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Article ; Online: The once-daily fixed-dose combination of olodaterol and tiotropium in the management of COPD: current evidence and future prospects.

Derom, Eric / Brusselle, Guy G / Joos, Guy F

Therapeutic advances in respiratory disease

2019 Volume 13, Page(s) 1753466619843426

Abstract: Long-acting bronchodilators are the cornerstone of pharmacologic treatment of chronic obstructive pulmonary disease (COPD). ... ...

Abstract	Long-acting bronchodilators are the cornerstone of pharmacologic treatment of chronic obstructive pulmonary disease (COPD). Spiolto
MeSH term(s)	Benzoxazines/administration & dosage ; Benzoxazines/pharmacology ; Bronchodilator Agents/administration & dosage ; Bronchodilator Agents/pharmacology ; Delayed-Action Preparations ; Drug Combinations ; Humans ; Muscarinic Antagonists/administration & dosage ; Muscarinic Antagonists/pharmacology ; Pulmonary Disease, Chronic Obstructive/drug therapy ; Pulmonary Disease, Chronic Obstructive/physiopathology ; Respiratory Function Tests ; Severity of Illness Index ; Tiotropium Bromide/administration & dosage ; Tiotropium Bromide/pharmacology
Chemical Substances	Benzoxazines ; Bronchodilator Agents ; Delayed-Action Preparations ; Drug Combinations ; Muscarinic Antagonists ; tiotropium-olodaterol ; Tiotropium Bromide (XX112XZP0J)
Language	English
Publishing date	2019-04-19
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	2476459-0
ISSN	1753-4666 ; 1753-4658
ISSN (online)	1753-4666
ISSN	1753-4658
DOI	10.1177/1753466619843426
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Zs.A 6753: Show issues

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Article ; Online: Efficacy of tiotropium-olodaterol fixed-dose combination in COPD.

Derom, Eric / Brusselle, Guy G / Joos, Guy F

International journal of chronic obstructive pulmonary disease

2016 Volume 11, Page(s) 3163–3177

Abstract: Tiotropium-olodaterol, formulated in the Respimat soft-mist inhaler, is an inhaled fixed-dose combination (FDC) of a long-acting muscarinic antagonist (LAMA) and a long-acting ... ...

Abstract	Tiotropium-olodaterol, formulated in the Respimat soft-mist inhaler, is an inhaled fixed-dose combination (FDC) of a long-acting muscarinic antagonist (LAMA) and a long-acting β
MeSH term(s)	Administration, Inhalation ; Adrenergic beta-2 Receptor Agonists/administration & dosage ; Adrenergic beta-2 Receptor Agonists/adverse effects ; Benzoxazines/administration & dosage ; Benzoxazines/adverse effects ; Bronchodilator Agents/administration & dosage ; Bronchodilator Agents/adverse effects ; Drug Combinations ; Exercise Tolerance/drug effects ; Forced Expiratory Volume ; Humans ; Lung/drug effects ; Lung/physiopathology ; Muscarinic Antagonists/administration & dosage ; Muscarinic Antagonists/adverse effects ; Pulmonary Disease, Chronic Obstructive/diagnosis ; Pulmonary Disease, Chronic Obstructive/drug therapy ; Pulmonary Disease, Chronic Obstructive/physiopathology ; Quality of Life ; Recovery of Function ; Time Factors ; Tiotropium Bromide/administration & dosage ; Tiotropium Bromide/adverse effects ; Treatment Outcome
Chemical Substances	Adrenergic beta-2 Receptor Agonists ; Benzoxazines ; Bronchodilator Agents ; Drug Combinations ; Muscarinic Antagonists ; tiotropium-olodaterol ; Tiotropium Bromide (XX112XZP0J)
Language	English
Publishing date	2016
Publishing country	New Zealand
Document type	Journal Article ; Review
ISSN	1178-2005
ISSN (online)	1178-2005
DOI	10.2147/COPD.S92840
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Article ; Online: Increased expression of ACE2, the SARS-CoV-2 entry receptor, in alveolar and bronchial epithelium of smokers and COPD subjects.

