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Article: Clinicopathologic features of a patient with primary monophasic synovial sarcoma of the jejunum.

Wang, Yajian / Jiang, Na / Jiang, Yina / Wang, Hongyan

Zhejiang da xue xue bao. Yi xue ban = Journal of Zhejiang University. Medical sciences

2023 Volume 51, Issue 4, Page(s) 474–479

Abstract: A case of primary synovial sarcoma of the jejunum was collected and analyzed retrospectively. A 19-year-old man who presented to hospital with abdominal pain. The CT scan showed a large mixed abdominal mass with bleeding. Laparotomy revealed that the ... ...

Abstract	A case of primary synovial sarcoma of the jejunum was collected and analyzed retrospectively. A 19-year-old man who presented to hospital with abdominal pain. The CT scan showed a large mixed abdominal mass with bleeding. Laparotomy revealed that the tumor originated from the jejunum, accompanied by rupture and hemorrhage. Microscopically, the tumor was composed of spindle cells. The tumor cells demonstrated diffuse expression of vimentin, transducin-like enhancer (TLE)-1, B-cell lymphoma protein (Bcl)-2, CD99 and focal expression of epithelial membrane antigen (EMA). The presence of specific
MeSH term(s)	Male ; Humans ; Young Adult ; Adult ; Sarcoma, Synovial/diagnosis ; Sarcoma, Synovial/genetics ; Sarcoma, Synovial/pathology ; Jejunum/metabolism ; Jejunum/pathology ; Retrospective Studies ; Biomarkers, Tumor/genetics
Chemical Substances	Biomarkers, Tumor
Language	English
Publishing date	2023-05-16
Publishing country	China
Document type	Case Reports ; Journal Article
ISSN	1008-9292
ISSN	1008-9292
DOI	10.3724/zdxbyxb-2022-0117
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Article ; Online: Amino-Acid-Encoded Bioinspired Supramolecular Self-Assembly of Multimorphological Nanocarriers.

Huang, Yifan / Yang, Guokun / Yu, Zian / Tong, Tong / Huang, Yan / Zhang, Qianzijing / Hong, Yajian / Jiang, Jun / Zhang, Guozhen / Yuan, Yue

Small (Weinheim an der Bergstrasse, Germany)

2024 , Page(s) e2311351

Abstract: Supramolecular self-assembly has emerged as an efficient tool to construct well-organized nanostructures for biomedical applications by small organic molecules. However, the physicochemical properties of self-assembled nanoarchitectures are greatly ... ...

Abstract	Supramolecular self-assembly has emerged as an efficient tool to construct well-organized nanostructures for biomedical applications by small organic molecules. However, the physicochemical properties of self-assembled nanoarchitectures are greatly influenced by their morphologies, mechanical properties, and working mechanisms, making it challenging to design and screen ideal building blocks. Herein, using a biocompatible firefly-sourced click reaction between the cyano group of 2-cyano-benzothiazole (CBT) and the 1,2-aminothiol group of cysteine (Cys), an amino-acid-encoded supramolecular self-assembly platform Cys(SEt)-X-CBT (X represents any amino acid) is developed to incorporate both covalent and noncovalent interactions for building diverse morphologies of nanostructures with bioinspired response mechanism, providing a convenient and rapid strategy to construct site-specific nanocarriers for drug delivery, cell imaging, and enzyme encapsulation. Additionally, it is worth noting that the biodegradation of Cys(SEt)-X-CBT generated nanocarriers can be easily tracked via bioluminescence imaging. By caging either the thiol or amino groups in Cys with other stimulus-responsive sites and modifying X with probes or drugs, a variety of multi-morphological and multifunctional nanomedicines can be readily prepared for a wide range of biomedical applications.
Language	English
Publishing date	2024-03-07
Publishing country	Germany
Document type	Journal Article
ZDB-ID	2168935-0
ISSN	1613-6829 ; 1613-6810
ISSN (online)	1613-6829
ISSN	1613-6810
DOI	10.1002/smll.202311351
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Article ; Online: An oncogenic enhancer encodes selective selenium dependency in AML.

