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  1. AU=Ahmad Shah Adil Ishtiyaq
  2. AU="Bock, Thomas"
  3. AU=Chang Hyejung
  4. AU="Messer, Alison"
  5. AU="Samarzija, Miroslav"
  6. AU="Oh, Yun-Hee"
  7. AU="Ramos, Jairo"
  8. AU="Chauhan Kushwah, Vinita"
  9. AU="Winter, Katrin"
  10. AU="Berro, Julien"
  11. AU=Cummins Claire B.
  12. AU="Damholt, A"
  13. AU="Muthu, Santhosh Kumar"
  14. AU="Tysinger, Emma"
  15. AU=Covarrubias David
  16. AU="Dino Papeš"
  17. AU="Assis, Daniel Barbosa"
  18. AU="Lauquin, Guy J-M"

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  1. Artikel ; Online: Bone Marrow Transplantation Dynamics: When Progenitor Expansion Prevails.

    Nakamura-Ishizu, Ayako / Ahmad, Shah Adil Ishtiyaq / Suda, Toshio

    Trends in cell biology

    2020  Band 30, Heft 11, Seite(n) 835–836

    Abstract: The transplantation of healthy hematopoietic stem cells (HSCs) contained in the bone marrow is a frontline treatment option for hematopoietic diseases. Detailed analysis of post-transplant HSC kinetics is crucial as the initial engraftment of HSCs ... ...

    Abstract The transplantation of healthy hematopoietic stem cells (HSCs) contained in the bone marrow is a frontline treatment option for hematopoietic diseases. Detailed analysis of post-transplant HSC kinetics is crucial as the initial engraftment of HSCs influences prognosis. Dong et al. have explored the dynamic change in HSC cell fate upon bone marrow transplantation through the utilization of single-cell transcriptomic analysis.
    Mesh-Begriff(e) Bone Marrow Transplantation ; Cell Differentiation ; Hematopoietic Stem Cell Transplantation ; Hematopoietic Stem Cells ; Humans ; Transcriptome
    Sprache Englisch
    Erscheinungsdatum 2020-09-10
    Erscheinungsland England
    Dokumenttyp Journal Article ; Comment
    ZDB-ID 30122-x
    ISSN 1879-3088 ; 0962-8924
    ISSN (online) 1879-3088
    ISSN 0962-8924
    DOI 10.1016/j.tcb.2020.08.006
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  2. Artikel ; Online: MITOL deficiency triggers hematopoietic stem cell apoptosis via ER stress response.

    Ma, Wenjuan / Ahmad, Shah Adil Ishtiyaq / Hashimoto, Michihiro / Khalilnezhad, Ahad / Kataoka, Miho / Arima, Yuichiro / Tanaka, Yosuke / Yanagi, Shigeru / Umemoto, Terumasa / Suda, Toshio

    The EMBO journal

    2024  Band 43, Heft 3, Seite(n) 339–361

    Abstract: Hematopoietic stem cell (HSC) divisional fate and function are determined by cellular metabolism, yet the contribution of specific cellular organelles and metabolic pathways to blood maintenance and stress-induced responses in the bone marrow remains ... ...

