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  1. Article ; Online: African Ancestry Individuals with Higher Educational Attainment Are Resilient to Alzheimer's Disease Measured by pTau181.

    Rajabli, Farid / Seixas, Azizi A / Akgun, Bilcag / Adams, Larry D / Inciute, Jovita / Hamilton, Kara L / Whithead, Patrice G / Konidari, Ioanna / Gu, Tianjie / Arvizu, Jamie / Golightly, Charles G / Starks, Takiyah D / Laux, Renee / Byrd, Goldie S / Haines, Jonathan L / Beecham, Gary W / Griswold, Anthony J / Vance, Jeffery M / Cuccaro, Michael L /
    Pericak-Vance, Margaret A

    Journal of Alzheimer's disease : JAD

    2024  Volume 98, Issue 1, Page(s) 221–229

    Abstract: Background: Cognitive and functional abilities in individuals with Alzheimer's disease (AD) pathology (ADP) are highly variable. Factors contributing to this variability are not well understood. Previous research indicates that higher educational ... ...

    Abstract Background: Cognitive and functional abilities in individuals with Alzheimer's disease (AD) pathology (ADP) are highly variable. Factors contributing to this variability are not well understood. Previous research indicates that higher educational attainment (EA) correlates with reduced cognitive impairments among those with ADP. While cognitive and functional impairments are correlated, they are distinguishable in their manifestations.
    Objective: To investigate whether levels of education are associated with functional impairments among those with ADP.
    Methods: This research involved 410 African American (AA) individuals (Institutional Review Boards 20070307, 01/27/2023) to ascertain whether EA correlates with functional resilience and if this effect varies between APOE ɛ4 carriers and non-carriers. Utilizing EA as a cognitive reserve proxy, CDR-FUNC as a functional difficulties measure, and blood pTau181 as an ADP proxy, the non-parametric Mann-Whitney U test assessed the relationship between EA and CDR-FUNC in individuals with advanced pTau181 levels.
    Results: The results showed that EA correlated with functional difficulties in AA individuals with high levels of pTau181, such that individuals with high EA are more likely to have better functional ability compared to those with lower EA (W = 730.5, p = 0.0007). Additionally, we found that the effect of high EA on functional resilience was stronger in ɛ4 non-carriers compared to ɛ4 carriers (W = 555.5, p = 0.022).
    Conclusion: This study extends the role of cognitive reserve and EA to functional performance showing that cognitive reserve influences the association between ADP burden and functional difficulties. Interestingly, this protective effect seems less pronounced in carriers of the strong genetic risk allele ɛ4.
    MeSH term(s) Humans ; Alzheimer Disease/genetics ; Resilience, Psychological ; Apolipoprotein E4/genetics ; Cognitive Dysfunction/genetics ; Educational Status
    Chemical Substances Apolipoprotein E4
    Language English
    Publishing date 2024-02-23
    Publishing country Netherlands
    Document type Journal Article
    ZDB-ID 1440127-7
    ISSN 1875-8908 ; 1387-2877
    ISSN (online) 1875-8908
    ISSN 1387-2877
    DOI 10.3233/JAD-231116
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  2. Article ; Online: Closing the Gap in Antiretroviral Initiation and Viral Suppression: Time Trends and Racial Disparities.

    Haines, Charles F / Fleishman, John A / Yehia, Baligh R / Lau, Bryan / Berry, Stephen A / Agwu, Allison L / Moore, Richard D / Gebo, Kelly A

    Journal of acquired immune deficiency syndromes (1999)

    2016  Volume 73, Issue 3, Page(s) 340–347

    Abstract: Background: In the current antiretroviral (ART) era, the evolution of HIV guidelines and emergence of new ART agents might be expected to impact the times to ART initiation and HIV virologic suppression. We sought to determine if times to AI and ... ...

