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  1. Book: Principles of bone biology / Volume 1

    Bilezikian, John P.

    2020  

    Author's details ed. by John P. Bilezikian
    Collection Principles of bone biology
    Language English
    Size xxi, 1060 Seiten, Illustrationen
    Edition Fourth edition
    Publisher Academic Press
    Publishing place San Diego u.a.
    Publishing country United States
    Document type Book
    HBZ-ID HT020191788
    ISBN 978-0-12-819932-9 ; 0-12-819932-6
    Database Catalogue ZB MED Medicine, Health

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  2. Book: Principles of bone biology / Volume 2

    Bilezikian, John P.

    2020  

    Author's details ed. by John P. Bilezikian
    Collection Principles of bone biology
    Language English
    Size xxi Seiten, Seite 1061 - 1986, Illustrationen
    Edition Fourth edition
    Publisher Academic Press
    Publishing place San Diego u.a.
    Publishing country United States
    Document type Book
    HBZ-ID HT020191801
    ISBN 978-0-12-819933-6 ; 0-12-819933-4
    Database Catalogue ZB MED Medicine, Health

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  3. Book: Vitamin D in clinical medicine

    Giustina, Andrea / Bilezikian, John P.

    (Frontiers of hormone research ; Vol. 50)

    2018  

    Series title Frontiers of hormone research ; Vol. 50
    Collection
    Keywords Vitamin D DEficinecy / complications ; Vitamin D / metabolism ; Endocrine System Diseases / etiology ; Pneumology ; Endocrinology ; Oncology ; Nutrition ; Cardiovascular System ; Vitamin-D-Mangel ; Endokrinopathie ; Systemerkrankung ; Ätiologie
    Subject Krankheitsursache ; Krankheit ; Systemkrankheit ; Systemische Störung ; Endokrine Krankheit ; Endokrine Drüse ; Endokrine Regulationsstörung ; Hormonstörung ; Hormonelle Störung ; Vitamin D
    Subject code 610
    Language English
    Size VIII, 206 Seiten, 10 fig., 4 in color, 14 tab., 730 g
    Edition 1. Auflage
    Publisher Karger
    Publishing place Basel
    Publishing country Switzerland
    Document type Book
    HBZ-ID HT019699581
    ISBN 978-3-318-06338-7 ; 3-318-06338-X ; 9783318063394 ; 3318063398
    Database Catalogue ZB MED Medicine, Health

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  4. Book: The parathyroids

    Bilezikian, John P.

    basic and clinical concepts

    2015  

    Author's details ed. John P. Bilezikian
    Keywords Parathyroid Diseases / physiopathology ; Parathyroid Diseases / therapy ; Parathyroid Glands / physiology ; Parathyroid Hormones / physiology
    Language English
    Size XXV, 919 S. : Ill., graph. Darst.
    Edition 3. ed.
    Publisher Elsevier Acad. Press
    Publishing place Amsterdam u.a.
    Publishing country Netherlands
    Document type Book
    Accompanying material Hinweis auf zusätzliches Internetmaterial im Buch
    HBZ-ID HT018445621
    ISBN 978-0-12-397166-1 ; 0-12-397166-7
    Database Catalogue ZB MED Medicine, Health

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  5. Book ; Online ; E-Book: Primer on the metabolic bone diseases and disorders of mineral metabolism

    Bilezikian, John P. / Bouillon, Roger / Clemens, Thomas / Compston, Juliet

    2019  

    Author's details editor-in-chief John P. Bilezikian, MD, PhD (hon) ; senior associate editors Roger Bouillon, MD, PhD, FRCP, Thomas Clemens, PhD; Juliet Compston, OBE, MD, FRCP, FRCPE, FRCPath, FMedSci ; associate editors Douglas C. Bauer, MD [und weitere]
    Language English
    Size 1 Online-Ressource (xxvii, 1105 Seiten), Illustrationen, Diagramme
    Edition Ninth edition
    Publisher Wiley Blackwell
    Publishing place Hoboken, NJ
    Publishing country United States
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT019860956
    ISBN 978-1-119-26658-7 ; 9781119266563 ; 9781119266570 ; 1-119-26658-0 ; 1119266564 ; 1119266572
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  6. Article ; Online: PTH and adiposity.

    Tay, Donovan / Bilezikian, John P

    Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research

    2024  

    Abstract: Although PTH is best known for its role as a regulator of skeletal remodelling and calcium homeostasis, more recent evidence supports a role for it in energy metabolism and other non-classical targets. In this report, we summarize evidence for an effect ... ...