    Abstract Hematopoietic stem cell (HSC) divisional fate and function are determined by cellular metabolism, yet the contribution of specific cellular organelles and metabolic pathways to blood maintenance and stress-induced responses in the bone marrow remains poorly understood. The outer mitochondrial membrane-localized E3 ubiquitin ligase MITOL/MARCHF5 (encoded by the Mitol gene) is known to regulate mitochondrial and endoplasmic reticulum (ER) interaction and to promote cell survival. Here, we investigated the functional involvement of MITOL in HSC maintenance by generating MX1-cre inducible Mitol knockout mice. MITOL deletion in the bone marrow resulted in HSC exhaustion and impairment of bone marrow reconstitution capability in vivo. Interestingly, MITOL loss did not induce major mitochondrial dysfunction in hematopoietic stem and progenitor cells. In contrast, MITOL deletion induced prolonged ER stress in HSCs, which triggered cellular apoptosis regulated by IRE1α. In line, dampening of ER stress signaling by IRE1α inihibitor KIRA6 partially rescued apoptosis of long-term-reconstituting HSC. In summary, our observations indicate that MITOL is a principal regulator of hematopoietic homeostasis and protects blood stem cells from cell death through its function in ER stress signaling.
    Mesh-Begriff(e) Mice ; Animals ; Endoribonucleases ; Protein Serine-Threonine Kinases/genetics ; Apoptosis ; Ubiquitin-Protein Ligases/genetics ; Ubiquitin-Protein Ligases/metabolism ; Hematopoietic Stem Cells/metabolism
    Chemische Substanzen Endoribonucleases (EC 3.1.-) ; Protein Serine-Threonine Kinases (EC 2.7.11.1) ; Ubiquitin-Protein Ligases (EC 2.3.2.27)
    Sprache Englisch
    Erscheinungsdatum 2024-01-18
    Erscheinungsland England
    Dokumenttyp Journal Article
    ZDB-ID 586044-1
    ISSN 1460-2075 ; 0261-4189
    ISSN (online) 1460-2075
    ISSN 0261-4189
    DOI 10.1038/s44318-024-00029-0
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  3. Artikel ; Online: Ribosome induces transdifferentiation of A549 and H-111-TC cancer cell lines.

    Anam, Mohammad Badrul / Istiaq, Arif / Kariya, Ryusho / Kudo, Mikiko / Ishtiyaq Ahmad, Shah Adil / Ito, Naofumi / Okada, Seiji / Ohta, Kunimasa

    Biochemistry and biophysics reports

    2021  Band 26, Seite(n) 100946

    Abstract: Previously we reported that, lactic acid bacteria (LAB) can induce human dermal fibroblast (HDF) cells to form multipotent cell clusters which are able to transdifferentiate into three germ layer derived cell lineages. Later on, we confirmed that ... ...

    Abstract Previously we reported that, lactic acid bacteria (LAB) can induce human dermal fibroblast (HDF) cells to form multipotent cell clusters which are able to transdifferentiate into three germ layer derived cell lineages. Later on, we confirmed that ribosome is responsible for the LAB-induced transdifferentiation and ribosomes from diverse organisms can mimic the LAB effect on HDF cells. In our present study we have shown that, upon incorporation of ribosomes, non-small cell lung cancer cell line A549 and gastric tubular adenocarcinoma cell line H-111-TC are transformed into spheroid like morphology those can be transdifferentiated into adipocytes and osteoblast. Our qPCR analysis has revealed that, during the formation of ribosome induced cancer cell spheroids, the expression of the cancer cell associated markers and cell cycle/proliferation markers were altered at different time point. Through our investigation, here we report a novel and a non-invasive approach for cancer cell reprogramming by incorporating ribosomes.
    Sprache Englisch
    Erscheinungsdatum 2021-02-12
    Erscheinungsland Netherlands
    Dokumenttyp Journal Article
    ZDB-ID 2831046-9
    ISSN 2405-5808 ; 2405-5808
    ISSN (online) 2405-5808
    ISSN 2405-5808
    DOI 10.1016/j.bbrep.2021.100946
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  4. Artikel ; Online: Ribosome Incorporation Induces EMT-like Phenomenon with Cell Cycle Arrest in Human Breast Cancer Cell.

    Kudo, Mikiko / Anam, Mohammad Badrul / Istiaq, Arif / Ahmad, Shah Adil Ishtiyaq / Ito, Naofumi / Ohta, Kunimasa

    Cells, tissues, organs

    2021  Band 211, Heft 2, Seite(n) 212–221

    Abstract: Although ribosomes are generally known to be a translational machinery, some ribosomal proteins also have accessory functions involving early development and differentiation. Previously, we reported that ribosome incorporation into human dermal ... ...

