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Article ; Online: Improving Health Equity in Rheumatology Through Workforce Diversification and Support for Health Equity Research and Education.

Vassileva, Maria T / Suresh, Vandana / Chan, Andrew C / Akinsete, Alisha Valdez / Blanco, Irene / Blazer, Ashira / Criscione-Schreiber, Lisa / Dowell, Sharon / Feldman, Candace H / FitzGerald, John / Gilbert, Mileka / Hughes, Grant / Husni, M Elaine / Kerr, Gail / Kwan, Olivia / Mantilla, Bryanna / Nilson, Susanne / Rivadeneira, Alfredo Carlos / Rodríguez, Martha /

Smith, Benjamin J / Soulsby, William Daniel / Wong, Stephen Chee-Yung / Yazdany, Jinoos / Ross, Will

Arthritis & rheumatology (Hoboken, N.J.)

2024

Language	English
Publishing date	2024-01-19
Publishing country	United States
Document type	Journal Article
ZDB-ID	2756371-6
ISSN	2326-5205 ; 2326-5191
ISSN (online)	2326-5205
ISSN	2326-5191
DOI	10.1002/art.42804
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Article ; Online: Cytokines, obesity, and cancer: new insights on mechanisms linking obesity to cancer risk and progression.

Gilbert, Candace A / Slingerland, Joyce M

Annual review of medicine

2013 Volume 64, Page(s) 45–57

Abstract: Obesity is a problem of epidemic proportions in many developed nations. Increased body mass index and obesity are associated with a significantly worse outcome for many cancers. Breast cancer risk in the postmenopausal setting and poor disease outcome ... ...

Abstract	Obesity is a problem of epidemic proportions in many developed nations. Increased body mass index and obesity are associated with a significantly worse outcome for many cancers. Breast cancer risk in the postmenopausal setting and poor disease outcome for all patients is significantly augmented in overweight and obese individuals. The expansion of fat tissue involves a complex interaction of endocrine factors known as adipokines and cytokines. High cytokine levels in primary breast cancers and in the circulation of affected patients have been associated with poor outcome. This review summarizes the how cytokine production in obese adipose tissue creates a chronic inflammatory microenvironment that favors tumor cell motility, invasion, and epithelial-mesenchymal transition to enhance the metastatic potential of tumor cells. Many of the cytokines associated with a proinflammatory state are not only upregulated in obese adipose tissue but may also stimulate the self-renewal of cancer stem cells. Thus, enhanced cytokine production in obese adipose tissue may serve both as a chemoattractant for invading cancers and to augment their malignant potential. These new mechanistic insights suggest that the current obesity epidemic will presage a significant increase in cancer incidence, morbidity, and mortality in the next few decades.
MeSH term(s)	Body Mass Index ; Cytokines/metabolism ; Disease Progression ; Global Health ; Humans ; Incidence ; Neoplasms/complications ; Neoplasms/epidemiology ; Neoplasms/metabolism ; Obesity/complications ; Obesity/epidemiology ; Obesity/metabolism ; Risk Factors
Chemical Substances	Cytokines
Language	English
Publishing date	2013
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	207930-6
ISSN	1545-326X ; 0066-4219
ISSN (online)	1545-326X
ISSN	0066-4219
DOI	10.1146/annurev-med-121211-091527
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Article ; Online: VEGFA activates an epigenetic pathway upregulating ovarian cancer-initiating cells.

Jang, Kibeom / Kim, Minsoon / Gilbert, Candace A / Simpkins, Fiona / Ince, Tan A / Slingerland, Joyce M

EMBO molecular medicine

2017 Volume 9, Issue 3, Page(s) 304–318

Abstract: The angiogenic factor, VEGFA, is a therapeutic target in ovarian cancer (OVCA). VEGFA can also stimulate stem-like cells in certain cancers, but mechanisms thereof are poorly understood. Here, we show that VEGFA mediates stem cell actions in primary ... ...

