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  1. Book: Physiologie

    Pape, Hans-Christian / Kurtz, Armin / Silbernagl, Stefan

    2023  

    Author's details herausgegeben von Hans-Christian Pape, Armin Kurtz, Stefan Silbernagl ; begründet von Rainer Klinke (†) und Stefan Silbernagl ; unter Mitarbeit von Markus Bleich [und weiteren]
    Keywords Medizinstudium ; Physiologie ; Physiologie-Lehrbuch ; Pathophysiologie ; Körperfunktionen ; physiologische Messgrößen ; physiologische Normalwerte ; 101 ; Hardcover, Softcover / Medizin/Nichtklinische Fächer
    Subject Humanphysiologie ; Mensch ; Körperfunktion
    Language German
    Size 1089 Seiten, Illustrationen, 27 cm x 19.5 cm
    Edition 10., vollständig überarbeitete Auflage
    Publisher Georg Thieme Verlag
    Publishing place Stuttgart ; New York
    Publishing country Germany
    Document type Book
    Accompanying material Zugang zu Online-Ausgabe über Code
    Old title Vorangegangen ist
    HBZ-ID HT030069771
    ISBN 978-3-13-244608-3 ; 3-13-244608-4 ; 9783132446106 ; 9783132446090 ; 3132446106 ; 3132446092
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2023 A 924 available for loan (reading room) Lesesaal EG
    Zugang zu Online-Ausgabe aus lizenzrechtlichen Gründen nicht verfügbar

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  2. Book: Physiologie

    Pape, Hans-Christian / Brenner, Bernhard / Klinke, Rainer

    2014  

    Author's details hrsg. von Hans-Christian Pape ... Mit Beitr. von Bernhard Brenner ... Begr. von Rainer Klinke
    Keywords Physiology ; Physiologie
    Subject Humanphysiologie ; Mensch ; Körperfunktion
    Language German
    Size 1024 S. : zahlr. Ill., graph. Darst.
    Edition 7., vollst. überarb. und erw. Aufl.
    Publisher Thieme
    Publishing place Stuttgart u.a.
    Publishing country Germany
    Document type Book
    Old title 6. Aufl. u. d. T. Klinke, Rainer: Physiologie
    HBZ-ID HT018353782
    ISBN 978-3-13-796007-2 ; 3-13-796007-X ; 9783131950772 ; 9783131514974 ; 9783131683175 ; 3131950773 ; 3131514973 ; 3131683171
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

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    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
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    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 on loan 0 Vormerkungen
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
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    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
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    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
    QT 104 S 6 available for loan (reading room) Lesesaal EG Lehrbuchsammlung
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    2009 A 10043 order for loan Magazin
    2014 A 4131 available for loan (reading room) Lesesaal EG

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  3. Book ; Online ; E-Book: Senior trauma patients

    Pape, Hans-Christoph / Kates, Stephen L. / Hierholzer, Christian / Bischoff-Ferrari, Heike

    an integrated approach

    2022  

    Abstract: This book describes a fully integrated approach to the management of elderly patients who are at risk of or suffer trauma, drawing on up-to-date knowledge from multiple specialties in recognition of the fact that the number and severity of comorbidities ... ...

