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  1. Article ; Online: Vitamin B

    Egan, William / Steinberg, Eric / Rose, Jeremy

    The American journal of emergency medicine

    2018  Volume 36, Issue 9, Page(s) 1717.e1–1717.e2

    Abstract: A 24-year-old female, otherwise healthy, presented to the Emergency Department (ED) with difficulty walking and bilateral leg pain. The patient was a recreational nitrous oxide ( ... ...

    Abstract A 24-year-old female, otherwise healthy, presented to the Emergency Department (ED) with difficulty walking and bilateral leg pain. The patient was a recreational nitrous oxide (NO
    MeSH term(s) Emergency Service, Hospital ; Female ; Humans ; Inhalant Abuse/complications ; Nervous System Diseases/chemically induced ; Nervous System Diseases/etiology ; Nitrous Oxide/adverse effects ; Vitamin B 12 Deficiency/chemically induced ; Vitamin B 12 Deficiency/complications ; Young Adult
    Chemical Substances Nitrous Oxide (K50XQU1029)
    Language English
    Publishing date 2018-05-24
    Publishing country United States
    Document type Case Reports ; Journal Article
    ZDB-ID 605890-5
    ISSN 1532-8171 ; 0735-6757
    ISSN (online) 1532-8171
    ISSN 0735-6757
    DOI 10.1016/j.ajem.2018.05.029
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  2. Article ; Online: Long-Term Follow-Up of CD19-CAR T-Cell Therapy in Children and Young Adults With B-ALL.

    Shah, Nirali N / Lee, Daniel W / Yates, Bonnie / Yuan, Constance M / Shalabi, Haneen / Martin, Staci / Wolters, Pamela L / Steinberg, Seth M / Baker, Eva H / Delbrook, Cindy P / Stetler-Stevenson, Maryalice / Fry, Terry J / Stroncek, David F / Mackall, Crystal L

    Journal of clinical oncology : official journal of the American Society of Clinical Oncology

    2021  Volume 39, Issue 15, Page(s) 1650–1659

    Abstract: ... and young adults (CAYAs) with B-cell acute lymphoblastic leukemia (B-ALL), but relapse rates are high ... of autologous CD19.28ζ-CAR T cells in CAYAs with relapsed or refractory B-ALL. Response and long-term clinical ... outcomes were assessed in relation to disease and treatment variables.: Results: Fifty CAYAs with B ...

    Abstract Purpose: CD19 chimeric antigen receptor (CD19-CAR) T cells induce high response rates in children and young adults (CAYAs) with B-cell acute lymphoblastic leukemia (B-ALL), but relapse rates are high. The role for allogeneic hematopoietic stem-cell transplant (alloHSCT) following CD19-CAR T-cell therapy to improve long-term outcomes in CAYAs has not been examined.
    Methods: We conducted a phase I trial of autologous CD19.28ζ-CAR T cells in CAYAs with relapsed or refractory B-ALL. Response and long-term clinical outcomes were assessed in relation to disease and treatment variables.
    Results: Fifty CAYAs with B-ALL were treated (median age, 13.5 years; range, 4.3-30.4). Thirty-one (62.0%) patients achieved a complete remission (CR), 28 (90.3%) of whom were minimal residual disease-negative by flow cytometry. Utilization of fludarabine/cyclophosphamide-based lymphodepletion was associated with improved CR rates (29/42, 69%) compared with non-fludarabine/cyclophosphamide-based lymphodepletion (2/8, 25%;
    Conclusion: We provide the longest follow-up in CAYAs with B-ALL after CD19-CAR T-cell therapy reported to date and demonstrate that sequential therapy with CD19.28ζ-CAR T cells followed by alloHSCT can mediate durable disease control in a sizable fraction of CAYAs with relapsed or refractory B-ALL (ClinicalTrials.gov identifier: NCT01593696).
    MeSH term(s) Adolescent ; Adult ; Child ; Child, Preschool ; Female ; Follow-Up Studies ; Humans ; Immunotherapy, Adoptive/methods ; Male ; Precursor B-Cell Lymphoblastic Leukemia-Lymphoma/therapy ; Young Adult
    Language English
    Publishing date 2021-03-25
    Publishing country United States
    Document type Clinical Trial, Phase I ; Journal Article ; Research Support, N.I.H., Intramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 604914-x
    ISSN 1527-7755 ; 0732-183X
    ISSN (online) 1527-7755
    ISSN 0732-183X
    DOI 10.1200/JCO.20.02262
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  3. Article: Single-cell profiling reveals preferential reduction of memory B cell subsets in cladribine patients that correlates with treatment response.

