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Book ; Online: Jeremy Bentham : le peuple comme fiction

Guillot, Armand

(La philosophie à l'œuvre)

2014

Series title	La philosophie à l'œuvre
Keywords	Philosophy ; philosophie ; interprétation ; utilitarisme ; critique ; Bentham ; théorie de la fiction ; peuple ; contrat social
Language	0\|f
Size	1 electronic resource (396 pages)
Publisher	Éditions de la Sorbonne
Publishing place	Paris
Document type	Book ; Online
Note	French ; Open Access
HBZ-ID	HT021615992
ISBN	9782859447908 ; 2859447903
Database	ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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Article ; Online: Deciding When to Start Renal Replacement Therapy After Cardiac Surgery in Real Life.

Guillot, Olivier / Vandroux, David / Tiquet, Bérénice / Abdallah, Sahar / Jouan, Jérome / Tricard, Jeremy / Pihan, Franck

Journal of cardiothoracic and vascular anesthesia

2022 Volume 37, Issue 4, Page(s) 675–677

MeSH term(s)	Humans ; Cardiac Surgical Procedures ; Renal Replacement Therapy ; Continuous Renal Replacement Therapy ; Acute Kidney Injury
Language	English
Publishing date	2022-12-26
Publishing country	United States
Document type	Letter
ZDB-ID	1067317-9
ISSN	1532-8422 ; 1053-0770
ISSN (online)	1532-8422
ISSN	1053-0770
DOI	10.1053/j.jvca.2022.12.021
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Zs.A 2203: Show issues			Location: Je nach Verfügbarkeit (siehe Angabe bei Bestand) bis Jg. 1994: Bestellungen von Artikeln über das Online-Bestellformular Jg. 1995 - 2021: Lesesall (1.OG) ab Jg. 2022: Lesesaal (EG)
Zs.MO 31: Show issues

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Article ; Online: X-ray-free protocol for pectus deformities based on magnetic resonance imaging and a low-cost portable three-dimensional scanning device: a preliminary study.

Guillot, Marc-Samir / Rouchaud, Aymeric / Mounayer, Charbel / Tricard, Jérémy / Belgacem, Alexis / Auditeau, Emilie / Omraam, Olivier / Fourcade, Laurent / Ballouhey, Quentin

Interactive cardiovascular and thoracic surgery

2021 Volume 33, Issue 1, Page(s) 110–118

Abstract: Objectives: To compare a standard protocol using chest computed tomography (CT) to a non-irradiant protocol involving a low-cost portable 3D scanner and magnetic resonance imaging (MRI) for all pectus deformities based on the Haller index (HI).: ... ...

Abstract	Objectives: To compare a standard protocol using chest computed tomography (CT) to a non-irradiant protocol involving a low-cost portable 3D scanner and magnetic resonance imaging (MRI) for all pectus deformities based on the Haller index (HI). Methods: From April 2019 to March 2020, all children treated for pectus excavatum or carinatum at our institution were evaluated by chest CT, 3D scanning (iPad with Structure Sensor and Captevia-Rodin4D) and MRI. The main objectives were to compare the HI determined by CT or MRI to a derived index evaluated with 3D scanning, the external Haller index (EHI). The secondary objectives were to assess the inter-rater variability and the concordance between CT and MRI for the HI and the correction index. Results: Eleven patients were evaluated. We identified a strong correlation between the HI with MRI and the EHI (Pearson correlation coefficient = 0.900; P < 0.001), with a strong concordance between a radiologist and a non-radiologist using intra-class correlation for the HI with MRI (intra-class correlation coefficient = 0.995; [0.983; 0.999]) and the EHI (intra-class correlation coefficient = 0.978; [0.823; 0.995]). We also identified a marked correlation between the HI with CT and the EHI (Pearson coefficient = 0.855; P = 0.002), with a strong inter-rater concordance (intra-class correlation coefficient = 0.975; [0.901; 0.993]), a reliable concordance between CT and MRI for the HI and the correction index (Pearson coefficient = 0.886; P = 0.033). Conclusions: Non-irradiant pectus deformity assessment is possible in clinical practice, replacing CT with MRI and 3D scanning as a possible readily-accessible monitoring tool.
MeSH term(s)	Child ; Funnel Chest/diagnostic imaging ; Humans ; Magnetic Resonance Imaging ; Severity of Illness Index ; Thorax ; Tomography, X-Ray Computed
Language	English
Publishing date	2021-03-03
Publishing country	England
Document type	Journal Article
ZDB-ID	2095298-3
ISSN	1569-9285 ; 1569-9293
ISSN (online)	1569-9285
ISSN	1569-9293
DOI	10.1093/icvts/ivab036
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Zs.A 5730: Show issues

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Article ; Online: Sympathetic axonal sprouting induces changes in macrophage populations and protects against pancreatic cancer.

