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  1. Article ; Online: snoRNAs in hematopoiesis and blood malignancies: A comprehensive review.

    Challakkara, Mohamed Fahad / Chhabra, Ravindresh

    Journal of cellular physiology

    2023  Volume 238, Issue 6, Page(s) 1207–1225

    Abstract: Small nucleolar RNAs (snoRNAs) are noncoding RNA molecules of highly variable size, usually ranging from 60 to 150 nucleotides. They are classified into H/ACA box snoRNAs, C/D box snoRNAs, and scaRNAs. Their functional profile includes biogenesis of ... ...

    Abstract Small nucleolar RNAs (snoRNAs) are noncoding RNA molecules of highly variable size, usually ranging from 60 to 150 nucleotides. They are classified into H/ACA box snoRNAs, C/D box snoRNAs, and scaRNAs. Their functional profile includes biogenesis of ribosomes, processing of rRNAs, 2'-O-methylation and pseudouridylation of RNAs, alternative splicing and processing of mRNAs and the generation of small RNA molecules like miRNA. The snoRNAs have been observed to have an important role in hematopoiesis and malignant hematopoietic conditions including leukemia, lymphoma, and multiple myeloma. Blood malignancies arise in immune system cells or the bone marrow due to chromosome abnormalities. It has been estimated that annually over 1.25 million cases of blood cancer occur worldwide. The snoRNAs often show a differential expression profile in blood malignancies. Recent reports associate the abnormal expression of snoRNAs with the inhibition of apoptosis, uncontrolled cell proliferation, angiogenesis, and metastasis. This implies that targeting snoRNAs could be a potential way to treat hematologic malignancies. In this review, we describe the various functions of snoRNAs, their role in hematopoiesis, and the consequences of their dysregulation in blood malignancies. We also evaluate the potential of the dysregulated snoRNAs as biomarkers and therapeutic targets for blood malignancies.
    MeSH term(s) Humans ; RNA, Small Nucleolar/genetics ; RNA, Small Nucleolar/metabolism ; RNA, Ribosomal ; Ribosomes/genetics ; Hematologic Neoplasms/genetics ; Hematologic Neoplasms/metabolism ; Hematopoiesis/genetics
    Chemical Substances RNA, Small Nucleolar ; RNA, Ribosomal
    Language English
    Publishing date 2023-05-14
    Publishing country United States
    Document type Journal Article ; Review ; Research Support, Non-U.S. Gov't
    ZDB-ID 3116-1
    ISSN 1097-4652 ; 0021-9541
    ISSN (online) 1097-4652
    ISSN 0021-9541
    DOI 10.1002/jcp.31032
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  2. Article ; Online: MSMEG_5850, a stress-induced TetR protein, involved in global transcription regulation in

    Singh, Parul / Kumar, Arbind / Chhabra, Ravindresh / Singh, Kashmir / Kaur, Jagdeep

    Future microbiology

    2023  Volume 18, Page(s) 563–580

    Abstract: Aim: ...

    Abstract Aim:
    MeSH term(s) Mycobacterium smegmatis/metabolism ; Bacterial Proteins/metabolism ; Gene Expression Profiling ; Gene Expression Regulation, Bacterial
    Chemical Substances Bacterial Proteins
    Language English
    Publishing date 2023-06-07
    Publishing country England
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 2254620-0
    ISSN 1746-0921 ; 1746-0913
    ISSN (online) 1746-0921
    ISSN 1746-0913
    DOI 10.2217/fmb-2022-0238
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  3. Article ; Online: Emerging role of mRNA methylation in regulating the hallmarks of cancer.

    Sarraf, Gargi / Chhabra, Ravindresh

    Biochimie

    2022  Volume 206, Page(s) 61–72

    Abstract: The dynamic chemical modifications of DNA, RNA, and proteins can transform normal cells into malignant ones. While the DNA and protein modifications in cancer have been described extensively in the literature, there are fewer reports about the role of ... ...

