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Article ; Online: Thrombosis in adult patients with acute leukemia.

Del Principe, Maria Ilaria / Del Principe, Domenico / Venditti, Adriano

2017 Volume 29, Issue 6, Page(s) 448–454

Abstract: Purpose of review: Recent studies indicate that the risk of thrombosis in hematologic patients may be similar or even higher than that found in patients with solid tumors. However, available information about pathogenesis and incidence of thrombosis in ... ...

Abstract	Purpose of review: Recent studies indicate that the risk of thrombosis in hematologic patients may be similar or even higher than that found in patients with solid tumors. However, available information about pathogenesis and incidence of thrombosis in acute leukemia is limited. This review focuses on mechanisms underlying thrombosis in acute leukemia and discusses recent literature data. Recent findings: In the last few years, proofs have been provided that leukemic cells release free prothrombotic products, such as micro-vesicles, tissue factors, circulating free DNA and RNA. Furthermore, leukemic blasts can activate the procoagulant population of platelets, which initiate and amplify coagulation, causing thrombosis. In addition to factors produced by acute leukemia itself, others concur to trigger thrombosis. Some drugs, infections and insertion of central venous catheter have been described to increase risk of thrombosis in patients with acute leukemia. Summary: Thrombosis represents a serious complication in patients affected by myeloid and lymphoid acute leukemia. A proper knowledge of its pathophysiology and of the predisposing risk factors may allow to implement strategies of prevention. Improving prevention of thrombosis appears a major goal in patients whose frequent conditions of thrombocytopenia impede an adequate delivery of anticoagulant therapy.
Language	English
Publishing date	2017-11
Publishing country	United States
Document type	Journal Article
ZDB-ID	1049384-0
ISSN	1531-703X ; 1040-8746
ISSN (online)	1531-703X
ISSN	1040-8746
DOI	10.1097/CCO.0000000000000402
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Article ; Online: Direct CD32 T-cell cytotoxicity: implications for breast cancer prognosis and treatment.

Sconocchia, Giuseppe / Lanzilli, Giulia / Cesarini, Valeriana / Silvestris, Domenico A / Rezvani, Katayoun / Arriga, Roberto / Caratelli, Sara / Chen, Ken / Dou, Jinzhuang / Cenciarelli, Carlo / Toietta, Gabriele / Baldari, Silvia / Sconocchia, Tommaso / De Paolis, Francesca / Aureli, Anna / Iezzi, Giandomenica / Irno Consalvo, Maria / Buccisano, Francesco / Del Principe, Maria I /

Maurillo, Luca / Venditti, Adriano / Ottaviani, Alessio / Spagnoli, Giulio C

Life science alliance

2022 Volume 5, Issue 12

Abstract: The FcγRII (CD32) ligands are IgFc fragments and pentraxins. The existence of additional ligands is unknown. We engineered T cells with human chimeric receptors resulting from the fusion between CD32 extracellular portion and transmembrane CD8α linked to ...

Abstract	The FcγRII (CD32) ligands are IgFc fragments and pentraxins. The existence of additional ligands is unknown. We engineered T cells with human chimeric receptors resulting from the fusion between CD32 extracellular portion and transmembrane CD8α linked to CD28/ζ chain intracellular moiety (CD32-CR). Transduced T cells recognized three breast cancer (BC) and one colon cancer cell line among 15 tested in the absence of targeting antibodies. Sensitive BC cell conjugation with CD32-CR T cells induced CD32 polarization and down-regulation, CD107a release, mutual elimination, and proinflammatory cytokine production unaffected by human IgGs but enhanced by cetuximab. CD32-CR T cells protected immunodeficient mice from subcutaneous growth of MDA-MB-468 BC cells. RNAseq analysis identified a 42 gene fingerprint predicting BC cell sensitivity and favorable outcomes in advanced BC. ICAM1 was a major regulator of CD32-CR T cell-mediated cytotoxicity. CD32-CR T cells may help identify cell surface CD32 ligand(s) and novel prognostically relevant transcriptomic signatures and develop innovative BC treatments.
MeSH term(s)	Animals ; Breast Neoplasms/genetics ; Breast Neoplasms/metabolism ; Breast Neoplasms/therapy ; CD28 Antigens/metabolism ; Cetuximab/metabolism ; Female ; Humans ; Ligands ; Mice ; T-Lymphocytes
Chemical Substances	CD28 Antigens ; Ligands ; Cetuximab (PQX0D8J21J)
Language	English
Publishing date	2022-10-14
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ISSN	2575-1077
ISSN (online)	2575-1077
DOI	10.26508/lsa.202201590
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Article ; Online: Fibroblast autophagy in fibrotic disorders.

