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Article: [Social Allostasis Regulation by Robots].

Sato, Wataru / Kanbara, Masayuki

Brain and nerve = Shinkei kenkyu no shinpo

2023 Volume 75, Issue 11, Page(s) 1219–1223

Abstract: Social allostasis, the process by which social interactions dynamically regulate psychophysiological states (e.g., emotions), is attracting attention. Social allostasis can significantly impact people's mental and physical wellbeing. However, adaptive ... ...

Abstract	Social allostasis, the process by which social interactions dynamically regulate psychophysiological states (e.g., emotions), is attracting attention. Social allostasis can significantly impact people's mental and physical wellbeing. However, adaptive social relationships are not necessarily available to all people. The use of robots is expected to solve this problem. In this paper, we present three studies of our group that prove the influence of social interactions with robots on human psychophysiological states. Our results showed that robots could amplify human subjective and physiological emotional responses through conversation, touch, and television co-viewing experiences. These results suggest that robots may be helpful for adaptive social allostasis regulation.
MeSH term(s)	Humans ; Allostasis ; Robotics/methods ; Emotions/physiology
Language	Japanese
Publishing date	2023-11-08
Publishing country	Japan
Document type	English Abstract ; Journal Article
ZDB-ID	390389-8
ISSN	1344-8129 ; 1881-6096 ; 0006-8969
ISSN (online)	1344-8129
ISSN	1881-6096 ; 0006-8969
DOI	10.11477/mf.1416202508
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Article ; Online: Design of structured H

Sato, Masayuki / Marcos, Andrés / Akasaka, Daisuke

ISA transactions

2023 Volume 143, Page(s) 20–37

Abstract: This article presents the structured ... ...

Abstract	This article presents the structured H
Language	English
Publishing date	2023-08-30
Publishing country	United States
Document type	Journal Article
ZDB-ID	2012746-7
ISSN	1879-2022 ; 0019-0578
ISSN (online)	1879-2022
ISSN	0019-0578
DOI	10.1016/j.isatra.2023.08.029
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Article ; Online: A distinctive new species of the genus Polyonyx Stimpson, 1858 (Crustacea: Decapoda: Anomura: Porcellanidae) from Okinawa, southwestern Japan.

Osawa, Masayuki / Sato, Taigi

Zootaxa

2022 Volume 5091, Issue 4, Page(s) 587–597

Abstract: The new porcellanid crab Polyonyx deezi n. sp. is described on the basis of two specimens from Okinawa Island, Ryukyu Islands, southwestern Japan. The new species belongs to the P. sinensis group and may be closest to P. socialis Werding Hiller, 2019 in ... ...

Abstract	The new porcellanid crab Polyonyx deezi n. sp. is described on the basis of two specimens from Okinawa Island, Ryukyu Islands, southwestern Japan. The new species belongs to the P. sinensis group and may be closest to P. socialis Werding Hiller, 2019 in the comparatively broad proportions of the carpi of the chelipeds and meri of the ambulatory legs. However, P. deezi n. sp. is immediately distinguished from all other congeners by the median branchial margins of the carapace being bluntly angular and produced laterally and the dorsal surfaces of the carapace and chelipeds with distinct protuberances. The occurrence of P. deezi n. sp. from coral reefs may be unusual in species of the P. sinensis group because many of the known species have been recorded from estuaries or coastal embayments. An identification key to the Indo-West Pacific species of the Polyonyx sinensis group is provided.
MeSH term(s)	Animal Distribution ; Animal Shells ; Animals ; Anomura ; Decapoda ; Japan
Language	English
Publishing date	2022-01-17
Publishing country	New Zealand
Document type	Journal Article
ISSN	1175-5334
ISSN (online)	1175-5334
DOI	10.11646/zootaxa.5091.4.6
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Article ; Online: Update on the treatment of cancer cachexia.

Nishie, Kenichi / Nishie, Tomomi / Sato, Seiichi / Hanaoka, Masayuki

Drug discovery today

2023 Volume 28, Issue 9, Page(s) 103689

Abstract: Cancer cachexia is a complex multifaceted syndrome involving functional impairment and changes in body composition that cannot be reversed by nutritional support. Cancer cachexia is characterized by decreased skeletal muscle mass, increased lipolysis, ... ...