    Author's details Hans-Christoph Pape, Stephen L. Kates, Christian Hierholzer, Heike A. Bischoff-Ferrari editors
    Abstract This book describes a fully integrated approach to the management of elderly patients who are at risk of or suffer trauma, drawing on up-to-date knowledge from multiple specialties in recognition of the fact that the number and severity of comorbidities in this population requires expertise in many fields. Readers will find comprehensive and detailed coverage of the prevention of complications related to aging, geriatric care concepts, specific treatments in acute care, including fracture stabilization and diagnostic procedures, and the surgical management of different types of fracture and soft tissue trauma. Intensive care management of the geriatric patient is also extensively addressed. Information is provided on diverse aspects of the physiology of ageing, and the coverage is completed by discussion of fracture care service models and specific models of rehabilitation and aftercare designed to prevent further falls and adverse late outcomes. The authors are a group of renowned experts from each of the relevant fields, and the book will be a valuable asset for surgeons, intensivists, geriatricians, gerontologists, and rehabilitation specialists. .
    Keywords Surgery ; Internal medicine ; Medical sciences ; Critical care medicine
    Language English
    Size 1 Online-Ressource(xiv, 365 Seiten), Illustrationen, Diagramme
    Publisher Springer
    Publishing place Cham
    Publishing country Switzerland
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT021299646
    ISBN 978-3-030-91483-7 ; 9783030914820 ; 3-030-91483-6 ; 3030914828
    DOI 10.1007/978-3-030-91483-7
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  4. Book: Physiologie

    Klinke, Rainer / Brenner, Bernhard / Pape, Hans-Christian / Kurtz, Armin / Silbernagl, Stefan

    2019  

    Author's details herausgegeben von Hans-Christian Pape, Armin Kurtz, Stefan Silbernagl ; begründet von Rainer Klinke (†) und Stefan Silbernagl ; mit Beiträgen von Bernhard Brenner (†) [und 29 weiteren]
    Keywords Physiologie ; Medizinstudium ; Physiologie-Lehrbuch ; Pathophysiologie ; physiologische Messgrößen ; physiologische Normalwerte ; Körperfunktionen ; Humanmedizin Vorklinik
    Subject Humanphysiologie ; Mensch ; Körperfunktion
    Subject code 610
    Language German
    Size 1028 Seiten, Illustrationen, Diagramme
    Edition 9., vollständig überarbeitete Auflage
    Publisher Georg Thieme Verlag
    Publishing place Stuttgart ; New York
    Publishing country Germany
    Document type Book
    HBZ-ID HT020164989
    ISBN 978-3-13-242391-6 ; 3-13-242391-2 ; 9783132423923 ; 9783132423930 ; 3132423920 ; 3132423939
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2019 A 1492 available for loan (reading room) Lesesaal EG
    Zugang zur Online-Ausgabe über Code aus lizenzrechtlichen Gründen nicht verfügbar

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  5. Book: Physiologie

    Klinke, Rainer / Brenner, Bernhard / Gay, Wolf-Rüdiger / Rothenburger, Astried / Pape, Hans-Christian / Kurtz, Armin / Silbernagl, Stefan

    2018  

    Author's details herausgegeben von Hans-Christian Pape, Armin Kurtz, Stefan Silbernagl ; begründet von Rainer Klinke und Stefan Silbernagl ; mit Beiträgen von Bernhard Brenner [und vielen anderen] ; Illustrationen von Rüdiger Gay und Astried Rothenburger
    Keywords Physiology ; Physiologie
    Subject Humanphysiologie ; Mensch ; Körperfunktion
    Language German
    Size 1024 Seiten, Illustrationen
    Edition 8., unveränderte Auflage
    Publisher Georg Thieme Verlag
    Publishing place Stuttgart ; New York
    Publishing country Germany
    Document type Book
    Note Zugang zur Online-Ausgabe über Code
    HBZ-ID HT019603467
    ISBN 978-3-13-242387-9 ; 9783132423923 ; 9783132423930 ; 3-13-242387-4 ; 3132423920 ; 3132423939
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    QT 104 S 6 on loan 0 Vormerkungen
    Zugang zur Online-Ausgabe über Code aus lizenzrechtlichen Gründen nicht verfügbar

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  6. Article ; Online: Regional specificity of cortico-thalamic coupling strength and directionality during waxing and waning of spike and wave discharges

    Annika Lüttjohann / Hans-Christian Pape

    Scientific Reports, Vol 9, Iss 1, Pp 1-

    2019  Volume 11

    Abstract: Abstract Spike-wave discharges (SWDs) on the EEG during absence epilepsy are waxing and waning stages of corticothalamic hypersynchrony. While the somatosensory cortex contains an epileptic focus, the role of thalamic nuclei in SWD generation is debated. ...