Del Principe, Domenico / Lista, Pasquale / Malorni, Walter / Giammarioli, Anna Maria

The Journal of pathology

2013 Volume 229, Issue 2, Page(s) 208–220

Abstract: Fibrotic disorders are multistage progressive processes that often arise from different causes and are commonly associated with chronic inflammation. Excessive deposition of extracellular matrix is the hallmark of many fibrotic diseases. This may be due ... ...

Abstract	Fibrotic disorders are multistage progressive processes that often arise from different causes and are commonly associated with chronic inflammation. Excessive deposition of extracellular matrix is the hallmark of many fibrotic diseases. This may be due to an excess of fibroblast recruitment and activation, as well as to their differentiation in myofibroblasts. These events may be triggered by cytokines, chemokines and growth factors released by lymphocytes or macrophages. The excessive production of extracellular matrix is apparently due to alterations of metabolic pathways in activated fibroblasts. It has been suggested that a defective autophagy, an important subcellular pathway with multiple homeostatic roles, also recognized as a key component of both innate and acquired immunity, could play a role. In this review we illustrate recent insights in the field, suggesting the possible implication of the immune system in orchestrating the fibrotic response via the modulation of autophagic pathways.
MeSH term(s)	Animals ; Autophagy ; Extracellular Matrix/metabolism ; Extracellular Matrix/pathology ; Fibroblasts/immunology ; Fibroblasts/metabolism ; Fibroblasts/pathology ; Fibrosis ; Homeostasis ; Humans ; Signal Transduction
Language	English
Publishing date	2013-01
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	3119-7
ISSN	1096-9896 ; 0022-3417
ISSN (online)	1096-9896
ISSN	0022-3417
DOI	10.1002/path.4115
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Article ; Online: Pathogenetic determinants in Kawasaki disease: the haematological point of view.

Del Principe, Domenico / Pietraforte, Donatella / Gambardella, Lucrezia / Marchesi, Alessandra / Tarissi de Jacobis, Isabella / Villani, Alberto / Malorni, Walter / Straface, Elisabetta

Journal of cellular and molecular medicine

2017 Volume 21, Issue 4, Page(s) 632–639

Abstract: Kawasaki disease is a multisystemic vasculitis that can result in coronary artery lesions. It predominantly affects young children and is characterized by prolonged fever, diffuse mucosal inflammation, indurative oedema of the hands and feet, a ... ...

Abstract	Kawasaki disease is a multisystemic vasculitis that can result in coronary artery lesions. It predominantly affects young children and is characterized by prolonged fever, diffuse mucosal inflammation, indurative oedema of the hands and feet, a polymorphous skin rash and non-suppurative lymphadenopathy. Coronary artery involvement is the most important complication of Kawasaki disease and may cause significant coronary stenosis resulting in ischemic heart disease. The introduction of intravenous immunoglobulin decreases the incidence of coronary artery lesions to less than 5%. The etiopathogenesis of this disease remains unclear. Several lines of evidence suggest that an interplay between a microbial infection and a genetic predisposition could take place in the development of the disease. In this review, we summarize the state of the art of pathogenetic mechanisms of Kawasaki disease underscoring the relevance of haematological features as a novel field of investigation.
MeSH term(s)	Biomarkers/blood ; Humans ; Immunity ; Mucocutaneous Lymph Node Syndrome/blood ; Mucocutaneous Lymph Node Syndrome/etiology ; Mucocutaneous Lymph Node Syndrome/genetics ; Mucocutaneous Lymph Node Syndrome/pathology ; Prognosis
Chemical Substances	Biomarkers
Keywords	covid19
Language	English
Publishing date	2017-04
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	2074559-X
ISSN	1582-4934 ; 1582-4934 ; 1582-1838
ISSN (online)	1582-4934
ISSN	1582-4934 ; 1582-1838
DOI	10.1111/jcmm.12992
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Zs.A 5752: Show issues

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Article ; Online: Platelets in Kawasaki patients: two different populations with different mitochondrial functions.

