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Article ; Online: Correlation between salivary estrogen levels and oral epithelial cytokeratin 5 expression.

Handajani, Juni / Effendi, Nuraini / Sosroseno, Wihaskoro

2020 Volume 9, Page(s) 186

Abstract: Background: ...

Abstract	Background:
MeSH term(s)	Adult ; Age Factors ; Aged ; Child ; Epithelial Cells/metabolism ; Epithelium ; Estrogens/analysis ; Female ; Humans ; Keratin-5/metabolism ; Saliva/chemistry
Chemical Substances	Estrogens ; Keratin-5
Language	English
Publishing date	2020-03-12
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2699932-8
ISSN	2046-1402 ; 2046-1402
ISSN (online)	2046-1402
ISSN	2046-1402
DOI	10.12688/f1000research.22536.2
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: Effect of colchicine on the murine immune response induced by Aggregatibacter actinomycetemcomitans.

Sosroseno, W

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

2009 Volume 63, Issue 3, Page(s) 221–227

Abstract: The aim of the present study was to test the hypothesis that colchicine may alter Aggregatibacter actinomycetemcomitans-induced immune response and abscess formation in mice. BALB/c mice were either sham-immunized or immunized with heat-killed A. ... ...

Abstract	The aim of the present study was to test the hypothesis that colchicine may alter Aggregatibacter actinomycetemcomitans-induced immune response and abscess formation in mice. BALB/c mice were either sham-immunized or immunized with heat-killed A. actinomycetemcomitans. Spleen cells were stimulated with heat-killed A. actinomycetemcomitans in the presence or absence of colchicine. Specific IgG subclass antibodies, interferon-gamma (IFN-gamma), interleukin-4 (IL-4) and cell proliferation were determined. The animals were sham-immunized (group I) or immunized with heat-killed A. actinomycetemcomitans (groups II-VII). Colchicine was administered intraperitoneally before (group III), on the same day of (group IV), or after (group V) the primary immunization and on the same day of (group VI) or after (group VII) the secondary immunization. All groups were challenged with viable A. actinomycetemcomitans. The levels of serum-specific IgG subclasses and both IFN-gamma and IL-4 before and after bacterial challenge were assessed. The diameter of skin lesions was assessed. The results showed that colchicine augmented splenic-specific IgG1 and IL-4 as well as cell proliferation but suppressed specific IgG2a and IFN-gamma levels. Enhancement of serum-specific IgG1 and IL-4 levels, suppression of specific IgG2a and IFN-gamma levels as well as DTH response, and delayed healing of the lesions were observed in groups IV and VI, but not in the remaining groups of animals. Therefore, these results suggest that colchicine may induce a T helper 2 (Th2)-like immunity specific to A. actinomycetemcomitans in vitro and that colchicine administered on the same day as the immunization may stimulate a non-protective Th2-like immunity in A. actinomycetemcomitans-induced infections in mice.
MeSH term(s)	Actinobacillus Infections/immunology ; Aggregatibacter actinomycetemcomitans/immunology ; Animals ; Antibodies/immunology ; Cell Proliferation/drug effects ; Colchicine/pharmacology ; Female ; Gout Suppressants/pharmacology ; Immunoglobulin G/immunology ; Injections, Intraperitoneal ; Interferon-gamma/immunology ; Interleukin-4/immunology ; Mice ; Mice, Inbred BALB C ; Spleen/cytology ; Spleen/immunology ; Th2 Cells/immunology ; Time Factors
Chemical Substances	Antibodies ; Gout Suppressants ; Immunoglobulin G ; Interleukin-4 (207137-56-2) ; Interferon-gamma (82115-62-6) ; Colchicine (SML2Y3J35T)
Language	English
Publishing date	2009-03
Publishing country	France
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	392415-4
ISSN	1950-6007 ; 0753-3322 ; 0300-0893
ISSN (online)	1950-6007
ISSN	0753-3322 ; 0300-0893
DOI	10.1016/j.biopha.2008.04.004
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Article: The effect of L-arginine on Porphyromonas gingivalis-induced phagocytosis of a murine macrophage-like RAW264.7 cell line.

Sosroseno, Wihaskoro

Immunopharmacology and immunotoxicology

2004 Volume 26, Issue 2, Page(s) 309–313

Abstract: The aim of this study was to determine the effect of L-arginine on Porphyromonas gingivalis-induced phagocytosis by RAW264.7 cells. The cells were pretreated with L-arginine or D-arginine prior to incubation with either unopsonized or opsonized P. ... ...

