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Article ; Online: Study on the mechanism of inhibiting patulin production by fengycin.

Fu, Ruimin / Tang, Wei / Zhang, Hong / Zhang, Yulian / Wang, Ding / Chen, Wuling

2022 Volume 17, Issue 1, Page(s) 372–379

Abstract: Penicillium ... ...

Abstract	Penicillium expansum
Language	English
Publishing date	2022-04-19
Publishing country	Poland
Document type	Journal Article
ZDB-ID	2817958-4
ISSN	2391-5412 ; 2391-5412
ISSN (online)	2391-5412
ISSN	2391-5412
DOI	10.1515/biol-2022-0041
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Article ; Online: Effect of

Dong, Sashuang / Zeng, Qi / He, Weimin / Cheng, Wei / Zhang, Ling / Zhong, Ruimin / He, Wen / Fang, Xiang / Wei, Hong

Food & function

2024 Volume 15, Issue 8, Page(s) 3993–4009

Abstract: Frailty, a complex geriatric syndrome, significantly impedes the goal of achieving 'healthy aging'. Increasing evidence suggests a connection between gut microbiota, systemic inflammation, and disease. However, it remains to be determined whether ... ...

Abstract	Frailty, a complex geriatric syndrome, significantly impedes the goal of achieving 'healthy aging'. Increasing evidence suggests a connection between gut microbiota, systemic inflammation, and disease. However, it remains to be determined whether interventions targeting the intestinal flora can effectively ameliorate frailty. Our research involved fecal microbiota transplantation (FMT) experiments on germ-free (GF) mice, dividing these mice into three groups: a group receiving transplants from healthy elderly individuals (HF group), a group of frailty patients (FF group), and the FF group supplemented with
MeSH term(s)	Animals ; Lactobacillus plantarum ; Fecal Microbiota Transplantation ; Mice ; Gastrointestinal Microbiome ; Frailty/therapy ; Frailty/metabolism ; Female ; Humans ; Germ-Free Life ; Probiotics/pharmacology ; Disease Models, Animal ; Mice, Inbred C57BL ; Aged ; Feces/microbiology
Language	English
Publishing date	2024-04-22
Publishing country	England
Document type	Journal Article
ZDB-ID	2612033-1
ISSN	2042-650X ; 2042-6496
ISSN (online)	2042-650X
ISSN	2042-6496
DOI	10.1039/d3fo05282f
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Article ; Online: Giant Ovarian Corpus Luteum Cysts Associated with Natural Pregnancy in Woman with Polycystic Ovary Syndrome.

Zhu, Linling / Yang, Xinyun / Zhao, Ruimin / Chen, Lu / Wen, Hong

Journal of minimally invasive gynecology

2022 Volume 30, Issue 1, Page(s) 5–6

MeSH term(s)	Pregnancy ; Female ; Humans ; Polycystic Ovary Syndrome/complications ; Ovarian Cysts/complications ; Ovarian Cysts/surgery
Language	English
Publishing date	2022-09-19
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2186934-0
ISSN	1553-4669 ; 1553-4650
ISSN (online)	1553-4669
ISSN	1553-4650
DOI	10.1016/j.jmig.2022.09.008
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Zs.A 4205: Show issues

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Article ; Online: A rare KMT2A::CBL transcript in an acute monoblastic leukemia patient with an unfavorable outcome.

Yu, Jinglei / Song, Fengmei / Zhang, Mingming / Xiao, Pingnan / Feng, Jingjing / Hong, Ruimin / Hu, Yongxian / Huang, He / Wei, Guoqing

Molecular biology reports

2024 Volume 51, Issue 1, Page(s) 561

Abstract: Background: Lysine [K] methyltransferase 2A (KMT2A, previously known as MLL) gene rearrangements are common in acute leukemias of various lineages and are associated with features such as chemotherapy resistance and rapid relapse. KMT2A::CBL is a rare ... ...