Pietraforte, Donatella / Gambardella, Lucrezia / Marchesi, Alessandra / Tarissi de Jacobis, Isabella / Villani, Alberto / Del Principe, Domenico / Malorni, Walter / Straface, Elisabetta

International journal of cardiology

2014 Volume 172, Issue 2, Page(s) 526–528

MeSH term(s)	Blood Platelets/physiology ; Child, Preschool ; Humans ; Infant ; Male ; Mitochondria/physiology ; Mitochondrial Membranes/physiology ; Mucocutaneous Lymph Node Syndrome/blood ; Mucocutaneous Lymph Node Syndrome/diagnosis ; Platelet Aggregation/physiology
Language	English
Publishing date	2014-03-15
Publishing country	Netherlands
Document type	Letter ; Research Support, Non-U.S. Gov't
ZDB-ID	779519-1
ISSN	1874-1754 ; 0167-5273
ISSN (online)	1874-1754
ISSN	0167-5273
DOI	10.1016/j.ijcard.2014.01.022
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Zs.A 1673: Show issues

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Article: CD16(low/negative) tumor-infiltrating lymphocyte: lymphoid or myeloid in origin?

Sconocchia, Giuseppe / Del Principe, Domenico / Barrett, A John

Clinical cancer research : an official journal of the American Association for Cancer Research

2007 Volume 13, Issue 5, Page(s) 1620; author reply 1621

MeSH term(s)	Cell Lineage/immunology ; Humans ; Killer Cells, Natural/cytology ; Killer Cells, Natural/immunology ; Killer Cells, Natural/metabolism ; Lymphocytes, Tumor-Infiltrating/cytology ; Lymphocytes, Tumor-Infiltrating/immunology ; Lymphocytes, Tumor-Infiltrating/metabolism ; Receptors, IgG/immunology ; Receptors, IgG/metabolism
Chemical Substances	Receptors, IgG
Language	English
Publishing date	2007-03-01
Publishing country	United States
Document type	Comment ; Letter
ZDB-ID	1225457-5
ISSN	1557-3265 ; 1078-0432
ISSN (online)	1557-3265
ISSN	1078-0432
DOI	10.1158/1078-0432.CCR-06-2247
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Zs.A 4223: Show issues

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Article ; Online: Trans-plasma membrane electron transport in mammals: functional significance in health and disease.

Del Principe, Domenico / Avigliano, Luciana / Savini, Isabella / Catani, Maria Valeria

Antioxidants & redox signaling

2011 Volume 14, Issue 11, Page(s) 2289–2318

Abstract: Trans-plasma membrane electron transport (t-PMET) has been established since the 1960s, but it has only been subject to more intensive research in the last decade. The discovery and characterization at the molecular level of its novel components has ... ...

Abstract	Trans-plasma membrane electron transport (t-PMET) has been established since the 1960s, but it has only been subject to more intensive research in the last decade. The discovery and characterization at the molecular level of its novel components has increased our understanding of how t-PMET regulates distinct cellular functions. This review will give an update on t-PMET, with particular emphasis on how its malfunction relates to some diseases, such as cancer, abnormal cell death, cardiovascular diseases, aging, obesity, neurodegenerative diseases, pulmonary fibrosis, asthma, and genetically linked pathologies. Understanding these relationships may provide novel therapeutic approaches for pathologies associated with unbalanced redox state.
MeSH term(s)	Aging/metabolism ; Animals ; Apoptosis ; Asthma/metabolism ; Autophagy ; Cardiovascular Diseases/metabolism ; Cell Membrane/metabolism ; Cytochrome Reductases/metabolism ; Cytochromes b/metabolism ; Electron Transport/physiology ; Fertilization ; Homeostasis ; Humans ; Mammals ; Metabolic Diseases/metabolism ; NADH, NADPH Oxidoreductases/metabolism ; Neoplasms/metabolism ; Neurodegenerative Diseases/metabolism ; Pulmonary Fibrosis/metabolism ; Spermatogenesis ; Voltage-Dependent Anion Channels/metabolism
Chemical Substances	Voltage-Dependent Anion Channels ; Cytochromes b (9035-37-4) ; NADH, NADPH Oxidoreductases (EC 1.6.-) ; Cytochrome Reductases (EC 1.6.2.-)
Language	English
Publishing date	2011-06
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	1483836-9
ISSN	1557-7716 ; 1523-0864
ISSN (online)	1557-7716
ISSN	1523-0864
DOI	10.1089/ars.2010.3247
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Article ; Online: The relevance of estrogen/estrogen receptor system on the gender difference in cardiovascular risk.