Abstract	The aim of this study was to determine the effect of L-arginine on Porphyromonas gingivalis-induced phagocytosis by RAW264.7 cells. The cells were pretreated with L-arginine or D-arginine prior to incubation with either unopsonized or opsonized P. gingivalis. In other experiments, the cells were pretreated with L-arginine and various concentrations of NMLA (N(G)-monomethyl-L-arginine) prior to incubation with the bacteria. The phagocytosis was microscopically assessed and determined by the phagocytic index. The results showed that L-arginine, but not D-arginine enhances the ability of RAW264.7 cells to engulf the bacteria. The upregulatory effect of L-arginine on P. gingivalis-induced phagocytosis was abolished by NMLA. The results of the present study suggest that L-arginine may upregulate the P. gingivalis-induced phagocytic activity of RAW264.7 cells, perhaps, via modulation of nitric oxide synthase.
MeSH term(s)	Animals ; Arginine/pharmacology ; Cell Line ; Macrophages/drug effects ; Macrophages/immunology ; Macrophages/microbiology ; Mice ; Phagocytosis/drug effects ; Porphyromonas gingivalis/immunology ; Porphyromonas gingivalis/pathogenicity ; omega-N-Methylarginine/pharmacology
Chemical Substances	omega-N-Methylarginine (27JT06E6GR) ; Arginine (94ZLA3W45F)
Language	English
Publishing date	2004-06-09
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	807033-7
ISSN	1532-2513 ; 0892-3973
ISSN (online)	1532-2513
ISSN	0892-3973
DOI	10.1081/iph-120037724
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Zs.A 1481: Show issues

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Article: Effect of sodium fluoride on the murine splenic immune response to Porphyromonas gingivalis in vitro.

Sosroseno, Wihaskoro

Immunopharmacology and immunotoxicology

2003 Volume 25, Issue 1, Page(s) 123–127

Abstract: Spleen cells from saline- and Porphyromonas gingivalis-primed mice were cultured and stimulated with or without P. gingivalis and added with or without various concentration of sodium fluoride (NaF). Cell proliferation, antigen-specific IgG antibodies ... ...

Abstract	Spleen cells from saline- and Porphyromonas gingivalis-primed mice were cultured and stimulated with or without P. gingivalis and added with or without various concentration of sodium fluoride (NaF). Cell proliferation, antigen-specific IgG antibodies and both IFN-gamma and IL-10 levels were determined by a colorimetric assay, ELISA and commercial ELISA kits respectively. The results showed that NaF at concentration of 1 x 10(-6) M enhanced but at concentration of 1 x 10(-1) M abolished the immune response to P. gingivalis, suggesting that NaF at low concentration may act as an adjuvant but at high concentration may be toxic to the P. gingivalis-induced murine splenic immune response in vitro.
MeSH term(s)	Animals ; Cells, Cultured ; Cytokines/drug effects ; Dose-Response Relationship, Drug ; Enzyme-Linked Immunosorbent Assay ; Female ; Immunity/drug effects ; Interleukin-10/metabolism ; Mice ; Mice, Inbred BALB C ; Porphyromonas/immunology ; Sodium Fluoride/pharmacology ; Spleen/cytology ; Spleen/drug effects ; Spleen/immunology
Chemical Substances	Cytokines ; Interleukin-10 (130068-27-8) ; Sodium Fluoride (8ZYQ1474W7)
Language	English
Publishing date	2003-04-01
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	807033-7
ISSN	1532-2513 ; 0892-3973
ISSN (online)	1532-2513
ISSN	0892-3973
DOI	10.1081/iph-120018287
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Article ; Online: The protective effects of antigen-specific IgY on pyocyanin-treated human lymphoma Raji cells.

Susilowati, Heni / Artanto, Sidna / Yulianto, Heribertus Dedy Kusuma / Sosroseno, Wihaskoro / Hutomo, Suryani

F1000Research

2019 Volume 8, Page(s) 1008

Abstract: Background: ...

