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  1. Book: Heptinstall's pathology of the kidney

    Jennette, J. Charles / D'Agati, Vivette

    2024  

    Title variant Pathology of the kidney
    Author's details editors J. Charles Jennette, MD, Vivette D. D'Agati MD, associate editors Agnes B. Fogo, MD, Volker Nickeleit, MD, M. Barry Stokes, MBBCh
    MeSH term(s) Kidney Diseases/pathology
    Language English
    Size xix, 1634 Seiten, Illustrationen
    Edition Eight edition
    Publisher Wolters Kluwer
    Publishing place Philadelphia, PA
    Publishing country United States
    Document type Book
    Accompanying material Zugang zur Online-Ausgabe über Code
    HBZ-ID HT021794059
    ISBN 978-1-9751-6153-8 ; 1-9751-6153-X
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2023 A 966 available for loan (reading room) Lesesaal EG
    Zugang zur Online-Ausgabe über Code aus lizenzrechtlichen Gründen nicht verfügbar

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  2. Book: Heptinstall's pathology of the kidney / 2

    Heptinstall, Robert H. / Jennette, J. Charles

    2015  

    Author's details ed. J. Charles Jennette
    Collection Heptinstall's pathology of the kidney
    Language English
    Size XVIII S., S. 715 - 1497, I-24 S. : zahlr. Ill., graph. Darst.
    Publisher Lippincott-Raven
    Publishing place Philadelphia u.a.
    Publishing country United States
    Document type Book
    HBZ-ID HT018431335
    ISBN 978-1-4511-4411-6 ; 1-4511-4411-3
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2014 A 3973 available for loan (reading room) Lesesaal EG

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  3. Book: Heptinstall's pathology of the kidney / 1

    Heptinstall, Robert H. / Jennette, J. Charles

    2015  

    Author's details ed. J. Charles Jennette
    Collection Heptinstall's pathology of the kidney
    Language English
    Size XVIII, 713, I-24 S. : zahlr. Ill., graph. Darst.
    Publisher Lippincott-Raven
    Publishing place Philadelphia u.a.
    Publishing country United States
    Document type Book
    Accompanying material Zugang zu Internetmaterial über Code
    HBZ-ID HT018431301
    ISBN 978-1-4511-4411-6 ; 1-4511-4411-3
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2014 A 3972 available for loan (reading room) Lesesaal EG

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  4. Book: Heptinstall's pathology of the kidney / 1

    Heptinstall, Robert H. / Jennette, J. Charles

    2007  

    Author's details ed. J. Charles Jennette
    Collection Heptinstall's pathology of the kidney
    Language English
    Size X, 852, 48 S. : zahlr. Ill., graph. Darst.
    Edition 6. ed.
    Publisher Lippincott-Raven
    Publishing place Philadelphia u.a.
    Publishing country United States
    Document type Book
    HBZ-ID HT014794218
    ISBN 978-0-7817-4750-9 ; 0-7817-4750-3
    Database Catalogue ZB MED Medicine, Health

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    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2006 A 6223 order for loan Magazin

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  5. Book: Heptinstall's pathology of the kidney / 2

    Heptinstall, Robert H. / Jennette, J. Charles

    2007  

    Author's details ed. J. Charles Jennette
    Collection Heptinstall's pathology of the kidney
    Language English
    Size X S., S. 854 - 1531, 48 S. : zahlr. Ill., graph. Darst.
    Edition 6. ed.
    Publisher Lippincott-Raven
    Publishing place Philadelphia u.a.
    Publishing country United States
    Document type Book
    HBZ-ID HT014794220
    ISBN 978-0-7817-4750-9 ; 0-7817-4750-3
    Database Catalogue ZB MED Medicine, Health

    Kategorien

    In stock of ZB MED Cologne/Königswinter

    Shelf markLoan statusLocation
    2006 A 6224 order for loan Magazin

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  6. Book ; Online: Heptinstall's pathology of the kidney

    Jennette, J. Charles / Heptinstall, Robert H., 1920-

    2007  

    Title variant Pathology of the kidney
    Author's details editors, J. Charles Jennette
    Keywords Kidney diseases - pathology
    Language English
    Size 1 Online-Ressource (2 Bände (x, 1531, i-48 Seiten)), Illustrationen
    Edition 6th ed.
    Publisher Lippincott Williams & Wilkins
    Publishing place Philadelphia
    Document type Book ; Online
    Note Includes bibliographical references and index
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    ISBN 978-0-7817-4750-9 ; 0-7817-4750-3
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

    Full text online

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  7. Article ; Online: Fabry Disease Associated With Antiglomerular Basement Membrane Disease: Chance or Consequence.

    Terinte-Balcan, George / Krishnasami, Zipporah / McMahon, Blaithin A / Jennette, J Charles

    Kidney international reports

    2022  Volume 7, Issue 3, Page(s) 658–659

    Language English
    Publishing date 2022-01-17
    Publishing country United States
    Document type Journal Article
    ISSN 2468-0249
    ISSN (online) 2468-0249
    DOI 10.1016/j.ekir.2022.01.1046
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Article ; Online: L17. What can we expect from the revised Chapel Hill consensus conference nomenclature of vasculitis?

