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  1. Article: 60 Second With Eileen White.

    White, Eileen

    Nursing times

    2016  Volume 112, Issue 14, Page(s) 24

    MeSH term(s) Attitude of Health Personnel ; England ; Hospice and Palliative Care Nursing ; Humans ; Job Satisfaction ; Nurses/psychology
    Language English
    Publishing date 2016-04
    Publishing country England
    Document type Interview
    ZDB-ID 391202-4
    ISSN 0954-7762 ; 0029-6589
    ISSN 0954-7762 ; 0029-6589
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  2. Article ; Online: Q&A: Eileen White on understanding autophagy.

    White, Eileen

    Cancer discovery

    2012  Volume 2, Issue 10, Page(s) 863

    MeSH term(s) Animals ; Antineoplastic Agents/therapeutic use ; Autophagy/drug effects ; Autophagy/physiology ; Cell Hypoxia ; Humans ; Mice ; Neoplasms/drug therapy ; Neoplasms/pathology ; Neoplasms/physiopathology
    Chemical Substances Antineoplastic Agents
    Language English
    Publishing date 2012-10
    Publishing country United States
    Document type Interview
    ZDB-ID 2625242-9
    ISSN 2159-8290 ; 2159-8274
    ISSN (online) 2159-8290
    ISSN 2159-8274
    DOI 10.1158/2159-8290.CD-ND2012-036
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article ; Online: Apoptosis, autophagy, and cancer: the critical role

    White, Eileen

    Genes & development

    2023  Volume 37, Issue 1-2, Page(s) 59–62

    MeSH term(s) Humans ; Apoptosis/genetics ; Neoplasms/genetics ; Autophagy/genetics ; Cell Line, Tumor
    Language English
    Publishing date 2023-04-16
    Publishing country United States
    Document type Journal Article
    ZDB-ID 806684-x
    ISSN 1549-5477 ; 0890-9369
    ISSN (online) 1549-5477
    ISSN 0890-9369
    DOI 10.1101/gad.350443.123
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Article ; Online: Role of Tumor Cell Intrinsic and Host Autophagy in Cancer.

    Guo, Jessie Yanxiang / White, Eileen

    Cold Spring Harbor perspectives in medicine

    2024  

    Abstract: Macroautophagy (autophagy hereafter) is an intracellular nutrient scavenging pathway induced by starvation and other stressors whereby cellular components such as organelles are captured in double-membrane vesicles (autophagosomes), whereupon their ... ...

    Abstract Macroautophagy (autophagy hereafter) is an intracellular nutrient scavenging pathway induced by starvation and other stressors whereby cellular components such as organelles are captured in double-membrane vesicles (autophagosomes), whereupon their contents are degraded through fusion with lysosomes. Two main purposes of autophagy are to recycle the intracellular breakdown products to sustain metabolism and survival during starvation and to eliminate damaged or excess cellular components to suppress inflammation and maintain homeostasis. In contrast to most normal cells and tissues in the fed state, tumor cells up-regulate autophagy to promote their growth, survival, and malignancy. This tumor-cell-autonomous autophagy supports elevated metabolic demand and suppresses tumoricidal activation of the innate and adaptive immune responses. Tumor-cell-nonautonomous (e.g., host) autophagy also supports tumor growth by maintaining essential tumor nutrients in the circulation and tumor microenvironment and by suppressing an antitumor immune response. In the setting of cancer therapy, autophagy is a resistance mechanism to chemotherapy, targeted therapy, and immunotherapy. Thus, tumor and host autophagy are protumorigenic and autophagy inhibition is being examined as a novel therapeutic approach to treat cancer.
    Language English
    Publishing date 2024-01-22
    Publishing country United States
    Document type Journal Article
    ISSN 2157-1422
    ISSN (online) 2157-1422
    DOI 10.1101/cshperspect.a041539
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  5. Book: Programmed cell death in cancer progression and therapy

    Khosravi-Far, Roya / White, Eileen

    (Advances in experimental medicine and biology ; 615)

    2008  

    Author's details Roya Khosravi-Far ; Eileen White
    Series title Advances in experimental medicine and biology ; 615
    Collection
    Language English
    Publisher Springer
    Publishing place New York u.a.
    Publishing country United States
    Document type Book
    HBZ-ID HT015418803
    ISBN 978-1-4020-6553-8 ; 9781402065545 ; 1-4020-6553-1 ; 140206554X
    Database Catalogue ZB MED Medicine, Health

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    Shelf markLoan statusLocation
    Zs A 3192 -615- not available for loan Zeitschriften-Lesesaal

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  6. Article ; Online: Blockade of RAF and autophagy is the one-two punch to take out Ras.