Eagle, Kenneth / Jiang, Yajian / Shi, Xiangguo / Li, Minhua / Obholzer, Nikolaus D / Hu, Tianyuan / Perez, Monika W / Koren, Jošt Vrabič / Kitano, Ayumi / Yi, Joanna S / Lin, Charles Y / Nakada, Daisuke

Cell stem cell

2022 Volume 29, Issue 4, Page(s) 650

Language	English
Publishing date	2022-04-08
Publishing country	United States
Document type	Published Erratum
ZDB-ID	2375354-7
ISSN	1875-9777 ; 1934-5909
ISSN (online)	1875-9777
ISSN	1934-5909
DOI	10.1016/j.stem.2022.03.006
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Article ; Online: Cell intrinsic and extrinsic regulation of leukemia cell metabolism.

Jiang, Yajian / Nakada, Daisuke

International journal of hematology

2016 Volume 103, Issue 6, Page(s) 607–616

Abstract: Metabolic homeostasis is a fundamental property of cells that becomes dysregulated in cancer to meet the altered, often heightened, demand for metabolism for increased growth and proliferation. Oncogenic mutations can directly change cellular metabolism ... ...

Abstract	Metabolic homeostasis is a fundamental property of cells that becomes dysregulated in cancer to meet the altered, often heightened, demand for metabolism for increased growth and proliferation. Oncogenic mutations can directly change cellular metabolism in a cell-intrinsic manner, priming cells for malignancy. Additionally, cell-extrinsic cues from the microenvironment, such as hypoxia, nutrient availability, oxidative stress, and crosstalk from surrounding cells can also affect cancer cell metabolism, and produce metabolic heterogeneity within the tumor. Here, we highlight recent findings revealing the complexity and adaptability of leukemia cells to coordinate metabolism.
MeSH term(s)	Cell Proliferation ; Cellular Microenvironment ; Homeostasis ; Humans ; Leukemia/metabolism ; Leukemia/pathology
Language	English
Publishing date	2016-06
Publishing country	Japan
Document type	Journal Article ; Review
ZDB-ID	1076875-0
ISSN	1865-3774 ; 0917-1258 ; 0925-5710
ISSN (online)	1865-3774
ISSN	0917-1258 ; 0925-5710
DOI	10.1007/s12185-016-1958-6
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Article ; Online: An oncogenic enhancer encodes selective selenium dependency in AML.

Eagle, Kenneth / Jiang, Yajian / Shi, Xiangguo / Li, Minhua / Obholzer, Nikolaus P / Hu, Tianyuan / Perez, Monika W / Koren, Jošt Vrabič / Kitano, Ayumi / Yi, Joanna S / Lin, Charles Y / Nakada, Daisuke

Cell stem cell

2022 Volume 29, Issue 3, Page(s) 386–399.e7

Abstract: Deregulation of transcription is a hallmark of acute myeloid leukemia (AML) that drives oncogenic expression programs and presents opportunities for therapeutic targeting. By integrating comprehensive pan-cancer enhancer landscapes with genetic ... ...

Abstract	Deregulation of transcription is a hallmark of acute myeloid leukemia (AML) that drives oncogenic expression programs and presents opportunities for therapeutic targeting. By integrating comprehensive pan-cancer enhancer landscapes with genetic dependency mapping, we find that AML-enriched enhancers encode for more selective tumor dependencies. We hypothesized that this approach could identify actionable dependencies downstream of oncogenic driver events and discovered a MYB-regulated AML-enriched enhancer regulating SEPHS2, a key component of the selenoprotein production pathway. Using a combination of patient samples and mouse models, we show that this enhancer upregulates SEPHS2, promoting selenoprotein production and antioxidant function required for AML survival. SEPHS2 and other selenoprotein pathway genes are required for AML growth in vitro. SEPHS2 knockout and selenium dietary restriction significantly delay leukemogenesis in vivo with little effect on normal hematopoiesis. These data validate the utility of enhancer mapping in target identification and suggest that selenoprotein production is an actionable target in AML.
MeSH term(s)	Animals ; Carcinogenesis/genetics ; Enhancer Elements, Genetic/genetics ; Humans ; Leukemia, Myeloid, Acute/pathology ; Mice ; Oncogenes ; Selenium/therapeutic use
Chemical Substances	Selenium (H6241UJ22B)
Language	English
Publishing date	2022-02-01
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	2375354-7
ISSN	1875-9777 ; 1934-5909
ISSN (online)	1875-9777
ISSN	1934-5909
DOI	10.1016/j.stem.2022.01.003
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Article ; Online: Nuclear NAD

Shi, Xiangguo / Jiang, Yajian / Kitano, Ayumi / Hu, Tianyuan / Murdaugh, Rebecca L / Li, Yuan / Hoegenauer, Kevin A / Chen, Rui / Takahashi, Koichi / Nakada, Daisuke

Science advances

2021 Volume 7, Issue 30

Abstract: Metabolic dysregulation underlies malignant phenotypes attributed to cancer stem cells, such as unlimited proliferation and differentiation blockade. Here, we demonstrate that ... ...