    Abstract Background: In the current antiretroviral (ART) era, the evolution of HIV guidelines and emergence of new ART agents might be expected to impact the times to ART initiation and HIV virologic suppression. We sought to determine if times to AI and virologic suppression decreased and if disparities exist by age, race/ethnicity, and HIV risk.
    Methods: We performed a retrospective cohort study of data from 12 sites of the HIV Research Network, a consortium of US clinics caring for HIV-infected patients. HIV-infected adults (≥18 year old) newly presenting for care between 2003 and 2013 were included in this study. Times to AI and virologic suppression were defined as time from enrollment to AI and HIV RNA <400 copies per milliliter, respectively. We conducted time-to-event analyses using competing risk regression in the HIV Research Network cohort from 2003 to 2012 in 2-year intervals, with follow-up through 2013.
    Results: Among 15,272 participants, 76.9% were male, 48.4% black, and 10.9% were injection drug use with median age of 38 years (interquartile range: 29-46 years). The adjusted subdistribution hazards ratios (SHRs) for AI and virologic suppression each increased for years 2007-2008 [SHR 1.23 (1.16-1.30), and SHR 1.25 (1.17-1.34), respectively], 2009-2010 [1.55 (1.46-1.64), and 1.54 (1.43-1.65), respectively], and 2011-2012 [1.94 (1.83-2.07), and 1.73 (1.61-1.86), respectively] compared with 2003-2004. Blacks had a lower probability of AI than whites and Hispanics.
    Conclusions: Since 2007, times from enrollment to AI and virologic suppression have decreased significantly compared with 2003-2004, but persisting disparities should be addressed.
    MeSH term(s) Adult ; African Continental Ancestry Group/statistics & numerical data ; Ambulatory Care ; Antiretroviral Therapy, Highly Active ; CD4 Lymphocyte Count ; European Continental Ancestry Group/statistics & numerical data ; Female ; Follow-Up Studies ; HIV Infections/drug therapy ; HIV Infections/ethnology ; Health Status Disparities ; Hispanic Americans/statistics & numerical data ; Humans ; Male ; Middle Aged ; Retrospective Studies ; Time Factors ; United States/epidemiology ; Viral Load
    Language English
    Publishing date 2016-10-24
    Publishing country United States
    Document type Journal Article ; Multicenter Study ; Research Support, Non-U.S. Gov't ; Research Support, N.I.H., Extramural ; Research Support, U.S. Gov't, P.H.S.
    ZDB-ID 645053-2
    ISSN 1944-7884 ; 1077-9450 ; 0897-5965 ; 0894-9255 ; 1525-4135
    ISSN (online) 1944-7884 ; 1077-9450
    ISSN 0897-5965 ; 0894-9255 ; 1525-4135
    DOI 10.1097/QAI.0000000000001114
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  3. Article: Generalizability of Tau and Amyloid Plasma Biomarkers in Alzheimer's Disease Cohorts of Diverse Genetic Ancestries.

    Griswold, Anthony J / Rajabli, Farid / Gu, Tianjie / Arvizu, Jamie / Golightly, Charles G / Whitehead, Patrice L / Hamilton-Nelson, Kara L / Adams, Larry D / Sanchez, Jose Javier / Mena, Pedro R / Starks, Takiyah D / Illanes-Manrique, Maryenela / Silva, Concepcion / Bush, William S / Cuccaro, Michael L / Vance, Jeffery M / Cornejo-Olivas, Mario R / Feliciano-Astacio, Briseida E / Byrd, Goldie S /
    Beecham, Gary W / Haines, Jonathan L / Pericak-Vance, Margaret A

    medRxiv : the preprint server for health sciences

    2024  

    Abstract: Introduction: Plasma phosphorylated threonine-181 of Tau and amyloid beta are biomarkers for differential diagnosis and preclinical detection of Alzheimer disease (AD). Given differences in AD risk across diverse populations, generalizability of ... ...

    Abstract Introduction: Plasma phosphorylated threonine-181 of Tau and amyloid beta are biomarkers for differential diagnosis and preclinical detection of Alzheimer disease (AD). Given differences in AD risk across diverse populations, generalizability of existing biomarker data is not assured.
    Methods: In 2,086 individuals of diverse genetic ancestries (African American, Caribbean Hispanic, and Peruvians) we measured plasma pTau-181 and Aβ42/Aβ40. Differences in biomarkers between cohorts and clinical diagnosis groups and the potential discriminative performance of the two biomarkers were assessed.
    Results: pTau-181 and Aβ42/Aβ40 were consistent across cohorts. Higher levels of pTau181 were associated with AD while Aβ42/Aβ40 had minimal differences. Correspondingly, pTau-181 had greater predictive value than Aβ42/Aβ40, however, the area under the curve differed between cohorts.
    Discussion: pTau-181 as a plasma biomarker for clinical AD is generalizable across genetic ancestries, but predictive value may differ. Combining genomic and biomarker data from diverse individuals will increase understanding of genetic risk and refine clinical diagnoses.
    Language English
    Publishing date 2024-04-12
    Publishing country United States
    Document type Preprint
    DOI 10.1101/2024.04.10.24305617
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  4. Article ; Online: Increase in CD4 count among new enrollees in HIV care in the modern antiretroviral therapy era.

