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  1. Article ; Online: Robert Weinberg.

    Weinberg, Robert

    Nature biotechnology

    2011  Volume 29, Issue 3, Page(s) 192

    MeSH term(s) Biomedical Research/trends ; Humans ; Medical Oncology/trends ; Neoplasms/diagnosis ; Neoplasms/therapy
    Language English
    Publishing date 2011-03
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1311932-1
    ISSN 1546-1696 ; 1087-0156
    ISSN (online) 1546-1696
    ISSN 1087-0156
    DOI 10.1038/nbt.1814
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  2. Book: Did Robert Bork understate the competitive impact of mergers?

    Ashenfelter, Orley C / Hosken, Daniel / Weinberg, Matthew C

    evidence from consummated mergers

    (NBER working paper series ; 19939)

    2014  

    Author's details Orley C. Ashenfelter; Daniel Hosken; Matthew C. Weinberg
    Series title NBER working paper series ; 19939
    Keywords Fusionskontrolle ; Wirkungsanalyse ; Oligopol ; Wettbewerbsbeschränkung ; USA
    Language English
    Size 33, [8] S., graph. Darst.
    Publishing place Cambridge, Mass
    Document type Book
    Database ECONomics Information System

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  3. Article: Did Robert Bork understate the competitive impact of mergers?

    Ashenfelter, Orley / Hosken, Daniel / Weinberg, Matthew

    The journal of law & economics Bd. 57.2014, Nr. 3, Part 2, (Aug.), S. S67-S100

    evidence from consummated mergers

    2014  

    Author's details Orley Ashenfelter; Daniel Hosken; Matthew Weinberg
    Keywords Fusionskontrolle ; Wirkungsanalyse ; Oligopol ; Wettbewerbsbeschränkung ; USA
    Language English
    Publisher Univ. of Chicago Press
    Publishing place Chicago, Ill
    Document type Article
    ZDB-ID 218304-3 ; 2035113-6
    ISSN 1537-5285 ; 0022-2186
    ISSN (online) 1537-5285
    ISSN 0022-2186
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  4. Article ; Online: It took a long, long time: Ras and the race to cure cancer.

    Weinberg, Robert A

    Cell

    2024  Volume 187, Issue 7, Page(s) 1574–1577

    Abstract: Discoveries of the roles of RAS oncogenes in cancer development four decades ago opened the door to proving that tumor development is driven by somatic mutations' altering the genomes of cancer cells. These discoveries led to illusions about the ... ...

    Abstract Discoveries of the roles of RAS oncogenes in cancer development four decades ago opened the door to proving that tumor development is driven by somatic mutations' altering the genomes of cancer cells. These discoveries led to illusions about the simplicity of cancer pathogenesis and how cancer could be cured.
    MeSH term(s) Humans ; Genes, ras ; Neoplasms/genetics ; Neoplasms/therapy ; Oncogenes ; Mutation
    Language English
    Publishing date 2024-03-28
    Publishing country United States
    Document type Journal Article
    ZDB-ID 187009-9
    ISSN 1097-4172 ; 0092-8674
    ISSN (online) 1097-4172
    ISSN 0092-8674
    DOI 10.1016/j.cell.2024.02.042
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  5. Book: The biology of cancer

    Weinberg, Robert A.

    2014  

    Author's details Robert A. Weinberg
    Keywords Neoplasms / genetics ; Genes, Neoplasm ; Cell Transformation, Neoplastic ; Cancer/Molecular aspects ; Cancer/Genetic aspects ; Cancer cells ; Neoplasms/Genetics ; Neoplastic cell transformation ; Genes/Neoplasm ; Krebs ; Genetik ; Biologie ; Biomedizin ; Carcinogenese ; Molekularbiologie
    Subject Molekulare Biologie ; Krebs ; Krebsentstehung ; Karzinogenese ; Kanzerogenese ; Onkogenese ; Carcinom ; Malignom ; Maligner Tumor ; Neoplasma ; Karzinom ; Bösartiger Tumor ; Krebserkrankung ; Allgemeine Biologie ; Allgemeine Genetik ; Erbbiologie ; Erbforschung ; Erblehre ; Vererbungslehre ; Vererbungswissenschaft ; Erblichkeitslehre
    Language English
    Size Getr. Zählung : zahlr. Ill., graph. Darst.
    Edition 2. ed.
    Publisher Garland Science
    Publishing place New York, NY u.a.
    Publishing country United States
    Document type Book
    Note Hier auch später erschienene, unveränderte Nachdrucke ; Poster-Beil. u.d.T.: Pathways in human cancer
    Accompanying material 1 DVD (12 cm); 1 Poster
    HBZ-ID HT017697923
    ISBN 978-0-8153-4528-2 ; 978-0-8153-4220-5 ; 978-0-8153-4219-9 ; 0-8153-4528-3 ; 0-8153-4220-9 ; 0-8153-4219-5
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2013 A 1793 available for loan (reading room) Lesesaal EG
    2013 MG 3 <<[Begleitmaterial]>> order for loan Magazin
    2013 MO 230 <<[Begleitmaterial]>> order for loan Magazin

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  6. Article ; Online: David M. Livingston (1941-2021).

