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Book ; Online ; E-Book: Interactive technologies and autism

Kientz, Julie A. / Hayes, Gillian R. / Goodwin, Matthew S. / Gelsomini, Mirko / Abowd, Gregory D.

(Synthesis lectures on assistive, rehabilitative, and health-preserving technologies)

2020

Author's details	Julie A. Kientz, Gillian R. Hayes, Matthew S. Goodwin, Mirko Gelsomini, Gregory D. Abowd
Series title	Synthesis lectures on assistive, rehabilitative, and health-preserving technologies
Keywords	Electronic books
Language	English
Size	1 Online-Ressource (xxv, 229 Seiten), Illustrationen
Edition	Second edition
Publisher	Morgan & Claypool Publishers
Publishing place	San Rafael
Publishing country	United States
Document type	Book ; Online ; E-Book
Remark	Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
HBZ-ID	HT020487592
ISBN	9783031016042 ; 9783031004766 ; 3031016041 ; 3031004760
Database	ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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Article ; Online: Metabolic hormone action in the VTA: Reward-directed behavior and mechanistic insights.

Geisler, Caroline E / Hayes, Matthew R

Physiology & behavior

2023 Volume 268, Page(s) 114236

Abstract: Dysfunctional signaling in midbrain reward circuits perpetuates diseases characterized by compulsive overconsumption of rewarding substances such as substance abuse, binge eating disorder, and obesity. Ventral tegmental area (VTA) dopaminergic activity ... ...

Abstract	Dysfunctional signaling in midbrain reward circuits perpetuates diseases characterized by compulsive overconsumption of rewarding substances such as substance abuse, binge eating disorder, and obesity. Ventral tegmental area (VTA) dopaminergic activity serves as an index for how rewarding stimuli are perceived and triggers behaviors necessary to obtain future rewards. The evolutionary linking of reward with seeking and consuming palatable foods ensured an organism's survival, and hormone systems that regulate appetite concomitantly developed to regulate motivated behaviors. Today, these same mechanisms serve to regulate reward-directed behavior around food, drugs, alcohol, and social interactions. Understanding how hormonal regulation of VTA dopaminergic output alters motivated behaviors is essential to leveraging therapeutics that target these hormone systems to treat addiction and disordered eating. This review will outline our current understanding of the mechanisms underlying VTA action of the metabolic hormones ghrelin, glucagon-like peptide-1, amylin, leptin, and insulin to regulate behavior around food and drugs of abuse, highlighting commonalities and differences in how these five hormones ultimately modulate VTA dopamine signaling.
MeSH term(s)	Humans ; Ventral Tegmental Area/metabolism ; Signal Transduction ; Appetite ; Obesity/metabolism ; Dopamine/metabolism ; Reward
Chemical Substances	Dopamine (VTD58H1Z2X)
Language	English
Publishing date	2023-05-12
Publishing country	United States
Document type	Review ; Journal Article
ZDB-ID	3907-x
ISSN	1873-507X ; 0031-9384
ISSN (online)	1873-507X
ISSN	0031-9384
DOI	10.1016/j.physbeh.2023.114236
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Article ; Online: The antiemetic actions of GIP receptor agonism.

Borner, Tito / De Jonghe, Bart C / Hayes, Matthew R

American journal of physiology. Endocrinology and metabolism

2024 Volume 326, Issue 4, Page(s) E528–E536

Abstract: Nausea and vomiting are primitive aspects of mammalian physiology and behavior that ensure survival. Unfortunately, both are ubiquitously present side effects of drug treatments for many chronic diseases with negative consequences on pharmacotherapy ... ...

