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Article ; Online: Responses of organ precursors to correct and incorrect inductive signals.

Yang, Yun / Li, Shuang / Luo, Lingfei

2023

Abstract: During embryonic development, the inductive molecules produced by local origins normally arrive at their target tissues in a nondirectional, diffusion manner. The target organ precursor cells must correctly interpret these inductive signals to ensure ... ...

Abstract	During embryonic development, the inductive molecules produced by local origins normally arrive at their target tissues in a nondirectional, diffusion manner. The target organ precursor cells must correctly interpret these inductive signals to ensure proper specification/differentiation, which is dependent on two prerequisites: (i) obtaining cell-intrinsic competence; and (ii) receiving correct inductive signals while resisting incorrect ones. Gain of intrinsic competence could avoid a large number of misinductions because the incompetent cells are nonresponsive to inductive signals. However, in cases of different precursor cells with similar competence and located in close proximity, resistance to incorrect inductive signals is essential for accurate determination of cell fate. Here we outline the mechanisms of how organ precursors respond to correct and incorrect inductive signals.
Language	English
Publishing date	2023-09-20
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	30122-x
ISSN	1879-3088 ; 0962-8924
ISSN (online)	1879-3088
ISSN	0962-8924
DOI	10.1016/j.tcb.2023.08.008
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Article ; Online: Polycystic kidney disease: novel insights into polycystin function.

Luo, Lingfei / Roy, Sudipto / Li, Li / Ma, Ming

Trends in molecular medicine

2023 Volume 29, Issue 4, Page(s) 268–281

Abstract: Autosomal dominant polycystic kidney disease (ADPKD) is a life-threatening monogenic disease caused by mutations in PKD1 and PKD2 that encode polycystin 1 (PC1) and polycystin 2 (PC2). PC1/2 localize to cilia of renal epithelial cells, and their function ...

Abstract	Autosomal dominant polycystic kidney disease (ADPKD) is a life-threatening monogenic disease caused by mutations in PKD1 and PKD2 that encode polycystin 1 (PC1) and polycystin 2 (PC2). PC1/2 localize to cilia of renal epithelial cells, and their function is believed to embody an inhibitory activity that suppresses the cilia-dependent cyst activation (CDCA) signal. Consequently, PC deficiency results in activation of CDCA and stimulates cyst growth. Recently, re-expression of PCs in established cysts has been shown to reverse PKD. Thus, the mode of action of PCs resembles a 'counterbalance in cruise control' to maintain lumen diameter within a designated range. Herein we review recent studies that point to novel arenas for future PC research with therapeutic potential for ADPKD.
MeSH term(s)	Humans ; Polycystic Kidney, Autosomal Dominant/genetics ; TRPP Cation Channels/genetics ; TRPP Cation Channels/metabolism ; Polycystic Kidney Diseases/genetics ; Polycystic Kidney Diseases/complications ; Signal Transduction ; Cysts/complications ; Kidney/metabolism
Chemical Substances	TRPP Cation Channels
Language	English
Publishing date	2023-02-15
Publishing country	England
Document type	Journal Article ; Review ; Research Support, Non-U.S. Gov't
ZDB-ID	2036490-8
ISSN	1471-499X ; 1471-4914
ISSN (online)	1471-499X
ISSN	1471-4914
DOI	10.1016/j.molmed.2023.01.005
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Article ; Online: Brain vascular damage-induced lymphatic ingrowth is directed by Cxcl12b/Cxcr4a.

Chen, Jingying / He, Jianbo / Luo, Lingfei

Development (Cambridge, England)

2022 Volume 149, Issue 13

Abstract: After ischemic stroke, promotion of vascular regeneration without causing uncontrolled vessel growth appears to be the major challenge for pro-angiogenic therapies. The molecular mechanisms underlying how nascent blood vessels (BVs) are correctly guided ... ...

