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Article: Prise en charge interdisciplinaire des adolescent-e-s transgenres et non binaires. L’exemple du Centre hospitalier universitaire vaudois (CHUV).

Schmitt, Patrick André / Busiah, Kanetee / Morisod Harari, Mathilde / Jacot-Guillarmod, Martine / Zufferey, Adèle / Ambresin, Anne-Emmanuelle

Revue medicale suisse

2024 Volume 20, Issue 870, Page(s) 802–806

Abstract: This article outlines the management of transgender and non-binary adolescents at CHUV. The current rise in visibility of this topic is associated with an increased demand for intervention and at the same time with an increased generation of continuously ...

Title translation	Interdisciplinary care for gender diverse adolescents. The example of the Centre hospitalier universitaire vaudois (CHUV).
Abstract	This article outlines the management of transgender and non-binary adolescents at CHUV. The current rise in visibility of this topic is associated with an increased demand for intervention and at the same time with an increased generation of continuously expanding medical evidence to guide interventions. The close collaboration among various specialized adolescent health services enables an interdisciplinary evaluation of diagnostic elements and indications for potential psychological, social or medical interventions. This article underscores the significance of an interdisciplinary and individualized approach to transgender and non-binary adolescents, respecting the diversity of the human being.
MeSH term(s)	Adolescent ; Humans ; Transgender Persons ; Interdisciplinary Studies
Language	French
Publishing date	2024-04-17
Publishing country	Switzerland
Document type	English Abstract ; Journal Article
ZDB-ID	2177010-4
ISSN	1660-9379
ISSN	1660-9379
DOI	10.53738/REVMED.2024.20.870.802
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Article ; Online: Molecular Evolution of the Glutathione S-Transferase Family in the Bemisia tabaci Species Complex.

Aidlin Harari, Ofer / Santos-Garcia, Diego / Musseri, Mirit / Moshitzky, Pnina / Patel, Mitulkumar / Visendi, Paul / Seal, Susan / Sertchook, Rotem / Malka, Osnat / Morin, Shai

Genome biology and evolution

2020 Volume 12, Issue 2, Page(s) 3857–3872

Abstract: The glutathione S-transferase (GST) family plays an important role in the adaptation of herbivorous ...

Abstract	The glutathione S-transferase (GST) family plays an important role in the adaptation of herbivorous insects to new host plants and other environmental constrains. The family codes for enzymes that neutralize reactive oxygen species and phytotoxins through the conjugation of reduced glutathione. Here, we studied the molecular evolution of the GST family in Bemisia tabaci, a complex of >35 sibling species, differing in their geographic and host ranges. We tested if some enzymes evolved different functionality, by comparing their sequences in six species, representing five of the six major genetic clades in the complex. Comparisons of the nonsynonymous to synonymous substitution ratios detected positive selection events in 11 codons of 5 cytosolic GSTs. Ten of them are located in the periphery of the GST dimer, suggesting a putative involvement in interactions with other proteins. Modeling the tertiary structure of orthologous enzymes, identified additional 19 mutations in 9 GSTs, likely affecting the enzymes' functionality. Most of the mutation events were found in the environmentally responsive classes Delta and Sigma, indicating a slightly different delta/sigma tool box in each species. At a broader genomic perspective, our analyses indicated a significant expansion of the Delta GST class in B. tabaci and a general association between the diet breadth of hemipteran species and their total number of GST genes. We raise the possibility that at least some of the identified changes improve the fitness of the B. tabaci species carrying them, leading to their better adaptation to specific environments.
MeSH term(s)	Animals ; Evolution, Molecular ; Glutathione Transferase/chemistry ; Glutathione Transferase/genetics ; Glutathione Transferase/metabolism ; Hemiptera/enzymology ; Hemiptera/genetics ; Mutation/genetics ; Phylogeny ; Protein Conformation
Chemical Substances	Glutathione Transferase (EC 2.5.1.18)
Language	English
Publishing date	2020-01-23
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ISSN	1759-6653
ISSN (online)	1759-6653
DOI	10.1093/gbe/evaa002
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Article ; Online: Spread of hyperplastic pulmonary neuroendocrine cells into air spaces (S.H.I.P.M.E.N.T.S): A proof for artifact.

