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  1. Article ; Online: GLP-1 receptor agonists in the treatment of diabetic non-alcoholic steatohepatitis patients.

    Adeghate, Ernest A

    Expert opinion on pharmacotherapy

    2024  , Page(s) 1–10

    Abstract: Introduction: Nonalcoholic fatty liver disease (NAFLD) is the most common hepatic disease affecting almost 30% of the world population. Approximately 25% of people with NAFLD develop nonalcoholic steatohepatitis (NASH), the fulminant version of the ... ...

    Abstract Introduction: Nonalcoholic fatty liver disease (NAFLD) is the most common hepatic disease affecting almost 30% of the world population. Approximately 25% of people with NAFLD develop nonalcoholic steatohepatitis (NASH), the fulminant version of the disease. Diabetes mellitus is present in 22.5% of people with NAFLD and 44.60% of individuals with NASH. This review was undertaken to examine the current contribution of glucagon-like peptide 1 (GLP-1) receptor agonists to the pharmacotherapy of diabetic nonalcoholic steatohepatitis.
    Areas covered: The author analyzed the current status of GLP-1 receptor agonists for pharmacotherapy of diabetic NASH. Research data and literature reports were taken from the database and or websites of Diabetes UK, American Diabetes Association, ClinicalTrials.gov, PubMed, and Scopus. The keywords utilized included type 2 diabetes, GLP-1, NASH, NAFLD, and clinical trials.
    Expert opinion: Since diabetic NASH is associated with obesity, diabetes mellitus, oxidative stress and inflammation, drugs capable of mitigating all of these conditions simultaneously, are most ideal for the treatment of diabetic NASH. These drugs include (in order of relevance), GLP-1 receptor agonists, GLP-1 and GIP dual receptor agonists, sodium-glucose co-transporter-2 (SGLT2) inhibitors, and pioglitazone. The future, FDA-approved drug for diabetic NASH treatment will likely be GLP-1 agonist, which could be used as monotherapy or in combination with other drugs.
    Language English
    Publishing date 2024-03-18
    Publishing country England
    Document type Journal Article
    ZDB-ID 2001535-5
    ISSN 1744-7666 ; 1465-6566
    ISSN (online) 1744-7666
    ISSN 1465-6566
    DOI 10.1080/14656566.2024.2328796
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 5130: Show issues Location:
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  2. Book ; Conference proceedings: Diabetes mellitus and its complications

    Adeghate, Ernest

    molecular mechanisms, epidemiology, and clinical medicine ; [result of a conference entitled International Conference on Recent Advances in Diabetes Mellitus and Its Complications, held on March 6 - 9, 2006 in Al Ain, United Arab Emirates]

    (Annals of the New York Academy of Sciences ; 1084)

    2006  

    Event/congress International Conference on Recent Advances in Diabetes Mellitus and Its Complications (2006, al-ʿAin)
    Author's details ed. by Ernest Adeghate
    Series title Annals of the New York Academy of Sciences ; 1084
    Collection
    Language English
    Size XIV, 532 S. : Ill., graph. Darst.
    Publisher Blackwell
    Publishing place Boston, Mass. u.a.
    Publishing country United States
    Document type Book ; Conference proceedings
    HBZ-ID HT014969110
    ISBN 1-57331-635-0 ; 978-1-57331-635-4
    Database Catalogue ZB MED Medicine, Health

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    Shelf markLoan statusLocation
    Se 110 -1084- verfügbar in Königswinter Königswinter

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  3. Book ; Conference proceedings: Special issue on Diabetes Mellitus and its complications: from molecular biology to clinical medicine

    Adeghate, Ernest

    [based on proceedings of the 3rd International Symposium on Diabetes Mellitus and its Complications, held in Budapest, Hungary, from July 2 - 6, 2001]

    (Archives of physiology and biochemistry ; 109,3)

    2001  

    Title variant Diabetes Mellitus and its complications: from molecular biology to clinical medicine
    Institution International Symposium on Diates Mellitus and its Complications
    Author's details [guest ed.: E. Adeghate]
    Series title Archives of physiology and biochemistry ; 109,3
    Collection
    Language English
    Size S. 195 - 291 : Ill., graph. Darst.
    Publisher Swets & Zeitlinger
    Publishing place Lisse
    Publishing country Netherlands
    Document type Book ; Conference proceedings
    HBZ-ID HT013369204
    Database Catalogue ZB MED Medicine, Health

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    Shelf markLoan statusLocation
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  4. Article ; Online: An Update on the Molecular and Cellular Basis of Pharmacotherapy in Type 2 Diabetes Mellitus.

