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Article ; Online: Niacin for reduction of cardiovascular risk.

van den Oever, Inge A M / Nurmohamed, Michael T / Lems, Willem F

2014 Volume 371, Issue 20, Page(s) 1942

MeSH term(s)	Atherosclerosis/drug therapy ; Female ; Humans ; Hypercholesterolemia/drug therapy ; Hypolipidemic Agents/administration & dosage ; Indoles/administration & dosage ; Infection/chemically induced ; Male ; Niacin/administration & dosage ; Niacin/adverse effects
Chemical Substances	Hypolipidemic Agents ; Indoles ; Niacin (2679MF687A)
Language	English
Publishing date	2014-11-13
Publishing country	United States
Document type	Comment ; Letter
ZDB-ID	207154-x
ISSN	1533-4406 ; 0028-4793
ISSN (online)	1533-4406
ISSN	0028-4793
DOI	10.1056/NEJMc1411240#SA4
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Article: Management of cardiovascular risk in patients with rheumatoid arthritis: evidence and expert opinion.

van den Oever, Inge A M / van Sijl, Alper M / Nurmohamed, Michael T

Therapeutic advances in musculoskeletal disease

2013 Volume 5, Issue 4, Page(s) 166–181

Abstract: The risk of cardiovascular morbidity and mortality is increased in rheumatoid arthritis. The classical cardiovascular risk factors, including smoking, hypertension, dyslipidaemia, insulin resistance and diabetes mellitus, obesity and physical inactivity ... ...

Abstract	The risk of cardiovascular morbidity and mortality is increased in rheumatoid arthritis. The classical cardiovascular risk factors, including smoking, hypertension, dyslipidaemia, insulin resistance and diabetes mellitus, obesity and physical inactivity do not appear to explain the excess cardiovascular risk in rheumatoid arthritis, although they do contribute, albeit in a different way or to a lesser extent, to rheumatoid arthritis in comparison with the general population. A very important link between rheumatoid arthritis and cardiovascular disease is inflammation as it plays a key role in all stages of atherosclerosis: from endothelial dysfunction to plaque rupture and thrombosis. It also has an influence on and accentuates some traditional cardiovascular risk factors, such as dyslipidaemia, obesity and insulin resistance. To date, the exact pathophysiologic mechanism by which this relation between cardiovascular disease and rheumatoid arthritis can be explained is not completely clear. Cardiovascular risk management in rheumatoid arthritis is mandatory. Unfortunately, the way this should be done remains a point of discussion. In this review issues regarding cardiovascular risk in rheumatoid arthritis and its management will be addressed, according to evidence presented in the latest studies and our own experience-based opinion.
Language	English
Publishing date	2013-07-12
Publishing country	England
Document type	Journal Article
ZDB-ID	2516075-8
ISSN	1759-7218 ; 1759-720X
ISSN (online)	1759-7218
ISSN	1759-720X
DOI	10.1177/1759720X13491025
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Article ; Online: Comparing inflammatory cell density in the myocardium and coronary arteries in rheumatoid arthritis patients versus controls with myocardial infarction: A post-mortem case-control study.

van den Oever, Inge A M / van Sijl, Alper M / Baylan, Umit / Ter Wee, Marieke M / Schalkwijk, Casper G / Krijnen, Paul A J / Nurmohamed, Michael T / Voskuyl, Alexandre E / Niessen, Hans W M / Simsek, Suat

International journal of cardiology

2016 Volume 209, Page(s) 74–76

MeSH term(s)	Aged ; Aged, 80 and over ; Arthritis, Rheumatoid/immunology ; Arthritis, Rheumatoid/pathology ; Autopsy ; Case-Control Studies ; Coronary Vessels/immunology ; Coronary Vessels/pathology ; Cytokines/immunology ; Female ; Humans ; Male ; Myocardial Infarction/immunology ; Myocardial Infarction/pathology ; Myocardium/immunology ; Myocardium/pathology
Chemical Substances	Cytokines
Language	English
Publishing date	2016-04-15
Publishing country	Netherlands
Document type	Comparative Study ; Letter ; Research Support, Non-U.S. Gov't
ZDB-ID	779519-1
ISSN	1874-1754 ; 0167-5273
ISSN (online)	1874-1754
ISSN	0167-5273
DOI	10.1016/j.ijcard.2016.02.065
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Zs.A 1673: Show issues

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Article ; Online: Endothelial dysfunction, inflammation, and apoptosis in diabetes mellitus.

van den Oever, Inge A M / Raterman, Hennie G / Nurmohamed, Mike T / Simsek, Suat

Mediators of inflammation

2010 Volume 2010, Page(s) 792393

Abstract: Endothelial dysfunction is regarded as an important factor in the pathogenesis of vascular disease in obesity-related type 2 diabetes. The imbalance in repair and injury (hyperglycemia, hypertension, dyslipidemia) results in microvascular changes, ... ...