    Teschner, Valerie E / Fleck, Ann-Katrin / Walter, Carolin / Schwarze, Anna-Sophie / Eschborn, Melanie / Wirth, Timo / Steinberg, Olga V / Schulte-Mecklenbeck, Andreas / Lu, I-Na / Herrera-Rivero, Marisol / Janoschka, Claudia / Lünemann, Jan D / Schwab, Nicholas / Meyer Zu Hörste, Gerd / Varghese, Julian / Gross, Catharina C / Pul, Refik / Kleinschnitz, Christoph / Mader, Simone /
    Meinl, Edgar / Stoll, Monika / Wiendl, Heinz / Klotz, Luisa

    Therapeutic advances in neurological disorders

    2023  Volume 16, Page(s) 17562864231211077

    Abstract: ... Flow cytometry revealed a predominant and sustained reduction of memory B cells compared to other B cell subsets ... The overall transcriptional profile of total blood B cells exhibited reduced expression of proinflammatory and ... within each B cell cluster did not change over time. Stable patients displayed stronger reductions of selected ...

    Abstract Background: Cladribine is a highly effective immunotherapy that is applied in two short-term courses over 2 years and reduces relapse rate and disease progression in patients with relapsing multiple sclerosis (MS). Despite the short treatment period, cladribine has a long-lasting effect on disease activity even after recovery of lymphocyte counts, suggesting a yet undefined long-term immune modulating effect.
    Objectives: Our aim was to provide a more profound understanding of the detailed effects of cladribine, also with regard to the patients' therapy response.
    Design: We performed an open-labeled, explorative, prospective, single-arm study, in which we examined the detailed lymphocyte subset development of MS patients who received cladribine treatment over 2 years.
    Methods: We performed in-depth profiling of the effects of cladribine on peripheral blood lymphocytes by flow cytometry, bulk RNA sequencing of sorted CD4
    Results: Flow cytometry revealed a predominant and sustained reduction of memory B cells compared to other B cell subsets after cladribine treatment, whereas T cell subsets were slightly reduced in a more uniform pattern. The overall transcriptional profile of total blood B cells exhibited reduced expression of proinflammatory and T cell activating genes, while single-cell transcriptomics revealed that gene expression within each B cell cluster did not change over time. Stable patients displayed stronger reductions of selected memory B cell clusters as compared to patients with clinical or cerebral MRI disease activity.
    Conclusion: We describe a pronounced and sustained effect of cladribine on the memory B cell compartment, and the resulting change in B cell subset composition causes a significant alteration of B cell transcriptional profiles resulting in reduced proinflammatory and T cell activating capacities. The extent of reduction in selected memory B cell clusters by cladribine may predict treatment response.
    Language English
    Publishing date 2023-12-09
    Publishing country England
    Document type Journal Article
    ZDB-ID 2442245-9
    ISSN 1756-2864 ; 1756-2856
    ISSN (online) 1756-2864
    ISSN 1756-2856
    DOI 10.1177/17562864231211077
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  4. Article ; Online: CD19/22 CAR T cells in children and young adults with B-ALL: phase 1 results and development of a novel bicistronic CAR.

    Shalabi, Haneen / Qin, Haiying / Su, Angela / Yates, Bonnie / Wolters, Pamela L / Steinberg, Seth M / Ligon, John A / Silbert, Sara / DéDé, Kniya / Benzaoui, Mehdi / Goldberg, Sophia / Achar, Sooraj / Schneider, Dina / Shahani, Shilpa A / Little, Lauren / Foley, Toni / Molina, John C / Panch, Sandhya / Mackall, Crystal L /
    Lee, Daniel W / Chien, Christopher D / Pouzolles, Marie / Ahlman, Mark / Yuan, Constance M / Wang, Hao-Wei / Wang, Yanyu / Inglefield, Jon / Toledo-Tamula, Mary Anne / Martin, Staci / Highfill, Steven L / Altan-Bonnet, Gregoire / Stroncek, David / Fry, Terry J / Taylor, Naomi / Shah, Nirali N

    Blood

    2022  Volume 140, Issue 5, Page(s) 451–463

    Abstract: ... upon our experiences targeting CD19 and CD22 in B-cell acute lymphoblastic leukemia (B-ALL), we report on our phase 1 ... CD19.22.BBζ) for children and young adults (CAYA) with B-cell malignancies. Primary objectives included ... Twenty patients, ages 5.4 to 34.6 years, with B-ALL received CD19.22.BBζ. The complete response (CR) rate ...

