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  1. Book ; Online ; E-Book: Lung disease in rheumatoid arthritis

    Fischer, Aryeh / Lee, Joyce S.

    (Respiratory medicine)

    2018  

    Author's details Aryeh Fischer, Joyce S. Lee editors
    Series title Respiratory medicine
    Language English
    Size 1 Online-Ressource (x, 178 Seiten), Illustrationen, Diagramme
    Publisher Humana Press
    Publishing place Cham
    Publishing country Switzerland
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT019657387
    ISBN 978-3-319-68888-6 ; 9783319688879 ; 3-319-68888-X ; 3319688871
    DOI 10.1007/978-3-319-68888-6
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  2. Article ; Online: Gazing into the Proteomic Crystal Ball: Predicting Survival in Idiopathic Pulmonary Fibrosis.

    Lee, Joyce S / Maher, Toby

    American journal of respiratory and critical care medicine

    2023  

    Language English
    Publishing date 2023-12-20
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1180953-x
    ISSN 1535-4970 ; 0003-0805 ; 1073-449X
    ISSN (online) 1535-4970
    ISSN 0003-0805 ; 1073-449X
    DOI 10.1164/rccm.202311-2108ED
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article ; Online: Overview of Rheumatoid Arthritis-Associated Interstitial Lung Disease and Its Treatment.

    Pugashetti, Janelle Vu / Lee, Joyce S

    Seminars in respiratory and critical care medicine

    2024  

    Abstract: Interstitial lung disease (ILD) is a common pulmonary complication of rheumatoid arthritis (RA), causing significant morbidity and mortality. Optimal treatment for RA-ILD is not yet well defined. Reliable prognostic indicators are largely byproducts of ... ...

    Abstract Interstitial lung disease (ILD) is a common pulmonary complication of rheumatoid arthritis (RA), causing significant morbidity and mortality. Optimal treatment for RA-ILD is not yet well defined. Reliable prognostic indicators are largely byproducts of prior ILD progression, including low or decreasing forced vital capacity and extensive or worsening fibrosis on imaging. In the absence of validated tools to predict treatment response, decisions about whether to initiate or augment treatment are instead based on clinical judgment. In general, treatment should be initiated in patients who are symptomatic, progressing, or at high risk of poor outcomes. Retrospective data suggest that mycophenolate mofetil, azathioprine, and rituximab are likely effective therapies for RA-ILD. Abatacept is also emerging as a potential first-line treatment option for patients with RA-ILD. Further, recent data demonstrate that immunosuppression may be beneficial even in patients with a usual interstitial pneumonia (UIP) pattern on imaging, suggesting that immunosuppression should be considered irrespective of imaging pattern. Recent randomized controlled trials have shown that antifibrotic medications, such as nintedanib and likely pirfenidone, slow forced vital capacity decline in RA-ILD. Consideration can be given to antifibrotic initiation in patients progressing despite immunosuppression, particularly in patients with a UIP pattern. Future research directions include developing tools to predict which patients will remain stable from patients who will progress, discriminating patients who will respond to treatment from nonresponders, and developing algorithms for starting immunosuppression, antifibrotics, or both as first-line therapies.
    Language English
    Publishing date 2024-03-14
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1183617-9
    ISSN 1098-9048 ; 1069-3424
    ISSN (online) 1098-9048
    ISSN 1069-3424
    DOI 10.1055/s-0044-1782218
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article ; Online: In search of the elusive biomarker(s): a proteomics analysis in rheumatoid arthritis-associated interstitial lung disease.

    Matson, Scott M / Lee, Joyce S

    Thorax

    2022  Volume 77, Issue 10, Page(s) 949

    MeSH term(s) Humans ; Proteomics ; Lung Diseases, Interstitial/complications ; Arthritis, Rheumatoid/complications ; Biomarkers ; Idiopathic Pulmonary Fibrosis/complications ; Lung
    Chemical Substances Biomarkers
    Language English
    Publishing date 2022-08-18
    Publishing country England
    Document type Journal Article ; Research Support, N.I.H., Extramural
    ZDB-ID 204353-1
    ISSN 1468-3296 ; 0040-6376
    ISSN (online) 1468-3296
    ISSN 0040-6376
    DOI 10.1136/thorax-2022-219354
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  5. Article ; Online: Recent advances in the genetics of idiopathic pulmonary fibrosis.

