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Article ; Online: Improving Quality of Congenital Heart Disease Research in Canada: Standardizing Nomenclature Across Canada.

Béland, Marie J / Harris, Kevin C / Marelli, Ariane J / Houyel, Lucile / Bailliard, Frédérique / Dallaire, Frédéric

2018 Volume 34, Issue 12, Page(s) 1674–1676

Abstract: In an effort to improve the quality of interinstitutional and nation-wide research into congenital heart disease (CHD) in Canada, the authors propose the national implementation of a single nomenclature list for CHD as a first step towards achieving a ... ...

Abstract	In an effort to improve the quality of interinstitutional and nation-wide research into congenital heart disease (CHD) in Canada, the authors propose the national implementation of a single nomenclature list for CHD as a first step towards achieving a common disease classification system in all Canadian institutions that deal with congenital heart malformations. The authors offer a brief overview of the history and state of nomenclature for CHD in Canada and recommend the national use of the CHD diagnostic list that was recently finalized by the International Society for Nomenclature of Paediatric and Congenital Heart Disease. This list was submitted to the World Health Organization for incorporation into the 11th iteration of the International Classification of Diseases and was recently translated into French by members of the International Society for Nomenclature of Paediatric and Congenital Heart Disease. The bilingual list of the 11th iteration of the International Classification of Diseases CHD terms is published online in this issue of the Canadian Journal of Cardiology. The national standardization of the nomenclature pertaining to CHD using the bilingual list of terms published herein will optimize national efforts to establish longitudinal CHD cohorts, capitalizing on Canada's health care infrastructure and solidifying Canadian leadership in CHD research.
MeSH term(s)	Biomedical Research ; Canada ; Heart Defects, Congenital ; Humans ; International Classification of Diseases ; Societies, Medical ; Terminology as Topic
Language	English
Publishing date	2018-08-29
Publishing country	England
Document type	Journal Article
ZDB-ID	632813-1
ISSN	1916-7075 ; 0828-282X
ISSN (online)	1916-7075
ISSN	0828-282X
DOI	10.1016/j.cjca.2018.08.034
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Article ; Online: Tetralogy of Fallot.

Bailliard, Frederique / Anderson, Robert H

Orphanet journal of rare diseases

2009 Volume 4, Page(s) 2

Abstract: Tetralogy of Fallot is a congenital cardiac malformation that consists of an interventricular communication, also known as a ventricular septal defect, obstruction of the right ventricular outflow tract, override of the ventricular septum by the aortic ... ...

Abstract	Tetralogy of Fallot is a congenital cardiac malformation that consists of an interventricular communication, also known as a ventricular septal defect, obstruction of the right ventricular outflow tract, override of the ventricular septum by the aortic root, and right ventricular hypertrophy. This combination of lesions occurs in 3 of every 10,000 live births, and accounts for 7-10% of all congenital cardiac malformations. Patients nowadays usually present as neonates, with cyanosis of varying intensity based on the degree of obstruction to flow of blood to the lungs. The aetiology is multifactorial, but reported associations include untreated maternal diabetes, phenylketonuria, and intake of retinoic acid. Associated chromosomal anomalies can include trisomies 21, 18, and 13, but recent experience points to the much more frequent association of microdeletions of chromosome 22. The risk of recurrence in families is 3%. Useful diagnostic tests are the chest radiograph, electrocardiogram, and echocardiogram. The echocardiogram establishes the definitive diagnosis, and usually provides sufficient information for planning of treatment, which is surgical. Approximately half of patients are now diagnosed antenatally. Differential diagnosis includes primary pulmonary causes of cyanosis, along with other cyanotic heart lesions, such as critical pulmonary stenosis and transposed arterial trunks. Neonates who present with ductal-dependent flow to the lungs will receive prostaglandins to maintain ductal patency until surgical intervention is performed. Initial intervention may be palliative, such as surgical creation of a systemic-to-pulmonary arterial shunt, but the trend in centres of excellence is increasingly towards neonatal complete repair. Centres that undertake neonatal palliation will perform the complete repair at the age of 4 to 6 months. Follow-up in patients born 30 years ago shows a rate of survival greater than 85%. Chronic issues that now face such adults include pulmonary regurgitation, recurrence of pulmonary stenosis, and ventricular arrhythmias. As the strategies for surgical and medical management have progressed, the morbidity and mortality of those born with tetralogy of Fallot in the current era is expected to be significantly improved.
MeSH term(s)	Adult ; Genetic Counseling ; Heart Defects, Congenital/diagnostic imaging ; Heart Defects, Congenital/genetics ; Heart Defects, Congenital/mortality ; Heart Defects, Congenital/surgery ; Humans ; Infant ; Infant, Newborn ; Prognosis ; Tetralogy of Fallot/diagnostic imaging ; Tetralogy of Fallot/genetics ; Tetralogy of Fallot/mortality ; Tetralogy of Fallot/surgery ; Ultrasonography
Language	English
Publishing date	2009-01-13
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	2225857-7
ISSN	1750-1172 ; 1750-1172
ISSN (online)	1750-1172
ISSN	1750-1172
DOI	10.1186/1750-1172-4-2
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Article ; Online: Tetralogy of Fallot

