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  1. Book ; Online ; E-Book: Occupational cancers

    Anttila, Sisko / Boffetta, Paolo

    2020  

    Author's details Sisko Anttila, Paolo Boffetta editors
    Keywords Oncology   ; Pathology ; Occupational medicine ; Epidemiology ; Public health ; Respiratory organs—Diseases
    Subject code 616.994
    Language English
    Size 1 Online-Ressource (xv, 640 Seiten), Illustrationen
    Edition Second edition
    Publisher Springer
    Publishing place Cham
    Publishing country Switzerland
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT020392965
    ISBN 978-3-030-30766-0 ; 9783030307653 ; 3-030-30766-2 ; 3030307654
    DOI 10.1007/978-3-030-30766-0
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  2. Book ; Online ; E-Book: Encyclopedia of cancer

    Boffetta, Paolo / Hainaut, Pierre

    2019  

    Author's details editors in chief Paolo Boffetta, Pierre Hainaut
    Keywords Cancer
    Subject code 616.994003
    Language English
    Size 1 Online-Ressource (xl, 1906 Seiten), Illustrationen, Diagramme, 28 cm
    Edition Third edition
    Publisher Elsevier AP
    Publishing place Amsterdam
    Publishing country Netherlands
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT020035938
    ISBN 978-0-128-12485-7 ; 9780128124840 ; 0-128-12485-7 ; 0128124849
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  3. Article ; Online: Re: TenHarmsel et al., Mortality among Individuals Diagnosed with Chronic Beryllium Disease and Beryllium Sensitization; J Occup Environ Med 2023 (in press).

    Boffetta, Paolo

    Journal of occupational and environmental medicine

    2024  

    Language English
    Publishing date 2024-03-19
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1223932-x
    ISSN 1536-5948 ; 1076-2752
    ISSN (online) 1536-5948
    ISSN 1076-2752
    DOI 10.1097/JOM.0000000000003097
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  4. Book: Occupational cancers

    Anttila, Sisko / Boffetta, Paolo

    2014  

    Author's details Sisko L. Anttila ; Paolo Boffetta
    Keywords Cancer ; Occupational diseases ; Medicine, Industrial
    Subject code 616.994
    Language English
    Size XVI, 609 S. : Ill., graph. Darst., 28 cm
    Publisher Springer
    Publishing place London u.a.
    Publishing country Great Britain
    Document type Book
    HBZ-ID HT018403376
    ISBN 978-1-4471-2824-3 ; 1-4471-2824-9 ; 9781447128250 ; 1447128257
    Database Catalogue ZB MED Medicine, Health

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    Shelf markLoan statusLocation
    2014 A 3681 available for loan (reading room) Lesesaal EG

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  5. Article ; Online: Occupational asbestos exposure and risk of esophageal cancer: A systematic review and meta-analysis.

    Seyyedsalehi, Monireh Sadat / Boffetta, Paolo

    International journal of cancer

    2024  Volume 154, Issue 11, Page(s) 1920–1929

    Abstract: Esophageal cancer (EC), which includes squamous cell carcinoma (ESCC) and adenocarcinoma (EAC), is an important cancer with poor prognosis and high mortality rate. Several occupational exposures have been associated with EC. We aim to investigate the ... ...

    Abstract Esophageal cancer (EC), which includes squamous cell carcinoma (ESCC) and adenocarcinoma (EAC), is an important cancer with poor prognosis and high mortality rate. Several occupational exposures have been associated with EC. We aim to investigate the association between occupational asbestos exposure and EC risk, considering types of asbestos and histology of the disease. We included studies mentioned in the list of references in previous reviews and pooled analyses, and we conducted an independent search in PubMed and Scopus. Forest plots of relative risks (RR) were constructed based on the association between occupational asbestos and EC risk. Random-effects models were used to address heterogeneity between 48 independent cohort and case-control studies. We found an association between occupational asbestos exposure and EC (meta-relative risk [RR] = 1.20, 95% confidence interval [CI] = 1.09-1.32; I2 = 58.8%, p-heterogeneity [het] <.001). The results of stratification by job (p-het = .20) indicate an increased RR among asbestos product workers (RR = 1.39, 95% CI = 1.07-1.81), asbestos applicators (RR = 1.41, 95% CI = 1.20-1.67), and construction workers (RR = 1.12, 95% CI = 1.02-1.24). There was no heterogeneity in meta-RR according to outcome (p = .29), geographic region (p = .69), year of publication (p = .59), quality score (p = .73), asbestos type (p = .93), study design (p = .87), and gender (p = .88), control for potential confounders (p = .20), year of first employment (p = .94) and exposure level (p = .43). The stratification analysis by histology type found an increased RR for both ESCC 1.33(1.03-1.71) and EAC 1.45(1.03-2.04) (p-het = .68). We didn't find evidence of publication bias (p = .07). The results of our study suggest that occupational asbestos exposure is associated with an increased risk of EC in both histology types.
    MeSH term(s) Humans ; Occupational Exposure/adverse effects ; Asbestos/toxicity ; Esophageal Neoplasms/etiology ; Esophageal Neoplasms/chemically induced ; Adenocarcinoma/etiology ; Carcinoma, Squamous Cell/complications ; Occupational Diseases/epidemiology ; Occupational Diseases/etiology
    Chemical Substances Asbestos (1332-21-4)
    Language English
    Publishing date 2024-02-10
    Publishing country United States
    Document type Meta-Analysis ; Systematic Review ; Journal Article
    ZDB-ID 218257-9
    ISSN 1097-0215 ; 0020-7136
    ISSN (online) 1097-0215
    ISSN 0020-7136
    DOI 10.1002/ijc.34881
    Database MEDical Literature Analysis and Retrieval System OnLINE