    Spagnolo, Paolo / Lee, Joyce S

    Current opinion in pulmonary medicine

    2023  Volume 29, Issue 5, Page(s) 399–405

    Abstract: Purpose of review: Genetics contributes substantially to the susceptibility to idiopathic pulmonary fibrosis (IPF). Genetic studies in sporadic and familial disease have identified several IPF-associated variants, mainly in telomere-related and ... ...

    Abstract Purpose of review: Genetics contributes substantially to the susceptibility to idiopathic pulmonary fibrosis (IPF). Genetic studies in sporadic and familial disease have identified several IPF-associated variants, mainly in telomere-related and surfactant protein genes.Here, we review the most recent literature on genetics of IPF and discuss how it may contribute to disease pathogenesis.
    Recent findings: Recent studies implicate genes involved in telomere maintenance, host defence, cell growth, mammalian target of rapamycin signalling, cell-cell adhesion, regulation of TGF-β signalling and spindle assembly as biological processes involved in the pathogenesis of IPF. Both common and rare genetic variants contribute to the overall risk of IPF; however, while common variants (i.e. polymorphisms) account for most of the heritability of sporadic disease, rare variants (i.e. mutations), mainly in telomere-related genes, are the main contributors to the heritability of familial disease. Genetic factors are likely to also influence disease behaviour and prognosis. Finally, recent data suggest that IPF shares genetic associations - and probably some pathogenetic mechanisms - with other fibrotic lung diseases.
    Summary: Common and rare genetic variants are associated with susceptibility and prognosis of IPF. However, many of the reported variants fall in noncoding regions of the genome and their relevance to disease pathobiology remains to be elucidated.
    MeSH term(s) Humans ; Idiopathic Pulmonary Fibrosis/genetics ; Mutation ; Pulmonary Surfactants ; Telomerase/genetics ; Signal Transduction/genetics
    Chemical Substances Pulmonary Surfactants ; Telomerase (EC 2.7.7.49)
    Language English
    Publishing date 2023-07-06
    Publishing country United States
    Document type Review ; Journal Article
    ZDB-ID 1285505-4
    ISSN 1531-6971 ; 1070-5287 ; 1078-1641
    ISSN (online) 1531-6971
    ISSN 1070-5287 ; 1078-1641
    DOI 10.1097/MCP.0000000000000989
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  6. Article ; Online: Cryptogenic Organizing Pneumonia.

    King, Talmadge E / Lee, Joyce S

    The New England journal of medicine

    2022  Volume 386, Issue 11, Page(s) 1058–1069

    MeSH term(s) Cryptogenic Organizing Pneumonia/diagnosis ; Cryptogenic Organizing Pneumonia/drug therapy ; Cryptogenic Organizing Pneumonia/pathology ; Cryptogenic Organizing Pneumonia/physiopathology ; Diagnosis, Differential ; Glucocorticoids/therapeutic use ; Humans ; Lung/diagnostic imaging ; Lung/pathology ; Prognosis
    Chemical Substances Glucocorticoids
    Language English
    Publishing date 2022-03-01
    Publishing country United States
    Document type Journal Article ; Review
    ZDB-ID 207154-x
    ISSN 1533-4406 ; 0028-4793
    ISSN (online) 1533-4406
    ISSN 0028-4793
    DOI 10.1056/NEJMra2116777
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  7. Article: Overview of Rheumatoid Arthritis-Associated Interstitial Lung Disease and Its Treatment

    Pugashetti, Janelle Vu / Lee, Joyce S.

    Seminars in Respiratory and Critical Care Medicine

    (Pulmonary Complications of Connective Tissue Diseases)

    2024  

    Abstract: Interstitial lung disease (ILD) is a common pulmonary complication of rheumatoid arthritis (RA), causing significant morbidity and mortality. Optimal treatment for RA-ILD is not yet well defined. Reliable prognostic indicators are largely byproducts of ... ...

