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  1. Article ; Online: Reply from Lykke Sylow, Lisbeth L. V. Møller, Maximilian Kleinert, Erik A. Richter and Thomas E. Jensen.

    Sylow, Lykke / Møller, Lisbeth L V / Kleinert, Maximilian / Richter, Erik A / Jensen, Thomas E

    The Journal of physiology

    2015  Volume 593, Issue 9, Page(s) 2239–2240

    MeSH term(s) Animals ; Female ; Glucose/metabolism ; Muscle Contraction ; Muscle, Skeletal/metabolism ; Neuropeptides/metabolism ; rac1 GTP-Binding Protein/metabolism
    Chemical Substances Neuropeptides ; rac1 GTP-Binding Protein (EC 3.6.5.2) ; Glucose (IY9XDZ35W2)
    Language English
    Publishing date 2015-05-01
    Publishing country England
    Document type Comment ; Letter
    ZDB-ID 3115-x
    ISSN 1469-7793 ; 0022-3751
    ISSN (online) 1469-7793
    ISSN 0022-3751
    DOI 10.1113/JP270414
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  2. Article: Erik G. Furubotn at 70

    Richter, Rudolf / Furubotn, Eirik Grundtvig

    Journal of institutional and theoretical economics : JITE 149 ,2, S. 363-364

    1993  

    Author's details Rudolf Richter
    Keywords 49 ; Furubotn
    Language English
    Publisher Mohr Siebeck
    Publishing place Tübingen
    Document type Article
    ZDB-ID 232799-5 ; 200227-9
    ISSN 0044-2550
    ISSN 0044-2550
    Database ECONomics Information System

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  3. Article ; Online: Exploratory physiological experiments are not clinical trials.

    Richter, Erik A / James, David E / Kirwan, John P / Zierath, Juleen R

    Cell metabolism

    2024  

    Language English
    Publishing date 2024-04-16
    Publishing country United States
    Document type Letter
    ZDB-ID 2176834-1
    ISSN 1932-7420 ; 1550-4131
    ISSN (online) 1932-7420
    ISSN 1550-4131
    DOI 10.1016/j.cmet.2024.04.005
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  4. Article ; Online: Is GLUT4 translocation the answer to exercise-stimulated muscle glucose uptake?

    Richter, Erik A

    American journal of physiology. Endocrinology and metabolism

    2020  Volume 320, Issue 2, Page(s) E240–E243

    Abstract: Exercise in humans increases muscle glucose uptake up to 100-fold compared with rest. The magnitude of increase depends on exercise intensity and duration. Although knockout of glucose transporter type 4 (GLUT4) convincingly has shown that GLUT4 is ... ...

    Abstract Exercise in humans increases muscle glucose uptake up to 100-fold compared with rest. The magnitude of increase depends on exercise intensity and duration. Although knockout of glucose transporter type 4 (GLUT4) convincingly has shown that GLUT4 is necessary for exercise to increase muscle glucose uptake, studies only show an approximate twofold increase in GLUT4 translocation to the muscle cell membrane when transitioning from rest to exercise. Therefore, there is a big discrepancy between the increase in glucose uptake and GLUT4 translocation. It is suggested that either the methods for measurements of GLUT4 translocation in muscle grossly underestimate the real translocation of GLUT4 or, alternatively, GLUT4 intrinsic activity increases in muscle during exercise, perhaps due to increased muscle temperature and/or mechanical effects during contraction/relaxation cycles.
    MeSH term(s) Animals ; Carbohydrate Metabolism/physiology ; Exercise/physiology ; Glucose/metabolism ; Glucose Transporter Type 4/metabolism ; Humans ; Insulin/metabolism ; Muscle Contraction/physiology ; Muscle, Skeletal/metabolism ; Protein Transport
    Chemical Substances Glucose Transporter Type 4 ; Insulin ; Glucose (IY9XDZ35W2)
    Language English
    Publishing date 2020-11-09
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 603841-4
    ISSN 1522-1555 ; 0193-1849
    ISSN (online) 1522-1555
    ISSN 0193-1849
    DOI 10.1152/ajpendo.00503.2020
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  5. Article ; Online: Transcellular Barriers to Glucose Delivery in the Body.

    Klip, Amira / De Bock, Katrien / Bilan, Philip J / Richter, Erik A

    Annual review of physiology

    2024  Volume 86, Page(s) 149–173

    Abstract: Glucose is the universal fuel of most mammalian cells, and it is largely replenished through dietary intake. Glucose availability to tissues is paramount for the maintenance of homeostatic energetics and, hence, supply should match demand by the ... ...

