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Book: Zhong guo guo min jing ji de she hui zhu yi gai zao

Xue, Muqiao / Su, Xing / Lin, Zili

1978

Author's details	Xue mu qiao, su xing, lin zi li deng zhu
Keywords	China
Language	Chinese
Size	148 pages, Tab., 21 cm
Edition	Di 3 ban, di 3 ci yin shua
Publisher	Ren min chu ban she
Publishing place	Bei jing
Document type	Book
Note	Includes bibliographical references
Database	Former special subject collection: coastal and deep sea fishing

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Article ; Online: Editor's Note:

Boukerche, Habib / Su, Zao-Zhong / Emdad, Luni / Baril, Patrick / Balme, Brigitte / Thomas, Luc / Randolph, Aaron / Valerie, Kristoffer / Sarkar, Devanand / Fisher, Paul B

Cancer research

2019 Volume 79, Issue 19, Page(s) 5127

Language	English
Publishing date	2019-09-30
Publishing country	United States
Document type	Journal Article ; Published Erratum
ZDB-ID	1432-1
ISSN	1538-7445 ; 0008-5472
ISSN (online)	1538-7445
ISSN	0008-5472
DOI	10.1158/0008-5472.CAN-19-2443
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Article: [Rapid identification of constituents of Urtica hyperborea using UPLC-ESI-Q-TOF-MS/MS method].

Su, Ri-Na / Luo, Wei-Zao / Wei, Rong-Rui / Ao, Wu-Li-Ji / Zhong, Guo-Yue

Zhongguo Zhong yao za zhi = Zhongguo zhongyao zazhi = China journal of Chinese materia medica

2019 Volume 44, Issue 8, Page(s) 1607–1614

Abstract: This paper deals with the application of ultra-performance liquid chromatography tandem quadrupole time of flight mass spectrometry(UPLC-ESI-Q-TOF-MS/MS) method to rapidly determine and analyze the chemical constituents of methanol extract of Urtica ... ...

Abstract	This paper deals with the application of ultra-performance liquid chromatography tandem quadrupole time of flight mass spectrometry(UPLC-ESI-Q-TOF-MS/MS) method to rapidly determine and analyze the chemical constituents of methanol extract of Urtica hyperborea. We employed UPLC YMC-Triart C18(2. 1 mm×100 mm,1. 9 μm) column to UPLC analysis with acetonitrile-water(containing 0. 4% formic acid) in gradient as mobile phase. The flow rate was 0. 3 m L·min-1 gradient elution and column temperature was 30℃; the injection volume was 4 μL. ESI ion source was used to ensure the data collected in anegative ion mode. The chemical components of U. hyperborea were identified through retention time,exact relative molecular mass,cleavage fragments of MS/MS and reported data.The results indicated that a total of 31 compounds were identified,including 8 flavonoids,14 phenolic compounds,8 phenylpropanoids(4 coumarins and 4 lignans),and 1 steroidal compound,13 of which were confirmed by comparison. The UPLC-ESI-Q-TOF-MS/MS method could rapid identify the chemical components of U. hyperborea. The above compounds were discovered in U. hyperborea for the first time,which could provide theoretical foundation for further research on the basis of the pharmacodynamics of U. hyperborea.
MeSH term(s)	Chromatography, High Pressure Liquid ; Drugs, Chinese Herbal/chemistry ; Flavonoids ; Lignans ; Phenols ; Phytochemicals/analysis ; Plant Extracts/analysis ; Tandem Mass Spectrometry ; Urticaceae/chemistry
Chemical Substances	Drugs, Chinese Herbal ; Flavonoids ; Lignans ; Phenols ; Phytochemicals ; Plant Extracts
Language	Chinese
Publishing date	2019-06-04
Publishing country	China
Document type	Journal Article
ZDB-ID	1004649-5
ISSN	1001-5302 ; 0254-0029
ISSN	1001-5302 ; 0254-0029
DOI	10.19540/j.cnki.cjcmm.20190118.005
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Article ; Online: Unique conditionally replication competent bipartite adenoviruses-cancer terminator viruses (CTV): efficacious reagents for cancer gene therapy.

Sarkar, Devanand / Su, Zao-Zhong / Fisher, Paul B

Cell cycle (Georgetown, Tex.)