    Weinberg, Robert A

    Cancer cell

    2021  Volume 39, Issue 12, Page(s) 1560–1561

    Language English
    Publishing date 2021-12-14
    Publishing country United States
    Document type Journal Article
    ZDB-ID 2078448-X
    ISSN 1878-3686 ; 1535-6108
    ISSN (online) 1878-3686
    ISSN 1535-6108
    DOI 10.1016/j.ccell.2021.11.008
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    Zs.MG 104: Show issues

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  7. Book: The biology of cancer

    Weinberg, Robert A.

    2007  

    Title variant Pathways in human cancer
    Author's details Robert A. Weinberg
    Keywords Neoplasms / genetics ; Genes, Neoplasm ; Cell Transformation, Neoplastic ; Krebs ; Cytogenetik
    Subject Zellgenetik ; Zytogenetik ; Carcinom ; Malignom ; Maligner Tumor ; Neoplasma ; Karzinom ; Bösartiger Tumor ; Krebserkrankung
    Language English
    Size Getr. Zählung : Ill., graph. Darst.
    Publisher Garland Science
    Publishing place New York, NY u.a.
    Publishing country United States
    Document type Book
    Note Poster-Beil. u.d.T.: Pathways in human cancer
    Accompanying material 1 CD-ROM (12 cm); 1 Poster
    HBZ-ID HT014831461
    ISBN 0-8153-4078-8 ; 0-8153-4076-1 ; 978-0-8153-4076-8 ; 978-0-8153-4078-2
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

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    2006 MQ 109 <<[Begleitmaterial]>> order for loan Magazin
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  8. Article ; Online: How TP53 (almost) became an oncogene.

    Weinberg, Robert A

    Journal of molecular cell biology

    2019  Volume 11, Issue 7, Page(s) 531–533

    MeSH term(s) Adult ; Female ; History, 20th Century ; History, 21st Century ; Humans ; Oncogenes ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/history ; Tumor Suppressor Protein p53/metabolism
    Chemical Substances TP53 protein, human ; Tumor Suppressor Protein p53
    Language English
    Publishing date 2019-07-24
    Publishing country United States
    Document type Historical Article ; Journal Article
    ZDB-ID 2500949-7
    ISSN 1759-4685 ; 1674-2788
    ISSN (online) 1759-4685
    ISSN 1674-2788
    DOI 10.1093/jmcb/mjz061
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  9. Article ; Online: Leveraging immunochemotherapy for treating pancreatic cancer.

    Dongre, Anushka / Weinberg, Robert A

    Cell research

    2021  Volume 31, Issue 12, Page(s) 1228–1229

    MeSH term(s) Humans ; Immunotherapy ; Pancreatic Neoplasms/drug therapy
    Language English
    Publishing date 2021-10-19
    Publishing country England
    Document type Journal Article ; Comment
    ZDB-ID 1319303-x
    ISSN 1748-7838 ; 1001-0602
    ISSN (online) 1748-7838
    ISSN 1001-0602
    DOI 10.1038/s41422-021-00574-x
    Database MEDical Literature Analysis and Retrieval System OnLINE

    Full text online

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  10. Article ; Online: Linking EMT programmes to normal and neoplastic epithelial stem cells.

    Lambert, Arthur W / Weinberg, Robert A

    Nature reviews. Cancer

    2021  Volume 21, Issue 5, Page(s) 325–338

    Abstract: Epithelial stem cells serve critical physiological functions in the generation, maintenance and repair of diverse tissues through their ability to self-renew and spawn more specialized, differentiated cell types. In an analogous fashion, cancer stem ... ...

    Abstract Epithelial stem cells serve critical physiological functions in the generation, maintenance and repair of diverse tissues through their ability to self-renew and spawn more specialized, differentiated cell types. In an analogous fashion, cancer stem cells have been proposed to fuel the growth, progression and recurrence of many carcinomas. Activation of an epithelial-mesenchymal transition (EMT), a latent cell-biological programme involved in development and wound healing, has been linked to the formation of both normal and neoplastic stem cells, but the mechanistic basis underlying this connection remains unclear. In this Perspective, we outline the instances where aspects of an EMT have been implicated in normal and neoplastic epithelial stem cells and consider the involvement of this programme during tissue regeneration and repair. We also discuss emerging concepts and evidence related to the heterogeneous and plastic cell states generated by EMT programmes and how these bear on our understanding of cancer stem cell biology and cancer metastasis. A more comprehensive accounting of the still-elusive links between EMT programmes and the stem cell state will surely advance our understanding of both normal stem cell biology and cancer pathogenesis.
    MeSH term(s) Animals ; Cell Differentiation ; Epithelial Cells/pathology ; Epithelial-Mesenchymal Transition ; Humans ; Neoplasms/pathology ; Neoplasms/therapy ; Neoplastic Stem Cells/pathology
    Language English
    Publishing date 2021-02-05
    Publishing country England
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 2062767-1
    ISSN 1474-1768 ; 1474-175X
    ISSN (online) 1474-1768
    ISSN 1474-175X
    DOI 10.1038/s41568-021-00332-6
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