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  1. Article ; Online: Water metabolism and vasopressin.

    Ishikawa, San-E

    Endocrine journal

    2023  Volume 70, Issue 4, Page(s) 341–342

    MeSH term(s) Water/metabolism ; Vasopressins/metabolism ; Arginine Vasopressin
    Chemical Substances Water (059QF0KO0R) ; Vasopressins (11000-17-2) ; Arginine Vasopressin (113-79-1)
    Language English
    Publishing date 2023-04-29
    Publishing country Japan
    Document type Journal Article
    ZDB-ID 1151918-6
    ISSN 1348-4540 ; 0918-8959
    ISSN (online) 1348-4540
    ISSN 0918-8959
    DOI 10.1507/endocrj.RMK70-4
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  2. Article: Hyponatremia Associated with Congestive Heart Failure: Involvement of Vasopressin and Efficacy of Vasopressin Receptor Antagonists.

    Ishikawa, San-E / Funayama, Hiroshi

    Journal of clinical medicine

    2023  Volume 12, Issue 4

    Abstract: Hyponatremia is frequently found in patients with congestive heart failure. A reduction in effective circulatory blood volume in a volume-expanded patient with decreased cardiac output is linked to a baroreceptor-mediated non-osmotic release of arginine ... ...

    Abstract Hyponatremia is frequently found in patients with congestive heart failure. A reduction in effective circulatory blood volume in a volume-expanded patient with decreased cardiac output is linked to a baroreceptor-mediated non-osmotic release of arginine vasopressin (AVP). The increased production of AVP and salt and water retention in the proximal and distal tubules of the kidney by humoral, hemodynamic, and neural mechanisms increase circulatory blood volume and contribute to hyponatremia. Recent studies have indicated that hyponatremia predicts the short-term and long-term prognosis of heart failure by increasing cardiac death and rehospitalization. In addition, the early development of hyponatremia in acute myocardial infarction also predicts the long-term prognosis of worsening heart failure. AVP V2 receptor antagonism may relieve water retention, but it is unknown whether the V2 receptor inhibitor, tolvaptan, improves the long-term prognosis of congestive heart failure. The newly identified natriuretic factor in renal salt wasting has the potential of improving clinical outcomes when combined with a distal diuretic.
    Language English
    Publishing date 2023-02-13
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 2662592-1
    ISSN 2077-0383
    ISSN 2077-0383
    DOI 10.3390/jcm12041482
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  3. Article: Transit analysis of thyroid antibodies in graves' disease following painless thyroiditis: a case report.

    Suto, Daisuke / Ebihara, Ken / Ishikawa, San-E

    Annals of medicine and surgery (2012)

    2023  Volume 85, Issue 5, Page(s) 2119–2122

    Abstract: Graves' disease and painless thyroiditis induce hyperthyroidism and are manifestations of an immunological disorder; however, their clinical entities differ. This case report illustrates a possible interrelation between the pathogenesis of these two ... ...

    Abstract Graves' disease and painless thyroiditis induce hyperthyroidism and are manifestations of an immunological disorder; however, their clinical entities differ. This case report illustrates a possible interrelation between the pathogenesis of these two disorders. A 34-year-old woman presented with palpitations, fatigue, and shortness of breath and was initially diagnosed with painless thyroiditis, which spontaneously normalized within 2 months. Within the euthyroid state, there were atypical alterations in thyroid autoantibodies, namely, activation of the thyroid stimulating hormone receptor antibody and inactivation of thyroid peroxidase and thyroglobulin antibodies. Ten months later, her hyperthyroidism recurred, with this second incidence deemed to be related to Graves' disease. Our patient had two types of painless thyroiditis with no successive hyperthyroidism, followed by Graves' disease, with her clinical manifestation transitioning from painless thyroiditis to Graves' disease over a 20-month period. Future studies are needed to elucidate the mechanisms and relationship between painless thyroiditis and Graves' disease.
    Language English
    Publishing date 2023-04-11
    Publishing country England
    Document type Journal Article
    ZDB-ID 2745440-X
    ISSN 2049-0801
    ISSN 2049-0801
    DOI 10.1097/MS9.0000000000000543
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  4. Article: Is Exaggerated Release of Arginine Vasopressin an Endocrine Disorder? Pathophysiology and Treatment.