    Abstract The dynamic chemical modifications of DNA, RNA, and proteins can transform normal cells into malignant ones. While the DNA and protein modifications in cancer have been described extensively in the literature, there are fewer reports about the role of RNA modifications in cancer. There are over 100 forms of RNA modifications and one of these, mRNA methylation, plays a critical role in the malignant properties of the cells. mRNA methylation is a reversible modification responsible for regulating protein expression at the post-transcriptional level. Despite being discovered in the 1970s, a complete understanding of the different proteins involved and the mechanism behind mRNA methylation remains largely unknown. However, these mRNA methylations have been shown to foster cancer hallmarks via specific cellular targets inside the cell. In this review, we provide a brief overview of mRNA methylation and its emerging role in regulating the various hallmarks of cancer.
    MeSH term(s) Humans ; Methylation ; RNA/metabolism ; DNA/metabolism ; RNA Processing, Post-Transcriptional ; Neoplasms/genetics ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; Methyltransferases/genetics ; Methyltransferases/metabolism
    Chemical Substances RNA (63231-63-0) ; DNA (9007-49-2) ; RNA, Messenger ; Methyltransferases (EC 2.1.1.-)
    Language English
    Publishing date 2022-10-13
    Publishing country France
    Document type Review ; Journal Article
    ZDB-ID 120345-9
    ISSN 1638-6183 ; 0300-9084
    ISSN (online) 1638-6183
    ISSN 0300-9084
    DOI 10.1016/j.biochi.2022.10.005
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  4. Article ; Online: let-7i-5p, miR-181a-2-3p and EGF/PI3K/SOX2 axis coordinate to maintain cancer stem cell population in cervical cancer

    Ravindresh Chhabra

    Scientific Reports, Vol 8, Iss 1, Pp 1-

    2018  Volume 9

    Abstract: Abstract The characteristics of cancer stem cells (CSCs) and the genes responsible for their maintenance are highly variable in different cancers. Here, we identify the coordination among miRNAs and EGF pathway genes which is critical for the maintenance ...

    Abstract Abstract The characteristics of cancer stem cells (CSCs) and the genes responsible for their maintenance are highly variable in different cancers. Here, we identify the coordination among miRNAs and EGF pathway genes which is critical for the maintenance of CSCs in cervical cancer. The transcript analysis of CSCs enriched from cervical cancer cell lines (CaSki and HeLa) revealed a significant upregulation of SOX2. Since EGF receptor is frequently over expressed in cervical cancer, we hypothesized that EGF pathway may be responsible for the upregulation of SOX2. Also, the media used for CSC enrichment was supplemented with EGF. The hypothesis was validated as inhibiting the EGF/PI3K pathway suppressed the expression of SOX2 and reduced the CSC population. In addition, miRNA profiling identified miR-181a-2-3p and let-7i-5p as markedly reduced in CSCs. The exogenous expression of either of these miRNAs in CaSki cells inhibited the expression of SOX2 and subsequently reduced CSC population. In conclusion, this study highlights for the first time the contrasting role of let-7i-5p/ miR-181a-2-3p and EGF/PI3K/SOX2 axis in maintaining cervical CSCs. While the EGF pathway promotes CSC formation in cervical cancer by inducing SOX2, miR-181a-2-3p/let-7i-5p counteracts the EGF pathway by inhibiting SOX2, thereby reducing the CSC population.
    Keywords Medicine ; R ; Science ; Q
    Subject code 616
    Language English
    Publishing date 2018-05-01T00:00:00Z
    Publisher Nature Publishing Group
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  5. Article ; Online: Modulation of gene expression by YTH domain family (YTHDF) proteins in human physiology and pathology.

    Kisan, Aju / Chhabra, Ravindresh

    Journal of cellular physiology

    2022  Volume 238, Issue 1, Page(s) 5–31

    Abstract: The advent of high throughput techniques in the past decade has significantly advanced the field of epitranscriptomics. The internal chemical modification of the target RNA at a specific site is a basic feature of epitranscriptomics and is critical for ... ...