Abstract	Nausea and vomiting are primitive aspects of mammalian physiology and behavior that ensure survival. Unfortunately, both are ubiquitously present side effects of drug treatments for many chronic diseases with negative consequences on pharmacotherapy tolerance, quality of life, and prognosis. One of the most critical clinical examples is the profound emesis and nausea that occur in patients undergoing chemotherapy, which continue to be among the most distressing side effects, even with the use of modern antiemetic medications. Similarly, antiobesity/diabetes medications that target the glucagon-like peptide-1 system, despite their remarkable metabolic success, also cause nausea and vomiting in a significant number of patients. These side effects hinder the ability to administer higher dosages for optimal glycemic and weight management and represent the major reasons for treatment discontinuation. Our inability to effectively control these side effects highlights the need to anatomically, molecularly, and functionally characterize novel neural substrates that drive and inhibit nausea and emesis. Here, we discuss clinical and preclinical evidence that highlights the glucose-dependent insulinotropic peptide receptor system as a novel therapeutic central target for the management of nausea and emesis.
MeSH term(s)	Animals ; Humans ; Antiemetics/adverse effects ; Vomiting/chemically induced ; Vomiting/drug therapy ; Quality of Life ; Nausea/chemically induced ; Nausea/drug therapy ; Mammals ; Receptors, Gastrointestinal Hormone
Chemical Substances	Antiemetics ; gastric inhibitory polypeptide receptor (D6H00MV7K8) ; Receptors, Gastrointestinal Hormone
Language	English
Publishing date	2024-03-13
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	603841-4
ISSN	1522-1555 ; 0193-1849
ISSN (online)	1522-1555
ISSN	0193-1849
DOI	10.1152/ajpendo.00330.2023
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Article ; Online: Hindbrain ghrelin and liver-expressed antimicrobial peptide 2, ligands for growth hormone secretagogue receptor, bidirectionally control food intake.

Wald, Hallie S / Ghidewon, Misgana Y / Hayes, Matthew R / Grill, Harvey J

American journal of physiology. Regulatory, integrative and comparative physiology

2023 Volume 324, Issue 4, Page(s) R547–R555

Abstract: Hindbrain growth hormone secretagogue receptor (GHSR) agonism increases food intake, yet the underlying neural mechanisms remain unclear. The functional effects of hindbrain GHSR antagonism by its endogenous antagonist liver-expressed antimicrobial ... ...

Abstract	Hindbrain growth hormone secretagogue receptor (GHSR) agonism increases food intake, yet the underlying neural mechanisms remain unclear. The functional effects of hindbrain GHSR antagonism by its endogenous antagonist liver-expressed antimicrobial peptide 2 (LEAP2) are also yet unexplored. To test the hypothesis that hindbrain GHSR agonism attenuates the food intake inhibitory effect of gastrointestinal (GI) satiation signals, ghrelin (at a feeding subthreshold dose) was administered to the fourth ventricle (4V) or directly to the nucleus tractus solitarius (NTS) before systemic delivery of the GI satiation signal cholecystokinin (CCK). Also examined, was whether hindbrain GHSR agonism attenuated CCK-induced NTS neural activation (c-Fos immunofluorescence). To investigate an alternate hypothesis that hindbrain GHSR agonism enhances feeding motivation and food seeking, intake stimulatory ghrelin doses were administered to the 4V and fixed ratio 5 (FR-5), progressive ratio (PR), and operant reinstatement paradigms for palatable food responding were evaluated. Also assessed were 4V LEAP2 delivery on food intake and body weight (BW) and on ghrelin-stimulated feeding. Both 4V and NTS ghrelin blocked the intake inhibitory effect of CCK and 4V ghrelin blocked CCK-induced NTS neural activation. Although 4V ghrelin increased low-demand FR-5 responding, it did not increase high-demand PR or reinstatement of operant responding. Fourth ventricle LEAP2 reduced chow intake and BW and blocked hindbrain ghrelin-stimulated feeding. Data support a role for hindbrain GHSR in bidirectional control of food intake through mechanisms that include interacting with the NTS neural processing of GI satiation signals but not food motivation and food seeking.
MeSH term(s)	Receptors, Ghrelin/metabolism ; Hepcidins ; Ghrelin/pharmacology ; Eating ; Solitary Nucleus/metabolism ; Cholecystokinin/pharmacology
Chemical Substances	Receptors, Ghrelin ; Hepcidins ; Ghrelin ; Cholecystokinin (9011-97-6)
Language	English
Publishing date	2023-02-27
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural
ZDB-ID	603839-6
ISSN	1522-1490 ; 0363-6119
ISSN (online)	1522-1490
ISSN	0363-6119
DOI	10.1152/ajpregu.00232.2022
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Article ; Online: Scientific familial lessons in ingestive behavior research: 2016 Alan N. Epstein research award.

