LIVIVO - Search results -

Search results

Result 1 - 10 of total 22

Search options

Article: CO2 emissions and energy technologies in Western Europe.

Barrera-Santana, J / Marrero, Gustavo A / Puch, Luis A / Díaz, Antonia

SERIEs : journal of the Spanish Economic Association

2021 Volume 12, Issue 2, Page(s) 105–150

Abstract: In this paper, we investigate the path to the green transition in Europe. In so doing, we implement an empirical model of dynamic panel data on a sample of sixteen Western European countries over the period 1980 to 2019. The model is consistent with ... ...

Abstract	In this paper, we investigate the path to the green transition in Europe. In so doing, we implement an empirical model of dynamic panel data on a sample of sixteen Western European countries over the period 1980 to 2019. The model is consistent with various features of neoclassical growth theory incorporating energy use. Our focus is on the short-run determinants of carbon emissions within that set of countries. We provide evidence that the relationship between economic activity and CO2 emissions is strong in economies where economic booms depend on energy-intensive sectors. Also, the mitigating role of renewable energy technologies is key when energy intensity rebounds. These circumstances may constitute a challenge for the climate transition goals targeted in the EU's Recovery Plan, whose main objective at this very moment is to mitigate the economic and social impact of the coronavirus pandemic.
Language	English
Publishing date	2021-05-25
Publishing country	Germany
Document type	Journal Article
ZDB-ID	2536381-5
ISSN	1869-4195 ; 1869-4187
ISSN (online)	1869-4195
ISSN	1869-4187
DOI	10.1007/s13209-021-00234-8
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

This service is chargeable due to the Delivery terms set by subito. Orders including an article and supplementary material will be classified as separate orders. In these cases, fees will be demanded for each order.

Book ; Article ; Online: CO2 emissions and energy technologies in Western Europe

Barrera-Santana, J. / Marrero, Gustavo A. / Puch, Luis / Díaz, Antonia

2021

Abstract: In this paper we investigate the path to the green transition in Europe. In so doing, we implement an empirical model of dynamic panel data on a sample of sixteen Western European countries over the period 1980 to 2019. The model is consistent with ... ...

Abstract	In this paper we investigate the path to the green transition in Europe. In so doing, we implement an empirical model of dynamic panel data on a sample of sixteen Western European countries over the period 1980 to 2019. The model is consistent with various features of neo-classical growth theory incorporating energy use. Our focus is on the short-run determinants of carbon emissions within that set of countries. We provide evidence that the relationship between economic activity and CO2 emissions is strong in economies where economic booms depend on energy-intensive sectors. Also, the mitigating role of renewable energy technologies is key when energy intensity rebounds. These circumstances may constitute a challenge for the climate transition goals targeted in the EU's Recovery Plan, whose main objective at this very moment is to mitigate the economic and social impact of the coronavirus pandemic.
Keywords	ddc:330 ; C23 ; Q43 ; Q5 ; CO2 Emissions ; Energy ; Business Cycles ; Panel Data
Subject code	339
Language	English
Publisher	Munich: ifo Institute - Leibniz Institute for Economic Research at the University of Munich
Publishing country	de
Document type	Book ; Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

Full text online

Full text

Inter-library loan at ZB MED

Your chosen title can be delivered directly to ZB MED Cologne location if you are registered as a user at ZB MED Cologne.

Article ; Online: CO2 emissions and energy technologies in Western Europe

Barrera-Santana, J. / Marrero, Gustavo A. / Puch, Luis / Díaz, Antonia

2021

Abstract	In this paper, we investigate the path to the green transition in Europe. In so doing, we implement an empirical model of dynamic panel data on a sample of sixteen Western European countries over the period 1980 to 2019. The model is consistent with various features of neoclassical growth theory incorporating energy use. Our focus is on the short-run determinants of carbon emissions within that set of countries. We provide evidence that the relationship between economic activity and CO2 emissions is strong in economies where economic booms depend on energy-intensive sectors. Also, the mitigating role of renewable energy technologies is key when energy intensity rebounds. These circumstances may constitute a challenge for the climate transition goals targeted in the EU's Recovery Plan, whose main objective at this very moment is to mitigate the economic and social impact of the coronavirus pandemic.
Keywords	ddc:330 ; C23 ; Q43 ; Q5 ; CO2 emissions ; Energy ; Business cycles ; Panel data
Subject code	339
Language	English
Publisher	Heidelberg: Springer
Publishing country	de
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

Full text online

Full text

Inter-library loan at ZB MED

Your chosen title can be delivered directly to ZB MED Cologne location if you are registered as a user at ZB MED Cologne.

