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  1. Article ; Online: Surgery or Endovascular Therapy for Chronic Limb Ischemia.

    Kida, Yujiro

    The New England journal of medicine

    2023  Volume 388, Issue 11, Page(s) e37

    MeSH term(s) Humans ; Chronic Limb-Threatening Ischemia ; Ischemia/surgery ; Endovascular Procedures ; Peripheral Vascular Diseases
    Language English
    Publishing date 2023-03-15
    Publishing country United States
    Document type Letter ; Comment
    ZDB-ID 207154-x
    ISSN 1533-4406 ; 0028-4793
    ISSN (online) 1533-4406
    ISSN 0028-4793
    DOI 10.1056/NEJMc2300713
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  2. Article ; Online: Baxdrostat for Treatment-Resistant Hypertension.

    Kida, Yujiro

    The New England journal of medicine

    2023  Volume 388, Issue 19, Page(s) 1820

    MeSH term(s) Humans ; Hypertension/drug therapy ; Hypertension/physiopathology ; Antihypertensive Agents/therapeutic use
    Chemical Substances Antihypertensive Agents
    Language English
    Publishing date 2023-05-08
    Publishing country United States
    Document type Letter ; Comment
    ZDB-ID 207154-x
    ISSN 1533-4406 ; 0028-4793
    ISSN (online) 1533-4406
    ISSN 0028-4793
    DOI 10.1056/NEJMc2302673
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  3. Article ; Online: Peritubular Capillary Rarefaction: An Underappreciated Regulator of CKD Progression.

    Kida, Yujiro

    International journal of molecular sciences

    2020  Volume 21, Issue 21

    Abstract: Peritubular capillary (PTC) rarefaction is commonly detected in chronic kidney disease (CKD) such as hypertensive nephrosclerosis and diabetic nephropathy. Moreover, PTC rarefaction prominently correlates with impaired kidney function and predicts the ... ...

    Abstract Peritubular capillary (PTC) rarefaction is commonly detected in chronic kidney disease (CKD) such as hypertensive nephrosclerosis and diabetic nephropathy. Moreover, PTC rarefaction prominently correlates with impaired kidney function and predicts the future development of end-stage renal disease in patients with CKD. However, it is still underappreciated that PTC rarefaction is a pivotal regulator of CKD progression, primarily because the molecular mechanisms of PTC rarefaction have not been well-elucidated. In addition to the established mechanisms (reduced proangiogenic factors and increased anti-angiogenic factors), recent studies discovered significant contribution of the following elements to PTC loss: (1) prompt susceptibility of PTC to injury, (2) impaired proliferation of PTC, (3) apoptosis/senescence of PTC, and (4) pericyte detachment from PTC. Mainly based on the recent and novel findings in basic research and clinical study, this review describes the roles of the above-mentioned elements in PTC loss and focuses on the major factors regulating PTC angiogenesis, the assessment of PTC rarefaction and its surrogate markers, and an overview of the possible therapeutic agents to mitigate PTC rarefaction during CKD progression. PTC rarefaction is not only a prominent histological characteristic of CKD but also a central driving force of CKD progression.
    MeSH term(s) Animals ; Apoptosis/physiology ; Capillaries/pathology ; Capillaries/physiology ; Cell Count ; Cellular Senescence/physiology ; Disease Progression ; Endothelial Cells/pathology ; Humans ; Kidney Failure, Chronic/etiology ; Kidney Failure, Chronic/pathology ; Kidney Tubules/blood supply ; Kidney Tubules/pathology ; Neovascularization, Physiologic/physiology ; Pericytes/pathology ; Pericytes/physiology ; Renal Insufficiency, Chronic/pathology ; Renal Insufficiency, Chronic/physiopathology
    Language English
    Publishing date 2020-11-04
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 2019364-6
    ISSN 1422-0067 ; 1422-0067 ; 1661-6596
    ISSN (online) 1422-0067
    ISSN 1422-0067 ; 1661-6596
    DOI 10.3390/ijms21218255
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  4. Article ; Online: Severe Anemia from Multiple Gastric Hyperplastic Polyps in a Hemodialysis Patient after Long-term Use of a Proton-pump Inhibitor.

    Ikeda, Shiyo / Takahashi, Toshiya / Tandoh, Toshitsugu / Ushiyama, Kaori / Kida, Yujiro

    Internal medicine (Tokyo, Japan)

    2024  Volume 63, Issue 5, Page(s) 649–657

    Abstract: A 90-year-old man on maintenance hemodialysis was admitted due to severe symptomatic anemia. Biopsies under esophagogastroduodenoscopy demonstrated that the cause of anemia was intermittent blood oozing from multiple gastric hyperplastic polyps. Even ... ...