    Abstract The advent of high throughput techniques in the past decade has significantly advanced the field of epitranscriptomics. The internal chemical modification of the target RNA at a specific site is a basic feature of epitranscriptomics and is critical for its structural stability and functional property. More than 170 modifications at the transcriptomic level have been reported so far, among which m6A methylation is one of the more conserved internal RNA modifications, abundantly found in eukaryotic mRNAs and frequently involved in enhancing the target messenger RNA's (mRNA) stability and translation. m6A modification of mRNAs is essential for multiple physiological processes including stem cell differentiation, nervous system development and gametogenesis. Any aberration in the m6A modification can often result in a pathological condition. The deregulation of m6A methylation has already been described in inflammation, viral infection, cardiovascular diseases and cancer. The m6A modification is reversible in nature and is carried out by specialized m6A proteins including writers (m6A methyltransferases) that add methyl groups and erasers (m6A demethylases) that remove methyl groups selectively. The fate of m6A-modified mRNA is heavily reliant on the various m6A-binding proteins ("readers") which recognize and generate a functional signal from m6A-modified mRNA. In this review, we discuss the role of a family of reader proteins, "YT521-B homology domain containing family" (YTHDF) proteins, in human physiology and pathology. In addition, we critically evaluate the potential of YTHDF proteins as therapeutic targets in human diseases.
    MeSH term(s) Humans ; Gene Expression ; Methylation ; RNA, Messenger/genetics ; RNA, Messenger/metabolism ; RNA-Binding Proteins/genetics ; RNA-Binding Proteins/metabolism
    Chemical Substances RNA, Messenger ; N-methyladenosine (CLE6G00625) ; RNA-Binding Proteins
    Language English
    Publishing date 2022-11-03
    Publishing country United States
    Document type Journal Article ; Review ; Research Support, Non-U.S. Gov't
    ZDB-ID 3116-1
    ISSN 1097-4652 ; 0021-9541
    ISSN (online) 1097-4652
    ISSN 0021-9541
    DOI 10.1002/jcp.30907
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    In stock of ZB MED Cologne/Königswinter

    Uc I Zs.212: Show issues Location:
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  6. Article ; Online: let-7i-5p, miR-181a-2-3p and EGF/PI3K/SOX2 axis coordinate to maintain cancer stem cell population in cervical cancer.

    Chhabra, Ravindresh

    Scientific reports

    2018  Volume 8, Issue 1, Page(s) 7840

    Abstract: The characteristics of cancer stem cells (CSCs) and the genes responsible for their maintenance are highly variable in different cancers. Here, we identify the coordination among miRNAs and EGF pathway genes which is critical for the maintenance of CSCs ... ...

    Abstract The characteristics of cancer stem cells (CSCs) and the genes responsible for their maintenance are highly variable in different cancers. Here, we identify the coordination among miRNAs and EGF pathway genes which is critical for the maintenance of CSCs in cervical cancer. The transcript analysis of CSCs enriched from cervical cancer cell lines (CaSki and HeLa) revealed a significant upregulation of SOX2. Since EGF receptor is frequently over expressed in cervical cancer, we hypothesized that EGF pathway may be responsible for the upregulation of SOX2. Also, the media used for CSC enrichment was supplemented with EGF. The hypothesis was validated as inhibiting the EGF/PI3K pathway suppressed the expression of SOX2 and reduced the CSC population. In addition, miRNA profiling identified miR-181a-2-3p and let-7i-5p as markedly reduced in CSCs. The exogenous expression of either of these miRNAs in CaSki cells inhibited the expression of SOX2 and subsequently reduced CSC population. In conclusion, this study highlights for the first time the contrasting role of let-7i-5p/ miR-181a-2-3p and EGF/PI3K/SOX2 axis in maintaining cervical CSCs. While the EGF pathway promotes CSC formation in cervical cancer by inducing SOX2, miR-181a-2-3p/let-7i-5p counteracts the EGF pathway by inhibiting SOX2, thereby reducing the CSC population.
    MeSH term(s) Cell Line, Tumor ; Chromones/pharmacology ; Epidermal Growth Factor/metabolism ; Erlotinib Hydrochloride/pharmacology ; Female ; Gene Expression Regulation, Neoplastic/drug effects ; HeLa Cells ; Humans ; MicroRNAs/genetics ; Morpholines/pharmacology ; Neoplastic Stem Cells/chemistry ; Neoplastic Stem Cells/drug effects ; Phosphatidylinositol 3-Kinases/metabolism ; SOXB1 Transcription Factors/genetics ; Signal Transduction/drug effects ; Up-Regulation ; Uterine Cervical Neoplasms/genetics ; Uterine Cervical Neoplasms/metabolism
    Chemical Substances Chromones ; MIrn181 microRNA, human ; MicroRNAs ; Morpholines ; SOX2 protein, human ; SOXB1 Transcription Factors ; mirnlet7 microRNA, human ; 2-(4-morpholinyl)-8-phenyl-4H-1-benzopyran-4-one (31M2U1DVID) ; Epidermal Growth Factor (62229-50-9) ; Erlotinib Hydrochloride (DA87705X9K) ; Phosphatidylinositol 3-Kinases (EC 2.7.1.-)
    Language English
    Publishing date 2018-05-18
    Publishing country England
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 2615211-3
    ISSN 2045-2322 ; 2045-2322
    ISSN (online) 2045-2322
    ISSN 2045-2322
    DOI 10.1038/s41598-018-26292-w
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  7. Article ; Online: Analysis of the Inhibitory Effect of hsa-miR-145-5p and hsa-miR-203a-5p on Imatinib-Resistant K562 Cells by GC/MS Metabolomics Method.

