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  1. Book ; Online ; E-Book: Neuropathology

    Prayson, Richard A. / Yeaney, Gabrielle A.

    a volume in the series foundations in diagnostic pathology

    (Foundations in diagnostic pathology)

    2024  

    Author's details edited by Richard A. Prayson, MD, MEd; Gabrielle Yeaney, MD
    Series title Foundations in diagnostic pathology
    Keywords Electronic books
    Language English
    Size 1 Online-Ressource (xiii, 610 Seiten), Illustrationen, Diagramme
    Edition Third edition
    Publisher Elsevier
    Publishing place Philadelphia, PA
    Publishing country United States
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT030008256
    ISBN 978-0-323-72228-5 ; 9780323713443 ; 0-323-72228-8 ; 0323713440
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  2. Book ; Online ; E-Book: Meningiomas

    Prayson, Richard A.

    from diagnosis to treatment

    (Rare disorders research progress ; Nova medicine & health)

    2022  

    Abstract: Meningiomas are among the most common primary tumors of the central neoplasm and are probably the most common primary nonglial tumor type. They are also the most common of the extra-axial tumors of the brain. Understanding of meningiomas and their ... ...

    Author's details Richard A. Prayson editor
    Series title Rare disorders research progress
    Nova medicine & health
    Abstract "Meningiomas are among the most common primary tumors of the central neoplasm and are probably the most common primary nonglial tumor type. They are also the most common of the extra-axial tumors of the brain. Understanding of meningiomas and their classification, grading and molecular biology and genetics has evolved significantly over the past few decades. Environmental and genetic factors have been implicated in the development of these neoplasms. They arise from meningothelial (arachnoidal cap cells), typically situated in the meninges, near the dura. However, these cells can be found in other locations less commonly which accounts for the development of meningiomas in unusual locations (sometimes outside the central nervous system). Although the majority of these tumors are considered low grade neoplasms, Classification classification and grading schemas have identified subtypes of meningiomas that are associated with more aggressive behavior or have unusual presentations. The fourteen chapters in this book address some of these various aspects of this diverse group of tumors"--
    Keywords Meningioma ; Meningioma/Diagnosis ; Meningioma/Treatment
    Subject code 616.99/481
    Language English
    Size 1 Online-Ressource (vi, 183 Seiten), Illustrationen
    Publisher Nova Medicine & Health
    Publishing place New York
    Publishing country United States
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT021392650
    ISBN 978-1-68507-807-2 ; 9781685077150 ; 1-68507-807-9 ; 1685077153
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  3. Book: Neuropathology

    Prayson, Richard A.

    (Foundations in diagnostic pathology ; Expert consult)

    2012  

    Author's details ed. by Richard A. Prayson
    Series title Foundations in diagnostic pathology
    Expert consult
    Keywords Nervous System Diseases / pathology ; Glioma / pathology
    Language English
    Edition 2. ed.
    Publisher Elsevier Saunders
    Publishing place Philadelphia, Pa
    Publishing country United States
    Document type Book
    Accompanying material Zugang zur Internetausgabe über Code
    HBZ-ID HT017100655
    ISBN 978-1-4377-0949-0 ; 1-4377-0949-4
    Database Catalogue ZB MED Medicine, Health

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  4. Book: Neuropathology review

    Prayson, Richard A.

    2008  

    Author's details Richard Prayson
    Keywords Nervous System Diseases ; Nervous system/Diseases
    Subject code 616.8047
    Language English
    Size XI, 252 S. : zahlr. Ill., 28cm
    Edition 2. ed.
    Publisher Humana Press
    Publishing place Totowa, NJ
    Publishing country United States
    Document type Book
    Accompanying material 1 CD-ROM (12 cm)
    HBZ-ID HT015388045
    ISBN 1-58829-895-7 ; 978-1-58829-895-9
    Database Catalogue ZB MED Medicine, Health

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  5. Article ; Online: Focal cortical dysplasia is a frequent coexistent pathology in patients with Rasmussen's encephalitis.

    Galev, Georgi / Prayson, Richard A

    Annals of diagnostic pathology

    2023  Volume 68, Page(s) 152224

    Abstract: Introduction: Rasmussen's encephalitis (RE) is a rare, predominantly pediatric epilepsy disorder of unknown etiology. It classically affects one of the cerebral hemispheres and histologically shows cortical chronic inflammation, gliosis, and neuronal ... ...