    Haines, Charles F / Fleishman, John A / Yehia, Baligh R / Berry, Stephen A / Moore, Richard D / Bamford, Laura P / Gebo, Kelly A

    Journal of acquired immune deficiency syndromes (1999)

    2014  Volume 67, Issue 1, Page(s) 84–90

    Abstract: Background: Earlier HIV diagnosis and engagement in care improve outcomes and is cost effective, as a result, in 2006, the Centers for Disease Control and Prevention (CDC) revised the HIV-screening guidelines. We sought to determine whether the CD4 ... ...

    Abstract Background: Earlier HIV diagnosis and engagement in care improve outcomes and is cost effective, as a result, in 2006, the Centers for Disease Control and Prevention (CDC) revised the HIV-screening guidelines. We sought to determine whether the CD4 count (CD4) at presentation, a surrogate for time to presentation, increased during the study period. Our a priori hypothesis was that the CD4 at presentation increased during the study period, particularly after the CDC guideline revision.
    Methods: We performed a retrospective cohort study and analyzed data from the HIV Research Network, a consortium of 18 US clinics caring for HIV-infected patients. HIV-infected adults (≥18 years old) newly presenting for care between 2003 and 2011 were included in this study. Multivariable linear regression examined associations with CD4 at enrollment. Calendar year was modeled as a linear spline with a change in slope at 2008, allowing determination of the mean change in CD4 per year during 2003-2007 and 2008-2011.
    Results: Over 13,543 newly presenting subjects enrolled from 2003 to 2011. Median CD4 at enrollment rose from 285 to 317 cells per cubic millimeter between 2003-2007 and 2008-2011 (P < 0.001). After adjusting for age, race/ethnicity, gender, HIV risk factor, and clinic site, the mean increase in the CD4 count at presentation per year was 13.3 cells per cubic millimeter per year (95% confidence interval 6.4 to 20.1 cells per cubic millimeter per year) greater during 2008-2011 than during 2003-2007.
    Conclusions: We demonstrate a small, but statistically significant, increase in CD4 at presentation after the CDC guideline revision. More efforts are needed to decrease time to presentation to HIV care.
    MeSH term(s) Adult ; Anti-Retroviral Agents/administration & dosage ; CD4 Lymphocyte Count/statistics & numerical data ; Cohort Studies ; Female ; HIV Infections/drug therapy ; HIV Infections/epidemiology ; HIV Infections/immunology ; HIV Infections/virology ; HIV-1/isolation & purification ; Humans ; Linear Models ; Male ; Middle Aged ; Multivariate Analysis ; RNA, Viral/blood ; Retrospective Studies ; United States/epidemiology
    Chemical Substances Anti-Retroviral Agents ; RNA, Viral
    Language English
    Publishing date 2014-05-27
    Publishing country United States
    Document type Journal Article ; Multicenter Study ; Research Support, N.I.H., Extramural ; Research Support, U.S. Gov't, P.H.S.
    ZDB-ID 645053-2
    ISSN 1944-7884 ; 1077-9450 ; 0897-5965 ; 0894-9255 ; 1525-4135
    ISSN (online) 1944-7884 ; 1077-9450
    ISSN 0897-5965 ; 0894-9255 ; 1525-4135
    DOI 10.1097/QAI.0000000000000228
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    Zs.A 2442: Show issues Location:
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  5. Article ; Online: Clostridium difficile in a HIV-infected cohort: incidence, risk factors, and clinical outcomes.

    Haines, Charles F / Moore, Richard D / Bartlett, John G / Sears, Cynthia L / Cosgrove, Sara E / Carroll, Karen / Gebo, Kelly A

    AIDS (London, England)

    2013  Volume 27, Issue 17, Page(s) 2799–2807

    Abstract: Objective: Clostridium difficile is the most commonly reported infectious diarrhoea in HIV-infected patients in the United States. We set out to determine the incidence, risk factors and clinical presentation of C. difficile infections (CDIs) in a ... ...