Article ; Online: CRISPRi-induced transcriptional regulation of IAH1 gene and its influence on volatile compounds profile in Kluyveromyces marxianus DU3.

Muñoz-Miranda, Luis A / Zepeda-Peña, Andrea Catalina / Casas-Godoy, Leticia / Pereira-Santana, Alejandro / Méndez-Zamora, Andrés / Barrera-Martínez, Iliana / Rodríguez-Zapata, Luis / Gschaedler-Mathis, Anne Christine / Figueroa-Yáñez, Luis J

World journal of microbiology & biotechnology

2024 Volume 40, Issue 4, Page(s) 121

Abstract: Mezcal is a traditional Mexican distilled beverage, known for its marked organoleptic profile, which is influenced by several factors, such as the fermentation process, where a wide variety of microorganisms are present. Kluyveromyces marxianus is one of ...

Abstract	Mezcal is a traditional Mexican distilled beverage, known for its marked organoleptic profile, which is influenced by several factors, such as the fermentation process, where a wide variety of microorganisms are present. Kluyveromyces marxianus is one of the main yeasts isolated from mezcal fermentations and has been associated with ester synthesis, contributing to the flavors and aromas of the beverage. In this study, we employed CRISPR interference (CRISPRi) technology, using dCas9 fused to the Mxi1 repressor factor domain, to down-regulate the expression of the IAH1 gene, encoding for an isoamyl acetate-hydrolyzing esterase, in K. marxianus strain DU3. The constructed CRISPRi plasmid successfully targeted the IAH1 gene, allowing for specific gene expression modulation. Through gene expression analysis, we assessed the impact of IAH1 down-regulation on the metabolic profile of volatile compounds. We also measured the expression of other genes involved in volatile compound biosynthesis, including ATF1, EAT1, ADH1, and ZWF1 by RT-qPCR. Results demonstrated successful down-regulation of IAH1 expression in K. marxianus strain DU3 using the CRISPRi system. The modulation of IAH1 gene expression resulted in alterations in the production of volatile compounds, specifically ethyl acetate, which are important contributors to the beverage's aroma. Changes in the expression levels of other genes involved in ester biosynthesis, suggesting that the knockdown of IAH1 may generate intracellular alterations in the balance of these metabolites, triggering a regulatory response. The application of CRISPRi technology in K. marxianus opens the possibility of targeted modulation of gene expression, metabolic engineering strategies, and synthetic biology in this yeast strain.
MeSH term(s)	Clustered Regularly Interspaced Short Palindromic Repeats ; Gene Expression Regulation ; Kluyveromyces/genetics ; Esters
Chemical Substances	Esters
Language	English
Publishing date	2024-03-05
Publishing country	Germany
Document type	Journal Article
ZDB-ID	1499109-3
ISSN	1573-0972 ; 0959-3993
ISSN (online)	1573-0972
ISSN	0959-3993
DOI	10.1007/s11274-023-03811-0
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

Article ; Online: How do we deliver our findings? Analysis of podium presentations at shoulder meetings.

Miquel, Joan / Santana, Fernando / Barrera, Albert / Torrens, Carlos

Journal of orthopaedic surgery and research

2018 Volume 13, Issue 1, Page(s) 234

Abstract: Background: The aim of the present study was to evaluate the time structure of oral presentations delivered at three shoulder congresses: shoulder sessions at the American Academy of Orthopaedic Surgeons (AAOS) Meeting, European Foundation of National ... ...

