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  1. Article ; Online: Unilateral tonsillar enlargement: Beware of hyperplasia after tonsilotomy.

    Liu, Yali / Ma, Guowei / Zhang, Yonghong / Li, Yumei

    Asian journal of surgery

    2024  

    Language English
    Publishing date 2024-04-10
    Publishing country Netherlands
    Document type Letter
    ZDB-ID 1068461-x
    ISSN 0219-3108 ; 1015-9584
    ISSN (online) 0219-3108
    ISSN 1015-9584
    DOI 10.1016/j.asjsur.2024.03.193
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    In stock of ZB MED Cologne/Königswinter

    Zs.B 2831: Show issues Location:
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    ab Jg. 2022: Lesesaal (EG)

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  2. Article ; Online: Deletion of Nox from

    Li, Dezhi / Ma, Wenwen / Chen, Guowei / Huang, Zhiqiang / Liu, Qing

    Foodborne pathogens and disease

    2024  

    Abstract: The foodborne pathogens have a serious threat to human health, ... ...

    Abstract The foodborne pathogens have a serious threat to human health, especially
    Language English
    Publishing date 2024-03-14
    Publishing country United States
    Document type Journal Article
    ZDB-ID 2148479-X
    ISSN 1556-7125 ; 1535-3141
    ISSN (online) 1556-7125
    ISSN 1535-3141
    DOI 10.1089/fpd.2023.0125
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    In stock of ZB MED Bonn / Germany

    Z 7517: Show issues

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  3. Article ; Online: W Deposited PdGa Catalyst with Tailored Hydrogen Adsorption and Reduction.

    Rao, Jiancun / Li, Guowei

    Microscopy and microanalysis : the official journal of Microscopy Society of America, Microbeam Analysis Society, Microscopical Society of Canada

    2023  Volume 29, Issue Supplement_1, Page(s) 1360–1361

    Language English
    Publishing date 2023-08-23
    Publishing country England
    Document type Journal Article
    ZDB-ID 1385710-1
    ISSN 1435-8115 ; 1431-9276
    ISSN (online) 1435-8115
    ISSN 1431-9276
    DOI 10.1093/micmic/ozad067.697
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article: Bioinformatics Analysis of Stem Cell circ-ASB3 Signaling Pathway and Its Affection on Glioma Biological Characteristics.

    Guowei, Li / Yanping, Jin

    Frontiers in neuroinformatics

    2022  Volume 16, Page(s) 859937

    Abstract: Objective: In our research we try to explore whether glioma stem cell containing circRNAs signal pathway could regulate glioma malignant progression and elaborate its possible mechanism.: Methods: In this study, we used biological information ... ...

    Abstract Objective: In our research we try to explore whether glioma stem cell containing circRNAs signal pathway could regulate glioma malignant progression and elaborate its possible mechanism.
    Methods: In this study, we used biological information analysis to build an RNA regulatory network and then proceeded RT-PCR to screen target RNAs, after that we clarified the targeting relationship between circRNA-miRNA-mRNA through double luciferase gene assay, RNA pull down experiment, PCR and Western Blot. Finally we adopted RNA transfection to identify its impact on glioma cell proliferation, invasion, migration, apoptosis and cell cycle.
    Results: circ-ASB3 was significantly up-regulated in glioma stem cells compared with glioma cells. The circ-ASB3/miR-543/Twist1 axis was discovered to be a possible regulatory pathway in glioma, circ-ASB3 could adsorb and targeted bind to miR-543, down-regulate miR-543 expression, thus release its targeted inhibition to Twist1. Circ-ASB3 was shown to increase glioma cell proliferation, invasion, and migration
    Conclusion: circ-ASB3 could enhance glioma malignancy
    Language English
    Publishing date 2022-04-12
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 2452979-5
    ISSN 1662-5196
    ISSN 1662-5196
    DOI 10.3389/fninf.2022.859937
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  5. Article ; Online: Construction of TiO

    Wang, Jun / Fu, Shuang / Hou, Peng / Liu, Jun / Li, Chao / Zhang, Hongguang / Wang, Guowei

    Molecules (Basel, Switzerland)

    2024  Volume 29, Issue 8

    Abstract: Utilizing solar energy for photocatalytic ... ...