Jacobs, Merel / Van Eeckhoutte, Hannelore P / Wijnant, Sara R A / Janssens, Wim / Joos, Guy F / Brusselle, Guy G / Bracke, Ken R

The European respiratory journal

2020 Volume 56, Issue 2

MeSH term(s)	Angiotensin-Converting Enzyme 2 ; Belgium ; Betacoronavirus/metabolism ; Biomarkers/blood ; Biopsy, Needle ; Cross-Sectional Studies ; Female ; Humans ; Immunohistochemistry ; In Vitro Techniques ; Linear Models ; Male ; Multivariate Analysis ; Peptidyl-Dipeptidase A/metabolism ; Pulmonary Disease, Chronic Obstructive/blood ; Pulmonary Disease, Chronic Obstructive/epidemiology ; Pulmonary Disease, Chronic Obstructive/pathology ; RNA, Messenger/blood ; Real-Time Polymerase Chain Reaction/methods ; Respiratory Mucosa/metabolism ; Respiratory Mucosa/pathology ; SARS-CoV-2 ; Sensitivity and Specificity ; Smoking/blood ; Smoking/epidemiology ; Specimen Handling ; Statistics, Nonparametric
Chemical Substances	Biomarkers ; RNA, Messenger ; Peptidyl-Dipeptidase A (EC 3.4.15.1) ; ACE2 protein, human (EC 3.4.17.23) ; Angiotensin-Converting Enzyme 2 (EC 3.4.17.23)
Keywords	covid19
Language	English
Publishing date	2020-08-20
Publishing country	England
Document type	Letter
ZDB-ID	639359-7
ISSN	1399-3003 ; 0903-1936
ISSN (online)	1399-3003
ISSN	0903-1936
DOI	10.1183/13993003.02378-2020
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Zs.A 2322: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 292: Show issues

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Article ; Online: Is there still hope for single therapies: how do we set up experimental systems to efficiently test combination therapies?

Joos, Guy F / Gabazza, Esteban C

Respirology (Carlton, Vic.)

2015 Volume 20, Issue 1, Page(s) 15–23

Abstract: Severe asthma, chronic obstructive pulmonary disease (COPD) and idiopathic pulmonary fibrosis (IPF) are chronic lung diseases with a clear need for development of new and more efficient therapy. In preclinical research, the mouse model has been ... ...

Abstract	Severe asthma, chronic obstructive pulmonary disease (COPD) and idiopathic pulmonary fibrosis (IPF) are chronic lung diseases with a clear need for development of new and more efficient therapy. In preclinical research, the mouse model has been instrumental in advancing our knowledge of the biology and immunology. However, it has been proven rather difficult and time consuming to develop new treatments that can impact on the clinical course of these diseases. Many challenges need to be overcome for upgrading the quality of currently available experimental disease models in order to enhance the translation rate of basic research to clinical practice. Establishment of transgenic mouse overexpressing disease-causing genes may provide tools to discover new pathological pathways and to evaluate the possibility of molecular targeted therapy in chronic lung diseases. Personalized medicine, if developed, might be the solution for 'disease heterogeneity' and for improving clinical outcome.
MeSH term(s)	Animals ; Asthma/drug therapy ; Biomedical Research ; Chronic Disease ; Disease Models, Animal ; Drug Therapy, Combination ; Humans ; Idiopathic Pulmonary Fibrosis/drug therapy ; Mice ; Molecular Targeted Therapy ; Pulmonary Disease, Chronic Obstructive/drug therapy
Language	English
Publishing date	2015-01
Publishing country	Australia
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	1435849-9
ISSN	1440-1843 ; 1323-7799
ISSN (online)	1440-1843
ISSN	1323-7799
DOI	10.1111/resp.12438
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Zs.A 4957: Show issues

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Article ; Online: Potential for long-acting muscarinic antagonists in chronic obstructive pulmonary disease.