Abstract	After ischemic stroke, promotion of vascular regeneration without causing uncontrolled vessel growth appears to be the major challenge for pro-angiogenic therapies. The molecular mechanisms underlying how nascent blood vessels (BVs) are correctly guided into the post-ischemic infarction area remain unknown. Here, using a zebrafish cerebrovascular injury model, we show that chemokine signaling provides crucial guidance cues to determine the growing direction of ingrown lymphatic vessels (iLVs) and, in turn, that of nascent BVs. The chemokine receptor Cxcr4a is transcriptionally activated in the iLVs after injury, whereas its ligand Cxcl12b is expressed in the residual central BVs, the destinations of iLV ingrowth. Mutant and mosaic studies indicate that Cxcl12b/Cxcr4a-mediated chemotaxis is necessary and sufficient to determine the growing direction of iLVs and nascent BVs. This study provides a molecular basis for how the vessel directionality of cerebrovascular regeneration is properly determined, suggesting potential application of Cxcl12b/Cxcr4a in the development of post-ischemic pro-angiogenic therapies.
MeSH term(s)	Animals ; Brain/metabolism ; Gene Expression Regulation, Developmental ; Lymphatic Vessels/metabolism ; Receptors, CXCR4/genetics ; Receptors, CXCR4/metabolism ; Zebrafish/genetics
Chemical Substances	Receptors, CXCR4
Language	English
Publishing date	2022-06-30
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	90607-4
ISSN	1477-9129 ; 0950-1991
ISSN (online)	1477-9129
ISSN	0950-1991
DOI	10.1242/dev.200729
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Article ; Online: Sulfamethoxazole induces brain capillaries toxicity in zebrafish by up-regulation of VEGF and chemokine signalling

Yuhang Xu / Lingfei Luo / Jingying Chen

Ecotoxicology and Environmental Safety, Vol 238, Iss , Pp 113620- (2022)

2022

Abstract: Sulfamethoxazole (SMX) is a widespread broad-spectrum bacteriostatic antibiotic. Its residual is frequently detected in the water and may therefore bioaccumulate in the brain of aquatic organisms via blood circulation. Brain capillaries toxicity is very ... ...

Abstract	Sulfamethoxazole (SMX) is a widespread broad-spectrum bacteriostatic antibiotic. Its residual is frequently detected in the water and may therefore bioaccumulate in the brain of aquatic organisms via blood circulation. Brain capillaries toxicity is very important for brain development. However, little information is available in the literature to show the toxicity of SMX to brain development. To study the SMX’s brain toxic effects and the related mechanisms, we exposed zebrafish embryos to SMX at different concentrations (0 ppm, 1 ppm, 25 ppm, 100 ppm and 250 ppm) and found that high concentration (250 ppm) of SMX would not only caused an abnormal in malformation rate, hatching rate, body length and survival rate of zebrafish embryos, but also lead to brain oedema. In addition, SMX also induced cerebral ischaemia, aggravates oxidative stress, and changes genes related to oxidative stress (sod1, cat, gpx4, and nrf2). Furthermore, ischaemia caused by SMX could promote ectopic angiogenesis in brain via activating the angiogenesis-related genes (vegfab, cxcr4a, cxcl12b) from 24 h to 53 h. Inhibition of VEGF signalling by SU5416, or inhibition of chemokine downstream PI3K signalling by LY294002, could rescue the brain capillaries toxicity and brain oedema induced by SMX. Our results provide new evidence for the brain toxicity of SMX and its residual danger in the environment and aquatic organisms.
Keywords	Sulfamethoxazole ; Brain capillaries toxicity ; Brain oedema ; VEGF ; Chemokine ; Environmental pollution ; TD172-193.5 ; Environmental sciences ; GE1-350
Subject code	616
Language	English
Publishing date	2022-06-01T00:00:00Z
Publisher	Elsevier
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: Sulfamethoxazole induces brain capillaries toxicity in zebrafish by up-regulation of VEGF and chemokine signalling.