Anderson Robert H / Bailliard Frederique

Orphanet Journal of Rare Diseases, Vol 4, Iss 1, p

2009 Volume 2

Abstract: Abstract Tetralogy of Fallot is a congenital cardiac malformation that consists of an interventricular communication, also known as a ventricular septal defect, obstruction of the right ventricular outflow tract, override of the ventricular septum by the ...

Abstract	Abstract Tetralogy of Fallot is a congenital cardiac malformation that consists of an interventricular communication, also known as a ventricular septal defect, obstruction of the right ventricular outflow tract, override of the ventricular septum by the aortic root, and right ventricular hypertrophy. This combination of lesions occurs in 3 of every 10,000 live births, and accounts for 7–10% of all congenital cardiac malformations. Patients nowadays usually present as neonates, with cyanosis of varying intensity based on the degree of obstruction to flow of blood to the lungs. The aetiology is multifactorial, but reported associations include untreated maternal diabetes, phenylketonuria, and intake of retinoic acid. Associated chromosomal anomalies can include trisomies 21, 18, and 13, but recent experience points to the much more frequent association of microdeletions of chromosome 22. The risk of recurrence in families is 3%. Useful diagnostic tests are the chest radiograph, electrocardiogram, and echocardiogram. The echocardiogram establishes the definitive diagnosis, and usually provides sufficient information for planning of treatment, which is surgical. Approximately half of patients are now diagnosed antenatally. Differential diagnosis includes primary pulmonary causes of cyanosis, along with other cyanotic heart lesions, such as critical pulmonary stenosis and transposed arterial trunks. Neonates who present with ductal-dependent flow to the lungs will receive prostaglandins to maintain ductal patency until surgical intervention is performed. Initial intervention may be palliative, such as surgical creation of a systemic-to-pulmonary arterial shunt, but the trend in centres of excellence is increasingly towards neonatal complete repair. Centres that undertake neonatal palliation will perform the complete repair at the age of 4 to 6 months. Follow-up in patients born 30 years ago shows a rate of survival greater than 85%. Chronic issues that now face such adults include pulmonary regurgitation, recurrence ...
Keywords	Medicine ; R
Subject code	610
Language	English
Publishing date	2009-01-01T00:00:00Z
Publisher	BMC
Document type	Article ; Online
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Article ; Online: The problems that exist when considering the anatomic variability between the channels that permit interventricular shunting.

Bailliard, Frédérique / Spicer, Diane E / Mohun, Timothy J / Henry, G William / Anderson, Robert H

Cardiology in the young

2014 Volume 25, Issue 1, Page(s) 15–28

Abstract: Although steps are being taken to produce a universally acceptable coding system for categorisation of the congenitally malformed hearts, obstacles remain in the search for consensus. One of the groups of lesions continuing to produce the greatest ... ...

