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Article ; Online: Spinal Cord Burst Stimulation vs Placebo Stimulation for Patients With Chronic Radicular Pain After Lumbar Spine Surgery.

Hunter, Corey W / Rosenow, Joshua / Russo, Marc

2023 Volume 329, Issue 10, Page(s) 847–848

MeSH term(s)	Humans ; Chronic Pain/etiology ; Chronic Pain/therapy ; Electric Stimulation Therapy ; Failed Back Surgery Syndrome/etiology ; Failed Back Surgery Syndrome/therapy ; Low Back Pain/etiology ; Low Back Pain/therapy ; Lumbar Vertebrae/surgery ; Radiculopathy/etiology ; Radiculopathy/therapy ; Spinal Cord
Language	English
Publishing date	2023-09-05
Publishing country	United States
Document type	Letter ; Comment
ZDB-ID	2958-0
ISSN	1538-3598 ; 0254-9077 ; 0002-9955 ; 0098-7484
ISSN (online)	1538-3598
ISSN	0254-9077 ; 0002-9955 ; 0098-7484
DOI	10.1001/jama.2022.24751
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Article ; Online: Initiation and Evolution of Early Onset Prostate Cancer.

Russo, Joshua W / Balk, Steven P

Cancer cell

2018 Volume 34, Issue 6, Page(s) 874–876

Abstract: In this issue of Cancer Cell, Gerhauser et al. analyze early-onset prostate cancers, showing roles for androgen receptor-driven rearrangements, an early APOBEC-driven mutational mechanism, and ESRP1 gene duplication. Through integration of whole-genome, ... ...

Abstract	In this issue of Cancer Cell, Gerhauser et al. analyze early-onset prostate cancers, showing roles for androgen receptor-driven rearrangements, an early APOBEC-driven mutational mechanism, and ESRP1 gene duplication. Through integration of whole-genome, transcriptome, and methylome data, they identify high-risk subgroups and develop an algorithm that may predict molecular evolution.
MeSH term(s)	Evolution, Molecular ; Humans ; Male ; Mutation ; Prostatic Neoplasms ; Receptors, Androgen/genetics ; Transcriptome
Chemical Substances	Receptors, Androgen
Language	English
Publishing date	2018-12-08
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, Non-P.H.S. ; Comment
ZDB-ID	2078448-X
ISSN	1878-3686 ; 1535-6108
ISSN (online)	1878-3686
ISSN	1535-6108
DOI	10.1016/j.ccell.2018.11.010
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Zs.A 5650: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)
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Article ; Online: Androgen Receptor Interaction with Mediator Complex Is Enhanced in Castration-Resistant Prostate Cancer by CDK7 Phosphorylation of MED1.

Russo, Joshua W / Nouri, Mannan / Balk, Steven P

Cancer discovery

2019 Volume 9, Issue 11, Page(s) 1490–1492

Abstract: In this issue ... ...

Abstract	In this issue of
MeSH term(s)	Cell Line, Tumor ; Drug Resistance, Neoplasm ; Humans ; Male ; Mediator Complex ; Mediator Complex Subunit 1 ; Phosphorylation ; Prostatic Neoplasms, Castration-Resistant ; Receptors, Androgen ; Signal Transduction
Chemical Substances	MED1 protein, human ; Mediator Complex ; Mediator Complex Subunit 1 ; Receptors, Androgen
Language	English
Publishing date	2019-10-30
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Comment
ZDB-ID	2625242-9
ISSN	2159-8290 ; 2159-8274
ISSN (online)	2159-8290
ISSN	2159-8274
DOI	10.1158/2159-8290.CD-19-1028
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Zs.A 6916: Show issues

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Article ; Online: Transient, Image-Guided Gel-dissection for Percutaneous Thermal Ablation.

Liu, Kathy / Russo, Mario / Ellis, Joshua S / Capua, John Di / Zhao, Sara / Kalva, Sanjeeva / Arellano, Ronald S / Irani, Zubin / Uppot, Raul / Linderman, Stephen W / Aizenberg, Joanna / Srinivasan, Shriya / Som, Avik

Advanced healthcare materials

2024 , Page(s) e2400272

Abstract: Image-guided tumor ablative therapies are mainstay cancer treatment options, but often require intra-procedural protective tissue displacement to reduce the risk of collateral damage to neighboring organs. Standard of care (SoC) strategies, such as ... ...

