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Article ; Online: When forgetting what happened at work matters: The role of affective rumination, problem-solving pondering, and self-control in work-family conflict and enrichment.

Junker, Nina M / Baumeister, Roy F / Straub, Kerstin / Greenhaus, Jeffrey H

2020 Volume 106, Issue 11, Page(s) 1750–1766

Abstract: Whether integrating work into home benefits or harms an employee's family role is a critical issue that has met with mixed findings in the extant literature. Work-home integration can be manifested in different ways. Unfortunately, prior research has ... ...

Abstract	Whether integrating work into home benefits or harms an employee's family role is a critical issue that has met with mixed findings in the extant literature. Work-home integration can be manifested in different ways. Unfortunately, prior research has tended to use global assessments of integration that may mask relationships between different types of integration and work-family outcomes. In 2 studies, the present research takes a step toward a more fine-grained analysis by focusing on the work-family consequences of affective rumination and problem-solving pondering, both of which represent psychological integration of work into home. In Study 1, using a between-person design with a 6-week time lag (
MeSH term(s)	Family Conflict ; Humans ; Problem Solving ; Self-Control
Language	English
Publishing date	2020-10-22
Publishing country	United States
Document type	Journal Article
ZDB-ID	219157-x
ISSN	1939-1854 ; 0021-9010
ISSN (online)	1939-1854
ISSN	0021-9010
DOI	10.1037/apl0000847
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Article: Non‐Monotonic Floodplain Responses to Changes in Flooding Intensity

Barefoot, Eric A. / Nittrouer, Jeffrey A. / Straub, Kyle M.

Journal of geophysical research. 2021 Oct., v. 126, no. 10

2021

Abstract: Overbank flooding is common along most rivers, and it influences the dispersal of sediment to floodplains. While variable discharge is a critical aspect of fluvial landscape evolution, it is typically modeled by simplifying the hydrograph to an ... ...

Abstract	Overbank flooding is common along most rivers, and it influences the dispersal of sediment to floodplains. While variable discharge is a critical aspect of fluvial landscape evolution, it is typically modeled by simplifying the hydrograph to an equivalent steady discharge: the channel‐forming discharge. However, for all formulations used to simplify hydrographs, many different inputs can produce the same channel‐forming discharge. Here, we investigate how hydrographs with different flood intensities affect channel mobility, sediment accumulation patterns, and alluvial morphology using a suite of physical experiments where a fan delta grew by dispersing a cohesive sediment mixture into a basin. The experiments spanned three levels: no flooding, low‐intensity flooding, and high‐intensity flooding, while the time‐averaged water and sediment discharge was equivalent between all flooding regimes. Across this gradient, channel mobility, alluvial morphology and sediment dispersal scaled non‐monotonically with flooding intensity, and the data suggest that levee‐building feedbacks are the cause. We found that flood intensity modulates the relative balance between sediment delivery to channel margins, which nourishes levee growth, and the intensity of overbank flow, which inhibits levee growth. When flooding was absent, levees experienced consistent overbank flow and sediment delivery, leading to moderate levee aggradation and sporadic levee breaches. In contrast, when low‐intensity flooding was imposed, levees experienced enhanced sediment delivery by low‐amplitude floods, but only intermittent scouring. A further increase in flood intensity generated intense overbank flows that inhibited levee growth altogether. These results imply the existence of an optimum levee‐building condition, where flooding conditions stabilize channels through levee‐building feedbacks.
Keywords	basins ; floodplains ; geophysics ; hydrograph ; landscapes ; research ; sediment yield ; sediments
Language	English
Dates of publication	2021-10
Publishing place	John Wiley & Sons, Ltd
Document type	Article
Note	JOURNAL ARTICLE
ISSN	2169-9003
DOI	10.1029/2021JF006310
Database	NAL-Catalogue (AGRICOLA)

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Article ; Online: Unconventional life history in a migratory shorebird: desegregating reproduction and migration.