    Abstract Although ribosomes are generally known to be a translational machinery, some ribosomal proteins also have accessory functions involving early development and differentiation. Previously, we reported that ribosome incorporation into human dermal fibroblasts generated embryoid body-like cell clusters, altered cellular fate, and differentiated into cells of all 3 germ layers. However, the molecular phenomena induced by ribosome incorporation in the cell remained unknown. Here, we demonstrate that ribosome incorporation into human breast cancer cell MCF7 leads to ribosome-induced cell clusters (RICs) formation accompanying with epithelial-mesenchymal transition (EMT)-like gene expression. Following ribosome incorporation, MCF7 cells cease proliferation, which is caused by inhibition of cell cycle transition from G0 to G1 phase. Further, MCF7 RICs show induced expression of EMT markers, TGF-β1 and Snail along with autophagy markers and tumor suppressor gene p53. These findings indicate that the incorporation of ribosome into cancer cells induces an EMT-like phenomenon and changes the cancer cell characteristics.
    Mesh-Begriff(e) Breast Neoplasms/genetics ; Breast Neoplasms/metabolism ; Cell Cycle Checkpoints ; Cell Differentiation ; Cell Line, Tumor ; Cell Movement ; Epithelial-Mesenchymal Transition/genetics ; Female ; Humans ; Ribosomes/metabolism ; Transforming Growth Factor beta1/metabolism ; Transforming Growth Factor beta1/pharmacology
    Chemische Substanzen Transforming Growth Factor beta1
    Sprache Englisch
    Erscheinungsdatum 2021-02-26
    Erscheinungsland Switzerland
    Dokumenttyp Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 1468141-9
    ISSN 1422-6421 ; 1422-6405
    ISSN (online) 1422-6421
    ISSN 1422-6405
    DOI 10.1159/000513908
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  5. Artikel ; Online: Ribosome induces transdifferentiation of A549 and H-111-TC cancer cell lines

    Mohammad Badrul Anam / Arif Istiaq / Ryusho Kariya / Mikiko Kudo / Shah Adil Ishtiyaq Ahmad / Naofumi Ito / Seiji Okada / Kunimasa Ohta

    Biochemistry and Biophysics Reports, Vol 26, Iss , Pp 100946- (2021)

    2021  

    Abstract: Previously we reported that, lactic acid bacteria (LAB) can induce human dermal fibroblast (HDF) cells to form multipotent cell clusters which are able to transdifferentiate into three germ layer derived cell lineages. Later on, we confirmed that ... ...

    Abstract Previously we reported that, lactic acid bacteria (LAB) can induce human dermal fibroblast (HDF) cells to form multipotent cell clusters which are able to transdifferentiate into three germ layer derived cell lineages. Later on, we confirmed that ribosome is responsible for the LAB-induced transdifferentiation and ribosomes from diverse organisms can mimic the LAB effect on HDF cells. In our present study we have shown that, upon incorporation of ribosomes, non-small cell lung cancer cell line A549 and gastric tubular adenocarcinoma cell line H-111-TC are transformed into spheroid like morphology those can be transdifferentiated into adipocytes and osteoblast. Our qPCR analysis has revealed that, during the formation of ribosome induced cancer cell spheroids, the expression of the cancer cell associated markers and cell cycle/proliferation markers were altered at different time point. Through our investigation, here we report a novel and a non-invasive approach for cancer cell reprogramming by incorporating ribosomes.
    Schlagwörter Cancer ; Ribosome ; Reprogramming ; Transdifferentiation ; Cancer cell spheroids ; Biology (General) ; QH301-705.5 ; Biochemistry ; QD415-436
    Sprache Englisch
    Erscheinungsdatum 2021-07-01T00:00:00Z
    Verlag Elsevier
    Dokumenttyp Artikel ; Online
    Datenquelle BASE - Bielefeld Academic Search Engine (Lebenswissenschaftliche Auswahl)

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  6. Artikel: Involvement of Tsukushi in diverse developmental processes.

    Ahmad, Shah Adil Ishtiyaq / Anam, Mohammad Badrul / Ito, Naofumi / Ohta, Kunimasa

    Journal of cell communication and signaling

    2018  Band 12, Heft 1, Seite(n) 205–210

    Abstract: Tsukushi (TSK) is a small signaling molecule which takes part in different developmental processes of multiple vertebrate organisms. The diverse activity of TSK depends on its ability to bind various intermediate molecules from different major signaling ... ...