    Mahgoub, Mohamed Omer / Ali, Ifrah Ismail / Adeghate, Jennifer O / Tekes, Kornélia / Kalász, Huba / Adeghate, Ernest A

    International journal of molecular sciences

    2023  Volume 24, Issue 11

    Abstract: Diabetes mellitus (DM) is a chronic illness with an increasing global prevalence. More than 537 million cases of diabetes were reported worldwide in 2021, and the number is steadily increasing. The worldwide number of people suffering from DM is ... ...

    Abstract Diabetes mellitus (DM) is a chronic illness with an increasing global prevalence. More than 537 million cases of diabetes were reported worldwide in 2021, and the number is steadily increasing. The worldwide number of people suffering from DM is projected to reach 783 million in 2045. In 2021 alone, more than USD 966 billion was spent on the management of DM. Reduced physical activity due to urbanization is believed to be the major cause of the increase in the incidence of the disease, as it is associated with higher rates of obesity. Diabetes poses a risk for chronic complications such as nephropathy, angiopathy, neuropathy and retinopathy. Hence, the successful management of blood glucose is the cornerstone of DM therapy. The effective management of the hyperglycemia associated with type 2 diabetes includes physical exercise, diet and therapeutic interventions (insulin, biguanides, second generation sulfonylureas, glucagon-like peptide 1 agonists, dipeptidyl-peptidase 4 inhibitors, thiazolidinediones, amylin mimetics, meglitinides, α-glucosidase inhibitors, sodium-glucose cotransporter-2 inhibitors and bile acid sequestrants). The optimal and timely treatment of DM improves the quality of life and reduces the severe burden of the disease for patients. Genetic testing, examining the roles of different genes involved in the pathogenesis of DM, may also help to achieve optimal DM management in the future by reducing the incidence of DM and by enhancing the use of individualized treatment regimens.
    MeSH term(s) Humans ; Diabetes Mellitus, Type 2/complications ; Hypoglycemic Agents/therapeutic use ; Hypoglycemic Agents/pharmacology ; Quality of Life ; Sodium-Glucose Transporter 2 Inhibitors/therapeutic use ; Dipeptidyl-Peptidase IV Inhibitors/pharmacology ; Dipeptidyl-Peptidase IV Inhibitors/therapeutic use
    Chemical Substances Hypoglycemic Agents ; Sodium-Glucose Transporter 2 Inhibitors ; Dipeptidyl-Peptidase IV Inhibitors
    Language English
    Publishing date 2023-05-26
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 2019364-6
    ISSN 1422-0067 ; 1422-0067 ; 1661-6596
    ISSN (online) 1422-0067
    ISSN 1422-0067 ; 1661-6596
    DOI 10.3390/ijms24119328
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  5. Article ; Online: Histamine H3 receptor antagonists - Roles in neurological and endocrine diseases and diabetes mellitus.

    Abdulrazzaq, Yousef M / Bastaki, Salim M A / Adeghate, Ernest

    Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

    2022  Volume 150, Page(s) 112947

    Abstract: Human histamine H3 receptor (H3R) was initially described in the brain of rat in 1983 and cloned in 1999. It can be found in the human brain and functions as a regulator of histamine synthesis and release. H3 receptors are predominantly resident in the ... ...

    Abstract Human histamine H3 receptor (H3R) was initially described in the brain of rat in 1983 and cloned in 1999. It can be found in the human brain and functions as a regulator of histamine synthesis and release. H3 receptors are predominantly resident in the presynaptic region of neurons containing histamine, where they modulate the synthesis and release of histamine (autoreceptor) or other neurotransmitters such as dopamine, norepinephrine, gamma-aminobutyric acid (GABA), glutamate, acetylcholine and serotonin (all heteroreceptors). The human histamine H3 receptor has twenty isoforms of which eight are functional. H3 receptor expression is seen in the cerebral cortex, neurons of the basal ganglia and hippocampus, which are important for process of cognition, sleep and homoeostatic regulation. In addition, histamine H3R antagonists stimulate insulin release, through inducing the release of acetylcholine and cause significant reduction in total body weight and triglycerides in obese subjects by causing a feeling of satiety in the hypothalamus. The ability of histamine H3R antagonist to reduce diabetes-induced hyperglycaemia is comparable to that of metformin. It is reasonable therefore, to claim that H3 receptor antagonists may play an important role in the therapy of disorders of cognition, the ability to sleep, oxidative stress, inflammation and anomaly of glucose homoeostasis. A large number of H3R antagonists are being developed by pharmaceutical companies and university research centres. As examples of these new drugs, this review will discuss a number of drugs, including the first histamine H3R receptor antagonist produced.
    MeSH term(s) Acetylcholine ; Animals ; Diabetes Mellitus ; Histamine ; Histamine Antagonists/pharmacology ; Histamine H3 Antagonists/pharmacology ; Histamine H3 Antagonists/therapeutic use ; Humans ; Rats ; Receptors, Histamine H3/metabolism
    Chemical Substances Histamine Antagonists ; Histamine H3 Antagonists ; Receptors, Histamine H3 ; Histamine (820484N8I3) ; Acetylcholine (N9YNS0M02X)
    Language English
    Publishing date 2022-04-19
    Publishing country France
    Document type Journal Article ; Review
    ZDB-ID 392415-4
    ISSN 1950-6007 ; 0753-3322 ; 0300-0893
    ISSN (online) 1950-6007
    ISSN 0753-3322 ; 0300-0893
    DOI 10.1016/j.biopha.2022.112947
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Ud II Zs.198: Show issues Location:
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  6. Article ; Online: Adropin's Role in Energy Homeostasis and Metabolic Disorders.