Abstract	Although steps are being taken to produce a universally acceptable coding system for categorisation of the congenitally malformed hearts, obstacles remain in the search for consensus. One of the groups of lesions continuing to produce the greatest problems is those that permit interventricular shunting. The difficulties relate partly to the words used to describe the group itself, as those using Germanic languages describe the holes as ventricular septal defects, whereas those using Romance languages consider them to represent interventricular communications. The two terms, however, are not necessarily synonymous. Further disagreements relate to whether the lesions placed within the group should be sub-categorised on the basis of their geographical location within the ventricular mass, as opposed to the anatomic nature of their borders. In reality, attention to both the features is necessary if we are to recognise the full extent of phenotypic variability. In this review, we first review the evolution and theories of analysis naming the channels that permit interventricular shunting. We then demonstrate that embryologic techniques provide evidence that the changing morphology of the developing murine heart parallels the anatomy of the different lesions encountered in the congenitally malformed human heart. We suggest that, with attention paid to the temporal development of the normal murine heart, combined with a strict definition of the plane of separation between the right and left ventricular cavities, it will be feasible to produce a categorisation that is acceptable to all.
MeSH term(s)	Cardiac Surgical Procedures ; Endocardial Cushion Defects/pathology ; Endocardial Cushion Defects/surgery ; Heart Septal Defects, Ventricular/pathology ; Heart Septal Defects, Ventricular/surgery ; Heart Septum/pathology ; Heart Septum/surgery ; Heart Ventricles/pathology ; Heart Ventricles/surgery ; Humans
Language	English
Publishing date	2014-05-27
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	1078466-4
ISSN	1467-1107 ; 1047-9511
ISSN (online)	1467-1107
ISSN	1047-9511
DOI	10.1017/S1047951114000869
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Article: Introduction to cardiac imaging in infants and children: techniques, potential, and role in the imaging work-up of various cardiac malformations and other pediatric heart conditions.

Bailliard, Frederique / Hughes, Marina L / Taylor, Andrew M

European journal of radiology

2008 Volume 68, Issue 2, Page(s) 191–198

Abstract: The increasing prevalence of congenital heart disease (CHD) can be attributed to major improvements in diagnosis and treatment. Although echocardiography is the most commonly used imaging modality for diagnosis and follow-up of subjects with CHD, the ... ...

Abstract	The increasing prevalence of congenital heart disease (CHD) can be attributed to major improvements in diagnosis and treatment. Although echocardiography is the most commonly used imaging modality for diagnosis and follow-up of subjects with CHD, the evolution of cardiovascular magnetic resonance (MR) imaging and increasingly computed tomography (CT) does offer new ways to visualize the heart and the great vessels. The development of cardiovascular MR techniques allows for a comprehensive assessment of cardiac anatomy and function. This provides information about the long-term sequlae of the underlying complex anatomy, hemodynamic assessment of residual post-operative lesions and complications of surgery. As much of the functional data in CHD patients is usually acquired with invasive X-ray angiography, non-invasive alternatives such as cardiovascular MR (and CT) are desirable. This review evaluates the role of MR imaging in the management of subjects with CHD, particularly detailing recent developments in imaging techniques as they relate to the various CHD diagnoses we commonly encounter in our practice.
MeSH term(s)	Child ; Child, Preschool ; Diagnostic Imaging ; Heart Defects, Congenital/diagnosis ; Heart Defects, Congenital/surgery ; Humans ; Infant ; Infant, Newborn ; Postoperative Complications/diagnosis
Language	English
Publishing date	2008-11
Publishing country	Ireland
Document type	Journal Article ; Review
ZDB-ID	138815-0
ISSN	1872-7727 ; 0720-048X
ISSN (online)	1872-7727
ISSN	0720-048X
DOI	10.1016/j.ejrad.2008.05.016
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Article ; Online: Worsening bradycardia following antithymocyte globulin treatment of severe aplastic anemia.

Godown, Justin / Deal, Allison M / Riley, Kathy / Bailliard, Frederique / Blatt, Julie

The journal of pediatric pharmacology and therapeutics : JPPT : the official journal of PPAG

2012 Volume 16, Issue 3, Page(s) 218–221

Abstract: Immunosuppressive regimens, which include antithymocyte globulin (ATG), are widely used for the treatment of severe aplastic anemia (SAA). However, bradycardia has been reported only as a rare side effect of ATG therapy in the manufacturer's product ... ...

Abstract	Immunosuppressive regimens, which include antithymocyte globulin (ATG), are widely used for the treatment of severe aplastic anemia (SAA). However, bradycardia has been reported only as a rare side effect of ATG therapy in the manufacturer's product information and, in rare cases, in the adult literature. We present an adolescent with SAA and preexisting bradycardia who underwent immunosuppression therapy with ATG, methylprednisolone, and tacrolimus and developed profound sinus bradycardia with successive doses of ATG.
Language	English
Publishing date	2012-03-27
Publishing country	United States
Document type	Case Reports
ISSN	2331-348X
ISSN (online)	2331-348X
DOI	10.5863/1551-6776-16.3.218
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Article ; Online: Characterisation of the long-term effects of anthracycline-associated myocardial toxicity using cardiac MRI; a pilot study