    Abstract Glucose is the universal fuel of most mammalian cells, and it is largely replenished through dietary intake. Glucose availability to tissues is paramount for the maintenance of homeostatic energetics and, hence, supply should match demand by the consuming organs. In its journey through the body, glucose encounters cellular barriers for transit at the levels of the absorbing intestinal epithelial wall, the renal epithelium mediating glucose reabsorption, and the tight capillary endothelia (especially in the brain). Glucose transiting through these cellular barriers must escape degradation to ensure optimal glucose delivery to the bloodstream or tissues. The liver, which stores glycogen and generates glucose de novo, must similarly be able to release it intact to the circulation. We present the most up-to-date knowledge on glucose handling by the gut, liver, brain endothelium, and kidney, and discuss underlying molecular mechanisms and open questions. Diseases associated with defects in glucose delivery and homeostasis are also briefly addressed. We propose that the universal problem of sparing glucose from catabolism in favor of translocation across the barriers posed by epithelia and endothelia is resolved through common mechanisms involving glucose transfer to the endoplasmic reticulum, from where glucose exits the cells via unconventional cellular mechanisms.
    MeSH term(s) Animals ; Humans ; Glucose/metabolism ; Epithelium/metabolism ; Brain/metabolism ; Biological Transport ; Intestines ; Mammals/metabolism
    Chemical Substances Glucose (IY9XDZ35W2)
    Language English
    Publishing date 2024-02-12
    Publishing country United States
    Document type Journal Article ; Review
    ZDB-ID 207933-1
    ISSN 1545-1585 ; 0066-4278
    ISSN (online) 1545-1585
    ISSN 0066-4278
    DOI 10.1146/annurev-physiol-042022-031657
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  6. Book ; Conference proceedings: Skeletal muscle metabolism in exercise and diabetes

    Richter, Erik A.

    [proceedings of the Copenhagen Muscle Research Centre Symposium on Regulation of Skeletal Muscle Metabolism: Focus on Glucose Transport, Exercise, and Diabetes, held October 23 - 26, 1997, in Copenhagen, Denmark]

    (Advances in experimental medicine and biology ; 441)

    1998  

    Institution Muscle Research Centre
    Symposium on Regulation of Skeletal Muscle Metabolism: Focus on Glucose Transport, Exercise, and Diabetes
    Author's details ed. by Erik A. Richter
    Series title Advances in experimental medicine and biology ; 441
    Collection
    Keywords Muscle, Skeletal / metabolism / congresses ; Glucose / metabolism / congresses ; Insulin / physiology / congresses ; Exercise / physiology / congresses ; Fatty Acids / metabolism / congresses ; Diabetes Mellitus / physiopathology / congresses ; Biological Transport / congresses ; Quer gestreifte Muskulatur ; Lipidstoffwechsel ; Lipidstoffwechselstörung ; Diabetes mellitus ; Glucosestoffwechsel ; Glucosestoffwechselstörung
    Subject Diabetes verus ; Zuckerharnruhr ; Zuckerkrankheit ; Glucose ; Glukosestoffwechselstörung ; Quergestreifte Muskulatur ; Quergestreifter Muskel ; Quer gestreifter Muskel ; Glukosestoffwechsel ; Glukosemetabolismus ; Glucosemetabolismus ; Fettstoffwechselstörung ; Dyslipämie ; Dyslipoproteinämie ; Dyslipidämie ; Fettstoffwechsel ; Lipide
    Language English
    Size IX, 328 S. : Ill., graph. Darst.
    Publisher Plenum Press
    Publishing place New York u.a.
    Publishing country United States
    Document type Book ; Conference proceedings
    HBZ-ID HT009061073
    ISBN 0-306-45920-5 ; 978-0-306-45920-7
    Database Catalogue ZB MED Medicine, Health

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    Shelf markLoan statusLocation
    Zs A 3192 -441- not available for loan Zeitschriften-Lesesaal

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  7. Article ; Online: Novel regulatory mechanisms in muscle metabolism during exercise.

    Richter, Erik A

    Experimental physiology

    2014  Volume 99, Issue 12, Page(s) 1559–1561

    MeSH term(s) Exercise/physiology ; Humans ; Muscle, Skeletal/metabolism
    Language English
    Publishing date 2014-12-01
    Publishing country England
    Document type Introductory Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 1016295-1
    ISSN 1469-445X ; 0958-0670
    ISSN (online) 1469-445X
    ISSN 0958-0670
    DOI 10.1113/expphysiol.2014.079111
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  8. Article ; Online: Interactions between insulin and exercise.

    Richter, Erik A / Sylow, Lykke / Hargreaves, Mark

    The Biochemical journal

    2021  Volume 478, Issue 21, Page(s) 3827–3846

    Abstract: The interaction between insulin and exercise is an example of balancing and modifying the effects of two opposing metabolic regulatory forces under varying conditions. While insulin is secreted after food intake and is the primary hormone increasing ... ...