Abstract	Image-guided tumor ablative therapies are mainstay cancer treatment options, but often require intra-procedural protective tissue displacement to reduce the risk of collateral damage to neighboring organs. Standard of care (SoC) strategies, such as hydrodissection (fluidic injection), are limited by rapid diffusion of fluid and poor retention time, risking injury to adjacent organs, increased cancer recurrence rates from incomplete tumor ablations, and limited patient qualification. Herein, we developed "gel-dissection," a technique leveraging injectable hydrogels for longer-lasting, shapeable, and transient tissue separation, empowering clinicians with improved ablation operation windows and greater control. A rheological model was designed to understand and tune gel-dissection parameters. In swine models, gel-dissection achieved 24 times longer-lasting tissue separation dynamics compared to saline, with 40% less injected volume. Gel-dissection achieved anti-dependent dissection between free-floating organs in the peritoneal cavity and clinically significant thermal protection, with the potential to expand minimally invasive therapeutic techniques, especially across locoregional therapies including radiation, cryoablation, endoscopy, and surgery. This article is protected by copyright. All rights reserved.
Language	English
Publishing date	2024-04-28
Publishing country	Germany
Document type	Journal Article
ZDB-ID	2649576-4
ISSN	2192-2659 ; 2192-2640
ISSN (online)	2192-2659
ISSN	2192-2640
DOI	10.1002/adhm.202400272
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Zs.A 6966: Show issues

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Article: Increased chromatin accessibility drives transition to androgen receptor splice variant dependence in castration-resistant prostate cancer.

Poluben, Larysa / Nouri, Mannan / Liang, Jiaqian / Varkaris, Andreas / Ersoy-Fazlioglu, Betul / Voznesensky, Olga / Lee, Irene I / Qiu, Xintao / Cato, Laura / Seo, Ji-Heui / Freedman, Matthew L / Sowalsky, Adam G / Lack, Nathan A / Corey, Eva / Nelson, Peter S / Brown, Myles / Long, Henry W / Balk, Steven P / Russo, Joshua W

bioRxiv : the preprint server for biology

2024

Abstract: Androgen receptor (AR) splice variants, of which ARv7 is the most common, are increased in prostate cancer (PC) that develops resistance to androgen signaling inhibitor drugs, but the extent to which these variants drive AR activity, and whether they ... ...

Abstract	Androgen receptor (AR) splice variants, of which ARv7 is the most common, are increased in prostate cancer (PC) that develops resistance to androgen signaling inhibitor drugs, but the extent to which these variants drive AR activity, and whether they have novel functions or dependencies, remain to be determined. We generated a subline of VCaP PC cells (VCaP16) that is resistant to the AR inhibitor enzalutamide (ENZ) and found that AR activity was independent of the full-length AR (ARfl), despite its continued high-level expression, and was instead driven by ARv7. The ARv7 cistrome and transcriptome in VCaP16 cells mirrored that of the ARfl in VCaP cells, although ARv7 chromatin binding was weaker, and strong ARv7 binding sites correlated with higher affinity ARfl binding sites across multiple models and clinical samples. Notably, although ARv7 expression in VCaP cells increased rapidly in response to ENZ, there was a long lag before it gained chromatin binding and transcriptional activity. This lag was associated with an increase in chromatin accessibility, with the AR and nuclear factor I (NFI) motifs being most enriched at these more accessible sites. Moreover, the transcriptional effects of combined NFIB and NFIX knockdown versus ARv7 knockdown were highly correlated. These findings indicate that ARv7 can drive the AR program, but that its activity is dependent on adaptations that increase chromatin accessibility to enhance its intrinsically weak chromatin binding.
Language	English
Publishing date	2024-01-12
Publishing country	United States
Document type	Preprint
DOI	10.1101/2024.01.10.575110
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Article: CCN5 in alveolar epithelial proliferation and differentiation during neonatal lung oxygen injury.

