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Book ; Thesis: Untersuchung der durch CEACAM1-4L vermittelten Angiogenese in Mausmodellen

Lovric, Svjetlana

2012

Author's details	vorgelegt von Svjetlana Lovric
Language	German
Size	90 Bl. : Ill., graph. Darst.
Publishing country	Germany
Document type	Book ; Thesis
Thesis / German Habilitation thesis	Hamburg, Univ., Diss., 2013
HBZ-ID	HT018702083
Database	Catalogue ZB MED Medicine, Health

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Article ; Online: Epithelial cell states associated with kidney and allograft injury.

Hinze, Christian / Lovric, Svjetlana / Halloran, Philip F / Barasch, Jonathan / Schmidt-Ott, Kai M

Nature reviews. Nephrology

2024

Abstract: The kidney epithelium, with its intricate arrangement of highly specialized cell types, constitutes the functional core of the organ. Loss of kidney epithelium is linked to the loss of functional nephrons and a subsequent decline in kidney function. In ... ...

Abstract	The kidney epithelium, with its intricate arrangement of highly specialized cell types, constitutes the functional core of the organ. Loss of kidney epithelium is linked to the loss of functional nephrons and a subsequent decline in kidney function. In kidney transplantation, epithelial injury signatures observed during post-transplantation surveillance are strong predictors of adverse kidney allograft outcomes. However, epithelial injury is currently neither monitored clinically nor addressed therapeutically after kidney transplantation. Several factors can contribute to allograft epithelial injury, including allograft rejection, drug toxicity, recurrent infections and postrenal obstruction. The injury mechanisms that underlie allograft injury overlap partially with those associated with acute kidney injury (AKI) and chronic kidney disease (CKD) in the native kidney. Studies using advanced transcriptomic analyses of single cells from kidney or urine have identified a role for kidney injury-induced epithelial cell states in exacerbating and sustaining damage in AKI and CKD. These epithelial cell states and their associated expression signatures are also observed in transplanted kidney allografts, suggesting that the identification and characterization of transcriptomic epithelial cell states in kidney allografts may have potential clinical implications for diagnosis and therapy.
Language	English
Publishing date	2024-04-17
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	2490366-8
ISSN	1759-507X ; 1759-5061
ISSN (online)	1759-507X
ISSN	1759-5061
DOI	10.1038/s41581-024-00834-0
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Zs.MG 107: Show issues

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Book ; Thesis: Untersuchung der durch CEACAM1-4L vermittelten Angiogenese in Mausmodellen

Lovric, Svjetlana

2012

Author's details	vorgelegt von Svjetlana Lovric
Language	German
Size	90 Bl., Ill., graph. Darst.
Document type	Book ; Thesis
Thesis / German Habilitation thesis	Univ., FB Medizin, Diss.--Hamburg, 2013
Database	Former special subject collection: coastal and deep sea fishing

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Article ; Online: 135. godišnjica rođenja Frana Bubanovića

Kristina Mlinac-Jerković / Vladimir Damjanović / Svjetlana Kalanj-Bognar / Jasna Lovrić

Kemija u Industriji, Vol 67, Iss 9-10, Pp 403-

slike iz života

2018 Volume 408

Abstract: U ovom radu prikazane su crtice iz života jednog od velikana hrvatskog prirodoslovlja, kemičara Frana Bubanovića. Ove 2018. godine obilježavamo 135 godina od njegova rođenja, 100 godina postojanja Zavoda za kemiju i biokemiju Medicinskog fakulteta ... ...

Abstract	U ovom radu prikazane su crtice iz života jednog od velikana hrvatskog prirodoslovlja, kemičara Frana Bubanovića. Ove 2018. godine obilježavamo 135 godina od njegova rođenja, 100 godina postojanja Zavoda za kemiju i biokemiju Medicinskog fakulteta Sveučilišta u Zagrebu, čiji je osnivač, te 100 godina od izdavanja njegove znanstveno-popularne knjige “Kemija živih bića”. Kroz godine napisan je velik broj tekstova o njegovom doprinosu popularizaciji znanosti, znanstvenom opusu, njegovom nastavničkom pozivu kao i sveučilišnim udžbenicima. Ovim tekstom želimo se osvrnuti na pojedine zanimljive trenutke njegova života te prikazati nekoliko dosad neobjavljenih dokumenata i fotografija ustupljenih od njegove obitelji.
Keywords	Fran Bubanović ; medicinska kemija i biokemija ; popularizacija znanosti ; Chemistry ; QD1-999
Language	English
Publishing date	2018-09-01T00:00:00Z
Publisher	Croatian Society of Chemical Engineers
Document type	Article ; Online
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Article ; Online: Treatment of autoimmunity: The impact of disease-modifying therapies in multiple sclerosis and comorbid autoimmune disorders.