    Singh, Priyanka / Yadav, Radheshyam / Verma, Malkhey / Chhabra, Ravindresh

    Journal of the American Society for Mass Spectrometry

    2023  Volume 34, Issue 10, Page(s) 2117–2126

    Abstract: Imatinib (IM) resistance is considered to be a significant challenge in the management of chronic myeloid leukemia (CML). Previous studies have reported that hsa-miR-145-5p and hsa-miR-203a-5p can overcome IM resistance and hsa-miR-203a-5p can alter ... ...

    Abstract Imatinib (IM) resistance is considered to be a significant challenge in the management of chronic myeloid leukemia (CML). Previous studies have reported that hsa-miR-145-5p and hsa-miR-203a-5p can overcome IM resistance and hsa-miR-203a-5p can alter glutathione metabolism in IM-resistant cells. The purpose of this study was to examine whether hsa-miR-145-5p or hsa-miR-203a-5p counters IM resistance by targeting the overall metabolic profile of IM-resistant K562 cells. The metablic profiling of cell lysates obtained from IM-sensitive, IM-resistant, and miR-transfected IM-resistant K562 cells was carried out using the GC-MS technique. Overall, 75 major metabolites were detected, of which 32 were present in all samples. The pathway analysis of MetaboAnalyst 5.0 revealed that the majorly enriched pathways included glucose metabolism, fatty acid biosynthesis, lipogenesis, and nucleotide metabolism. Eleven of identified metabolites, l-glutamine, l-glutamic acid, l-lactic acid, phosphoric acid, 9,12-octadecadienoic acid, 9-octadecenoic acid, myristic acid, palmitic acid, cholesterol, and β-alanine, appeared in enriched pathways. IM-resistant cells had comparatively higher concentrations of all of these metabolites. Notably, the introduction of hsa-miR-145-5p or hsa-miR-203a-5p into resistant cells resulted in a decrease in levels of these metabolites. The efficacy of miR-203a-5p was particularly remarkable in comparison with miR-145-5p, as evidenced by partial least-squares-discriminant analysis (PLS-DA), which showed a high level of similarity in metabolic profile between IM-sensitive K562 cells and IM-resistant cells transfected with hsa-miR-203a-5p. The results indicate that GC-MS-based metabolic profiling has the potential to distinguish between drug-resistant and -sensitive cells. This approach can also be used to routinely monitor therapeutic response in drug-resistant patients, thus, enabling personalized therapy.
    MeSH term(s) Humans ; Imatinib Mesylate/pharmacology ; Imatinib Mesylate/therapeutic use ; K562 Cells ; Gas Chromatography-Mass Spectrometry ; MicroRNAs/metabolism ; MicroRNAs/therapeutic use ; Leukemia, Myelogenous, Chronic, BCR-ABL Positive/drug therapy ; Leukemia, Myelogenous, Chronic, BCR-ABL Positive/genetics
    Chemical Substances Imatinib Mesylate (8A1O1M485B) ; MicroRNAs ; MIRN145 microRNA, human
    Language English
    Publishing date 2023-09-14
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1073671-2
    ISSN 1879-1123 ; 1044-0305
    ISSN (online) 1879-1123
    ISSN 1044-0305
    DOI 10.1021/jasms.3c00103
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  8. Article ; Online: Correction to: Overview of genetic and epigenetic regulation of human papillomavirus and apoptosis in cervical cancer.

