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Book ; Online: Frühkindliche Entwicklung stärken

Schütte, Kerstin / Rose, Heidemarie / Köller, Olaf / Köller, Olaf

Eine Zukunftsallianz aus Fachpraxis, Wissenschaft, Verwaltung, Politik und Zivilgesellschaft

2022

Keywords	Education ; BRISE - Bremer Initiative zur Stärkung frühkindlicher Entwicklung ; Familiäre Gesundheitsförderung ; Präventionsketten ; Wohlergehen aller Kinder
Language	German
Size	1 electronic resource (136 pages)
Publisher	Waxmann Verlag
Document type	Book ; Online
Note	German
HBZ-ID	HT030381046
ISBN	9783830945673 ; 3830945671
Database	ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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Article ; Online: Marked reduction of Na(+), K(+)-ATPase and creatine kinase activities induced by acute lysine administration in glutaryl-CoA dehydrogenase deficient mice.

Amaral, Alexandre Umpierrez / Cecatto, Cristiane / Seminotti, Bianca / Zanatta, Ângela / Fernandes, Carolina Gonçalves / Busanello, Estela Natacha Brandt / Braga, Luisa Macedo / Ribeiro, César Augusto João / de Souza, Diogo Onofre Gomes / Woontner, Michael / Koeller, David M / Goodman, Stephen / Wajner, Moacir

Molecular genetics and metabolism

2012 Volume 107, Issue 1-2, Page(s) 81–86

Abstract: ... α-ketoglutarate dehydrogenase (α-KGDH), creatine kinase (CK) and synaptic Na(+), K(+)-ATPase. No ... However, the most marked effects provoked by Lys administration were marked decreases of the activities of Na(+), K ... of Na(+), K(+)-ATPase and CK activities may play an important role in the pathogenesis ...

Abstract	Glutaric acidemia type I (GA I) is an inherited neurometabolic disorder caused by a severe deficiency of the mitochondrial glutaryl-CoA dehydrogenase activity leading to accumulation of predominantly glutaric (GA) and 3-hydroxyglutaric (3HGA) acids in the brain and other tissues. Affected patients usually present with hypotonia and brain damage and acute encephalopathic episodes whose pathophysiology is not yet fully established. In this study we investigated important parameters of cellular bioenergetics in brain, heart and skeletal muscle from 15-day-old glutaryl-CoA dehydrogenase deficient mice (Gcdh(-/-)) submitted to a single intra-peritoneal injection of saline (Sal) or lysine (Lys - 8 μmol/g) as compared to wild type (WT) mice. We evaluated the activities of the respiratory chain complexes II, II-III and IV, α-ketoglutarate dehydrogenase (α-KGDH), creatine kinase (CK) and synaptic Na(+), K(+)-ATPase. No differences of all evaluated parameters were detected in the Gcdh(-/-) relatively to the WT mice injected at baseline (Sal). Furthermore, mild increases of the activities of some respiratory chain complexes (II-III and IV) were observed in heart and skeletal muscle of Gcdh(-/-) and WT mice after Lys administration. However, the most marked effects provoked by Lys administration were marked decreases of the activities of Na(+), K(+)-ATPase in brain and CK in brain and skeletal muscle of Gcdh(-/-) mice. In contrast, brain α-KGDH activity was not altered in WT and Gcdh(-/-) injected with Sal or Lys. Our results demonstrate that reduction of Na(+), K(+)-ATPase and CK activities may play an important role in the pathogenesis of the neurodegenerative changes in GA I.
MeSH term(s)	Amino Acid Metabolism, Inborn Errors/drug therapy ; Amino Acid Metabolism, Inborn Errors/metabolism ; Animals ; Brain/drug effects ; Brain/metabolism ; Brain Diseases, Metabolic/drug therapy ; Brain Diseases, Metabolic/metabolism ; Creatine Kinase/metabolism ; Disease Models, Animal ; Electron Transport/drug effects ; Electron Transport/physiology ; Glutaryl-CoA Dehydrogenase/deficiency ; Glutaryl-CoA Dehydrogenase/metabolism ; Heart/drug effects ; Ketoglutarate Dehydrogenase Complex/metabolism ; Lysine/administration & dosage ; Lysine/pharmacology ; Mice ; Mice, Knockout ; Muscle, Skeletal/drug effects ; Muscle, Skeletal/metabolism ; Myocardium/metabolism ; Sodium-Potassium-Exchanging ATPase/metabolism
Chemical Substances	Ketoglutarate Dehydrogenase Complex (EC 1.2.4.2) ; Glutaryl-CoA Dehydrogenase (EC 1.3.8.6) ; Creatine Kinase (EC 2.7.3.2) ; Sodium-Potassium-Exchanging ATPase (EC 3.6.3.9) ; Lysine (K3Z4F929H6)
Language	English
Publishing date	2012-09
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1418518-0
ISSN	1096-7206 ; 1096-7192
ISSN (online)	1096-7206
ISSN	1096-7192
DOI	10.1016/j.ymgme.2012.04.015
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Article: Time course of inwardly rectifying K(+) current reduction in glial cells surrounding ischemic brain lesions.