    Abstract Abstract Spike-wave discharges (SWDs) on the EEG during absence epilepsy are waxing and waning stages of corticothalamic hypersynchrony. While the somatosensory cortex contains an epileptic focus, the role of thalamic nuclei in SWD generation is debated. Here we assess the contribution of distinct thalamic nuclei through multiple-site unit recordings in a genetic rat model of absence epilepsy and cross-correlation analysis, revealing coupling strength and directionality of neuronal activity at high temporal resolution. Corticothalamic coupling increased and decreased during waxing and waning of SWD, respectively. A cortical drive on either sensory or higher order thalamic nuclei distinguished between onset and offset of SWD, respectively. Intrathalamic coupling steadily increased during maintained SWD activity, peaked at SWD offset, and subsequently displayed a sharp decline to baseline. The peak in intrathalamic coupling coincided with a sharp increase in coupling strength between reticular thalamic nucleus and somatosensory cortex. This increased influence of the inhibitory reticular thalamic nucleus is suggested to serve as a break for SWD activity. Overall, the data extend the cortical focus theory of absence epilepsy by identifying a regionally specific cortical lead over distinct thalamic nuclei, particularly also during waning of generalized epileptic discharges, thereby revealing a potential window and location for intervention.
    Keywords Medicine ; R ; Science ; Q
    Language English
    Publishing date 2019-02-01T00:00:00Z
    Publisher Nature Publishing Group
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

    Full text online

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  7. Article ; Online: Regional specificity of cortico-thalamic coupling strength and directionality during waxing and waning of spike and wave discharges.

    Lüttjohann, Annika / Pape, Hans-Christian

    Scientific reports

    2019  Volume 9, Issue 1, Page(s) 2100

    Abstract: Spike-wave discharges (SWDs) on the EEG during absence epilepsy are waxing and waning stages of corticothalamic hypersynchrony. While the somatosensory cortex contains an epileptic focus, the role of thalamic nuclei in SWD generation is debated. Here we ... ...

    Abstract Spike-wave discharges (SWDs) on the EEG during absence epilepsy are waxing and waning stages of corticothalamic hypersynchrony. While the somatosensory cortex contains an epileptic focus, the role of thalamic nuclei in SWD generation is debated. Here we assess the contribution of distinct thalamic nuclei through multiple-site unit recordings in a genetic rat model of absence epilepsy and cross-correlation analysis, revealing coupling strength and directionality of neuronal activity at high temporal resolution. Corticothalamic coupling increased and decreased during waxing and waning of SWD, respectively. A cortical drive on either sensory or higher order thalamic nuclei distinguished between onset and offset of SWD, respectively. Intrathalamic coupling steadily increased during maintained SWD activity, peaked at SWD offset, and subsequently displayed a sharp decline to baseline. The peak in intrathalamic coupling coincided with a sharp increase in coupling strength between reticular thalamic nucleus and somatosensory cortex. This increased influence of the inhibitory reticular thalamic nucleus is suggested to serve as a break for SWD activity. Overall, the data extend the cortical focus theory of absence epilepsy by identifying a regionally specific cortical lead over distinct thalamic nuclei, particularly also during waning of generalized epileptic discharges, thereby revealing a potential window and location for intervention.
    MeSH term(s) Action Potentials/physiology ; Animals ; Cerebral Cortex/physiology ; Disease Models, Animal ; Electroencephalography/methods ; Epilepsy, Absence/physiopathology ; Male ; Neural Pathways ; Neurons/physiology ; Rats ; Rats, Wistar ; Somatosensory Cortex/physiology ; Thalamic Nuclei/physiology ; Thalamus/physiology
    Language English
    Publishing date 2019-02-14
    Publishing country England
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 2615211-3
    ISSN 2045-2322 ; 2045-2322
    ISSN (online) 2045-2322
    ISSN 2045-2322
    DOI 10.1038/s41598-018-37985-7
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Article ; Online: Neuropeptide S-Mediated Modulation of Prepulse Inhibition Depends on Age, Gender, Stimulus-Timing, and Attention