    Abstract Remission durability following single-antigen targeted chimeric antigen receptor (CAR) T-cells is limited by antigen modulation, which may be overcome with combinatorial targeting. Building upon our experiences targeting CD19 and CD22 in B-cell acute lymphoblastic leukemia (B-ALL), we report on our phase 1 dose-escalation study of a novel murine stem cell virus (MSCV)-CD19/CD22-4-1BB bivalent CAR T-cell (CD19.22.BBζ) for children and young adults (CAYA) with B-cell malignancies. Primary objectives included toxicity and dose finding. Secondary objectives included response rates and relapse-free survival (RFS). Biologic correlatives included laboratory investigations, CAR T-cell expansion and cytokine profiling. Twenty patients, ages 5.4 to 34.6 years, with B-ALL received CD19.22.BBζ. The complete response (CR) rate was 60% (12 of 20) in the full cohort and 71.4% (10 of 14) in CAR-naïve patients. Ten (50%) developed cytokine release syndrome (CRS), with 3 (15%) having ≥ grade 3 CRS and only 1 experiencing neurotoxicity (grade 3). The 6- and 12-month RFS in those achieving CR was 80.8% (95% confidence interval [CI]: 42.4%-94.9%) and 57.7% (95% CI: 22.1%-81.9%), respectively. Limited CAR T-cell expansion and persistence of MSCV-CD19.22.BBζ compared with EF1α-CD22.BBζ prompted laboratory investigations comparing EF1α vs MSCV promoters, which did not reveal major differences. Limited CD22 targeting with CD19.22.BBζ, as evaluated by ex vivo cytokine secretion and leukemia eradication in humanized mice, led to development of a novel bicistronic CD19.28ζ/CD22.BBζ construct with enhanced cytokine production against CD22. With demonstrated safety and efficacy of CD19.22.BBζ in a heavily pretreated CAYA B-ALL cohort, further optimization of combinatorial antigen targeting serves to overcome identified limitations (www.clinicaltrials.gov #NCT03448393).
    MeSH term(s) Animals ; Antigens, CD19 ; Burkitt Lymphoma ; Cytokine Release Syndrome ; Cytokines ; Humans ; Immunotherapy, Adoptive/adverse effects ; Immunotherapy, Adoptive/methods ; Lymphoma, B-Cell ; Mice ; Precursor Cell Lymphoblastic Leukemia-Lymphoma ; Receptors, Antigen, T-Cell/genetics ; Receptors, Chimeric Antigen/genetics ; Recurrence ; T-Lymphocytes
    Chemical Substances Antigens, CD19 ; Cytokines ; Receptors, Antigen, T-Cell ; Receptors, Chimeric Antigen
    Language English
    Publishing date 2022-05-17
    Publishing country United States
    Document type Clinical Trial, Phase I ; Journal Article
    ZDB-ID 80069-7
    ISSN 1528-0020 ; 0006-4971
    ISSN (online) 1528-0020
    ISSN 0006-4971
    DOI 10.1182/blood.2022015795
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    Zs.MO 364: Show issues

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  5. Article ; Online: Blinatumomab Nonresponse and High-Disease Burden Are Associated With Inferior Outcomes After CD19-CAR for B-ALL.

    Myers, Regina M / Taraseviciute, Agne / Steinberg, Seth M / Lamble, Adam J / Sheppard, Jennifer / Yates, Bonnie / Kovach, Alexandra E / Wood, Brent / Borowitz, Michael J / Stetler-Stevenson, Maryalice / Yuan, Constance M / Pillai, Vinodh / Foley, Toni / Chung, Perry / Chen, Lee / Lee, Daniel W / Annesley, Colleen / DiNofia, Amanda / Grupp, Stephan A /
    John, Samuel / Bhojwani, Deepa / Brown, Patrick A / Laetsch, Theodore W / Gore, Lia / Gardner, Rebecca A / Rheingold, Susan R / Pulsipher, Michael A / Shah, Nirali N

    Journal of clinical oncology : official journal of the American Society of Clinical Oncology

    2021  Volume 40, Issue 9, Page(s) 932–944

    Abstract: ... induce remission in relapsed or refractory B-cell acute lymphoblastic leukemia (ALL) but are also associated ...