    Yadav, Chetna / Yadav, Ritu / Chhabra, Ravindresh / Nanda, Smiti / Ranga, Shalu / Kadian, Lokesh / Ahuja, Parul

    Apoptosis : an international journal on programmed cell death

    2023  Volume 28, Issue 9-10, Page(s) 1500

    Language English
    Publishing date 2023-02-09
    Publishing country Netherlands
    Document type Published Erratum
    ZDB-ID 1452360-7
    ISSN 1573-675X ; 1360-8185
    ISSN (online) 1573-675X
    ISSN 1360-8185
    DOI 10.1007/s10495-023-01822-8
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  9. Article ; Online: Nanoemulsions (O/W) prepared from essential oil extracted from

    Sharma, Arun Dev / Chhabra, Ravindresh / Jain, Puneet / Kaur, Inderjeet / Amrita / Bhawna

    Journal of biomaterials science. Polymer edition

    2023  Volume 34, Issue 17, Page(s) 2438–2461

    Abstract: Essential oil ... ...

    Abstract Essential oil from
    MeSH term(s) Humans ; Oils, Volatile/pharmacology ; Oils, Volatile/chemistry ; Antioxidants/pharmacology ; Melaleuca/chemistry ; HeLa Cells ; Spectroscopy, Fourier Transform Infrared ; Tea Tree Oil/pharmacology ; Tea Tree Oil/chemistry ; Anti-Inflammatory Agents/pharmacology ; Tea
    Chemical Substances Oils, Volatile ; Antioxidants ; Tea Tree Oil (68647-73-4) ; Anti-Inflammatory Agents ; Tea
    Language English
    Publishing date 2023-09-04
    Publishing country England
    Document type Journal Article
    ZDB-ID 1014190-x
    ISSN 1568-5624 ; 1568-5616 ; 0920-5063
    ISSN (online) 1568-5624 ; 1568-5616
    ISSN 0920-5063
    DOI 10.1080/09205063.2023.2253584
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  10. Article ; Online: miRNA and methylation: a multifaceted liaison.

    Chhabra, Ravindresh

    Chembiochem : a European journal of chemical biology

    2015  Volume 16, Issue 2, Page(s) 195–203

    Abstract: miRNAs and DNA methylation are both critical regulators of gene expression. Aberration in miRNA expression or DNA methylation is a causal factor for numerous pathological conditions. DNA methylation can inhibit the transcription of miRNAs, just like ... ...

    Abstract miRNAs and DNA methylation are both critical regulators of gene expression. Aberration in miRNA expression or DNA methylation is a causal factor for numerous pathological conditions. DNA methylation can inhibit the transcription of miRNAs, just like coding genes, by methylating the CpG islands in the promoter regions of miRNAs. Conversely, certain miRNAs can directly target DNA methyltransferases and bring about their inhibition, thereby affecting the whole genome methylation pattern. Recently, methylation patterns have also been revealed in mRNA. Surprisingly, the two most commonly studied methylation states in mRNA (m6A and m5C) are found to be enriched in 3'-UTRs (untranslated regions), the target site for the majority of miRNAs. Whereas m5C is reported to stabilise mRNA, m6A has a destabilising effect on mRNA. However, the effect of mRNA methylation on its interaction with miRNAs is largely unexplored. The review highlights the complex interplay between microRNA and methylation at DNA and mRNA level.
    MeSH term(s) 3' Untranslated Regions ; CpG Islands ; Cytosine/metabolism ; DNA Methylation ; Epigenesis, Genetic ; Gene Expression Regulation ; Gene Expression Regulation, Neoplastic ; Humans ; MicroRNAs/genetics ; MicroRNAs/metabolism ; Promoter Regions, Genetic ; RNA/metabolism ; RNA, Messenger
    Chemical Substances 3' Untranslated Regions ; MicroRNAs ; RNA, Messenger ; RNA (63231-63-0) ; Cytosine (8J337D1HZY)
    Language English
    Publishing date 2015-01-19
    Publishing country Germany
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 2020469-3
    ISSN 1439-7633 ; 1439-4227
    ISSN (online) 1439-7633
    ISSN 1439-4227
    DOI 10.1002/cbic.201402449
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