Nature communications

2022 Volume 13, Issue 1, Page(s) 1985

Abstract: Neuronal nerve processes in the tumor microenvironment were highlighted recently. However, the origin of intra-tumoral nerves remains poorly known, in part because of technical difficulties in tracing nerve fibers via conventional histological ... ...

Abstract	Neuronal nerve processes in the tumor microenvironment were highlighted recently. However, the origin of intra-tumoral nerves remains poorly known, in part because of technical difficulties in tracing nerve fibers via conventional histological preparations. Here, we employ three-dimensional (3D) imaging of cleared tissues for a comprehensive analysis of sympathetic innervation in a murine model of pancreatic ductal adenocarcinoma (PDAC). Our results support two independent, but coexisting, mechanisms: passive engulfment of pre-existing sympathetic nerves within tumors plus an active, localized sprouting of axon terminals into non-neoplastic lesions and tumor periphery. Ablation of the innervating sympathetic nerves increases tumor growth and spread. This effect is explained by the observation that sympathectomy increases intratumoral CD163
MeSH term(s)	Animals ; Carcinoma, Pancreatic Ductal ; Macrophages ; Mice ; Pancreatic Neoplasms ; Sympathetic Nervous System/physiology ; Tumor Microenvironment ; Pancreatic Neoplasms
Language	English
Publishing date	2022-04-13
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	2553671-0
ISSN	2041-1723 ; 2041-1723
ISSN (online)	2041-1723
ISSN	2041-1723
DOI	10.1038/s41467-022-29659-w
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Article ; Online: Sympathetic axonal sprouting induces changes in macrophage populations and protects against pancreatic cancer

Nature Communications, Vol 13, Iss 1, Pp 1-

2022 Volume 19

Abstract: The autonomic nervous systems densely innervate the pancreas, but its contribution to pancreatic ductal adenocarcinoma (PDAC) progression is not fully understood. Here, the authors characterize the pattern of sympathetic innervation by 3D imaging in a ... ...

Abstract	The autonomic nervous systems densely innervate the pancreas, but its contribution to pancreatic ductal adenocarcinoma (PDAC) progression is not fully understood. Here, the authors characterize the pattern of sympathetic innervation by 3D imaging in a murine model of PDAC and show that sympathectomy aggravates cancer progression.
Keywords	Science ; Q
Language	English
Publishing date	2022-04-01T00:00:00Z
Publisher	Nature Portfolio
Document type	Article ; Online
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: Aglycosylated extracellular loop of inwardly rectifying potassium channel 4.1 (KCNJ10) provides a target for autoimmune neuroinflammation.

Brain communications

2023 Volume 5, Issue 2, Page(s) fcad044

Abstract: Multiple sclerosis is an autoimmune disease of the central nervous system. Yet, the autoimmune targets are still undefined. The extracellular e1 sequence of KCNJ10, the inwardly rectifying potassium channel 4.1, has been subject to fierce debate for its ... ...

Abstract	Multiple sclerosis is an autoimmune disease of the central nervous system. Yet, the autoimmune targets are still undefined. The extracellular e1 sequence of KCNJ10, the inwardly rectifying potassium channel 4.1, has been subject to fierce debate for its role as a candidate autoantigen in multiple sclerosis. Inwardly rectifying potassium channel 4.1 is expressed in the central nervous system but also in peripheral tissues, raising concerns about the central nervous system-specificity of such autoreactivity. Immunization of C57Bl6/J female mice with the e1 peptide (amino acids 83-120 of Kir4.1) induced anti-e1 immunoglobulin G- and T-cell responses and promoted demyelinating encephalomyelitis with B cell central nervous system enrichment in leptomeninges and T cells/macrophages in central nervous system parenchyma from forebrain to spinal cord, mostly in the white matter. Within our cohort of multiple sclerosis patients (
Language	English
Publishing date	2023-02-22
Publishing country	England
Document type	Journal Article
ISSN	2632-1297
ISSN (online)	2632-1297
DOI	10.1093/braincomms/fcad044
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Article: High Glucose Activates YAP Signaling to Promote Vascular Inflammation.