    Abstract Objective: Clostridium difficile is the most commonly reported infectious diarrhoea in HIV-infected patients in the United States. We set out to determine the incidence, risk factors and clinical presentation of C. difficile infections (CDIs) in a cohort of HIV-infected individuals.
    Design: We performed a nested, case-control analysis with four non-CDI controls randomly selected for each case.
    Methods: We assessed the incidence of CDI in the Johns Hopkins HIV Clinical Cohort between 1 July 2003 and 31 December 2010. Incident cases were defined as first positive C. difficile cytotoxin assay or PCR for toxin B gene. We used conditional logistic regression models to assess risk factors for CDI. We abstracted data on the clinical presentation and outcomes from case chart review.
    Results: We identified 154 incident CDI cases for an incidence of 8.3 cases per 1000 patient years. No unique clinical features of HIV-associated CDI were identified. In multivariate analysis, risk of CDI was independently increased for CD4 cell count of 50 cells/μl or less [adjusted odds ratio (AOR) 20.7, 95% confidence interval (CI) 2.8-151.4], hospital onset CDI (AOR 26.7, 95% CI 3.1-231.2) and use of clindamycin (AOR 27.6, 95% CI 2.2-339.4), fluoroquinolones (AOR 4.5, 95% CI 1.2-17.5), macrolides (AOR 6.3, 95% CI 1.8-22.1), gastric acid suppressants (AOR 3.1, 95% CI 1.4-6.9) or immunosuppressive agents (AOR 6.8, 95% CI 1.2-39.6).
    Conclusion: The incidence of CDI in HIV-infected patients was twice that previously reported. Our data show that compromised cellular immunity, as defined by CD4 cell count of 50 cells/μl or less, is a risk factor for CDI. Clinicians should be aware of the increased CDI risk, particularly in those with severe CD4 cell count suppression.
    MeSH term(s) Adolescent ; Adult ; Aged ; CD4 Lymphocyte Count ; Case-Control Studies ; Clostridioides difficile/isolation & purification ; Clostridium Infections/epidemiology ; Clostridium Infections/microbiology ; Clostridium Infections/pathology ; Diarrhea/epidemiology ; Diarrhea/microbiology ; Diarrhea/pathology ; HIV Infections/complications ; Humans ; Incidence ; Male ; Middle Aged ; Retrospective Studies ; Risk Factors ; Treatment Outcome ; United States ; Young Adult
    Language English
    Publishing date 2013-07-09
    Publishing country England
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 639076-6
    ISSN 1473-5571 ; 0269-9370 ; 1350-2840
    ISSN (online) 1473-5571
    ISSN 0269-9370 ; 1350-2840
    DOI 10.1097/01.aids.0000432450.37863.e9
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  6. Article ; Online: Inhibition of estrogen signaling in myeloid cells increases tumor immunity in melanoma.

    Chakraborty, Binita / Byemerwa, Jovita / Shepherd, Jonathan / Haines, Corinne N / Baldi, Robert / Gong, Weida / Liu, Wen / Mukherjee, Debarati / Artham, Sandeep / Lim, Felicia / Bae, Yeeun / Brueckner, Olivia / Tavares, Kendall / Wardell, Suzanne E / Hanks, Brent A / Perou, Charles M / Chang, Ching-Yi / McDonnell, Donald P

    The Journal of clinical investigation

    2021  Volume 131, Issue 23

    Abstract: Immune checkpoint blockade (ICB) therapies have significantly prolonged patient survival across multiple tumor types, particularly in melanoma. Interestingly, sex-specific differences in response to ICB have been observed, with males receiving a greater ... ...