    Abstract Purpose: CD19-targeted chimeric antigen receptor T cells (CD19-CAR) and blinatumomab effectively induce remission in relapsed or refractory B-cell acute lymphoblastic leukemia (ALL) but are also associated with CD19 antigen modulation. There are limited data regarding the impact of prior blinatumomab exposure on subsequent CD19-CAR outcomes.
    Patients and methods: We conducted a multicenter, retrospective review of children and young adults with relapsed or refractory ALL who received CD19-CAR between 2012 and 2019. Primary objectives addressed 6-month relapse-free survival (RFS) and event-free survival (EFS), stratified by blinatumomab use. Secondary objectives included comparison of longer-term survival outcomes, complete remission rates, CD19 modulation, and identification of factors associated with EFS.
    Results: Of 420 patients (median age, 12.7 years; interquartile range, 7.1-17.5) treated with commercial tisagenlecleucel or one of three investigational CD19-CAR constructs, 77 (18.3%) received prior blinatumomab. Blinatumomab-exposed patients more frequently harbored
    Conclusion: With the largest series to date in pediatric CD19-CAR, and, to our knowledge, the first to study the impact of sequential CD19 targeting, we demonstrate that blinatumomab nonresponse and high-disease burden were independently associated with worse RFS and EFS, identifying important indicators of long-term outcomes following CD19-CAR.
    MeSH term(s) Acute Disease ; Antibodies, Bispecific/adverse effects ; Antigens, CD19 ; Child ; Cost of Illness ; Humans ; Immunotherapy, Adoptive/adverse effects ; Lymphoma, B-Cell/drug therapy ; Precursor Cell Lymphoblastic Leukemia-Lymphoma/drug therapy ; Recurrence ; Young Adult
    Chemical Substances Antibodies, Bispecific ; Antigens, CD19 ; blinatumomab (4FR53SIF3A)
    Language English
    Publishing date 2021-11-12
    Publishing country United States
    Document type Journal Article ; Multicenter Study ; Research Support, N.I.H., Intramural
    ZDB-ID 604914-x
    ISSN 1527-7755 ; 0732-183X
    ISSN (online) 1527-7755
    ISSN 0732-183X
    DOI 10.1200/JCO.21.01405
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  6. Article: CD19-Targeted Chimeric Antigen Receptor T-cell Therapy for Concomitant Diffuse Large B-cell Lymphoma and Multiple Myeloma.

    D'Ovidio, Tyler / Ciccolini, Kathryn / Kalac, Matko / Osman, Keren / Steinberg, Amir

    Cureus

    2023  Volume 15, Issue 9, Page(s) e44542

    Abstract: Multiple myeloma (MM) and diffuse large B-cell lymphoma (DLBCL) comprise a large fraction ... CD19- and B-cell maturation antigen-targeted chimeric antigen receptor (CAR) T-cell therapies have been ...

    Abstract Multiple myeloma (MM) and diffuse large B-cell lymphoma (DLBCL) comprise a large fraction of hematologic malignancies diagnosed each year. However, the co-occurrence of these conditions in the same patient is rare. CD19- and B-cell maturation antigen-targeted chimeric antigen receptor (CAR) T-cell therapies have been approved in recent years with promising responses. Here, we present a patient who presented following a bone marrow biopsy that revealed MM with 20% lambda-restricted plasma cells with no evidence of lymphoma involvement in the marrow. A subsequent lymph node biopsy of a right thigh mass was done and revealed DLBCL. The patient received CD19-targeted CAR T-cell therapy and has no detectable MM or DLBCL. To our knowledge, this is the first case report in the literature describing a patient with concomitant MM and DLBCL who received CD19-targeted CAR T-cell therapy.
    Language English
    Publishing date 2023-09-01
    Publishing country United States
    Document type Case Reports
    ZDB-ID 2747273-5
    ISSN 2168-8184
    ISSN 2168-8184
    DOI 10.7759/cureus.44542
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  7. Article ; Online: Refinement of the Reflective Function Questionnaire for Youth (RFQY) Scale B Using Item Response Theory.

    Sharp, Carla / Steinberg, Lynne / McLaren, Veronica / Weir, Stuart / Ha, Carolyn / Fonagy, Peter

    Assessment

    2021  Volume 29, Issue 6, Page(s) 1204–1215

    Abstract: ... for Youth (RFQY) Scale B. Data from a non-clinical sample of young people ( ...

    Abstract We conducted item response theory analyses to refine the Reflective Function Questionnaire for Youth (RFQY) Scale B. Data from a non-clinical sample of young people (
    MeSH term(s) Adolescent ; Adult ; Humans ; Psychometrics ; Reproducibility of Results ; Surveys and Questionnaires ; Young Adult
    Language English
    Publishing date 2021-04-02
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1362144-0
    ISSN 1552-3489 ; 1073-1911
    ISSN (online) 1552-3489
    ISSN 1073-1911
    DOI 10.1177/10731911211003971
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  8. Article ; Online: Search for New Phenomena in Final States with Two Leptons and One or No b-Tagged Jets at sqrt[s]=13  TeV Using the ATLAS Detector.

