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Book: Pneumocystis carinii

Sattler, Fred R.

(Baillière's clinical infectious diseases ; 2,3)

1995

Author's details	F. R. Sattler ..., guest ed
Series title	Baillière's clinical infectious diseases ; 2,3
Collection
Keywords	Pneumocystis carinii ; Pneumocystis carinii Infections ; Interstitielle plasmazelluläre Pneumonie
Subject	Pneumozystose
Language	English
Size	X S., S. 409 - 576 : graph. Darst.
Publisher	Baillière Tindall
Publishing place	London u.a.
Publishing country	Great Britain
Document type	Book
HBZ-ID	HT006832433
ISBN	0-7020-2086-9 ; 978-0-7020-2086-5
Database	Catalogue ZB MED Medicine, Health

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Article ; Online: Growth hormone in the aging male.

Sattler, Fred R

Best practice & research. Clinical endocrinology & metabolism

2013 Volume 27, Issue 4, Page(s) 541–555

Abstract: Secretion of growth hormone (GH) and IGF-1 levels decline during advancing years-of-life. These changes (somatopause) are associated with loss of vitality, muscle mass, physical function, together with the occurrence of frailty, central adiposity, ... ...

Abstract	Secretion of growth hormone (GH) and IGF-1 levels decline during advancing years-of-life. These changes (somatopause) are associated with loss of vitality, muscle mass, physical function, together with the occurrence of frailty, central adiposity, cardiovascular complications, and deterioration of mental function. For GH treatment to be considered for anti-aging, improved longevity, organ-specific function, or quality of life should be demonstrable. A limited number of controlled studies suggest that GH supplementation in older men increases lean mass by ∼2 kg with similar reductions in fat mass. There is little evidence that GH treatment improves muscle strength and performance (e.g. walking speed or ability to climb stairs) or quality of life. The GHRH agonist (tesamorelin) restores normal GH pulsatility and amplitude, selectively reduces visceral fat, intima media thickness and triglycerides, and improves cognitive function in older persons. This report critically reviews the potential for GH augmentation during aging with emphasis on men since women appear more resistant to treatment.
MeSH term(s)	Aged ; Aged, 80 and over ; Aging/physiology ; Animals ; Atherosclerosis/prevention & control ; Carbohydrate Metabolism/drug effects ; Carotid Intima-Media Thickness ; Cognition/drug effects ; Growth Hormone-Releasing Hormone/analogs & derivatives ; Growth Hormone-Releasing Hormone/therapeutic use ; Human Growth Hormone/deficiency ; Human Growth Hormone/genetics ; Human Growth Hormone/therapeutic use ; Humans ; Insulin-Like Growth Factor I/pharmacology ; Lipid Metabolism/drug effects ; Longevity/drug effects ; Male ; Muscles/physiology ; Sarcopenia/complications ; Sarcopenia/prevention & control
Chemical Substances	Human Growth Hormone (12629-01-5) ; Insulin-Like Growth Factor I (67763-96-6) ; Growth Hormone-Releasing Hormone (9034-39-3) ; tesamorelin (MQG94M5EEO)
Language	English
Publishing date	2013-06-18
Publishing country	Netherlands
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, U.S. Gov't, Non-P.H.S. ; Review
ZDB-ID	2052339-7
ISSN	1878-1594 ; 1532-1908 ; 1521-690X
ISSN (online)	1878-1594 ; 1532-1908
ISSN	1521-690X
DOI	10.1016/j.beem.2013.05.003
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Article: Growth hormone in the aging male

Sattler, Fred R

Best Practice & Research Clinical Endocrinology & Metabolism. 2013 Aug., v. 27, no. 4