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    Zs.MO 576: Show issues

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  6. Article ; Online: Re: Exposure to asbestos and the risk of colorectal cancer mortality: a systematic review and meta-analysis by Kwak

    Boffetta, Paolo

    Occupational and environmental medicine

    2020  Volume 77, Issue 9, Page(s) 655

    MeSH term(s) Asbestos/adverse effects ; Colonic Neoplasms ; Colorectal Neoplasms/etiology ; Humans ; Mesothelioma
    Chemical Substances Asbestos (1332-21-4)
    Language English
    Publishing date 2020-07-24
    Publishing country England
    Document type Letter ; Comment
    ZDB-ID 1180733-7
    ISSN 1470-7926 ; 1351-0711
    ISSN (online) 1470-7926
    ISSN 1351-0711
    DOI 10.1136/oemed-2020-106588
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  7. Article: Per- and Poly-fluoroalkyl Substances (PFAS) Exposure and Risk of Kidney, Liver, and Testicular Cancers: A Systematic Review and Meta-Analysis.

    Seyyedsalehi, Monireh Sadat / Boffetta, Paolo

    La Medicina del lavoro

    2023  Volume 114, Issue 5, Page(s) e2023040

    Abstract: Introduction: Per- and poly-fluoroalkyl substances (PFASs) are a large, complex group of synthetic chemicals humans can be exposed to from occupational or environmental sources. In this systematic review and meta-analysis, we examined the association ... ...

    Abstract Introduction: Per- and poly-fluoroalkyl substances (PFASs) are a large, complex group of synthetic chemicals humans can be exposed to from occupational or environmental sources. In this systematic review and meta-analysis, we examined the association between PFAS exposure, particularly Perfluorooctanoic Acid (PFOA), and Perfluorooctane Sulfonic Acid (PFOS), and risk of kidney, liver, and testicular cancer.
    Methods: We systematically searched PubMed to identify cohort and case-control studies reported after the Monograph of the International Agency for Research on Cancer and the Toxicological Profile of the Agency for Toxic Substances and Disease Registry. We assessed the quality of the studies by using a modified version of the Newcastle-Ottawa Scale (NOS). Forest relative risk (RR) plots were constructed for liver, kidney, and testicular cancer. We conducted stratified analyses by geographic region, study design, quality score, outcome, years of publication, exposure source, and PFAS type. A random-effects model was used to address heterogeneity between studies.
    Results: Fifteen studies, including ten cohort studies, three case-control studies nested in a cohort, and two case-control studies were included after removing duplicate and irrelevant reports. We found an association between overall PFAS exposure and the risk of kidney cancers (RR=1.18, 95% CI =1.05-1.32; I =52.8%, 11 studies). Also, we showed an association between high-level exposure to PFAS and kidney cancer (RR=1.74, 95% CI =1.23-2.47; p=0.005) and testicular cancer (RR=2.22, 95% CI =1.12-4.39; p=0.057). There was no association with liver cancer. We found no heterogeneity by geographical region, PFAS type, study design, outcome, quality score, year of publication, or exposure source. Only two studies reported results among women.
    Conclusions: We detected an association between overall PFAS exposure and kidney cancer and high doses of PFAS with testicular cancer. However, bias and confounding cannot be excluded, precluding a conclusion in terms of causality.
    MeSH term(s) Female ; Humans ; Male ; Testicular Neoplasms/chemically induced ; Testicular Neoplasms/epidemiology ; Kidney ; Liver ; Kidney Neoplasms/chemically induced ; Kidney Neoplasms/epidemiology ; Carcinoma, Renal Cell ; Fluorocarbons/adverse effects
    Chemical Substances Fluorocarbons
    Language English
    Publishing date 2023-10-24
    Publishing country Italy
    Document type Meta-Analysis ; Systematic Review ; Journal Article
    ZDB-ID 123678-7
    ISSN 0025-7818
    ISSN 0025-7818
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    Zs.MO 395: Show issues

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  8. Article ; Online: Occupational exposure to diesel exhausts and liver and pancreatic cancers: a systematic review and meta-analysis.