Köller, H / Schroeter, M / Jander, S / Stoll, G / Siebler, M

Brain research

2000 Volume 872, Issue 1-2, Page(s) 194–198

Abstract: K(+) currents of activated glial cells surrounding ischemic infarcts are investigated using acutely ... rectifying K(+) currents (K(IR)) were markedly reduced in cells neighboring infarcts with maximal alteration ... at day 3 after infarct followed by a partial recovery. This reduction of glial K(IR) currents ...

Abstract	K(+) currents of activated glial cells surrounding ischemic infarcts are investigated using acutely dissociated cells from the periinfarct area after permanent middle cerebral artery occlusion in rats. Inwardly rectifying K(+) currents (K(IR)) were markedly reduced in cells neighboring infarcts with maximal alteration at day 3 after infarct followed by a partial recovery. This reduction of glial K(IR) currents may contribute to the functional disturbances in the periinfarct area.
MeSH term(s)	Animals ; Barium/pharmacology ; Brain Ischemia/metabolism ; Brain Ischemia/pathology ; Cells, Cultured ; Glial Fibrillary Acidic Protein/metabolism ; Infarction, Middle Cerebral Artery ; Ion Transport/drug effects ; Ion Transport/physiology ; Nerve Growth Factors ; Neuroglia/metabolism ; Neuroglia/pathology ; Patch-Clamp Techniques ; Potassium/metabolism ; Potassium Channels/metabolism ; Potassium Channels, Inwardly Rectifying ; Rats ; Rats, Wistar ; S100 Calcium Binding Protein beta Subunit ; S100 Proteins/metabolism
Chemical Substances	Glial Fibrillary Acidic Protein ; Nerve Growth Factors ; Potassium Channels ; Potassium Channels, Inwardly Rectifying ; S100 Calcium Binding Protein beta Subunit ; S100 Proteins ; Barium (24GP945V5T) ; Potassium (RWP5GA015D)
Language	English
Publishing date	2000-07-28
Publishing country	Netherlands
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1200-2
ISSN	1872-6240 ; 0006-8993
ISSN (online)	1872-6240
ISSN	0006-8993
DOI	10.1016/s0006-8993(00)02434-3
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Book ; Online: PISA 2018

Reiss, Kristina / Weis, Mirjam / Klieme, Eckhard / Köller, Olaf

2019

Abstract: Mit der PISA-Studie werden grundlegende Kompetenzen von Fünfzehnjährigen gegen Ende der Pflichtschulzeit erfasst. Ziel ist es, Aussagen darüber zu treffen, wie gut die Jugendlichen auf eine erfolgreiche Teilhabe an der modernen Gesellschaft vorbereitet ... ...

