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Article ; Online: A Life Study in Academic Leadership: A Conversation Between Carolyn C. Meltzer and Hossein Jadvar.

Meltzer, Carolyn C / Jadvar, Hossein

Journal of nuclear medicine : official publication, Society of Nuclear Medicine

2023 Volume 64, Issue 8, Page(s) 1167–1170

MeSH term(s)	Leadership ; Communication
Language	English
Publishing date	2023-07-13
Publishing country	United States
Document type	Journal Article
ZDB-ID	80272-4
ISSN	1535-5667 ; 0097-9058 ; 0161-5505 ; 0022-3123
ISSN (online)	1535-5667
ISSN	0097-9058 ; 0161-5505 ; 0022-3123
DOI	10.2967/jnumed.123.266097
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Article ; Online: The clinical value of C-reactive protein and its association with tumour location in patients undergoing curative surgery for colorectal cancer - a ScotScan collaborative study.

Fuglestad, Anniken J / Meltzer, Sebastian / Ree, Anne Hansen / McMillan, Donald C / Park, James H / Kersten, Christian

Acta oncologica (Stockholm, Sweden)

2022 Volume 61, Issue 10, Page(s) 1248–1255

Abstract: ... negative prognostic factor for patients diagnosed with colorectal cancer (CRC). C-reactive protein (CRP) is ...

Abstract	Introduction: The presence of preoperative systemic inflammatory response (SIR) is an established negative prognostic factor for patients diagnosed with colorectal cancer (CRC). C-reactive protein (CRP) is known to be implicated in detrimental immune responses. The biological differences between right-sided and left-sided CRC are gaining increasing attention. Our aim was to analyse the prognostic value of CRP and explore the association between tumour location and SIR. Material and methods: A total of 2059 patients treated for stage I-III CRC, identified from the prospectively sampled ScotScan Collaborative dataset, were included. The clinical and prognostic value of five CRP levels (<10/11-30/31-60/61-100/>100 mg/l) were examined. Additionally, the relationship between SIR and tumour location was explored. Results: Increasing levels of CRP were associated with impaired overall and cancer-specific outcome. Presence of SIR was independently associated with right-sided tumour location ( Conclusions: This study confirms CRP as a routinely available, valid, and clinically relevant strong prognostic marker of SIR in CRC patients. Right-sided tumours were more often associated with SIR, but the prognostic impact was stronger in left-sided tumours.
MeSH term(s)	Humans ; C-Reactive Protein/analysis ; Colorectal Neoplasms/pathology ; Prognosis
Chemical Substances	C-Reactive Protein (9007-41-4)
Language	English
Publishing date	2022-09-06
Publishing country	England
Document type	Journal Article
ZDB-ID	896449-x
ISSN	1651-226X ; 0349-652X ; 0284-186X ; 1100-1704
ISSN (online)	1651-226X
ISSN	0349-652X ; 0284-186X ; 1100-1704
DOI	10.1080/0284186X.2022.2117572
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Article: 5-Aza-4'-thio-2'-deoxycytidine induces C>G transversions in a specific trinucleotide context and leads to acute lymphoid leukemia.

Aplan, Peter / Bertoli, Ryan / Chung, Yang Jo / Difilippantonio, Michael / Wokasch, Anthony / Marasco, Madison / Klimaszewski, Haley / Garber, Susannah / Zhu, Yuelin / Walker, Robert / Cao, Dengchao / Doroshow, James / Meltzer, Paul

Research square

2023

Abstract: ... lymphoid leukemia. Whole exome sequencing revealed thousands of acquired mutations, almost all of which were C > G ...

Abstract	DNA methyltransferase inhibitors (DNMTi), most commonly cytidine analogs, are compounds that are used clinically to decrease 5'-cytosine methylation, with the aim of re-expression of tumor suppressor genes. We used a murine pre-clinical model of myelodysplastic syndrome based on transplantation of cells expressing a NUP98::HOXD13 transgene to investigate 5-Aza-4'-thio-2'-deoxycytidine (Aza TdCyd or ATC), a thiol substituted DNMTi, as a potential therapy. We found that ATC treatment led to lymphoid leukemia in wild-type recipient cells; further study revealed that healthy mice treated with ATC also developed lymphoid leukemia. Whole exome sequencing revealed thousands of acquired mutations, almost all of which were C > G transversions in a previously unrecognized, specific 5'-NCG-3' context. These mutations involved dozens of genes well-known to be involved in human lymphoid leukemia, such as
Language	English
Publishing date	2023-12-19
Publishing country	United States
Document type	Preprint
DOI	10.21203/rs.3.rs-3186246/v1
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Article ; Online: Deposition of Centromeric Histone H3 Variant CENP-A/Cse4 into Chromatin Is Facilitated by Its C-Terminal Sumoylation.