Xu, Yuhang / Luo, Lingfei / Chen, Jingying

Ecotoxicology and environmental safety

2022 Volume 238, Page(s) 113620

Abstract	Sulfamethoxazole (SMX) is a widespread broad-spectrum bacteriostatic antibiotic. Its residual is frequently detected in the water and may therefore bioaccumulate in the brain of aquatic organisms via blood circulation. Brain capillaries toxicity is very important for brain development. However, little information is available in the literature to show the toxicity of SMX to brain development. To study the SMX's brain toxic effects and the related mechanisms, we exposed zebrafish embryos to SMX at different concentrations (0 ppm, 1 ppm, 25 ppm, 100 ppm and 250 ppm) and found that high concentration (250 ppm) of SMX would not only caused an abnormal in malformation rate, hatching rate, body length and survival rate of zebrafish embryos, but also lead to brain oedema. In addition, SMX also induced cerebral ischaemia, aggravates oxidative stress, and changes genes related to oxidative stress (sod1, cat, gpx4, and nrf2). Furthermore, ischaemia caused by SMX could promote ectopic angiogenesis in brain via activating the angiogenesis-related genes (vegfab, cxcr4a, cxcl12b) from 24 h to 53 h. Inhibition of VEGF signalling by SU5416, or inhibition of chemokine downstream PI3K signalling by LY294002, could rescue the brain capillaries toxicity and brain oedema induced by SMX. Our results provide new evidence for the brain toxicity of SMX and its residual danger in the environment and aquatic organisms.
MeSH term(s)	Animals ; Aquatic Organisms ; Brain ; Brain Edema/chemically induced ; Capillaries ; Sulfamethoxazole/toxicity ; Up-Regulation ; Vascular Endothelial Growth Factor A/genetics ; Water Pollutants, Chemical/toxicity ; Zebrafish
Chemical Substances	Vascular Endothelial Growth Factor A ; Water Pollutants, Chemical ; Sulfamethoxazole (JE42381TNV)
Language	English
Publishing date	2022-05-10
Publishing country	Netherlands
Document type	Journal Article
ZDB-ID	436536-7
ISSN	1090-2414 ; 0147-6513
ISSN (online)	1090-2414
ISSN	0147-6513
DOI	10.1016/j.ecoenv.2022.113620
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Article ; Online: The Lysosomal Storage Disorder Due to

Bao, Wandong / Wang, Xinjuan / Luo, Lingfei / Ni, Rui

Zebrafish

2021 Volume 18, Issue 3, Page(s) 175–183

Abstract: The phospholipid phosphatase FIG4/Fig4 is a subunit of PIKFYVE/Pikfyve kinase complex that synthesizes phosphatidylinositol 3,5-bisphosphate (PI(3,5) ... ...

Abstract	The phospholipid phosphatase FIG4/Fig4 is a subunit of PIKFYVE/Pikfyve kinase complex that synthesizes phosphatidylinositol 3,5-bisphosphate (PI(3,5)P
MeSH term(s)	Animals ; Liver ; Lysosomes ; Mutation ; Phosphoric Monoester Hydrolases/genetics ; Zebrafish/genetics
Chemical Substances	Phosphoric Monoester Hydrolases (EC 3.1.3.2)
Language	English
Publishing date	2021-04-26
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2156020-1
ISSN	1557-8542 ; 1545-8547
ISSN (online)	1557-8542
ISSN	1545-8547
DOI	10.1089/zeb.2020.1911
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Article ; Online: Concentrations and influencing factors of 17 elements in placenta, cord blood, and maternal blood of women from an e-waste recycling area.

Luo, Yacui / Zhang, Haijun / Gui, Fangzhong / Fang, Jiayang / Lin, Haijiang / Qiu, Danhong / Ge, Lingfei / Wang, Qiong / Xu, Peiwei / Tang, Jun

Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS)

2024 Volume 84, Page(s) 127449

Abstract: Background: The effects of prenatal element exposure on mothers and fetuses have generated concern. Profiles of trace and toxic elements in biological material are urgently desired, especially for women who reside near e-waste recycling facilities. The ... ...