Abstract	Mit der PISA-Studie werden grundlegende Kompetenzen von Fünfzehnjährigen gegen Ende der Pflichtschulzeit erfasst. Ziel ist es, Aussagen darüber zu treffen, wie gut die Jugendlichen auf eine erfolgreiche Teilhabe an der modernen Gesellschaft vorbereitet sind. Die Ergebnisse der PISA-Studie ermöglichen alle drei Jahre den internationalen Vergleich von Kompetenzen Fünfzehnjähriger in den Bereichen Naturwissenschaften, Mathematik und Lesen und damit Aussagen über die Wirksamkeit von Bildungssystemen. Der nationale Berichtsband stellt die Ergebnisse der Schülerinnen und Schüler in Deutschland aus der PISA-Studie 2018 vor und setzt sie in Relation zu den Ergebnissen in anderen Staaten. Der Schwerpunkt der Erhebungen und Auswertungen liegt dabei auf der Lesekompetenz.Die Rahmenkonzeption der Lesekompetenz wurde für die PISA-Studie 2018 im Vergleich zu den vorigen Runden einer Revision unterzogen und berücksichtigt nun die sich verändernde Lesepraxis, die mit der Nutzung digitaler Medien einhergeht. Es werden leistungsbezogene Variablen genauso wie motivationale Orientierungen, Einstellungen und Verhalten erfasst, die für die Lesekompetenz wichtig sein können. Daneben wird der Beitrag der sozialen Herkunft und des Zuwanderungshintergrunds für die Lesekompetenzen untersucht. Darüber hinaus werden die Kompetenzen der Jugendlichen in der Mathematik und den Naturwissenschaften vorgestellt und diskutiert
Keywords	Education (General) ; Theory and practice of education ; Psychology
Size	1 electronic resource (266 pages)
Publisher	Waxmann Verlag
Document type	Book ; Online
Note	german ; Open Access
HBZ-ID	HT020481661
ISBN	9783830941002 ; 3830941005
DOI	10.31244/9783830991007
Database	ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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Article ; Online: Reduction of Na+, K+-ATPase activity and expression in cerebral cortex of glutaryl-CoA dehydrogenase deficient mice: a possible mechanism for brain injury in glutaric aciduria type I.

Amaral, Alexandre Umpierrez / Seminotti, Bianca / Cecatto, Cristiane / Fernandes, Carolina Gonçalves / Busanello, Estela Natacha Brandt / Zanatta, Ângela / Kist, Luiza Wilges / Bogo, Maurício Reis / de Souza, Diogo Onofre Gomes / Woontner, Michael / Goodman, Stephen / Koeller, David M / Wajner, Moacir

Molecular genetics and metabolism

2012 Volume 107, Issue 3, Page(s) 375–382

Abstract: ... reduction of Na(+), K(+)-ATPase activity associated with a lower degree of its expression in cerebral cortex ... observed in Gcdh-/- mice fed with a normal diet. Since Na(+), K(+)-ATPase activity is required ... ketoglutarate dehydrogenase (α-KGDH), creatine kinase (CK) and Na+, K+-ATPase in cerebral cortex, striatum and ...