Pelosi, Giuseppe / Nesa, Francesco / Taietti, Davide / Servillo, Sascia Pietro / Papanikolaou, Nikolaos / Zompatori, Maurizio / Meroni, Alberto / Harari, Sergio / Incarbone, Matteo

Lung cancer (Amsterdam, Netherlands)

2019 Volume 137, Page(s) 43–47

Abstract: Objectives: Spread through air spaces (STAS) is a recently proposed invasion way of lung cancer, including neuroendocrine (NE) neoplasms. However, if this phenomenon is a real one or an artifact while manipulating lung specimens, it is still matter of ... ...

Abstract	Objectives: Spread through air spaces (STAS) is a recently proposed invasion way of lung cancer, including neuroendocrine (NE) neoplasms. However, if this phenomenon is a real one or an artifact while manipulating lung specimens, it is still matter of debate. Material and methods: Three consecutive patients with newly diagnosed diffuse idiopathic pulmonary NE cell hyperplasia (DIPNECH) were reviewed for STAS. Results: In well-fixed lung specimens, DIPNECH was seen to coexist with atypical carcinoid, bifocal typical carcinoid and adenocarcinoma in the three patients, respectively. While STAS was not observed at the growing edges of tumors, a few freely-floating aggregates of hyperplastic NE cells within air spaces were noticed to emanate from foci of NE hyperplasia and tumorlets and in intimate association with normal bronchiolar cells and erythrocytes to denote artifactual derivation upon tissue manipulation. Conclusions: Traveling of hyperplastic NE cells through air spaces is likely to artifactually occur via knife, surgeon or other way, thus challenging invasion by STAS.
MeSH term(s)	Adenocarcinoma of Lung/pathology ; Aged ; Artifacts ; Carcinoid Tumor/pathology ; Female ; Humans ; Hyperplasia/pathology ; Lung Neoplasms/pathology ; Male ; Middle Aged ; Neoplasm Invasiveness ; Neuroendocrine Cells/pathology ; Neuroendocrine Tumors/pathology ; Prognosis
Language	English
Publishing date	2019-09-13
Publishing country	Ireland
Document type	Case Reports ; Journal Article
ZDB-ID	632771-0
ISSN	1872-8332 ; 0169-5002
ISSN (online)	1872-8332
ISSN	0169-5002
DOI	10.1016/j.lungcan.2019.09.006
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Zs.MO 423: Show issues

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Article: Prototype of social-ecological system’s resilience analysis using a dynamic index

Oliveira, Bruno M. / Boumans, Roelof / Fath, Brian D. / Othoniel, Benoit / Liu, Wei / Harari, Joseph

Ecological indicators. 2022 Aug., v. 141

2022

Abstract: ... A system’s property is composed of ecological and social limits representing boundaries that, if trespassed ... result in the system’s regime change with increasing uncertainties. These changes can impact ... the reliability of delivering a set of desired ecosystem services, and consequently, society’s wellbeing can be ...