    Sassano, Michele / Collatuzzo, Giulia / Teglia, Federica / Boffetta, Paolo

    European journal of epidemiology

    2024  Volume 39, Issue 3, Page(s) 241–255

    Abstract: Background: Diesel exhaust (DE) is human carcinogen with sufficient evidence only for lung cancer. Systematic evidence on other cancer types is scarce, thus we aimed to systematically review current literature on the association between occupational DE ... ...

    Abstract Background: Diesel exhaust (DE) is human carcinogen with sufficient evidence only for lung cancer. Systematic evidence on other cancer types is scarce, thus we aimed to systematically review current literature on the association between occupational DE exposure and risk of liver and pancreatic cancers.
    Methods: We performed a systematic literature review to identify cohort studies on occupational DE exposure and risk of cancers other than lung. We computed pooled relative risks (RRs) and corresponding 95% confidence intervals (CIs) for liver and pancreatic cancers using DerSimonian and Laird random-effects model.
    Results: Fifteen studies reporting results on pancreatic cancer and fourteen on liver cancer were included. We found a weakly increased risk of pancreatic cancer in workers exposed to DE (RR: 1.07, 95% CI: 1.00, 1.14), mainly driven by results on incidence (RR: 1.11, 95% CI: 1.02, 1.22). As for liver cancer, results were suggestive of a positive association (RR: 1.09; 95% CI: 0.99, 1.19), although a significant estimate was present in studies published before 2000 (RR: 1.41; 95% CI: 1.09, 1.82). We found no compelling evidence of publication bias.
    Conclusions: Our findings suggest an association between occupational DE exposure and liver and pancreatic cancer. Further studies with detailed exposure assessment, environmental monitoring data, and appropriate control for confounders are warranted.
    MeSH term(s) Humans ; Vehicle Emissions/toxicity ; Occupational Exposure/adverse effects ; Pancreatic Neoplasms/chemically induced ; Pancreatic Neoplasms/epidemiology ; Liver Neoplasms/chemically induced ; Liver Neoplasms/epidemiology ; Occupational Diseases/epidemiology
    Chemical Substances Vehicle Emissions
    Language English
    Publishing date 2024-01-30
    Publishing country Netherlands
    Document type Systematic Review ; Meta-Analysis ; Journal Article ; Review
    ZDB-ID 632614-6
    ISSN 1573-7284 ; 0393-2990
    ISSN (online) 1573-7284
    ISSN 0393-2990
    DOI 10.1007/s10654-024-01099-4
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  9. Article ; Online: Systematic review of studies on exposure to arsenic in drinking water and cognitive and neurobehavioral effects.

    Boffetta, Paolo / Sambati, Luisa / Sassano, Michele

    Critical reviews in toxicology

    2024  Volume 54, Issue 3, Page(s) 174–193

    Abstract: An association between exposure to arsenic (As) and neurologic and behavioral effects has been reported in some studies, but no systematic review is available of the evidence linking As in drinking water and neurobehavioral effects after consideration of ...