Konen, Franz Felix / Möhn, Nora / Witte, Torsten / Schefzyk, Matthias / Wiestler, Miriam / Lovric, Svjetlana / Hufendiek, Karsten / Schwenkenbecher, Philipp / Sühs, Kurt-Wolfram / Friese, Manuel A / Klotz, Luisa / Pul, Refik / Pawlitzki, Marc / Hagin, David / Kleinschnitz, Christoph / Meuth, Sven G / Skripuletz, Thomas

Autoimmunity reviews

2023 Volume 22, Issue 5, Page(s) 103312

Abstract: More than 10 disease-modifying therapies (DMT) are approved by the European Medicines Agency (EMA) and the US Food and Drug Administration (FDA) for the treatment of multiple sclerosis (MS) and new therapeutic options are on the horizon. Due to different ...

Abstract	More than 10 disease-modifying therapies (DMT) are approved by the European Medicines Agency (EMA) and the US Food and Drug Administration (FDA) for the treatment of multiple sclerosis (MS) and new therapeutic options are on the horizon. Due to different underlying therapeutic mechanisms, a more individualized selection of DMTs in MS is possible, taking into account the patient's current situation. Therefore, concomitant treatment of various comorbid conditions, including autoimmune mediated disorders such as rheumatoid arthritis, should be considered in MS patients. Because the pathomechanisms of autoimmunity partially overlap, DMT could also treat concomitant inflammatory diseases and simplify the patient's treatment. In contrast, the exacerbation and even new occurrence of several autoimmune diseases have been reported as a result of immunomodulatory treatment of MS. To simplify treatment and avoid disease exacerbation, knowledge of the beneficial and adverse effects of DMT in other autoimmune disorders is critical. Therefore, we conducted a literature search and described the beneficial and adverse effects of approved and currently studied DMT in a large number of comorbid autoimmune diseases, including rheumatoid arthritis, ankylosing spondylitis, inflammatory bowel diseases, cutaneous disorders including psoriasis, Sjögren´s syndrome, systemic lupus erythematosus, systemic vasculitis, autoimmune hepatitis, and ocular autoimmune disorders. Our review aims to facilitate the selection of an appropriate DMT in patients with MS and comorbid autoimmune diseases.
MeSH term(s)	Humans ; Multiple Sclerosis/complications ; Multiple Sclerosis/drug therapy ; Multiple Sclerosis/epidemiology ; Autoimmunity ; Autoimmune Diseases/complications ; Autoimmune Diseases/drug therapy ; Arthritis, Rheumatoid ; Lupus Erythematosus, Systemic
Language	English
Publishing date	2023-03-15
Publishing country	Netherlands
Document type	Journal Article ; Review
ZDB-ID	2144145-5
ISSN	1873-0183 ; 1568-9972
ISSN (online)	1873-0183
ISSN	1568-9972
DOI	10.1016/j.autrev.2023.103312
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Article ; Online: Genetic testing in steroid-resistant nephrotic syndrome: when and how?

Lovric, Svjetlana / Ashraf, Shazia / Tan, Weizhen / Hildebrandt, Friedhelm

Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association

2015 Volume 31, Issue 11, Page(s) 1802–1813

Abstract: Steroid-resistant nephrotic syndrome (SRNS) represents the second most frequent cause of chronic kidney disease in the first three decades of life. It manifests histologically as focal segmental glomerulosclerosis (FSGS) and carries a 33% risk of relapse ...