Abstract	Resilience is understood as a social-ecological system (SES) property that embodies nature and society and a research perspective with high potential to be applied in reaching sustainability goals. A system’s property is composed of ecological and social limits representing boundaries that, if trespassed, result in the system’s regime change with increasing uncertainties. These changes can impact the reliability of delivering a set of desired ecosystem services, and consequently, society’s wellbeing can be negatively affected. Thus, modeling a complex and adaptive SES, with feedback, nonlinearities, and path–dependence becomes a crucial tool to inform building a responsible governance behavior that tackles SES resilience. This work built a prototype model of SES resilience for a case study in a Brazilian coastal city with the following aims: 1) to formalize the principles underpinning resilience into a dynamic index, 2) to assess the extent to which this understanding highlights system interdependencies and tradeoffs, and 3) to learn about the benefits of making quantitative assessments of such socio–institutional principles. Multiscale Integrated Model of Ecosystem Services – MIMES (Boumans et al., 2015; Oliveira et al., 2022) is a SES modeling framework using System Dynamics that embraces complexities’ attributes in an interdisciplinary and integrated model. Constructing a causal loop diagram embracing the social sphere represented by the seven resilience principles proposed by Biggs et al. (2015), revealed the necessity to include social goals in the model. It was considered that the Homo economicus represents the most common social perspective and determinant for those goals. Ten different types of ecosystem services were extracted from the ecological part of the simulation (Oliveira et al. 2022) and then combined with those seven resilience principles into the Dynamic Resilience Index (DRI) using a Cobb Douglas–like production function. The numerical simulation produced four insights about resilience that are described and discussed: 1st insight: resilience of what to what? The resilience of the whole system in providing a set of Ecosystem Services against changes in slow variables; 2nd insight: resilience presents seasonal variations; 3rd insight: the system is operating as if it is in the K phase during ecological succession; 4th insight: not all resilience principles have the same weight in resilience. Conclusions point out that resilience can present seasonal variations, and that response diversity and functional redundancy are leverage principles with higher influence in resilience.
Keywords	Homo ; case studies ; ecological succession ; ecosystems ; governance ; mathematical models ; production functions ; prototypes ; society
Language	English
Dates of publication	2022-08
Publishing place	Elsevier Ltd
Document type	Article
ZDB-ID	2036774-0
ISSN	1872-7034 ; 1470-160X
ISSN (online)	1872-7034
ISSN	1470-160X
DOI	10.1016/j.ecolind.2022.109113
Database	NAL-Catalogue (AGRICOLA)

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Article ; Online: Primary umbilical endometriosis (Villar’s nodule): Case report

Benardete-Harari, Denise Niza / Muñoz-de Cote Frade, José Manuel / Hidalgo-Carrera, Jaime Antonio / Felgueres-Hermida, Alberto / Meraz-Ávila, Diego

Revista medica del Instituto Mexicano del Seguro Social

2018 Volume 56, Issue 2, Page(s) 203–206

Abstract: Background: Umbilical endometriosis or Villar’s nodule is uncommon. Incidence goes up to 0.5-1.2 ... worldwide, there is no incidence reports in Mexico. Clinically, it’s a solitary node, solid and well defined ... colored red, blue or black and it’s ocassionally accompanied by bleeding and dysmenorrhea. The treatment ...

Title translation	Endometriosis umbilical primaria (nódulo de Villar): reporte de un caso
Abstract	Background: Umbilical endometriosis or Villar’s nodule is uncommon. Incidence goes up to 0.5-1.2% of all patients with endometriosis. As far as 2012, only 109 cases of umbilical endometriosis have been reported worldwide, there is no incidence reports in Mexico. Clinically, it’s a solitary node, solid and well defined, colored red, blue or black and it’s ocassionally accompanied by bleeding and dysmenorrhea. The treatment can be medical or surgical and it consists of painkillers, and hormonal suppression using GnRH analogues, hormonal contraceptives, danazol or progesterone. Surgical treatment consists in complete resection of the umbilical lesion. The aim of this paper is to present a clinical report case and have a bibliographic review of this pathology. Clinical case: A 34 year-old patient comes to the clinic due to severe umbilical pain of some months, associated with enlargement, swelling and cyclic menstrual bleeding. Escision of the lesion is performed and sent to pathology having the diagnosis of umbilical endometriosis. There is no evidence of endometriosic implants under laparoscopic control. Conclusions: Regards of being a rare pathology, umbilical endometriosis must be taken as a differential diagnosis in patients with umbilical lesions associated with menstrual cycle pain.
MeSH term(s)	Adult ; Diagnosis, Differential ; Endometriosis/diagnosis ; Endometriosis/pathology ; Female ; Humans ; Umbilicus/pathology
Language	Spanish
Publishing date	2018-06-15
Publishing country	Mexico
Document type	Case Reports ; Journal Article
ZDB-ID	732133-8
ISSN	2448-5667 ; 0443-5117 ; 0484-7849
ISSN (online)	2448-5667
ISSN	0443-5117 ; 0484-7849
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Article ; Online: Linkage, whole genome sequence, and biological data implicate variants in RAB10 in Alzheimer’s disease resilience

Perry G. Ridge / Celeste M. Karch / Simon Hsu / Ivan Arano / Craig C. Teerlink / Mark T. W. Ebbert / Josue D. Gonzalez Murcia / James M. Farnham / Anna R. Damato / Mariet Allen / Xue Wang / Oscar Harari / Victoria M. Fernandez / Rita Guerreiro / Jose Bras / John Hardy / Ronald Munger / Maria Norton / Celeste Sassi /