Hayes, Matthew R

Physiology & behavior

2017 Volume 176, Page(s) 214–216

Abstract: While energy balance is under the control of the central nervous system (CNS), a major source of neural regulation for the behavioral, physiological and endocrine processes governing energy balance originates in the periphery. Indeed, the organs of the ... ...

Abstract	While energy balance is under the control of the central nervous system (CNS), a major source of neural regulation for the behavioral, physiological and endocrine processes governing energy balance originates in the periphery. Indeed, the organs of the gastrointestinal (GI) tract, supporting organs of the peritoneal cavity and adipose tissue are the source of numerous neurotransmitter and neuroendocrine signals released from non-neuronal peripheral tissue that signal in a paracrine and endocrine fashion to regulate the physiological and behavioral processes that affect energy balance. Given the ever increasing appreciation that chronic hyperphagia of highly-palatable/rewarding food is a major contributing factor to the obesity epidemic, it is not surprising that the field has increased research efforts focusing on understanding what role peripherally-derived neuroendocrine signals play in modulating food reward and motivated behaviors. Research throughout my career has focused on understanding gut-to-brain communication of relevance to energy balance control. Through very fortuitous opportunities and amazing collaborations, my research program has also expanded widely to include analyses of multiple GI-, pancreatic- and adipose tissue-derived anorectic signals involved in food intake and energy balance control, as well as analyses of higher-order determinants of food reward, nausea, aversion and maladaptive motivated behaviors. I am honored to be the recipient of the 2016 Alan N. Epstein Research Award from the Society for the Study of Ingestive Behavior, and express much appreciation for the amazing collaborations I have had with my mentors, colleagues and trainees.
Language	English
Publishing date	2017-07-01
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	3907-x
ISSN	1873-507X ; 0031-9384
ISSN (online)	1873-507X
ISSN	0031-9384
DOI	10.1016/j.physbeh.2017.01.042
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Article ; Online: Metabolic hormone action in the VTA: Reward-directed behavior and mechanistic insights

Geisler, Caroline E. / Hayes, Matthew R.

Physiology & Behavior. 2023 Sept., v. 268 p.114236-

2023

Abstract	Dysfunctional signaling in midbrain reward circuits perpetuates diseases characterized by compulsive overconsumption of rewarding substances such as substance abuse, binge eating disorder, and obesity. Ventral tegmental area (VTA) dopaminergic activity serves as an index for how rewarding stimuli are perceived and triggers behaviors necessary to obtain future rewards. The evolutionary linking of reward with seeking and consuming palatable foods ensured an organism's survival, and hormone systems that regulate appetite concomitantly developed to regulate motivated behaviors. Today, these same mechanisms serve to regulate reward-directed behavior around food, drugs, alcohol, and social interactions. Understanding how hormonal regulation of VTA dopaminergic output alters motivated behaviors is essential to leveraging therapeutics that target these hormone systems to treat addiction and disordered eating. This review will outline our current understanding of the mechanisms underlying VTA action of the metabolic hormones ghrelin, glucagon-like peptide-1, amylin, leptin, and insulin to regulate behavior around food and drugs of abuse, highlighting commonalities and differences in how these five hormones ultimately modulate VTA dopamine signaling.
Keywords	alcohols ; appetite ; behavior ; brain ; dopamine ; ghrelin ; glucagon-like peptide 1 ; hormonal regulation ; insulin ; leptin ; obesity ; substance abuse ; therapeutics ; Reward ; Motivation ; Drugs of abuse ; Addiction
Language	English
Dates of publication	2023-09
Publishing place	Elsevier Inc.
Document type	Article ; Online
ZDB-ID	3907-x
ISSN	1873-507X ; 0031-9384
ISSN (online)	1873-507X
ISSN	0031-9384
DOI	10.1016/j.physbeh.2023.114236
Database	NAL-Catalogue (AGRICOLA)

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Article ; Online: An Inversion Affecting the GCH1 Gene as a Novel Finding in Dopa-Responsive Dystonia.