Abstract	The angiogenic factor, VEGFA, is a therapeutic target in ovarian cancer (OVCA). VEGFA can also stimulate stem-like cells in certain cancers, but mechanisms thereof are poorly understood. Here, we show that VEGFA mediates stem cell actions in primary human OVCA culture and OVCA lines via VEGFR2-dependent Src activation to upregulate Bmi1, tumor spheres, and ALDH1 activity. The VEGFA-mediated increase in spheres was abrogated by Src inhibition or
MeSH term(s)	Aldehyde Dehydrogenase 1 Family ; Cell Line, Tumor ; Cell Proliferation ; Epigenesis, Genetic ; Female ; Humans ; Isoenzymes/metabolism ; MicroRNAs/metabolism ; Neoplastic Stem Cells/physiology ; Ovarian Neoplasms/pathology ; Polycomb Repressive Complex 1/metabolism ; Retinal Dehydrogenase/metabolism ; Up-Regulation ; Vascular Endothelial Growth Factor A/metabolism
Chemical Substances	BMI1 protein, human ; Isoenzymes ; MIRN128 microRNA, human ; MicroRNAs ; VEGFA protein, human ; Vascular Endothelial Growth Factor A ; Aldehyde Dehydrogenase 1 Family (EC 1.2.1) ; ALDH1A1 protein, human (EC 1.2.1.36) ; Retinal Dehydrogenase (EC 1.2.1.36) ; Polycomb Repressive Complex 1 (EC 2.3.2.27)
Language	English
Publishing date	2017-02-08
Publishing country	England
Document type	Journal Article
ZDB-ID	2467145-9
ISSN	1757-4684 ; 1757-4676
ISSN (online)	1757-4684
ISSN	1757-4676
DOI	10.15252/emmm.201606840
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Article ; Online: Detecting personal microbiota signatures at artificial crime scenes.

Hampton-Marcell, Jarrad T / Larsen, Peter / Anton, Tifani / Cralle, Lauren / Sangwan, Naseer / Lax, Simon / Gottel, Neil / Salas-Garcia, Mariana / Young, Candace / Duncan, George / Lopez, Jose V / Gilbert, Jack A

Forensic science international

2020 Volume 313, Page(s) 110351

Abstract: When mapped to the environments we interact with on a daily basis, the 36 million microbial cells per hour that humans emit leave a trail of evidence that can be leveraged for forensic analysis. We employed 16S rRNA amplicon sequencing to map unique ... ...

Abstract	When mapped to the environments we interact with on a daily basis, the 36 million microbial cells per hour that humans emit leave a trail of evidence that can be leveraged for forensic analysis. We employed 16S rRNA amplicon sequencing to map unique microbial sequence variants between human skin and building surfaces in three experimental conditions: over time during controlled and uncontrolled incidental interactions with a door handle, and during multiple mock burglaries in ten real residences. We demonstrate that humans (n = 30) leave behind microbial signatures that can be used to track interaction with various surfaces within a building, but the likelihood of accurately detecting the specific burglar for a given home was between 20-25%. Also, the human microbiome contains rare microbial taxa that can be combined to create a unique microbial profile, which when compared to 600 other individuals can improve our ability to link an individual 'burglar' to a residence. In total, 5512 discriminating, non-singleton unique exact sequence variants (uESVs) were identified as unique to an individual, with a minimum of 1 and a maximum of 568, suggesting some people maintain a greater degree of unique taxa compared to our population of 600. Approximate 60-77% of the unique exact sequence variants originated from the hands of participants, and these microbial discriminators spanned 36 phyla but were dominated by the Proteobacteria (34%). A fitted regression generated to determine whether an intruder's uESVs found on door handles in an office decayed over time in the presence or absence of office workers, found no significant shift in proportion of uESVs over time irrespective of the presence of office workers. While it was possible to detect the correct burglars' microbiota as having contributed to the invaded space, the predictions were very weak in comparison to accepted forensic standards. This suggests that at this time 16S rRNA amplicon sequencing of the built environment microbiota cannot be used as a reliable trace evidence standard for criminal investigations.
MeSH term(s)	Crime ; Forensic Sciences/methods ; Humans ; Microbiota/genetics ; RNA, Ribosomal, 16S/genetics ; Sequence Analysis, DNA ; Skin/microbiology ; Statistics as Topic ; Touch
Chemical Substances	RNA, Ribosomal, 16S
Language	English
Publishing date	2020-05-30
Publishing country	Ireland
Document type	Journal Article
ZDB-ID	424042-x
ISSN	1872-6283 ; 0379-0738
ISSN (online)	1872-6283
ISSN	0379-0738
DOI	10.1016/j.forsciint.2020.110351
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Article ; Online: Cancer stem cells: cell culture, markers, and targets for new therapies.

Gilbert, Candace A / Ross, Alonzo H

Journal of cellular biochemistry

2009 Volume 108, Issue 5, Page(s) 1031–1038

Abstract: A cancer stem cell (CSC) is defined as an undifferentiated cell with the ability to self-renew, differentiate to multiple lineages and initiate tumors that mimic the parent tumor. In this review, we focus on glioblastomas, describing recent progress and ... ...