2013

Abstract	Secretion of growth hormone (GH) and IGF-1 levels decline during advancing years-of-life. These changes (somatopause) are associated with loss of vitality, muscle mass, physical function, together with the occurrence of frailty, central adiposity, cardiovascular complications, and deterioration of mental function. For GH treatment to be considered for anti-aging, improved longevity, organ-specific function, or quality of life should be demonstrable. A limited number of controlled studies suggest that GH supplementation in older men increases lean mass by ∼2 kg with similar reductions in fat mass. There is little evidence that GH treatment improves muscle strength and performance (e.g. walking speed or ability to climb stairs) or quality of life. The GHRH agonist (tesamorelin) restores normal GH pulsatility and amplitude, selectively reduces visceral fat, intima media thickness and triglycerides, and improves cognitive function in older persons. This report critically reviews the potential for GH augmentation during aging with emphasis on men since women appear more resistant to treatment.
Keywords	adiposity ; agonists ; cognition ; elderly ; insulin-like growth factor I ; longevity ; males ; men ; muscle strength ; muscles ; quality of life ; secretion ; somatoliberin ; somatotropin ; triacylglycerols ; visceral fat ; walking ; women
Language	English
Dates of publication	2013-08
Size	p. 541-555.
Publishing place	Elsevier Ltd
Document type	Article
ZDB-ID	1493559-4
ISSN	1521-690X
ISSN	1521-690X
DOI	10.1016/j.beem.2013.05.003
Database	NAL-Catalogue (AGRICOLA)

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Article: Pathogenesis and treatment of lipodystrophy: what clinicians need to know.

Sattler, Fred R

Topics in HIV medicine : a publication of the International AIDS Society, USA

2008 Volume 16, Issue 4, Page(s) 127–133

Abstract: ... This article summarizes a presentation on lipodystrophy made by Fred R. Sattler, MD, at an International AIDS ...

Abstract	The pathogenesis of lipodystrophy in HIV-infected patients is likely multifactorial, involving effects of antiretroviral medications, HIV itself, as well as genetic and other host factors. Protease inhibitors have been associated with fat accumulation, and the nucleoside analogue reverse transcriptase inhibitors (nRTIs) stavudine, didanosine, and zidovudine have been associated with fat loss (lipoatrophy). Strategies that have met with some success in reducing central fat accumulation include treatment with growth hormone or growth hormone-releasing hormone. Strategies that have met with some success for lipoatrophy include switching from nRTIs associated with lipoatrophy or starting treatment with regimens that include drugs associated with lower risk of lipoatrophy (tenofovir, abacavir, lamivudine, emtricitabine). This article summarizes a presentation on lipodystrophy made by Fred R. Sattler, MD, at an International AIDS Society--USA Continuing Medical Education course in Washington, DC, in May 2008. The original presentation is available as a Webcast at www.iasusa.org.
MeSH term(s)	Anti-HIV Agents/adverse effects ; Anti-HIV Agents/therapeutic use ; HIV Infections/complications ; HIV Infections/drug therapy ; HIV-Associated Lipodystrophy Syndrome/drug therapy ; HIV-Associated Lipodystrophy Syndrome/etiology ; HIV-Associated Lipodystrophy Syndrome/prevention & control ; Humans
Chemical Substances	Anti-HIV Agents
Language	English
Publishing date	2008-10
Publishing country	United States
Document type	Journal Article ; Review
ISSN	1542-8826
ISSN	1542-8826
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: Inflammation and complications of HIV disease.

Dubé, Michael P / Sattler, Fred R

The Journal of infectious diseases

2010 Volume 201, Issue 12, Page(s) 1783–1785

MeSH term(s)	AIDS Dementia Complex/epidemiology ; AIDS-Related Opportunistic Infections/epidemiology ; Anti-HIV Agents/adverse effects ; Blood Coagulation Disorders/epidemiology ; Endothelium/pathology ; HIV Infections/complications ; HIV Infections/drug therapy ; HIV Infections/pathology ; HIV-Associated Lipodystrophy Syndrome/epidemiology ; Humans ; Inflammation/pathology ; Lymphoma, AIDS-Related/epidemiology
Chemical Substances	Anti-HIV Agents
Language	English
Publishing date	2010-06-15
Publishing country	United States
Document type	Comment ; Editorial
ZDB-ID	3019-3
ISSN	1537-6613 ; 0022-1899
ISSN (online)	1537-6613
ISSN	0022-1899
DOI	10.1086/652751
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Article ; Online: Feasibility of quantifying change in immune white cells in abdominal adipose tissue in response to an immune modulator in clinical obesity.

Sattler, Fred R / Mert, Melissa / Sankaranarayanan, Ishwarya / Mack, Wendy J / Galle-Treger, Lauriane / Gonzalez, Evelyn / Baronikian, Lilit / Lee, Kyuwan / Jahani, Pedram Shafiei / Hodis, Howard N / Dieli-Conwright, Christina / Akbari, Omid

PloS one

2020 Volume 15, Issue 9, Page(s) e0237496

Abstract: Background: Obesity is often associated with inflammation in adipose tissue (AT) with release of mediators of atherogenesis. We postulated that it would be feasible to collect sufficient abdominal AT to quantify changes in a broad array of adaptive and ... ...