Slezak, Colby R / Blomberg, Erik J / Roth, Amber M / Berigan, Liam A / Fish, Alexander C / Darling, Rachel / Clements, Sarah J / Balkcom, Greg / Carpenter, Bobbi / Costanzo, Gary / Duguay, Jeffrey / Graham, Clayton L / Harvey, William / Hook, Michael / Howell, Douglas L / Maddox, Seth / Meyer, Shawn W / Nichols, Theodore C / Pollard, J Bruce /

Roy, Christian / Stiller, Joshua C / Straub, Jacob N / Tetreault, Mathieu / Tyl, Reina / Williams, Lisa / Kilburn, Jennifer E / McWilliams, Scott R

Proceedings. Biological sciences

2024 Volume 291, Issue 2021, Page(s) 20240021

Abstract: Conventional life-history theory predicts that energy-demanding events such as reproduction and migration must be temporally segregated to avoid resource limitation. Here, we provide, to our knowledge, the first direct evidence of 'itinerant breeding' in ...

Abstract	Conventional life-history theory predicts that energy-demanding events such as reproduction and migration must be temporally segregated to avoid resource limitation. Here, we provide, to our knowledge, the first direct evidence of 'itinerant breeding' in a migratory bird, an incredibly rare breeding strategy (less than 0.1% of extant bird species) that involves the temporal overlap of migratory and reproductive periods of the annual cycle. Based on GPS-tracking of over 200 female American woodcock, most female woodcock (greater than 80%) nested more than once (some up to six times) with short re-nest intervals, and females moved northwards on average 800 km between first and second nests, and then smaller distances (
MeSH term(s)	Animals ; Female ; Seasons ; Reproduction ; Birds ; Ecosystem ; Life History Traits ; Charadriiformes ; Animal Migration
Language	English
Publishing date	2024-04-17
Publishing country	England
Document type	Journal Article
ZDB-ID	209242-6
ISSN	1471-2954 ; 0080-4649 ; 0962-8452 ; 0950-1193
ISSN (online)	1471-2954
ISSN	0080-4649 ; 0962-8452 ; 0950-1193
DOI	10.1098/rspb.2024.0021
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Article ; Online: Highly Conductive and Permeable Nanocomposite Ultrafiltration Membranes Using Laser-Reduced Graphene Oxide.

Straub, Anthony P / Bergsman, David S / Getachew, Bezawit A / Leahy, Liam M / Patil, Jatin J / Ferralis, Nicola / Grossman, Jeffrey C

Nano letters

2021 Volume 21, Issue 6, Page(s) 2429–2435

Abstract: Electrically conductive membranes are a promising avenue to reduce water treatment costs due to their ability to minimize the detrimental impact of fouling, to degrade contaminants, and to provide other additional benefits during filtration. Here, we ... ...

Abstract	Electrically conductive membranes are a promising avenue to reduce water treatment costs due to their ability to minimize the detrimental impact of fouling, to degrade contaminants, and to provide other additional benefits during filtration. Here, we demonstrate the facile and low-cost fabrication of electrically conductive membranes using laser-reduced graphene oxide (GO). In this method, GO is filtered onto a poly(ether sulfone) membrane support before being pyrolyzed via laser into a conductive film. Laser-reduced GO composite membranes are shown to be equally as permeable to water as the underlying membrane support and possess sheet resistances as low as 209 Ω/□. Application of the laser-reduced GO membranes is demonstrated through greater than 97% removal of a surrogate water contaminant, 25 μM methyl orange dye, with an 8 V applied potential. Furthermore, we show that laser-reduced GO membranes can be further tuned with the addition of
Language	English
Publishing date	2021-03-09
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, Non-P.H.S.
ISSN	1530-6992
ISSN (online)	1530-6992
DOI	10.1021/acs.nanolett.0c04512
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Article: The safety of in-office laryngologic procedures during active antithrombotic therapy.

Straub, Jeffrey M / Calamari, Kevin A / Shin, Timothy J / Janse, Sarah A / Forrest, Lowell A / deSilva, Brad W / Matrka, Laura A

Laryngoscope investigative otolaryngology

2020 Volume 5, Issue 5, Page(s) 890–894

Abstract: Objectives: To determine whether patients undergoing in-office laryngologic procedures on antithrombotic therapy are at increased risk for treatment-related complications.: Methods: Patients were those who underwent at least one in-office ... ...