Andrew Singleton / Steven G. Younkin / Dennis W. Dickson / Todd E. Golde / Nathan D. Price / Nilüfer Ertekin-Taner / Carlos Cruchaga / Alison M. Goate / Christopher Corcoran / JoAnn Tschanz / Lisa A. Cannon-Albright / John S. K. Kauwe / for the Alzheimer’s Disease Neuroimaging Initiative

Genome Medicine, Vol 9, Iss 1, Pp 1-

2017 Volume 14

Abstract: Abstract Background While age and the APOE ε4 allele are major risk factors for Alzheimer’s ...

Abstract	Abstract Background While age and the APOE ε4 allele are major risk factors for Alzheimer’s disease (AD), a small percentage of individuals with these risk factors exhibit AD resilience by living well beyond 75 years of age without any clinical symptoms of cognitive decline. Methods We used over 200 “AD resilient” individuals and an innovative, pedigree-based approach to identify genetic variants that segregate with AD resilience. First, we performed linkage analyses in pedigrees with resilient individuals and a statistical excess of AD deaths. Second, we used whole genome sequences to identify candidate SNPs in significant linkage regions. Third, we replicated SNPs from the linkage peaks that reduced risk for AD in an independent dataset and in a gene-based test. Finally, we experimentally characterized replicated SNPs. Results Rs142787485 in RAB10 confers significant protection against AD (p value = 0.0184, odds ratio = 0.5853). Moreover, we replicated this association in an independent series of unrelated individuals (p value = 0.028, odds ratio = 0.69) and used a gene-based test to confirm a role for RAB10 variants in modifying AD risk (p value = 0.002). Experimentally, we demonstrated that knockdown of RAB10 resulted in a significant decrease in Aβ42 (p value = 0.0003) and in the Aβ42/Aβ40 ratio (p value = 0.0001) in neuroblastoma cells. We also found that RAB10 expression is significantly elevated in human AD brains (p value = 0.04). Conclusions Our results suggest that RAB10 could be a promising therapeutic target for AD prevention. In addition, our gene discovery approach can be expanded and adapted to other phenotypes, thus serving as a model for future efforts to identify rare variants for AD and other complex human diseases.
Keywords	Alzheimer’s disease ; Protective variants ; Whole genome sequencing ; Utah Population Database ; Linkage analyses ; Medicine ; R ; Genetics ; QH426-470
Subject code	616
Language	English
Publishing date	2017-11-01T00:00:00Z
Publisher	BMC
Document type	Article ; Online
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Article ; Online: The war in Ukraine is an environmental catastrophe.

Harari, S / Annesi-Maesano, I

The international journal of tuberculosis and lung disease : the official journal of the International Union against Tuberculosis and Lung Disease

2023 Volume 27, Issue 2, Page(s) 94–95

Language	English
Publishing date	2023-02-28
Publishing country	France
Document type	Journal Article
ZDB-ID	1385624-8
ISSN	1815-7920 ; 1027-3719
ISSN (online)	1815-7920
ISSN	1027-3719
DOI	10.5588/ijtld.22.0603
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Zs.MO 261: Show issues

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Article ; Online: Genetic variants associated with Alzheimer’s disease confer different cerebral cortex cell-type population structure

Zeran Li / Jorge L. Del-Aguila / Umber Dube / John Budde / Rita Martinez / Kathleen Black / Qingli Xiao / Nigel J. Cairns / The Dominantly Inherited Alzheimer Network (DIAN) / Joseph D. Dougherty / Jin-Moo Lee / John C. Morris / Randall J. Bateman / Celeste M. Karch / Carlos Cruchaga / Oscar Harari

Genome Medicine, Vol 10, Iss 1, Pp 1-

2018 Volume 19

Abstract: Abstract Background Alzheimer’s disease (AD) is characterized by neuronal loss and astrocytosis ...

