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  1. Article ; Online: Recent advances in the management of hepatocellular carcinoma: First, cut … is the deepest or is it "no-touch" and exercise?

    Teoh, Narci C

    Journal of gastroenterology and hepatology

    2019  Volume 34, Issue 3, Page(s) 481–482

    MeSH term(s) Carcinoma, Hepatocellular/therapy ; Chemoembolization, Therapeutic/methods ; Exercise Therapy ; Hepatectomy ; Humans ; Liver Neoplasms/therapy ; Radiofrequency Ablation
    Language English
    Publishing date 2019-03-01
    Publishing country Australia
    Document type Editorial
    ZDB-ID 632882-9
    ISSN 1440-1746 ; 0815-9319
    ISSN (online) 1440-1746
    ISSN 0815-9319
    DOI 10.1111/jgh.14632
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  2. Article ; Online: Statins as early therapy to mitigate COVID-19 (SARS-CoV-2)-associated ARDS and cytokine storm syndrome - time is of the essence.

    Teoh, Narci / Farrell, Geoff

    Journal of clinical and translational research

    2020  Volume 5, Issue 5, Page(s) 227–229

    Keywords covid19
    Language English
    Publishing date 2020-04-18
    Publishing country Singapore
    Document type Journal Article
    ZDB-ID 3019815-X
    ISSN 2424-810X ; 2382-6533
    ISSN (online) 2424-810X
    ISSN 2382-6533
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  3. Article: Statins as early therapy to mitigate COVID-19 (SARS-CoV-2)-associated ARDS and cytokine storm syndrome - time is of the essence

    Teoh, Narci / Farrell, Geoff

    Journal of clinical and translational research

    Keywords covid19
    Publisher WHO
    Document type Article
    Note WHO #Covidence: #632291
    Database COVID19

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  4. Article ; Online: Should we stop "MaS"ing about with steatotic liver grafts and "MiS" out on potential donor livers?

    Teoh, Narci C

    Journal of gastroenterology and hepatology

    2012  Volume 27, Issue 3, Page(s) 422–424

    MeSH term(s) Fatty Liver/complications ; Fatty Liver/diagnosis ; Fatty Liver/epidemiology ; Fatty Liver/pathology ; Fatty Liver/therapy ; Female ; Graft Survival ; Humans ; Liver Transplantation/physiology ; Male ; Practice Guidelines as Topic ; Renal Insufficiency/etiology
    Language English
    Publishing date 2012-03
    Publishing country Australia
    Document type Comment ; Editorial
    ZDB-ID 632882-9
    ISSN 1440-1746 ; 0815-9319
    ISSN (online) 1440-1746
    ISSN 0815-9319
    DOI 10.1111/j.1440-1746.2012.07061.x
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  5. Article ; Online: Ku70 senses cytosolic DNA and assembles a tumor-suppressive signalosome.

    Pandey, Abhimanu / Shen, Cheng / Feng, Shouya / Enosi Tuipulotu, Daniel / Ngo, Chinh / Liu, Cheng / Kurera, Melan / Mathur, Anukriti / Venkataraman, Shweta / Zhang, Jing / Talaulikar, Dipti / Song, Renhua / Wong, Justin J-L / Teoh, Narci / Kaakoush, Nadeem O / Man, Si Ming

    Science advances

    2024  Volume 10, Issue 4, Page(s) eadh3409

    Abstract: The innate immune response contributes to the development or attenuation of acute and chronic diseases, including cancer. Microbial DNA and mislocalized DNA from damaged host cells can activate different host responses that shape disease outcomes. Here, ... ...

    Abstract The innate immune response contributes to the development or attenuation of acute and chronic diseases, including cancer. Microbial DNA and mislocalized DNA from damaged host cells can activate different host responses that shape disease outcomes. Here, we show that mice and humans lacking a single allele of the DNA repair protein Ku70 had increased susceptibility to the development of intestinal cancer. Mechanistically, Ku70 translocates from the nucleus into the cytoplasm where it binds to cytosolic DNA and interacts with the GTPase Ras and the kinase Raf, forming a tripartite protein complex and docking at Rab5
    MeSH term(s) Animals ; Humans ; Mice ; Cell Proliferation ; DNA ; MAP Kinase Signaling System ; Neoplasms/genetics ; Signal Transduction
    Chemical Substances DNA (9007-49-2) ; Xrcc6 protein, mouse (EC 3.6.4.12) ; Xrcc6 protein, human (EC 3.6.4.12)
    Language English
    Publishing date 2024-01-26
    Publishing country United States
    Document type Journal Article
    ZDB-ID 2810933-8
    ISSN 2375-2548 ; 2375-2548
    ISSN (online) 2375-2548
    ISSN 2375-2548
    DOI 10.1126/sciadv.adh3409
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  6. Article ; Online: Non-alcoholic fatty liver disease.