Del Principe, Domenico / Ruggieri, Anna / Pietraforte, Donatella / Villani, Alberto / Vitale, Cristiana / Straface, Elisabetta / Malorni, Walter

International journal of cardiology

2015 Volume 187, Page(s) 291–298

Abstract: It has been reported that the incidence of thrombotic events can display a gender disparity. In particular, a lower thrombotic risk has been described in female gender. The mechanisms underlying this disparity are still poorly understood. Of great ... ...

Abstract	It has been reported that the incidence of thrombotic events can display a gender disparity. In particular, a lower thrombotic risk has been described in female gender. The mechanisms underlying this disparity are still poorly understood. Of great interest is the hypothesis that hormones, estrogen in particular, could play a key role. In fact, the possibility that some hormonal factors could protect women from thrombotic events appears well documented in literature. For instance, several studies aimed at the analysis of the impact of estrogen and estrogen receptors in thrombogenesis claim for the implication of these hormones either in megakaryocyte differentiation or, more intriguingly, directly affecting platelet integrity and function. In consideration of the absence of the nucleus, platelet susceptibility appears quite striking and probably due to the non-nuclear estrogen receptor function. In this review we briefly summarize our knowledge as concerns the role of estrogen and estrogen receptors in determining megakaryocyte/platelet functions and thrombogenicity.
MeSH term(s)	Blood Coagulation/physiology ; Blood Platelets/physiology ; Cardiovascular Diseases/epidemiology ; Cardiovascular Diseases/etiology ; Estrogens/physiology ; Female ; Humans ; Male ; Receptors, Estrogen/physiology ; Risk Factors ; Sex Factors ; Thrombosis/epidemiology ; Thrombosis/etiology
Chemical Substances	Estrogens ; Receptors, Estrogen
Language	English
Publishing date	2015
Publishing country	Netherlands
Document type	Journal Article ; Review
ZDB-ID	779519-1
ISSN	1874-1754 ; 0167-5273
ISSN (online)	1874-1754
ISSN	0167-5273
DOI	10.1016/j.ijcard.2015.03.145
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Article ; Online: Invasive aspergillosis in relapsed/refractory acute myeloid leukaemia patients: Results from SEIFEM 2016-B survey.

Del Principe, Maria Ilaria / Dragonetti, Giulia / Conti, Allegra / Verga, Luisa / Ballanti, Stelvio / Fanci, Rosa / Candoni, Anna / Marchesi, Francesco / Cattaneo, Chiara / Lessi, Federica / Fracchiolla, Nicola / Spolzino, Angelica / Prezioso, Lucia / Delia, Mario / Potenza, Leonardo / Decembrino, Nunzia / Castagnola, Carlo / Nadali, Gianpaolo / Picardi, Marco /

Zama, Daniele / Orciulo, Enrico / Veggia, Barbara / Garzia, Mariagrazia / Dargenio, Michelina / Melillo, Lorella / Manetta, Sara / Russo, Domenico / Mancini, Valentina / Piedimonte, Monica / Tisi, Maria Chiara / Toschi, Nicola / Busca, Alessandro / Pagano, Livio

Mycoses

2021 Volume 65, Issue 2, Page(s) 171–177

Abstract: Background: In patients with relapsed/refractory acute myeloid leukaemia (R/R AML) who received salvage chemotherapy, limited and not updated studies explored the incidence of invasive aspergillosis (IA) and the role of antifungal prophylaxis (AP). The ... ...

