Article ; Online: Copper exposure induces mitochondrial dysfunction and hepatotoxicity via the induction of oxidative stress and PERK/ATF4 -mediated endoplasmic reticulum stress.
Environmental pollution (Barking, Essex : 1987)
2024 Volume 352, Page(s) 124145
Abstract: Copper is an essential trace element, and excessive exposure could result in hepatoxicity, however, the underlying molecular mechanisms remain incompletely understood. The present study is aimed to investigate the molecular mechanisms of copper sulfate ( ... ...
Abstract | Copper is an essential trace element, and excessive exposure could result in hepatoxicity, however, the underlying molecular mechanisms remain incompletely understood. The present study is aimed to investigate the molecular mechanisms of copper sulfate (CuSO |
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Language | English |
Publishing date | 2024-05-10 |
Publishing country | England |
Document type | Journal Article |
ZDB-ID | 280652-6 |
ISSN | 1873-6424 ; 0013-9327 ; 0269-7491 |
ISSN (online) | 1873-6424 |
ISSN | 0013-9327 ; 0269-7491 |
DOI | 10.1016/j.envpol.2024.124145 |
Database | MEDical Literature Analysis and Retrieval System OnLINE |
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