Abstract	Cancer cachexia is a complex multifaceted syndrome involving functional impairment and changes in body composition that cannot be reversed by nutritional support. Cancer cachexia is characterized by decreased skeletal muscle mass, increased lipolysis, and decreased food intake. Cancer cachexia decreases chemotherapy tolerance as well as quality of life. However, because no fully effective interventions are available, cancer cachexia remains an unmet need in cancer treatment. In recent years, several discoveries and treatments for cancer cachexia have been studied, and guidelines have been published. We believe that the development of effective strategies for the diagnosis and treatment of cancer cachexia will lead to breakthroughs in cancer treatment.
MeSH term(s)	Humans ; Cachexia/drug therapy ; Cachexia/etiology ; Quality of Life ; Anorexia/etiology ; Neoplasms/complications
Language	English
Publishing date	2023-06-28
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	1324988-5
ISSN	1878-5832 ; 1359-6446
ISSN (online)	1878-5832
ISSN	1359-6446
DOI	10.1016/j.drudis.2023.103689
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Article ; Online: KEGG tools for classification and analysis of viral proteins.

Jin, Zhao / Sato, Yoko / Kawashima, Masayuki / Kanehisa, Minoru

Protein science : a publication of the Protein Society

2023 Volume 32, Issue 12, Page(s) e4820

Abstract: The KEGG database and analysis tools (https://www.kegg.jp) have been developed mostly for understanding genes and genomes of cellular organisms. The KO (KEGG Orthology) dataset, which is a collection of functional orthologs, plays the role of linking ... ...

Abstract	The KEGG database and analysis tools (https://www.kegg.jp) have been developed mostly for understanding genes and genomes of cellular organisms. The KO (KEGG Orthology) dataset, which is a collection of functional orthologs, plays the role of linking genes in the genome to pathways and other molecular networks, enabling KEGG mapping to uncover hidden features in the genome. Although viruses were part of KEGG for some time, they were not fully integrated in the KEGG analysis tools, because the KO assignment rate is very low for virus genes. To supplement KOs a new dataset named virus ortholog clusters (VOCs) is computationally generated, covering 90% of viral proteins in KEGG. VOCs can be used, in place of KOs, for taxonomy mapping to uncover relationships of sequence similarity groups and taxonomic groups and for identifying conserved gene orders in virus genomes. Furthermore, selected VOCs are used to define tentative KOs for characterizing protein functions. Here an overview of KEGG tools is presented focusing on these extensions for viral protein analysis.
MeSH term(s)	Viral Proteins/genetics ; Genome ; Databases, Factual ; Viruses/genetics
Chemical Substances	Viral Proteins
Language	English
Publishing date	2023-10-25
Publishing country	United States
Document type	Journal Article
ZDB-ID	1106283-6
ISSN	1469-896X ; 0961-8368
ISSN (online)	1469-896X
ISSN	0961-8368
DOI	10.1002/pro.4820
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Zs.A 3407: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)
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Article ; Online: Neck and shoulder pain in thoracic adolescent idiopathic scoliosis 10 years after posterior spinal fusion.

Ohashi, Masayuki / Watanabe, Kei / Hirano, Toru / Hasegawa, Kazuhiro / Tashi, Hideki / Makino, Tatsuo / Minato, Keitaro / Sato, Masayuki / Kawashima, Hiroyuki

European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society

2024

Abstract: Purpose: We aimed to determine the clinical significance of neck and shoulder pain (NSP) 10 years after posterior spinal fusion (PSF) for thoracic adolescent idiopathic scoliosis (AIS) and the relationship between radiographic parameters and NSP.: ... ...