Abstract	Background: The aim of the present study was to evaluate the time structure of oral presentations delivered at three shoulder congresses: shoulder sessions at the American Academy of Orthopaedic Surgeons (AAOS) Meeting, European Foundation of National Associations of Orthopaedics and Traumatology (EFORT) Congress, and International Congress of Shoulder and Elbow Surgery (ICSES). Methods: A total of 160 oral presentations at the 2016 AAOS Annual Meeting, 17th EFORT Congress, and 13th ICSES were included. Podium presentations were categorized by topic, congress, inclusion of video support, and nationality of the speaker. Total time and time dedicated to each section of the presentation (introduction, methods, results, discussion and conclusions) were collected for all podium presentations. Results: Approximately 34% of speakers exceeded time constraints. No differences were found in the times that presenters used for the introduction, methods, results, and conclusions sections (p > 0.05). However, when extended introductions were delivered, the results and conclusions sections were shortened (r = - 0.2 and r = - 0.21, respectively). Inclusion of video support tended to result in exceedance of time limits (p < 0.01). Conclusions: One third of the shoulder surgeons exceeded time constraints in their conference presentations, and no distinctions were found in time allocations for different sections of the presentations. Longer introductions may lead to time restriction in the results and conclusions sections.
MeSH term(s)	Congresses as Topic/standards ; Joint Diseases/surgery ; Orthopedics ; Shoulder Joint/surgery ; Time Factors
Language	English
Publishing date	2018-09-14
Publishing country	England
Document type	Journal Article
ISSN	1749-799X
ISSN (online)	1749-799X
DOI	10.1186/s13018-018-0942-7
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

Inter-library loan at ZB MED

Your chosen title can be delivered directly to ZB MED Cologne location if you are registered as a user at ZB MED Cologne.

Article ; Online: Quinolinic Acid Induces Alterations in Neuronal Subcellular Compartments, Blocks Autophagy Flux and Activates Necroptosis and Apoptosis in Rat Striatum.

Silva-Islas, Carlos Alfredo / Santana-Martínez, Ricardo Alberto / León-Contreras, Juan Carlos / Barrera-Oviedo, Diana / Pedraza-Chaverri, Jose / Hernández-Pando, Rogelio / Maldonado, Perla D

Molecular neurobiology

2022 Volume 59, Issue 11, Page(s) 6632–6651

Abstract: Quinolinic acid (QUIN) is an agonist of N-methyl-D-aspartate receptor (NMDAr) used to study the underlying mechanism of excitotoxicity in animal models. There is evidence indicating that impairment in autophagy at early times contributes to cellular ... ...

Abstract	Quinolinic acid (QUIN) is an agonist of N-methyl-D-aspartate receptor (NMDAr) used to study the underlying mechanism of excitotoxicity in animal models. There is evidence indicating that impairment in autophagy at early times contributes to cellular damage in excitotoxicity; however, the status of autophagy in QUIN model on day 7 remains unexplored. In this study, the ultrastructural analysis of subcellular compartments and the status of autophagy, necroptosis, and apoptosis in the striatum of rats administered with QUIN (120 nmol and 240 nmol) was performed on day 7. QUIN induced circling behavior, neurodegeneration, and cellular damage; also, it promoted swollen mitochondrial crests, spherical-like morphology, and mitochondrial fragmentation; decreased ribosomal density in the rough endoplasmic reticulum; and altered the continuity of myelin sheaths in axons with separation of the compact lamellae. Furthermore, QUIN induced an increase and a decrease in ULK1 and p-70-S6K phosphorylation, respectively, suggesting autophagy activation; however, the increased microtubule-associated protein 1A/1B-light chain 3-II (LC3-II) and sequestosome-1/p62 (SQSTM1/p62), the coexistence of p62 and LC3 in the same structures, and the decrease in Beclin 1 and mature cathepsin D also indicates a blockage in autophagy flux. Additionally, QUIN administration increased tumor necrosis factor alpha (TNFα) and receptor-interacting protein kinase 3 (RIPK3) levels and its phosphorylation (p-RIPK3), as well as decreased B-cell lymphoma 2 (Bcl-2) and increased Bcl-2-associated X protein (Bax) levels and c-Jun N-terminal kinase (JNK) phosphorylation, suggesting an activation of necroptosis and apoptosis, respectively. These results suggest that QUIN activates the autophagy, but on day 7, it is blocked and organelle and cellular damage, neurodegeneration, and behavior alterations could be caused by necroptosis and apoptosis activation.
MeSH term(s)	Animals ; Apoptosis/physiology ; Autophagy/physiology ; Beclin-1/metabolism ; Cathepsin D/metabolism ; JNK Mitogen-Activated Protein Kinases/metabolism ; Lysosomes/metabolism ; Microtubule-Associated Proteins/metabolism ; Necroptosis ; Quinolinic Acid/toxicity ; Rats ; Receptors, N-Methyl-D-Aspartate/metabolism ; Sequestosome-1 Protein/metabolism ; Tumor Necrosis Factor-alpha/metabolism ; bcl-2-Associated X Protein/metabolism
Chemical Substances	Beclin-1 ; Microtubule-Associated Proteins ; Receptors, N-Methyl-D-Aspartate ; Sequestosome-1 Protein ; Tumor Necrosis Factor-alpha ; bcl-2-Associated X Protein ; JNK Mitogen-Activated Protein Kinases (EC 2.7.11.24) ; Cathepsin D (EC 3.4.23.5) ; Quinolinic Acid (F6F0HK1URN)
Language	English
Publishing date	2022-08-18
Publishing country	United States
Document type	Journal Article
ZDB-ID	645020-9
ISSN	1559-1182 ; 0893-7648
ISSN (online)	1559-1182
ISSN	0893-7648
DOI	10.1007/s12035-022-02986-1
Database	MEDical Literature Analysis and Retrieval System OnLINE