Abstract	Background: Obesity is often associated with inflammation in adipose tissue (AT) with release of mediators of atherogenesis. We postulated that it would be feasible to collect sufficient abdominal AT to quantify changes in a broad array of adaptive and innate mononuclear white cells in obese non-diabetic adults in response to a dipeptidyl protease inhibitor (DPP4i), known to inhibit activation of immune white cells. Methods: Adults 18-55 years-of-age were screened for abdominal obesity and insulin resistance or impaired glucose tolerance but without known inflammatory conditions. Twenty-one eligible participants consented for study and were randomized 3:1 to receive sitagliptin (DPP4i) at 100mg or matching placebo daily for 28 days. Abdominal AT collected by percutaneous biopsy and peripheral blood mononuclear cell fractions were evaluated before and after treatment; plasma was stored for batch testing. Results: Highly sensitive C-reactive protein, a global marker of inflammation, was not elevated in the study population. Innate lymphoid cells (ILC) type 3 (ILC-3) in abdominal AT decreased with active treatment compared with placebo (p = 0.04). Other immune white cells in AT and peripheral blood mononuclear cell (PBMC) fractions did not change with treatment compared to placebo (p>0.05); although ILC-2 declined in PBMCs (p = 0.007) in the sitagliptin treatment group. Two circulating biomarkers of atherogenesis, interferon-inducible protein-10 (IP-10) and sCD40L declined in plasma (p = 0.02 and p = 0.07, respectively) in the active treatment group, providing indirect validation of a net reduction in inflammation. Conclusions: In this pilot study, two cell types of the innate lymphoid system, ILC-3 in AT and ILC-2 PBMCs declined during treatment and as did circulating biomarkers of atherogenesis. Changes in other immune cells were not demonstrable. The study showed that sufficient abdominal AT could be obtained to quantify white cells of both innate and adaptive immunity and to demonstrate changes during therapy with an immune inhibitor. Trial registration: ClinicalTrials.gov identifier (NCT number): NCT02576.
MeSH term(s)	Abdominal Fat/pathology ; Adult ; Biomarkers/blood ; Feasibility Studies ; Female ; Flow Cytometry ; Humans ; Immunity, Innate ; Leukocytes, Mononuclear/pathology ; Lymphocytes/pathology ; Male ; Middle Aged ; Obesity/blood ; Obesity/immunology ; Sitagliptin Phosphate/pharmacology ; Treatment Outcome
Chemical Substances	Biomarkers ; Sitagliptin Phosphate (TS63EW8X6F)
Language	English
Publishing date	2020-09-03
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ISSN	1932-6203
ISSN (online)	1932-6203
DOI	10.1371/journal.pone.0237496
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: Feasibility of quantifying change in immune white cells in abdominal adipose tissue in response to an immune modulator in clinical obesity.

Fred R Sattler / Melissa Mert / Ishwarya Sankaranarayanan / Wendy J Mack / Lauriane Galle-Treger / Evelyn Gonzalez / Lilit Baronikian / Kyuwan Lee / Pedram Shafiei Jahani / Howard N Hodis / Christina Dieli-Conwright / Omid Akbari

PLoS ONE, Vol 15, Iss 9, p e

2020 Volume 0237496

Abstract: Background Obesity is often associated with inflammation in adipose tissue (AT) with release of mediators of atherogenesis. We postulated that it would be feasible to collect sufficient abdominal AT to quantify changes in a broad array of adaptive and ... ...