    Abstract Utilizing solar energy for photocatalytic CO
    Language English
    Publishing date 2024-04-22
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 1413402-0
    ISSN 1420-3049 ; 1431-5165 ; 1420-3049
    ISSN (online) 1420-3049
    ISSN 1431-5165 ; 1420-3049
    DOI 10.3390/molecules29081899
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    In stock of ZB MED Cologne/Königswinter

    Zs.MO 81: Show issues

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  6. Article: Editorial: Advances in peanut research.

    Chen, Tingting / Li, Guowei / Zhang, Hong

    Frontiers in plant science

    2023  Volume 14, Page(s) 1180009

    Language English
    Publishing date 2023-04-04
    Publishing country Switzerland
    Document type Editorial
    ZDB-ID 2613694-6
    ISSN 1664-462X
    ISSN 1664-462X
    DOI 10.3389/fpls.2023.1180009
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  7. Article ; Online: Synthesis of the [6-6-7-5-5] Pentacyclic Core of Calyciphylline N.

    Zhao, Zhiwen / Deng, Guowei / Li, Chuang-Chuang

    Organic letters

    2024  Volume 26, Issue 11, Page(s) 2238–2242

    Abstract: A new approach for the concise 11-step synthesis of the [6-6-7-5-5] BCDEF pentacyclic core of calyciphylline N is described. A type II [5 + 2] cycloaddition was employed to construct the strained BCD skeleton, which encompasses the challenging bicyclo[2 ... ...

    Abstract A new approach for the concise 11-step synthesis of the [6-6-7-5-5] BCDEF pentacyclic core of calyciphylline N is described. A type II [5 + 2] cycloaddition was employed to construct the strained BCD skeleton, which encompasses the challenging bicyclo[2.2.2] and bicyclo[4.3.1] ring systems. With a regio- and diastereoselective Lu's [3 + 2] cycloaddition, followed by intramolecular aldol cyclization and elimination, the desired [5-5]-fused EF ring system has been successfully installed, resulting in the complete carbocyclic skeleton of calyciphylline N.
    Language English
    Publishing date 2024-03-04
    Publishing country United States
    Document type Journal Article
    ISSN 1523-7052
    ISSN (online) 1523-7052
    DOI 10.1021/acs.orglett.4c00437
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  8. Article ; Online: The FDX1 methylation regulatory mechanism in the malignant phenotype of glioma.

    Guowei, Li / Xiufang, Lu / Qianqian, Xu / Yanping, Jin

    Genomics

    2023  Volume 115, Issue 2, Page(s) 110601

    Abstract: To explore FDX1 methylation as a regulatory mechanism in the malignant phenotype of glioma, we screened for pathways involved through bioinformatic analysis, then proceeded with RIP and cell models to verify the regulation of RNAs and mitophagy. We chose ...

    Abstract To explore FDX1 methylation as a regulatory mechanism in the malignant phenotype of glioma, we screened for pathways involved through bioinformatic analysis, then proceeded with RIP and cell models to verify the regulation of RNAs and mitophagy. We chose Clone and Transwell assays to evaluate the malignant phenotype of glioma cells. MMP was detected by flow cytometry and mitochondrial morphology was observed by TEM. We also constructed animal models to study the sensitivity of glioma cells to cuproptosis. We successfully identified the signalling pathway: our cell model showed that C-MYC could upregulate FDX1 through YTHDF1 and inhibit mitophagy in glioma cells. Functional experiments revealed C-MYC could also enhance glioma cell proliferation and invasion via YTHDF1 and FDX1. In vivo experiments showed glioma cells were highly sensitive to cuproptosis. We concluded that C-MYC could upregulate FDX1 by m6A methylation, thus promoting the malignant phenotype in glioma cells.
    MeSH term(s) Animals ; Cell Line, Tumor ; Cell Proliferation/genetics ; Glioma/metabolism ; Methylation ; MicroRNAs/metabolism ; Phenotype ; Ferredoxins/metabolism
    Chemical Substances MicroRNAs ; Ferredoxins
    Language English
    Publishing date 2023-03-06
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 356334-0
    ISSN 1089-8646 ; 0888-7543
    ISSN (online) 1089-8646
    ISSN 0888-7543
    DOI 10.1016/j.ygeno.2023.110601
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 2367: Show issues Location:
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    Jg. 1995 - 2021: Lesesall (2.OG)
    ab Jg. 2022: Lesesaal (EG)