Joos, Guy F

Expert opinion on investigational drugs

2010 Volume 19, Issue 2, Page(s) 257–264

Abstract: Importance of the field: The prevention and relief of symptoms by regular use of bronchodilators is central to the pharmacological management of chronic obstructive pulmonary disease (COPD).: Areas covered in this review: The aim of this article is ... ...

Abstract	Importance of the field: The prevention and relief of symptoms by regular use of bronchodilators is central to the pharmacological management of chronic obstructive pulmonary disease (COPD). Areas covered in this review: The aim of this article is to review the effects of inhaled muscarinic antagonists in the treatment of stable COPD. What the reader will gain: An update of the clinical studies performed with the long-acting inhaled muscarinic antagonist (LAMA) tiotropium bromide in patients with COPD is given. In recent years, combinations of a LAMA and a long-acting inhaled beta2-agonist (LABA), and 'triple therapy' consisting of a LAMA, a LABA, and an inhaled steroid are being developed. Issues of safety of inhaled anticholinergics in COPD are discussed and a short overview of new LAMAs being developed for COPD is given. Take home messages: The importance of anticholinergic drug treatment in COPD was largely advanced by the development of the first LAMA, tiotropium bromide. The vast experience obtained with tiotropium bromide has paved the way for new LAMAs such as aclidinium bromide and glycopyrrolate (NVA-237).
MeSH term(s)	Administration, Inhalation ; Adrenergic beta-Agonists/administration & dosage ; Androstadienes/administration & dosage ; Bronchodilator Agents/administration & dosage ; Bronchodilator Agents/adverse effects ; Bronchodilator Agents/pharmacology ; Clinical Trials as Topic ; Delayed-Action Preparations/administration & dosage ; Delayed-Action Preparations/adverse effects ; Delayed-Action Preparations/pharmacology ; Drug Evaluation, Preclinical ; Drug Therapy, Combination/adverse effects ; Drugs, Investigational ; Fluticasone ; Humans ; Muscarinic Antagonists/administration & dosage ; Muscarinic Antagonists/adverse effects ; Muscarinic Antagonists/pharmacology ; Pulmonary Disease, Chronic Obstructive/drug therapy ; Receptors, Muscarinic/drug effects ; Scopolamine Derivatives/administration & dosage ; Scopolamine Derivatives/adverse effects ; Scopolamine Derivatives/pharmacology ; Tiotropium Bromide
Chemical Substances	Adrenergic beta-Agonists ; Androstadienes ; Bronchodilator Agents ; Delayed-Action Preparations ; Drugs, Investigational ; Muscarinic Antagonists ; Receptors, Muscarinic ; Scopolamine Derivatives ; Fluticasone (CUT2W21N7U) ; Tiotropium Bromide (XX112XZP0J)
Language	English
Publishing date	2010-02
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	1182884-5
ISSN	1744-7658 ; 0967-8298 ; 1354-3784
ISSN (online)	1744-7658
ISSN	0967-8298 ; 1354-3784
DOI	10.1517/13543780903505084
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Zs.A 3739: Show issues

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Article ; Online: Expression of ACE2, the SARS-CoV-2 Receptor, in Lung Tissue of Patients With Type 2 Diabetes.

Wijnant, Sara R A / Jacobs, Merel / Van Eeckhoutte, Hannelore P / Lapauw, Bruno / Joos, Guy F / Bracke, Ken R / Brusselle, Guy G

Diabetes

2020 Volume 69, Issue 12, Page(s) 2691–2699

Abstract: Increased expression of pulmonary ACE2, the SARS-CoV-2 receptor, could contribute to increased infectivity of COVID-19 in patients with diabetes, but ACE2 expression has not been studied in lung tissue of subjects with diabetes. We therefore studied ACE2 ...