    Abstract Tsukushi (TSK) is a small signaling molecule which takes part in different developmental processes of multiple vertebrate organisms. The diverse activity of TSK depends on its ability to bind various intermediate molecules from different major signaling pathways. Interactions of TSK with BMP, FGF, TGF-β and Wnt pathways have already been confirmed. In this review, we will introduce the latest information regarding the involvement of TSK in developmental events. We suggest a fine tuning role for TSK in multiple signaling cascades. Also, we recommend further studies on the developmental role of TSK to fully reveal its potential.
    Sprache Englisch
    Erscheinungsdatum 2018-01-19
    Erscheinungsland Netherlands
    Dokumenttyp Journal Article
    ZDB-ID 2299380-0
    ISSN 1873-961X ; 1873-9601
    ISSN (online) 1873-961X
    ISSN 1873-9601
    DOI 10.1007/s12079-018-0452-8
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  7. Artikel ; Online: Tsukushi is essential for proper maintenance and terminal differentiation of mouse hippocampal neural stem cells.

    Ahmad, Shah Adil Ishtiyaq / Anam, Mohammad Badrul / Istiaq, Arif / Ito, Naofumi / Ohta, Kunimasa

    Development, growth & differentiation

    2020  Band 62, Heft 2, Seite(n) 108–117

    Abstract: Secreted proteoglycan molecule Tsukushi (TSK) regulates various developmental processes, such as early body patterning and neural plate formation by interacting with major signaling pathways like Wnt, BMP, Notch etc. In central nervous system, TSK ... ...

    Abstract Secreted proteoglycan molecule Tsukushi (TSK) regulates various developmental processes, such as early body patterning and neural plate formation by interacting with major signaling pathways like Wnt, BMP, Notch etc. In central nervous system, TSK inhibits Wnt signaling to control chick retinal development. It also plays important roles for axon guidance and anterior commissure formation in mouse brain. In the present study, we investigated the role of TSK for the development and proper functioning of mouse hippocampus. We found that TSK expression is prominent at hippocampal regions of early postnatal mouse until postnatal day 15 and gradually declines at later stages. Hippocampal dimensions are affected in TSK knockout mice (TSK-KO) as shown by reduced size of hippocampus and dentate gyrus (DG). Interestingly, neural stem cell (NSC) density at the neural niche of DG was higher in TSK-KO compared with wild-type. The ratio of proliferating NSCs as well as the rate of overall cell proliferation was also higher in TSK-KO hippocampus. Our in vitro study also suggests an increased number of neural stem/progenitor cells residing in TSK-KO hippocampus. Finally, we found that the terminal differentiation of NSCs in TSK-KO was disturbed as the differentiation to neuronal cell lineage was increased while the percentages of astrocytes and oligodendrocytes were decreased. Overall, our study establishes the involvement of TSK in hippocampal development, NSC maintenance and terminal differentiation at perinatal stages.
    Mesh-Begriff(e) Animals ; Cell Differentiation/genetics ; Cell Differentiation/physiology ; Cell Proliferation/genetics ; Cell Proliferation/physiology ; Gene Expression Regulation, Developmental ; Hippocampus/cytology ; Hippocampus/metabolism ; Immunochemistry ; Mice ; Mice, Knockout ; Neural Stem Cells/cytology ; Neural Stem Cells/metabolism ; Neurogenesis/genetics ; Neurogenesis/physiology ; Proteoglycans/genetics ; Proteoglycans/metabolism
    Chemische Substanzen Proteoglycans ; tsukushi protein, mouse
    Sprache Englisch
    Erscheinungsdatum 2020-01-26
    Erscheinungsland Japan
    Dokumenttyp Journal Article
    ZDB-ID 280433-5
    ISSN 1440-169X ; 0012-1592
    ISSN (online) 1440-169X
    ISSN 0012-1592
    DOI 10.1111/dgd.12649
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  8. Artikel ; Online: ATP citrate lyase controls hematopoietic stem cell fate and supports bone marrow regeneration.

    Umemoto, Terumasa / Johansson, Alban / Ahmad, Shah Adil Ishtiyaq / Hashimoto, Michihiro / Kubota, Sho / Kikuchi, Kenta / Odaka, Haruki / Era, Takumi / Kurotaki, Daisuke / Sashida, Goro / Suda, Toshio

    The EMBO journal

    2022  Band 41, Heft 8, Seite(n) e109463

    Abstract: In order to support bone marrow regeneration after myeloablation, hematopoietic stem cells (HSCs) actively divide to provide both stem and progenitor cells. However, the mechanisms regulating HSC function and cell fate choice during hematopoietic ... ...