Abstract	Mitochondrial dysfunction has been proposed to play an important role in the neuropathology of glutaric acidemia type I (GA I). However, the relevance of bioenergetics disruption and the exact mechanisms responsible for the cortical leukodystrophy and the striatum degeneration presented by GA I patients are not yet fully understood. Therefore, in the present work we measured the respiratory chain complexes activities I-IV, mitochondrial respiratory parameters state 3, state 4, the respiratory control ratio and dinitrophenol (DNP)-stimulated respiration (uncoupled state), as well as the activities of α-ketoglutarate dehydrogenase (α-KGDH), creatine kinase (CK) and Na+, K+-ATPase in cerebral cortex, striatum and hippocampus from 30-day-old Gcdh-/- and wild type (WT) mice fed with a normal or a high Lys (4.7%) diet. When a baseline (0.9% Lys) diet was given, we verified mild alterations of the activities of some respiratory chain complexes in cerebral cortex and hippocampus, but not in striatum from Gcdh-/- mice as compared to WT animals. Furthermore, the mitochondrial respiratory parameters and the activities of α-KGDH and CK were not modified in all brain structures from Gcdh-/- mice. In contrast, we found a significant reduction of Na(+), K(+)-ATPase activity associated with a lower degree of its expression in cerebral cortex from Gcdh-/- mice. Furthermore, a high Lys (4.7%) diet did not accentuate the biochemical alterations observed in Gcdh-/- mice fed with a normal diet. Since Na(+), K(+)-ATPase activity is required for cell volume regulation and to maintain the membrane potential necessary for a normal neurotransmission, it is presumed that reduction of this enzyme activity may represent a potential underlying mechanism involved in the brain swelling and cortical abnormalities (cortical atrophy with leukodystrophy) observed in patients affected by GA I.
MeSH term(s)	Amino Acid Metabolism, Inborn Errors/enzymology ; Amino Acid Metabolism, Inborn Errors/genetics ; Amino Acid Metabolism, Inborn Errors/pathology ; Animals ; Brain Diseases, Metabolic/enzymology ; Brain Diseases, Metabolic/genetics ; Brain Diseases, Metabolic/pathology ; Cerebral Cortex/enzymology ; Cerebral Cortex/pathology ; Corpus Striatum/enzymology ; Corpus Striatum/pathology ; Creatine Kinase/genetics ; Creatine Kinase/metabolism ; Down-Regulation ; Electron Transport/genetics ; Food, Formulated ; Gene Expression ; Glutaryl-CoA Dehydrogenase/deficiency ; Glutaryl-CoA Dehydrogenase/genetics ; Hippocampus/enzymology ; Hippocampus/pathology ; Humans ; Ketoglutarate Dehydrogenase Complex/genetics ; Ketoglutarate Dehydrogenase Complex/metabolism ; Mice ; Mice, Knockout ; Mitochondria/genetics ; Mitochondria/metabolism ; Oxidative Phosphorylation ; Sodium-Potassium-Exchanging ATPase/genetics ; Sodium-Potassium-Exchanging ATPase/metabolism
Chemical Substances	Ketoglutarate Dehydrogenase Complex (EC 1.2.4.2) ; Glutaryl-CoA Dehydrogenase (EC 1.3.8.6) ; Creatine Kinase (EC 2.7.3.2) ; Sodium-Potassium-Exchanging ATPase (EC 3.6.3.9)
Language	English
Publishing date	2012-11
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1418518-0
ISSN	1096-7206 ; 1096-7192
ISSN (online)	1096-7206
ISSN	1096-7192
DOI	10.1016/j.ymgme.2012.08.016
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Book ; Thesis: Effekt der frühen enteralen Ernährung mit Glutamin nach hämorrhagischem Schock und Sepsis im Rattenmodell

Köller, Kirsten

2002

Author's details	vorgelegt von Kirsten Köller
Language	German
Size	II, 51 Bl., graph. Darst.
Publishing country	Germany
Document type	Book ; Thesis
Thesis / German Habilitation thesis	Kiel, Univ., Diss., 2002
HBZ-ID	HT013361098
Database	Catalogue ZB MED Medicine, Health

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2002 D 1958	order for loan	Magazin

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Article: Marked reduction of Na⁺, K⁺-ATPase and creatine kinase activities induced by acute lysine administration in glutaryl-CoA dehydrogenase deficient mice

Molecular genetics and metabolism. 2012 , v. 107, no. 1-2

2012

Abstract: ... dehydrogenase (α-KGDH), creatine kinase (CK) and synaptic Na⁺, K⁺-ATPase. No differences of all evaluated ... effects provoked by Lys administration were marked decreases of the activities of Na⁺, K⁺-ATPase in brain ... altered in WT and Gcdh⁻/⁻ injected with Sal or Lys. Our results demonstrate that reduction of Na⁺, K⁺ ...