Fiaturi, Najla / Russo, Joshua W / Nielsen, Heber C / Castellot, John J

Journal of cell communication and signaling

2018 Volume 12, Issue 1, Page(s) 217–229

Abstract: Lung immaturity is the major cause of morbidity and mortality in premature infants, especially those born <28 weeks of gestation. These infants are at high risk of developing respiratory distress syndrome (RDS), a lung disease caused by insufficient ... ...

Abstract	Lung immaturity is the major cause of morbidity and mortality in premature infants, especially those born <28 weeks of gestation. These infants are at high risk of developing respiratory distress syndrome (RDS), a lung disease caused by insufficient surfactant production and immaturity of saccular/alveolar type II epithelial cells in the lung. RDS treatment includes oxygen and respiratory support that improve survival but also increase the risk for bronchopulmonary dysplasia (BPD), a chronic lung disease characterized by arrested alveolarization, airway hyperreactivity, and pulmonary hypertension. The mechanisms regulating normal alveolar development and how injury disrupts normal development to cause BPD are not well understood. We examined the role of the matricellular protein CCN5 (Cysteine-rich protein 61/Connective tissue growth factor/Nephroblastoma-overexpressed protein) in the development of BPD. Cultured non-proliferating alveolar type II cells expressed low levels of CCN5 protein, and displayed higher levels during proliferation. siRNA targeting of CCN5 reduced alveolar type II cell proliferation and migration in cell culture. In a mouse model of hyperoxia-induced BPD, CCN5 protein was increased only in proliferating alveolar type I cells. Alveolar epithelial cells co-expressing markers of type II cells and type I cells also appeared. The results suggest that hyperoxic injury in immature lungs induces proliferation of type I cells and trans-differentiation of type II cells into type I cells. We propose that the mechanism of the injury response in BPD includes CCN5 expression. Study of CCN5 in neonatal alveolar injury will further our understanding of BPD pathophysiology while providing a mechanistic foundation for therapeutic approaches.
Language	English
Publishing date	2018-01-18
Publishing country	Netherlands
Document type	Journal Article
ZDB-ID	2299380-0
ISSN	1873-961X ; 1873-9601
ISSN (online)	1873-961X
ISSN	1873-9601
DOI	10.1007/s12079-017-0443-1
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Article ; Online: The underappreciated diversity of bile acid modifications.

Mohanty, Ipsita / Mannochio-Russo, Helena / Schweer, Joshua V / El Abiead, Yasin / Bittremieux, Wout / Xing, Shipei / Schmid, Robin / Zuffa, Simone / Vasquez, Felipe / Muti, Valentina B / Zemlin, Jasmine / Tovar-Herrera, Omar E / Moraïs, Sarah / Desai, Dhimant / Amin, Shantu / Koo, Imhoi / Turck, Christoph W / Mizrahi, Itzhak / Kris-Etherton, Penny M /

Petersen, Kristina S / Fleming, Jennifer A / Huan, Tao / Patterson, Andrew D / Siegel, Dionicio / Hagey, Lee R / Wang, Mingxun / Aron, Allegra T / Dorrestein, Pieter C

Cell

2024 Volume 187, Issue 7, Page(s) 1801–1818.e20

Abstract: The repertoire of modifications to bile acids and related steroidal lipids by host and microbial metabolism remains incompletely characterized. To address this knowledge gap, we created a reusable resource of tandem mass spectrometry (MS/MS) spectra by ... ...