Abstract	Background: Lysine [K] methyltransferase 2A (KMT2A, previously known as MLL) gene rearrangements are common in acute leukemias of various lineages and are associated with features such as chemotherapy resistance and rapid relapse. KMT2A::CBL is a rare fusion of unknown pathogenesis generated by a unique interstitial deletion of chromosome 11 that has been reported across a wide age range in both acute myeloid leukemia (AML) and acute lymphoblastic leukemia (ALL) patients. The leukemogenic effect of the KMT2A::CBL rearrangement and its association with clinical prognosis have not been well clarified. Methods and results: We report the case of a 64-year-old female who was diagnosed with acute monoblastic leukemia (M5a) and who acquired the rare KMT2A::CBL fusion. The patient received multiple cycles of therapy but did not achieve remission and eventually succumbed to severe infection and disease progression. Additionally, we characterized the predicted KMT2A-CBL protein structure in this case to reveal the underlying leukemogenic mechanisms and summarized reported cases of hematological malignancies with KMT2A::CBL fusion to investigate the correlation of gene rearrangements with clinical outcomes. Conclusions: This report provides novel insights into the leukemogenic potential of the KMT2A::CBL rearrangement and the correlation between gene rearrangements and clinical outcomes.
MeSH term(s)	Female ; Humans ; Middle Aged ; Leukemia, Monocytic, Acute/genetics ; Leukemia ; Disease Progression ; Gene Rearrangement/genetics ; Hematologic Neoplasms
Language	English
Publishing date	2024-04-21
Publishing country	Netherlands
Document type	Case Reports ; Journal Article
ZDB-ID	186544-4
ISSN	1573-4978 ; 0301-4851
ISSN (online)	1573-4978
ISSN	0301-4851
DOI	10.1007/s11033-024-09543-0
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Zs.A 1140: Show issues

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Article ; Online: Precocious puberty or growth hormone deficiency as initial presentation in Mayer-Rokitansky-kuster-Hauser syndrome: a clinical report of 5 cases.

Ai, Zhuanzhuan / Zhu, Xiaoyun / Chen, Hong / Chen, Ruimin

BMC pediatrics

2022 Volume 22, Issue 1, Page(s) 418

Abstract: Background: We report five patients with Mayer-Rokitansky-Küster-Hauser syndrome (MRKHS), four of whom presented with precocious puberty and one with growth hormone deficiency (GHD. Our five children add to the growing endocrine data base of MRKHS.: ... ...

Abstract	Background: We report five patients with Mayer-Rokitansky-Küster-Hauser syndrome (MRKHS), four of whom presented with precocious puberty and one with growth hormone deficiency (GHD. Our five children add to the growing endocrine data base of MRKHS. Case presentation: We retrospectively reviewed clinical data of 5 MRKHS patients from 2017 to 2020. The clinical features, hormonal profiles, radiological imaging and genetic analyses were collated. The age range of the 5 patients at diagnosis was 6.7-9.1 years. Four presented with premature thelarche, and one presented with short stature. External genitalia were normal in all patients. Gonadotropin-releasing hormone stimulation tests for the 5 patients revealed peak luteinizing hormone and follicular stimulating hormone levels of 3.57, 6.24, 11.5, 4.44 and 4.97 IU/L and 9.41, 16.7, 13.8, 14.2 and 10.3 mIU/mL, respectively. Growth hormone stimulation for one patient with short stature was consistent with GHD with a peak level of GH was 7.30 ng/mL. Imaging disclosed advanced bone age in four patients and no skeletal abnormalities in any of the patients. Ultrasonography of the abdomen revealed bilateral polycystic kidneys in one patient. Pelvic magnetic resonance imaging confirmed no uterus in five patients. All of the patients had a normal karyotype (46, XX). In one patient, whole-exome sequencing detected a deletion of 17q12(chr17:36,046,434-36,105,050, hg19) encompassing the HNF1B gene. Conclusions: We report the unusual co-occurrence of precocious puberty and GHD in patients with MRKHS, highlighting that abnormal puberty and growth development may represent initial unexplained manifestations. Whether the deletion of 17q 22 begat GHD is unclear.
MeSH term(s)	46, XX Disorders of Sex Development/complications ; 46, XX Disorders of Sex Development/diagnosis ; 46, XX Disorders of Sex Development/genetics ; Child ; Child, Preschool ; Congenital Abnormalities/diagnosis ; Congenital Abnormalities/genetics ; Female ; Growth Hormone ; Humans ; Mullerian Ducts/abnormalities ; Puberty, Precocious/diagnosis ; Puberty, Precocious/etiology ; Retrospective Studies ; Vagina
Chemical Substances	Growth Hormone (9002-72-6)
Language	English
Publishing date	2022-07-14
Publishing country	England
Document type	Case Reports ; Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2041342-7
ISSN	1471-2431 ; 1471-2431
ISSN (online)	1471-2431
ISSN	1471-2431
DOI	10.1186/s12887-022-03474-0
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Article ; Online: Two novel AMHR2 gene variants in monozygotic twins with persistent Müllerian duct syndrome: A case report and functional study.