Abstract	Abstract Background Alzheimer’s disease (AD) is characterized by neuronal loss and astrocytosis in the cerebral cortex. However, the specific effects that pathological mutations and coding variants associated with AD have on the cellular composition of the brain are often ignored. Methods We developed and optimized a cell-type-specific expression reference panel and employed digital deconvolution methods to determine brain cellular distribution in three independent transcriptomic studies. Results We found that neuronal and astrocyte relative proportions differ between healthy and diseased brains and also among AD cases that carry specific genetic risk variants. Brain carriers of pathogenic mutations in APP, PSEN1, or PSEN2 presented lower neuron and higher astrocyte relative proportions compared to sporadic AD. Similarly, the APOE ε4 allele also showed decreased neuronal and increased astrocyte relative proportions compared to AD non-carriers. In contrast, carriers of variants in TREM2 risk showed a lower degree of neuronal loss compared to matched AD cases in multiple independent studies. Conclusions These findings suggest that genetic risk factors associated with AD etiology have a specific imprinting in the cellular composition of AD brains. Our digital deconvolution reference panel provides an enhanced understanding of the fundamental molecular mechanisms underlying neurodegeneration, enabling the analysis of large bulk RNA-sequencing studies for cell composition and suggests that correcting for the cellular structure when performing transcriptomic analysis will lead to novel insights of AD.
Keywords	Digital deconvolution ; Alzheimer’s disease ; Brain cellular composition ; Bulk RNA-sequencing ; Autosomal dominant AD ; TREM2 ; Medicine ; R ; Genetics ; QH426-470
Subject code	616
Language	English
Publishing date	2018-06-01T00:00:00Z
Publisher	BMC
Document type	Article ; Online
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Article ; Online: Measurement of the proton-air cross section at √s=57 TeV with the Pierre Auger Observatory.

Abreu, P / Aglietta, M / Ahn, E J / Albuquerque, I F M / Allard, D / Allekotte, I / Allen, J / Allison, P / Almeda, A / Alvarez Castillo, J / Alvarez-Muñiz, J / Ambrosio, M / Aminaei, A / Anchordoqui, L / Andringa, S / Antičić, T / Aramo, C / Arganda, E / Arqueros, F /