Abstract	Endothelial dysfunction is regarded as an important factor in the pathogenesis of vascular disease in obesity-related type 2 diabetes. The imbalance in repair and injury (hyperglycemia, hypertension, dyslipidemia) results in microvascular changes, including apoptosis of microvascular cells, ultimately leading to diabetes related complications. This review summarizes the mechanisms by which the interplay between endothelial dysfunction, inflammation, and apoptosis may cause (micro)vascular damage in patients with diabetes mellitus.
MeSH term(s)	Apoptosis/physiology ; Diabetes Complications/pathology ; Diabetes Complications/physiopathology ; Diabetes Mellitus/pathology ; Diabetes Mellitus/physiopathology ; Endothelium, Vascular/pathology ; Endothelium, Vascular/physiopathology ; Humans ; Inflammation/pathology ; Inflammation/physiopathology ; Signal Transduction/immunology
Language	English
Publishing date	2010-06-15
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	1137605-3
ISSN	1466-1861 ; 0962-9351
ISSN (online)	1466-1861
ISSN	0962-9351
DOI	10.1155/2010/792393
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Article ; Online: Cardiovascular risk management in rheumatoid arthritis patients still suboptimal: the Implementation of Cardiovascular Risk Management in Rheumatoid Arthritis project.

van den Oever, Inge A M / Heslinga, Maaike / Griep, Ed N / Griep-Wentink, Hanneke R M / Schotsman, Rob / Cambach, Walter / Dijkmans, Ben A C / Smulders, Yvo M / Lems, Willem F / Boers, Maarten / Voskuyl, Alexandre E / Peters, Mike J L / van Schaardenburg, Dirkjan / Nurmohamed, Micheal T

Rheumatology (Oxford, England)

2017 Volume 56, Issue 9, Page(s) 1472–1478

Abstract: Objective: To assess the 10-year cardiovascular (CV) risk score and to identify treatment and undertreatment of CV risk factors in patients with established RA.: Methods: Demographics, CV risk factors and prevalence of cardiovascular disease (CVD) ... ...

Abstract	Objective: To assess the 10-year cardiovascular (CV) risk score and to identify treatment and undertreatment of CV risk factors in patients with established RA. Methods: Demographics, CV risk factors and prevalence of cardiovascular disease (CVD) were assessed by questionnaire. To calculate the 10-year CV risk score according to the Dutch CV risk management guideline, systolic blood pressure was measured and cholesterol levels were determined from fasting blood samples. Patients were categorized into four groups: indication for treatment but not treated; inadequately treated, so not meeting goals (systolic blood pressure ⩽140 mmHg and/or low-density lipoprotein ⩽2.5 mmol/l); adequately treated; or no treatment necessary. Results: A total of 720 consecutive RA patients were included, 375 from Reade and 345 from the Antonius Hospital. The mean age of patients was 59 years (s.d. 12) and 73% were female. Seventeen per cent of the patients had a low 10-year CV risk (<10%), 21% had an intermediate risk (10-19%), 53% a high risk (⩾20%) and 9% had CVD. In total, 69% had an indication for preventive treatment (cholesterol-lowering or antihypertensive drugs). Of those, 42% received inadequate treatment and 40% received no treatment at all. Conclusion: Optimal CV risk management remains a major challenge and better awareness and management are urgently needed to reduce the high risk of CVD in the RA population.
MeSH term(s)	Aged ; Antihypertensive Agents/therapeutic use ; Arthritis, Rheumatoid/complications ; Arthritis, Rheumatoid/epidemiology ; Arthritis, Rheumatoid/physiopathology ; Blood Pressure/physiology ; Cardiovascular Diseases/epidemiology ; Cardiovascular Diseases/etiology ; Cardiovascular Diseases/physiopathology ; Cardiovascular Diseases/prevention & control ; Cholesterol/blood ; Cross-Sectional Studies ; Drug Therapy, Combination ; Female ; Humans ; Hydroxymethylglutaryl-CoA Reductase Inhibitors/therapeutic use ; Male ; Middle Aged ; Netherlands/epidemiology ; Prevalence ; Risk Assessment/methods ; Risk Factors ; Risk Management/methods ; Risk Management/standards
Chemical Substances	Antihypertensive Agents ; Hydroxymethylglutaryl-CoA Reductase Inhibitors ; Cholesterol (97C5T2UQ7J)
Language	English
Publishing date	2017-09-01
Publishing country	England
Document type	Journal Article
ZDB-ID	1464822-2
ISSN	1462-0332 ; 1462-0324
ISSN (online)	1462-0332
ISSN	1462-0324
DOI	10.1093/rheumatology/kew497
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Zs.B 240: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 2021: Bestellungen von Artikeln über das Online-Bestellformular ab Jg. 2022: Lesesaal (EG)
Zs.MO 497: Show issues