Abstract	Increased expression of pulmonary ACE2, the SARS-CoV-2 receptor, could contribute to increased infectivity of COVID-19 in patients with diabetes, but ACE2 expression has not been studied in lung tissue of subjects with diabetes. We therefore studied ACE2 mRNA and protein expression in lung tissue samples of subjects with and without diabetes that were collected between 2002 and 2020 from patients undergoing lobectomy for lung tumors. For RT-PCR analyses, samples from 15 subjects with diabetes were compared with 91 randomly chosen control samples. For immunohistochemical staining, samples from 26 subjects with diabetes were compared with 66 randomly chosen control samples. mRNA expression of ACE2 was measured by quantitative RT-PCR. Protein levels of ACE2 were visualized by immunohistochemistry on paraffin-embedded lung tissue samples and quantified in alveolar and bronchial epithelium. Pulmonary ACE2 mRNA expression was not different between subjects with or without diabetes. In contrast, protein levels of ACE2 were significantly increased in both alveolar tissue and bronchial epithelium of patients with diabetes compared with control subjects, independent of smoking, chronic obstructive pulmonary disease, BMI, renin-angiotensin-aldosterone system inhibitor use, and other potential confounders. To conclude, we show increased bronchial and alveolar ACE2 protein expression in patients with diabetes. Further research is needed to elucidate whether upregulation of ACE2 expression in airways and lungs has consequences on infectivity and clinical outcomes of COVID-19.
MeSH term(s)	Angiotensin-Converting Enzyme 2/genetics ; Angiotensin-Converting Enzyme 2/metabolism ; COVID-19/complications ; COVID-19/virology ; Diabetes Mellitus, Type 2/complications ; Gene Expression Regulation ; Humans ; Lung/metabolism ; RNA, Messenger ; SARS-CoV-2
Chemical Substances	RNA, Messenger ; ACE2 protein, human (EC 3.4.17.23) ; Angiotensin-Converting Enzyme 2 (EC 3.4.17.23)
Keywords	covid19
Language	English
Publishing date	2020-10-06
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	80085-5
ISSN	1939-327X ; 0012-1797
ISSN (online)	1939-327X
ISSN	0012-1797
DOI	10.2337/db20-0669
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Article: Which patients with asthma could benefit from using anti-leukotriene drugs: an evidence based review.

Joos, Guy F

Polskie Archiwum Medycyny Wewnetrznej

2008 Volume 118, Issue 12, Page(s) 689–690

MeSH term(s)	Administration, Inhalation ; Anti-Asthmatic Agents/administration & dosage ; Anti-Asthmatic Agents/therapeutic use ; Asthma/drug therapy ; Dose-Response Relationship, Drug ; Evidence-Based Medicine ; Humans ; Leukotriene Antagonists/administration & dosage ; Leukotriene Antagonists/therapeutic use ; Treatment Outcome
Chemical Substances	Anti-Asthmatic Agents ; Leukotriene Antagonists
Language	English
Publishing date	2008-12
Publishing country	Poland
Document type	Editorial
ZDB-ID	123500-x
ISSN	1897-9483 ; 1897-9483 ; 0032-3772
ISSN (online)	1897-9483
ISSN	1897-9483 ; 0032-3772
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Zs.A 360: Show issues

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Article: Are beta2-agonists safe in patients with acute exacerbations of COPD?

Joos, Guy F

American journal of respiratory and critical care medicine

2007 Volume 176, Issue 4, Page(s) 322–323

MeSH term(s)	Adrenergic beta-Agonists/therapeutic use ; Albuterol/therapeutic use ; Cholinergic Antagonists/therapeutic use ; Humans ; Nebulizers and Vaporizers ; Pulmonary Disease, Chronic Obstructive/drug therapy ; Pulmonary Disease, Chronic Obstructive/physiopathology ; Pulmonary Gas Exchange/drug effects ; Pulmonary Gas Exchange/physiology
Chemical Substances	Adrenergic beta-Agonists ; Cholinergic Antagonists ; Albuterol (QF8SVZ843E)
Language	English
Publishing date	2007-08-15
Publishing country	United States
Document type	Comment ; Editorial
ZDB-ID	1180953-x
ISSN	1535-4970 ; 1073-449X ; 0003-0805
ISSN (online)	1535-4970
ISSN	1073-449X ; 0003-0805
DOI	10.1164/rccm.200704-643ED
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Article ; Online: MiR-223 is increased in lungs of patients with COPD and modulates cigarette smoke-induced pulmonary inflammation.