Abstract	Steroid-resistant nephrotic syndrome (SRNS) represents the second most frequent cause of chronic kidney disease in the first three decades of life. It manifests histologically as focal segmental glomerulosclerosis (FSGS) and carries a 33% risk of relapse in a renal transplant. No efficient treatment exists. Identification of single-gene (monogenic) causes of SRNS has moved the glomerular epithelial cell (podocyte) to the center of its pathogenesis. Recently, mutations in >30 recessive or dominant genes were identified as causing monogenic forms of SRNS, thereby revealing the encoded proteins as essential for glomerular function. These findings helped define protein interaction complexes and functional pathways that could be targeted for treatment of SRNS. Very recently, it was discovered that in the surprisingly high fraction of ∼30% of all individuals who manifest with SRNS before 25 years of age, a causative mutation can be detected in one of the ∼30 different SRNS-causing genes. These findings revealed that SRNS and FSGS are not single disease entities but rather are part of a spectrum of distinct diseases with an identifiable genetic etiology. Mutation analysis should be offered to all individuals who manifest with SRNS before the age of 25 years, because (i) it will provide the patient and families with an unequivocal cause-based diagnosis, (ii) it may uncover a form of SRNS that is amenable to treatment (e.g. coenzyme Q
MeSH term(s)	DNA Mutational Analysis ; Genetic Markers/genetics ; Genetic Testing/methods ; Humans ; Mutation/genetics ; Nephrotic Syndrome/congenital ; Nephrotic Syndrome/diagnosis ; Nephrotic Syndrome/genetics
Chemical Substances	Genetic Markers
Language	English
Publishing date	2015-10-27
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	90594-x
ISSN	1460-2385 ; 0931-0509
ISSN (online)	1460-2385
ISSN	0931-0509
DOI	10.1093/ndt/gfv355
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Zs.A 2198: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (1.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 329: Show issues

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Article ; Online: Steroid-Resistant Nephrotic Syndrome-Associated

Liu, Pei-Ju / Gunther, Laura K / Garone, Michael E / Zhang, Chunling / Perez, Diana / Bi-Karchin, Jing / Pellenz, Christopher D / Chase, Sharon E / Presti, Maria F / Plante, Eric L / Martin, Claire E / Lovric, Svjetlana / Yengo, Christopher M / Hildebrandt, Friedhelm / Krendel, Mira

Journal of the American Society of Nephrology : JASN

2022 Volume 33, Issue 11, Page(s) 1989–2007

Abstract: Background: Myo1e is a nonmuscle motor protein enriched in podocytes. Mutations in : Methods: EGFP-tagged human : Results: Both mutants were expressed as full-length proteins in the Myo1e-KO cells. However, unlike wild-type (WT) Myo1e, the T119I ... ...

Abstract	Background: Myo1e is a nonmuscle motor protein enriched in podocytes. Mutations in Methods: EGFP-tagged human Results: Both mutants were expressed as full-length proteins in the Myo1e-KO cells. However, unlike wild-type (WT) Myo1e, the T119I variant was not enriched at the cell junctions or clathrin-coated vesicles (CCVs). In contrast, D388H variant localization was similar to that of WT. The rate of dissociation of the D388H variant from cell-cell junctions and CCVs was decreased, suggesting this mutation affects Myo1e interactions with binding partners. ATPase activity and ability to translocate actin filaments were drastically reduced for the D388H mutant, supporting findings from cell-based experiments. Conclusions: T119I and D388H mutations are deleterious to Myo1e functions. The experimental approaches used in this study can be applied to future characterization of novel
MeSH term(s)	Animals ; Humans ; Mice ; Mutation ; Myosin Type I/genetics ; Myosin Type I/metabolism ; Nephrotic Syndrome/genetics ; Steroids
Chemical Substances	Myosin Type I (EC 3.6.1.-) ; Steroids ; MYO1E protein, human (EC 3.6.1.-)
Language	English
Publishing date	2022-09-09
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	1085942-1
ISSN	1533-3450 ; 1046-6673
ISSN (online)	1533-3450
ISSN	1046-6673
DOI	10.1681/ASN.2021111505
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Article ; Online: Long-term B cell depletion associates with regeneration of kidney function.

Fleig, Susanne V / Konen, Franz F / Schröder, Christoph / Schmitz, Jessica / Gingele, Stefan / Bräsen, Jan H / Lovric, Svjetlana / Schmidt, Bernhard M W / Haller, Hermann / Skripuletz, Thomas / von Vietinghoff, Sibylle

Immunity, inflammation and disease

2021 Volume 9, Issue 4, Page(s) 1479–1488

Abstract: Background: Chronic kidney disease (CKD) is a common condition that increases mortality and the risk of cardiovascular and other morbidities regardless of underlying renal condition. Chronic inflammation promotes renal fibrosis. Recently, renal B cell ... ...