Abstract	Objectives: To determine whether patients undergoing in-office laryngologic procedures on antithrombotic therapy are at increased risk for treatment-related complications. Methods: Patients were those who underwent at least one in-office laryngologic procedure with any of three fellowship-trained laryngologists. Procedures were identified by current procedural terminology (CPT) code and included biopsies, excisions, laser ablations, and injections (therapeutic and augmentative). Patients were divided into two groups based on the use of antithrombotic therapy at the time of their procedure. Retrospective chart review was performed to identify any complications, with an average follow-up of 186 days. Results: Five hundred-sixty-four unique individuals were identified with ages ranging from 18 to 93 years old and with a relatively even distribution between females (45%) and males (55%). They underwent 647 procedures in total, 310 of which were performed while on some form of antithrombotic therapy. Sixteen procedures were associated with complications either during or after the procedure. In comparing overall complication rates, there was no significant difference between non-antithrombotic (2.4%) and antithrombotic (3.3%) cohorts (OR 1.09, 95% CI [0.46-2.60], Conclusions: In spite of known risks in other settings, antithrombotic agents do not appear to confer increased risk of treatment-related complications during in-office laryngologic procedures, obviating the need for cessation of therapy prior to these interventions. Level of evidence: 4.
Language	English
Publishing date	2020-09-02
Publishing country	United States
Document type	Journal Article
ISSN	2378-8038
ISSN	2378-8038
DOI	10.1002/lio2.451
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Article ; Online: Nitric oxide: what's new to NO?

Ghimire, Kedar / Altmann, Helene M / Straub, Adam C / Isenberg, Jeffrey S

American journal of physiology. Cell physiology

2016 Volume 312, Issue 3, Page(s) C254–C262

Abstract: Nitric oxide (NO) is one of the critical components of the vasculature, regulating key signaling pathways in health. In macrovessels, NO functions to suppress cell inflammation as well as adhesion. In this way, it inhibits thrombosis and promotes blood ... ...

Abstract	Nitric oxide (NO) is one of the critical components of the vasculature, regulating key signaling pathways in health. In macrovessels, NO functions to suppress cell inflammation as well as adhesion. In this way, it inhibits thrombosis and promotes blood flow. It also functions to limit vessel constriction and vessel wall remodeling. In microvessels and particularly capillaries, NO, along with growth factors, is important in promoting new vessel formation, a process termed angiogenesis. With age and cardiovascular disease, animal and human studies confirm that NO is dysregulated at multiple levels including decreased production, decreased tissue half-life, and decreased potency. NO has also been implicated in diseases that are related to neurotransmission and cancer although it is likely that these processes involve NO at higher concentrations and from nonvascular cell sources. Conversely, NO and drugs that directly or indirectly increase NO signaling have found clinical applications in both age-related diseases and in younger individuals. This focused review considers recently reported advances being made in the field of NO signaling regulation at several levels including enzymatic production, receptor function, interacting partners, localization of signaling, matrix-cellular and cell-to-cell cross talk, as well as the possible impact these newly described mechanisms have on health and disease.
MeSH term(s)	Animals ; Blood Flow Velocity ; Blood Vessels/physiopathology ; Cardiovascular Diseases/physiopathology ; Humans ; Models, Cardiovascular ; Neovascularization, Physiologic/physiology ; Nitric Oxide/metabolism
Chemical Substances	Nitric Oxide (31C4KY9ESH)
Language	English
Publishing date	2016-12-14
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	392098-7
ISSN	1522-1563 ; 0363-6143
ISSN (online)	1522-1563
ISSN	0363-6143
DOI	10.1152/ajpcell.00315.2016
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Article ; Online: CD47 Promotes Age-Associated Deterioration in Angiogenesis, Blood Flow and Glucose Homeostasis.

Ghimire, Kedar / Li, Yao / Chiba, Takuto / Julovi, Sohel M / Li, Jennifer / Ross, Mark A / Straub, Adam C / O'Connell, Philip J / Rüegg, Curzio / Pagano, Patrick J / Isenberg, Jeffrey S / Rogers, Natasha M

Cells

2020 Volume 9, Issue 7

Abstract: The aged population is currently at its highest level in human history and is expected to increase further in the coming years. In humans, aging is accompanied by impaired angiogenesis, diminished blood flow and altered metabolism, among others. A ... ...