    Ishikawa, San-E

    Journal of clinical medicine

    2017  Volume 6, Issue 11

    Abstract: Exaggerated release of arginine vasopressin (AVP) is profoundly involved in impaired water excretion and related hyponatremia. Such disorders underlie syndromes of inappropriate secretion of antidiuretic hormone (SIADH) and edematous diseases, such as ... ...

    Abstract Exaggerated release of arginine vasopressin (AVP) is profoundly involved in impaired water excretion and related hyponatremia. Such disorders underlie syndromes of inappropriate secretion of antidiuretic hormone (SIADH) and edematous diseases, such as congestive heart failure and decompensated liver cirrhosis. All the causes are fundamentally from non-endocrine diseases. AVP-induced water retention could produce hyponatremia, and further accelerate poor long-term outcome of edematous diseases. Administration of AVP V₂ receptor antagonists verifies how much AVP is involved in the pathogenesis of the impaired water excretion. The present paper demonstrated that exaggerated release of AVP plays a crucial role as an accessory endocrine disorder in pathological states of water retention and dilutional hyponatremia in non-endocrine disorders.
    Language English
    Publishing date 2017-10-31
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 2662592-1
    ISSN 2077-0383
    ISSN 2077-0383
    DOI 10.3390/jcm6110102
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  5. Article ; Online: Is Exaggerated Release of Arginine Vasopressin an Endocrine Disorder? Pathophysiology and Treatment

    San-e Ishikawa

    Journal of Clinical Medicine, Vol 6, Iss 11, p

    2017  Volume 102

    Abstract: Exaggerated release of arginine vasopressin (AVP) is profoundly involved in impaired water excretion and related hyponatremia. Such disorders underlie syndromes of inappropriate secretion of antidiuretic hormone (SIADH) and edematous diseases, such as ... ...

    Abstract Exaggerated release of arginine vasopressin (AVP) is profoundly involved in impaired water excretion and related hyponatremia. Such disorders underlie syndromes of inappropriate secretion of antidiuretic hormone (SIADH) and edematous diseases, such as congestive heart failure and decompensated liver cirrhosis. All the causes are fundamentally from non-endocrine diseases. AVP-induced water retention could produce hyponatremia, and further accelerate poor long-term outcome of edematous diseases. Administration of AVP V2 receptor antagonists verifies how much AVP is involved in the pathogenesis of the impaired water excretion. The present paper demonstrated that exaggerated release of AVP plays a crucial role as an accessory endocrine disorder in pathological states of water retention and dilutional hyponatremia in non-endocrine disorders.
    Keywords impaired water excretion ; hyponatremia ; aquaporin 2 ; arterial underfilling ; heart failure ; SIADH ; vasopressin (AVP) V2 receptor antagonist ; Medicine ; R
    Language English
    Publishing date 2017-10-01T00:00:00Z
    Publisher MDPI AG
    Document type Article ; Online
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  6. Article: Hyponatremia Associated with Heart Failure: Pathological Role of Vasopressin-Dependent Impaired Water Excretion.

    Ishikawa, San-E

    Journal of clinical medicine

    2015  Volume 4, Issue 5, Page(s) 933–947

    Abstract: An exaggerated increase in circulatory blood volume is linked to congestive heart failure. Despite this increase, reduction of the "effective circulatory blood volume" in congestive heart failure is associated with decreased cardiac output, and can ... ...

    Abstract An exaggerated increase in circulatory blood volume is linked to congestive heart failure. Despite this increase, reduction of the "effective circulatory blood volume" in congestive heart failure is associated with decreased cardiac output, and can weaken the sensitivity of baroreceptors. Thereafter, tonic inhibition of the baroreceptor-mediated afferent pathway of vagal nerves is removed, providing an increase in non-osmotic release of arginine vasopressin (AVP). In the renal collecting duct, the aquaporin-2 (AQP2) water channel is regulated by sustained elevation of AVP release, and this leads to augmented hydroosmotic action of AVP, that results in exaggerated water retention and dilutional hyponatremia. Hyponatremia is also a predictor for worsening heart failure in patients with known/new onset heart failure. Therefore, such a dilutional hyponatremia associated with organ damage is predictive of the short- and long-term outcome of heart failure.
    Language English
    Publishing date 2015-05-08
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 2662592-1
    ISSN 2077-0383
    ISSN 2077-0383
    DOI 10.3390/jcm4050933
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  7. Article ; Online: Arginine vasopressin in heart failure.