Abstract	A cancer stem cell (CSC) is defined as an undifferentiated cell with the ability to self-renew, differentiate to multiple lineages and initiate tumors that mimic the parent tumor. In this review, we focus on glioblastomas, describing recent progress and problems in characterizing these cells. There have been advances in CSC culture, but tumor cell heterogeneity has made purification of CSCs difficult. Indeed, it may be that CSCs significantly vary from tumor to tumor. We also discuss the proposal that CSCs are resistant to radiotherapy and chemotherapy and play a major role in repopulating tumors following treatment. To overcome their resistance to conventional therapies, we may be able to use our extensive knowledge of the signaling pathways essential for stem cells during development. These pathways have potential as targets for new glioblastoma therapies. Hence, although there is an ongoing debate on the nature of CSCs, the theory continues to suggest new ideas for both the lab and the clinic.
MeSH term(s)	Biomarkers, Tumor ; Brain Neoplasms/drug therapy ; Brain Neoplasms/pathology ; Brain Neoplasms/radiotherapy ; Cell Culture Techniques ; Cell Differentiation ; Cell Lineage ; Cell Proliferation ; Cell Transformation, Neoplastic ; Culture Media ; Drug Resistance, Neoplasm/physiology ; Glioblastoma/drug therapy ; Glioblastoma/pathology ; Glioblastoma/radiotherapy ; Humans ; Models, Biological ; Neoplastic Stem Cells/pathology ; Neoplastic Stem Cells/physiology ; Signal Transduction/physiology
Chemical Substances	Biomarkers, Tumor ; Culture Media
Language	English
Publishing date	2009-09-16
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Review
ZDB-ID	392402-6
ISSN	1097-4644 ; 0730-2312
ISSN (online)	1097-4644
ISSN	0730-2312
DOI	10.1002/jcb.22350
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Article: Hypothermia as a Possible Symptom of Serotonin Toxicity: A Case Report.

McKeirnan, Kimberly C / Vaitla, Kavya / Gilbert, Rubi / Anderson, Candace B / Undeberg, Megan R

The Senior care pharmacist

2023 Volume 38, Issue 6, Page(s) 223–232

Abstract: ... ...

Abstract	Background
MeSH term(s)	Humans ; Serotonin/adverse effects ; Serotonin Syndrome/chemically induced ; Serotonin Syndrome/diagnosis ; Serotonin Syndrome/therapy ; Hypothermia/chemically induced ; Hypothermia/diagnosis ; Selective Serotonin Reuptake Inhibitors/adverse effects ; Fluoxetine/adverse effects
Chemical Substances	Serotonin (333DO1RDJY) ; Selective Serotonin Reuptake Inhibitors ; Fluoxetine (01K63SUP8D)
Language	English
Publishing date	2023-05-26
Publishing country	United States
Document type	Case Reports ; Review ; Journal Article
ISSN	2639-9636
ISSN	2639-9636
DOI	10.4140/TCP.n.2023.223
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Article ; Online: Clinical Indications for Rapid Sequence MRI in Pediatric Neurosurgical Patients and the Limitations and Barriers to Implementation.

Franklin, Deveney / Barr, Candace / Nguyen, Diana / O'Shaughnessy, Declan / Gilbert, Olivia E / Quinsey, Carolyn

Journal of visualized experiments : JoVE

2024 , Issue 203

Abstract: Rapid and fast magnetic resonance imaging (MRI) protocols have become increasingly popular for pediatric neurosurgical patients as they are a great way to reduce ionizing radiation and sedation. While their popularity has increased, there are hurdles to ... ...

Abstract	Rapid and fast magnetic resonance imaging (MRI) protocols have become increasingly popular for pediatric neurosurgical patients as they are a great way to reduce ionizing radiation and sedation. While their popularity has increased, there are hurdles to overcome when transitioning to using them clinically, such as cost, staffing training, and motion artifact. Through this paper, we developed a protocol for clinical applications where rapid MRI can be a substitute or adjuvant in diagnostic workup. Further, we outline the relevant literature for the use of RS-MRI for the spine, TBI, and hydrocephalus pathologies while expanding upon the limitations and logistical barriers when transitioning to their use, a few of which are discussed above. Through this, we conclude that RS-MRI can be used diagnostically for spinal pathologies such as syrinx and hydrocephalus. Further, its lack of sensitivity for TBI findings makes rapid sequence magnetic resonance imaging (RS-MRI) a strong adjuvant with other advanced imaging or computed tomography (CT) for traumatic brain injury (TBI) pathologies.
MeSH term(s)	Child ; Humans ; Brain Injuries, Traumatic/diagnostic imaging ; Brain Injuries, Traumatic/surgery ; Hydrocephalus/pathology ; Hydrocephalus/surgery ; Magnetic Resonance Imaging/methods ; Tomography, X-Ray Computed/methods ; Artifacts ; Adjuvants, Immunologic
Chemical Substances	Adjuvants, Immunologic
Language	English
Publishing date	2024-01-12
Publishing country	United States
Document type	Journal Article ; Video-Audio Media
ZDB-ID	2259946-0
ISSN	1940-087X ; 1940-087X
ISSN (online)	1940-087X
ISSN	1940-087X
DOI	10.3791/65797
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Article ; Online: COVID-19 in hospitalized lung and non-lung solid organ transplant recipients: A comparative analysis from a multicenter study.