Abstract	Background: Chronic kidney disease (CKD) is a common condition that increases mortality and the risk of cardiovascular and other morbidities regardless of underlying renal condition. Chronic inflammation promotes renal fibrosis. Recently, renal B cell infiltrates were described in chronic kidney disease of various etiologies beyond autoimmunity. Methods: We here investigated B cells and indicators of tertiary lymphoid structure formation in human renal biopsies. Renal function was studied during long-term B cell depletion in human patients with membranous nephropathy and with CKD of unknown origin. Results: Cytokine profiles of tertiary lymphoid structure formation were detected in human renal interstitium in a range of kidney diseases. Complex B cell structures consistent with tertiary lymphoid organ formation were evident in human membranous nephropathy. Here, B cell density did not significantly associate with proteinuria severity, but with worse excretory renal function. Proteinuria responses mostly occurred within the first 6 months of B cell depletion. In contrast, recovery of excretory kidney function was observed only after 18 months of continuous therapy, consistent with a structural process. Renal tertiary lymphatic structures were also detected in the absence of autoimmune kidney disease. To start to address whether B cell depletion may affect CKD in a broader population, we assessed kidney function in neurologic patients with CKD of unknown origin. In this cohort, eGFR significantly increased within 24 months of B cell depletion. Conclusion: Long-term B cell depletion associated with significant improvement of excretory kidney function in human CKD. Kinetics and mechanisms of renal B cell aggregation should be investigated further to stratify the impact of B cells and their aggregates as therapeutic targets.
MeSH term(s)	Cohort Studies ; Disease Progression ; Humans ; Kidney ; Regeneration ; Renal Insufficiency, Chronic
Language	English
Publishing date	2021-07-29
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ISSN	2050-4527
ISSN (online)	2050-4527
DOI	10.1002/iid3.499
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Article ; Online: Osteopontin in antineutrophil cytoplasmic autoantibody-associated vasculitis: relation to disease activity, organ manifestation and immunosuppressive therapy.

Lorenzen, Johan / Lovric, Svjetlana / Krämer, Robert / Haller, Hermann / Haubitz, Marion

Annals of the rheumatic diseases

2010 Volume 69, Issue 6, Page(s) 1169–1171

Abstract: Background: Osteopontin is a pleiotropic cytokine involved in the recruitment and retention of neutrophils to sites of inflammation, which are the primary targets cells in antineutrophil cytoplasmic autoantibody-associated vasculitis (AAV). Osteopontin ... ...

Abstract	Background: Osteopontin is a pleiotropic cytokine involved in the recruitment and retention of neutrophils to sites of inflammation, which are the primary targets cells in antineutrophil cytoplasmic autoantibody-associated vasculitis (AAV). Osteopontin may play a role in the pathogenesis of AAV. Methods: 24 patients with systemic AAV and six patients with limited granulomatous disease were included. 19 patients were followed up at 6 and 12 months after the initiation of immunosuppressive therapy. 21 matched healthy volunteers and 20 body mass index and glomerular filtration rate-matched patients with IgA nephropathy were included as controls. Plasma levels of osteopontin were measured by ELISA. Disease activity was gauged by the Birmingham vasculitis activity score (BVAS) and C-reactive protein (CRP). Results: Osteopontin levels are elevated compared with controls (healthy p<0.001; IgA p<0.001).Osteopontin levels decrease significantly during follow-up (p=0.02). Osteopontin levels correlate with disease activity (BVAS r=0.93; CRP r=0.73; all p<0.001) as well as erythrocyturia (r=0.7, p<0.001) and proteinuria (r=0.54, p=0.007). Conclusions: Active AAV is characterised by increased plasma levels of osteopontin, which decrease dramatically with successful therapy. Osteopontin may mediate the inflammatory process in AAV through the recruitment of neutrophils.
MeSH term(s)	Adult ; Aged ; Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/blood ; Anti-Neutrophil Cytoplasmic Antibody-Associated Vasculitis/drug therapy ; Antibodies, Antineutrophil Cytoplasmic/blood ; Biomarkers/blood ; Body Mass Index ; C-Reactive Protein/metabolism ; Female ; Follow-Up Studies ; Glomerular Filtration Rate ; Humans ; Immunosuppressive Agents/therapeutic use ; Male ; Middle Aged ; Osteopontin/blood ; Severity of Illness Index
Chemical Substances	Antibodies, Antineutrophil Cytoplasmic ; Biomarkers ; Immunosuppressive Agents ; Osteopontin (106441-73-0) ; C-Reactive Protein (9007-41-4)
Language	English
Publishing date	2010-06
Publishing country	England
Document type	Journal Article
ZDB-ID	7090-7
ISSN	1468-2060 ; 0003-4967
ISSN (online)	1468-2060
ISSN	0003-4967
DOI	10.1136/ard.2009.113621
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Zs.MO 167: Show issues

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Article ; Online: Spectrum of mutations in Chinese children with steroid-resistant nephrotic syndrome.