Abstract	Purpose: We aimed to determine the clinical significance of neck and shoulder pain (NSP) 10 years after posterior spinal fusion (PSF) for thoracic adolescent idiopathic scoliosis (AIS) and the relationship between radiographic parameters and NSP. Methods: Of 72 patients who underwent PSF for thoracic AIS (Lenke 1 or 2) between 2000 and 2013, we included 52 (46 females; Lenke type 1 in 34 patients and type 2 in 18; mean age, 25.6 years) who underwent NSP evaluation using visual analog scale (VAS, 10 cm) 10 years postoperatively (follow-up rate, 72.2%). Correlation analyses were performed using Spearman's rank correlation coefficient (r). Results: The VAS for NSP was 2.6 cm in median and 3.4 cm in mean at 10 years. The VAS had significant negative correlations with several SRS-22 domain scores (rs = - 0.348 for pain, - 0.347 for function, - 0.308 for mental health, and - 0.372 for total) (p < 0.05). In addition, the VAS score was significantly correlated with cervical lordosis (CL) (rs = 0.296), lumbar lordosis (rs = - 0.299), and sacral slope (rs = 0.362) (p < 0.05). Furthermore, at the 10-year follow-up, CL was significantly negatively correlated with T1 slope (rs = - 0.763) and thoracic kyphosis (TK) (- 0.554 for T1-12 and - 0.344 for T5-12) (p < 0.02). Conclusion: NSP was associated with deterioration in SRS-22 scores, indicating that NSP is a clinically significant long-term issue in PSF for thoracic AIS. Restoring or maintaining the TK and T1 slopes, which are controllable factors during PSF, may improve cervical lordosis and alleviate NSP at 10-year follow-up.
Language	English
Publishing date	2024-04-04
Publishing country	Germany
Document type	Journal Article
ZDB-ID	1115375-1
ISSN	1432-0932 ; 0940-6719
ISSN (online)	1432-0932
ISSN	0940-6719
DOI	10.1007/s00586-024-08233-6
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Article ; Online: Decoding the basis of histological variation in human cancer.

Fujii, Masayuki / Sekine, Shigeki / Sato, Toshiro

Nature reviews. Cancer

2023 Volume 24, Issue 2, Page(s) 141–158

Abstract: Molecular abnormalities that shape human neoplasms dissociate their phenotypic landscape from that of the healthy counterpart. Through the lens of a microscope, tumour pathology optically captures such aberrations projected onto a tissue slide and has ... ...

Abstract	Molecular abnormalities that shape human neoplasms dissociate their phenotypic landscape from that of the healthy counterpart. Through the lens of a microscope, tumour pathology optically captures such aberrations projected onto a tissue slide and has categorized human epithelial neoplasms into distinct histological subtypes based on the diverse morphogenetic and molecular programmes that they manifest. Tumour histology often reflects tumour aggressiveness, patient prognosis and therapeutic vulnerability, and thus has been used as a de facto diagnostic tool and for making clinical decisions. However, it remains elusive how the diverse histological subtypes arise and translate into pleiotropic biological phenotypes. Molecular analysis of clinical tumour tissues and their culture, including patient-derived organoids, and add-back genetic reconstruction of tumorigenic pathways using gene engineering in culture models and rodents further elucidated molecular mechanisms that underlie morphological variations. Such mechanisms include genetic mutations and epigenetic alterations in cellular identity codes that erode hard-wired morphological programmes and histologically digress tumours from the native tissues. Interestingly, tumours acquire the ability to grow independently of the niche-driven stem cell ecosystem along with these morphological alterations, providing a biological rationale for histological diversification during tumorigenesis. This Review comprehensively summarizes our current understanding of such plasticity in the histological and lineage commitment fostered cooperatively by molecular alterations and the tumour environment, and describes basic and clinical implications for future cancer therapy.
MeSH term(s)	Humans ; Carcinogenesis ; Mutation ; Phenotype ; Stem Cells
Language	English
Publishing date	2023-12-22
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	2062767-1
ISSN	1474-1768 ; 1474-175X
ISSN (online)	1474-1768
ISSN	1474-175X
DOI	10.1038/s41568-023-00648-5
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Zs.A 5563: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (2.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MG 24: Show issues

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Article ; Online: Effect of ASP8062 on morphine self-administration and morphine-induced respiratory suppression in monkeys.

Akuzawa, Shinobu / Irie, Megumi / Kanki, Masayuki / Shirakawa, Takafumi / Sato, Yuichiro

Journal of pharmacological sciences

2023 Volume 151, Issue 4, Page(s) 171–176

Abstract: ASP8062 is an orally available ... ...

Abstract	ASP8062 is an orally available GABA
MeSH term(s)	Animals ; Morphine ; Macaca fascicularis ; Chromatography, Liquid ; Tandem Mass Spectrometry ; Dose-Response Relationship, Drug
Chemical Substances	Morphine (76I7G6D29C) ; ASP8062
Language	English
Publishing date	2023-02-15
Publishing country	Japan
Document type	Journal Article
ZDB-ID	2104264-0
ISSN	1347-8648 ; 1347-8613
ISSN (online)	1347-8648
ISSN	1347-8613
DOI	10.1016/j.jphs.2023.02.003
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Article ; Online: Contribution of Complement, Microangiopathy and Inflammation in Idiopathic Inflammatory Myopathies.