    Abstract Immune checkpoint blockade (ICB) therapies have significantly prolonged patient survival across multiple tumor types, particularly in melanoma. Interestingly, sex-specific differences in response to ICB have been observed, with males receiving a greater benefit from ICB than females, although the mechanism or mechanisms underlying this difference are unknown. Mining published transcriptomic data sets, we determined that the response to ICBs is influenced by the functionality of intratumoral macrophages. This puts into context our observation that estrogens (E2) working through the estrogen receptor α (ERα) stimulated melanoma growth in murine models by skewing macrophage polarization toward an immune-suppressive state that promoted CD8+ T cell dysfunction and exhaustion and ICB resistance. This activity was not evident in mice harboring macrophage-specific depletion of ERα, confirming a direct role for estrogen signaling within myeloid cells in establishing an immunosuppressed state. Inhibition of ERα using fulvestrant, a selective estrogen receptor downregulator (SERD), decreased tumor growth, stimulated adaptive immunity, and increased the antitumor efficacy of ICBs. Further, a gene signature that determines ER activity in macrophages predicted survival in patients with melanoma treated with ICB. These results highlight the importance of E2/ER signaling as a regulator of intratumoral macrophage polarization, an activity that can be therapeutically targeted to reverse immune suppression and increase ICB efficacy.
    MeSH term(s) Animals ; CD8-Positive T-Lymphocytes/cytology ; Cell Line, Tumor ; Estrogen Receptor alpha/metabolism ; Estrogens/metabolism ; Female ; Fulvestrant/pharmacology ; Humans ; Immune System ; Macrophages/metabolism ; Melanoma/immunology ; Melanoma/metabolism ; Melanoma, Experimental/metabolism ; Mice ; Mice, Inbred C57BL ; Myeloid Cells/metabolism ; Neoplasm Metastasis ; RNA, Small Cytoplasmic/metabolism ; Receptors, Estrogen ; Signal Transduction ; Skin Neoplasms/immunology ; Skin Neoplasms/metabolism ; Tumor Microenvironment
    Chemical Substances Estrogen Receptor alpha ; Estrogens ; RNA, Small Cytoplasmic ; Receptors, Estrogen ; Fulvestrant (22X328QOC4)
    Language English
    Publishing date 2021-10-25
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 3067-3
    ISSN 1558-8238 ; 0021-9738
    ISSN (online) 1558-8238
    ISSN 0021-9738
    DOI 10.1172/JCI151347
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  7. Article ; Online: Male-specific epistasis between WWC1 and TLN2 genes is associated with Alzheimer's disease.

    Gusareva, Elena S / Twizere, Jean-Claude / Sleegers, Kristel / Dourlen, Pierre / Abisambra, Jose F / Meier, Shelby / Cloyd, Ryan / Weiss, Blaine / Dermaut, Bart / Bessonov, Kyrylo / van der Lee, Sven J / Carrasquillo, Minerva M / Katsumata, Yuriko / Cherkaoui, Majid / Asselbergh, Bob / Ikram, M Arfan / Mayeux, Richard / Farrer, Lindsay A / Haines, Jonathan L /
    Pericak-Vance, Margaret A / Schellenberg, Gerard D / Sims, Rebecca / Williams, Julie / Amouyel, Philippe / van Duijn, Cornelia M / Ertekin-Taner, Nilüfer / Van Broeckhoven, Christine / Dequiedt, Franck / Fardo, David W / Lambert, Jean-Charles / Van Steen, Kristel

    Neurobiology of aging

    2018  Volume 72, Page(s) 188.e3–188.e12

    Abstract: Systematic epistasis analyses in multifactorial disorders are an important step to better characterize complex genetic risk structures. We conducted a hypothesis-free sex-stratified genome-wide screening for epistasis contributing to Alzheimer's disease ( ...

    Abstract Systematic epistasis analyses in multifactorial disorders are an important step to better characterize complex genetic risk structures. We conducted a hypothesis-free sex-stratified genome-wide screening for epistasis contributing to Alzheimer's disease (AD) susceptibility. We identified a statistical epistasis signal between the single nucleotide polymorphisms rs3733980 and rs7175766 that was associated with AD in males (genome-wide significant p
    MeSH term(s) Alzheimer Disease/genetics ; Cohort Studies ; Epistasis, Genetic/genetics ; Female ; Humans ; Intracellular Signaling Peptides and Proteins/genetics ; Male ; Meta-Analysis as Topic ; Phosphoproteins/genetics ; Sex Characteristics ; Sex Factors ; Talin/genetics
    Chemical Substances Intracellular Signaling Peptides and Proteins ; Phosphoproteins ; TLN2 protein, human ; Talin ; WWC1 protein, human
    Language English
    Publishing date 2018-08-09
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 604505-4
    ISSN 1558-1497 ; 0197-4580
    ISSN (online) 1558-1497
    ISSN 0197-4580
    DOI 10.1016/j.neurobiolaging.2018.08.001
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  8. Article ; Online: The human papillomavirus type 16 E7 oncoprotein activates the Fanconi anemia (FA) pathway and causes accelerated chromosomal instability in FA cells.