Abstract	Glutaric acidemia type I (GA I) is an inherited neurometabolic disorder caused by a severe deficiency of the mitochondrial glutaryl-CoA dehydrogenase activity leading to accumulation of predominantly glutaric (GA) and 3-hydroxyglutaric (3HGA) acids in the brain and other tissues. Affected patients usually present with hypotonia and brain damage and acute encephalopathic episodes whose pathophysiology is not yet fully established. In this study we investigated important parameters of cellular bioenergetics in brain, heart and skeletal muscle from 15-day-old glutaryl-CoA dehydrogenase deficient mice (Gcdh⁻/⁻) submitted to a single intra-peritoneal injection of saline (Sal) or lysine (Lys — 8μmol/g) as compared to wild type (WT) mice. We evaluated the activities of the respiratory chain complexes II, II-III and IV, α-ketoglutarate dehydrogenase (α-KGDH), creatine kinase (CK) and synaptic Na⁺, K⁺-ATPase. No differences of all evaluated parameters were detected in the Gcdh⁻/⁻ relatively to the WT mice injected at baseline (Sal). Furthermore, mild increases of the activities of some respiratory chain complexes (II-III and IV) were observed in heart and skeletal muscle of Gcdh⁻/⁻ and WT mice after Lys administration. However, the most marked effects provoked by Lys administration were marked decreases of the activities of Na⁺, K⁺-ATPase in brain and CK in brain and skeletal muscle of Gcdh⁻/⁻ mice. In contrast, brain α-KGDH activity was not altered in WT and Gcdh⁻/⁻ injected with Sal or Lys. Our results demonstrate that reduction of Na⁺, K⁺-ATPase and CK activities may play an important role in the pathogenesis of the neurodegenerative changes in GA I.
Keywords	alpha-ketoglutaric acid ; brain ; brain damage ; creatine kinase ; electron transport chain ; heart ; lysine ; mice ; pathogenesis ; pathophysiology ; patients ; skeletal muscle ; sodium
Language	English
Dates of publication	2012-09
Size	p. 81-86.
Publishing place	Elsevier Inc.
Document type	Article
ZDB-ID	1418518-0
ISSN	1096-7206 ; 1096-7192
ISSN (online)	1096-7206
ISSN	1096-7192
DOI	10.1016/j.ymgme.2012.04.015
Database	NAL-Catalogue (AGRICOLA)

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Article ; Online: Intramedullary Masses of the Spinal Cord: Radiologic-Pathologic Correlation.

Shih, Robert Y / Koeller, Kelly K

Radiographics : a review publication of the Radiological Society of North America, Inc

2020 Volume 40, Issue 4, Page(s) 1125–1145

Abstract: Spinal cord tumors are a challenge for patients and neurosurgeons because of the high risk of neurologic deficits from the disease process and surgical interventions. Spinal cord tumors are uncommon, and approximately 2%-3% of primary intra-axial tumors ... ...

Abstract	Spinal cord tumors are a challenge for patients and neurosurgeons because of the high risk of neurologic deficits from the disease process and surgical interventions. Spinal cord tumors are uncommon, and approximately 2%-3% of primary intra-axial tumors of the central nervous system occur in the spinal cord. Primary intra-axial tumors are usually derived from neuroepithelial tissue, especially glial cells. This often leads to a classic intramedullary mass differential diagnosis of ependymoma or astrocytoma, which together constitute up to 70% of spinal cord tumors. For example, ependymomas occur predominantly in adults, and astrocytomas (specifically pilocytic astrocytomas) occur predominantly in children. While that is an excellent starting point, in order to refine the differential diagnosis, the authors review the radiologic-pathologic features of specific neoplastic categories and entities recognized by the World Health Organization (WHO) in the 2016
MeSH term(s)	Contrast Media ; Diagnosis, Differential ; Humans ; Multimodal Imaging ; Spinal Cord Neoplasms/diagnostic imaging ; Spinal Cord Neoplasms/pathology
Chemical Substances	Contrast Media
Language	English
Publishing date	2020-06-12
Publishing country	United States
Document type	Case Reports ; Journal Article
ZDB-ID	603172-9
ISSN	1527-1323 ; 0271-5333
ISSN (online)	1527-1323
ISSN	0271-5333
DOI	10.1148/rg.2020190196
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Article: Depolarization of cultured astrocytes by leukotriene B4. Evidence for the induction of a K+ conductance inhibitor.

Köller, H / Siebler, M / Pekel, M / Müller, H W

Brain research

1993 Volume 612, Issue 1-2, Page(s) 28–34

Abstract: ... higher concentrations (1 microM). When the K+ conductance blocker, Ba2+ (2 mM), was added to the bath ... which, in turn, inhibits K+ conductances. This effect could impair glial, as well as neuronal, functions during ...