Abstract	Background: The effects of prenatal element exposure on mothers and fetuses have generated concern. Profiles of trace and toxic elements in biological material are urgently desired, especially for women who reside near e-waste recycling facilities. The aim of this study was to investigate elements concentrations in placenta, cord blood, and maternal blood of women and to evaluate the influencing factors. Methods: A group of 48 women from an e-waste recycling site and a group of 31 women from a non-e-waste recycling site were recruited. Basic characteristics were collected by questionnaire and the concentrations of 17 elements in placenta, cord blood, and maternal blood samples were analyzed by inductively coupled plasma mass spectrometry (ICP-MS). Finally, the generalized linear model regression analysis (GLM) was used to test the association between element concentrations and possible factors. Results: Compared to the control group, the exposed group had significantly elevated cadmium (Cd), zinc (Zn), nickel (Ni), and antimony (Sb) in placenta, and higher lead (Pb) in maternal blood and cord blood (P<0.05). Sb concentration in maternal blood was significantly lower than in the control group (P<0.05). GLM analysis showed that element concentrations were mainly associated with maternal age [chromium (Cr), iron (Fe), selenium (Se), cobalt (Co), mercury (Hg) in placenta, copper (Cu) in maternal blood], education (Se, Sb in placenta), family income (Cu in maternal blood and Ni in placenta), passive smoking [Cu and Zn in placenta, Pb in maternal blood], and e-waste contact history (Hg in cord blood, Cu, Zn, and Cd in maternal blood). Conclusions: Women in the e-waste recycling area had higher toxic element levels in the placenta and blood samples. More preventive measures were needed to reduce the risk of element exposure for mothers and fetuses in these areas.
Language	English
Publishing date	2024-04-12
Publishing country	Germany
Document type	Journal Article
ZDB-ID	1236267-0
ISSN	1878-3252 ; 1611-602X ; 0946-672X
ISSN (online)	1878-3252 ; 1611-602X
ISSN	0946-672X
DOI	10.1016/j.jtemb.2024.127449
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Article ; Online: Non-destructive measurements for the evaluation of beef freshness based on airflow interaction and a structured light 3D imaging technique and comparison with pH value and total volatile basic nitrogen content

Luo, Xiuzhi / Sun, Qinming / Zhou, Lingfei / He, Ke / Gao, Xin / Tang, Xiuying

IAgrE Biosystems Engineering. 2023 Feb., v. 226 p.182-193

2023

Abstract: This study aimed to investigate the potential ability to predict total volatile basic nitrogen (TVB-N) content and pH value for the freshness evaluation of beef by examining instantaneous elastoplastic deformation of the meat caused by airflow using a ... ...

Abstract	This study aimed to investigate the potential ability to predict total volatile basic nitrogen (TVB-N) content and pH value for the freshness evaluation of beef by examining instantaneous elastoplastic deformation of the meat caused by airflow using a structured light 3D imaging technique. A constant airflow impacted the beef surface whilst a 3D camera continuously captured point cloud images of the concave area. The volume, surface area, and depth of spatial deformation, as well as the projected area of three planes, were obtained using the K-nearest neighbour algorithm, down-sampling processing, the oriented bounding box algorithm, and the Alpha Shape algorithm. The instantaneous recovery and residual of each quantified deformation feature were obtained at the moment of air unloading to get the instantaneous elastic–plastic deformation and degree of instantaneous elastic–plastic deformation of beef's spatial deformation feature. A correlation analysis of the obtained features and two freshness indicators (pH value and TVB-N content) was carried out for feature selection and modelling. The results showed that the models using the degree of instantaneous elastic deformation of spatial deformation features of beef with feature selection produced the best prediction results in this study. For beef pH values and TVB-N content evaluation, the correlation coefficient in the prediction set were 0.745 and 0.844, respectively.
Keywords	air ; air flow ; algorithms ; beef ; cameras ; data collection ; elastic deformation ; freshness ; nitrogen content ; pH ; prediction ; surface area ; total volatile basic nitrogen ; TVB-N and pH ; Airflow ; Spatial deformation ; Structured light 3D imaging ; Instantaneous elastoplasticity
Language	English
Dates of publication	2023-02
Size	p. 182-193.
Publishing place	Elsevier Ltd
Document type	Article ; Online
ZDB-ID	2075942-3
ISSN	1537-5110
ISSN	1537-5110
DOI	10.1016/j.biosystemseng.2023.01.006
Database	NAL-Catalogue (AGRICOLA)

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Article ; Online: Pax4-Ghrelin mediates the conversion of pancreatic ε-cells to β-cells after extreme β-cell loss in zebrafish.

Yu, Junqin / Ma, Jianlong / Li, Yanfeng / Zhou, Yang / Luo, Lingfei / Yang, Yun

Development (Cambridge, England)

2023 Volume 150, Issue 6

Abstract: Pancreatic ε-cells producing ghrelin are one type of endocrine cell found in islets, which have been shown to influence other intra-islet cells, especially in regulating the function of β cells. However, the role of such cells during β-cell regeneration ... ...