    Jennette, J Charles

    Presse medicale (Paris, France : 1983)

    2013  Volume 42, Issue 4 Pt 2, Page(s) 550–555

    MeSH term(s) Child ; Diagnosis, Differential ; Humans ; Terminology as Topic ; Vasculitis/classification ; Vasculitis/diagnosis ; Vasculitis/etiology
    Language English
    Publishing date 2013-04
    Publishing country France
    Document type Consensus Development Conference ; Journal Article ; Review
    ZDB-ID 120943-7
    ISSN 2213-0276 ; 0032-7867 ; 0755-4982 ; 0301-1518
    ISSN (online) 2213-0276
    ISSN 0032-7867 ; 0755-4982 ; 0301-1518
    DOI 10.1016/j.lpm.2013.01.016
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    In stock of ZB MED Cologne/Königswinter

    Zs.A 794: Show issues Location:
    Je nach Verfügbarkeit (siehe Angabe bei Bestand)
    bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular
    Jg. 1995 - 2021: Lesesall (1.OG)
    ab Jg. 2022: Lesesaal (EG)
    Ua VI Zs.87: Show issues Location:
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  9. Article ; Online: Glomerulonephritis in Animal Models and Human Medicine: Discovery, Pathogenesis, and Diagnostics.

    Cianciolo, Rachel E / Jennette, J Charles

    Toxicologic pathology

    2018  Volume 46, Issue 8, Page(s) 898–903

    Abstract: Glomerulonephritis (GN) is inflammation of glomeruli. The four major categories that cause human GN are mediated by immunoglobulin or complement or both, and they include (1) immune complex-mediated GN, (2) anti-glomerular basement membrane-mediated GN, ( ...

    Abstract Glomerulonephritis (GN) is inflammation of glomeruli. The four major categories that cause human GN are mediated by immunoglobulin or complement or both, and they include (1) immune complex-mediated GN, (2) anti-glomerular basement membrane-mediated GN, (3) antineutrophil cytoplasmic autoantibody-mediated GN, and (4) complement factor 3 glomerulopathy mediated by complement dysregulation. Initiating processes include infection, autoimmunity, exogenous antigens, and neoplasia. Often there are predisposing and modulating genetic, epigenetic, and/or environmental factors. Animal models facilitated the recognition and elucidation of the pathogeneses of all four categories of GN, and they continue to be used in preclinical studies to identify and validate therapies for all four types of GN. Advanced diagnostic modalities (e.g., transmission electron microscopy and immunofluorescence) are helpful and sometimes required for the correct categorization of GN in humans and animals. This review provides historical background on the discovery of the different GN pathogeneses, describes some of the animal models used to discover and understand each GN pathogenic category, reviews the diagnostic classification of each category of GN, and compares human GN to spontaneous forms of nonhuman GN.
    MeSH term(s) Animals ; Disease Models, Animal ; Glomerulonephritis/classification ; Glomerulonephritis/diagnosis ; Glomerulonephritis/pathology ; Humans
    Language English
    Publishing date 2018-10-03
    Publishing country United States
    Document type Journal Article
    ZDB-ID 841009-4
    ISSN 1533-1601 ; 0192-6233
    ISSN (online) 1533-1601
    ISSN 0192-6233
    DOI 10.1177/0192623318800714
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 1975: Show issues Location:
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    Zs.MO 473: Show issues

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  10. Article ; Online: Mechanisms of vascular damage in ANCA vasculitis.

    Massicotte-Azarniouch, David / Herrera, Carolina A / Jennette, J Charles / Falk, Ronald J / Free, Meghan E

    Seminars in immunopathology

    2022  Volume 44, Issue 3, Page(s) 325–345

    Abstract: The discovery of anti-neutrophil cytoplasmic antibodies (ANCA) and their antigenic targets, myeloperoxidase (MPO) and proteinase 3 (PR3), has led to further understanding as to the pathophysiologic processes that underlie vascular and tissue damage in ... ...

    Abstract The discovery of anti-neutrophil cytoplasmic antibodies (ANCA) and their antigenic targets, myeloperoxidase (MPO) and proteinase 3 (PR3), has led to further understanding as to the pathophysiologic processes that underlie vascular and tissue damage in ANCA vasculitis. ANCA trigger neutrophil activation leading to vascular damage in ANCA vasculitis. However, decades of study have determined that neutrophil activation alone is not sufficient to cause disease. Inflammatory stimuli are drivers of ANCA autoantigen expression and ANCA production. Certain infections or bacterial peptides may be crucial players in the initial steps of ANCA immunopathogenesis. Genetic and epigenetic alterations of gene encoding for MPO and PR3 provide additional disturbances to the immune homeostasis which provide a substrate for pathogenic ANCA formation from an adaptive immune system predisposed to autoreactivity. Promoted by inflammatory cytokines, ANCA binding leads to neutrophil activation, a process characterized by conformational changes, production and release of cytotoxic substances, and alternative complement pathway activation, thus creating an intense inflammatory milieu. This cascade of events perpetuates a vicious cycle of further inflammatory cell recruitment and activation, culminating in tissue necrosis. Our understanding of the pathogenic process in ANCA vasculitis paves the way for the development of therapies targeting crucial steps in this process. The greater appreciation of the role for complement, monocytes, and the adaptive immune system has already led to novel complement blockers and is poised to lead to further innovations which will allow for tailored antigen- or cell-specific immunotherapy targeting the autoimmune process without exposure to undue risks or toxicities.
    MeSH term(s) Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/etiology ; Antibodies, Antineutrophil Cytoplasmic ; Autoantigens ; Humans ; Monocytes/metabolism ; Myeloblastin/genetics ; Vasculitis/metabolism
    Chemical Substances Antibodies, Antineutrophil Cytoplasmic ; Autoantigens ; Myeloblastin (EC 3.4.21.76)
    Language English
    Publishing date 2022-03-07
    Publishing country Germany
    Document type Journal Article ; Review
    ZDB-ID 2316828-6
    ISSN 1863-2300 ; 1863-2297
    ISSN (online) 1863-2300
    ISSN 1863-2297
    DOI 10.1007/s00281-022-00920-0
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.A 1425: Show issues Location:
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