Abstract	The aged population is currently at its highest level in human history and is expected to increase further in the coming years. In humans, aging is accompanied by impaired angiogenesis, diminished blood flow and altered metabolism, among others. A cellular mechanism that impinges upon these manifestations of aging can be a suitable target for therapeutic intervention. Here we identify cell surface receptor CD47 as a novel age-sensitive driver of vascular and metabolic dysfunction. With the natural aging process, CD47 and its ligand thrombospondin-1 were increased, concurrent with a reduction of self-renewal transcription factors OCT4, SOX2, KLF4 and cMYC (OSKM) in arteries from aged wild-type mice and older human subjects compared to younger controls. These perturbations were prevented in arteries from aged CD47-null mice. Arterial endothelial cells isolated from aged wild-type mice displayed cellular exhaustion with decreased proliferation, migration and tube formation compared to cells from aged CD47-null mice. CD47 suppressed ex vivo sprouting, in vivo angiogenesis and skeletal muscle blood flow in aged wild-type mice. Treatment of arteries from older humans with a CD47 blocking antibody mitigated the age-related deterioration in angiogenesis. Finally, aged CD47-null mice were resistant to age- and diet-associated weight gain, glucose intolerance and insulin desensitization. These results indicate that the CD47-mediated signaling maladapts during aging to broadly impair endothelial self-renewal, angiogenesis, perfusion and glucose homeostasis. Our findings provide a strong rationale for therapeutically targeting CD47 to minimize these dysfunctions during aging.
MeSH term(s)	Aging/pathology ; Animals ; Arteries/pathology ; CD47 Antigen/metabolism ; Cell Movement/genetics ; Cell Proliferation/genetics ; Cell Self Renewal ; Endothelial Cells/metabolism ; Endothelial Cells/pathology ; Gene Expression Regulation ; Glucose/metabolism ; Homeostasis ; Humans ; Male ; Matrix Metalloproteinases/metabolism ; Metabolic Syndrome/pathology ; Mice, Inbred C57BL ; Neovascularization, Physiologic/genetics ; Regional Blood Flow ; Thrombospondin 1/metabolism ; Transcription Factors/metabolism
Chemical Substances	CD47 Antigen ; Thrombospondin 1 ; Transcription Factors ; Matrix Metalloproteinases (EC 3.4.24.-) ; Glucose (IY9XDZ35W2)
Language	English
Publishing date	2020-07-15
Publishing country	Switzerland
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	2661518-6
ISSN	2073-4409 ; 2073-4409
ISSN (online)	2073-4409
ISSN	2073-4409
DOI	10.3390/cells9071695
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Article ; Online: As in Real Estate, Location Matters: Cellular Expression of Complement Varies Between Macular and Peripheral Regions of the Retina and Supporting Tissues.

Frontiers in immunology

2022 Volume 13, Page(s) 895519

Abstract: The cellular events that dictate the initiation of the complement pathway in ocular degeneration, such as age-related macular degeneration (AMD), is poorly understood. Using gene expression analysis (single cell and bulk), mass spectrometry, and ... ...

Abstract	The cellular events that dictate the initiation of the complement pathway in ocular degeneration, such as age-related macular degeneration (AMD), is poorly understood. Using gene expression analysis (single cell and bulk), mass spectrometry, and immunohistochemistry, we dissected the role of multiple retinal and choroidal cell types in determining the complement homeostasis. Our scRNA-seq data show that the cellular response to early AMD is more robust in the choroid, particularly in fibroblasts, pericytes and endothelial cells. In late AMD, complement changes were more prominent in the retina especially with the expression of the classical pathway initiators. Notably, we found a spatial preference for these differences. Overall, this study provides insights into the heterogeneity of cellular responses for complement expression and the cooperation of neighboring cells to complete the pathway in healthy and AMD eyes. Further, our findings provide new cellular targets for therapies directed at complement.
MeSH term(s)	Choroid ; Complement System Proteins ; Endothelial Cells ; Humans ; Macular Degeneration/genetics ; Retina
Chemical Substances	Complement System Proteins (9007-36-7)
Language	English
Publishing date	2022-06-15
Publishing country	Switzerland
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	2606827-8
ISSN	1664-3224 ; 1664-3224
ISSN (online)	1664-3224
ISSN	1664-3224
DOI	10.3389/fimmu.2022.895519
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Article ; Online: CD47 Promotes Age-Associated Deterioration in Angiogenesis, Blood Flow and Glucose Homeostasis

Kedar Ghimire / Yao Li / Takuto Chiba / Sohel M. Julovi / Jennifer Li / Mark A. Ross / Adam C. Straub / Philip J. O’Connell / Curzio Rüegg / Patrick J. Pagano / Jeffrey S. Isenberg / Natasha M. Rogers