Ortillon, Jeremy / Le Bail, Jean-Christophe / Villard, Elise / Léger, Bertrand / Poirier, Bruno / Girardot, Christine / Beeske, Sandra / Ledein, Laetitia / Blanchard, Véronique / Brieu, Patrice / Naimi, Souâd / Janiak, Philip / Guillot, Etienne / Meloni, Marco

Frontiers in physiology

2021 Volume 12, Page(s) 665994

Abstract: Background and aims: The YAP/TAZ signaling is known to regulate endothelial activation and vascular inflammation in response to shear stress. Moreover, YAP/TAZ signaling plays a role in the progression of cancers and renal damage associated with ... ...

Abstract	Background and aims: The YAP/TAZ signaling is known to regulate endothelial activation and vascular inflammation in response to shear stress. Moreover, YAP/TAZ signaling plays a role in the progression of cancers and renal damage associated with diabetes. However, whether YAP/TAZ signaling is also implicated in diabetes-associated vascular complications is not known. Methods: The effect of high glucose on YAP/TAZ signaling was firstly evaluated Results: In vitro Conclusion: With the current study we demonstrated that YAP signaling is activated by high glucose in endothelial cells
Language	English
Publishing date	2021-06-04
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2564217-0
ISSN	1664-042X
ISSN	1664-042X
DOI	10.3389/fphys.2021.665994
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Article ; Online: Two-Stage Bronchoscopic Endobronchial Valve Treatment Can Lead to Progressive Lung Volume Reduction and May Decrease Pneumothorax Risk.

Egenod, Thomas / Tricard, Jeremy / Fumat, Romane / Simonneau, Yannick / Favard, Florent / Guillot, Marc-Samir / Collot, Samia / Dupuis, Marion / Melloni, Boris / Vergnenegre, Alain / Guibert, Nicolas / Dusselier, Matthieu

International journal of chronic obstructive pulmonary disease

2021 Volume 16, Page(s) 1957–1965

Abstract: Background: Since successful development of endobronchial valves (EBV) as treatment for severe emphysema, its main complication, pneumothorax, remains an important concern.: Objective: We hypothesized that a two-step EBV implantation, during two ... ...

Abstract	Background: Since successful development of endobronchial valves (EBV) as treatment for severe emphysema, its main complication, pneumothorax, remains an important concern. Objective: We hypothesized that a two-step EBV implantation, during two distinct iterative procedures could lead to a more progressive target lobe volume reduction (TLVR) and thus ipsilateral lobe re-expansion, resulting in a significant decrease in the pneumothorax rate. Methods: This retrospective bi-center study carried out by Limoges and Toulouse University Hospitals included patients following the inclusion criteria established by the BLVR expert panel. All patients were treated by two distinct procedures: first, EBVs were placed in all but the most proximal segment or sub-segment. The remaining segment was treated subsequently. All patients had a complete evaluation before treatment, and 3 months after the second procedure. Results: Out of 58 patients included, only 4 pneumothoraxes (7%) occurred during the study. The other complications were pneumonia and severe COPD exacerbation (8.6% and 13.7% of patients, respectively). Significant improvement was found for FEV Conclusion: This new approach using EBV could reduce the incidence of pneumothorax without increasing other complication rates. Clinical and physiological outcomes are similar to those reported in studies using the conventional single-step treatment.
MeSH term(s)	Bronchoscopy ; Forced Expiratory Volume ; Humans ; Pneumonectomy/adverse effects ; Pneumothorax/diagnostic imaging ; Pneumothorax/etiology ; Pneumothorax/prevention & control ; Pulmonary Disease, Chronic Obstructive/diagnosis ; Pulmonary Disease, Chronic Obstructive/surgery ; Pulmonary Emphysema/diagnostic imaging ; Pulmonary Emphysema/surgery ; Retrospective Studies ; Treatment Outcome
Language	English
Publishing date	2021-06-28
Publishing country	New Zealand
Document type	Journal Article
ZDB-ID	2212419-6
ISSN	1178-2005 ; 1176-9106
ISSN (online)	1178-2005
ISSN	1176-9106
DOI	10.2147/COPD.S307829
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Article ; Online: Myasthenia Gravis Induced by Immune Checkpoint Inhibitors.