Ohkuni, Kentaro / Suva, Evelyn / Au, Wei-Chun / Walker, Robert L / Levy-Myers, Reuben / Meltzer, Paul S / Baker, Richard E / Basrai, Munira A

Genetics

2020 Volume 214, Issue 4, Page(s) 839–854

Abstract: Centromeric localization of CENP-A (Cse4 ... ...

Abstract	Centromeric localization of CENP-A (Cse4 in
MeSH term(s)	Chromatin/metabolism ; Chromatin Assembly Factor-1/genetics ; Chromatin Assembly Factor-1/metabolism ; Chromosomal Proteins, Non-Histone/chemistry ; Chromosomal Proteins, Non-Histone/genetics ; Chromosomal Proteins, Non-Histone/metabolism ; DNA-Binding Proteins/chemistry ; DNA-Binding Proteins/genetics ; DNA-Binding Proteins/metabolism ; Peptide Elongation Factors/genetics ; Peptide Elongation Factors/metabolism ; Protein Domains ; Saccharomyces cerevisiae ; Saccharomyces cerevisiae Proteins/chemistry ; Saccharomyces cerevisiae Proteins/genetics ; Saccharomyces cerevisiae Proteins/metabolism ; Sumoylation ; Synthetic Lethal Mutations ; Ubiquitin-Protein Ligases/genetics ; Ubiquitin-Protein Ligases/metabolism
Chemical Substances	CSE4 protein, S cerevisiae ; Chromatin ; Chromatin Assembly Factor-1 ; Chromosomal Proteins, Non-Histone ; DNA-Binding Proteins ; Peptide Elongation Factors ; Saccharomyces cerevisiae Proteins ; Scm3 protein, S cerevisiae ; Psh1 protein, S cerevisiae (EC 2.3.2.27) ; Ubiquitin-Protein Ligases (EC 2.3.2.27)
Language	English
Publishing date	2020-02-28
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Intramural
ZDB-ID	2167-2
ISSN	1943-2631 ; 0016-6731
ISSN (online)	1943-2631
ISSN	0016-6731
DOI	10.1534/genetics.120.303090
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Article: Rapid (13)C Urea Breath Test to Identify Helicobacter pylori Infection in Emergency Department Patients with Upper Abdominal Pain.

Meltzer, Andrew C / Pierce, Rebecca / Cummings, Derek A T / Pines, Jesse M / May, Larissa / Smith, Meaghan A / Marcotte, Joseph / McCarthy, Melissa L

The western journal of emergency medicine

2013 Volume 14, Issue 3, Page(s) 278–282

Abstract: ... were tested for H. pylori using a rapid point of care (13)C Urea Breath Test (UBT) [Exalenz Bioscience ...

Abstract	Introduction: In emergency department (ED) patients with upper abdominal pain, management includes ruling out serious diseases and providing symptomatic relief. One of the major causes of upper abdominal pain is an ulcer caused by Helicobacter pylori (H. pylori), which can be treated and cured with antibiotics. We sought to estimate the prevalence of H. pylori infection in symptomatic patients using a convenience sample at a single urban academic ED and demonstrate the feasibility of ED-based testing. Methods: We prospectively enrolled patients with a chief complaint of pain or discomfort in the upper abdomen for 1 year from February 2011 until February 2012 at a single academic urban ED. Enrolled subjects were tested for H. pylori using a rapid point of care (13)C Urea Breath Test (UBT) [Exalenz Bioscience]. We compared patient characteristics between those who tested positive versus negative for the disease. Results: A total of 205 patients with upper abdominal pain were tested over 12 months, and 24% (95% confidence interval: 19% to 30%) tested positive for H. pylori. Black subjects were more likely to test positive than white subjects (28% v. 6%, P < 0.001). Other factors, such as age and sex, were not different between the 2 groups. Conclusion: In our ED, H. pylori infection was present in 1 in 4 patients with epigastric pain, and testing with a UBT was feasible. Further study is needed to determine the risk factors associated with infection, the prevalence of H. pylori in other EDs, the effect of the test on ED length of stay and the costeffectiveness of an ED-based test-and-treat strategy.
Language	English
Publishing date	2013-02-12
Publishing country	United States
Document type	Journal Article
ZDB-ID	2375700-0
ISSN	1936-9018 ; 1936-900X
ISSN (online)	1936-9018
ISSN	1936-900X
DOI	10.5811/westjem.2012.12.15173
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Article ; Online: Clinical impact of treatment timing for chronic hepatitis C infection: a decision model.