Cells, Vol 9, Iss 1695, p

2020 Volume 1695

Abstract	The aged population is currently at its highest level in human history and is expected to increase further in the coming years. In humans, aging is accompanied by impaired angiogenesis, diminished blood flow and altered metabolism, among others. A cellular mechanism that impinges upon these manifestations of aging can be a suitable target for therapeutic intervention. Here we identify cell surface receptor CD47 as a novel age-sensitive driver of vascular and metabolic dysfunction. With the natural aging process, CD47 and its ligand thrombospondin-1 were increased, concurrent with a reduction of self-renewal transcription factors OCT4, SOX2, KLF4 and cMYC (OSKM) in arteries from aged wild-type mice and older human subjects compared to younger controls. These perturbations were prevented in arteries from aged CD47-null mice. Arterial endothelial cells isolated from aged wild-type mice displayed cellular exhaustion with decreased proliferation, migration and tube formation compared to cells from aged CD47-null mice. CD47 suppressed ex vivo sprouting, in vivo angiogenesis and skeletal muscle blood flow in aged wild-type mice. Treatment of arteries from older humans with a CD47 blocking antibody mitigated the age-related deterioration in angiogenesis. Finally, aged CD47-null mice were resistant to age- and diet-associated weight gain, glucose intolerance and insulin desensitization. These results indicate that the CD47-mediated signaling maladapts during aging to broadly impair endothelial self-renewal, angiogenesis, perfusion and glucose homeostasis. Our findings provide a strong rationale for therapeutically targeting CD47 to minimize these dysfunctions during aging.
Keywords	aging ; angiogenesis ; CD47 ; thrombospondin-1 ; glucose homeostasis ; metabolism ; Biology (General) ; QH301-705.5
Subject code	610
Language	English
Publishing date	2020-07-01T00:00:00Z
Publisher	MDPI AG
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: Radiation-induced fibrosis: mechanisms and implications for therapy.

Straub, Jeffrey M / New, Jacob / Hamilton, Chase D / Lominska, Chris / Shnayder, Yelizaveta / Thomas, Sufi M

Journal of cancer research and clinical oncology

2015 Volume 141, Issue 11, Page(s) 1985–1994

Abstract: Purpose: Radiation-induced fibrosis (RIF) is a long-term side effect of external beam radiation therapy for the treatment of cancer. It results in a multitude of symptoms that significantly impact quality of life. Understanding the mechanisms of RIF- ... ...

Abstract	Purpose: Radiation-induced fibrosis (RIF) is a long-term side effect of external beam radiation therapy for the treatment of cancer. It results in a multitude of symptoms that significantly impact quality of life. Understanding the mechanisms of RIF-induced changes is essential to developing effective strategies to prevent long-term disability and discomfort following radiation therapy. In this review, we describe the current understanding of the etiology, clinical presentation, pathogenesis, treatment, and directions of future therapy for this condition. Methods: A literature review of publications describing mechanisms or treatments of RIF was performed. Specific databases utilized included PubMed and clinicaltrials.gov, using keywords "Radiation-Induced Fibrosis," "Radiotherapy Complications," "Fibrosis Therapy," and other closely related terms. Results: RIF is the result of a misguided wound healing response. In addition to causing direct DNA damage, ionizing radiation generates reactive oxygen and nitrogen species that lead to localized inflammation. This inflammatory process ultimately evolves into a fibrotic one characterized by increased collagen deposition, poor vascularity, and scarring. Tumor growth factor beta serves as the primary mediator in this response along with a host of other cytokines and growth factors. Current therapies have largely been directed toward these molecular targets and their associated signaling pathways. Conclusion: Although RIF is widely prevalent among patients undergoing radiation therapy and significantly impacts quality of life, there is still much to learn about its pathogenesis and mechanisms. Current treatments have stemmed from this understanding, and it is anticipated that further elucidation will be essential for the development of more effective therapies.
MeSH term(s)	DNA Damage/radiation effects ; Fibrosis ; Humans ; Inflammation/etiology ; Inflammation/metabolism ; Neoplasms/radiotherapy ; Radiation Injuries/etiology ; Radiation Injuries/physiopathology ; Radiation Injuries/therapy ; Radiotherapy/adverse effects ; Transforming Growth Factor beta/metabolism
Chemical Substances	Transforming Growth Factor beta
Language	English
Publishing date	2015-11
Publishing country	Germany
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	134792-5
ISSN	1432-1335 ; 0171-5216 ; 0084-5353 ; 0943-9382
ISSN (online)	1432-1335
ISSN	0171-5216 ; 0084-5353 ; 0943-9382
DOI	10.1007/s00432-015-1974-6
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