    Wei Si / Xiaobin Liu / Hans-Christian Pape / Rainer K. Reinscheid

    Pharmaceuticals, Vol 14, Iss 489, p

    2021  Volume 489

    Abstract: Conflicting reports about the role of neuropeptide S (NPS) in animal models of psychotic-like behavior and inconsistent results from human genetic studies seeking potential associations with schizophrenia prompted us to reevaluate the effects of NPS in ... ...

    Abstract Conflicting reports about the role of neuropeptide S (NPS) in animal models of psychotic-like behavior and inconsistent results from human genetic studies seeking potential associations with schizophrenia prompted us to reevaluate the effects of NPS in the prepulse inhibition (PPI) paradigm in mice. Careful examination of NPS receptor (NPSR1) knockout mice at different ages revealed that PPI deficits are only expressed in young male knockout animals (<12 weeks of age), that can be replicated in NPS precursor knockout mice and appear strain-independent, but are absent in female mice. PPI deficits can be aggravated by MK-801 and alleviated by clozapine. Importantly, treatment of wildtype mice with a centrally-active NPSR1 antagonist was able to mimic PPI deficits. PPI impairment in young male NPSR1 and NPS knockout mice may be caused by attentional deficits that are enhanced by increasing interstimulus intervals. Our data reveal a substantial NPS-dependent developmental influence on PPI performance and confirm a significant role of attentional processes for sensory-motor gating. Through its influence on attention and arousal, NPS appears to positively modulate PPI in young animals, whereas compensatory mechanisms may alleviate NPS-dependent deficits in older mice.
    Keywords NPSR1 ; NPS ; knockout mouse ; PPI ; schizophrenia ; clozapine ; Medicine ; R ; Pharmacy and materia medica ; RS1-441
    Language English
    Publishing date 2021-05-01T00:00:00Z
    Publisher MDPI AG
    Document type Article ; Online
    Database BASE - Bielefeld Academic Search Engine (life sciences selection)

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  9. Article ; Online: Stimulation of 5-HT receptors in anterodorsal BNST guides fear to predictable and unpredictable threat.

    Hessel, Margarita / Pape, Hans-Christian / Seidenbecher, Thomas

    European neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology

    2020  Volume 39, Page(s) 56–69

    Abstract: Through pharmacological manipulation of the serotonergic (5-Hydroxytryptamin, 5-HT) system, combined with behavioral analysis, we tested the hypothesis that fear responses to predictable and unpredictable threat are regulated through stimulation of 5-HT ... ...