2006 Volume 5, Issue 14, Page(s) 1531–1536

Abstract: The frequent resistance of aggressive cancers to currently available therapies, such as radiotherapy and chemotherapy, mandates development of targeted, nontoxic and more efficacious treatment protocols. Conditionally replication competent adenoviruses ( ... ...

Abstract	The frequent resistance of aggressive cancers to currently available therapies, such as radiotherapy and chemotherapy, mandates development of targeted, nontoxic and more efficacious treatment protocols. Conditionally replication competent adenoviruses (CRCAs) that induce oncolysis by cancer-specific replication are currently being evaluated in clinical trials. However, a single modality approach may not be sufficient to completely eradicate cancer in a patient, because most cancers arise from abnormalities in multiple genetic and signal transduction pathways. The promoter region of rodent progression elevated gene-3 (PEG-3), cloned and characterized in our laboratory, embodies the unique property of increased activity in a broad range of tumor cells, both rodent and human, when compared to normal counterparts. Bipartite adenoviruses were engineered to express the E1A gene, necessary for viral replication, under control of the PEG-3 promoter (PEG-Prom) and simultaneously express a second transgene in the E3 region that encodes an apoptosis-inducing and immunomodulatory cytokine, either immune interferon (IFN-gamma) or melanoma differentiation associated gene-7/interleukin-24 (mda-7/IL-24). These conditionally replication competent bipartite adenoviruses, referred to as cancer terminator viruses (CTVs), facilitated cancer-selective adenovirus replication, robust transgene expression and apoptosis induction with complete eradication of both primary and distant (metastatic) human cancers xenotransplanted in athymic nude mice. These findings suggest that CTVs might prove efficacious for the therapy of primary and advanced neoplastic diseases.
MeSH term(s)	Adenoviridae/genetics ; Animals ; Antineoplastic Agents/therapeutic use ; Genetic Therapy/methods ; Humans ; Mice ; Mice, Nude ; Neoplasms/pathology ; Neoplasms/therapy ; Neoplasms, Experimental/pathology ; Neoplasms, Experimental/therapy ; Oncolytic Virotherapy/methods ; Oncolytic Viruses/genetics ; Transplantation, Heterologous ; Virus Replication
Chemical Substances	Antineoplastic Agents
Language	English
Publishing date	2006-07-17
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, Non-P.H.S. ; Review
ZDB-ID	2146183-1
ISSN	1551-4005 ; 1538-4101 ; 1554-8627
ISSN (online)	1551-4005
ISSN	1538-4101 ; 1554-8627
DOI	10.4161/cc.5.14.3095
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Article ; Online: mda-9/Syntenin: more than just a simple adapter protein when it comes to cancer metastasis.

Sarkar, Devanand / Boukerche, Habib / Su, Zao-Zhong / Fisher, Paul B

Cancer research

2008 Volume 68, Issue 9, Page(s) 3087–3093

Abstract: Cancer is a progressive disease that, in many instances, if untreated, can culminate in metastatic spread of primary tumor cells to distant sites in the body. Metastasis frequently confers virulence and therapy resistance to cancer cells, and defining ... ...

Abstract	Cancer is a progressive disease that, in many instances, if untreated, can culminate in metastatic spread of primary tumor cells to distant sites in the body. Metastasis frequently confers virulence and therapy resistance to cancer cells, and defining the molecular events that control metastasis will be mandatory to develop rational, targeted therapies for effective intervention, prevention of recurrence, and the "holy grail" of engendering a cure. Adapter proteins are physiologically pertinent molecules that, through interactions with key regulatory proteins via specific conserved domains, control important cellular events. Melanoma differentiation associated gene-9 (mda-9), also known as syntenin, is a PDZ domain-containing adapter protein that is involved in organization of protein complexes in the plasma membranes, regulation of B-cell development, intracellular trafficking and cell-surface targeting, synaptic transmission, and axonal outgrowth. Recent studies now define a seminal role for mda-9/syntenin in cancer metastasis. The present review provides a current perspective of our understanding of this important aspect of mda-9/syntenin, suggesting that this gene and its encoded protein and interacting protein partners may provide viable targets for intervening in the final and invariably the most lethal stage of cancer progression, namely, cancer metastasis.
MeSH term(s)	Adaptor Proteins, Signal Transducing/genetics ; Adaptor Proteins, Signal Transducing/physiology ; Animals ; Axons/metabolism ; Axons/physiology ; Cell Adhesion/genetics ; Cloning, Molecular ; Genes, Tumor Suppressor/physiology ; Glutamic Acid/metabolism ; Humans ; Models, Biological ; Neoplasm Metastasis/genetics ; Neurites/metabolism ; Neurites/physiology ; Protein Binding ; Signal Transduction/genetics ; Syntenins/genetics ; Syntenins/metabolism ; Syntenins/physiology ; Tissue Distribution
Chemical Substances	Adaptor Proteins, Signal Transducing ; SDCBP protein, human ; Syntenins ; Glutamic Acid (3KX376GY7L)
Language	English
Publishing date	2008-05-01
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	1432-1
ISSN	1538-7445 ; 0008-5472
ISSN (online)	1538-7445
ISSN	0008-5472
DOI	10.1158/0008-5472.CAN-07-6210
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Article: Cloning differentially expressed genes using rapid subtraction hybridization (RaSH).