Asorey, H / Assis, P / Aublin, J / Ave, M / Avenier, M / Avila, G / Bäcker, T / Balzer, M / Barber, K B / Barbosa, A F / Bardenet, R / Barroso, S L C / Baughman, B / Bäuml, J / Beatty, J J / Becker, B R / Becker, K H / Bellétoile, A / Bellido, J A / Benzvi, S / Berat, C / Bertou, X / Biermann, P L / Billoir, P / Blanco, F / Blanco, M / Bleve, C / Blümer, H / Boháčová, M / Boncioli, D / Bonifazi, C / Bonino, R / Borodai, N / Brack, J / Brogueira, P / Brown, W C / Bruijn, R / Buchholz, P / Bueno, A / Burton, R E / Caballero-Mora, K S / Caramete, L / Caruso, R / Castellina, A / Catalano, O / Cataldi, G / Cazon, L / Cester, R / Chauvin, J / Cheng, S H / Chiavassa, A / Chinellato, J A / Chirinos Diaz, J / Chudoba, J / Clay, R W / Coluccia, M R / Conceição, R / Contreras, F / Cook, H / Cooper, M J / Coppens, J / Cordier, A / Coutu, S / Covault, C E / Creusot, A / Criss, A / Cronin, J / Curutiu, A / Dagoret-Campagne, S / Dallier, R / Dasso, S / Daumiller, K / Dawson, B R / de Almeida, R M / De Domenico, M / De Donato, C / de Jong, S J / De La Vega, G / de Mello Junior, W J M / de Mello Neto, J R T / De Mitri, I / de Souza, V / de Vries, K D / Decerprit, G / del Peral, L / del Río, M / Deligny, O / Dembinski, H / Dhital, N / Di Giulio, C / Díaz Castro, M L / Diep, P N / Dobrigkeit, C / Docters, W / D'Olivo, J C / Dong, P N / Dorofeev, A / dos Anjos, J C / Dova, M T / D'Urso, D / Dutan, I / Ebr, J / Engel, R / Erdmann, M / Escobar, C O / Espadanal, J / Etchegoyen, A / Facal San Luis, P / Fajardo Tapia, I / Falcke, H / Farrar, G / Fauth, A C / Fazzini, N / Ferguson, A P / Ferrero, A / Fick, B / Filevich, A / Filipčič, A / Fliescher, S / Fracchiolla, C E / Fraenkel, E D / Fröhlich, U / Fuchs, B / Gaior, R / Gamarra, R F / Gambetta, S / García, B / Garcia-Gamez, D / Garcia-Pinto, D / Gascon, A / Gemmeke, H / Gesterling, K / Ghia, P L / Giaccari, U / Giller, M / Glass, H / Gold, M S / Golup, G / Gomez Albarracin, F / Gómez Berisso, M / Gonçalves, P / Gonzalez, D / Gonzalez, J G / Gookin, B / Góra, D / Gorgi, A / Gouffon, P / Gozzini, S R / Grashorn, E / Grebe, S / Griffith, N / Grigat, M / Grillo, A F / Guardincerri, Y / Guarino, F / Guedes, G P / Guzman, A / Hague, J D / Hansen, P / Harari, D / Harmsma, S / Harrison, T A / Harton, J L / Haungs, A / Hebbeker, T / Heck, D / Herve, A E / Hojvat, C / Hollon, N / Holmes, V C / Homola, P / Hörandel, J R / Horneffer, A / Horvath, P / Hrabovský, M / Huege, T / Insolia, A / Ionita, F / Italiano, A / Jarne, C / Jiraskova, S / Josebachuili, M / Kadija, K / Kampert, K H / Karhan, P / Kasper, P / Kégl, B / Keilhauer, B / Keivani, A / Kelley, J L / Kemp, E / Kieckhafer, R M / Klages, H O / Kleifges, M / Kleinfeller, J / Knapp, J / Koang, D-H / Kotera, K / Krohm, N / Krömer, O / Kruppke-Hansen, D / Kuehn, F / Kuempel, D / Kulbartz, J K / Kunka, N / La Rosa, G / Lachaud, C / Lauer, R / Lautridou, P / Le Coz, S / Leão, M S A B / Lebrun, D / Lebrun, P / Leigui de Oliveira, M A / Lemiere, A / Letessier-Selvon, A / Lhenry-Yvon, I / Link, K / López, R / Lopez Agüera, A / Louedec, K / Lozano Bahilo, J / Lu, L / Lucero, A / Ludwig, M / Lyberis, H / Macolino, C / Maldera, S / Mandat, D / Mantsch, P / Mariazzi, A G / Marin, J / Marin, V / Maris, I C / Marquez Falcon, H R / Marsella, G / Martello, D / Martin, L / Martinez, H / Martínez Bravo, O / Mathes, H J / Matthews, J / Matthews, J A J / Matthiae, G / Maurizio, D / Mazur, P O / Medina-Tanco, G / Melissas, M / Melo, D / Menichetti, E / Menshikov, A / Mertsch, P / Meurer, C / Mićanović, S / Micheletti, M I / Miller, W / Miramonti, L / Molina-Bueno, L / Mollerach, S / Monasor, M / Monnier Ragaigne, D / Montanet, F / Morales, B / Morello, C / Moreno, E / Moreno, J C / Morris, C / Mostafá, M / Moura, C A / Mueller, S / Muller, M A / Müller, G / Münchmeyer, M / Mussa, R / Navarra, G / Navarro, J L / Navas, S / Necesal, P / Nellen, L / Nelles, A / Neuser, J / Nhung, P T / Niemietz, L / Nierstenhoefer, N / Nitz, D / Nosek, D / Nožka, L / Nyklicek, M / Oehlschläger, J / Olinto, A / Olmos-Gilbaja, V M / Ortiz, M / Pacheco, N / Pakk Selmi-Dei, D / Palatka, M / Pallotta, J / Palmieri, N / Parente, G / Parizot, E / Parra, A / Parsons, R D / Pastor, S / Paul, T / Pech, M / Pekala, J / Pelayo, R / Pepe, I M / Perrone, L / Pesce, R / Petermann, E / Petrera, S / Petrinca, P / Petrolini, A / Petrov, Y / Petrovic, J / Pfendner, C / Phan, N / Piegaia, R / Pierog, T / Pieroni, P / Pimenta, M / Pirronello, V / Platino, M / Ponce, V H / Pontz, M / Privitera, P / Prouza, M / Quel, E J / Querchfeld, S / Rautenberg, J / Ravel, O / Ravignani, D / Revenu, B / Ridky, J / Riggi, S / Risse, M / Ristori, P / Rivera, H / Rizi, V / Roberts, J / Robledo, C / Rodrigues de Carvalho, W / Rodriguez, G / Rodriguez Martino, J / Rodriguez Rojo, J / Rodriguez-Cabo, I / Rodríguez-Frías, M D / Ros, G / Rosado, J / Rossler, T / Roth, M / Rouillé-d'Orfeuil, B / Roulet, E / Rovero, A C / Rühle, C / Salamida, F / Salazar, H / Salesa Greus, F / Salina, G / Sánchez, F / Santo, C E / Santos, E / Santos, E M / Sarazin, F / Sarkar, B / Sarkar, S / Sato, R / Scharf, N / Scherini, V / Schieler, H / Schiffer, P / Schmidt, A / Scholten, O / Schoorlemmer, H / Schovancova, J / Schovánek, P / Schröder, F / Schulte, S / Schuster, D / Sciutto, S J / Scuderi, M / Segreto, A / Settimo, M / Shadkam, A / Shellard, R C / Sidelnik, I / Sigl, G / Silva Lopez, H H / Smiałkowski, A / Smída, R / Snow, G R / Sommers, P / Sorokin, J / Spinka, H / Squartini, R / Stanic, S / Stapleton, J / Stasielak, J / Stephan, M / Stutz, A / Suarez, F / Suomijärvi, T / Supanitsky, A D / Suša, T / Sutherland, M S / Swain, J / Szadkowski, Z / Szuba, M / Tamashiro, A / Tapia, A / Tartare, M / Taşcău, O / Tavera Ruiz, C G / Tcaciuc, R / Tegolo, D / Thao, N T / Thomas, D / Tiffenberg, J / Timmermans, C / Tiwari, D K / Tkaczyk, W / Todero Peixoto, C J / Tomé, B / Tonachini, A / Travnicek, P / Tridapalli, D B / Tristram, G / Trovato, E / Tueros, M / Ulrich, R / Unger, M / Urban, M / Valdés Galicia, J F / Valiño, I / 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Physical review letters