Full text online

Accessible to users with ZB MED library card

In stock of ZB MED Cologne/Königswinter

Zs.A 2411: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (2.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾

Article ; Online: Sustained Activation of JNK Induced by Quinolinic Acid Alters the BDNF/TrkB Axis in the Rat Striatum.

Santana-Martínez, Ricardo A / León-Contreras, Juan Carlos / Barrera-Oviedo, Diana / Pedraza-Chaverri, José / Hernández-Pando, Rogelio / Maldonado, Perla D

Neuroscience

2018 Volume 383, Page(s) 22–32

Abstract: Oxidative stress secondary to excitotoxicity is a common factor in the physiopathology of a variety of neurological disorders. In response to oxidative stress, several signaling pathways, such as MAPK, are activated or inactivated. Mitogen-activated ... ...

Abstract	Oxidative stress secondary to excitotoxicity is a common factor in the physiopathology of a variety of neurological disorders. In response to oxidative stress, several signaling pathways, such as MAPK, are activated or inactivated. Mitogen-activated protein kinase (MAPK) family activation must be finely regulated in time and intensity, as this pathway may either preserve cell survival or promote cell death. In the present study, the activation of MAPK in the excitotoxic injury induced by quinolinic acid (QUIN) was examined in vivo, at short and long times. We used different doses (30, 60, 120 and 240 nmol) of QUIN injected intrastriatally in the right rat striatum and the effect of this treatment on motor deficits, cellular damage, MAPK activation and BDNF/TrkB axis, were evaluated at 2 h and 7 days post-lesion. Higher doses of QUIN (120 and 240 nmol) induced rat motor deficits and caused morphological changes in neurons around the lesion core. QUIN decreased the activation of ERK1/2 in a dose-dependent manner at 7 days post-injection, and induced a sustained increase of c-Jun NH2-terminal kinase (JNK) activation from 2 h to 7 days post-injury. JNK activation was dependent on the QUIN-induced NMDAr activation (only 120 nmol). No significant difference in p38 activation with QUIN was observed. QUIN (120 and 240 nmol) decreased BDNF/TrkB levels at 7 days post-injury. JNK inhibition (by an intracerebroventricular injection of SP600125) prevented the QUIN-induced reduction in BDNF and TrkB at 7 day post-injury, suggesting a role for the QUIN-induced JNK activation on the observed decrease in BDNF levels.
MeSH term(s)	Animals ; Brain-Derived Neurotrophic Factor/metabolism ; Corpus Striatum/drug effects ; Corpus Striatum/metabolism ; Enzyme Activation/drug effects ; MAP Kinase Signaling System/drug effects ; MAP Kinase Signaling System/physiology ; Male ; Oxidative Stress/physiology ; Quinolinic Acid/toxicity ; Rats ; Rats, Wistar ; Receptor, trkB/metabolism ; Signal Transduction/drug effects ; Signal Transduction/physiology
Chemical Substances	Bdnf protein, rat ; Brain-Derived Neurotrophic Factor ; Ntrk2 protein, rat (EC 2.7.10.1) ; Receptor, trkB (EC 2.7.10.1) ; Quinolinic Acid (F6F0HK1URN)
Language	English
Publishing date	2018-05-03
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	196739-3
ISSN	1873-7544 ; 0306-4522
ISSN (online)	1873-7544
ISSN	0306-4522
DOI	10.1016/j.neuroscience.2018.04.034
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Zs.A 1215: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (1.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article ; Online: Enhancing smoking cessation in Mexico using an e-Health tool in primary healthcare.