    Abstract The interaction between insulin and exercise is an example of balancing and modifying the effects of two opposing metabolic regulatory forces under varying conditions. While insulin is secreted after food intake and is the primary hormone increasing glucose storage as glycogen and fatty acid storage as triglycerides, exercise is a condition where fuel stores need to be mobilized and oxidized. Thus, during physical activity the fuel storage effects of insulin need to be suppressed. This is done primarily by inhibiting insulin secretion during exercise as well as activating local and systemic fuel mobilizing processes. In contrast, following exercise there is a need for refilling the fuel depots mobilized during exercise, particularly the glycogen stores in muscle. This process is facilitated by an increase in insulin sensitivity of the muscles previously engaged in physical activity which directs glucose to glycogen resynthesis. In physically trained individuals, insulin sensitivity is also higher than in untrained individuals due to adaptations in the vasculature, skeletal muscle and adipose tissue. In this paper, we review the interactions between insulin and exercise during and after exercise, as well as the effects of regular exercise training on insulin action.
    MeSH term(s) Animals ; Exercise ; Glucose/metabolism ; Glycogen/metabolism ; Humans ; Insulin/metabolism ; Muscles/metabolism
    Chemical Substances Insulin ; Glycogen (9005-79-2) ; Glucose (IY9XDZ35W2)
    Language English
    Publishing date 2021-11-03
    Publishing country England
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 2969-5
    ISSN 1470-8728 ; 0006-2936 ; 0306-3275 ; 0264-6021
    ISSN (online) 1470-8728
    ISSN 0006-2936 ; 0306-3275 ; 0264-6021
    DOI 10.1042/BCJ20210185
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  9. Article ; Online: Plant Membrane-On-A-Chip: A Platform for Studying Plant Membrane Proteins and Lipids.

    Stuebler, Martin / Manzer, Zachary A / Liu, Han-Yuan / Miller, Julia / Richter, Annett / Krishnan, Srinivasan / Selivanovitch, Ekaterina / Banuna, Barituziga / Jander, Georg / Reimhult, Erik / Zipfel, Warren R / Roeder, Adrienne H K / Piñeros, Miguel A / Daniel, Susan

    ACS applied materials & interfaces

    2024  

    Abstract: The cell plasma membrane is a two-dimensional, fluid mosaic material composed of lipids and proteins that create a semipermeable barrier defining the cell from its environment. Compared with soluble proteins, the methodologies for the structural and ... ...

    Abstract The cell plasma membrane is a two-dimensional, fluid mosaic material composed of lipids and proteins that create a semipermeable barrier defining the cell from its environment. Compared with soluble proteins, the methodologies for the structural and functional characterization of membrane proteins are challenging. An emerging tool for studies of membrane proteins in mammalian systems is a "plasma membrane on a chip," also known as a supported lipid bilayer. Here, we create the "plant-membrane-on-a-chip,″ a supported bilayer made from the plant plasma membranes of
    Language English
    Publishing date 2024-04-09
    Publishing country United States
    Document type Journal Article
    ISSN 1944-8252
    ISSN (online) 1944-8252
    DOI 10.1021/acsami.3c18562
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  10. Article ; Online: Soil warming increases the number of growing bacterial taxa but not their growth rates.

    Metze, Dennis / Schnecker, Jörg / de Carlan, Coline Le Noir / Bhattarai, Biplabi / Verbruggen, Erik / Ostonen, Ivika / Janssens, Ivan A / Sigurdsson, Bjarni D / Hausmann, Bela / Kaiser, Christina / Richter, Andreas

    Science advances

    2024  Volume 10, Issue 8, Page(s) eadk6295

    Abstract: Soil microorganisms control the fate of soil organic carbon. Warming may accelerate their activities putting large carbon stocks at risk of decomposition. Existing knowledge about microbial responses to warming is based on community-level measurements, ... ...

    Abstract Soil microorganisms control the fate of soil organic carbon. Warming may accelerate their activities putting large carbon stocks at risk of decomposition. Existing knowledge about microbial responses to warming is based on community-level measurements, leaving the underlying mechanisms unexplored and hindering predictions. In a long-term soil warming experiment in a Subarctic grassland, we investigated how active populations of bacteria and archaea responded to elevated soil temperatures (+6°C) and the influence of plant roots, by measuring taxon-specific growth rates using quantitative stable isotope probing and
    MeSH term(s) Soil ; Carbon ; Soil Microbiology ; Bacteria ; Archaea
    Chemical Substances Soil ; Carbon (7440-44-0)
    Language English
    Publishing date 2024-02-23
    Publishing country United States
    Document type Journal Article
    ZDB-ID 2810933-8
    ISSN 2375-2548 ; 2375-2548
    ISSN (online) 2375-2548
    ISSN 2375-2548
    DOI 10.1126/sciadv.adk6295
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