Boukerche, Habib / Su, Zao-Zhong / Kang, Dong-Chul / Fisher, Paul B

Methods in molecular biology (Clifton, N.J.)

2007 Volume 383, Page(s) 15–29

Abstract: Differential gene expression represents the entry point for comprehending complex biological processes. In this context, identification and cloning of differentially expressed genes represent critical elements in this process. Many techniques have been ... ...

Abstract	Differential gene expression represents the entry point for comprehending complex biological processes. In this context, identification and cloning of differentially expressed genes represent critical elements in this process. Many techniques have been developed to facilitate achieving these objectives. Although effective in many situations, most currently described approaches are not trouble-free and have limitations, including complexity of performance, redundancy of gene identification (reflecting cloning biases) and false-positive gene identification. A detailed methodology to perform a rapid and efficient cloning approach, called rapid subtraction hybridization is described in this chapter. This strategy has been applied successfully to a number of cell culture systems and biological processes, including terminal differentiation and cancer progression in human melanoma cells, resistance or sensitivity to HIV-1 in human T cells and gene expression changes following infection of normal human fetal astrocytes with HIV-1 or treatment with neutrotoxic agents. Based on its simplicity of performance and high frequency of genuine differential gene identification, the rapid subtraction hybridization (RaSH) approach will allow wide applications in diverse systems and biological contexts.
MeSH term(s)	Blotting, Northern ; Cloning, Molecular/methods ; DNA, Complementary ; Gene Expression Profiling/methods ; Genomics/methods ; Nucleic Acid Hybridization
Chemical Substances	DNA, Complementary
Language	English
Publishing date	2007
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ISSN	1064-3745
ISSN	1064-3745
DOI	10.1007/978-1-59745-335-6_2
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article: [Study on effect of extract from Tibetan medicine Urtica hyperborean on anti-prostatic hyperplasia].

Su, Ri-Na / Wei, Rong-Rui / Luo, Wei-Zao / Zhu, Ji-Xiao / Wang, Lu / Zhong, Guo-Yue

Zhongguo Zhong yao za zhi = Zhongguo zhongyao zazhi = China journal of Chinese materia medica

2019 Volume 44, Issue 9, Page(s) 1953–1959

Abstract: In this study,mouse models of benign prostatic hyperplasia induced by subcutaneous injection of testosterone propionate was used to investigate the therapeutic effect and mechanism of Urtica hyperborean( UW) extracts on prostate hyperplasia in mice. The ... ...

Abstract	In this study,mouse models of benign prostatic hyperplasia induced by subcutaneous injection of testosterone propionate was used to investigate the therapeutic effect and mechanism of Urtica hyperborean( UW) extracts on prostate hyperplasia in mice. The effects of UW extracts on prostate index,serum epidermal growth factor( EGF) and dihydrotestosterone( DHT) in model mice were observed,and the EGF and anti-apoptotic factor( Bcl-2) mRNA expression levels were detected as well as pathological changes in prostate tissue. The results showed that the ethyl acetate extraction and alcohol soluble fraction of the UW could significantly reduce the prostate index,reduce the serum DHT and EGF levels( P<0. 01),and significantly decrease the EGF and Bcl-2 mRNA expression( P<0. 01),significantly improved the morphological structure of prostate tissue. The above results confirmed that ethyl acetate extract and alcohol-soluble parts of UW have a good preventive effect on mice prostatic hyperplasia model,and its mechanism may be to reduce androgen levels by regulating polypeptide growth factors and/or inhibiting cell hyperproliferation and promoting apoptosis. This study laid the foundation for the further research on UW.
MeSH term(s)	Animals ; Dihydrotestosterone/blood ; Epidermal Growth Factor/blood ; Male ; Medicine, Tibetan Traditional ; Mice ; Plant Extracts/pharmacology ; Prostatic Hyperplasia/chemically induced ; Prostatic Hyperplasia/drug therapy ; Proto-Oncogene Proteins c-bcl-2/metabolism ; Testosterone Propionate ; Urticaceae/chemistry
Chemical Substances	Plant Extracts ; Proto-Oncogene Proteins c-bcl-2 ; Dihydrotestosterone (08J2K08A3Y) ; Bcl2 protein, mouse (114100-40-2) ; Epidermal Growth Factor (62229-50-9) ; Testosterone Propionate (WI93Z9138A)
Language	Chinese
Publishing date	2019-07-24
Publishing country	China
Document type	Journal Article
ZDB-ID	1004649-5
ISSN	1001-5302 ; 0254-0029
ISSN	1001-5302 ; 0254-0029
DOI	10.19540/j.cnki.cjcmm.20190304.004
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Article ; Online: Astrocyte elevated gene-1: far more than just a gene regulated in astrocytes.