    Abstract In order to support bone marrow regeneration after myeloablation, hematopoietic stem cells (HSCs) actively divide to provide both stem and progenitor cells. However, the mechanisms regulating HSC function and cell fate choice during hematopoietic recovery remain unclear. We herein provide novel insights into HSC regulation during regeneration by focusing on mitochondrial metabolism and ATP citrate lyase (ACLY). After 5-fluorouracil-induced myeloablation, HSCs highly expressing endothelial protein C receptor (EPCR
    Mesh-Begriff(e) ATP Citrate (pro-S)-Lyase/genetics ; ATP Citrate (pro-S)-Lyase/metabolism ; Bone Marrow ; Endothelial Protein C Receptor/metabolism ; Hematopoietic Stem Cells/physiology ; Histones/metabolism
    Chemische Substanzen Endothelial Protein C Receptor ; Histones ; ATP Citrate (pro-S)-Lyase (EC 2.3.3.8)
    Sprache Englisch
    Erscheinungsdatum 2022-03-01
    Erscheinungsland England
    Dokumenttyp Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 586044-1
    ISSN 1460-2075 ; 0261-4189
    ISSN (online) 1460-2075
    ISSN 0261-4189
    DOI 10.15252/embj.2021109463
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  9. Artikel ; Online: Transdifferentiation of human somatic cells by ribosome.

    Ito, Naofumi / Anam, Mohammad Badrul / Ahmad, Shah Adil Ishtiyaq / Ohta, Kunimasa

    Development, growth & differentiation

    2018  Band 60, Heft 5, Seite(n) 241–247

    Abstract: Ribosomes are intracellular organelles ubiquitous in all organisms, which translate information from mRNAs to synthesize proteins. They are complex macromolecules composed of dozens of proteins and ribosomal RNAs. Other than translation, some ribosomal ... ...

    Abstract Ribosomes are intracellular organelles ubiquitous in all organisms, which translate information from mRNAs to synthesize proteins. They are complex macromolecules composed of dozens of proteins and ribosomal RNAs. Other than translation, some ribosomal proteins also have side-jobs called "Moonlighting" function. The majority of these moonlighting functions influence cancer progression, early development and differentiation. Recently, we discovered that ribosome is involved in the regulation of cellular transdifferentiation of human dermal fibroblasts (HDFs). In vitro incorporation of ribosomes into HDFs arrests cell proliferation and induces the formation of cell clusters, that differentiate into three germ layer derived cells upon induction by differentiation mediums. The discovery of ribosome induced transdifferentiation, that is not based on genetic modification, find new possibilities for the treatment of cancer and congenital diseases, as well as to understand early development and cellular lineage differentiation.
    Mesh-Begriff(e) Animals ; Cell Proliferation/genetics ; Cell Proliferation/physiology ; Cell Transdifferentiation/genetics ; Cell Transdifferentiation/physiology ; Germ Layers/cytology ; Germ Layers/metabolism ; Humans ; Ribosomes/metabolism
    Sprache Englisch
    Erscheinungsdatum 2018-05-29
    Erscheinungsland Japan
    Dokumenttyp Journal Article ; Review
    ZDB-ID 280433-5
    ISSN 1440-169X ; 0012-1592
    ISSN (online) 1440-169X
    ISSN 0012-1592
    DOI 10.1111/dgd.12538
    Datenquelle MEDical Literature Analysis and Retrieval System OnLINE

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  10. Artikel ; Online: An In Silico Approach for Characterization of an Aminoglycoside Antibiotic-Resistant Methyltransferase Protein from Pyrococcus furiosus (DSM 3638)

    Arafat Rahman Oany / Tahmina Pervin Jyoti / Shah Adil Ishtiyaq Ahmad

    Bioinformatics and Biology Insights, Vol 2014, Iss 8, Pp 65-

    2014  Band 72

    Schlagwörter Biology (General) ; QH301-705.5
    Sprache Englisch
    Erscheinungsdatum 2014-03-01T00:00:00Z
    Verlag SAGE Publishing
    Dokumenttyp Artikel ; Online
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