Abstract	The repertoire of modifications to bile acids and related steroidal lipids by host and microbial metabolism remains incompletely characterized. To address this knowledge gap, we created a reusable resource of tandem mass spectrometry (MS/MS) spectra by filtering 1.2 billion publicly available MS/MS spectra for bile-acid-selective ion patterns. Thousands of modifications are distributed throughout animal and human bodies as well as microbial cultures. We employed this MS/MS library to identify polyamine bile amidates, prevalent in carnivores. They are present in humans, and their levels alter with a diet change from a Mediterranean to a typical American diet. This work highlights the existence of many more bile acid modifications than previously recognized and the value of leveraging public large-scale untargeted metabolomics data to discover metabolites. The availability of a modification-centric bile acid MS/MS library will inform future studies investigating bile acid roles in health and disease.
MeSH term(s)	Animals ; Humans ; Bile Acids and Salts/chemistry ; Metabolomics/methods ; Polyamines ; Tandem Mass Spectrometry/methods ; Gastrointestinal Microbiome ; Databases, Chemical
Chemical Substances	Bile Acids and Salts ; Polyamines
Language	English
Publishing date	2024-03-11
Publishing country	United States
Document type	Journal Article
ZDB-ID	187009-9
ISSN	1097-4172 ; 0092-8674
ISSN (online)	1097-4172
ISSN	0092-8674
DOI	10.1016/j.cell.2024.02.019
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Zs.A 1167: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (1.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MG 58: Show issues

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Article ; Online: Androgen receptor splice variant 7 functions independently of the full length receptor in prostate cancer cells.

Liang, Jiaqian / Wang, Liyang / Poluben, Larysa / Nouri, Mannan / Arai, Seiji / Xie, Lisha / Voznesensky, Olga S / Cato, Laura / Yuan, Xin / Russo, Joshua W / Long, Henry W / Brown, Myles / Chen, Shaoyong / Balk, Steven P

Cancer letters

2021 Volume 519, Page(s) 172–184

Abstract: One mechanism for reactivation of androgen receptor (AR) activity after androgen deprivation therapy in castration-resistant prostate cancer (CRPC) is expression of splice variants such as ARv7 that delete the ligand binding domain and have constitutive ... ...

Abstract	One mechanism for reactivation of androgen receptor (AR) activity after androgen deprivation therapy in castration-resistant prostate cancer (CRPC) is expression of splice variants such as ARv7 that delete the ligand binding domain and have constitutive activity. Exogenous overexpressed ARv7 can function as a homodimer or heterodimer with full length AR (ARfl), which is highly expressed with ARv7 in CRPC. However, the extent to which endogenous ARv7 function is dependent on heterodimerization with ARfl remains to be determined. We used double-crosslinking to stabilize AR complexes on chromatin in a CRPC cell line expressing endogenous ARfl and ARv7 (LN95 cells), and established that only trace levels of ARfl were associated with ARv7 on chromatin. Consistent with this result, depletion of ARfl with an AR degrader targeting the AR ligand binding domain did not decrease ARv7 binding to chromatin or its association with HOXB13, but did decrease overall AR transcriptional activity. Comparable results were obtained in CWR22RV1 cells, another CRPC cell line expressing ARfl and ARv7. These results indicate that ARv7 function in CRPC is not dependent on ARfl, and that both contribute independently to overall AR activity.
MeSH term(s)	Cell Line ; Cell Line, Tumor ; Chromatin/genetics ; HEK293 Cells ; Homeodomain Proteins/genetics ; Humans ; Ligands ; Male ; Prostate/metabolism ; Prostatic Neoplasms/genetics ; Protein Domains/genetics ; Receptors, Androgen/genetics
Chemical Substances	Chromatin ; Homeodomain Proteins ; Ligands ; Receptors, Androgen
Language	English
Publishing date	2021-07-10
Publishing country	Ireland
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	195674-7
ISSN	1872-7980 ; 0304-3835
ISSN (online)	1872-7980
ISSN	0304-3835
DOI	10.1016/j.canlet.2021.07.013
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Article ; Online: Metastatic Castration-Resistant Prostate Cancer Remains Dependent on Oncogenic Drivers Found in Primary Tumors.

Einstein, David J / Arai, Seiji / Calagua, Carla / Xie, Fang / Voznesensky, Olga / Capaldo, Brian J / Luffman, Christina / Hecht, Jonathan L / Balk, Steven P / Sowalsky, Adam G / Russo, Joshua W

JCO precision oncology

2021 Volume 5

Abstract: Metastatic prostate cancer is initially sensitive to androgen receptor inhibition, but eventually becomes castration-resistant prostate cancer (mCRPC). Early use of more intensive therapies targeting androgen receptor and other oncogenic drivers in ... ...

