablasts along with activated and terminally differentiated T and NK cells and high levels of SARS-CoV-2-specific antibodies. The core signature of seven plasma proteins revealed a highly inflammatory microenvironment in addition to endothelial injury in severe COVID-19. Changes within this signature were associated with either disease progression or recovery. In summary, our data suggest that besides a strong inflammatory response, severe COVID-19 is driven by endothelial activation and barrier disruption, whereby recovery depends on the regeneration of the endothelial integrity.
MeSH term(s)	Antibodies, Viral/blood ; Biomarkers/blood ; Blood Proteins/metabolism ; C-Reactive Protein/metabolism ; COVID-19/diagnosis ; COVID-19/immunology ; COVID-19/mortality ; COVID-19/virology ; Chemokine CXCL10/blood ; Chemokine CXCL9/blood ; Cluster Analysis ; Convalescence ; Cytokine Release Syndrome/diagnosis ; Cytokine Release Syndrome/immunology ; Cytokine Release Syndrome/mortality ; Cytokine Release Syndrome/virology ; Disease Progression ; Endothelium, Vascular/immunology ; Endothelium, Vascular/virology ; Granulocytes/immunology ; Granulocytes/virology ; Hematopoietic Cell Growth Factors/blood ; Hepatocyte Growth Factor/blood ; Humans ; Intensive Care Units ; Interleukin-12 Subunit p40/blood ; Interleukin-6/blood ; Interleukin-8/blood ; Killer Cells, Natural/immunology ; Killer Cells, Natural/virology ; Lectins, C-Type/blood ; Lymphopenia/diagnosis ; Lymphopenia/immunology ; Lymphopenia/mortality ; Lymphopenia/virology ; Plasma Cells/immunology ; Plasma Cells/virology ; SARS-CoV-2/pathogenicity ; Survival Analysis ; T-Lymphocytes/immunology ; T-Lymphocytes/virology
Chemical Substances	Antibodies, Viral ; Biomarkers ; Blood Proteins ; CLEC11A protein, human ; CXCL10 protein, human ; CXCL8 protein, human ; CXCL9 protein, human ; Chemokine CXCL10 ; Chemokine CXCL9 ; HGF protein, human ; Hematopoietic Cell Growth Factors ; IL6 protein, human ; Interleukin-12 Subunit p40 ; Interleukin-6 ; Interleukin-8 ; Lectins, C-Type ; Hepatocyte Growth Factor (67256-21-7) ; C-Reactive Protein (9007-41-4)
Language	English
Publishing date	2021-12-10
Publishing country	England
Document type	Journal Article ; Observational Study ; Research Support, Non-U.S. Gov't
ZDB-ID	2886872-9
ISSN	2059-3635 ; 2095-9907
ISSN (online)	2059-3635
ISSN	2095-9907
DOI	10.1038/s41392-021-00819-6
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Article ; Online: Cortical Thickness and Depressive Symptoms in Cognitively Normal Individuals: The Mayo Clinic Study of Aging.

Pink, Anna / Przybelski, Scott A / Krell-Roesch, Janina / Stokin, Gorazd B / Roberts, Rosebud O / Mielke, Michelle M / Knopman, David S / Jack, Clifford R / Petersen, Ronald C / Geda, Yonas E

Journal of Alzheimer's disease : JAD

2017 Volume 58, Issue 4, Page(s) 1273–1281

Abstract: Altered cortical thickness has been observed in aging and various neurodegenerative disorders. Furthermore, reduced hippocampal volume has been reported in late-life depression. Even mild depressive symptoms are common in the elderly. However, little is ... ...

Abstract	Altered cortical thickness has been observed in aging and various neurodegenerative disorders. Furthermore, reduced hippocampal volume has been reported in late-life depression. Even mild depressive symptoms are common in the elderly. However, little is known about the structural MRI measures of depressive symptoms in normal cognitive aging. Thus we sought to examine the association between depressive symptoms with cortical thickness and hippocampal volume as measured by brain MRI among community-dwelling participants. We conducted a cross-sectional study derived from the ongoing population-based Mayo Clinic Study of Aging, involving cognitively normal participants (N = 1,507) aged≥70 years. We observed that depressive symptoms were associated with lower global cortical thickness and lower thickness in specific prefrontal and temporal cortical regions, labeled by FreeSurfer software, version 5.3. As expected, the strength of correlation was very small, given that participants were community-dwelling with only mild depressive symptoms. We did not observe associations between hippocampal volume and depressive symptoms. These findings may provide insight into the structural correlates of mild depressive symptoms in elderly participants.
MeSH term(s)	Aged ; Aged, 80 and over ; Aging/pathology ; Aging/psychology ; Cerebral Cortex/diagnostic imaging ; Cerebral Cortex/pathology ; Cognition/physiology ; Cross-Sectional Studies ; Depression/pathology ; Depression/physiopathology ; Female ; Hippocampus/diagnostic imaging ; Hippocampus/pathology ; Humans ; Magnetic Resonance Imaging ; Male ; Neuropsychological Tests ; Psychiatric Status Rating Scales
Language	English
Publishing date	2017-05-26
Publishing country	Netherlands
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Research Support, N.I.H., Extramural
ZDB-ID	1440127-7
ISSN	1875-8908 ; 1387-2877
ISSN (online)	1875-8908
ISSN	1387-2877
DOI	10.3233/JAD-170041
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Article ; Online: Cortical Thickness and Anxiety Symptoms Among Cognitively Normal Elderly Persons: The Mayo Clinic Study of Aging.

Pink, Anna / Przybelski, Scott A / Krell-Roesch, Janina / Stokin, Gorazd B / Roberts, Rosebud O / Mielke, Michelle M / Spangehl, Kathleen A / Knopman, David S / Jack, Clifford R / Petersen, Ronald C / Geda, Yonas E

The Journal of neuropsychiatry and clinical neurosciences

2016 Volume 29, Issue 1, Page(s) 60–66

Abstract: The authors conducted a cross-sectional study to investigate the association between anxiety symptoms and cortical thickness, as well as amygdalar volume. A total of 1,505 cognitively normal participants, aged ≥70 years, were recruited from the Mayo ... ...

Abstract	The authors conducted a cross-sectional study to investigate the association between anxiety symptoms and cortical thickness, as well as amygdalar volume. A total of 1,505 cognitively normal participants, aged ≥70 years, were recruited from the Mayo Clinic Study of Aging in Olmsted County, Minnesota, on whom Beck Anxiety Inventory and 3T brain MRI data were available. Even though the effect sizes were small in this community-dwelling group of participants, anxiety symptoms were associated with reduced global cortical thickness and reduced thickness within the frontal and temporal cortex. However, after additionally adjusting for comorbid depressive symptoms, only the association between anxiety symptoms and reduced insular thickness remained significant.
MeSH term(s)	Aged ; Aging/pathology ; Aging/psychology ; Anxiety/diagnostic imaging ; Anxiety/epidemiology ; Apolipoprotein E4/genetics ; Cerebral Cortex/diagnostic imaging ; Comorbidity ; Cross-Sectional Studies ; Depression/diagnostic imaging ; Depression/epidemiology ; Female ; Humans ; Magnetic Resonance Imaging ; Male ; Minnesota/epidemiology ; Organ Size ; Psychiatric Status Rating Scales
Chemical Substances	Apolipoprotein E4
Language	English
Publishing date	2016-08-31
Publishing country	United States
Document type	Journal Article
ZDB-ID	1036340-3
ISSN	1545-7222 ; 0895-0172
ISSN (online)	1545-7222
ISSN	0895-0172
DOI	10.1176/appi.neuropsych.15100378
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