    Abstract An association between exposure to arsenic (As) and neurologic and behavioral effects has been reported in some studies, but no systematic review is available of the evidence linking As in drinking water and neurobehavioral effects after consideration of study quality and potential confounding, with focus on low-level circumstances of exposure. We conducted a systematic review and reported it in compliance with the Preferred Reporting Items for Systematic reviews and Meta-Analyses (PRISMA) guidelines, through a search of the databases PubMed, Web of Science, Scopus, and Embase. We included in the review the studies reporting results based on exposure from drinking water in humans. Endpoints were heterogeneous across studies, so we classified them into eight broad domains and developed an ad-hoc system to evaluate their methodological quality, based on three tiers. It was not possible to conduct meta-analysis because of the heterogeneity in exposure assessment and in the definition and assessment of outcomes. The search identified 18,518 articles. After elimination of duplicates and irrelevant articles, we retained 106 articles which reported results on As exposure and neurobehavioral effects, of which 22 reported risk estimates from exposure in drinking water (six among adults and 16 among children). None of the studies was conducted blindly. Among the studies in adults, two, which were conducted in highly exposed populations, were classified as high quality. These two studies were broadly consistent in reporting an association between exposure to As and decline in cognitive function; however, they provide no evidence of an association for exposure below 75 μg/L. The four lower-quality studies were based on populations with low exposure; these studies reported associations with inconsistent outcomes, few of which remained statistically significant after adjustment for multiple comparisons. Among the five high-quality studies of children, one reported an association between As in drinking water and intellectual function, whereas none of the other studies reported an association with different neurobehavioral indicators, after adjusting for potential confounders and multiple comparisons. Out of seven intermediate-quality studies, three reported an association with cognitive function or other outcomes; but sources of bias were not adequately controlled. The remaining studies were negative. The four low-quality studies did not contribute to the overall evidence because of methodological limitations. Our assessment of the available literature showed a lack of evidence for a causal association between exposure to As in drinking water and neurobehavioral effects. To clarify whether such an association exists, further studies prospectively evaluating changes in both the concentration of As in drinking water during the life course, and neurobehavioral outcomes, as well as appropriately controlling for potential confounders, are needed.
    MeSH term(s) Child ; Adult ; Humans ; Arsenic/toxicity ; Drinking Water ; Cognition
    Chemical Substances Arsenic (N712M78A8G) ; Drinking Water
    Language English
    Publishing date 2024-03-27
    Publishing country England
    Document type Systematic Review ; Journal Article ; Review
    ZDB-ID 1097071-x
    ISSN 1547-6898 ; 1040-8444
    ISSN (online) 1547-6898
    ISSN 1040-8444
    DOI 10.1080/10408444.2023.2297751
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  10. Article ; Online: Risk of bladder, kidney and prostate cancer from occupational exposure to welding fumes: a systematic review and meta-analysis.

    Collatuzzo, Giulia / Hamdani, Maha / Boffetta, Paolo

    International archives of occupational and environmental health

    2024  Volume 97, Issue 3, Page(s) 221–230

    Abstract: Background: Our aimed to conduct a meta-analysis of cohort studies on risk of genitourinary (GU) cancers in workers exposed to welding fumes (WF).: Methods: We performed a systematic review of studies published on Pubmed, Scopus and Embase following ... ...

    Abstract Background: Our aimed to conduct a meta-analysis of cohort studies on risk of genitourinary (GU) cancers in workers exposed to welding fumes (WF).
    Methods: We performed a systematic review of studies published on Pubmed, Scopus and Embase following PRISMA criteria. Two researchers selected cohort studies on WF exposure. From 2582 articles, 7 non-overlapping studies were included. Quality of studies was scored according to CASP. We run a random effects meta-analysis to calculate the relative risk (RR) and 95% confidence intervals (CI) of GU cancer, overall and stratified by cancer, country, and quality score.
    Results: We included seven studies reporting results on GU cancers, including prostate, bladder and kidney cancer (PC, BC, and KC). The RR was 1.19 (95% CI = 1.07-1.32, 16 risk estimates) for GU cancer; 1.13 (95% CI = 0.90-1.42, 4 risk estimates) for PC; 1.26 (95% CI = 0.98-1.60, 7 risk estimates) for BC and 1.28 (95% CI = 1.12-1.47, 5 risk estimates) for KC. Heterogeneity was present in all meta-analyses (p < 0.001). The increased risk was more pronounced in North American than in European studies (respectively, OR = 1.35, 95% CI = 1.18-1.55; OR = 1.13, 95% CI = 1.01-1.27 p heterogeneity = 0.03). There was no heterogeneity according to quality score (p = 0.4). Data were insufficient to investigate associations by industry or welding type. Publication bias for each cancer was excluded.
    Conclusion: This meta-analysis suggests increased risk of KC and BC, but not of PC, in workers exposed to WF. Confounding by other occupational and non-occupational risk factors could not be excluded. Data were not adequate to address the risk of specific exposure circumstances.
    MeSH term(s) Male ; Humans ; Urinary Bladder ; Welding ; Occupational Exposure/adverse effects ; Prostatic Neoplasms/epidemiology ; Kidney
    Language English
    Publishing date 2024-01-17
    Publishing country Germany
    Document type Meta-Analysis ; Systematic Review ; Journal Article ; Review
    ZDB-ID 129038-1
    ISSN 1432-1246 ; 0340-0131 ; 0367-9977
    ISSN (online) 1432-1246
    ISSN 0340-0131 ; 0367-9977
    DOI 10.1007/s00420-023-02040-0
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