    Farrell, Geoffrey C / Wardell, Rebecca / Teoh, Narci / Chitturi, Shiv

    Internal medicine journal

    2019  Volume 49, Issue 5, Page(s) 681–683

    MeSH term(s) Australia ; Hospitals, Urban ; Humans ; Non-alcoholic Fatty Liver Disease ; Prevalence ; Risk Factors
    Language English
    Publishing date 2019-05-12
    Publishing country Australia
    Document type Letter ; Comment
    ZDB-ID 2045436-3
    ISSN 1445-5994 ; 1444-0903
    ISSN (online) 1445-5994
    ISSN 1444-0903
    DOI 10.1111/imj.14283
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    Zs.MO 424: Show issues

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  7. Article ; Online: Pre-"EMT"ing key processes in liver carcinogenesis: Growing evidence for how malignant hepatocytes invade and conquer.

    Teoh, Narci C

    Hepatology (Baltimore, Md.)

    2010  Volume 52, Issue 1, Page(s) 384–388

    Language English
    Publishing date 2010-07
    Publishing country United States
    Document type Comment ; Journal Article
    ZDB-ID 604603-4
    ISSN 1527-3350 ; 0270-9139
    ISSN (online) 1527-3350
    ISSN 0270-9139
    DOI 10.1002/hep.23777
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    Zs.A 1660: Show issues Location:
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  8. Article ; Online: Much ado about Nutlin.

    Teoh, Narci C

    Journal of gastroenterology and hepatology

    2011  Volume 26, Issue 2, Page(s) 213–215

    MeSH term(s) Animals ; Antineoplastic Agents/pharmacology ; Apoptosis/drug effects ; Carcinoma, Hepatocellular/enzymology ; Carcinoma, Hepatocellular/genetics ; Carcinoma, Hepatocellular/pathology ; Cell Cycle/drug effects ; Cell Proliferation/drug effects ; Cell Survival/drug effects ; DNA-Binding Proteins/genetics ; DNA-Binding Proteins/metabolism ; Enzyme Inhibitors/pharmacology ; Humans ; Imidazoles/pharmacology ; Liver Neoplasms/enzymology ; Liver Neoplasms/genetics ; Liver Neoplasms/pathology ; Mutation ; Nuclear Proteins/genetics ; Nuclear Proteins/metabolism ; Piperazines/pharmacology ; Proto-Oncogene Proteins c-mdm2/antagonists & inhibitors ; Proto-Oncogene Proteins c-mdm2/metabolism ; Tumor Protein p73 ; Tumor Suppressor Protein p53/genetics ; Tumor Suppressor Protein p53/metabolism ; Tumor Suppressor Proteins/genetics ; Tumor Suppressor Proteins/metabolism
    Chemical Substances Antineoplastic Agents ; DNA-Binding Proteins ; Enzyme Inhibitors ; Imidazoles ; Nuclear Proteins ; Piperazines ; TP53 protein, human ; Tumor Protein p73 ; Tumor Suppressor Protein p53 ; Tumor Suppressor Proteins ; nutlin 3 (53IA0V845C) ; MDM2 protein, human (EC 2.3.2.27) ; Proto-Oncogene Proteins c-mdm2 (EC 2.3.2.27)
    Language English
    Publishing date 2011-02
    Publishing country Australia
    Document type Editorial ; Comment
    ZDB-ID 632882-9
    ISSN 1440-1746 ; 0815-9319
    ISSN (online) 1440-1746
    ISSN 0815-9319
    DOI 10.1111/j.1440-1746.2010.06588.x
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  9. Article ; Online: Hepatic ischemia reperfusion injury: Contemporary perspectives on pathogenic mechanisms and basis for hepatoprotection-the good, bad and deadly.

    Teoh, Narci C

    Journal of gastroenterology and hepatology

    2011  Volume 26 Suppl 1, Page(s) 180–187

    Abstract: Hepatic ischemia reperfusion (IR) injury is an important clinical problem complicating liver surgery and transplantation. The pathogenesis underlying reperfusion injury after warm ischemia is complex, encompassing a multitude of different cell types and ... ...