Abstract	Pancreatic ε-cells producing ghrelin are one type of endocrine cell found in islets, which have been shown to influence other intra-islet cells, especially in regulating the function of β cells. However, the role of such cells during β-cell regeneration is currently unknown. Here, using a zebrafish nitroreductase (NTR)-mediated β-cell ablation model, we reveal that ghrelin-positive ε-cells in the pancreas act as contributors to neogenic β-cells after extreme β-cell loss. Further studies show that the overexpression of ghrelin or the expansion of ε-cells potentiates β-cell regeneration. Lineage tracing confirms that a proportion of embryonic ε-cells can transdifferentiate to β-cells, and that the deletion of Pax4 enhances this transdifferentiation of ε-cells to β-cells. Mechanistically, Pax4 binds to the ghrelin regulatory region and represses its transcription. Thus, deletion of Pax4 derepresses ghrelin expression and causes producing more ghrelin-positive cells, enhancing the transdifferentiation of ε-cells to β-cells and consequently potentiating β-cell regeneration. Our findings reveal a previously unreported role for ε-cells during zebrafish β-cell regeneration, indicating that Pax4 regulates ghrelin transcription and mediates the conversion of embryonic ε-cells to β-cells after extreme β-cell loss.
MeSH term(s)	Animals ; Ghrelin/metabolism ; Homeodomain Proteins/metabolism ; Pancreas ; Transcription Factors/metabolism ; Zebrafish/genetics ; Zebrafish/metabolism
Chemical Substances	Ghrelin ; Homeodomain Proteins ; Transcription Factors ; pax4 protein, zebrafish
Language	English
Publishing date	2023-03-27
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	90607-4
ISSN	1477-9129 ; 0950-1991
ISSN (online)	1477-9129
ISSN	0950-1991
DOI	10.1242/dev.201306
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Article ; Online: VEGF signaling governs the initiation of biliary-mediated liver regeneration through the PI3K-mTORC1 axis.

Cai, Pengcheng / Ni, Rui / Lv, Mengzhu / Liu, Huijuan / Zhao, Jieqiong / He, Jianbo / Luo, Lingfei

Cell reports

2023 Volume 42, Issue 9, Page(s) 113028

Abstract: Biliary epithelial cells (BECs) are a potential source to repair the damaged liver when hepatocyte proliferation is compromised. Promotion of BEC-to-hepatocyte transdifferentiation could be beneficial to the clinical therapeutics of patients with end- ... ...

Abstract	Biliary epithelial cells (BECs) are a potential source to repair the damaged liver when hepatocyte proliferation is compromised. Promotion of BEC-to-hepatocyte transdifferentiation could be beneficial to the clinical therapeutics of patients with end-stage liver diseases. However, mechanisms underlying the initiation of BEC transdifferentiation remain largely unknown. Here, we show that upon extreme hepatocyte injury, vegfaa and vegfr2/kdrl are notably induced in hepatic stellate cells and BECs, respectively. Pharmacological and genetic inactivation of vascular endothelial growth factor (VEGF) signaling would disrupt BEC dedifferentiation and proliferation, thus restraining hepatocyte regeneration. Mechanically, VEGF signaling regulates the activation of the PI3K-mammalian target of rapamycin complex 1 (mTORC1) axis, which is essential for BEC-to-hepatocyte transdifferentiation. In mice, VEGF signaling exerts conserved roles in oval cell activation and BEC-to-hepatocyte differentiation. Taken together, this study shows VEGF signaling as an initiator of biliary-mediated liver regeneration through activating the PI3K-mTORC1 axis. Modulation of VEGF signaling in BECs could be a therapeutic approach for patients with end-stage liver diseases.
MeSH term(s)	Humans ; Animals ; Mice ; Vascular Endothelial Growth Factor A ; Phosphatidylinositol 3-Kinases ; Liver Regeneration/physiology ; Hepatocytes ; Cell Proliferation ; Liver Diseases ; Mechanistic Target of Rapamycin Complex 1 ; Liver ; Mammals
Chemical Substances	Vascular Endothelial Growth Factor A ; Phosphatidylinositol 3-Kinases (EC 2.7.1.-) ; Mechanistic Target of Rapamycin Complex 1 (EC 2.7.11.1)
Language	English
Publishing date	2023-08-24
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2649101-1
ISSN	2211-1247 ; 2211-1247
ISSN (online)	2211-1247
ISSN	2211-1247
DOI	10.1016/j.celrep.2023.113028
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