Pho, M T / Jensen, D M / Meltzer, D O / Kim, A Y / Linas, B P

Journal of viral hepatitis

2015 Volume 22, Issue 8, Page(s) 630–638

Abstract: Recent advances in the treatment of hepatitis C virus (HCV) infection have led to the availability ...

Abstract	Recent advances in the treatment of hepatitis C virus (HCV) infection have led to the availability of both highly efficacious interferon-containing and interferon-sparing regimens. However, the use of such therapies faces restrictions due to high costs. For patients who are medically eligible to receive interferon, the choice between the two will likely be impacted by preferences surrounding interferon, severity of disease, coverage policies and out-of-pocket costs. We developed a decision model to quantify the trade-offs between immediate, interferon-containing therapy and delayed, interferon-free therapy for patients with chronic, genotype 1 HCV infection. We projected the quality-adjusted life expectancy stratified by the presence or absence of cirrhosis for four strategies: (i) no treatment; (ii) immediate, one-time treatment with an interferon-containing regimen; (iii) immediate treatment as above with the opportunity for retreatment in patients who fail to achieve sustained virologic response with interferon-free therapy in 1 year; and (iv) delayed therapy with interferon-free therapy in 1 year. When compared to one-time immediate treatment with the interferon-containing regimen, delayed treatment with the interferon-free regimen in 1 year resulted in longer life expectancy, with a 0.2 quality-adjusted life year (QALY) increase in noncirrhotic patients, and a 1.1 QALY increase in patients with cirrhosis. This superiority in health benefits was lost when wait time for interferon-free therapy was greater than 3-3.2 years. In this modelling analysis, interferon-free therapy resulted in superior health benefits compared to immediate therapy with interferon until wait time exceeded 3-3.2 years. Such data can inform decision-making regarding treatment initiation for HCV as healthcare financing evolves.
MeSH term(s)	Adult ; Aged ; Aged, 80 and over ; Antiviral Agents/administration & dosage ; Decision Support Systems, Clinical ; Drug Therapy/methods ; Female ; Hepatitis C, Chronic/drug therapy ; Humans ; Life Expectancy ; Male ; Middle Aged ; Quality of Life ; Time Factors ; Treatment Outcome
Chemical Substances	Antiviral Agents
Language	English
Publishing date	2015-06-17
Publishing country	England
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, U.S. Gov't, P.H.S.
ZDB-ID	1212497-7
ISSN	1365-2893 ; 1352-0504
ISSN (online)	1365-2893
ISSN	1352-0504
DOI	10.1111/jvh.12412
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Zs.A 4119: Show issues

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Article ; Online: Overlapping ETS and CRE Motifs ((G/C)CGGAAGTGACGTCA) preferentially bound by GABPα and CREB proteins.

Chatterjee, Raghunath / Zhao, Jianfei / He, Ximiao / Shlyakhtenko, Andrey / Mann, Ishminder / Waterfall, Joshua J / Meltzer, Paul / Sathyanarayana, B K / FitzGerald, Peter C / Vinson, Charles

G3 (Bethesda, Md.)

2012 Volume 2, Issue 10, Page(s) 1243–1256

Abstract: ... C/G)CCGGAAGCGGAA) and the ETS⇔CRE motif ((C/G)CGGAAGTGACGTCAC). The nucleotides in bold are part ...