    Abstract Hepatic ischemia reperfusion (IR) injury is an important clinical problem complicating liver surgery and transplantation. The pathogenesis underlying reperfusion injury after warm ischemia is complex, encompassing a multitude of different cell types and signalling mechanisms innate and/or mobilized to the liver. Since the author's 2003 review in the Journal, considerable progress has been achieved in enhancing our understanding of some of the pathogenic pathways and crucial mediators of hepatic inflammation such as the heme oxygenase system, CXC chemokines, Toll-like receptors as well as the mode of parenchymal cell death in IR injury. A better appreciation of these mechanisms will accelerate efforts in designing optimal interventions to prevent hepatic IR injury and improve outcomes after liver transplantation.
    MeSH term(s) Animals ; Apoptosis ; Chemokines, CXC/metabolism ; Disease Models, Animal ; Heme Oxygenase (Decyclizing)/metabolism ; Humans ; Inflammation Mediators/metabolism ; Liver/blood supply ; Liver/immunology ; Liver/metabolism ; Liver Transplantation/adverse effects ; Necrosis ; Receptor for Advanced Glycation End Products ; Receptors, Immunologic/metabolism ; Reperfusion Injury/etiology ; Reperfusion Injury/immunology ; Reperfusion Injury/metabolism ; Reperfusion Injury/prevention & control ; Signal Transduction ; Toll-Like Receptors/metabolism ; Warm Ischemia/adverse effects
    Chemical Substances Chemokines, CXC ; Inflammation Mediators ; Receptor for Advanced Glycation End Products ; Receptors, Immunologic ; Toll-Like Receptors ; Heme Oxygenase (Decyclizing) (EC 1.14.14.18)
    Language English
    Publishing date 2011-01
    Publishing country Australia
    Document type Journal Article
    ZDB-ID 632882-9
    ISSN 1440-1746 ; 0815-9319
    ISSN (online) 1440-1746
    ISSN 0815-9319
    DOI 10.1111/j.1440-1746.2010.06584.x
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  10. Article ; Online: Remote hindlimb preconditioning and hepatoprotection: NO-table strides against liver ischaemia/reperfusion injury.

    Teoh, Narci C

    Clinical science (London, England : 1979)

    2011  Volume 121, Issue 6, Page(s) 253–255

    Abstract: Hepatic IR (ischaemia/reperfusion) injury is an important clinical problem complicating liver surgery and transplantation. IPC (ischaemic preconditioning) is a strategy whereby brief episodes of IR in an organ can induce an adaptive response to protect ... ...

    Abstract Hepatic IR (ischaemia/reperfusion) injury is an important clinical problem complicating liver surgery and transplantation. IPC (ischaemic preconditioning) is a strategy whereby brief episodes of IR in an organ can induce an adaptive response to protect against subsequent prolonged IR injury. However, trauma to vessels supplying the target organ is unavoidable using the technique of direct IPC. One amenable strategy would be to apply the protective preconditioning stimulus to an organ distant or remote from the target organ of interest, a technique known as RIPC (remote IPC). In the present issue of Clinical Science, Abu-Amara and co-workers utilize hindlimb RIPC as a novel therapeutic strategy against liver IR injury and investigate the mechanistic contribution of NO to hepatoprotection by administering C-PTIO [2-(4-carboxyphenyl)-4,4,5,5-tetramethylimidazoline-1-oxyl-3-oxide potassium salt], an NO scavenger. Their experiments set the stage for more definitive studies to demonstrate a discernible benefit for the utility of RIPC in liver surgery and transplantation.
    MeSH term(s) Animals ; Benzoates/pharmacology ; Hindlimb/blood supply ; Imidazoles/pharmacology ; Ischemic Preconditioning/methods ; Liver/blood supply ; Mice ; Nitric Oxide/antagonists & inhibitors ; Nitric Oxide/physiology ; Reperfusion Injury/physiopathology ; Reperfusion Injury/prevention & control
    Chemical Substances Benzoates ; Imidazoles ; 1,3-dihydroxy-4,4,5,5-tetramethyl-2-(4-carboxyphenyl)tetrahydroimidazole (145757-47-7) ; Nitric Oxide (31C4KY9ESH)
    Language English
    Publishing date 2011-09
    Publishing country England
    Document type Comment ; Journal Article
    ZDB-ID 206835-7
    ISSN 1470-8736 ; 0301-0538 ; 0009-0360 ; 0143-5221
    ISSN (online) 1470-8736
    ISSN 0301-0538 ; 0009-0360 ; 0143-5221
    DOI 10.1042/CS20110243
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