Abstract	Since astrocytes have been shown to participate in intracerebral immunological processes we investigated the effect of the immune mediators, leukotrienes (LT) B4, LTC4 and LTD4 on membrane properties of cultured astrocytes from neonatal rat brain. When LTB4 was added to the bath solution the membrane potential slowly decreased from -96 mV to -38 mV. While LTB4 at a concentration of 500 nM was ineffective, depolarization occurred when concentrations of 750 nM and above were used. The depolarizing effect was specific for LTB4, since LTC4 and LTD4, other arachidonic acid derivates, failed to depolarize astrocytes at even higher concentrations (1 microM). When the K+ conductance blocker, Ba2+ (2 mM), was added to the bath solution, astrocytes depolarized to the same degree but no further depolarization was achieved when LTB4 was added. Bath application of Co2+ (1 mM), in order to reduce putative Ca2+ inward currents or reduced internal chloride concentration, did not alter the LTB4-induced depolarization, thus arguing against additional Ca(2+)- or Cl(-)-dependent depolarizing effects. The LTB4-induced depolarization could be markedly reduced, however, by preincubation of the cells with cycloheximide (2 microM), which blocks translation and thereby protein synthesis. Cycloheximide alone had no effect on the membrane potential. These data indicate that, in astrocytes, LTB4 stimulates the synthesis of a protein, which, in turn, inhibits K+ conductances. This effect could impair glial, as well as neuronal, functions during CNS diseases accompanied by immunological processes.
MeSH term(s)	Animals ; Animals, Newborn ; Astrocytes/drug effects ; Astrocytes/metabolism ; Astrocytes/physiology ; Cells, Cultured ; Chlorides/pharmacology ; Cobalt/pharmacology ; Cycloheximide/pharmacology ; Electrophysiology ; Leukotriene B4/pharmacology ; Membrane Potentials/drug effects ; Nerve Tissue Proteins/biosynthesis ; Neuromuscular Depolarizing Agents/pharmacology ; Potassium Channels/drug effects ; Rats ; Rats, Wistar ; SRS-A/pharmacology
Chemical Substances	Chlorides ; Nerve Tissue Proteins ; Neuromuscular Depolarizing Agents ; Potassium Channels ; SRS-A ; Leukotriene B4 (1HGW4DR56D) ; Cobalt (3G0H8C9362) ; Cycloheximide (98600C0908)
Language	English
Publishing date	1993-05-28
Publishing country	Netherlands
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1200-2
ISSN	1872-6240 ; 0006-8993
ISSN (online)	1872-6240
ISSN	0006-8993
DOI	10.1016/0006-8993(93)91640-e
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Article ; Online: Estudo de algumas reações oxoacidobásicas no solvente NaCl fundido entre 1100K e 1200K Study of some oxoacidbasic reactions in molten NaCl in the temperature range of 1100K to 1200 K

Sérgio Luiz Koeller / Richard Louis Combes

Química Nova, Vol 25, Iss 2, Pp 226-

2002 Volume 230

Abstract: Equilibrium constants (K) of some oxoacidbasic reactions in molten NaCl in the temperature range ...

Abstract	Equilibrium constants (K) of some oxoacidbasic reactions in molten NaCl in the temperature range of 1100K to 1200K, have been measured potentiometrically with a calcia stabilized zirconia oxide ion (O2-) indicator electrode. In molten NaCl at 1100K the pKs values (in molality scale) for HCl/H2O and HO-/H2O are respectively 11.0 ± 0.3; 1.6 ± 0.3 and the pKs for CaO is 4.3 ± 0.3. The results have been compared with those determined previously by Combes for the molten equimolar NaCl-KCl mixture and are in good agreement with literature data and gives some qualitative explanation of the comparison of oxoacidbasic properties between molten NaCl and NaCl-KCl.
Keywords	oxoacidic ; molten salts ; solubility ; Chemistry ; QD1-999 ; Science ; Q ; DOAJ:Chemistry (General) ; DOAJ:Chemistry
Subject code	540
Language	English
Publishing date	2002-05-01T00:00:00Z
Publisher	Sociedade Brasileira de Química
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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