Abstract	Background Obesity is often associated with inflammation in adipose tissue (AT) with release of mediators of atherogenesis. We postulated that it would be feasible to collect sufficient abdominal AT to quantify changes in a broad array of adaptive and innate mononuclear white cells in obese non-diabetic adults in response to a dipeptidyl protease inhibitor (DPP4i), known to inhibit activation of immune white cells. Methods Adults 18-55 years-of-age were screened for abdominal obesity and insulin resistance or impaired glucose tolerance but without known inflammatory conditions. Twenty-one eligible participants consented for study and were randomized 3:1 to receive sitagliptin (DPP4i) at 100mg or matching placebo daily for 28 days. Abdominal AT collected by percutaneous biopsy and peripheral blood mononuclear cell fractions were evaluated before and after treatment; plasma was stored for batch testing. Results Highly sensitive C-reactive protein, a global marker of inflammation, was not elevated in the study population. Innate lymphoid cells (ILC) type 3 (ILC-3) in abdominal AT decreased with active treatment compared with placebo (p = 0.04). Other immune white cells in AT and peripheral blood mononuclear cell (PBMC) fractions did not change with treatment compared to placebo (p>0.05); although ILC-2 declined in PBMCs (p = 0.007) in the sitagliptin treatment group. Two circulating biomarkers of atherogenesis, interferon-inducible protein-10 (IP-10) and sCD40L declined in plasma (p = 0.02 and p = 0.07, respectively) in the active treatment group, providing indirect validation of a net reduction in inflammation. Conclusions In this pilot study, two cell types of the innate lymphoid system, ILC-3 in AT and ILC-2 PBMCs declined during treatment and as did circulating biomarkers of atherogenesis. Changes in other immune cells were not demonstrable. The study showed that sufficient abdominal AT could be obtained to quantify white cells of both innate and adaptive immunity and to demonstrate changes during therapy ...
Keywords	Medicine ; R ; Science ; Q
Subject code	610
Language	English
Publishing date	2020-01-01T00:00:00Z
Publisher	Public Library of Science (PLoS)
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article: Biomarkers Associated with Death After Initiating Treatment for Tuberculosis and HIV in Patients with Very Low CD

Sattler, Fred R / Chelliah, Daniel / Wu, Xingye / Sanchez, Alejandro / Kendall, Michelle A / Hogg, Evelyn / Lagat, David / Lalloo, Umesh / Veloso, Valdilea / Havlir, Diane V / Landay, Alan

Pathogens & immunity

2018 Volume 3, Issue 1, Page(s) 46–62

Abstract: Background: The risk of short-term death for treatment naive patients dually infected with : Methods: We utilized a retrospective case-cohort design among participants of the ACTG A5221 study who had baseline CD4 < 50 cells/mm: Results: The case- ... ...

Abstract	Background: The risk of short-term death for treatment naive patients dually infected with Methods: We utilized a retrospective case-cohort design among participants of the ACTG A5221 study who had baseline CD4 < 50 cells/mm Results: The case-cohort sample was similar to the 282 participants within the parent cohort with CD4 <50 cells/mm Conclusions: Unlike patients only infected with
Language	English
Publishing date	2018-04-26
Publishing country	United States
Document type	Journal Article
ISSN	2469-2964
ISSN	2469-2964
DOI	10.20411/pai.v3i1.235
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Article ; Online: Update of penetrance estimates in Birt-Hogg-Dubé syndrome.

Bruinsma, Fiona Jane / Dowty, James G / Win, Aung Ko / Goddard, Laura C / Agrawal, Prachi / Attina', Domenico / Bissada, Nabil / De Luise, Monica / Eisen, Daniel B / Furuya, Mitsuko / Gasparre, Giuseppe / Genuardi, Maurizio / Gerdes, Anne-Marie / Hansen, Thomas Van Overeem / Houweling, Arjan C / Johannesma, Paul Christiaan / Lencastre, André / Lim, Derek / Lindor, Noralane M /

Journal of medical genetics

2023 Volume 60, Issue 4, Page(s) 317–326

Abstract: Background: Birt-Hogg-Dubé (BHD) syndrome is a rare genetic syndrome caused by pathogenic or likely pathogenic germline variants in the : Methods: A comprehensive review was conducted to identify studies that had recruited families carrying ... ...