Roffel, Mirjam P / Maes, Tania / Brandsma, Corry-Anke / van den Berge, Maarten / Vanaudenaerde, Bart M / Joos, Guy F / Brusselle, Guy G / Heijink, Irene H / Bracke, Ken R

American journal of physiology. Lung cellular and molecular physiology

2021 Volume 321, Issue 6, Page(s) L1091–L1104

Abstract: Since microRNA (miR)-223-3p modulates inflammatory responses and chronic obstructive pulmonary disease (COPD) is associated with amplified pulmonary inflammation, we hypothesized that miR-223-3p plays a role in COPD pathogenesis. Expression of miR-223-3p ...

Abstract	Since microRNA (miR)-223-3p modulates inflammatory responses and chronic obstructive pulmonary disease (COPD) is associated with amplified pulmonary inflammation, we hypothesized that miR-223-3p plays a role in COPD pathogenesis. Expression of miR-223-3p was measured in lung tissue of two independent cohorts with patients with GOLD stage II-IV COPD, never smokers, and smokers without COPD. The functional role of miR-223-3p was studied in deficient mice and on overexpression in airway epithelial cells from COPD and controls. We observed higher miR-223-3p levels in patients with COPD stage II-IV compared with (non)-smoking controls, and levels were associated with higher neutrophil numbers in bronchial biopsies of patients with COPD. MiR-223-3p expression was also increased in lungs and bronchoalveolar lavage of cigarette smoke (CS)-exposed mice. CS-induced neutrophil and monocyte lung infiltration was stronger in miR-223-deficient mice on acute (5 days) exposure, but attenuated on subchronic (4 wk) exposure. Additionally, miR-223 deficiency attenuated acute and subchronic CS-induced lung infiltration of dendritic cells and T lymphocytes. Finally, in vitro overexpression of miR-223-3p in non-COPD airway epithelial cells suppressed C-X-C motif chemokine ligand 8 (CXCL8) and granulocyte monocyte-colony stimulation factor (GM-CSF) secretion and gene expression of the proinflammatory transcription factor
MeSH term(s)	Aged ; Animals ; Cigarette Smoking/adverse effects ; Cytokines/metabolism ; Female ; Humans ; Lung/drug effects ; Lung/metabolism ; Lung/pathology ; Male ; Mice ; Mice, Knockout ; MicroRNAs/genetics ; Middle Aged ; Neutrophils/drug effects ; Neutrophils/immunology ; Neutrophils/metabolism ; Pneumonia/chemically induced ; Pneumonia/genetics ; Pneumonia/metabolism ; Pneumonia/pathology ; Pulmonary Disease, Chronic Obstructive/genetics ; Pulmonary Disease, Chronic Obstructive/metabolism ; Pulmonary Disease, Chronic Obstructive/pathology
Chemical Substances	Cytokines ; MIRN223 microRNA, human ; MIRN223 microRNA, mouse ; MicroRNAs
Language	English
Publishing date	2021-10-20
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1013184-x
ISSN	1522-1504 ; 1040-0605
ISSN (online)	1522-1504
ISSN	1040-0605
DOI	10.1152/ajplung.00252.2021
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Uc I Zs.89: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular ab Jg. 2022: Lesesaal (EG)
Zs.A 2749: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)

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Article ; Online: Increased Expression of ACE2, the SARS-CoV-2 Entry Receptor, in Alveolar and Bronchial Epithelium of Smokers and Patients with COPD

Jacobs, Merel / Van Eeckhoutte, Hannelore P / Wijnant, Sara RA / Janssens, Wim / Joos, Guy F / Brusselle, Guy / Bracke, Ken

SSRN Electronic Journal ; ISSN 1556-5068

2020

Keywords	covid19
Language	English
Publisher	Elsevier BV
Publishing country	us
Document type	Article ; Online
DOI	10.2139/ssrn.3592674
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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