    Aad, G / Abbott, B / Abbott, D C / Abed Abud, A / Abeling, K / Abhayasinghe, D K / Abidi, S H / Abramowicz, H / Abreu, H / Abulaiti, Y / Abusleme Hoffman, A C / Acharya, B S / Achkar, B / Adam, L / Adam Bourdarios, C / Adamczyk, L / Adamek, L / Adelman, J / Adiguzel, A /
    Adorni, S / Adye, T / Affolder, A A / Afik, Y / Agapopoulou, C / Agaras, M N / Agarwala, J / Aggarwal, A / Agheorghiesei, C / Aguilar-Saavedra, J A / Ahmad, A / Ahmadov, F / Ahmed, W S / Ai, X / Aielli, G / Akatsuka, S / Akbiyik, M / Åkesson, T P A / Akimov, A V / Al Khoury, K / Alberghi, G L / Albert, J / Alconada Verzini, M J / Alderweireldt, S / Aleksa, M / Aleksandrov, I N / Alexa, C / Alexopoulos, T / Alfonsi, A / Alfonsi, F / Alhroob, M / Ali, B / Ali, S / Aliev, M / Alimonti, G / Allaire, C / Allbrooke, B M M / Allport, P P / Aloisio, A / Alonso, F / Alpigiani, C / Alunno Camelia, E / Alvarez Estevez, M / Alviggi, M G / Amaral Coutinho, Y / Ambler, A / Ambroz, L / Amelung, C / Amidei, D / Amor Dos Santos, S P / Amoroso, S / Amrouche, C S / Anastopoulos, C / Andari, N / Andeen, T / Anders, J K / Andrean, S Y / Andreazza, A / Andrei, V / Angelidakis, S / Angerami, A / Anisenkov, A V / Annovi, A / Antel, C / Anthony, M T / Antipov, E / Antonelli, M / Antrim, D J A / Anulli, F / Aoki, M / Aparisi Pozo, J A / Aparo, M A / Aperio Bella, L / Aranzabal, N / Araujo Ferraz, V / Arcangeletti, C / Arce, A T H / Arena, E / Arguin, J-F / Argyropoulos, S / Arling, J-H / Armbruster, A J / Armstrong, A / Arnaez, O / Arnold, H / Arrubarrena Tame, Z P / Artoni, G / Asada, H / Asai, K / Asai, S / Asbah, N A / Asimakopoulou, E M / Asquith, L / Assahsah, J / Assamagan, K / Astalos, R / Atkin, R J / Atkinson, M / Atlay, N B / Atmani, H / Atmasiddha, P A / Augsten, K / Auricchio, S / Austrup, V A / Avolio, G / Ayoub, M K / Azuelos, G / Babal, D / Bachacou, H / Bachas, K / Backman, F / Badea, A / Bagnaia, P / Bahrasemani, H / Bailey, A J / Bailey, V R / Baines, J T / Bakalis, C / Baker, O K / Bakker, P J / Bakos, E / Bakshi Gupta, D / Balaji, S / Balasubramanian, R / Baldin, E M / Balek, P / Ballabene, E / Balli, F / Balunas, W K / Balz, J / Banas, E / Bandieramonte, M / Bandyopadhyay, A / Barak, L / Barberio, E L / Barberis, D / Barbero, M / Barbour, G / Barends, K N / Barillari, 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Bergeaas Kuutmann, E / Berger, N / Bergmann, B / Bergsten, L J / Beringer, J / Berlendis, S / Bernardi, G / Bernius, C / Bernlochner, F U / Berry, T / Berta, P / Berthold, A / Bertram, I A / Bessidskaia Bylund, O / Bethke, S / Betti, A / Bevan, A J / Bhatta, S / Bhattacharya, D S / Bhattarai, P / Bhopatkar, V S / Bi, R / Bianchi, R M / Biebel, O / Bielski, R / Biesuz, N V / Biglietti, M / Billoud, T R V / Bindi, M / Bingul, A / Bini, C / Biondi, S / Birch-Sykes, C J / Bird, G A / Birman, M / Bisanz, T / Biswal, J P / Biswas, D / Bitadze, A / Bittrich, C / Bjørke, K / Bloch, I / Blocker, C / Blue, A / Blumenschein, U / Bobbink, G J / Bobrovnikov, V S / Bogavac, D / Bogdanchikov, A G / Bohm, C / Boisvert, V / Bokan, P / Bold, T / Bomben, M / Bona, M / Boonekamp, M / Booth, C D / Borbély, A G / Borecka-Bielska, H M / Borgna, L S / Borissov, G / Bortoletto, D / Boscherini, D / Bosman, M / Bossio Sola, J D / Bouaouda, K / Boudreau, J / Bouhova-Thacker, E V / Boumediene, D / Bouquet, R / Boveia, A / Boyd, J / Boye, D / Boyko, I R / Bozson, A J / Bracinik, J / Brahimi, N / Brandt, G / Brandt, O / Braren, F / Brau, B / Brau, J E / Breaden Madden, W D / Brendlinger, K / Brener, R / Brenner, L / Brenner, R / Bressler, S / Brickwedde, B / Briglin, D L / Britton, D / Britzger, D / Brock, I / Brock, R / Brooijmans, G / Brooks, W K / Brost, E / Bruckman de Renstrom, P A / Brüers, B / Bruncko, D / Bruni, A / Bruni, G / Bruschi, M / Bruscino, N / Bryngemark, L / Buanes, T / Buat, Q / Buchholz, P / Buckley, A G / Budagov, I A / Bugge, M K / Bulekov, O / Bullard, B A / Burch, T J / Burdin, S / Burgard, C D / Burger, A M / Burghgrave, B / Burr, J T P / Burton, C D / Burzynski, J C / Büscher, V / Bussey, P J / Butler, J M / Buttar, C M / Butterworth, J M / Buttinger, W / Buxo Vazquez, C J / Buzykaev, A R / Cabras, G / Cabrera Urbán, S / Caforio, D / Cai, H / Cairo, V M M / Cakir, O / Calace, N / Calafiura, P / Calderini, G / Calfayan, P / Callea, G / Caloba, L P / Caltabiano, A / 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/ Sekula, S J / Selem, L P / Semprini-Cesari, N / Sen, S / Serfon, C / Serin, L / Serkin, L / Sessa, M / Severini, H / Sevova, S / Sforza, F / Sfyrla, A / Shabalina, E / Shaheen, R / Shahinian, J D / Shaikh, N W / Shaked Renous, D / Shan, L Y / Shapiro, M / Sharma, A / Sharma, A S / Sharma, S / Shatalov, P B / Shaw, K / Shaw, S M / Sherwood, P / Shi, L / Shimmin, C O / Shimogama, Y / Shinner, J D / Shipsey, I P J / Shirabe, S / Shiyakova, M / Shlomi, J / Shochet, M J / Shojaii, J / Shope, D R / Shrestha, S / Shrif, E M / Shroff, M J / Shulga, E / Sicho, P / Sickles, A M / Sideras Haddad, E / Sidiropoulou, O / Sidoti, A / Siegert, F / Sijacki, Dj / Silva Oliveira, M V / Silverstein, S B / Simion, S / Simoniello, R / Simsek, S / Sinervo, P / Sinetckii, V / Singh, S / Sinha, S / Sioli, M / Siral, I / Sivoklokov, S Yu / Sjölin, J / Skaf, A / Skorda, E / Skubic, P / Slawinska, M / Sliwa, K / Smakhtin, V / Smart, B H / Smiesko, J / Smirnov, S Yu / Smirnov, Y / Smirnova, L N / Smirnova, O / 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/ Tsybychev, D / Tu, Y / Tudorache, A / Tudorache, V / Tuna, A N / Turchikhin, S / Turgeman, D / Turk Cakir, I / Turner, R J / Turra, R / Tuts, P M / Tzamarias, S / Tzanis, P / Tzovara, E / Uchida, K / Ukegawa, F / Unal, G / Unal, M / Undrus, A / Unel, G / Ungaro, F C / Uno, K / Urban, J / Urquijo, P / Usai, G / Ushioda, R / Usman, M / Uysal, Z / Vacek, V / Vachon, B / Vadla, K O H / Vafeiadis, T / Valderanis, C / Valdes Santurio, E / Valente, M / Valentinetti, S / Valero, A / Valéry, L / Vallance, R A / Vallier, A / Valls Ferrer, J A / Van Daalen, T R / Van Gemmeren, P / Van Stroud, S / Van Vulpen, I / Vanadia, M / Vandelli, W / Vandenbroucke, M / Vandewall, E R / Vannicola, D / Vannoli, L / Vari, R / Varnes, E W / Varni, C / Varol, T / Varouchas, D / Varvell, K E / Vasile, M E / Vaslin, L / Vasquez, G A / Vazeille, F / Vazquez Furelos, D / Vazquez Schroeder, T / Veatch, J / Vecchio, V / Veen, M J / Veliscek, I / Veloce, L M / Veloso, F / Veneziano, S / Ventura, A / Verbytskyi, A / Verducci, M / Vergis, C / Verissimo De Araujo, M / Verkerke, W / Vermeulen, A T / Vermeulen, J C / Vernieri, C / Verschuuren, P J / Vesterbacka, M L / Vetterli, M C / Viaux Maira, N / Vickey, T / Vickey Boeriu, O E / Viehhauser, G H A / Vigani, L / Villa, M / Villaplana Perez, M / Villhauer, E M / Vilucchi, E / Vincter, M G / Virdee, G S / Vishwakarma, A / Vittori, C / Vivarelli, I / Vladimirov, V / Voevodina, E / Vogel, M / Vokac, P / Von Ahnen, J / von Buddenbrock, S E / Von Toerne, E / Vorobel, V / Vorobev, K / Vos, M / Vossebeld, J H / Vozak, M / Vranjes, N / Vranjes Milosavljevic, M / Vrba, V / Vreeswijk, M / Vu, N K / Vuillermet, R / Vukotic, I / Wada, S / Wagner, C / Wagner, P / Wagner, W / Wahdan, S / Wahlberg, H / Wakasa, R / Wakida, M / Walbrecht, V M / Walder, J / Walker, R / Walker, S D / Walkowiak, W / Wang, A M / Wang, A Z / Wang, C / Wang, H / Wang, J / Wang, P / Wang, R-J / Wang, R / Wang, S M / Wang, S / Wang, T / Wang, W T / Wang, W X / Wang, X / Wang, Y / Wang, Z / 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Zhukov, K / Zhulanov, V / Zieminska, D / Zimine, N I / Zimmermann, S / Ziolkowski, M / Živković, L / Zoccoli, A / Zoch, K / Zorbas, T G / Zormpa, O / Zou, W / Zwalinski, L