    Abstract Although PTH is best known for its role as a regulator of skeletal remodelling and calcium homeostasis, more recent evidence supports a role for it in energy metabolism and other non-classical targets. In this report, we summarize evidence for an effect of PTH on adipocytes. This review is based upon all peer-reviewed papers, published in the English language with PubMed as the primary search engine. Recent preclinical studies have documented an effect of PTH to stimulate lipolysis in both adipocytes and liver cells and to cause browning of adipocytes. PTH also reduces bone marrow adiposity and hepatic steatosis. Although clinical studies are limited, disease models of PTH excess and PTH deficiency lend support to these preclinical findings. This review supports the concept of PTH as a polyfunctional hormone that influences energy metabolism as well as bone metabolism.
    Language English
    Publishing date 2024-04-18
    Publishing country England
    Document type Journal Article
    ZDB-ID 632783-7
    ISSN 1523-4681 ; 0884-0431
    ISSN (online) 1523-4681
    ISSN 0884-0431
    DOI 10.1093/jbmr/zjae056
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  7. Book ; Conference proceedings: Bisphosphonates and osteonecrosis of the jaw

    Bilezikian, John P.

    manuscripts stemming from the Conference Entitled "Bisphosphonates & Osteonecrosis of the Jaw" held May 19, 2007 at the New York Academy of Sciences

    (Annals of the New York Academy of Sciences ; 1218)

    2011  

    Event/congress Conference Entiteled Bisphosphonates & Osteonecrosis of the Jaw (2007, NewYorkNY)
    Author's details iss. ed. John P. Bilezikian
    Series title Annals of the New York Academy of Sciences ; 1218
    Collection
    Language English
    Size 87 S. : Ill., graph. Darst.
    Publisher Wiley
    Publishing place Malden, MA u.a.
    Publishing country United States
    Document type Book ; Conference proceedings
    HBZ-ID HT016744591
    ISBN 1-57331-710-1 ; 978-1-57331-710-8
    Database Catalogue ZB MED Medicine, Health

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  8. Book: Principles of bone biology / 2

    Bilezikian, John P.

    2008  

    Author's details ed. by John P. Bilezikian
    Collection Principles of bone biology
    Language English
    Size LIV S., S. 955 - 1942, [4] Bl. : Ill., graph. Darst.
    Edition 3. ed.
    Publisher Academic Press
    Publishing place San Diego u.a.
    Publishing country United States
    Document type Book
    HBZ-ID HT015737188
    ISBN 978-0-12-373886-8 ; 0-12-373886-5
    Database Catalogue ZB MED Medicine, Health

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  9. Book: Principles of bone biology / 1

    Bilezikian, John P.

    2008  

    Author's details ed. by John P. Bilezikian
    Collection Principles of bone biology
    Language English
    Size LIV, 954 S., [7] Bl. : Ill., graph. Darst.
    Edition 3. ed.
    Publisher Academic Press
    Publishing place San Diego u.a.
    Publishing country United States
    Document type Book
    HBZ-ID HT015737117
    ISBN 978-0-12-373885-1 ; 0-12-373885-7
    Database Catalogue ZB MED Medicine, Health

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  10. Article ; Online: Hypoparathyroidism.

    Bilezikian, John P

    The Journal of clinical endocrinology and metabolism

    2020  Volume 105, Issue 6

    Abstract: Background: Hypoparathyroidism is a rare endocrine disorder characterized by hypocalcemia and low or undetectable levels of parathyroid hormone.: Methods: This review is an evidence-based summary of hypoparathyroidism in terms of relevant ... ...

    Abstract Background: Hypoparathyroidism is a rare endocrine disorder characterized by hypocalcemia and low or undetectable levels of parathyroid hormone.
    Methods: This review is an evidence-based summary of hypoparathyroidism in terms of relevant pathophysiological, clinical, and therapeutic concepts.
    Results: Many clinical manifestations of hypoparathyroidism are due to the lack of the physiological actions of parathyroid hormone on its 2 major target organs: the skeleton and the kidney. The skeleton is inactive, accruing bone without remodeling it. The kidneys lose the calcium-conserving actions of parathyroid hormone and, thus, excrete a greater fraction of calcium. Biochemical manifestations, besides hypocalcemia and low or undetectable levels of parathyroid hormone, include hyperphosphatemia and low levels of 1,25-dihydroxyvitamin D. Calcifications in the kidney, brain, and other soft tissues are common. Removal of, or damage to, the parathyroid glands at the time of anterior neck surgery is, by far, the most likely etiology. Autoimmune destruction of the parathyroid glands and other genetic causes represent most of the other etiologies. Conventional treatment with calcium and active vitamin D can maintain the serum calcium level but high doses may be required, adding to the risk of long-term soft tissue calcifications. The advent of replacement therapy with recombinant human PTH(1-84) represents a major step in the therapeutics of this disease.
    Conclusions: Advances in our knowledge of hypoparathyroidism have led to greater understanding of the disease itself and our approach to it.
    MeSH term(s) Humans ; Hypocalcemia/complications ; Hypoparathyroidism/etiology ; Hypoparathyroidism/metabolism ; Hypoparathyroidism/pathology ; Parathyroid Hormone/metabolism ; Prognosis
    Chemical Substances Parathyroid Hormone
    Language English
    Publishing date 2020-04-23
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 3029-6
    ISSN 1945-7197 ; 0021-972X
    ISSN (online) 1945-7197
    ISSN 0021-972X
    DOI 10.1210/clinem/dgaa113
    Database MEDical Literature Analysis and Retrieval System OnLINE

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