Heldman, Madeleine R / Kates, Olivia S / Safa, Kassem / Kotton, Camille N / Georgia, Sarah J / Steinbrink, Julie M / Alexander, Barbara D / Hemmersbach-Miller, Marion / Blumberg, Emily A / Crespo, Maria M / Multani, Ashrit / Lewis, Angelica V / Eugene Beaird, Omer / Haydel, Brandy / La Hoz, Ricardo M / Moni, Lisset / Condor, Yesabeli / Flores, Sandra / Munoz, Carlos G /

Guitierrez, Juan / Diaz, Esther I / Diaz, Daniela / Vianna, Rodrigo / Guerra, Giselle / Loebe, Matthias / Rakita, Robert M / Malinis, Maricar / Azar, Marwan M / Hemmige, Vagish / McCort, Margaret E / Chaudhry, Zohra S / Singh, Pooja / Hughes, Kailey / Velioglu, Arzu / Yabu, Julie M / Morillis, Jose A / Mehta, Sapna A / Tanna, Sajal D / Ison, Michael G / Tomic, Rade / Candace Derenge, Ariella / van Duin, David / Maximin, Adrienne / Gilbert, Carlene / Goldman, Jason D / Sehgal, Sameep / Weisshaar, Dana / Girgis, Reda E / Nelson, Joanna / Lease, Erika D / Limaye, Ajit P / Fisher, Cynthia E

American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons

2021 Volume 21, Issue 8, Page(s) 2774–2784

Abstract: Lung transplant recipients (LTR) with coronavirus disease 2019 (COVID-19) may have higher mortality than non-lung solid organ transplant recipients (SOTR), but direct comparisons are limited. Risk factors for mortality specifically in LTR have not been ... ...

Abstract	Lung transplant recipients (LTR) with coronavirus disease 2019 (COVID-19) may have higher mortality than non-lung solid organ transplant recipients (SOTR), but direct comparisons are limited. Risk factors for mortality specifically in LTR have not been explored. We performed a multicenter cohort study of adult SOTR with COVID-19 to compare mortality by 28 days between hospitalized LTR and non-lung SOTR. Multivariable logistic regression models were used to assess comorbidity-adjusted mortality among LTR vs. non-lung SOTR and to determine risk factors for death in LTR. Of 1,616 SOTR with COVID-19, 1,081 (66%) were hospitalized including 120/159 (75%) LTR and 961/1457 (66%) non-lung SOTR (p = .02). Mortality was higher among LTR compared to non-lung SOTR (24% vs. 16%, respectively, p = .032), and lung transplant was independently associated with death after adjusting for age and comorbidities (aOR 1.7, 95% CI 1.0-2.6, p = .04). Among LTR, chronic lung allograft dysfunction (aOR 3.3, 95% CI 1.0-11.3, p = .05) was the only independent risk factor for mortality and age >65 years, heart failure and obesity were not independently associated with death. Among SOTR hospitalized for COVID-19, LTR had higher mortality than non-lung SOTR. In LTR, chronic allograft dysfunction was independently associated with mortality.
MeSH term(s)	Adult ; Aged ; COVID-19 ; Cohort Studies ; Humans ; Lung ; Organ Transplantation/adverse effects ; SARS-CoV-2 ; Transplant Recipients
Language	English
Publishing date	2021-07-24
Publishing country	United States
Document type	Journal Article ; Multicenter Study ; Research Support, N.I.H., Extramural
ZDB-ID	2060594-8
ISSN	1600-6143 ; 1600-6135
ISSN (online)	1600-6143
ISSN	1600-6135
DOI	10.1111/ajt.16692
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Zs.A 5542: Show issues

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Article: The Role of the Pharmacist in Patient Self-Advocacy for Osteoporosis Screening.

