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  1. Article ; Online: The rise of precision cellular therapies.

    Konig, Maximilian F

    Nature reviews. Rheumatology

    2024  Volume 20, Issue 2, Page(s) 69–70

    MeSH term(s) Humans ; Precision Medicine ; Molecular Targeted Therapy
    Language English
    Publishing date 2024-01-08
    Publishing country United States
    Document type Journal Article
    ZDB-ID 2491532-4
    ISSN 1759-4804 ; 1759-4790
    ISSN (online) 1759-4804
    ISSN 1759-4790
    DOI 10.1038/s41584-023-01073-6
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    Zs.A 6338: Show issues Location:
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  2. Article ; Online: Sarcoid myopathy: an insidious diagnosis that can mimic inclusion body myositis.

    Mathias, Kristen / Konig, Maximilian F / Lloyd, Thomas / Albayda, Jemima

    Rheumatology (Oxford, England)

    2024  

    Language English
    Publishing date 2024-03-11
    Publishing country England
    Document type Journal Article
    ZDB-ID 1464822-2
    ISSN 1462-0332 ; 1462-0324
    ISSN (online) 1462-0332
    ISSN 1462-0324
    DOI 10.1093/rheumatology/keae155
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  3. Article ; Online: Joint Pain in a Woman Wearing a Face Mask.

    Liebowitz, Jason E / Konig, Maximilian F

    JAMA

    2022  Volume 327, Issue 14, Page(s) 1392–1393

    MeSH term(s) Arthralgia/etiology ; COVID-19/prevention & control ; Female ; Humans ; Masks ; Personal Protective Equipment
    Language English
    Publishing date 2022-03-17
    Publishing country United States
    Document type Case Reports ; Journal Article
    ZDB-ID 2958-0
    ISSN 1538-3598 ; 0254-9077 ; 0002-9955 ; 0098-7484
    ISSN (online) 1538-3598
    ISSN 0254-9077 ; 0002-9955 ; 0098-7484
    DOI 10.1001/jama.2022.2644
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  4. Article ; Online: Clinical Images: Frosted branch angiitis in systemic lupus erythematosus.

    Tsou, Brittany C / Thangamathesvaran, Loka / Pandit, Ravi / Konig, Maximilian F / Haque, Uzma Jalal

    ACR open rheumatology

    2024  Volume 6, Issue 4, Page(s) 171

    Language English
    Publishing date 2024-01-04
    Publishing country United States
    Document type Journal Article
    ISSN 2578-5745
    ISSN (online) 2578-5745
    DOI 10.1002/acr2.11648
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  5. Article ; Online: The microbiome in autoimmune rheumatic disease.

    Konig, Maximilian F

    Best practice & research. Clinical rheumatology

    2020  Volume 34, Issue 1, Page(s) 101473

    Abstract: Microbial contributions to the immunopathogenesis of autoimmune rheumatic diseases have been studied since the advent of germ theory in the 19th century. With the exception of Group A Streptococcus in rheumatic fever, early studies failed to establish ... ...

    Abstract Microbial contributions to the immunopathogenesis of autoimmune rheumatic diseases have been studied since the advent of germ theory in the 19th century. With the exception of Group A Streptococcus in rheumatic fever, early studies failed to establish causal relationships between specific pathobionts and rheumatic disease. Today, systemic autoimmune diseases are thought to result from a complex interplay of environmental factors, individual genetic risk, and stochastic events. Interactions of microbiota and the immune system have been shown to promote and sustain chronic inflammation and autoimmunity. In mechanistic studies, microbe-immune cell interactions have been implicated in the initiation of autoimmune rheumatic diseases, e.g., through the posttranslational modification of autoantigens in rheumatoid arthritis or through neutrophil cell death and cross-reactivity with commensal orthologs in systemic lupus erythematosus. In parallel, modern molecular techniques have catalyzed the study of the microbiome in systemic autoimmune diseases. Here, I review current insights gained into the skin, oral, gut, lung, and vascular microbiome in connective tissue diseases and vasculitis. Mechanism relevant to the development and propagation of autoimmunity will be discussed whenever explored. While studies on autoimmune rheumatic disease have almost invariably shown abnormal microbiome structure (dysbiosis), substantial variability in microbial composition between studies makes generalization difficult. Moreover, an etiopathogenic role of specific pathobionts cannot be inferred by association alone. Integrating descriptive studies of microbial communities with hypothesis-driven research informed by immunopathogenesis will be important in elucidating targetable mechanisms in preclinical and established rheumatic disease.
    MeSH term(s) Autoimmune Diseases/microbiology ; Autoimmunity ; Dysbiosis ; Humans ; Lupus Erythematosus, Systemic/microbiology ; Microbiota ; Rheumatic Diseases/microbiology
    Language English
    Publishing date 2020-02-07
    Publishing country Netherlands
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Review
    ZDB-ID 2052323-3
    ISSN 1532-1770 ; 1521-6942
    ISSN (online) 1532-1770
    ISSN 1521-6942
    DOI 10.1016/j.berh.2019.101473
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  6. Article ; Online: More on CD19-CAR T Cells in Systemic Lupus Erythematosus.