Ponciano-Rodríguez, Guadalupe / Reynales-Shigematsu, Luz Myriam / Rodríguez-Bolaños, Rosibel / Pruñonosa-Santana, Javier / Cartujano-Barrera, Francisco / Cupertino, Ana Paula

Salud publica de Mexico

2018 Volume 60, Issue 5, Page(s) 549–558

Abstract: Objective: To evaluate an e-Health tool designed to enhance smoking cessation in Mexico in primary healthcare.: Materials and methods: Smokers 18 years of age and older were recruited in the waiting room of two primary healthcare clinics in Mexico ... ...

Abstract	Objective: To evaluate an e-Health tool designed to enhance smoking cessation in Mexico in primary healthcare. Materials and methods: Smokers 18 years of age and older were recruited in the waiting room of two primary healthcare clinics in Mexico City. Participants used an eHealth smoking cessation tool that included smoking-related assessments, education on pharmacotherapy, and motivational videos. A follow-up assessment was conducted at 12 weeks week on smoking status. Logistic regression models were performed to identify factors associated with smoking cessation or consumption reduction. Results: A total of 132 smokers were enrolled in the study. At follow-up, 23.5% of participants self-reported smoking cessation. Among those who did not quit smoking, 65.0% decreased the number of cigarettes. Factors associated significantly with smoking cessation were: being a non-daily smoker, being interested in quitting smoking, having low level of physical dependence, and participating in cessation treatment. Conclusions: The e-Health tool produced a high rate of smoking cessation. Better outcomes are obtained when this tool is used with conventional cessation programs.
MeSH term(s)	Adolescent ; Adult ; Aged ; Female ; Humans ; Male ; Mexico ; Middle Aged ; Primary Health Care ; Smoking Cessation/methods ; Telemedicine ; Young Adult
Language	English
Publishing date	2018-12-14
Publishing country	Mexico
Document type	Journal Article
ZDB-ID	954220-6
ISSN	1606-7916 ; 0036-3634
ISSN (online)	1606-7916
ISSN	0036-3634
DOI	10.21149/9348
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Zs.B 1309: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

Article: The Therapeutic Effect of Curcumin in Quinolinic Acid-Induced Neurotoxicity in Rats is Associated with BDNF, ERK1/2, Nrf2, and Antioxidant Enzymes.

Santana-Martínez, Ricardo A / Silva-Islas, Carlos A / Fernández-Orihuela, Yessica Y / Barrera-Oviedo, Diana / Pedraza-Chaverri, José / Hernández-Pando, Rogelio / Maldonado, Perla D

Antioxidants (Basel, Switzerland)

2019 Volume 8, Issue 9

Abstract: In the present study we investigated the participation of brain-derived neurotropic factor (BDNF) on the activation of the mitogen activated protein kinase (MAPK) protein extracellular signal-regulated kinase-1/2 (ERK1/2) as a mechanism of curcumin (CUR) ...

Abstract	In the present study we investigated the participation of brain-derived neurotropic factor (BDNF) on the activation of the mitogen activated protein kinase (MAPK) protein extracellular signal-regulated kinase-1/2 (ERK1/2) as a mechanism of curcumin (CUR) to provide an antioxidant defense system mediated by the nuclear factor erythroid 2-related factor 2 (Nrf2) in the neurotoxic model induced by quinolinic acid (QUIN). Wistar rats received CUR (400 mg/kg, intragastrically) for 6 days after intrastriatal injection with QUIN (240 nmol). CUR improved the motor deficit and morphological alterations induced by QUIN and restored BDNF, ERK1/2, and Nrf2 levels. CUR treatment avoided the decrease in the protein levels of glutathione peroxidase (GPx), glutathione reductase (GR), γ-glutamylcysteine ligase (γ-GCL), and glutathione (GSH) levels. Only, the QUIN-induced decrease in the GR activity was prevented by CUR treatment. Finally, QUIN increased superoxide dismutase 2 (SOD2) and catalase (CAT) levels, and the γGCL and CAT activities; however, this increase was major in the QUIN+CUR group for γ-GCL, CAT, and SOD activities. These data suggest that the therapeutic effect of CUR could involve BDNF action on the activation of ERK1/2 to induce increased levels of protein and enzyme activity of antioxidant proteins regulated by Nrf2 and GSH levels.
Language	English
Publishing date	2019-09-11
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2704216-9
ISSN	2076-3921
ISSN	2076-3921
DOI	10.3390/antiox8090388
Database	MEDical Literature Analysis and Retrieval System OnLINE