    Abstract Through pharmacological manipulation of the serotonergic (5-Hydroxytryptamin, 5-HT) system, combined with behavioral analysis, we tested the hypothesis that fear responses to predictable and unpredictable threat are regulated through stimulation of 5-HT receptors (5-HT-R) in the anterodorsal section of the bed nucleus of the stria terminalis (adBNST). Local adBNST application of 5-HT1A-R antagonist WAY100635 and 5-HT1B-R antagonist NAS-181 before fear retrieval enhanced freezing, 24 h after predictable fear conditioning. In contrast, increased fear responses to unpredictable threat were blocked by 5-HT1A-R agonist Buspirone (given before conditioning or retrieval) and 5-HT1B-R agonist CP-94253 (applied before training). Prolonged fear responses were also blocked by local application of the 5-HT2A-R antagonist R-96544 before fear retrieval, and conversely, local application of the 5-HT2A-R agonist NBOH-2C-CN hydrochloride before fear retrieval enhanced freezing 24 h after predictable conditioning, indicating augmented fear responses. Activation of inhibitory 5-HT1A- or 5-HT1B-Rs and the blockade of the excitatory 5-HT2A-R before unpredictable fear conditioning significantly reduced freezing during retrieval. The results from this study suggest that modulation of inhibitory 5-HT1A/1B-R and/or excitatory 5-HT2A-R activity in the adBNST may represent potential targets for the development of new treatment strategies in anxiety disorders. In addition, this study supports the validity and reliability of the mouse model of modulated fear to predictable and unpredictable threats to study mechanisms of fear and anxiety in combination with pharmacological manipulations.
    MeSH term(s) Animals ; Fear/drug effects ; Fear/physiology ; Fear/psychology ; Injections, Intraventricular ; Male ; Mice ; Mice, Inbred C57BL ; Receptors, Serotonin/metabolism ; Septal Nuclei/drug effects ; Septal Nuclei/metabolism ; Serotonin 5-HT1 Receptor Agonists/administration & dosage ; Serotonin 5-HT1 Receptor Antagonists/administration & dosage ; Serotonin 5-HT2 Receptor Agonists/administration & dosage ; Serotonin 5-HT2 Receptor Antagonists/administration & dosage
    Chemical Substances Receptors, Serotonin ; Serotonin 5-HT1 Receptor Agonists ; Serotonin 5-HT1 Receptor Antagonists ; Serotonin 5-HT2 Receptor Agonists ; Serotonin 5-HT2 Receptor Antagonists
    Language English
    Publishing date 2020-08-29
    Publishing country Netherlands
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 1082947-7
    ISSN 1873-7862 ; 0924-977X
    ISSN (online) 1873-7862
    ISSN 0924-977X
    DOI 10.1016/j.euroneuro.2020.08.006
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 3288: Show issues Location:
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    bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
    Jg. 1995 - 2021: Lesesall (2.OG)
    ab Jg. 2022: Lesesaal (EG)
    Zs.MO 630: Show issues

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  10. Article: Neuropeptide S-Mediated Modulation of Prepulse Inhibition Depends on Age, Gender, Stimulus-Timing, and Attention.

    Si, Wei / Liu, Xiaobin / Pape, Hans-Christian / Reinscheid, Rainer K

    Pharmaceuticals (Basel, Switzerland)

    2021  Volume 14, Issue 5

    Abstract: Conflicting reports about the role of neuropeptide S (NPS) in animal models of psychotic-like behavior and inconsistent results from human genetic studies seeking potential associations with schizophrenia prompted us to reevaluate the effects of NPS in ... ...

    Abstract Conflicting reports about the role of neuropeptide S (NPS) in animal models of psychotic-like behavior and inconsistent results from human genetic studies seeking potential associations with schizophrenia prompted us to reevaluate the effects of NPS in the prepulse inhibition (PPI) paradigm in mice. Careful examination of NPS receptor (NPSR1) knockout mice at different ages revealed that PPI deficits are only expressed in young male knockout animals (<12 weeks of age), that can be replicated in NPS precursor knockout mice and appear strain-independent, but are absent in female mice. PPI deficits can be aggravated by MK-801 and alleviated by clozapine. Importantly, treatment of wildtype mice with a centrally-active NPSR1 antagonist was able to mimic PPI deficits. PPI impairment in young male NPSR1 and NPS knockout mice may be caused by attentional deficits that are enhanced by increasing interstimulus intervals. Our data reveal a substantial NPS-dependent developmental influence on PPI performance and confirm a significant role of attentional processes for sensory-motor gating. Through its influence on attention and arousal, NPS appears to positively modulate PPI in young animals, whereas compensatory mechanisms may alleviate NPS-dependent deficits in older mice.
    Language English
    Publishing date 2021-05-20
    Publishing country Switzerland
    Document type Journal Article
    ZDB-ID 2193542-7
    ISSN 1424-8247
    ISSN 1424-8247
    DOI 10.3390/ph14050489
    Database MEDical Literature Analysis and Retrieval System OnLINE

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