Sarkar, Devanand / Emdad, Luni / Lee, Seok-Geun / Yoo, Byoung Kwon / Su, Zao-Zhong / Fisher, Paul B

Cancer research

2009 Volume 69, Issue 22, Page(s) 8529–8535

Abstract: Since its original cloning by subtraction hybridization in 2002, it is now evident that Astrocyte elevated gene-1 (AEG-1) is a key contributor to the carcinogenic process in diverse organs. AEG-1 protein expression is elevated in advanced stages of many ... ...

Abstract	Since its original cloning by subtraction hybridization in 2002, it is now evident that Astrocyte elevated gene-1 (AEG-1) is a key contributor to the carcinogenic process in diverse organs. AEG-1 protein expression is elevated in advanced stages of many cancers, which correlates with poor survival. In specific cancers, such as breast and liver cancer, the AEG-1 gene itself is amplified, further supporting a seminal role in tumorigenesis. Overexpression and inhibition studies both in in vitro and in in vivo models reveal the importance of AEG-1 in regulating multiple physiologically and pathologically relevant processes including proliferation, invasion, metastasis, and gene expression. AEG-1 is a single-pass transmembrane protein with multiple nuclear localization signals and no known domains or motifs. Although pertinent roles of AEG-1 in the carcinogenic process are established, its potential function (promotion of metastasis only versus functioning as a bona fide oncogene) as well as localization (cell surface versus nucleus) remain areas requiring further clarification. The present review critically evaluates what is currently known about AEG-1 and provides new perspectives relative to this intriguing molecule that may provide a rational target for intervening in the cancer phenotype.
MeSH term(s)	Animals ; Astrocytes/physiology ; Cell Adhesion Molecules/physiology ; Humans ; Membrane Proteins ; Neoplasms/genetics ; RNA-Binding Proteins
Chemical Substances	Cell Adhesion Molecules ; MTDH protein, human ; Membrane Proteins ; RNA-Binding Proteins
Language	English
Publishing date	2009-11-10
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	1432-1
ISSN	1538-7445 ; 0008-5472
ISSN (online)	1538-7445
ISSN	0008-5472
DOI	10.1158/0008-5472.CAN-09-1846
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Article: Emerging roles of centrosomal amplification and genomic instability in cancer.

Emdad, Luni / Sarkar, Devanand / Su, Zao-Zhong / Fisher, Paul B

Frontiers in bioscience : a journal and virtual library

2005 Volume 10, Page(s) 728–742

Abstract: The carcinogenic process is multistep in terms of its etiology and multifactor in terms of its evolution. In this context, the temporal accumulation of multiple genetic changes during multistage carcinogenesis that can be mediated at least in part by ... ...