Abstract	Metabolic dysregulation underlies malignant phenotypes attributed to cancer stem cells, such as unlimited proliferation and differentiation blockade. Here, we demonstrate that NAD
MeSH term(s)	Animals ; Apoptosis/genetics ; Homeostasis ; Humans ; Leukemia, Myeloid, Acute/drug therapy ; Leukemia, Myeloid, Acute/genetics ; Mice ; NAD/metabolism ; Neoplastic Stem Cells/metabolism ; Nicotinamide-Nucleotide Adenylyltransferase/genetics ; Nicotinamide-Nucleotide Adenylyltransferase/metabolism
Chemical Substances	NAD (0U46U6E8UK) ; NMNAT1 protein, human (EC 2.7.7.1) ; Nicotinamide-Nucleotide Adenylyltransferase (EC 2.7.7.1) ; Nmnat1 protein, mouse (EC 2.7.7.1)
Language	English
Publishing date	2021-07-21
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	2810933-8
ISSN	2375-2548 ; 2375-2548
ISSN (online)	2375-2548
ISSN	2375-2548
DOI	10.1126/sciadv.abf3895
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Article ; Online: A stone miner with both silicosis and constrictive pericarditis: case report and review of the literature.

Jiang, Yajian / Shao, Fangchun

BMC pulmonary medicine

2013 Volume 13, Page(s) 71

Abstract: Background: The working environment of stone miners has been believed to cause their susceptibility to respiratory diseases. Silicosis is an occupational disease caused by exposure to crystalline silica dust which is marked by inflammation and scarring ... ...

Abstract	Background: The working environment of stone miners has been believed to cause their susceptibility to respiratory diseases. Silicosis is an occupational disease caused by exposure to crystalline silica dust which is marked by inflammation and scarring in the lung. The immune system boosted after the silica invasion led to self-damage and lay the foundation of silicosis pathogenesis. Silicosis coexisting with other diseases in one patient has been reported, however, was not reported to coexist with constrictive pericarditis. We, for the first time, reported a patient with silicosis and constrictive pericarditis and thought the immune response was probably the link between the two. Case presentation: A 59-year-old Chinese stone miner complained of chest distress was found to have lung nodules which were found to be silica deposits by biopsy. This patient was also found to have constrictive pericarditis at the same time. Later surgical decortication cured his symptoms. Conclusion: We provided the first case having constrictive pericarditis concomitant with silicosis. A probable link between the two diseases was the immune response boosted by the silica deposits.
MeSH term(s)	Air Pollutants, Occupational/immunology ; Air Pollutants, Occupational/toxicity ; Dust ; Humans ; Male ; Middle Aged ; Mining ; Pericarditis, Constrictive/complications ; Pericarditis, Constrictive/immunology ; Pericarditis, Constrictive/surgery ; Silicon Dioxide/immunology ; Silicon Dioxide/toxicity ; Silicosis/complications ; Silicosis/immunology
Chemical Substances	Air Pollutants, Occupational ; Dust ; Silicon Dioxide (7631-86-9)
Language	English
Publishing date	2013-12-06
Publishing country	England
Document type	Case Reports ; Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	2059871-3
ISSN	1471-2466 ; 1471-2466
ISSN (online)	1471-2466
ISSN	1471-2466
DOI	10.1186/1471-2466-13-71
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Article ; Online: Decreased Ratio of VEGF165b/VEGF in Aqueous Humor Predicts Progression of Diabetic Retinopathy.

Jiang, Feng / Chong, Liuyun / Du, Shufang / Duan, Yajian / Wang, Ying / Wang, Jiaxing / Chen, Song / He, Tiangeng

Ophthalmic research

2020 Volume 63, Issue 6, Page(s) 517–523

Abstract: Background: Different splicing of vascular endothelial growth factor (VEGF) gene results in 2 families of VEGF, the proangiogenic isoforms (VEGFxxxa) and the antiangiogenic isoforms (VEGFxxxb). VEGF165b is the major antiangiogenic isoform of VEGF and ... ...