    Abstract A 90-year-old man on maintenance hemodialysis was admitted due to severe symptomatic anemia. Biopsies under esophagogastroduodenoscopy demonstrated that the cause of anemia was intermittent blood oozing from multiple gastric hyperplastic polyps. Even after successful eradication of Helicobacter pylori, he showed hypergastrinemia (480 pg/mL) owing to esomeprazole (proton-pump inhibitor) therapy for the past 4.5 years to treat reflux esophagitis. Seven months after we switched esomeprazole to famotidine (H
    MeSH term(s) Male ; Humans ; Aged, 80 and over ; Proton Pump Inhibitors/adverse effects ; Esomeprazole/adverse effects ; Anemia/chemically induced ; Biopsy ; Hyperplasia/chemically induced ; Renal Dialysis/adverse effects
    Chemical Substances Proton Pump Inhibitors ; Esomeprazole (N3PA6559FT)
    Language English
    Publishing date 2024-03-01
    Publishing country Japan
    Document type Case Reports ; Journal Article
    ZDB-ID 32371-8
    ISSN 1349-7235 ; 0021-5120 ; 0918-2918
    ISSN (online) 1349-7235
    ISSN 0021-5120 ; 0918-2918
    DOI 10.2169/internalmedicine.2091-23
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  5. Article ; Online: Efficacy and safety of sirolimus in lymphangioleiomyomatosis.

    Kida, Yujiro

    The New England journal of medicine

    2011  Volume 365, Issue 3, Page(s) 271; author reply 272

    MeSH term(s) Forced Expiratory Volume/drug effects ; Humans ; Lymphangioleiomyomatosis/drug therapy ; Lymphangioleiomyomatosis/physiopathology ; Patient Dropouts ; Sirolimus/pharmacology ; Sirolimus/therapeutic use ; TOR Serine-Threonine Kinases/antagonists & inhibitors
    Chemical Substances TOR Serine-Threonine Kinases (EC 2.7.1.1) ; Sirolimus (W36ZG6FT64)
    Language English
    Publishing date 2011-07-21
    Publishing country United States
    Document type Comment ; Letter
    ZDB-ID 207154-x
    ISSN 1533-4406 ; 0028-4793
    ISSN (online) 1533-4406
    ISSN 0028-4793
    DOI 10.1056/NEJMc1106358#SA1
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  6. Article ; Online: VTACH trial.

    Kida, Yujiro

    Lancet (London, England)

    2010  Volume 375, Issue 9718, Page(s) 890; author reply 890–1

    MeSH term(s) Anti-Arrhythmia Agents/therapeutic use ; Catheter Ablation ; Defibrillators, Implantable ; Humans ; Secondary Prevention ; Tachycardia, Ventricular/therapy
    Chemical Substances Anti-Arrhythmia Agents
    Language English
    Publishing date 2010-03-13
    Publishing country England
    Document type Comment ; Letter
    ZDB-ID 3306-6
    ISSN 1474-547X ; 0023-7507 ; 0140-6736
    ISSN (online) 1474-547X
    ISSN 0023-7507 ; 0140-6736
    DOI 10.1016/S0140-6736(10)60380-7
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  7. Article ; Online: Polymyxin B hemoperfusion and mortality in abdominal septic shock.

    Kida, Yujiro

    JAMA

    2009  Volume 302, Issue 18, Page(s) 1969; author reply 1969–70

    MeSH term(s) Anti-Bacterial Agents/therapeutic use ; Gram-Negative Bacterial Infections/mortality ; Gram-Negative Bacterial Infections/therapy ; Gram-Positive Bacterial Infections/mortality ; Gram-Positive Bacterial Infections/therapy ; Hemoperfusion ; Humans ; Polymyxin B/therapeutic use ; Shock, Septic/mortality ; Shock, Septic/therapy
    Chemical Substances Anti-Bacterial Agents ; Polymyxin B (1404-26-8)
    Language English
    Publishing date 2009-11-11
    Publishing country United States
    Document type Comment ; Letter
    ZDB-ID 2958-0
    ISSN 1538-3598 ; 0254-9077 ; 0002-9955 ; 0098-7484
    ISSN (online) 1538-3598
    ISSN 0254-9077 ; 0002-9955 ; 0098-7484
    DOI 10.1001/jama.2009.1608
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  8. Article ; Online: Sirtuins, Cell Senescence, and Vascular Aging.