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Article ; Online: Endothelial Dysfunction, Inflammation, and Apoptosis in Diabetes Mellitus

Suat Simsek / Inge A. M. van den Oever / Hennie G. Raterman / Mike T. Nurmohamed

Mediators of Inflammation, Vol

2010 Volume 2010

Keywords	Pathology ; RB1-214 ; Medicine ; R ; DOAJ:Pathology ; DOAJ:Medicine (General) ; DOAJ:Health Sciences
Language	English
Publishing date	2010-01-01T00:00:00Z
Publisher	Hindawi Publishing Corporation
Document type	Article ; Online
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Article ; Online: Cardiovascular risk management in patients with active ankylosing spondylitis: a detailed evaluation.

Heslinga, Sjoerd C / Van den Oever, Inge A / Van Sijl, Alper M / Peters, Mike J / Van der Horst-Bruinsma, Irene E / Smulders, Yvo M / Nurmohamed, Michael T

BMC musculoskeletal disorders

2015 Volume 16, Page(s) 80

Abstract: Background: Ankylosing spondylitis (AS) is an inflammatory disease with documented elevated cardiovascular (CV) risk due to systemic inflammation and a higher prevalence of CV risk factors. CV risk management (CV-RM) could be an effective method to ... ...

Abstract	Background: Ankylosing spondylitis (AS) is an inflammatory disease with documented elevated cardiovascular (CV) risk due to systemic inflammation and a higher prevalence of CV risk factors. CV risk management (CV-RM) could be an effective method to reduce CV mortality and morbidity in AS patients. We assessed CV risk and evaluated guideline adherence according to the Dutch CV-RM guideline. Methods: This study was conducted with a cohort of consecutive AS patients eligible for treatment with a tumor necrosis factor (TNF) -α inhibitor. Data from the Dutch National Institute for Public Health and Environment was used to compare the prevalence of CV risk factors in AS patients with the Dutch background population. Results: In total, 254 consecutive AS patients were included. The prevalences of hypertension (41% vs 31%) and smoking (43% vs 27%) were substantially higher in AS patients as compared to the general Dutch background population. Of 138 AS patients older than 40 years the 10-years CV risk could be calculated. Fifty-one of these 138 patients (37%) had an indication for CV risk treatment. CV risk treatment was initiated in 42 of the 51 (82%), however, in only 12 of the 51 (24%) patients treatment targets for either hypertension or hypercholesterolemia were reached. Conclusion: The increased rates of hypertension and smoking illustrate the importance of CV-RM in AS patients. Although the majority of all AS patients eligible for CV-RM received CV risk medication, CV-RM remains a challenge for treating physicians, as treatment targets were not achieved in three-quarter of the eligible patients.
MeSH term(s)	Adult ; Cardiovascular Diseases/epidemiology ; Cohort Studies ; Cross-Sectional Studies ; Female ; Humans ; Hypertension/complications ; Longitudinal Studies ; Male ; Middle Aged ; Netherlands/epidemiology ; Patient Compliance ; Prevalence ; Risk Factors ; Risk Management/trends ; Smoking/adverse effects ; Spondylitis, Ankylosing/complications
Language	English
Publishing date	2015-04-09
Publishing country	England
Document type	Comparative Study ; Journal Article
ISSN	1471-2474
ISSN (online)	1471-2474
DOI	10.1186/s12891-015-0532-3
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Article ; Online: Complement Activation in the Disease Course of Coronavirus Disease 2019 and Its Effects on Clinical Outcomes

de Nooijer, Aline H / Grondman, Inge / Janssen, Nico A F / Netea, Mihai G / Willems, Loek / van de Veerdonk, Frank L / Giamarellos-Bourboulis, Evangelos J / Toonen, Erik J M / Joosten, Leo A B / Jaeger, Martin / Dijkstra, Helga / Lemmers, Heidi / van Emst, Liesbeth / Schraa, Kiki / Jacobs, Cor / Hijmans, Anneke / Jansen, Trees / Weren, Fieke / Fransman, Liz /