Abstract	Background:
MeSH term(s)	Animals ; Chickens ; Egg Yolk ; Humans ; Immunoglobulins ; Infant ; Lymphoma ; Pyocyanine
Chemical Substances	IgY ; Immunoglobulins ; Pyocyanine (9OQM399341)
Language	English
Publishing date	2019-07-04
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2699932-8
ISSN	2046-1402 ; 2046-1402
ISSN (online)	2046-1402
ISSN	2046-1402
DOI	10.12688/f1000research.19327.2
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Article: Nitric oxide production by murine spleen cells stimulated with Porphyromonas gingivalis-derived lipopolysaccharide.

Sosroseno, W

Asian Pacific journal of allergy and immunology

2000 Volume 18, Issue 4, Page(s) 209–214

Abstract: The aim of the present study was to determine whether Porphyromonas gingivalis-lipopolysaccharide (Pg-LPS) may stimulate nitric oxide (NO) production by murine spleen cells. Spleen cells derived from Balb/c mice were cultured in the presence of Pg-LPS or ...

Abstract	The aim of the present study was to determine whether Porphyromonas gingivalis-lipopolysaccharide (Pg-LPS) may stimulate nitric oxide (NO) production by murine spleen cells. Spleen cells derived from Balb/c mice were cultured in the presence of Pg-LPS or LPS from Salmonella Typhosa. The cell were also cultured in the presence of Pg-LPS with or without L-arginine, L-arginine plus NG-monomethyl-L-arginine (NMMA), or IFN-gamma. Furthermore, the plastic non-adherent spleen cells were stimulated with Pg-LPS and L-arginine. The results showed that Pg-LPS failed to stimulate splenic NO production by themselves. Exogenous L-arginine or IFN-gamma up-regulated the NO production of Pg-LPS-stimulated spleen cells, but the stimulatory effects of L-arginine were completely blocked by NMMA. It was also demonstrated that in the presence of Pg-LPS and L-arginine, splenic macrophages were the cellular source of NO. These results suggest, therefore, that P. gingivalis-LPS may induce murine splenic macrophages to produce NO in a L-arginine and an IFN-gamma-dependent mechanism.
MeSH term(s)	Animals ; Antineoplastic Agents/pharmacology ; Arginine/pharmacology ; Cells, Cultured ; Enzyme Inhibitors/pharmacology ; Female ; Interferon-gamma/pharmacology ; Lipopolysaccharides/pharmacology ; Mice ; Mice, Inbred BALB C ; Nitric Oxide/biosynthesis ; Nitric Oxide/metabolism ; Porphyromonas gingivalis ; Salmonella typhi ; Spleen/cytology ; Spleen/drug effects ; Spleen/metabolism ; omega-N-Methylarginine/pharmacology
Chemical Substances	Antineoplastic Agents ; Enzyme Inhibitors ; Lipopolysaccharides ; omega-N-Methylarginine (27JT06E6GR) ; Nitric Oxide (31C4KY9ESH) ; Interferon-gamma (82115-62-6) ; Arginine (94ZLA3W45F)
Language	English
Publishing date	2000-12
Publishing country	Thailand
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	605782-2
ISSN	2228-8694 ; 0125-877X
ISSN (online)	2228-8694
ISSN	0125-877X
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Zs.A 1966: Show issues

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Article ; Online: The effect of immune status, age and genetic background on induction of oral tolerance to Actinomyces viscosus in mice.

Sosroseno, Wihaskoro / Herminajeng, Endang / Bird, Phil

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

2015 Volume 70, Page(s) 294–298

Abstract: The aim of the present study was to determine the effect of immune status, age and genetic background on the induction of oral tolerance to Actinomyces viscosus. Suppression of delayed type hypersensitivity (DTH) response and antigen-specific serum ... ...

