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  1. Article ; Online: Hydroxychloroquine and azithromycin as a treatment of COVID-19.

    Fanin, Alice / Calegari, Jessica / Beverina, Anna / Tiraboschi, Silvia

    Internal and emergency medicine

    2020  Volume 15, Issue 5, Page(s) 841–843

    MeSH term(s) Azithromycin ; Betacoronavirus ; COVID-19 ; Coronavirus Infections/drug therapy ; France ; Humans ; Hydroxychloroquine ; Pandemics ; Pneumonia, Viral ; Retrospective Studies ; SARS-CoV-2 ; COVID-19 Drug Treatment
    Chemical Substances Hydroxychloroquine (4QWG6N8QKH) ; Azithromycin (83905-01-5)
    Keywords covid19
    Language English
    Publishing date 2020-05-30
    Publishing country Italy
    Document type Journal Article ; Comment
    ZDB-ID 2454173-4
    ISSN 1970-9366 ; 1828-0447
    ISSN (online) 1970-9366
    ISSN 1828-0447
    DOI 10.1007/s11739-020-02388-y
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article ; Online: Hydroxychloroquine and azithromycin as a treatment of COVID-19

    Fanin, Alice / Calegari, Jessica / Beverina, Anna / Tiraboschi, Silvia

    Internal and Emergency Medicine

    2020  Volume 15, Issue 5, Page(s) 841–843

    Keywords Internal Medicine ; Emergency Medicine ; covid19
    Language English
    Publisher Springer Science and Business Media LLC
    Publishing country us
    Document type Article ; Online
    ZDB-ID 2454173-4
    ISSN 1970-9366 ; 1828-0447
    ISSN (online) 1970-9366
    ISSN 1828-0447
    DOI 10.1007/s11739-020-02388-y
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  4. Article ; Online: Liver alterations in anorexia nervosa are not caused by insulin resistance.

    Fanin, Alice / Miele, Lucia / Bertolini, Emanuela / Giorgini, Alessia / Pontiroli, Antonio Ettore / Benetti, Alberto

    Internal and emergency medicine

    2019  Volume 15, Issue 2, Page(s) 337–339

    Abstract: Background: Liver dysfunction has been widely reported in connection with anorexia nervosa (AN) but the pathogenesis of these alterations has never been fully understood despite reported theories about the presence of insulin resistance (IR) and non- ... ...

    Abstract Background: Liver dysfunction has been widely reported in connection with anorexia nervosa (AN) but the pathogenesis of these alterations has never been fully understood despite reported theories about the presence of insulin resistance (IR) and non-alcoholic fatty liver disease (NAFLD). The aim of this study is to investigate if hypertransaminasemia in AN is linked to IR and NAFLD.
    Methods: Anthropometric data and laboratory exams of 34 patients and 34 controls were analyzed, including alanine-aminotransferase, aspartate-aminotransferase and homeostatic model assessment of insulin resistance (HOMA-IR) index. All subjects also underwent magnetic resonance imaging (MRI), ultrasonography (US), and transient elastography (TE).
    Results: Evidence of increased alanine aminotransferase in AN patients was confirmed in our sample together with a lower HOMA-IR index compared to controls. Positive results in US appeared in 16 patients vs none in controls (p = 0.0007); patients with liver parenchyma abnormalities in US were not different than normal-US patients in any of the studied variables. Only one patient showed non-alcoholic fatty liver disease in MRI while abnormal TE was found in four patients and never in controls.
    Conclusions: Liver damage suggested by increased serum liver enzymes cannot be due to liver steatosis but potentially to a different liver disease (not identified by MRI) or to an early liver fibrosis not associated with an insulin-resistant status.
    MeSH term(s) Alanine Transaminase/analysis ; Alanine Transaminase/blood ; Anorexia Nervosa/complications ; Anorexia Nervosa/physiopathology ; Anthropometry/methods ; Fatty Liver/blood ; Fatty Liver/etiology ; Humans ; Insulin Resistance/physiology ; Liver/abnormalities ; Liver/physiopathology ; Ultrasonography/methods
    Chemical Substances Alanine Transaminase (EC 2.6.1.2)
    Language English
    Publishing date 2019-11-16
    Publishing country Italy
    Document type Letter
    ZDB-ID 2454173-4
    ISSN 1970-9366 ; 1828-0447
    ISSN (online) 1970-9366
    ISSN 1828-0447
    DOI 10.1007/s11739-019-02227-9
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  5. Article ; Online: Response to Zoppini et al.

    Fanin, Alice / Benetti, Alberto / Zakaria, Ahmed / Ceriani, Valerio / Pontiroli, Antonio E

    The American journal of gastroenterology

    2015  Volume 110, Issue 2, Page(s) 353–354

    MeSH term(s) Diabetes Complications/mortality ; End Stage Liver Disease/mortality ; Female ; Humans ; Male
    Language English
    Publishing date 2015-02
    Publishing country United States
    Document type Comment ; Letter
    ZDB-ID 390122-1
    ISSN 1572-0241 ; 0002-9270
    ISSN (online) 1572-0241
    ISSN 0002-9270
    DOI 10.1038/ajg.2014.420
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  6. Article ; Online: Becker muscular dystrophy due to an intronic splicing mutation inducing a dual dystrophin transcript.

    Todeschini, Alice / Gualandi, Francesca / Trabanelli, Cecilia / Armaroli, Annarita / Ravani, Anna / Fanin, Marina / Rota, Silvia / Bello, Luca / Ferlini, Alessandra / Pegoraro, Elena / Padovani, Alessandro / Filosto, Massimiliano

    Neuromuscular disorders : NMD

    2016  Volume 26, Issue 10, Page(s) 662–665

    Abstract: We describe a 29-year-old patient who complained of left thigh muscle weakness since he was 23 and of moderate proximal weakness of both lower limbs with difficulty in climbing stairs and running since he was 27. Mild weakness of iliopsoas and quadriceps ...

    Abstract We describe a 29-year-old patient who complained of left thigh muscle weakness since he was 23 and of moderate proximal weakness of both lower limbs with difficulty in climbing stairs and running since he was 27. Mild weakness of iliopsoas and quadriceps muscles and muscle atrophy of both the distal forearm and thigh were observed upon clinical examination. He harboured a novel c.1150-3C>G substitution in the DMD gene, affecting the intron 10 acceptor splice site and causing exon 11 skipping and an out-of-frame transcript. However, protein of normal molecular weight but in reduced amounts was observed on Western Blot analysis. Reverse transcription analysis on muscle RNA showed production, via alternative splicing, of a transcript missing exon 11 as well as a low abundant full-length transcript which is enough to avoid the severe Duchenne phenotype. Our study showed that a reduced amount of full length dystrophin leads to a mild form of Becker muscular dystrophy. These results confirm earlier findings that low amounts of dystrophin can be associated with a milder phenotype, which is promising for therapies aiming at dystrophin restoration.
    Language English
    Publishing date 2016-10
    Publishing country England
    Document type Journal Article
    ZDB-ID 1077681-3
    ISSN 1873-2364 ; 0960-8966
    ISSN (online) 1873-2364
    ISSN 0960-8966
    DOI 10.1016/j.nmd.2016.08.007
    Database MEDical Literature Analysis and Retrieval System OnLINE

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