Chen, Hong / Lin, Peng / Yuan, Xin / Chen, Ruimin

Molecular genetics & genomic medicine

2022 Volume 10, Issue 8, Page(s) e1999

Abstract: Background: Persistent Müllerian duct syndrome (PMDS) is an autosomal recessive congenital abnormality in which Müllerian derivatives, uterus, cervix, upper two-thirds of the vagina, and fallopian tubes persist in otherwise normally virilized males. ... ...

Abstract	Background: Persistent Müllerian duct syndrome (PMDS) is an autosomal recessive congenital abnormality in which Müllerian derivatives, uterus, cervix, upper two-thirds of the vagina, and fallopian tubes persist in otherwise normally virilized males. Mutations in anti-Müllerian hormone (AMH) and AMH receptor type II (AMHR2) genes have been identified as causative. However, functional experimental analysis of AMHR2 or AMH variants that cause PMDS is still lacking. Materials and methods: A Chinese Han family affected by PMDS was identified. To assess the history and clinical manifestations of PMDS, physical, operational, ultrasonographical, pathological, and other examinations were performed on family members. The variant screening was conducted using trio whole-exome sequencing (trio WES) and Sanger sequencing. Complementation-based NanoLuciferase Binary Technology (NanoBiT) was used to examine the interaction between AMH and AMHR2 variants in vivo. The effect of the two variants on the transcriptional activity of the TGFβ/BMP pathway was evaluated using a luciferase assay. Results: Classic phenotypic manifestations of PMDS in a pair of identical twins were described and confirmed by genetic sequence analysis. Molecular studies revealed two novel variants c.118G > C [p.(Gly40Arg)], c.1222G > C [p.(Ala408Pro)] in the AMHR2 gene. The AMHR2 p.Gly40Arg variant reduces its ability to bind to AMH, while the p.Ala408Pro variant alters the kinase domain structure. Both variants significantly reduce TGFβ/BMP signaling. Conclusion: Two missense AMHR2 variants associated with PMDS were identified. These findings provide novel insights toward better clinical evaluation and further understanding of the molecular basis of PMDS.
MeSH term(s)	Anti-Mullerian Hormone/genetics ; Anti-Mullerian Hormone/metabolism ; Disorder of Sex Development, 46,XY/genetics ; Female ; Humans ; Male ; Transforming Growth Factor beta ; Twins, Monozygotic
Chemical Substances	Transforming Growth Factor beta ; Anti-Mullerian Hormone (80497-65-0)
Language	English
Publishing date	2022-06-02
Publishing country	United States
Document type	Case Reports ; Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2734884-2
ISSN	2324-9269 ; 2324-9269
ISSN (online)	2324-9269
ISSN	2324-9269
DOI	10.1002/mgg3.1999
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Article ; Online: FOXD3 and GAB2 as a pair of rivals antagonistically control hepatocellular carcinogenesis.

Liu, Ruimin / Sun, Yan / Chen, Shuai / Hong, Yun / Lu, Zhongxian

The FEBS journal

2022 Volume 289, Issue 15, Page(s) 4536–4548

Abstract: Our previous study demonstrated that GAB2 promoted tumorigenesis in liver tissue and was a potential target for the treatment of hepatocellular carcinoma (HCC). Here, we identified that the tumour suppressor protein Forkhead box D3 (Foxd3) is a ... ...