Becquart, Ondine / Lacotte, Julie / Malissart, Pauline / Nadal, Jeremy / Lesage, Candice / Guillot, Bernard / Du Thanh, Aurélie

Journal of immunotherapy (Hagerstown, Md. : 1997)

2019 Volume 42, Issue 8, Page(s) 309–312

Abstract: Immune checkpoint inhibitors deeply improved the prognosis of metastatic melanoma or other types of cancer, but their related adverse effects (AEs) can be very severe, especially when the neurological system is touched, as in myasthenia gravis (MG). It ... ...

Abstract	Immune checkpoint inhibitors deeply improved the prognosis of metastatic melanoma or other types of cancer, but their related adverse effects (AEs) can be very severe, especially when the neurological system is touched, as in myasthenia gravis (MG). It is a rare immune AE that can be life-threatening and can be revealed by several symptoms. We report a case of our experience and review the current literature of MG exacerbated or occurring during immunotherapy to describe characteristics of this AE, warn the oncologist about this toxicity, and summarize the treatments conducted. Thirty-four cases of MG were reported, mostly with anti-programmed cell death protein 1 checkpoint inhibitor, and with melanoma. Onset was quick after the first or second infusion. Treatment comprised corticosteroids, prostigmine, and more or less plasmapheresis or immunoglobulins. Prognosis is poor, as 13 patients died after MG. MG is a rare immune-related AE that must be rapidly evoked and treated in case of neurological symptoms emerging after immunotherapy.
MeSH term(s)	Aged ; Antineoplastic Agents, Immunological/adverse effects ; Female ; Humans ; Melanoma/drug therapy ; Myasthenia Gravis/drug therapy ; Myasthenia Gravis/etiology ; Neostigmine/therapeutic use ; Nivolumab/adverse effects ; Programmed Cell Death 1 Receptor/antagonists & inhibitors
Chemical Substances	Antineoplastic Agents, Immunological ; PDCD1 protein, human ; Programmed Cell Death 1 Receptor ; Nivolumab (31YO63LBSN) ; Neostigmine (3982TWQ96G)
Language	English
Publishing date	2019-06-26
Publishing country	United States
Document type	Case Reports ; Journal Article ; Review
ZDB-ID	1064067-8
ISSN	1537-4513 ; 1053-8550 ; 1524-9557
ISSN (online)	1537-4513
ISSN	1053-8550 ; 1524-9557
DOI	10.1097/CJI.0000000000000278
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Article ; Online: Aberrant hyperexpression of the RNA binding protein FMRP in tumors mediates immune evasion.

Science (New York, N.Y.)

2022 Volume 378, Issue 6621, Page(s) eabl7207

Abstract: Many human cancers manifest the capability to circumvent attack by the adaptive immune system. In this work, we identified a component of immune evasion that involves frequent up-regulation of fragile X mental retardation protein (FMRP) in solid tumors. ... ...

Abstract	Many human cancers manifest the capability to circumvent attack by the adaptive immune system. In this work, we identified a component of immune evasion that involves frequent up-regulation of fragile X mental retardation protein (FMRP) in solid tumors. FMRP represses immune attack, as revealed by cancer cells engineered to lack its expression. FMRP-deficient tumors were infiltrated by activated T cells that impaired tumor growth and enhanced survival in mice. Mechanistically, FMRP's immunosuppression was multifactorial, involving repression of the chemoattractant C-C motif chemokine ligand 7 (CCL7) concomitant with up-regulation of three immunomodulators-interleukin-33 (IL-33), tumor-secreted protein S (PROS1), and extracellular vesicles. Gene signatures associate FMRP's cancer network with poor prognosis and response to therapy in cancer patients. Collectively, FMRP is implicated as a regulator that orchestrates a multifaceted barrier to antitumor immune responses.
MeSH term(s)	Animals ; Humans ; Mice ; Fragile X Mental Retardation Protein/genetics ; Fragile X Mental Retardation Protein/metabolism ; Immune Evasion ; Neoplasms/immunology ; Immune Tolerance ; Chemokine CCL7/metabolism ; Interleukin-33 ; Protein S/metabolism
Chemical Substances	Fragile X Mental Retardation Protein (139135-51-6) ; FMR1 protein, human ; Chemokine CCL7 ; Interleukin-33 ; PROS1 protein, human ; Protein S
Language	English
Publishing date	2022-11-18
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	128410-1
ISSN	1095-9203 ; 0036-8075
ISSN (online)	1095-9203
ISSN	0036-8075
DOI	10.1126/science.abl7207
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Zs.MG 77: Show issues

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