    Spardy, Nicole / Duensing, Anette / Charles, Domonique / Haines, Nathan / Nakahara, Tomomi / Lambert, Paul F / Duensing, Stefan

    Journal of virology

    2007  Volume 81, Issue 23, Page(s) 13265–13270

    Abstract: Fanconi anemia (FA) patients have an increased risk for squamous cell carcinomas (SCCs) at sites of predilection for infection with high-risk human papillomavirus (HPV) types, including the oral cavity and the anogenital tract. We show here that ... ...

    Abstract Fanconi anemia (FA) patients have an increased risk for squamous cell carcinomas (SCCs) at sites of predilection for infection with high-risk human papillomavirus (HPV) types, including the oral cavity and the anogenital tract. We show here that activation of the FA pathway is a frequent event in cervical SCCs. We found that FA pathway activation is triggered mainly by the HPV type 16 (HPV-16) E7 oncoprotein and is associated with an enhanced formation of large FANCD2 foci and recruitment of FANCD2 as well as FANCD1/BRCA2 to chromatin. Episomal expression of HPV-16 oncoproteins was sufficient to activate the FA pathway. Importantly, the expression of HPV-16 E7 in FA-deficient cells led to accelerated chromosomal instability. Taken together, our findings establish the FA pathway as an early host cell response to high-risk HPV infection and may help to explain the greatly enhanced susceptibility of FA patients to squamous cell carcinogenesis at anatomic sites that are frequently infected by high-risk HPVs.
    MeSH term(s) Apoptosis Regulatory Proteins ; BRCA2 Protein/metabolism ; Cell Line ; Chromatin/metabolism ; Chromosomal Instability ; Fanconi Anemia Complementation Group D2 Protein/metabolism ; Human papillomavirus 16/metabolism ; Humans ; Oncogene Proteins, Viral/metabolism ; Papillomavirus E7 Proteins ; Protein Binding
    Chemical Substances Apoptosis Regulatory Proteins ; BLID protein, human ; BRCA2 Protein ; BRCA2 protein, human ; Chromatin ; FANCD2 protein, human ; Fanconi Anemia Complementation Group D2 Protein ; Oncogene Proteins, Viral ; Papillomavirus E7 Proteins ; oncogene protein E7, Human papillomavirus type 16
    Language English
    Publishing date 2007-12
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural
    ZDB-ID 80174-4
    ISSN 1098-5514 ; 0022-538X
    ISSN (online) 1098-5514
    ISSN 0022-538X
    DOI 10.1128/JVI.01121-07
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  9. Article ; Online: Measurement of CP Violation in B^{0}→ψ(→ℓ^{+}ℓ^{-})K_{S}^{0}(→π^{+}π^{-}) Decays.

    Aaij, R / Abdelmotteleb, A S W / Abellan Beteta, C / Abudinén, F / Ackernley, T / Adeva, B / Adinolfi, M / Adlarson, P / Afsharnia, H / Agapopoulou, C / Aidala, C A / Ajaltouni, Z / Akar, S / Akiba, K / Albicocco, P / Albrecht, J / Alessio, F / Alexander, M / Alfonso Albero, A /
    Aliouche, Z / Alvarez Cartelle, P / Amalric, R / Amato, S / Amey, J L / Amhis, Y / An, L / Anderlini, L / Andersson, M / Andreianov, A / Andreola, P / Andreotti, M / Andreou, D / Ao, D / Archilli, F / Artamonov, A / Artuso, M / Aslanides, E / Atzeni, M / Audurier, B / Bacher, D / Bachiller Perea, I / Bachmann, S / Bachmayer, M / Back, J J / Bailly-Reyre, A / Baladron Rodriguez, P / Balagura, V / Baldini, W / Baptista de Souza Leite, J / Barbetti, M / Barbosa, I R / Barlow, R J / Barsuk, S / Barter, W / Bartolini, M / Baryshnikov, F / Basels, J M / Bassi, G / Batsukh, B / Battig, A / Bay, A / Beck, A / Becker, M / Bedeschi, F / Bediaga, I B / Beiter, A / Belin, S / Bellee, V / Belous, K / Belov, I / Belyaev, I / Benane, G / Bencivenni, G / Ben-Haim, E / Berezhnoy, A / Bernet, R / Bernet Andres, S / Berninghoff, D / Bernstein, H C / Bertella, C / Bertolin, A / Betancourt, C / Betti, F / Bex, J / Bezshyiko, Ia / Bhom, J / Bian, L / Bieker, M S / Biesuz, N V / Billoir, P / Biolchini, A 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    Physical review letters

    2024  Volume 132, Issue 2, Page(s) 21801

    Abstract: A measurement of time-dependent CP violation in the decays of B^{0} and B[over ¯]^{0} mesons to the final states J/ψ(→μ^{+}μ^{-})K_{S}^{0}, ψ(2S)(→μ^{+}μ^{-})K_{S}^{0} and J/ψ(→e^{+}e^{-})K_{S}^{0} with K_{S}^{0}→π^{+}π^{-} is presented. The data ... ...