    Ali, Ifrah Ismail / D'Souza, Crystal / Singh, Jaipaul / Adeghate, Ernest

    International journal of molecular sciences

    2022  Volume 23, Issue 15

    Abstract: Adropin is a novel 76-amino acid-peptide that is expressed in different tissues and cells including the liver, pancreas, heart and vascular tissues, kidney, milk, serum, plasma and many parts of the brain. Adropin, encoded by ... ...

    Abstract Adropin is a novel 76-amino acid-peptide that is expressed in different tissues and cells including the liver, pancreas, heart and vascular tissues, kidney, milk, serum, plasma and many parts of the brain. Adropin, encoded by the
    MeSH term(s) Animals ; Blood Proteins/genetics ; Cholesterol ; Glucose/metabolism ; Homeostasis ; Intercellular Signaling Peptides and Proteins/genetics ; Metabolic Diseases ; Mice
    Chemical Substances Blood Proteins ; Intercellular Signaling Peptides and Proteins ; Cholesterol (97C5T2UQ7J) ; Glucose (IY9XDZ35W2)
    Language English
    Publishing date 2022-07-28
    Publishing country Switzerland
    Document type Journal Article ; Review
    ZDB-ID 2019364-6
    ISSN 1422-0067 ; 1422-0067 ; 1661-6596
    ISSN (online) 1422-0067
    ISSN 1422-0067 ; 1661-6596
    DOI 10.3390/ijms23158318
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  7. Article ; Online: A pilot study: effect of irisin on trabecular bone in a streptozotocin-induced animal model of type 1 diabetic osteopathy utilizing a micro-CT.

    Mohsin, Sahar / Brock, Fiona / Kaimala, Suneesh / Greenwood, Charlene / Sulaiman, Mohsin / Rogers, Keith / Adeghate, Ernest

    PeerJ

    2023  Volume 11, Page(s) e16278

    Abstract: Background: Osteoporosis is a significant co-morbidity of type 1 diabetes mellitus (DM1) leading to increased fracture risk. Exercise-induced hormone 'irisin' in low dosage has been shown to have a beneficial effect on bone metabolism by increasing ... ...

    Abstract Background: Osteoporosis is a significant co-morbidity of type 1 diabetes mellitus (DM1) leading to increased fracture risk. Exercise-induced hormone 'irisin' in low dosage has been shown to have a beneficial effect on bone metabolism by increasing osteoblast differentiation and reducing osteoclast maturation, and inhibiting apoptosis and inflammation. We investigated the role of irisin in treating diabetic osteopathy by observing its effect on trabecular bone.
    Methods: DM1 was induced by intraperitoneal injection of streptozotocin 60 mg/kg body weight. Irisin in low dosage (5 µg twice a week for 6 weeks I/P) was injected into half of the control and 4-week diabetic male Wistar rats. Animals were sacrificed six months after induction of diabetes. The trabecular bone in the femoral head and neck was analyzed using a micro-CT technique. Bone turnover markers were measured using ELISA, Western blot, and RT-PCR techniques.
    Results: It was found that DM1 deteriorates the trabecular bone microstructure by increasing trabecular separation (Tb-Sp) and decreasing trabecular thickness (Tb-Th), bone volume fraction (BV/TV), and bone mineral density (BMD). Irisin treatment positively affects bone quality by increasing trabecular number
    Conclusions: This is the first pilot study to our knowledge that shows that a low dose of irisin marginally improves the trabecular bone in DM1 and is an effective peptide in reducing sclerostin levels.
    MeSH term(s) Rats ; Animals ; Male ; X-Ray Microtomography ; Fibronectins ; Pilot Projects ; Streptozocin ; Osteocalcin ; Diabetes Mellitus, Type 1/drug therapy ; Cancellous Bone/diagnostic imaging ; Rats, Wistar ; Models, Animal
    Chemical Substances Fibronectins ; Streptozocin (5W494URQ81) ; Osteocalcin (104982-03-8)
    Language English
    Publishing date 2023-10-17
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 2703241-3
    ISSN 2167-8359 ; 2167-8359
    ISSN (online) 2167-8359
    ISSN 2167-8359
    DOI 10.7717/peerj.16278
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Article ; Online: Lycopodium Mitigates Oxidative Stress and Inflammation in the Colonic Mucosa of Acetic Acid-Induced Colitis in Rats.