    Series title Pulmonary Complications of Connective Tissue Diseases
    Abstract Interstitial lung disease (ILD) is a common pulmonary complication of rheumatoid arthritis (RA), causing significant morbidity and mortality. Optimal treatment for RA-ILD is not yet well defined. Reliable prognostic indicators are largely byproducts of prior ILD progression, including low or decreasing forced vital capacity and extensive or worsening fibrosis on imaging. In the absence of validated tools to predict treatment response, decisions about whether to initiate or augment treatment are instead based on clinical judgment. In general, treatment should be initiated in patients who are symptomatic, progressing, or at high risk of poor outcomes. Retrospective data suggest that mycophenolate mofetil, azathioprine, and rituximab are likely effective therapies for RA-ILD. Abatacept is also emerging as a potential first-line treatment option for patients with RA-ILD. Further, recent data demonstrate that immunosuppression may be beneficial even in patients with a usual interstitial pneumonia (UIP) pattern on imaging, suggesting that immunosuppression should be considered irrespective of imaging pattern. Recent randomized controlled trials have shown that antifibrotic medications, such as nintedanib and likely pirfenidone, slow forced vital capacity decline in RA-ILD. Consideration can be given to antifibrotic initiation in patients progressing despite immunosuppression, particularly in patients with a UIP pattern. Future research directions include developing tools to predict which patients will remain stable from patients who will progress, discriminating patients who will respond to treatment from nonresponders, and developing algorithms for starting immunosuppression, antifibrotics, or both as first-line therapies.
    Keywords rheumatoid arthritis ; interstitial lung disease ; usual interstitial pneumonia ; treatment ; management ; mycophenolate ; azathioprine ; rituximab ; abatacept
    Language English
    Publishing date 2024-03-14
    Publisher Thieme Medical Publishers, Inc.
    Publishing place Stuttgart ; New York
    Document type Article
    ZDB-ID 1183617-9
    ISSN 1098-9048 ; 1069-3424
    ISSN (online) 1098-9048
    ISSN 1069-3424
    DOI 10.1055/s-0044-1782218
    Database Thieme publisher's database

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  8. Article ; Online: Risk association paradox after pediatric liver transplantation: Can we identify the causes that bring back patients to the operating room?

    Lee, Joyce S / Stewart, Kenneth E / Tanaka, Kenichi A / Butt, Amir L

    Paediatric anaesthesia

    2023  Volume 34, Issue 2, Page(s) 185–186

    MeSH term(s) Child ; Humans ; Liver Transplantation ; Operating Rooms ; Retrospective Studies ; Length of Stay
    Language English
    Publishing date 2023-10-18
    Publishing country France
    Document type Letter ; Comment
    ZDB-ID 1086049-6
    ISSN 1460-9592 ; 1155-5645
    ISSN (online) 1460-9592
    ISSN 1155-5645
    DOI 10.1111/pan.14784
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  9. Article: Editorial: Interstitial Lung Disease Around the World.

    Wijsenbeek, Marlies / Lee, Joyce S / Udwadia, Zarir / Kreuter, Michael

    Frontiers in medicine

    2022  Volume 9, Page(s) 865334

    Language English
    Publishing date 2022-03-18
    Publishing country Switzerland
    Document type Editorial
    ZDB-ID 2775999-4
    ISSN 2296-858X
    ISSN 2296-858X
    DOI 10.3389/fmed.2022.865334
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  10. Article ; Online: "An Ounce of Prevention . . . ": Will This Be the Future for Idiopathic Pulmonary Fibrosis?

    Lee, Joyce S

    American journal of respiratory and critical care medicine

    2017  Volume 196, Issue 10, Page(s) 1240–1241

    Language English
    Publishing date 2017-11-15
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1180953-x
    ISSN 1535-4970 ; 0003-0805 ; 1073-449X
    ISSN (online) 1535-4970
    ISSN 0003-0805 ; 1073-449X
    DOI 10.1164/rccm.201706-1116ED
    Database MEDical Literature Analysis and Retrieval System OnLINE

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