Wang, Fang / Zhang, Yanqin / Mao, Jianhua / Yu, Zihua / Yi, Zhuwen / Yu, Li / Sun, Jun / Wei, Xiuxiu / Ding, Fangrui / Zhang, Hongwen / Xiao, Huijie / Yao, Yong / Tan, Weizhen / Lovric, Svjetlana / Ding, Jie / Hildebrandt, Friedhelm

Pediatric nephrology (Berlin, Germany)

2017 Volume 32, Issue 7, Page(s) 1181–1192

Abstract: Background: The aim of this study was to elucidate whether genetic screening test results of pediatric patients with steroid-resistant nephrotic syndrome (SRNS) vary with ethnicity.: Methods: Using high-throughput DNA sequencing, 28 nephrotic ... ...

Abstract	Background: The aim of this study was to elucidate whether genetic screening test results of pediatric patients with steroid-resistant nephrotic syndrome (SRNS) vary with ethnicity. Methods: Using high-throughput DNA sequencing, 28 nephrotic syndrome-related genes were analyzed in 110 chil-dren affected by SRNS and 10 children with isolated proteinuria enrolled by 5 centers in China (67 boys, 53 girls). Their age at disease onset ranged from 1 day to 208 months (median, 48.8 months). Patients were excluded if their age at onset of disease was over 18 years or if they were diagnosed as having Alport syndrome. Results: A genetic etiology was identified in 28.3% of our cohort and the likelihood of establishing a genetic diagnosis decreased as the age at onset of nephrotic syndrome increased. The most common mutated genes were ADCK4 (6.67%), NPHS1 (5.83%), WT1 (5.83%), and NPHS2 (3.33%), and the difference in the frequencies of ADCK4 and NPHS2 mutations between this study and a study on monogenic causes of SRNS in the largest international cohort of 1,783 different families was significant. A case of congenital nephrotic syndrome was attributed to a homozygous missense mutation in ADCK4, and a de novo missense mutation in TRPC6 was detected in a case of infantile nephrotic syndrome. Conclusions: Our results showed that, in the first and the largest multicenter cohort of Chinese pediatric SRNS reported to date, ADCK4 is the most common causative gene, whereas there is a low prevalence of NPHS2 mutations. Our data indicated that the genetic testing results for pediatric SRNS patients vary with different ethnicities, and this information will help to improve management of the disease in clinical practice.
MeSH term(s)	Adolescent ; Age of Onset ; Asian Continental Ancestry Group/genetics ; Child ; Child, Preschool ; China ; Cohort Studies ; DNA Mutational Analysis/methods ; Drug Resistance/genetics ; Female ; Genetic Predisposition to Disease ; Genetic Testing/methods ; Glucocorticoids/pharmacology ; Glucocorticoids/therapeutic use ; High-Throughput Nucleotide Sequencing ; Homozygote ; Humans ; Infant ; Infant, Newborn ; Intracellular Signaling Peptides and Proteins/genetics ; Male ; Membrane Proteins/genetics ; Mutation, Missense ; Nephrotic Syndrome/congenital ; Nephrotic Syndrome/drug therapy ; Nephrotic Syndrome/genetics ; Protein Kinases/genetics ; Proteinuria/genetics ; Sequence Analysis, DNA/methods ; TRPC6 Cation Channel/genetics ; WT1 Proteins/genetics
Chemical Substances	Glucocorticoids ; Intracellular Signaling Peptides and Proteins ; Membrane Proteins ; NPHS2 protein ; TRPC6 Cation Channel ; TRPC6 protein, human ; WT1 Proteins ; WT1 protein, human ; nephrin ; COQ8B protein, human (EC 2.7.-) ; Protein Kinases (EC 2.7.-)
Language	English
Publishing date	2017-02-15
Publishing country	Germany
Document type	Journal Article ; Multicenter Study
ZDB-ID	631932-4
ISSN	1432-198X ; 0931-041X
ISSN (online)	1432-198X
ISSN	0931-041X
DOI	10.1007/s00467-017-3590-y
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