Gerretsen, Jelle / van de Maat, Josephine / Nijman, Gerine / Moorlag, Simone / Taks, Esther / Debisarun, Priya / Kouijzer, Ilse / Wertheim, Heiman / Hopman, Joost / Rahamat-Langendoen, Janette / Bleeker-Rovers, Chantal / ten Oever, Jaap / van Crevel, Reinout / Hoogerwerf, Jacobien / de Mast, Quirijn / van der Hoeven, Hans / Pickkers, Peter / Kox, Matthijs / Frenzel, Tim / Schouten, Jeroen / Hemelaar, Pleun / Beunders, Remi / van der Velde, Sjef / Kooistra, Emma / Waalders, Nicole / Claassen, Wout / Heesakkers, Hidde / van Schaik, Tirsa / van der Eng, Hetty / Rovers, Noortje / Klop-Riehl, Margreet

The Journal of Infectious Diseases ; ISSN 0022-1899 1537-6613

2020

Abstract: Abstract Background Excessive activation of immune responses in coronavirus disease 2019 (COVID-19) is considered to be related to disease severity, complications, and mortality rate. The complement system is an important component of innate immunity and ...

Abstract	Abstract Background Excessive activation of immune responses in coronavirus disease 2019 (COVID-19) is considered to be related to disease severity, complications, and mortality rate. The complement system is an important component of innate immunity and can stimulate inflammation, but its role in COVID-19 is unknown. Methods A prospective, longitudinal, single center study was performed in hospitalized patients with COVID-19. Plasma concentrations of complement factors C3a, C3c, and terminal complement complex (TCC) were assessed at baseline and during hospital admission. In parallel, routine laboratory and clinical parameters were collected from medical files and analyzed. Results Complement factors C3a, C3c, and TCC were significantly increased in plasma of patients with COVID-19 compared with healthy controls (P < .05). These complement factors were especially elevated in intensive care unit patients during the entire disease course (P < .005 for C3a and TCC). More intense complement activation was observed in patients who died and in those with thromboembolic events. Conclusions Patients with COVID-19 demonstrate activation of the complement system, which is related to disease severity. This pathway may be involved in the dysregulated proinflammatory response associated with increased mortality rate and thromboembolic complications. Components of the complement system might have potential as prognostic markers for disease severity and as therapeutic targets in COVID-19.
Keywords	Immunology and Allergy ; Infectious Diseases ; covid19
Language	English
Publisher	Oxford University Press (OUP)
Publishing country	uk
Document type	Article ; Online
DOI	10.1093/infdis/jiaa646
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Article ; Online: Noninvasive prenatal diagnosis of Huntington disease: detection of the paternally inherited expanded CAG repeat in maternal plasma.

van den Oever, Jessica M E / Bijlsma, Emilia K / Feenstra, Ilse / Muntjewerff, Nienke / Mathijssen, Inge B / Bakker, Egbert / van Belzen, Martine J / Boon, Elles M J

Prenatal diagnosis

2015 Volume 35, Issue 10, Page(s) 945–949

Abstract: Objective: With a shift towards noninvasive testing, we have explored and validated the use of noninvasive prenatal diagnosis (NIPD) for Huntington disease (HD).: Methods: Fifteen couples have been included, assessing a total of n = 20 pregnancies. ... ...

Abstract	Objective: With a shift towards noninvasive testing, we have explored and validated the use of noninvasive prenatal diagnosis (NIPD) for Huntington disease (HD). Methods: Fifteen couples have been included, assessing a total of n = 20 pregnancies. Fetal paternally inherited CAG repeat length was determined in total cell-free DNA from maternal plasma using a direct approach by PCR and subsequent fragment analysis. Results: All fetal HD (n = 7) and intermediate (n = 3) CAG repeats could be detected in maternal plasma. Detection of repeats in the normal range (n = 10) was successful in n = 5 cases where the paternal repeat size could be distinguished from maternal repeat patterns after fragment analysis. In all other cases (n = 5), the paternal peaks coincided with the maternal peak pattern. All NIPD results were concordant with results from routine diagnostics on fetal genomic DNA from chorionic villi. Conclusion: In this validation study, we demonstrated that all fetuses at risk for HD could be identified noninvasively in maternal plasma. Additionally, we have confirmed results from previously described case reports that NIPD for HD can be performed using a direct approach by PCR. For future diagnostics, parental CAG profiles can be used to predict the success rate for NIPD prior to testing.
MeSH term(s)	Female ; Humans ; Huntingtin Protein ; Huntington Disease/blood ; Huntington Disease/diagnosis ; Huntington Disease/genetics ; Male ; Maternal Serum Screening Tests ; Nerve Tissue Proteins/genetics ; Pregnancy
Chemical Substances	HTT protein, human ; Huntingtin Protein ; Nerve Tissue Proteins
Language	English
Publishing date	2015-10
Publishing country	England
Document type	Journal Article ; Validation Studies
ZDB-ID	82031-3
ISSN	1097-0223 ; 0197-3851
ISSN (online)	1097-0223
ISSN	0197-3851
DOI	10.1002/pd.4593
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Zs.A 1625: Show issues