    Konig, Maximilian F / Paul, Suman

    The New England journal of medicine

    2021  Volume 385, Issue 19, Page(s) e67

    MeSH term(s) Antigens, CD19 ; Cell Count ; Humans ; Lupus Erythematosus, Systemic ; T-Lymphocytes
    Chemical Substances Antigens, CD19
    Language English
    Publishing date 2021-11-03
    Publishing country United States
    Document type Letter ; Comment
    ZDB-ID 207154-x
    ISSN 1533-4406 ; 0028-4793
    ISSN (online) 1533-4406
    ISSN 0028-4793
    DOI 10.1056/NEJMc2114593
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  7. Article ; Online: Sulfasalazine: a risk factor for severe COVID-19?

    Konig, Maximilian F / Grzes, Katarzyna M / Robinson, Philip C / Pearce, Edward J

    The Lancet. Rheumatology

    2022  Volume 4, Issue 6, Page(s) e388–e389

    Language English
    Publishing date 2022-03-15
    Publishing country England
    Document type Journal Article
    ISSN 2665-9913
    ISSN (online) 2665-9913
    DOI 10.1016/S2665-9913(22)00067-4
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  8. Article ; Online: Microbial pathways to subvert host immunity generate citrullinated neoantigens targeted in rheumatoid arthritis.

    Gómez-Bañuelos, Eduardo / Konig, Maximilian F / Andrade, Felipe

    Current opinion in structural biology

    2022  Volume 75, Page(s) 102423

    Abstract: The specific association between antibodies to citrullinated proteins and rheumatoid arthritis (RA) has centered interest on understanding why citrullinated proteins become immunogenic in this disease, which is believed to inform the origins of ... ...

    Abstract The specific association between antibodies to citrullinated proteins and rheumatoid arthritis (RA) has centered interest on understanding why citrullinated proteins become immunogenic in this disease, which is believed to inform the origins of autoimmunity in RA. Since citrullination is a physiologic post-translational modification (PTM), one theory is that conditions promoting abnormal citrullination are initiators of self-reactive immune responses to citrullinated proteins in RA. Foremost candidates that dysregulate the normal balance of citrullination are microbial agents, which can exploit citrullination as an effector mechanism to subvert host antimicrobial activities and maximize their progeny. Here, we will use the host-pathogen interface as a unifying model to link microbe-induced citrullination and the loss of immunological tolerance to citrullinated antigens in RA.
    MeSH term(s) Arthritis, Rheumatoid/metabolism ; Autoimmunity ; Citrullination ; Citrulline/metabolism ; Humans ; Protein Processing, Post-Translational
    Chemical Substances Citrulline (29VT07BGDA)
    Language English
    Publishing date 2022-07-11
    Publishing country England
    Document type Journal Article ; Review ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 1068353-7
    ISSN 1879-033X ; 0959-440X
    ISSN (online) 1879-033X
    ISSN 0959-440X
    DOI 10.1016/j.sbi.2022.102423
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    Zs.A 3206: Show issues Location:
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  9. Article ; Online: Acute Bilateral Pseudogout of the Temporomandibular Joint.

    Konig, Maximilian F / Faller, Garrey T

    Arthritis & rheumatology (Hoboken, N.J.)

    2020  Volume 72, Issue 7, Page(s) 1159

    MeSH term(s) Acute Disease ; Aged, 80 and over ; Arthralgia/physiopathology ; Blood Sedimentation ; C-Reactive Protein/metabolism ; Chondrocalcinosis/diagnostic imaging ; Chondrocalcinosis/metabolism ; Chondrocalcinosis/pathology ; Chondrocalcinosis/physiopathology ; Female ; Fever/physiopathology ; Humans ; Neck Pain/physiopathology ; Temporomandibular Joint Disorders/diagnostic imaging ; Temporomandibular Joint Disorders/metabolism ; Temporomandibular Joint Disorders/pathology ; Temporomandibular Joint Disorders/physiopathology ; Tomography, X-Ray Computed ; Trismus/physiopathology
    Chemical Substances C-Reactive Protein (9007-41-4)
    Language English
    Publishing date 2020-05-27
    Publishing country United States
    Document type Case Reports ; Journal Article ; Research Support, N.I.H., Extramural
    ZDB-ID 2756371-6
    ISSN 2326-5205 ; 2326-5191
    ISSN (online) 2326-5205
    ISSN 2326-5191
    DOI 10.1002/art.41279
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  10. Article: Editorial: Interstitial Lung Disease in the Context of Systemic Disease: Pathophysiology, Treatment and Outcomes.

    Korsten, Peter / Konig, Maximilian F / Tampe, Björn / Mirsaeidi, Mehdi

    Frontiers in medicine

    2021  Volume 7, Page(s) 644075

    Language English
    Publishing date 2021-01-20
    Publishing country Switzerland
    Document type Editorial
    ZDB-ID 2775999-4
    ISSN 2296-858X
    ISSN 2296-858X
    DOI 10.3389/fmed.2020.644075
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