McKeirnan, Kimberly C / Anderson, Candace B / Powell, Alexa J / Gilbert, Rubi / Undeberg, Megan R

The Senior care pharmacist

2022 Volume 37, Issue 12, Page(s) 612–622

Abstract: ... ...

Abstract	Background
MeSH term(s)	Humans ; Female ; Pharmacists ; Osteoporosis/chemically induced ; Osteoporosis/diagnosis ; Osteoporosis/drug therapy ; Bone Density ; Physicians ; Fractures, Bone ; Proton Pump Inhibitors
Chemical Substances	Proton Pump Inhibitors
Language	English
Publishing date	2022-12-02
Publishing country	United States
Document type	Case Reports ; Journal Article
ISSN	2639-9636
ISSN	2639-9636
DOI	10.4140/TCP.n.2022.612
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Article ; Online: Gamma-secretase inhibitors enhance temozolomide treatment of human gliomas by inhibiting neurosphere repopulation and xenograft recurrence.

Gilbert, Candace A / Daou, Marie-Claire / Moser, Richard P / Ross, Alonzo H

Cancer research

2010 Volume 70, Issue 17, Page(s) 6870–6879

Abstract: Malignant gliomas are treated with a combination of surgery, radiation, and temozolomide (TMZ), but these therapies ultimately fail due to tumor recurrence. In glioma cultures, TMZ treatment significantly decreases neurosphere formation; however, a small ...

Abstract	Malignant gliomas are treated with a combination of surgery, radiation, and temozolomide (TMZ), but these therapies ultimately fail due to tumor recurrence. In glioma cultures, TMZ treatment significantly decreases neurosphere formation; however, a small percentage of cells survive and repopulate the culture. A promising target for glioma therapy is the Notch signaling pathway. Notch activity is upregulated in many gliomas and can be suppressed using gamma-secretase inhibitors (GSI). Using a neurosphere recovery assay and xenograft experiments, we analyzed if the addition of GSIs with TMZ treatment could inhibit repopulation and tumor recurrence. We show that TMZ + GSI treatment decreased neurosphere formation and inhibited neurosphere recovery. This enhancement of TMZ treatment occurred through inhibition of the Notch pathway and depended on the sequence of drug administration. In addition, ex vivo TMZ + GSI treatment of glioma xenografts in immunocompromised mice extended tumor latency and survival, and in vivo TMZ + GSI treatment blocked tumor progression in 50% of mice with preexisting tumors. These data show the importance of the Notch pathway in chemoprotection and repopulation of TMZ-treated gliomas. The addition of GSIs to current treatments is a promising approach to decrease brain tumor recurrence.
MeSH term(s)	Amyloid Precursor Protein Secretases/antagonists & inhibitors ; Animals ; Antineoplastic Agents, Alkylating/pharmacology ; Antineoplastic Combined Chemotherapy Protocols/pharmacology ; Brain Neoplasms/drug therapy ; Brain Neoplasms/enzymology ; Brain Neoplasms/pathology ; Cell Line, Tumor ; Dacarbazine/administration & dosage ; Dacarbazine/analogs & derivatives ; Dacarbazine/pharmacology ; Dipeptides/administration & dosage ; Dipeptides/pharmacology ; Glioblastoma/drug therapy ; Glioblastoma/enzymology ; Glioblastoma/pathology ; Humans ; Mice ; Neoplasm Recurrence, Local/drug therapy ; Neoplasm Recurrence, Local/enzymology ; Neoplasm Recurrence, Local/pathology ; RNA, Messenger/biosynthesis ; RNA, Messenger/genetics ; Receptors, Notch/biosynthesis ; Receptors, Notch/genetics ; Receptors, Notch/metabolism ; Signal Transduction/drug effects ; Spheroids, Cellular ; Temozolomide ; Tumor Cells, Cultured ; Xenograft Model Antitumor Assays
Chemical Substances	Antineoplastic Agents, Alkylating ; Dipeptides ; N-(N-(3,5-difluorophenacetyl)alanyl)phenylglycine tert-butyl ester ; RNA, Messenger ; Receptors, Notch ; Dacarbazine (7GR28W0FJI) ; Amyloid Precursor Protein Secretases (EC 3.4.-) ; Temozolomide (YF1K15M17Y)
Language	English
Publishing date	2010-08-24
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	1432-1
ISSN	1538-7445 ; 0008-5472
ISSN (online)	1538-7445
ISSN	0008-5472
DOI	10.1158/0008-5472.CAN-10-1378
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