Abstract	Our previous study demonstrated that GAB2 promoted tumorigenesis in liver tissue and was a potential target for the treatment of hepatocellular carcinoma (HCC). Here, we identified that the tumour suppressor protein Forkhead box D3 (Foxd3) is a transcriptional repressor of the Gab2 gene. In human HCC cells, FOXD3 expression is low, but GAB2 expression is abundant. Increased Foxd3 expression inhibited the expression of Gab2 in a dose-dependent manner. Ectopic expression of Foxd3 in HCC cells reduced Gab2-mediated promotion of cell proliferation and migration in vitro. Foxd3 also inhibited Gab2-stimulated phosphorylation of Jak2 and Stat3. Furthermore, the protein levels of Foxd3 and Gab2 had a clear negative correlation: Gab2 expression was induced, whereas Foxd3 expression was suppressed in most tumour tissues in mice with diethylnitrosamine (DEN)-induced hepatocellular carcinoma. These results suggest that the tumour suppressor Foxd3 and tumour enhancer Gab2 mutually inhibit each other to synergistically control the occurrence of HCC, providing a novel mechanism for treating this disease.
MeSH term(s)	Adaptor Proteins, Signal Transducing/genetics ; Adaptor Proteins, Signal Transducing/metabolism ; Animals ; Carcinogenesis/genetics ; Carcinoma, Hepatocellular/pathology ; Cell Line, Tumor ; Cell Proliferation/genetics ; Forkhead Transcription Factors/genetics ; Forkhead Transcription Factors/metabolism ; Gene Expression Regulation, Neoplastic ; Humans ; Liver Neoplasms/pathology ; Mice
Chemical Substances	Adaptor Proteins, Signal Transducing ; FOXD3 protein, human ; Forkhead Transcription Factors ; GAB2 protein, human ; Gab2 protein, mouse
Language	English
Publishing date	2022-02-25
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2173655-8
ISSN	1742-4658 ; 1742-464X
ISSN (online)	1742-4658
ISSN	1742-464X
DOI	10.1111/febs.16403
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Article: Acetobacter sp. improves the undesirable odors of fermented noni (Morinda citrifolia L.) juice

Zhang, Lin / Hong, Qiongping / Yu, Chengfeng / Wang, Ruimin / Li, Congfa / Liu, Sixin

Food chemistry. 2023 Feb. 01, v. 401

2023

Abstract: To clarify the role of Acetobacter sp. in fermented noni juice, the physiochemical properties, main active ingredients and volatile constituents were comprehensively analyzed. The sugar content and acidity tended to be stable after 12 days of ... ...

Abstract	To clarify the role of Acetobacter sp. in fermented noni juice, the physiochemical properties, main active ingredients and volatile constituents were comprehensively analyzed. The sugar content and acidity tended to be stable after 12 days of fermentation. Acetobacter sp. had no significant influence on major active ingredients of products. The headspace-gas chromatography-ion mobility spectrometry (HS-GC-IMS) were performed to describe the characteristic flavor profiles during fermentation. A total of 55 flavor compounds were screened with odour threshold and Kruskal-Wallis p < 0.05. Among them, 14 different biomarkers were selected with Variable Importance in Projection (VIP) greater than 1. The concentrations of ketones and aldehydes increased significantly, mainly contributing to the floral, fruit and green features. The content of hexanoic acid, octanoic acid and butanoic acid as the main source of peculiar odor were significantly reduced, indicating Acetobacter sp. could improve the unpleasant odor of fermented noni juice.
Keywords	Acetobacter ; Morinda citrifolia ; acidity ; biomarkers ; fermentation ; flavor ; food chemistry ; fruits ; hexanoic acid ; juices ; octanoic acid ; odors ; spectroscopy ; sugar content
Language	English
Dates of publication	2023-0201
Publishing place	Elsevier Ltd
Document type	Article
ZDB-ID	243123-3
ISSN	1873-7072 ; 0308-8146
ISSN (online)	1873-7072
ISSN	0308-8146
DOI	10.1016/j.foodchem.2022.134126
Database	NAL-Catalogue (AGRICOLA)

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Article ; Online: CTCF variant begets to short stature by down-regulation of IGF1.

Chen, Hong / Li, Weiyu / Zhang, Suping / Sun, Yunteng / Shen, Yiping / Chen, Ruimin

Journal of molecular endocrinology

2023 Volume 70, Issue 4

Abstract: Pathogenic variants in the transcription factor CCCTC-binding factor (CTCF) are associated with mental retardation, autosomal dominant 21 (MRD21, MIM#615502). Current studies supported the strong relationship between CTCF variants and growth, yet the ... ...