Abstract	Background: Birt-Hogg-Dubé (BHD) syndrome is a rare genetic syndrome caused by pathogenic or likely pathogenic germline variants in the Methods: A comprehensive review was conducted to identify studies that had recruited families carrying pathogenic or likely pathogenic variants in Results: Our final dataset contained 204 families that were informative for at least one manifestation of BHD (67 families informative for skin manifestations, 63 for lung, 88 for renal carcinoma and 29 for polyps). By age 70 years, male carriers of the Conclusions: These updated penetrance estimates, based on a large number of families, are important for the genetic counselling and clinical management of BHD syndrome.
MeSH term(s)	Humans ; Male ; Female ; Aged ; Birt-Hogg-Dube Syndrome/genetics ; Birt-Hogg-Dube Syndrome/pathology ; Penetrance ; Colonic Polyps ; Proto-Oncogene Proteins/genetics ; Tumor Suppressor Proteins/genetics ; Kidney Neoplasms/epidemiology ; Kidney Neoplasms/genetics ; Carcinoma, Renal Cell/epidemiology ; Carcinoma, Renal Cell/genetics
Chemical Substances	Proto-Oncogene Proteins ; Tumor Suppressor Proteins
Language	English
Publishing date	2023-02-27
Publishing country	England
Document type	Review ; Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	220881-7
ISSN	1468-6244 ; 0022-2593
ISSN (online)	1468-6244
ISSN	0022-2593
DOI	10.1136/jmg-2022-109104
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Article ; Online: Acute Response of PGC-1α and IGF-1 Isoforms to Maximal Eccentric Exercise in Skeletal Muscle of Postmenopausal Women.

Dieli-Conwright, Christina M / Kiwata, Jacqueline L / Tuzon, Creighton T / Spektor, Tanya M / Sattler, Fred R / Rice, Judd C / Schroeder, Edward Todd

Journal of strength and conditioning research

2016 Volume 30, Issue 4, Page(s) 1161–1170

Abstract: ... associated with reduced mRNA expression of the catabolic gene myostatin (R = -0.88, p < 0.01), whereas MGF ... mRNA expression was associated with reduced mRNA expression of the catabolic gene FOXO3A (R = -0.81, p ...

Abstract	PGC-1α4, a novel isoform of the transcriptional coactivator PGC-1α, was recently postulated to modulate the expression of anabolic and catabolic genes and therefore regulate skeletal muscle hypertrophy. Resting levels of PGC-1α4 messenger RNA (mRNA) expression were found to increase in healthy adults after resistance training. However, the acute effect of resistance exercise (RE) on PGC-1α4 expression in populations prone to progressive muscle loss, such as postmenopausal women, has not been evaluated. Here, we investigated alterations in mRNA expression of PGC-1α4 and PGC-1α1, a regulator of muscle oxidative changes, in postmenopausal women after high-intensity eccentric RE and analyzed these findings with respect to changes in insulin-like growth factor (IGF)-1 and catabolic gene expression. Nine postmenopausal women (age, 57.9 ± 3.2 years) performed 10 sets of 10 maximal eccentric repetitions of single-leg extension with 20-second rest periods between sets. Muscle biopsies were obtained from the vastus lateralis of the exercised leg before and 4 hours after the RE bout with mRNA expression determined by quantitative real-time polymerase chain reaction. No significant changes in the mRNA expression of either PGC-1α isoform were observed after acute eccentric RE (p > 0.05). IGF-1Ea mRNA expression significantly increased (p ≤ 0.05), whereas IGF-1Eb and mechano-growth factor (MGF) did not significantly change (p > 0.05). PGC-1α4 mRNA expression was associated with reduced mRNA expression of the catabolic gene myostatin (R = -0.88, p < 0.01), whereas MGF mRNA expression was associated with reduced mRNA expression of the catabolic gene FOXO3A (R = -0.81, p ≤ 0.05). These data demonstrate an attenuated response of PGC-1α isoforms to an acute bout of maximal eccentric exercise with short rest periods in postmenopausal women.
MeSH term(s)	Aged ; Biopsy ; Female ; Forkhead Box Protein O3/genetics ; Forkhead Box Protein O3/metabolism ; Gene Expression ; Humans ; Insulin-Like Growth Factor I/genetics ; Insulin-Like Growth Factor I/metabolism ; Middle Aged ; Muscle, Skeletal/metabolism ; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha/genetics ; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha/metabolism ; Postmenopause/metabolism ; Protein Isoforms/genetics ; Protein Isoforms/metabolism ; RNA, Messenger/metabolism ; Resistance Training
Chemical Substances	FOXO3 protein, human ; Forkhead Box Protein O3 ; PPARGC1A protein, human ; Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha ; Protein Isoforms ; RNA, Messenger ; Insulin-Like Growth Factor I (67763-96-6)
Language	English
Publishing date	2016-04
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural
ZDB-ID	1156349-7
ISSN	1533-4287 ; 1064-8011
ISSN (online)	1533-4287
ISSN	1064-8011
DOI	10.1519/JSC.0000000000001171
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