    Abstract Introduction: Rasmussen's encephalitis (RE) is a rare, predominantly pediatric epilepsy disorder of unknown etiology. It classically affects one of the cerebral hemispheres and histologically shows cortical chronic inflammation, gliosis, and neuronal loss. The etiopathogenesis of RE remains unknown, with genetic, infectious, and autoimmune factors all speculated to play a role. Although the histologic findings in RE are well described, few studies have investigated a large cohort of cases looking for the coexistence of RE with focal cortical dysplasia (FCD).
    Design: The study is a retrospective review of RE patients who underwent surgical resection of brain tissue between 1979 and 2021. Relevant patient history was retrieved, and available histologic slides were reviewed. The histologic severity of RE was described according to the Pardo criteria. In cases where FCD was present, the observed patterns of FCD (namely Ia, Ib, IIa, IIb, etc.) were described using the International League Against Epilepsy (ILAE) classification.
    Results: Thirty-eight resection specimens from 31 patients formed the study cohort. Seventeen patients (54.8 %) were male; average age at surgery was 8 years (range: 2-28 years). Twenty-seven resection specimens (71.1 %) from 23 patients (74 %) showed evidence of coexistent FCD. Most cases with FCD resembled the ILAE type Ib (n = 23) pattern. Cases of RE that did not show FCD were either Pardo stage 1 (n = 5) or 4 (n = 6), with all Pardo stage 2 and 3 cases demonstrating FCD.
    Conclusions: FCD was found in most patients with RE (74 %). The most observed pattern of FCD was ILAE Ib.
    MeSH term(s) Child ; Humans ; Male ; Child, Preschool ; Adolescent ; Young Adult ; Adult ; Female ; Focal Cortical Dysplasia ; Epilepsy/complications ; Epilepsy/pathology ; Encephalitis/complications ; Retrospective Studies ; Inflammation ; Malformations of Cortical Development/complications ; Malformations of Cortical Development/pathology ; Malformations of Cortical Development/surgery ; Magnetic Resonance Imaging
    Language English
    Publishing date 2023-11-03
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1440011-x
    ISSN 1532-8198 ; 1092-9134
    ISSN (online) 1532-8198
    ISSN 1092-9134
    DOI 10.1016/j.anndiagpath.2023.152224
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  6. Article: Metastatic gastrointestinal stromal tumor to the thoracic spine.

    Prayson, Richard A

    Clinical neuropathology

    2019  Volume 38, Issue 6, Page(s) 294–296

    MeSH term(s) Adult ; Gastrointestinal Neoplasms/pathology ; Gastrointestinal Stromal Tumors/secondary ; Humans ; Male ; Spinal Neoplasms/secondary ; Thoracic Vertebrae
    Language English
    Publishing date 2019-07-19
    Publishing country Germany
    Document type Case Reports ; Journal Article
    ZDB-ID 603167-5
    ISSN 0722-5091
    ISSN 0722-5091
    DOI 10.5414/NP301184
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  7. Book ; Online: Neuropathology Review

    Prayson, Richard

    2008  

    Author's details by Richard Prayson
    Keywords Neurology ; Neurosurgery ; Pathology
    Language English
    Edition Second Edition
    Publisher Humana Press
    Publishing place Totowa, NJ
    Document type Book ; Online
    HBZ-ID TT050387188
    ISBN 978-1-588-29895-9 ; 978-1-597-45219-9 ; 1-588-29895-7 ; 1-597-45219-X
    DOI 10.1007/978-1-59745-219-9
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  8. Article ; Online: IDH-1 polymorphisms in pilocytic astrocytomas.

    Prayson, Richard A

    Annals of diagnostic pathology

    2019  Volume 43, Page(s) 151411

    Abstract: As of 2016, isocitrate dehydrogenase (IDH)-1 and IDH-2 mutations are part of the definition of an oligodendroglioma and may be seen in a significant subset of grade II-IV fibrillary astrocytomas. Reports of IDH-1 and IDH-2 alterations in pilocytic ... ...