Abstract	Pathogenic variants in the transcription factor CCCTC-binding factor (CTCF) are associated with mental retardation, autosomal dominant 21 (MRD21, MIM#615502). Current studies supported the strong relationship between CTCF variants and growth, yet the mechanism of CTCF mutation leading to short stature is not known. Clinical information, treatment regimens, and follow-up outcomes of a patient with MRD21 were collected. The possible pathogenic mechanisms of CTCF variants leading to short stature were investigated using immortalized lymphocyte cell lines (LCLs), HEK-293T, and immortalized normal human liver cell lines (LO2). This patient received long-term treatment with recombinant human growth hormone (rhGH) which resulted in an increased height of 1.0 SDS. She had low serum insulin-like growth factor 1 (IGF1) before the treatment and the IGF1 level was not significantly increased during the treatment (-1.38 ± 0.61 SDS). The finding suggested that the CTCF R567W variant could have impaired IGF1 production pathway. We further demonstrated that the mutant CTCF had a reduced ability to bind to the promoter region of IGF1, consequently significantly reducing the transcriptional activation and expression of IGF1. Our novel results demonstrated a direct positive regulation of CTCF on the transcription of the IGF1 promoter. The impaired IGF1 expression due to CTCF mutation may explain the substandard effect of rhGH treatment on MRD21 patients. This study provided novel insights into the molecular basis of CTCF-associated disorder.
MeSH term(s)	Female ; Humans ; CCCTC-Binding Factor/genetics ; CCCTC-Binding Factor/metabolism ; Down-Regulation/genetics ; Human Growth Hormone/genetics ; Insulin-Like Growth Factor I/genetics ; Insulin-Like Growth Factor I/metabolism
Chemical Substances	CCCTC-Binding Factor ; Human Growth Hormone (12629-01-5) ; IGF1 protein, human ; Insulin-Like Growth Factor I (67763-96-6) ; CTCF protein, human
Language	English
Publishing date	2023-04-05
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	645012-x
ISSN	1479-6813 ; 0952-5041
ISSN (online)	1479-6813
ISSN	0952-5041
DOI	10.1530/JME-22-0193
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Zs.A 2406: Show issues

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Article ; Online: Biomarkers for Chimeric Antigen Receptor T Cell Therapy in Acute Lymphoblastic Leukemia: Prospects for Personalized Management and Prognostic Prediction.

Hong, Ruimin / Hu, Yongxian / Huang, He

Frontiers in immunology

2021 Volume 12, Page(s) 627764

Abstract: Chimeric antigen receptor (CAR) T cell therapy represents a breakthrough in immunotherapy with the potential of ushering in a new era in cancer treatment. Remarkable therapeutic response and complete remission of this innovative management have been ... ...

Abstract	Chimeric antigen receptor (CAR) T cell therapy represents a breakthrough in immunotherapy with the potential of ushering in a new era in cancer treatment. Remarkable therapeutic response and complete remission of this innovative management have been observed in patients with relapse/refractory acute lymphoblastic leukemia. With CAR-T cell therapy becoming widely used both in multicenter clinical trials and as a commercial treatment, therapeutic efficacy monitoring and management of toxicities will be indispensable for ensuring safety and improving overall survival. Biomarkers can act not only as effective indicators reflecting patients' baseline characteristics, CAR-T cell potency, and the immune microenvironment, but can also assess side effects during treatment. In this review, we will elaborate on a series of biomarkers associated with therapeutic response as well as treatment-related toxicities, and present their current condition and latent value with respect to the clinical utility. The combination of biomarker research and CAR-T cell therapy will contribute to establishing a safer and more powerful monitoring system and prolonging the event-free survival of patients.
MeSH term(s)	Animals ; Biomarkers, Pharmacological ; Humans ; Immunotherapy, Adoptive/methods ; Monitoring, Physiologic ; Precision Medicine ; Precursor Cell Lymphoblastic Leukemia-Lymphoma/diagnosis ; Precursor Cell Lymphoblastic Leukemia-Lymphoma/immunology ; Precursor Cell Lymphoblastic Leukemia-Lymphoma/therapy ; Prognosis ; Remission Induction ; T-Lymphocytes/immunology ; T-Lymphocytes/transplantation
Chemical Substances	Biomarkers, Pharmacological
Language	English
Publishing date	2021-02-25
Publishing country	Switzerland
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	2606827-8
ISSN	1664-3224 ; 1664-3224
ISSN (online)	1664-3224
ISSN	1664-3224
DOI	10.3389/fimmu.2021.627764
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