Abstract	The aim of the present study was to determine the effect of immune status, age and genetic background on the induction of oral tolerance to Actinomyces viscosus. Suppression of delayed type hypersensitivity (DTH) response and antigen-specific serum antibody levels could be induced in DBA/2 mice intragastrically and systemically immunized with A. viscocus, suggesting the induction of oral tolerance. In contrast, this immune suppression could be abrogated if the animals had been systemically immunized prior to the induction of oral tolerance with the same bacterium. Long-term systemic immunization prior to intragastric immunization with A. viscocus suppressed DTH response only. Cell transfer of this group of animals also suppressed DTH response in the donors, indicating the action of suppressor cells for inhibition of DTH response. Furthermore, oral tolerance to A. viscocus failed to occur in mice aged at 3 days and 1, 2, 4, 6 and 36 weeks old. Mice bearing H-2(d) haplotype were the most susceptible to oral tolerization, followed by H-2(b) and H-2(k). Therefore, the results of the presence study suggest that the induction of oral tolerance to A. viscosus in mice may be dependence on the immune status and genetic background but not age.
MeSH term(s)	Actinomyces viscosus ; Administration, Oral ; Age Factors ; Animals ; Female ; Genetic Background ; Immune Tolerance/genetics ; Immune Tolerance/immunology ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C3H ; Mice, Inbred C57BL ; Mice, Inbred DBA ; Mouth Mucosa/immunology ; Mouth Mucosa/microbiology ; Species Specificity
Language	English
Publishing date	2015-03
Publishing country	France
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	392415-4
ISSN	1950-6007 ; 0753-3322 ; 0300-0893
ISSN (online)	1950-6007
ISSN	0753-3322 ; 0300-0893
DOI	10.1016/j.biopha.2014.12.039
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Ud II Zs.198: Show issues

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Article: A review of the mechanisms of oral tolerance and immunotherapy.

Sosroseno, W

Journal of the Royal Society of Medicine

1995 Volume 88, Issue 1, Page(s) 14–17

Abstract: The induction of oral tolerance by oral immunization has been well recognized. Accumulated evidence shows that oral tolerance can be mediated by orally activated humoral and cellular factors. In animal models, the development of several T cell-mediated ... ...

Abstract	The induction of oral tolerance by oral immunization has been well recognized. Accumulated evidence shows that oral tolerance can be mediated by orally activated humoral and cellular factors. In animal models, the development of several T cell-mediated autoimmune diseases, such as multiple sclerosis, rheumatoid arthritis, uveitis and diabetes type 1 can be inhibited by oral immunization of the respective antigens. In allergy, oral administration of certain allergens can prevent and reduce both contact and atopic dermatitis. Oral tolerance to alloantigen also reduces graft rejection. In spite of these encouraging results, the usefulness of this approach for an alternative immunotherapy in humans needs to be investigated further.
MeSH term(s)	Administration, Oral ; Autoimmune Diseases/therapy ; Humans ; Hypersensitivity/therapy ; Immune Tolerance ; Immunosuppression ; Immunotherapy ; T-Lymphocytes/immunology ; T-Lymphocytes, Regulatory/immunology ; Transplantation Immunology
Language	English
Publishing date	1995-01
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	6731-3
ISSN	1758-1095 ; 0141-0768 ; 0035-9157
ISSN (online)	1758-1095
ISSN	0141-0768 ; 0035-9157
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Article: The immunology of nickel-induced allergic contact dermatitis.

Sosroseno, W

Asian Pacific journal of allergy and immunology

1995 Volume 13, Issue 2, Page(s) 173–181

Abstract: Attempts have been made to elucidate the immunopathogenesis of contact allergy; yet, the exact mechanism by which nickel-induced allergic contact dermatitis (NACD) occurs is far from clear and is discussed herein. It seems to suggest that a direct nickel- ...

Abstract	Attempts have been made to elucidate the immunopathogenesis of contact allergy; yet, the exact mechanism by which nickel-induced allergic contact dermatitis (NACD) occurs is far from clear and is discussed herein. It seems to suggest that a direct nickel-MHC class II molecule binding on the skin antigen presenting cells such as Langerhans cells (LCs) would result in Th1 cell activation. Substances such as serotonin and cytokines such as TNF-alpha produced by activated mast cells may increase adhesion molecule expression and thus, enhance T cell trafficking in the skin. Cytokines such as IFN-gamma and IL-1 and perhaps IL-12 certainly play a crucial role in the activation of Th1 cells. Along with possible function of CD8 cells, downregulation of NACD may be mediated by suppressed function of LCs via the action of activated keratinocytes-derived IL-10. Inhibition of NACD can also be generated by feeding with nickel, suggesting that the induction of oral tolerance to nickel may be beneficial for an alternative immunotherapy of nickel allergy. Nevertheless, this testable model provides a direction for further investigation.
MeSH term(s)	Animals ; Antigen-Presenting Cells/immunology ; Cell Adhesion Molecules/immunology ; Cytokines/immunology ; Dermatitis, Allergic Contact/etiology ; Dermatitis, Allergic Contact/immunology ; Dermatitis, Allergic Contact/prevention & control ; Humans ; Nickel/administration & dosage ; Nickel/adverse effects ; Skin/immunology ; T-Lymphocytes/immunology
Chemical Substances	Cell Adhesion Molecules ; Cytokines ; Nickel (7OV03QG267)
Language	English
Publishing date	1995-12
Publishing country	Thailand
Document type	Journal Article ; Review
ZDB-ID	605782-2
ISSN	2228-8694 ; 0125-877X
ISSN (online)	2228-8694
ISSN	0125-877X
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Article ; Online: Nitric oxide production by a murine macrophage cell line (RAW264.7 cells) stimulated with Aggregatibacter actinomycetemcomitans surface-associated material.