    Ishikawa, San-e

    Circulation journal : official journal of the Japanese Circulation Society

    2014  Volume 78, Issue 9, Page(s) 2159–2161

    MeSH term(s) Cardiac Output, Low ; Heart Failure ; Humans ; Hyponatremia ; Vasopressins/blood ; Water-Electrolyte Balance
    Chemical Substances Vasopressins (11000-17-2)
    Language English
    Publishing date 2014-07-29
    Publishing country Japan
    Document type Editorial ; Comment
    ZDB-ID 2068090-9
    ISSN 1347-4820 ; 1346-9843
    ISSN (online) 1347-4820
    ISSN 1346-9843
    DOI 10.1253/circj.cj-14-0752
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    Ui IV Zs.74: Show issues Location:
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  8. Article ; Online: Hyponatremia Associated with Heart Failure

    San-e Ishikawa

    Journal of Clinical Medicine, Vol 4, Iss 5, Pp 933-

    Pathological Role of Vasopressin-Dependent Impaired Water Excretion

    2015  Volume 947

    Abstract: An exaggerated increase in circulatory blood volume is linked to congestive heart failure. Despite this increase, reduction of the “effective circulatory blood volume” in congestive heart failure is associated with decreased cardiac output, and can ... ...

    Abstract An exaggerated increase in circulatory blood volume is linked to congestive heart failure. Despite this increase, reduction of the “effective circulatory blood volume” in congestive heart failure is associated with decreased cardiac output, and can weaken the sensitivity of baroreceptors. Thereafter, tonic inhibition of the baroreceptor-mediated afferent pathway of vagal nerves is removed, providing an increase in non-osmotic release of arginine vasopressin (AVP). In the renal collecting duct, the aquaporin-2 (AQP2) water channel is regulated by sustained elevation of AVP release, and this leads to augmented hydroosmotic action of AVP, that results in exaggerated water retention and dilutional hyponatremia. Hyponatremia is also a predictor for worsening heart failure in patients with known/new onset heart failure. Therefore, such a dilutional hyponatremia associated with organ damage is predictive of the short- and long-term outcome of heart failure.
    Keywords hyponatremia ; arginine vasopressin (AVP) ; aquaporin2 (AQP2) ; non-osmotic release ; heart failure ; cardiac output ; circulatory blood volume ; impaired water excretion ; AVP V2 receptor antagonist ; Medicine ; R
    Subject code 616 ; 610
    Language English
    Publishing date 2015-05-01T00:00:00Z
    Publisher MDPI AG
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  9. Article: [The cutting-edge of medicine; hyponatremia and water metabolism disorders in elderly].

    Ishikawa, San-e

    Nihon Naika Gakkai zasshi. The Journal of the Japanese Society of Internal Medicine

    2013  Volume 102, Issue 7, Page(s) 1807–1813

    MeSH term(s) Aged ; Blood Volume/physiology ; Humans ; Hyponatremia/diagnosis ; Hyponatremia/metabolism ; Hyponatremia/physiopathology ; Inappropriate ADH Syndrome/physiopathology ; Sodium/metabolism ; Water/metabolism
    Chemical Substances Water (059QF0KO0R) ; Sodium (9NEZ333N27)
    Language Japanese
    Publishing date 2013-07-07
    Publishing country Japan
    Document type Journal Article ; Review
    ZDB-ID 952816-7
    ISSN 1883-2083 ; 0021-5384
    ISSN (online) 1883-2083
    ISSN 0021-5384
    DOI 10.2169/naika.102.1807
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    Zs.B 594: Show issues Location:
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  10. Article: [SIADH].

    Ishikawa, San-e

    Nihon rinsho. Japanese journal of clinical medicine

    2012  Volume 70 Suppl 6, Page(s) 715–719

    MeSH term(s) Humans ; Inappropriate ADH Syndrome/chemically induced ; Inappropriate ADH Syndrome/diagnosis ; Inappropriate ADH Syndrome/etiology
    Language Japanese
    Publishing date 2012-08
    Publishing country Japan
    Document type Journal Article
    ZDB-ID 390903-7
    ISSN 0047-1852
    ISSN 0047-1852
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