    Abstract A measurement of time-dependent CP violation in the decays of B^{0} and B[over ¯]^{0} mesons to the final states J/ψ(→μ^{+}μ^{-})K_{S}^{0}, ψ(2S)(→μ^{+}μ^{-})K_{S}^{0} and J/ψ(→e^{+}e^{-})K_{S}^{0} with K_{S}^{0}→π^{+}π^{-} is presented. The data correspond to an integrated luminosity of 6  fb^{-1} collected at a center-of-mass energy of sqrt[s]=13  TeV with the LHCb detector. The CP-violation parameters are measured to be S_{ψK_{S}^{0}}=0.717±0.013(stat)±0.008(syst) and C_{ψK_{S}^{0}}=0.008±0.012(stat)±0.003(syst). This measurement of S_{ψK_{S}^{0}} represents the most precise single measurement of the CKM angle β to date and is more precise than the current world average. In addition, measurements of the CP-violation parameters of the individual channels are reported and a combination with the LHCb Run 1 measurements is performed.
    Language English
    Publishing date 2024-01-26
    Publishing country United States
    Document type Journal Article
    ZDB-ID 208853-8
    ISSN 1079-7114 ; 0031-9007
    ISSN (online) 1079-7114
    ISSN 0031-9007
    DOI 10.1103/PhysRevLett.132.021801
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  10. Article ; Online: Improved Measurement of CP Violation Parameters in B_{s}^{0}→J/ψK^{+}K^{-} Decays in the Vicinity of the ϕ(1020) Resonance.

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    Physical review letters

    2024  Volume 132, Issue 5, Page(s) 51802

    Abstract: The decay-time-dependent CP asymmetry in B_{s}^{0}→J/ψ(→μ^{+}μ^{-})K^{+}K^{-} decays is measured using proton-proton collision data, corresponding to an integrated luminosity of 6  fb^{-1}, collected with the LHCb detector at a center-of-mass energy of ... ...

    Abstract The decay-time-dependent CP asymmetry in B_{s}^{0}→J/ψ(→μ^{+}μ^{-})K^{+}K^{-} decays is measured using proton-proton collision data, corresponding to an integrated luminosity of 6  fb^{-1}, collected with the LHCb detector at a center-of-mass energy of 13 TeV. Using a sample of approximately 349 000 B_{s}^{0} signal decays with an invariant K^{+}K^{-} mass in the vicinity of the ϕ(1020) resonance, the CP-violating phase ϕ_{s} is measured, along with the difference in decay widths of the light and heavy mass eigenstates of the B_{s}^{0}-B[over ¯]_{s}^{0} system, ΔΓ_{s}, and the difference of the average B_{s}^{0} and B^{0} meson decay widths, Γ_{s}-Γ_{d}. The values obtained are ϕ_{s}=-0.039±0.022±0.006  rad, ΔΓ_{s}=0.0845±0.0044±0.0024  ps^{-1}, and Γ_{s}-Γ_{d}=-0.0056_{-0.0015}^{+0.0013}±0.0014  ps^{-1}, where the first uncertainty is statistical and the second systematic. These are the most precise single measurements to date and are consistent with expectations based on the Standard Model and with the previous LHCb analyses of this decay. These results are combined with previous independent LHCb measurements. The phase ϕ_{s} is also measured independently for each polarization state of the K^{+}K^{-} system and shows no evidence for polarization dependence.
    Language English
    Publishing date 2024-02-16
    Publishing country United States
    Document type Journal Article
    ZDB-ID 208853-8
    ISSN 1079-7114 ; 0031-9007
    ISSN (online) 1079-7114
    ISSN 0031-9007
    DOI 10.1103/PhysRevLett.132.051802
    Database MEDical Literature Analysis and Retrieval System OnLINE

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