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Article ; Online: Cytokine production assays reveal discriminatory immune defects in adults with recurrent infections and noninfectious inflammation.

Ten Oever, Jaap / van de Veerdonk, Frank L / Joosten, Leo A B / Simon, Anna / van Crevel, Reinout / Kullberg, Bart-Jan / Gyssens, Inge C / van der Meer, Jos W M / van Deuren, Marcel / Netea, Mihai G

Clinical and vaccine immunology : CVI

2014 Volume 21, Issue 8, Page(s) 1061–1069

Abstract: Cytokine production assays have been primarily used in research settings studying novel immunodeficiencies. We sought to determine the diagnostic value of cytokine production assays in patients with recurrent and/or severe infectious diseases (IDs) ... ...

Abstract	Cytokine production assays have been primarily used in research settings studying novel immunodeficiencies. We sought to determine the diagnostic value of cytokine production assays in patients with recurrent and/or severe infectious diseases (IDs) without known immunodeficiencies and unclassified noninfectious inflammatory disorders (NIIDs). We retrospectively examined cytokine production in whole-blood and peripheral blood mononuclear cell samples from 157 adult patients. A cytokine production rate of <5% of that of healthy controls was considered defective. While monocyte-derived cytokine (tumor necrosis factor alpha [TNF-α], interleukin-1β [IL-1β], and IL-6) production was rarely affected, 30% of all included patients had deficient production of interferon gamma (IFN-γ), IL-17A, or IL-22. Twenty-five percent of the NIID patients displayed defective IFN-γ production, whereas IL-17A production was generally unaffected. In the group of ID patients, defective IFN-γ production was found in 19% and 14% of the patients with viral and bacterial infections, respectively, and in 38%, 24%, and 50% of patients with mycobacterial, mucocutaneous, and invasive fungal infections, respectively. Defective IL-17A and IL-22 production was mainly confined to ID patients with mucocutaneous fungal infections. In conclusion, cytokine production assays frequently detect defective Th1 responses in patients with mycobacterial or fungal infections, in contrast to patients with respiratory tract infections or isolated bacterial infections. Defective IL-17A and IL-22 production was primarily found in patients with fungal infections, while monocyte-derived cytokine production was unaffected. Thus, lymphocyte-derived cytokine production assays are helpful in the diagnostic workup of patients with recurrent infections and suspected immunodeficiencies and have the potential to reveal immune defects that might guide adjunctive immunomodulatory therapy.
MeSH term(s)	Adolescent ; Adult ; Aged ; Aged, 80 and over ; Bacterial Infections/immunology ; Cytokines/analysis ; Cytokines/biosynthesis ; Cytokines/blood ; Female ; Humans ; Immunologic Deficiency Syndromes/immunology ; Inflammation/immunology ; Interferon-gamma/biosynthesis ; Interleukin-17/biosynthesis ; Interleukin-1beta/biosynthesis ; Interleukin-6/biosynthesis ; Interleukins/biosynthesis ; Male ; Middle Aged ; Mycobacterium Infections/immunology ; Mycoses/immunology ; Retrospective Studies ; Serologic Tests ; Th1 Cells/immunology ; Th17 Cells/immunology ; Tumor Necrosis Factor-alpha/biosynthesis ; Young Adult ; Interleukin-22
Chemical Substances	Cytokines ; IL17A protein, human ; IL1B protein, human ; IL6 protein, human ; Interleukin-17 ; Interleukin-1beta ; Interleukin-6 ; Interleukins ; Tumor Necrosis Factor-alpha ; Interferon-gamma (82115-62-6)
Language	English
Publishing date	2014-05-28
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2221082-9
ISSN	1556-679X ; 1556-6811
ISSN (online)	1556-679X
ISSN	1556-6811
DOI	10.1128/CVI.00152-14
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