    Kida, Yujiro / Goligorsky, Michael S

    The Canadian journal of cardiology

    2016  Volume 32, Issue 5, Page(s) 634–641

    Abstract: The sirtuins (SIRTs) constitute a class of proteins with nicotinamide adenine dinucleotide-dependent deacetylase or adenosine diphosphate-ribosyltransferase activity. Seven SIRT family members have been identified in mammals, from SIRT1, the best studied ...

    Abstract The sirtuins (SIRTs) constitute a class of proteins with nicotinamide adenine dinucleotide-dependent deacetylase or adenosine diphosphate-ribosyltransferase activity. Seven SIRT family members have been identified in mammals, from SIRT1, the best studied for its role in vascular aging, to SIRT7. SIRT1 and SIRT2 are localized in the nucleus and cytoplasm. SIRT3, SIRT4, and SIRT5 are mitochondrial, and SIRT6 and SIRT7 are nuclear. Extensive studies have clearly revealed that SIRT proteins regulate diverse cell functions and responses to stressors. Vascular aging involves the aging process (senescence) of endothelial and vascular smooth muscle cells. Two types of cell senescence have been identified: (1) replicative senescence with telomere attrition; and (2) stress-induced premature senescence without telomere involvement. Both types of senescence induce vascular cell growth arrest and loss of vascular homeostasis, and contribute to the initiation and progression of cardiovascular diseases. Previous mechanistic studies have revealed in detail that SIRT1, SIRT3, and SIRT6 show protective functions against vascular aging, and definite vascular function of other SIRTs is under investigation. Thus, direct SIRT modulation and nicotinamide adenine dinucleotide stimulation of SIRT are promising candidates for cardiovascular disease therapy. A small number of pilot studies have been conducted to assess SIRT modulation in humans. These clinical studies have not yet provided convincing evidence that SIRT proteins alleviate morbidity and mortality in patients with cardiovascular diseases. The outcomes of multiple ongoing clinical trials are awaited to define the efficacy of SIRT modulators and SIRT activators in cardiovascular diseases, along with the potential adverse effects of chronic SIRT modulation.
    MeSH term(s) Aging/physiology ; Cardiovascular Diseases/metabolism ; Cardiovascular Diseases/physiopathology ; Cell Survival ; Cellular Senescence/genetics ; Cellular Senescence/physiology ; Endothelium, Vascular/metabolism ; Homeostasis ; Humans ; Myocytes, Smooth Muscle/metabolism ; Sirtuins/metabolism ; Telomere/physiology
    Chemical Substances Sirtuins (EC 3.5.1.-)
    Language English
    Publishing date 2016
    Publishing country England
    Document type Journal Article ; Review ; Research Support, N.I.H., Extramural
    ZDB-ID 632813-1
    ISSN 1916-7075 ; 0828-282X
    ISSN (online) 1916-7075
    ISSN 0828-282X
    DOI 10.1016/j.cjca.2015.11.022
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  9. Article ; Online: Critically ill patients and acute renal failure.

    Kida, Yujiro

    JAMA

    2006  Volume 295, Issue 6, Page(s) 624; author reply 624–5

    MeSH term(s) Acute Kidney Injury/etiology ; Acute Kidney Injury/mortality ; Comorbidity ; Critical Illness ; Diabetes Complications ; Humans ; Renal Replacement Therapy ; Risk Factors
    Language English
    Publishing date 2006-02-08
    Publishing country United States
    Document type Comment ; Letter
    ZDB-ID 2958-0
    ISSN 1538-3598 ; 0254-9077 ; 0002-9955 ; 0098-7484
    ISSN (online) 1538-3598
    ISSN 0254-9077 ; 0002-9955 ; 0098-7484
    DOI 10.1001/jama.295.6.624-b
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  10. Article ; Online: Target blood pressure and kidney disease.

    Kida, Yujiro

    Annals of internal medicine

    2005  Volume 143, Issue 4, Page(s) 310–1; author reply 311

    MeSH term(s) Blood Pressure/physiology ; Disease Progression ; Humans ; Hypertension/drug therapy ; Kidney Diseases/physiopathology
    Language English
    Publishing date 2005-06-20
    Publishing country United States
    Document type Comment ; Letter
    ZDB-ID 336-0
    ISSN 1539-3704 ; 0003-4819
    ISSN (online) 1539-3704
    ISSN 0003-4819
    DOI 10.7326/0003-4819-143-4-200508160-00018
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