Honda, Masaya / Shimizu, Fumitaka / Sato, Ryota / Nakamori, Masayuki

Journal of neuromuscular diseases

2023 Volume 11, Issue 1, Page(s) 5–16

Abstract: Purpose of review: Idiopathic inflammatory myopathies (IIMs) are a heterogeneous group characterized by muscle weakness and skin symptoms and are categorized into six subtypes: dermatomyositis (DM), polymyositis (PM), anti-synthetase syndrome (ASS), ... ...

Abstract	Purpose of review: Idiopathic inflammatory myopathies (IIMs) are a heterogeneous group characterized by muscle weakness and skin symptoms and are categorized into six subtypes: dermatomyositis (DM), polymyositis (PM), anti-synthetase syndrome (ASS), immune-mediated myopathy (IMNM), inclusion body myopathy (IBM), and overlap myositis. Myositis-specific autoantibodies were detected for the diagnosis and classification of IIM. This review highlights the pathogenic contributions of the complement system, microangiopathy, and inflammation in IIM. Recent findings: Deposition of complement around capillaries and/or the sarcolemma was observed in muscle biopsy specimens from patients with DM, ASS, and IMNM, suggesting the pathomechanism of complement-dependent muscle and endothelial cell injury. A recent study using human muscle microvascular endothelial cells showed that Jo-1 antibodies from ASS induce complement-dependent cellular cytotoxicity in vitro. Based on both clinical and pathological observations, antibody- and complement-mediated microangiopathy may contribute to the development of DM and anti-Jo-1 ASS. Juvenile DM is characterized by the loss of capillaries, perivascular inflammation, and small-vessel angiopathies, which may be related to microinfarction and perifascicular atrophy. Several serum biomarkers that reflect the IFN1 signature and microangiopathy are elevated in patients with DM. The pathological observation of myxovirus resistance protein A (MxA), which suggests a type 1 interferon (IFN1) signature in DM, supports the diagnosis and further understanding of the pathomechanism of IIM. A recent report showed that an increase in triggering receptor expressed on myeloid cells (TREM-1) around perimysial blood vessels and muscles in patients with IIM plays a role in triggering inflammation and promoting the migration of inflammatory cells by secreting proinflammatory cytokines, such as tumor necrosis factor α. Summary: The deposition of complement in muscles and capillaries is a characteristic feature of DM, ASS, and IMNM. Microangiopathy plays a pathogenic role in DM, possibly resulting in perifascicular atrophy. Further understanding of the detailed pathomechanism regarding complement, microangiopathy, and inflammation may lead to novel therapeutic approaches for IIM.
MeSH term(s)	Humans ; Capillaries/pathology ; Endothelial Cells/pathology ; Myositis/diagnosis ; Muscle, Skeletal/pathology ; Muscular Diseases/pathology ; Inflammation/pathology ; Atrophy/pathology
Language	English
Publishing date	2023-12-16
Publishing country	Netherlands
Document type	Journal Article ; Review
ISSN	2214-3602
ISSN (online)	2214-3602
DOI	10.3233/JND-230168
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Article ; Online: Author Correction: Reduced glutathione level in the aqueous humor of patients with primary open-angle glaucoma and normal-tension glaucoma.

Sato, Kota / Saigusa, Daisuke / Kokubun, Taiki / Fujioka, Amane / Feng, Qiwei / Saito, Ritsumi / Uruno, Akira / Matsukawa, Naomi / Ohno-Oishi, Michiko / Kunikata, Hiroshi / Yokoyama, Yu / Yasuda, Masayuki / Himori, Noriko / Omodaka, Kazuko / Tsuda, Satoru / Maekawa, Shigeto / Yamamoto, Masayuki / Nakazawa, Toru

npj aging

2024 Volume 10, Issue 1, Page(s) 8

Language	English
Publishing date	2024-01-20
Publishing country	England
Document type	Published Erratum
ISSN	2731-6068
ISSN (online)	2731-6068
DOI	10.1038/s41514-024-00137-5
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