Abstract	The carcinogenic process is multistep in terms of its etiology and multifactor in terms of its evolution. In this context, the temporal accumulation of multiple genetic changes during multistage carcinogenesis that can be mediated at least in part by genomic instability may represent crucial components of tumor cell evolution. Evidence is accumulating indicating a close link between genomic instability and cancer initiation and progression. Neoplastic cells typically possess numerous genomic lesions, which may include sequence alterations (point mutations, small deletions, and insertions) and/or gross structural abnormalities in one or more chromosomes (large-scale deletions, rearrangements, gene amplifications). Furthermore karyotypic alterations, including whole chromosome loss or gain, ploidy changes (aneuploidy and polyploidy) and a variety of chromosome aberrations are common in tumor cells. Genomic instability also involves mitotic defects associated with centrosome abnormalities. However, the question of whether abnormal centrosomes cause genomic instability or develop secondary to other changes has not been conclusively resolved. In this review, the recent studies investigating genomic instability and aneuploidy in human cancer, centrosome amplification and the role of centrosomal duplication in chromosomal mis-segregetion, and genes implicated in regulating chromosome segregation, centrosomal amplification and progression in cancer cells are discussed.
MeSH term(s)	Aneuploidy ; Animals ; Antigens, Differentiation/genetics ; Cell Cycle ; Cell Cycle Proteins/genetics ; Centrosome/ultrastructure ; Disease Progression ; Genome ; Genomic Instability ; Humans ; Neoplasms/genetics ; Neoplasms/pathology ; Phosphorylation ; Protein Phosphatase 1
Chemical Substances	Antigens, Differentiation ; Cell Cycle Proteins ; PPP1R15A protein, human (EC 3.1.3.16) ; Protein Phosphatase 1 (EC 3.1.3.16)
Language	English
Publishing date	2005-01-01
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, P.H.S. ; Review
ZDB-ID	2141320-4
ISSN	1093-9946
ISSN	1093-9946
DOI	10.2741/1567
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Article ; Online: Astrocyte elevated gene-1 (AEG-1): A multifunctional regulator of normal and abnormal physiology.

Yoo, Byoung Kwon / Emdad, Luni / Lee, Seok-Geun / Su, Zao-zhong / Santhekadur, Prasanna / Chen, Dong / Gredler, Rachel / Fisher, Paul B / Sarkar, Devanand

Pharmacology & therapeutics

2011 Volume 130, Issue 1, Page(s) 1–8

Abstract: Since its initial identification and cloning in 2002, Astrocyte Elevated Gene-1 (AEG-1), also known as metadherin (MTDH), 3D3 and LYsine-RIch CEACAM1 co-isolated (LYRIC), has emerged as an important oncogene that is overexpressed in all cancers analyzed ... ...

Abstract	Since its initial identification and cloning in 2002, Astrocyte Elevated Gene-1 (AEG-1), also known as metadherin (MTDH), 3D3 and LYsine-RIch CEACAM1 co-isolated (LYRIC), has emerged as an important oncogene that is overexpressed in all cancers analyzed so far. Examination of a large cohort of patient samples representing diverse cancer indications has revealed progressive increase in AEG-1 expression with stages and grades of the disease and an inverse relationship between AEG-1 expression level and patient prognosis. AEG-1 functions as a bona fide oncogene by promoting transformation. In addition, it plays a significant role in invasion, metastasis, angiogenesis and chemoresistance, all important hallmarks of an aggressive cancer. AEG-1 is also implicated in diverse physiological and pathological processes, such as development, inflammation, neurodegeneration, migraine and Huntington's disease. AEG-1 is a highly basic protein with a transmembrane domain and multiple nuclear localization signals and it is present in the cell membrane, cytoplasm, nucleus, nucleolus and endoplasmic reticulum. In each location, AEG-1 interacts with specific proteins thereby modulating diverse intracellular processes the combination of which contributes to its pleiotrophic properties. The present review provides a snapshot of the current literature along with future perspectives on this unique molecule.
MeSH term(s)	Animals ; Biomarkers, Tumor ; Cell Adhesion Molecules/genetics ; Cell Adhesion Molecules/physiology ; Gene Expression ; Humans ; Intracellular Signaling Peptides and Proteins/metabolism ; Membrane Proteins ; Molecular Targeted Therapy ; Neoplasms/diagnosis ; Neoplasms/drug therapy ; Neoplasms/genetics ; Neoplasms/metabolism ; Oncogenes ; RNA-Binding Proteins ; Signal Transduction/physiology
Chemical Substances	Biomarkers, Tumor ; Cell Adhesion Molecules ; Intracellular Signaling Peptides and Proteins ; MTDH protein, human ; Membrane Proteins ; RNA-Binding Proteins
Language	English
Publishing date	2011-01-20
Publishing country	England
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	194735-7
ISSN	1879-016X ; 0163-7258
ISSN (online)	1879-016X
ISSN	0163-7258
DOI	10.1016/j.pharmthera.2011.01.008
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