2012 Volume 109, Issue 6, Page(s) 62002

Abstract: We report a measurement of the proton-air cross section for particle production at the center-of-mass energy per nucleon of 57 TeV. This is derived from the distribution of the depths of shower maxima observed with the Pierre Auger Observatory: ... ...

Abstract	We report a measurement of the proton-air cross section for particle production at the center-of-mass energy per nucleon of 57 TeV. This is derived from the distribution of the depths of shower maxima observed with the Pierre Auger Observatory: systematic uncertainties are studied in detail. Analyzing the tail of the distribution of the shower maxima, a proton-air cross section of [505±22(stat)(-36)(+28)(syst)] mb is found.
Language	English
Publishing date	2012-08-10
Publishing country	United States
Document type	Journal Article
ZDB-ID	208853-8
ISSN	1079-7114 ; 0031-9007
ISSN (online)	1079-7114
ISSN	0031-9007
DOI	10.1103/PhysRevLett.109.062002
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Article ; Online: Now we know: chronic exposure to air pollutants is a risk factor for the development of idiopathic pulmonary fibrosis.

Sesé, Lucile / Harari, Sergio

The European respiratory journal

2023 Volume 61, Issue 2

MeSH term(s)	Humans ; Air Pollutants/adverse effects ; Air Pollutants/analysis ; Genetic Predisposition to Disease ; Idiopathic Pulmonary Fibrosis/epidemiology ; Idiopathic Pulmonary Fibrosis/etiology ; Air Pollution/adverse effects ; Air Pollution/analysis ; Risk Factors
Chemical Substances	Air Pollutants
Language	English
Publishing date	2023-02-02
Publishing country	England
Document type	Editorial ; Comment
ZDB-ID	639359-7
ISSN	1399-3003 ; 0903-1936
ISSN (online)	1399-3003
ISSN	0903-1936
DOI	10.1183/13993003.02113-2022
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Zs.A 2322: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 292: Show issues

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