    White, Eileen

    Proceedings of the National Academy of Sciences of the United States of America

    2019  Volume 116, Issue 10, Page(s) 3965–3967

    MeSH term(s) Autophagy ; Cell Survival ; Humans ; Mitogen-Activated Protein Kinases ; Neoplasms
    Chemical Substances Mitogen-Activated Protein Kinases (EC 2.7.11.24)
    Language English
    Publishing date 2019-02-19
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Comment
    ZDB-ID 209104-5
    ISSN 1091-6490 ; 0027-8424
    ISSN (online) 1091-6490
    ISSN 0027-8424
    DOI 10.1073/pnas.1900800116
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  7. Article ; Online: Autophagy in PDGFRA

    Yang, Yang / White, Eileen

    Autophagy

    2022  Volume 19, Issue 2, Page(s) 726–728

    Abstract: Macroautophagy/autophagy defects are a risk factor for inflamatory bowel disease (IBD), but the mechanism remains unclear. We previously demonstrated that conditional whole-body deletion of the ... ...

    Abstract Macroautophagy/autophagy defects are a risk factor for inflamatory bowel disease (IBD), but the mechanism remains unclear. We previously demonstrated that conditional whole-body deletion of the essential
    MeSH term(s) Mice ; Animals ; Autophagy/physiology ; Intestines ; Stem Cells/metabolism ; Autophagy-Related Protein 7/genetics ; Autophagy-Related Protein 7/metabolism ; Autophagy-Related Protein 5/genetics ; Receptor Protein-Tyrosine Kinases ; Mammals/metabolism ; Homeostasis
    Chemical Substances Autophagy-Related Protein 7 (EC 6.2.1.45) ; Autophagy-Related Protein 5 ; Receptor Protein-Tyrosine Kinases (EC 2.7.10.1)
    Language English
    Publishing date 2022-07-06
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 2454135-7
    ISSN 1554-8635 ; 1554-8627
    ISSN (online) 1554-8635
    ISSN 1554-8627
    DOI 10.1080/15548627.2022.2090694
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  8. Article ; Online: The presidential partnership: leaving a trail.

    White, Kenneth / Sullivan-Marx, Eileen

    Nursing outlook

    2021  Volume 69, Issue 6, Page(s) 935–936

    MeSH term(s) Cooperative Behavior ; Creativity ; Female ; Humans ; Leadership ; Male ; Organizational Innovation
    Language English
    Publishing date 2021-12-11
    Publishing country United States
    Document type Editorial
    ZDB-ID 300568-9
    ISSN 1528-3968 ; 0029-6554
    ISSN (online) 1528-3968
    ISSN 0029-6554
    DOI 10.1016/j.outlook.2021.10.001
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  9. Article ; Online: MDVs to the rescue: How autophagy-deficient cancer cells adapt to defective mitophagy.

    Poillet-Perez, Laura / White, Eileen

    Developmental cell

    2021  Volume 56, Issue 14, Page(s) 2010–2012

    Abstract: Cancers are dependent on mitochondria, the powerhouse of the cell, and autophagy, the mechanism to preserve mitochondrial quality and function. In this issue of Developmental Cell, Towers et al. identify mitochondria-derived vesicles (MDVs) as a new ... ...

    Abstract Cancers are dependent on mitochondria, the powerhouse of the cell, and autophagy, the mechanism to preserve mitochondrial quality and function. In this issue of Developmental Cell, Towers et al. identify mitochondria-derived vesicles (MDVs) as a new adaptive mechanism enabling cancer cells to compensate for autophagy loss and to maintain mitochondrial function.
    MeSH term(s) Autophagy ; Mitochondria ; Mitophagy ; Neoplasms
    Language English
    Publishing date 2021-08-29
    Publishing country United States
    Document type Journal Article ; Comment
    ZDB-ID 2054967-2
    ISSN 1878-1551 ; 1534-5807
    ISSN (online) 1878-1551
    ISSN 1534-5807
    DOI 10.1016/j.devcel.2021.06.022
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  10. Article ; Online: Autophagy suppresses TRP53/p53 and oxidative stress to enable mammalian survival.

    Yang, Yang / White, Eileen

    Autophagy

    2020  Volume 16, Issue 7, Page(s) 1355–1357

    Abstract: Macroautophagy (hereafter autophagy) plays an important role in maintaining cellular homeostasis under stress conditions. We previously demonstrated that conditional autophagy deficiency in adult mice causes selective tissue damage, is lethal upon ... ...

    Abstract Macroautophagy (hereafter autophagy) plays an important role in maintaining cellular homeostasis under stress conditions. We previously demonstrated that conditional autophagy deficiency in adult mice causes selective tissue damage, is lethal upon fasting, and shortens lifespan to less than three months primarily due to neurodegeneration, but not all the mechanisms are known. We conditionally deleted
    Language English
    Publishing date 2020-05-23
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural
    ZDB-ID 2454135-7
    ISSN 1554-8635 ; 1554-8627
    ISSN (online) 1554-8635
    ISSN 1554-8627
    DOI 10.1080/15548627.2020.1765522
    Database MEDical Literature Analysis and Retrieval System OnLINE

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