Abstract	Previously, we identified 8-bps long DNA sequences (8-mers) that localize in human proximal promoters and grouped them into known transcription factor binding sites (TFBS). We now examine split 8-mers consisting of two 4-mers separated by 1-bp to 30-bps (X(4)-N(1-30)-X(4)) to identify pairs of TFBS that localize in proximal promoters at a precise distance. These include two overlapping TFBS: the ETS⇔ETS motif ((C/G)CCGGAAGCGGAA) and the ETS⇔CRE motif ((C/G)CGGAAGTGACGTCAC). The nucleotides in bold are part of both TFBS. Molecular modeling shows that the ETS⇔CRE motif can be bound simultaneously by both the ETS and the B-ZIP domains without protein-protein clashes. The electrophoretic mobility shift assay (EMSA) shows that the ETS protein GABPα and the B-ZIP protein CREB preferentially bind to the ETS⇔CRE motif only when the two TFBS overlap precisely. In contrast, the ETS domain of ETV5 and CREB interfere with each other for binding the ETS⇔CRE. The 11-mer (CGGAAGTGACG), the conserved part of the ETS⇔CRE motif, occurs 226 times in the human genome and 83% are in known regulatory regions. In vivo GABPα and CREB ChIP-seq peaks identified the ETS⇔CRE as the most enriched motif occurring in promoters of genes involved in mRNA processing, cellular catabolic processes, and stress response, suggesting that a specific class of genes is regulated by this composite motif.
MeSH term(s)	Animals ; Base Sequence ; Binding Sites ; Conserved Sequence ; Cyclic AMP Response Element-Binding Protein/chemistry ; Cyclic AMP Response Element-Binding Protein/metabolism ; DNA Methylation ; GA-Binding Protein Transcription Factor/chemistry ; GA-Binding Protein Transcription Factor/metabolism ; Humans ; Mice ; Molecular Docking Simulation ; Nucleic Acid Conformation ; Nucleotide Motifs ; Promoter Regions, Genetic ; Protein Binding ; Protein Conformation ; Proto-Oncogene Proteins c-ets/chemistry ; Proto-Oncogene Proteins c-ets/genetics
Chemical Substances	Cyclic AMP Response Element-Binding Protein ; GA-Binding Protein Transcription Factor ; GABPA protein, human ; Proto-Oncogene Proteins c-ets
Language	English
Publishing date	2012-10-01
Publishing country	England
Document type	Journal Article
ZDB-ID	2629978-1
ISSN	2160-1836 ; 2160-1836
ISSN (online)	2160-1836
ISSN	2160-1836
DOI	10.1534/g3.112.004002
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Article: No interruption of lactation is needed after (11)C-WAY 100635 or (11)C-raclopride PET.

Moses-Kolko, Eydie L / Meltzer, Carolyn Cidis / Helsel, Joseph C / Sheetz, Michael / Mathis, Chestera / Ruszkiewicz, James / Bogen, Debra / Confer, Andrea L / Wisner, Katherine L

Journal of nuclear medicine : official publication, Society of Nuclear Medicine

2005 Volume 46, Issue 10, Page(s) 1765

MeSH term(s)	Adult ; Female ; Humans ; Lactation/metabolism ; Milk, Human/chemistry ; Piperazines/analysis ; Piperazines/pharmacokinetics ; Positron-Emission Tomography/methods ; Pyridines/analysis ; Pyridines/pharmacokinetics ; Raclopride/analysis ; Raclopride/pharmacokinetics ; Radiation Dosage ; Radiometry/methods ; Radiopharmaceuticals/analysis ; Radiopharmaceuticals/pharmacokinetics ; Risk Assessment/methods ; Risk Factors
Chemical Substances	Piperazines ; Pyridines ; Radiopharmaceuticals ; Raclopride (430K3SOZ7G) ; N-(2-(4-(2-methoxyphenyl)-1-piperazinyl)ethyl)-N-(2-pyridinyl)cyclohexanecarboxamide (71IH826FEG)
Language	English
Publishing date	2005-10
Publishing country	United States
Document type	Clinical Trial ; Letter
ZDB-ID	80272-4
ISSN	1535-5667 ; 0161-5505 ; 0097-9058 ; 0022-3123
ISSN (online)	1535-5667
ISSN	0161-5505 ; 0097-9058 ; 0022-3123
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Zs.A 114: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (1.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 328: Show issues

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Article ; Online: Genome scan of clot lysis time and its association with thrombosis in a protein C-deficient kindred.