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  9. Article ; Online: Preliminary experimental study on the prevention of all-trans retinoic acid induced cleft palate in mice by nicotinamide

    LI Guowei / TANG Shijie

    口腔疾病防治, Vol 31, Iss 9, Pp 634-

    2023  Volume 640

    Abstract: Objective To explore the preventive effect of nicotinamide (NAM) on cleft palate induced by all-trans retinoic acid (RA), to provide research evidence for the prevention of cleft palate. Methods The mouse cleft palate model was induced by intragastric ... ...

    Abstract Objective To explore the preventive effect of nicotinamide (NAM) on cleft palate induced by all-trans retinoic acid (RA), to provide research evidence for the prevention of cleft palate. Methods The mouse cleft palate model was induced by intragastric administration of 70 mg/kg all-trans retinoic acid at embryonic day 10.5 (E10.5) in the control group. The mouse cleft palate model was treated by caudal vein injection of 20 mg/kg NAM at E8.5 to E13.5 in the experimental group (1). The cleft palate model was treated by caudal vein injection of 40 mg/kg NAM at E8.5-E13.5 in the experimental group (2). The cleft palate of fetal rats was observed by laparotomy on E16.5 and statistically analyzed. Annexin V-FITC/PI double staining was used to detect the apoptosis of mouse embryonic palatal mesenchyme (MEPM) cells treated with RA 1 μmol/L (RA 1 group), NAM 200 μmol/L (NAM 200 group), and both NAM 200 μmol/L and RA 1 μmol/L (NAM 200+RA 1 group) for 24 hours by flow cytometry and the apoptosis rate in groups were compared. Culture without RA or NAM was used as a control. Results The cleft palate rate in the control group was 98%. The cleft palate rate in experimental group (1) was 87%. There was no significant difference between groups (P>0.05). The cleft palate rate in the experimental group (2) was 63%, compared with the control group, there was a significant difference (P<0.01). The cell apoptosis rate was 16.53%±2.89% in the CONTROL group. The cell apoptosis rate was 22.9%±1.85% in the RA 1 group, which was a significant increase compared with the CONTROL group (P<0.01). The apoptotic rate of the NAM 200 group was 9.23%±1.39%, which was a significant decrease compared with NA 1 group (P<0.01). The apoptosis rate of the NAM 200+RA 1 group was 14.9%±7.67%, which was a significant decrease compared with the RA 1 group (P<0.01). Conclusion NAM can prevent cleft palate. 40 mg/kg nicotinamide during pregnancy is an effective concentration for the prevention of RA-induced cleft palate. The mechanism by which ...
    Keywords nicotinamide ， ; all-trans retinoic acid ， ; cleft palate ， ; cell apoptosis ， ; congenital malformation ， ; mouse embryonic palatal mesenchyme ， ; pregnancy care ， ; disease prevention， ; Medicine ; R
    Subject code 630
    Language Chinese
    Publishing date 2023-09-01T00:00:00Z
    Publisher Editorial Department of Journal of Prevention and Treatment for Stomatological Diseases
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  10. Article ; Online: Dietary Habits and Metabolic Health.

    Li, Bowen / Tang, Xue / Le, Guowei

    Nutrients

    2023  Volume 15, Issue 18

    Abstract: Dietary habits refer to the long-term dietary patterns and habits that an individual forms and maintains in their daily life [ ... ]. ...

    Abstract Dietary habits refer to the long-term dietary patterns and habits that an individual forms and maintains in their daily life [...].
    MeSH term(s) Diet ; Feeding Behavior
    Language English
    Publishing date 2023-09-14
    Publishing country Switzerland
    Document type Editorial
    ZDB-ID 2518386-2
    ISSN 2072-6643 ; 2072-6643
    ISSN (online) 2072-6643
    ISSN 2072-6643
    DOI 10.3390/nu15183975
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