El-Wahsh, Shadi / Fellner, Avi / Hobbs, Matthew / Copty, Joe / Deveson, Ira / Stevanovski, Igor / Stoll, Marion / Zhu, Danqing / Narayanan, Ramesh K / Grosz, Bianca / Worgan, Lisa / Cheong, Pak Leng / Yeow, Dennis / Rudaks, Laura / Hasan, Md Mehedi / Hayes, Vanessa M / Kennerson, Marina / Kumar, Kishore R / Hayes, Michael

Movement disorders clinical practice

2024

Language	English
Publishing date	2024-03-18
Publishing country	United States
Document type	Letter
ISSN	2330-1619
ISSN (online)	2330-1619
DOI	10.1002/mdc3.14023
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Article ; Online: Updated systematic review of the approach to pelvic exenteration for locally advanced primary rectal cancer.

Fahy, Matthew R / Hayes, Cathal / Kelly, Michael E / Winter, Desmond C

European journal of surgical oncology : the journal of the European Society of Surgical Oncology and the British Association of Surgical Oncology

2022 Volume 48, Issue 11, Page(s) 2284–2291

Abstract: Objectives: To review the evidence regarding surgical advances in the management of primary locally advanced rectal cancer.: Background: The management of rectal cancer has evolved significantly in recent decades, with improved (neo)adjuvant ... ...

Abstract	Objectives: To review the evidence regarding surgical advances in the management of primary locally advanced rectal cancer. Background: The management of rectal cancer has evolved significantly in recent decades, with improved (neo)adjuvant treatment strategies and enhanced perioperative protocols. Centralization of care for complex, advanced cases has enabled surgeons in these units to undertake more ambitious surgical procedures. Methods: A Pubmed, Ovid, Embase and Cochrane database search was conducted according to the predetermined search strategy. The review protocol was prospectively registered with PROSPERO (CRD42021245582). Results: 14 studies were identified which reported on the outcomes of 3,188 patients who underwent pelvic exenteration (PE) for primary rectal cancer. 50% of patients had neoadjuvant radiotherapy. 24.2% underwent flap reconstruction, 9.4% required a bony resection and 34 patients underwent a major vascular excision. 73.9% achieved R0 resection, with 33.1% experiencing a major complication. Median length of hospital stay ranged from 13 to 19 days. 1.6% of patients died within 30 days of their operation. Five-year overall survival (OS) rates ranged 29%-78%. Limitations: The studies included in our review were mostly single-centre observational studies published prior to the introduction of modern neoadjuvant treatment regimens. It was not possible to perform a meta-analysis on the basis that most were non-randomized, non-comparative studies. Conclusions: Pelvic exenteration offers patients with locally advanced rectal cancer the chance of long-term survival with acceptable levels of morbidity. Increased experience facilitates more radical procedures, with the introduction of new platforms and/or reconstructive options.
MeSH term(s)	Humans ; Pelvic Exenteration/methods ; Retrospective Studies ; Rectal Neoplasms/surgery ; Rectum/surgery ; Neoadjuvant Therapy ; Neoplasm Recurrence, Local/surgery ; Treatment Outcome
Language	English
Publishing date	2022-01-05
Publishing country	England
Document type	Review ; Journal Article ; Systematic Review
ZDB-ID	632519-1
ISSN	1532-2157 ; 0748-7983
ISSN (online)	1532-2157
ISSN	0748-7983
DOI	10.1016/j.ejso.2021.12.471
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Article ; Online: The Association of Laryngeal Position on Videolaryngoscopy and Time Taken to Intubate Using Spatial Point Pattern Analysis of Prospectively Collected Quality Assurance Data.

Miller, Matthew R / Gemal, Hugo / Ware, Sandra / Hayes-Bradley, Clare

Anesthesia and analgesia

2022 Volume 134, Issue 6, Page(s) 1288–1296

Abstract: Background: During videolaryngoscopy (VL), the larynx appears within the defined area of the video screen, and its location can be measured as a point within this space. Spatial statistics offer methods to explore the relationship between location data ... ...