Order via subito

Article ; Online: Apocynin protects against neurological damage induced by quinolinic acid by an increase in glutathione synthesis and Nrf2 levels.

Cruz-Álvarez, Silvia / Santana-Martínez, Ricardo / Avila-Chávez, Euclides / Barrera-Oviedo, Diana / Hernández-Pando, Rogelio / Pedraza-Chaverri, José / Maldonado, Perla D

Neuroscience

2017 Volume 350, Page(s) 65–74

Abstract: Apocynin (APO) is a well-known NADPH oxidase (NOX) inhibitor. However, several studies have reported its ability to increase glutathione (GSH) levels. Due to GSH is a major non-enzymatic antioxidant in brain, the aim of this study was to evaluate, in the ...

Abstract	Apocynin (APO) is a well-known NADPH oxidase (NOX) inhibitor. However, several studies have reported its ability to increase glutathione (GSH) levels. Due to GSH is a major non-enzymatic antioxidant in brain, the aim of this study was to evaluate, in the striatum of control and quinolinic acid (QUIN) injected rats, the effect of APO administration on: (1) GSH levels, (2) activity of some enzymes involved in the GSH metabolism, and (3) nuclear factor erythroid-2-related factor 2 (Nrf2) mRNA levels. Animals received QUIN 240nmol in right striatum and APO (5mg/kg, i.p.), 30min before and 60min after intrastriatal injection. APO treatment prevented the QUIN-induced histological damage to the striatum. In control rats, APO treatment increased GSH and Nrf2 mRNA levels and the activities of gamma-glutamylcysteine ligase (γ-GCL), glutathione-S-transferase (GST) and glutathione peroxidase (GPx). On the other hand, APO treatment prevented the QUIN-induced decrease in GSH and Nrf2 levels, and in γ-GCL and GPx activities. These data indicate that APO is able to increase GSH levels and the activity of proteins involved in its metabolism, which could be associated with its ability to increase the Nrf2 mRNA levels.
MeSH term(s)	Acetophenones/pharmacology ; Animals ; Antioxidants/pharmacology ; Corpus Striatum/drug effects ; Corpus Striatum/metabolism ; Glutathione/metabolism ; Glutathione Peroxidase/metabolism ; Male ; NF-E2-Related Factor 2/metabolism ; Quinolinic Acid/pharmacology ; Rats, Wistar
Chemical Substances	Acetophenones ; Antioxidants ; NF-E2-Related Factor 2 ; Nfe2l2 protein, rat ; acetovanillone (B6J7B9UDTR) ; Glutathione Peroxidase (EC 1.11.1.9) ; Quinolinic Acid (F6F0HK1URN) ; Glutathione (GAN16C9B8O)
Language	English
Publishing date	2017-03-19
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	196739-3
ISSN	1873-7544 ; 0306-4522
ISSN (online)	1873-7544
ISSN	0306-4522
DOI	10.1016/j.neuroscience.2017.03.011
Database	MEDical Literature Analysis and Retrieval System OnLINE

In stock of ZB MED Cologne/Königswinter

Zs.A 1215: Show issues

Location:
Je nach Verfügbarkeit (siehe Angabe bei Bestand)
bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
Jg. 1995 - 2021: Lesesall (1.OG)
ab Jg. 2022: Lesesaal (EG)

Order via subito

Details ▾
- See ZB MED holdings
- Order with fees

To top

More links

Kategorien

Order via subito

Full text online

More links

Kategorien

Inter-library loan at ZB MED

Full text online

More links

Kategorien

Inter-library loan at ZB MED

More links

Kategorien

Order via subito

More links

Kategorien

Order via subito

Inter-library loan at ZB MED

Full text online

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito

More links

Kategorien

Order via subito

More links

Kategorien

In stock of ZB MED Cologne/Königswinter

Order via subito