Abstract	Background: Different splicing of vascular endothelial growth factor (VEGF) gene results in 2 families of VEGF, the proangiogenic isoforms (VEGFxxxa) and the antiangiogenic isoforms (VEGFxxxb). VEGF165b is the major antiangiogenic isoform of VEGF and the most studied member of the VEGFxxxb family so far. Objectives: To determine the concentration of VEGF165b and VEGF in the aqueous humor (AH) in diabetic eyes with or without diabetic retinopathy (DR) and to address the predictive value of VEGF165b/VEGF ratio for progression of DR. Methods: AH samples from 20 eyes in healthy controls (CON group), 40 eyes in diabetic patients without DR (nDR group), and 30 eyes in diabetic patients with mild nonproliferative DR (DR group) were collected. All of the patients were followed up for at least 5 years. VEGF165b and VEGF levels of AH samples were measured by enzyme-linked immunosorbent assay (ELISA). The predictive value of the initial VEGF165b/VEGF ratio for progression of DR was studied. Results: The mean concentration of VEGF165b significantly decreased in diabetic eyes vs. controls. The mean concentration of VEGF significantly increased in the DR group vs. the CON group. The VEGF165b/VEGF ratio was significantly lower in diabetic patients compared to the CON group. The VEGF165b/VEGF ratio was significantly lower in diabetic patients compared to the control group. The mean follow-up was 66.1months (range 60-71 months). The risk of DR progression was greater with a lower VEGF165b/VEGF ratio. Conclusion: The VEGF165b/VEGF ratio is lower in the AH of DR patients and the decreased ratio of VEGF165b/VEGF predicts DR progression.
MeSH term(s)	Aged ; Aged, 80 and over ; Aqueous Humor/metabolism ; Biomarkers/metabolism ; Diabetic Retinopathy/metabolism ; Disease Progression ; Female ; Humans ; Male ; Middle Aged ; Retrospective Studies ; Vascular Endothelial Growth Factor A/metabolism
Chemical Substances	Biomarkers ; Vascular Endothelial Growth Factor A
Language	English
Publishing date	2020-04-28
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	205708-6
ISSN	1423-0259 ; 0030-3747
ISSN (online)	1423-0259
ISSN	0030-3747
DOI	10.1159/000508250
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Article ; Online: Clonal expansion and myeloid leukemia progression modeled by multiplex gene editing of murine hematopoietic progenitor cells.

Shi, Xiangguo / Kitano, Ayumi / Jiang, Yajian / Luu, Victor / Hoegenauer, Kevin A / Nakada, Daisuke

Experimental hematology

2018 Volume 64, Page(s) 33–44.e5

Abstract: Recent advances in next-generation sequencing have identified novel mutations and revealed complex genetic architectures in human hematological malignancies. Moving forward, new methods to quickly generate animal models that recapitulate the complex ... ...

Abstract	Recent advances in next-generation sequencing have identified novel mutations and revealed complex genetic architectures in human hematological malignancies. Moving forward, new methods to quickly generate animal models that recapitulate the complex genetics of human hematological disorders are needed to transform the genetic information to new therapies. Here, we used a ribonucleoprotein-based CRISPR/Cas9 system to model human clonal hematopoiesis of indeterminate potential and acute myeloid leukemia (AML). We edited multiple genes recurrently mutated in hematological disorders, including those encoding epigenetic regulators, transcriptional regulators, and signaling components in murine hematopoietic stem/progenitor cells. Tracking the clonal dynamics by sequencing the indels induced by CRISPR/Cas9 revealed clonal expansion in some recipient mice that progressed to AML initiated by leukemia-initiating cells. Our results establish that the CRISPR/Cas9-mediated multiplex mutagenesis can be used to engineer a variety of murine models of hematological malignancies with complex genetic architectures seen in human disease.
MeSH term(s)	Animals ; Bone Marrow Transplantation ; CRISPR-Associated Protein 9 ; CRISPR-Cas Systems ; Clone Cells/pathology ; Disease Models, Animal ; Disease Progression ; Female ; Gene Editing/methods ; Genes, Neoplasm ; Hematopoietic Stem Cells/pathology ; Humans ; INDEL Mutation ; Leukemia, Myeloid, Acute/genetics ; Leukemia, Myeloid, Acute/pathology ; Male ; Mice ; Mice, Inbred C57BL ; Mutation ; Neoplastic Stem Cells/pathology ; Preleukemia/genetics ; Preleukemia/pathology ; Ribonucleoproteins/genetics ; Specific Pathogen-Free Organisms
Chemical Substances	Ribonucleoproteins ; CRISPR-Associated Protein 9 (EC 3.1.-)
Language	English
Publishing date	2018-05-08
Publishing country	Netherlands
Document type	Journal Article ; Research Support, N.I.H., Extramural
ZDB-ID	185107-x
ISSN	1873-2399 ; 0531-5573 ; 0301-472X
ISSN (online)	1873-2399
ISSN	0531-5573 ; 0301-472X
DOI	10.1016/j.exphem.2018.04.009
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Article ; Online: Mutation in BRAF and SMAD4 associated with resistance to neoadjuvant chemoradiation therapy in locally advanced rectal cancer.