Meltzer, M E / Hasstedt, S J / Vossen, C Y / Callas, P W / DE Groot, Ph G / Rosendaal, F R / Lisman, T / Bovill, E G

Journal of thrombosis and haemostasis : JTH

2010 Volume 9, Issue 7, Page(s) 1383–1390

Abstract: ... to a type I protein C deficiency. Furthermore, we tested for quantitative trait loci (QTLs) for CLT, using ... variance component linkage analysis.: Results: Protein C-deficient family members had shorter CLTs ... the risk of venous thrombosis 2.4-fold in non-deficient family members. Protein C deficiency ...

Abstract	Background: Previously, we found increased clot-lysis time (CLT), as measured with a plasma-based assay, to increase the risk of venous thrombosis in two population-based case-control studies. The genes influencing CLT are as yet unknown. Patients/methods: We tested CLT as risk factor for venous thrombosis in Kindred Vermont II (n = 346), a pedigree suffering from a high thrombosis risk, partially attributable to a type I protein C deficiency. Furthermore, we tested for quantitative trait loci (QTLs) for CLT, using variance component linkage analysis. Results: Protein C-deficient family members had shorter CLTs than non-deficient members (median CLT 67 min vs. 75 min). One standard deviation increase in CLT increased the risk of venous thrombosis 2.4-fold in non-deficient family members. Protein C deficiency without elevated CLT increased the risk 6.9-fold. Combining both risk factors yielded a 27.8-fold increased risk. The heritability of CLT was 42-52%. We found suggestive evidence of linkage on chromosome 11 (62 cM), partly explained by the prothrombin 20210A mutation, and on chromosome 13 (52 cM). Thrombin-activatable fibrinolysis inhibitor genotypes did not explain the variation in CLT. Conclusion: Hypofibrinolysis appears to increase thrombosis risk in this family, especially in combination with protein C deficiency. Protein C deficiency is associated with short CLT. CLT is partly genetically regulated. Suggestive QTLs were found on chromosomes 11 and 13.
MeSH term(s)	Blood Coagulation Tests ; Carboxypeptidase B2/genetics ; Chromosomes, Human, Pair 11 ; Chromosomes, Human, Pair 13 ; Family ; Fibrinolysis/genetics ; Genetic Linkage ; Genome, Human/physiology ; Humans ; Mutation ; Protein C Deficiency/genetics ; Protein C Deficiency/physiopathology ; Prothrombin/genetics ; Quantitative Trait Loci ; Thrombosis/genetics
Chemical Substances	Prothrombin (9001-26-7) ; Carboxypeptidase B2 (EC 3.4.17.20)
Language	English
Publishing date	2010-12-23
Publishing country	England
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	2112661-6
ISSN	1538-7836 ; 1538-7933
ISSN (online)	1538-7836
ISSN	1538-7933
DOI	10.1111/j.1538-7836.2011.04343.x
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Zs.A 5805: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 295: Show issues

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Article ; Online: Addition of [(eta(5)-C(5)Me(5))IrH(4)] to a zwitterionic silylene: stepwise formation of iridium(V)-silyl and iridium(III)-silylene complexes.

Jungton, Ann-Katrin / Meltzer, Antje / Präsang, Carsten / Braun, Thomas / Driess, Matthias / Penner, Anna

Dalton transactions (Cambridge, England : 2003)

2010 Volume 39, Issue 23, Page(s) 5436–5438

Abstract: ... into an iridium-hydrogen bond of the iridium(v) hydride , [(eta(5)-C(5)Me(5))IrH(4)]. Complex undergoes proton ...

Abstract	The first zwitterionic silyl-iridium(v) complex is generated by insertion of silylene into an iridium-hydrogen bond of the iridium(v) hydride , [(eta(5)-C(5)Me(5))IrH(4)]. Complex undergoes proton migration from an IrH moiety to the terminal CH(2) group of the silyl ligand to furnish the N-donor stabilised Ir(iii)-silylene complex .
Language	English
Publishing date	2010-06-21
Publishing country	England
Document type	Journal Article
ZDB-ID	1472887-4
ISSN	1477-9234 ; 1364-5447 ; 0300-9246 ; 1477-9226
ISSN (online)	1477-9234 ; 1364-5447
ISSN	0300-9246 ; 1477-9226
DOI	10.1039/c0dt00236d
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