    Bastaki, Salim M A / Amir, Naheed / Adeghate, Ernest / Ojha, Shreesh

    Molecules (Basel, Switzerland)

    2022  Volume 27, Issue 9

    Abstract: Inflammatory bowel diseases (IBDs) such as ulcerative colitis (UC) and Crohn's disease (CD) are diseases of the gastrointestinal system involving genetic and environmental factors attributed to oxidative stress and inflammation. Targeting oxidative ... ...

    Abstract Inflammatory bowel diseases (IBDs) such as ulcerative colitis (UC) and Crohn's disease (CD) are diseases of the gastrointestinal system involving genetic and environmental factors attributed to oxidative stress and inflammation. Targeting oxidative stress and inflammation by novel dietary compounds of natural origin convincingly appears to be one of the important therapeutic strategies to keep the disease in remission. As there is no permanent cure for IBD except for chronic long-term treatment or surgery, it is therefore imperative to investigate plant-based agents that are receiving attention for their therapeutic benefits to overcome the debilitating clinical conditions of IBD. Lycopodium (LYCO), a plant of tropical and subtropical origin and known by numerous names such as ground pine, club moss, or devil's claw, has been popularly used for centuries in traditional medicine including Chinese and Indian medicines. In the present study, the effect of LYCO has been investigated in an acetic acid (AA)-induced colitis model in Wistar rats. LYCO was orally administered at the dose of 50 mg/kg/day either 3 days before or 30 min after the induction of IBD and continued for 7 days by intrarectal administration of AA. The changes in body weight and macroscopic and microscopic analysis of the colon of rats of different experimental groups were observed on days 0, 2, 4, and 7. The levels of myeloperoxidase (MPO), reduced glutathione (GSH), and malondialdehyde (MDA) were measured. AA caused a significant reduction in body weight and increased macroscopic and microscopic ulcer scores along with a significant decline in antioxidant enzymes, superoxide dismutase (SOD), and catalase and antioxidant substrate, glutathione (GSH). There was a concomitant increased formation of malondialdehyde (MDA), a marker of lipid peroxidation, and raised myeloperoxidase (MPO) activity, a marker of neutrophil activation. Treatment with LYCO significantly improved IBD-induced reduction in body weight, improved histology, inhibited MDA formation, and restored antioxidants along with reduced MPO activity. AA also caused the release of proinflammatory cytokines such as interleukin-1β (IL-1β) and interleukin-23 (IL-23). Furthermore, AA also increased the levels of calprotectin, a protein released by neutrophils under inflammatory conditions of the gastrointestinal tract. LYCO treatment significantly reduced the release of calprotectin and proinflammatory cytokines. The results demonstrate that LYCO treatment has the potential to improve disease activity by inhibiting oxidative stress, lipid peroxidation, and inflammation along with histological preservation of colonic tissues.
    MeSH term(s) Acetic Acid/metabolism ; Animals ; Anti-Inflammatory Agents/therapeutic use ; Antioxidants/metabolism ; Body Weight ; Colitis/chemically induced ; Colitis/drug therapy ; Colitis/metabolism ; Colitis, Ulcerative/chemically induced ; Colitis, Ulcerative/drug therapy ; Colitis, Ulcerative/metabolism ; Cytokines/metabolism ; Glutathione/metabolism ; Inflammation/metabolism ; Inflammatory Bowel Diseases/pathology ; Intestinal Mucosa/metabolism ; Leukocyte L1 Antigen Complex/metabolism ; Leukocyte L1 Antigen Complex/pharmacology ; Leukocyte L1 Antigen Complex/therapeutic use ; Lycopodium ; Malondialdehyde/metabolism ; Oxidative Stress ; Peroxidase/metabolism ; Rats ; Rats, Wistar
    Chemical Substances Anti-Inflammatory Agents ; Antioxidants ; Cytokines ; Leukocyte L1 Antigen Complex ; Malondialdehyde (4Y8F71G49Q) ; Peroxidase (EC 1.11.1.7) ; Glutathione (GAN16C9B8O) ; Acetic Acid (Q40Q9N063P)
    Language English
    Publishing date 2022-04-26
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 1413402-0
    ISSN 1420-3049 ; 1431-5165 ; 1420-3049
    ISSN (online) 1420-3049
    ISSN 1431-5165 ; 1420-3049
    DOI 10.3390/molecules27092774
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.MO 81: Show issues