    Physical review letters

    2021  Volume 127, Issue 14, Page(s) 141801

    Abstract: A search for new phenomena is presented in final states with two leptons and one or no b-tagged ... is observed in the data. Inspired by the B-meson decay anomalies, a four-fermion contact interaction ... between two quarks (b, s) and two leptons (ee or μμ) is used as a benchmark signal model, which is ...

    Abstract A search for new phenomena is presented in final states with two leptons and one or no b-tagged jets. The event selection requires the two leptons to have opposite charge, the same flavor (electrons or muons), and a large invariant mass. The analysis is based on the full run-2 proton-proton collision dataset recorded at a center-of-mass energy of sqrt[s]=13  TeV by the ATLAS experiment at the LHC, corresponding to an integrated luminosity of 139  fb^{-1}. No significant deviation from the expected background is observed in the data. Inspired by the B-meson decay anomalies, a four-fermion contact interaction between two quarks (b, s) and two leptons (ee or μμ) is used as a benchmark signal model, which is characterized by the energy scale and coupling, Λ and g_{*}, respectively. Contact interactions with Λ/g_{*} lower than 2.0 (2.4) TeV are excluded for electrons (muons) at the 95% confidence level, still far below the value that is favored by the B-meson decay anomalies. Model-independent limits are set as a function of the minimum dilepton invariant mass, which allow the results to be reinterpreted in various signal scenarios.
    Language English
    Publishing date 2021-10-15
    Publishing country United States
    Document type Journal Article
    ZDB-ID 208853-8
    ISSN 1079-7114 ; 0031-9007
    ISSN (online) 1079-7114
    ISSN 0031-9007
    DOI 10.1103/PhysRevLett.127.141801
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  9. Article ; Online: Risk of hepatitis B reactivation and cytomegalovirus related infections with Mogamulizumab: A retrospective study of international pharmacovigilance database.

    Wang, Shuai / Jayarangaiah, Apoorva / Malone, Mariuxi / Elrafei, Tarek / Steinberg, Lewis / Kumar, Abhishek

    EClinicalMedicine

    2020  Volume 28, Page(s) 100601

    Abstract: ... risk of hepatitis B reactivation and cytomegalovirus (CMV) related infection post-Moga ... all cases of hepatitis B reactivation and CMV related infection between January 1, 2011, and December 31 ... Eight cases were reported for hepatitis B reactivation with Moga use, compared to 2290 cases ...