Abstract	Metastatic prostate cancer is initially sensitive to androgen receptor inhibition, but eventually becomes castration-resistant prostate cancer (mCRPC). Early use of more intensive therapies targeting androgen receptor and other oncogenic drivers in treatment-naïve primary prostate cancer (PC) may be more effective than that in advanced mCRPC. However, analysis of primary tumors may not reveal targetable metastatic drivers that are subclonal in the primary tumor or acquired at metastatic sites. Methods: PC samples spanning one patient's clinical course: diagnostic biopsies, pre- or post-enzalutamide metastatic biopsies, and rapid autopsy samples including a patient-derived xenograft (PDX) were analyzed by targeted exome sequencing followed by phylogenetic analysis. Results: Left- and right-lobe primary PC tumors appeared to diverge, with the right acquiring additional shared mutations and striking differences in copy number alterations that later appeared in metastatic samples during the treatment course and at autopsy, whereas the left base tumor maintained a quiet copy number alteration landscape and partitioned into a dead-end node. Conclusion: These findings indicate that truncal alterations identified in primary PC can drive advanced mCRPC, even in the presence of additional strong oncogenic drivers (ie,
MeSH term(s)	Biopsy ; Carcinogenesis ; Humans ; Male ; Phylogeny ; Prostatic Neoplasms, Castration-Resistant/genetics
Language	English
Publishing date	2021-09-16
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, Non-P.H.S.
ISSN	2473-4284
ISSN (online)	2473-4284
DOI	10.1200/PO.21.00059
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Article ; Online: Prostate cancer research in the 21st century; report from the 2021 Coffey-Holden prostate cancer academy meeting.

Miyahira, Andrea K / Zarif, Jelani C / Coombs, Catherine C / Flavell, Robert R / Russo, Joshua W / Zaidi, Samir / Zhao, Di / Zhao, Shuang G / Pienta, Kenneth J / Soule, Howard R

The Prostate

2021 Volume 82, Issue 2, Page(s) 169–181

Abstract: Introduction: The 2021 Coffey-Holden Prostate Cancer Academy (CHPCA) Meeting, "Prostate Cancer Research in the 21st Century," was held virtually, from June 24-25, 2021.: Methods: The CHPCA Meeting is organized by the Prostate Cancer Foundation as a ... ...

Abstract	Introduction: The 2021 Coffey-Holden Prostate Cancer Academy (CHPCA) Meeting, "Prostate Cancer Research in the 21st Century," was held virtually, from June 24-25, 2021. Methods: The CHPCA Meeting is organized by the Prostate Cancer Foundation as a unique discussion-oriented meeting focusing on critical topics in prostate cancer research envisioned to bridge the next major advances in prostate cancer biology and treatment. The 2021 CHPCA Meeting was virtually attended by 89 investigators and included 31 talks over nine sessions. Results: Major topic areas discussed at the meeting included: cancer genomics and sequencing, functional genomic approaches to studying mediators of plasticity, emerging signaling pathways in metastatic castration resistant prostate cancer, Wnt signaling biology and the challenges of targeted therapy, clonal hematopoiesis, neuroendocrine cell plasticity and antitumor immunity, cancer immunotherapy and its synergizers, and imaging the tumor microenvironment and metabolism. Discussion: This meeting report summarizes the research presented at the 2021 CHPCA Meeting. We hope that publication of this knowledge will accelerate new understandings and the development of new biomarkers and treatments for prostate cancer.
MeSH term(s)	Congresses as Topic ; Humans ; Immunotherapy/methods ; Male ; Prostate/diagnostic imaging ; Prostate/immunology ; Prostate/metabolism ; Prostatic Neoplasms/genetics ; Prostatic Neoplasms/pathology ; Prostatic Neoplasms/therapy ; Research/trends
Language	English
Publishing date	2021-11-03
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, U.S. Gov't, Non-P.H.S. ; Review
ZDB-ID	604707-5
ISSN	1097-0045 ; 0270-4137
ISSN (online)	1097-0045
ISSN	0270-4137
DOI	10.1002/pros.24262
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