Abstract	Background: During videolaryngoscopy (VL), the larynx appears within the defined area of the video screen, and its location can be measured as a point within this space. Spatial statistics offer methods to explore the relationship between location data and associated variables of interest. The aims of this study were to use spatial point pattern analysis to explore if the position of the larynx on VL is associated with longer times to intubate, increased risk of a needing >1 intubation attempt, or percentage of glottic opening. Methods: Quality assurance data and clinical notes from all prehospital intubations using C-MAC Pocket Monitor with CMAC-4 blade (Karl Storz) from January 1, 2018, to July 31, 2020, were reviewed. We extracted 6 measurements corresponding to the time taken to obtain the initial and then best laryngeal view, time to manipulate a bougie, and time to place the endotracheal tube, as well a percentage of glottic opening and a number of intubation attempts. Larynx location was the middle of the base of glottis, in cm from the left and bottom on the C-MAC screen. Two plots were produced to summarize the base of glottis location and time to perform each time component of intubation. Next, a cross mark function and a maximum absolute deviation hypothesis test were performed to assess the null hypotheses that the spatial distributions were random. The association between glottis location and >1 intubation attempt was assessed by a spatial relative risk plot. Results: Of 619 eligible intubations, 385 had a video for analysis. The following time variables had a nonrandom spatial distribution with a tendency for longer times when the larynx was off-center to the top or right of the screen: laryngoscope passing from teeth to glottis, glottis first view to best view of the larynx, time from bougie appearing to being placed in the cords, and overall time from teeth to endotracheal tube passing through cords. There was no increased relative risk for >1 intubation attempt. Conclusions: Spatial point pattern analysis identified a relationship between the position of the larynx during VL and prolonged intubation times. We did not find a relationship between larynx location and >1 attempt. Whether the location of the larynx on the screen is a marker for difficult VL or if optimizing the larynx position to the center of the screen improves intubation times would require further prospective studies.
MeSH term(s)	Humans ; Intubation, Intratracheal/methods ; Laryngoscopes ; Laryngoscopy/methods ; Larynx ; Prospective Studies
Language	English
Publishing date	2022-01-12
Publishing country	United States
Document type	Journal Article
ZDB-ID	80032-6
ISSN	1526-7598 ; 0003-2999
ISSN (online)	1526-7598
ISSN	0003-2999
DOI	10.1213/ANE.0000000000005868
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Article ; Online: The Role of GIP in the Regulation of GLP-1 Satiety and Nausea.

Hayes, Matthew R / Borner, Tito / De Jonghe, Bart C

Diabetes

2021 Volume 70, Issue 9, Page(s) 1956–1961

Abstract: Gastric inhibitory peptide (GIP) is best known for its role as an incretin hormone in control of blood glucose concentrations. As a classic satiation signal, however, the literature illustrates a mixed picture of GIP involvement with an at best weak ... ...

Abstract	Gastric inhibitory peptide (GIP) is best known for its role as an incretin hormone in control of blood glucose concentrations. As a classic satiation signal, however, the literature illustrates a mixed picture of GIP involvement with an at best weak anorectic response profile being reported for GIP receptor (GIPR) signaling. Not surprisingly, the pursuit of exploiting the GIP system as a therapeutic target for diabetes and obesity has fallen behind that of the other gastrointestinal-derived incretin, glucagon-like peptide 1 (GLP-1). However, recent discoveries highlighted here support potential therapeutic advantages of combinatorial therapies targeting GIP and GLP-1 systems together, with perhaps the most surprising finding that GIPR agonism may have antiemetic properties. As nausea and vomiting are the most common side effects of all existing GLP-1 pharmacotherapies, the ability for GIP agonism to reduce GLP-1-induced illness behaviors but retain (if not enhance) weight loss and glycemic control may offer a new era in the treatment of obesity and diabetes.
MeSH term(s)	Animals ; Diabetes Mellitus, Type 2/metabolism ; Gastric Inhibitory Polypeptide/metabolism ; Glucagon-Like Peptide 1/metabolism ; Humans ; Nausea/metabolism ; Obesity/metabolism ; Satiation/physiology
Chemical Substances	Gastric Inhibitory Polypeptide (59392-49-3) ; Glucagon-Like Peptide 1 (89750-14-1)
Language	English
Publishing date	2021-06-27
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
ZDB-ID	80085-5
ISSN	1939-327X ; 0012-1797
ISSN (online)	1939-327X
ISSN	0012-1797
DOI	10.2337/dbi21-0004
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