    Abstract As of 2016, isocitrate dehydrogenase (IDH)-1 and IDH-2 mutations are part of the definition of an oligodendroglioma and may be seen in a significant subset of grade II-IV fibrillary astrocytomas. Reports of IDH-1 and IDH-2 alterations in pilocytic astrocytomas have been rare. This study reports two cases of pilocytic astrocytomas which harbored IDH-1 polymorphisms (G105G) (c.315C > T) discovered on polymerase chain reaction (PCR) testing and sequencing. The first was encountered in a 21-year-old male with a right orbital frontal pole mass. The second occurred in a 19-year-old female with a right frontal tumor. Neither tumor stained with antibody to IDH-1 (R132H). No BRAF V600E immunostaining, minimal p53 staining (<5%) and no loss of ATRX staining was noted in both cases. The significance of the IDH-1 findings at this juncture is uncertain. Misdiagnosis of the tumor as a fibrillary astrocytoma or oligodendroglioma due to the presence of an IDH alteration should be avoided.
    MeSH term(s) Astrocytoma/genetics ; Astrocytoma/pathology ; Astrocytoma/surgery ; Central Nervous System Neoplasms/diagnostic imaging ; Central Nervous System Neoplasms/pathology ; Central Nervous System Neoplasms/surgery ; Diagnostic Errors ; Female ; Humans ; Isocitrate Dehydrogenase/genetics ; Male ; Mutation ; Oligodendroglioma/genetics ; Oligodendroglioma/pathology ; Oligodendroglioma/surgery ; Polymorphism, Genetic/genetics ; Treatment Outcome ; Young Adult
    Chemical Substances Isocitrate Dehydrogenase (EC 1.1.1.41) ; IDH1 protein, human (EC 1.1.1.42.)
    Language English
    Publishing date 2019-10-23
    Publishing country United States
    Document type Case Reports ; Journal Article
    ZDB-ID 1440011-x
    ISSN 1532-8198 ; 1092-9134
    ISSN (online) 1532-8198
    ISSN 1092-9134
    DOI 10.1016/j.anndiagpath.2019.151411
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  9. Book: Frozen section libary: central nervous system

    Prayson, Richard A. / Napekoski, Karl M.

    (Frozen section library ; [6])

    2011  

    Author's details Richard A. Prayson ; Karl M. Napekoski
    Series title Frozen section library ; [6]
    Frozen section library series
    Collection Frozen section library series
    Keywords Central Nervous System / anatomy & histology ; Central Nervous System / pathology ; Frozen Sections
    Language English
    Size XI, 152 S. : Ill., graph. Darst.
    Publisher Springer
    Publishing place New York u.a.
    Publishing country United States
    Document type Book
    Note Bd.-Zählung im Internet ermittelt
    HBZ-ID HT016691469
    ISBN 978-1-4419-7578-2 ; 9781441975799 ; 1-4419-7578-0 ; 1441975799
    Database Catalogue ZB MED Medicine, Health

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  10. Article: Tumor-to-tumor metastasis of an adenocarcinoma to a glioblastoma with review of the literature on tumor-to-tumor metastasis to gliomas.

    Galev, Georgi / Weigelt, Maximillian A / Prayson, Richard A

    Clinical neuropathology

    2023  Volume 42, Issue 5, Page(s) 190–196

    Abstract: Tumor-to-tumor metastasis (TTM) is a process where one tumor metastasizes to another tumor. It is an exceedingly rare phenomenon, particularly in the central nervous system, where it most commonly occurs with meningiomas as the recipient. Herein, we ... ...

    Abstract Tumor-to-tumor metastasis (TTM) is a process where one tumor metastasizes to another tumor. It is an exceedingly rare phenomenon, particularly in the central nervous system, where it most commonly occurs with meningiomas as the recipient. Herein, we present a case of tumor-to-tumor metastasis of an adenocarcinoma to a glioblastoma in a 75-year-old female. The patient had a history of high-grade ductal carcinoma in situ of the breast 8 years prior, treated with lumpectomy and radiation. She presented with a left fronto-parietal mass. Histologically, the lesion showed a glioblastoma, IDH-wildtype, WHO grade 4, associated with a metastatic adenocarcinoma (positive for estrogen receptor, progesterone receptor, and mammaglobin), suggesting a breast primary. The patient passed away 5 months after surgery. Involvement of glioblastoma by TTM is especially rare; only 1 case of TTM to glioblastoma is thus far reported in the English literature. The mechanism by which TTM occurs is poorly understood. TTM may be the first presentation of an occult malignancy and warrants thorough clinical, laboratory, and imaging investigation.
    MeSH term(s) Female ; Humans ; Aged ; Glioblastoma ; Adenocarcinoma/pathology ; Adenocarcinoma/secondary ; Adenocarcinoma/therapy ; Neoplasms, Second Primary
    Language English
    Publishing date 2023-10-01
    Publishing country Germany
    Document type Review ; Case Reports ; Journal Article
    ZDB-ID 603167-5
    ISSN 0722-5091
    ISSN 0722-5091
    DOI 10.5414/NP301578
    Database MEDical Literature Analysis and Retrieval System OnLINE

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