    Abstract Background: Mogamulizumab (Moga) is a C-C chemokine receptor-4 antibody approved in the United States for relapsed /refractory mycosis fungoides and Sézary syndrome. Few cases reported an increased risk of hepatitis B reactivation and cytomegalovirus (CMV) related infection post-Moga. However, literature is limited to mainly case reports and series, while no study has used the Food and Drug Administration adverse events reporting system (FARES) database to investigate the relationship.
    Methods: Using United States Food and Drug Administration adverse events reporting system database, we collected all cases of hepatitis B reactivation and CMV related infection between January 1, 2011, and December 31, 2019, for Moga and other drugs. The reporting odds ratio (ROR) was calculated, which was considered significant when the lower limit of 95% confidence interval (CI) >1.
    Findings: Three hundred and thirty-eight total adverse cases were reported for Moga during the study period, with 261 cases reported indication for use, including cutaneous T cell lymphoma (47.04%), and adult T cell leukemia/lymphoma (30.18%). Eight cases were reported for hepatitis B reactivation with Moga use, compared to 2290 cases with other medications. The ROR is 143.67 (
    Interpretation: A signal has been identified between Moga exposure and hepatitis B reactivation as well as CMV related infection. A consideration in future studies should be placed on determining the relationship and investigating the need for pre-treatment screening, close monitoring, and utilization of prophylaxis in this population-based on pre-treatment risks.
    Funding: None.
    Language English
    Publishing date 2020-10-16
    Publishing country England
    Document type Journal Article
    ISSN 2589-5370
    ISSN (online) 2589-5370
    DOI 10.1016/j.eclinm.2020.100601
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  10. Article ; Online: Bruton's tyrosine kinase promotes persistence of mature anti-insulin B cells.

    Bonami, Rachel H / Sullivan, Allison M / Case, James B / Steinberg, Hannah E / Hoek, Kristen L / Khan, Wasif N / Kendall, Peggy L

    Journal of immunology (Baltimore, Md. : 1950)

    2014  Volume 192, Issue 4, Page(s) 1459–1470

    Abstract: Autoreactive B lymphocytes are essential for the development of T cell-mediated type 1 diabetes ... T1D). Cytoplasmic Bruton's tyrosine kinase (BTK) is a key component of B cell signaling, and ... function of autoreactive B cells is not clear. To evaluate the contributions of BTK, we used mice ...

    Abstract Autoreactive B lymphocytes are essential for the development of T cell-mediated type 1 diabetes (T1D). Cytoplasmic Bruton's tyrosine kinase (BTK) is a key component of B cell signaling, and its deletion in T1D-prone NOD mice significantly reduces diabetes. However, the role of BTK in the survival and function of autoreactive B cells is not clear. To evaluate the contributions of BTK, we used mice in which B cells express an anti-insulin BCR (125Tg) and promote T1D, despite being anergic. Crossing Btk deficiency onto 125Tg mice reveals that, in contrast to immature B cells, mature anti-insulin B cells are exquisitely dependent upon BTK, because their numbers are reduced by 95%. BTK kinase domain inhibition reproduces this effect in mature anti-insulin B cells, with less impact at transitional stages. The increased dependence of anti-insulin B cells on BTK became particularly evident in an Igκ locus site-directed model, in which 50% of B cells edit their BCRs to noninsulin specificities; Btk deficiency preferentially depletes insulin binders from the follicular and marginal zone B cell subsets. The persistent few Btk-deficient anti-insulin B cells remain competent to internalize Ag and invade pancreatic islets. As such, loss of BTK does not significantly reduce diabetes incidence in 125Tg/NOD mice as it does in NOD mice with a normal B cell repertoire. Thus, BTK targeting may not impair autoreactive anti-insulin B cell function, yet it may provide protection in an endogenous repertoire by decreasing the relative availability of mature autoreactive B cells.
    MeSH term(s) Agammaglobulinaemia Tyrosine Kinase ; Animals ; B-Lymphocytes/immunology ; Cells, Cultured ; Diabetes Mellitus, Type 1/immunology ; Immunoglobulins/immunology ; Insulin/immunology ; Insulin Antibodies/immunology ; Mice ; Mice, Inbred C57BL ; Mice, Inbred NOD ; Mice, Transgenic ; Protein-Tyrosine Kinases/genetics ; Protein-Tyrosine Kinases/immunology ; Receptors, Antigen, B-Cell/metabolism
    Chemical Substances IgK ; Immunoglobulins ; Insulin ; Insulin Antibodies ; Receptors, Antigen, B-Cell ; Protein-Tyrosine Kinases (EC 2.7.10.1) ; Agammaglobulinaemia Tyrosine Kinase (EC 2.7.10.2) ; Btk protein, mouse (EC 2.7.10.2)
    Language English
    Publishing date 2014-01-22
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
    ZDB-ID 3056-9
    ISSN 1550-6606 ; 0022-1767 ; 1048-3233 ; 1047-7381
    ISSN (online) 1550-6606
    ISSN 0022-1767 ; 1048-3233 ; 1047-7381
    DOI 10.4049/jimmunol.1300125
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