Abstract	Background: In patients with relapsed/refractory acute myeloid leukaemia (R/R AML) who received salvage chemotherapy, limited and not updated studies explored the incidence of invasive aspergillosis (IA) and the role of antifungal prophylaxis (AP). The aims of this multicentre retrospective 'SEIFEM 2016-B' study were as follows: (1) to evaluate the current rate and the outcome of proven/probable IA and (2) to assess the efficacy of AP, in a large 'real life' series of patient with R/R AML submitted to salvage chemotherapy. Results: Of 2250 R/R AML patients, a total of 74 cases of IA (5.1%) were recorded as follows: 10 (0.7%) proven and 64 (4.3%) probable. Information about AP were available in 73/74 (99%) patients. Fifty-eight (79%) breakthrough infections occurred, mainly during AP with posaconazole [25 (43%)]. The patients who received AP during salvage chemotherapy showed a benefit from antifungal therapy (AT) than patients who did not received AP [43 (86%) vs 7 (14%); p < .033]. In a multivariate analysis, AP and absence of severe mucositis had a significant favourable effect on overall response rate. Conclusion: Our data demonstrated that the incidence of IA during the salvage chemotherapy is similar to the past. Nevertheless, the attributable mortality rate (AMR) appears to be lower than that previously reported in R/R AML. Further prospective studies should be performed to confirm our preliminary observation and understand and the why a decreased AMR is reported in this setting of high-risk patients.
MeSH term(s)	Antifungal Agents/therapeutic use ; Aspergillosis/drug therapy ; Aspergillosis/epidemiology ; Humans ; Invasive Fungal Infections/drug therapy ; Invasive Fungal Infections/epidemiology ; Leukemia, Myeloid, Acute/complications ; Leukemia, Myeloid, Acute/drug therapy ; Leukemia, Myeloid, Acute/microbiology ; Retrospective Studies
Chemical Substances	Antifungal Agents
Language	English
Publishing date	2021-12-03
Publishing country	Germany
Document type	Journal Article ; Multicenter Study
ZDB-ID	392487-7
ISSN	1439-0507 ; 0933-7407
ISSN (online)	1439-0507
ISSN	0933-7407
DOI	10.1111/myc.13384
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Article ; Online: Downstream effects of endocannabinoid on blood cells: implications for health and disease.

Gasperi, Valeria / Evangelista, Daniela / Savini, Isabella / Del Principe, Domenico / Avigliano, Luciana / Maccarrone, Mauro / Catani, Maria Valeria

Cellular and molecular life sciences : CMLS

2015 Volume 72, Issue 17, Page(s) 3235–3252

Abstract: Endocannabinoids (eCBs), among which N-arachidonoylethanolamine (AEA) and 2-arachidonoylglycerol (2-AG) are the most biologically active members, are polyunsaturated lipids able to bind cannabinoid, vanilloid and peroxisome proliferator-activated ... ...

Abstract	Endocannabinoids (eCBs), among which N-arachidonoylethanolamine (AEA) and 2-arachidonoylglycerol (2-AG) are the most biologically active members, are polyunsaturated lipids able to bind cannabinoid, vanilloid and peroxisome proliferator-activated receptors. Depending on the target engaged, these bioactive mediators can regulate different signalling pathways, at both central and peripheral levels. The biological action of eCBs is tightly controlled by a plethora of metabolic enzymes which, together with the molecular targets of these substances, form the so-called "endocannabinoid system". The ability of eCBs to control manifold peripheral functions has received a great deal of attention, especially in the light of their widespread distribution in the body. In particular, eCBs are important regulators in blood, where they modulate haematopoiesis, platelet aggregation and apoptosis, as well as chemokine release and migration of immunocompetent cells. Here, we shall review the current knowledge on the pathophysiological roles of eCBs in blood. We shall also discuss the involvement of eCBs in those disorders affecting the haematological system, including cancer and inflammation. Knowledge gained to date underlines a fundamental role of the eCB system in blood, thus suggesting that it may represent a therapeutic promise for a broad range of diseases involving impaired hematopoietic cell functions.
MeSH term(s)	Blood Cells/metabolism ; Blood Cells/physiology ; Endocannabinoids/chemistry ; Endocannabinoids/metabolism ; Endocannabinoids/physiology ; Hematologic Diseases/physiopathology ; Hematopoiesis/physiology ; Humans ; Models, Biological ; Molecular Structure ; Signal Transduction/physiology
Chemical Substances	Endocannabinoids
Language	English
Publishing date	2015-09
Publishing country	Switzerland
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	1358415-7
ISSN	1420-9071 ; 1420-682X
ISSN (online)	1420-9071
ISSN	1420-682X
DOI	10.1007/s00018-015-1924-0
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