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  9. Article ; Online: Pancreatic β-cell: the beauty of being plastic.

    Adeghate, Ernest

    Experimental physiology

    2012  Volume 97, Issue 8, Page(s) 906–907

    MeSH term(s) Animals ; Blood Glucose/metabolism ; Hyperglycemia/blood ; Insulin-Secreting Cells/drug effects
    Chemical Substances Blood Glucose
    Language English
    Publishing date 2012-08
    Publishing country England
    Document type Comment ; Journal Article
    ZDB-ID 1016295-1
    ISSN 1469-445X ; 0958-0670
    ISSN (online) 1469-445X
    ISSN 0958-0670
    DOI 10.1113/expphysiol.2012.064766
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    Uc I Zs.102: Show issues Location:
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  10. Article ; Online: Early (5-Day) Onset of Diabetes Mellitus Causes Degeneration of Photoreceptor Cells, Overexpression of Incretins, and Increased Cellular Bioenergetics in Rat Retina.

    Adeghate, Jennifer O / D'Souza, Crystal / Kántor, Orsolya / Tariq, Saeed / Souid, Abdul-Kader / Adeghate, Ernest

    Cells

    2021  Volume 10, Issue 8

    Abstract: The effects of early (5-day) onset of diabetes mellitus (DM) on retina ultrastructure and cellular bioenergetics were examined. The retinas of streptozotocin-induced diabetic rats were compared to those of non-diabetic rats using light and transmission ... ...

    Abstract The effects of early (5-day) onset of diabetes mellitus (DM) on retina ultrastructure and cellular bioenergetics were examined. The retinas of streptozotocin-induced diabetic rats were compared to those of non-diabetic rats using light and transmission electron microscopy. Tissue localization of glucagon-like-peptide-1 (GLP-1), exendin-4 (EXE-4), and catalase (CAT) in non-diabetic and diabetic rat retinas was conducted using immunohistochemistry, while the retinal and plasma concentration of GLP-1, EXE-4, and CAT were measured with ELISA. Lipid profiles and kidney and liver function markers were measured from the blood of non-diabetic and diabetic rats with an automated biochemical analyzer. Oxygen consumption was monitored using a phosphorescence analyzer, and the adenosine triphosphate (ATP) level was determined using the Enliten ATP assay kit. Blood glucose and cholesterol levels were significantly higher in diabetic rats compared to control. The number of degenerated photoreceptor cells was significantly higher in the diabetic rat retina. Tissue levels of EXE-4, GLP-1 and CAT were significantly (
    MeSH term(s) Adenosine Triphosphate/metabolism ; Animals ; Biomarkers/blood ; Blood Glucose/analysis ; Catalase/blood ; Catalase/metabolism ; Diabetes Mellitus, Experimental/metabolism ; Diabetes Mellitus, Experimental/pathology ; Glucagon-Like Peptide 1/blood ; Glucagon-Like Peptide 1/metabolism ; Incretins/blood ; Incretins/genetics ; Incretins/metabolism ; Male ; Microscopy, Electron, Transmission ; Oxygen Consumption ; Photoreceptor Cells/cytology ; Photoreceptor Cells/metabolism ; Rats ; Rats, Wistar ; Retina/metabolism ; Retina/pathology ; Retina/ultrastructure
    Chemical Substances Biomarkers ; Blood Glucose ; Incretins ; Glucagon-Like Peptide 1 (89750-14-1) ; Adenosine Triphosphate (8L70Q75FXE) ; Catalase (EC 1.11.1.6)
    Language English
    Publishing date 2021-08-04
    Publishing country Switzerland
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 2661518-6
    ISSN 2073-4409 ; 2073-4409
    ISSN (online) 2073-4409
    ISSN 2073-4409
    DOI 10.3390/cells10081981
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