Sosroseno, W / Bird, P S / Seymour, G J

Anaerobe

2011 Volume 17, Issue 5, Page(s) 246–251

Abstract: Nitric oxide (NO) may play a crucial role in the pathogenesis of periodontal disease and, hence, the aim of the present study was to test the hypothesis that Aggregatibacter actinomycetemcomitans surface-associated material (SAM) stimulates inducible ... ...

Abstract	Nitric oxide (NO) may play a crucial role in the pathogenesis of periodontal disease and, hence, the aim of the present study was to test the hypothesis that Aggregatibacter actinomycetemcomitans surface-associated material (SAM) stimulates inducible nitric oxide synthase (iNOS) activity and NO production by the murine macrophage cell line RAW264.7. Cells were stimulated with untreated or heat-treated A. actinomycetemcomitans SAM and with or without pre-treatment with L-N(6)-(1-Iminoethyl)-lysine (L-NIL) (an iNOS inhibitor), polymyxin B, interferon-gamma (IFN-γ) and Interleukin-4 (IL-4), IL-10, genistein [a protein tyrosine kinase (PTK) inhibitor], bisindolylmaleimide [a protein kinase C (PKC) inhibitor], bromophenacyl bromide (BPB) [a phospholipase A(2) (PLA2) inhibitor] or wortmannin [phosphatidylinositol 3-kinase (PI-3K) inhibitor]. The iNOS activity and nitrite production in the cell cultures were determined. Untreated but not heat-treated A. actinomycetemcomitans SAM-stimulated both iNOS activity and nitrite production in RAW264.7 cells. L-NIL, IL-4, IL-10, genistein, bisindolylmaleimide, or BPB, suppressed but IFN-γ enhanced both iNOS activity and nitrite production by A. actinomycetemcomitans SAM-stimulated cells. Wortmannin and polymyxin B failed to alter both iNOS activity or nitrite production by A. actinomycetemcomitans SAM treated cells. Therefore, the present study suggests that a heat-sensitive protein constituent(s) of A. actinomycetemcomitans SAM stimulates both iNOS activity and nitrite production by RAW264.7 cells in a cytokine, PTK, PKC, and PLA(2) but not PI-3K-dependent fashion.
MeSH term(s)	Aggregatibacter actinomycetemcomitans/immunology ; Aggregatibacter actinomycetemcomitans/metabolism ; Animals ; Bacterial Proteins/immunology ; Cell Line ; Cytokines/metabolism ; Cytokines/pharmacology ; Enzyme Inhibitors/immunology ; Humans ; Lipopolysaccharides/immunology ; Macrophages/immunology ; Macrophages/metabolism ; Mice ; Nitric Oxide/biosynthesis ; Nitric Oxide Synthase Type II/antagonists & inhibitors ; Nitric Oxide Synthase Type II/immunology ; Nitric Oxide Synthase Type II/metabolism ; Nitrites/metabolism ; Phospholipases A2/metabolism ; Protein Kinase C/metabolism ; Protein-Tyrosine Kinases/metabolism
Chemical Substances	Bacterial Proteins ; Cytokines ; Enzyme Inhibitors ; Lipopolysaccharides ; Nitrites ; Nitric Oxide (31C4KY9ESH) ; Nitric Oxide Synthase Type II (EC 1.14.13.39) ; Protein-Tyrosine Kinases (EC 2.7.10.1) ; Protein Kinase C (EC 2.7.11.13) ; Phospholipases A2 (EC 3.1.1.4)
Language	English
Publishing date	2011-10
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1237621-8
ISSN	1095-8274 ; 1075-9964
ISSN (online)	1095-8274
ISSN	1075-9964
DOI	10.1016/j.anaerobe.2011.06.006
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