Jiang, Dan / Wang, Xin / Wang, Yajian / Philips, Dana / Meng, Wenjian / Xiong, Moli / Zhao, Junyi / Sun, Linyong / He, Du / Li, Kun

Virchows Archiv : an international journal of pathology

2019 Volume 475, Issue 1, Page(s) 39–47

Abstract: Our study was done in order to identify novel molecular markers to predict which locally advanced rectal cancers (LARCs) might be resistant to neoadjuvant chemoradiotherapy (nCRT). Seventy-four patients with LARCs treated with nCRT were collected. ... ...

Abstract	Our study was done in order to identify novel molecular markers to predict which locally advanced rectal cancers (LARCs) might be resistant to neoadjuvant chemoradiotherapy (nCRT). Seventy-four patients with LARCs treated with nCRT were collected. Pathological evaluation after nCRT was performed according to the tumor regression grading (TRG) system. Next-generation sequencing kit including 279 exons of 59 genes was performed on Illumina Miseq Platform. Sanger sequencing was performed to confirm some mutations. Four of the tumors (4/74, 5.4%) had BRAF mutation, which presented in one TRG 2 tumor and three TRG 3 tumors but was not observed in TRG 0-1 tumors. Higher mutational frequency of BRAF gene in TRG 3 tumors (3/12, 25%) was found in comparison with the TRG 0-2 tumors (1/62, 1.6%; p = 0.012). Eight tumors (8/74, 10.8%) harbored SMAD4 mutations, which was mutated across all TRG groups. However, SMAD4 mutated more in TRG 3 tumors (4/12, 33.3%) compared with that in TRG 0-2 tumors (4/62, 6.5%; p = 0.020). The patients with BRAF-mutated LARCs had shorter progression-free survival (PFS) (p = 0.045) and shorter overall survival (OS) (p = 0.000) than the BRAF wild-type (WT) ones. The patients with SMAD4-mutated tumors had shorter PFS than the WT cases (p = 0.008). BRAF and SMAD4 genetic mutations might be important molecular markers to predict resistance to nCRT and poor prognosis in LARCs. More cases are needed to confirm these findings in the near future.
MeSH term(s)	Adult ; Aged ; Biomarkers, Tumor/genetics ; Chemoradiotherapy, Adjuvant/adverse effects ; DNA Mutational Analysis ; Disease Progression ; Drug Resistance, Neoplasm/genetics ; Exons ; Female ; Genetic Predisposition to Disease ; Humans ; Male ; Middle Aged ; Mutation ; Mutation Rate ; Neoadjuvant Therapy/adverse effects ; Neoplasm Grading ; Neoplasm Staging ; Phenotype ; Progression-Free Survival ; Proto-Oncogene Proteins B-raf/genetics ; Radiation Tolerance/genetics ; Rectal Neoplasms/genetics ; Rectal Neoplasms/mortality ; Rectal Neoplasms/pathology ; Rectal Neoplasms/therapy ; Retrospective Studies ; Risk Factors ; Smad4 Protein/genetics ; Time Factors
Chemical Substances	Biomarkers, Tumor ; SMAD4 protein, human ; Smad4 Protein ; BRAF protein, human (EC 2.7.11.1) ; Proto-Oncogene Proteins B-raf (EC 2.7.11.1)
Language	English
Publishing date	2019-05-06
Publishing country	Germany
Document type	Comparative Study ; Journal Article
ZDB-ID	1184867-4
ISSN	1432-2307 ; 0945-6317
ISSN (online)	1432-2307
ISSN	0945-6317
DOI	10.1007/s00428-019-02576-y
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