Taylor Andrew / Bailliard Frederique / Hughes Marina L / Levitt Gill

Journal of Cardiovascular Magnetic Resonance, Vol 11, Iss Suppl 1, p P

2009 Volume 95

Keywords	Diseases of the circulatory (Cardiovascular) system ; RC666-701 ; Specialties of internal medicine ; RC581-951 ; Internal medicine ; RC31-1245 ; Medicine ; R ; DOAJ:Cardiovascular ; DOAJ:Medicine (General) ; DOAJ:Health Sciences
Language	English
Publishing date	2009-01-01T00:00:00Z
Publisher	BioMed Central
Document type	Article ; Online
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Article ; Online: Evaluation of coronary artery disease in congenital heart disease and pediatrics utilizing adenosine stress perfusion

Bailliard Frederique / Barker Piers CA / Campbell Michael J / Kim Ray J

Journal of Cardiovascular Magnetic Resonance, Vol 13, Iss Suppl 1, p P

2011 Volume 200

Keywords	Diseases of the circulatory (Cardiovascular) system ; RC666-701 ; Specialties of internal medicine ; RC581-951 ; Internal medicine ; RC31-1245 ; Medicine ; R ; DOAJ:Cardiovascular ; DOAJ:Medicine (General) ; DOAJ:Health Sciences
Language	English
Publishing date	2011-02-01T00:00:00Z
Publisher	BioMed Central
Document type	Article ; Online
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Article ; Online: Evolving surgical strategy for sinus venosus atrial septal defect: effect on sinus node function and late venous obstruction.

Stewart, Robert D / Bailliard, Frédérique / Kelle, Angela M / Backer, Carl L / Young, Luciana / Mavroudis, Constantine

The Annals of thoracic surgery

2007 Volume 84, Issue 5, Page(s) 1651–5; discussion 1655

Abstract: Background: Our surgical strategy for repair of sinus venosus atrial septal defect has evolved chiefly to avoid sinus node dysfunction. We reviewed our experience with the single-patch, two-patch, and Warden repairs.: Methods: We identified 54 ... ...

Abstract	Background: Our surgical strategy for repair of sinus venosus atrial septal defect has evolved chiefly to avoid sinus node dysfunction. We reviewed our experience with the single-patch, two-patch, and Warden repairs. Methods: We identified 54 patients with repair of sinus venosus atrial septal defect from 1990 to 2006. Mean age was 9.5 +/- 12.6 years; median age was 4.2 years. Partial anomalous pulmonary venous connection was found in 52 patients (96%); drainage was to the right atrium in 8, right atrial-superior vena cava (SVC) junction in 17, and directly to the SVC in 27. Techniques were single-patch repair (24), two-patch repair (25), and Warden repair (5). Autologous pericardium was used in all patients. Echocardiogram and electrocardiogram follow-up were available for 48 patients (89%). Results: There were no early or late deaths and no reoperations. No patient had pulmonary vein stenosis. Five patients had SVC stenosis: 2 mild after two-patch repair; 1 moderate and 1 mild after single-patch repair; and 1 severe stenosis after Warden procedure (p = 0.3). The incidence of rhythm change from sinus to low atrial or junctional rhythm was 35% and was significantly greater among patients with two-patch repair (12 of 22, 55%) compared with single-patch repair (5 of 21, 24%), or the Warden repair (0 of 5, p = 0.02). Conclusions: Repair of sinus venosus atrial septal defect with autologous pericardium is associated with a low incidence of late SVC or pulmonary vein stenosis with all techniques. Use of the two-patch technique, however, is associated with a significantly greater incidence of sinus node dysfunction. For the patients with partial anomalous pulmonary venous connection entering the SVC, the Warden procedure avoids interfering with the sinus node and should be used preferentially. The single-patch technique remains the procedure of choice for sinus venosus atrial septal defect with partial anomalous pulmonary venous connection entering the right atrium or right atrium-SVC junction.
MeSH term(s)	Adolescent ; Adult ; Cardiac Surgical Procedures/adverse effects ; Cardiac Surgical Procedures/methods ; Child ; Child, Preschool ; Female ; Heart Septal Defects, Atrial/surgery ; Humans ; Infant ; Male ; Middle Aged ; Postoperative Complications/etiology ; Pulmonary Veins/abnormalities ; Pulmonary Veins/surgery ; Pulmonary Veno-Occlusive Disease/etiology ; Sinoatrial Node/physiopathology ; Vena Cava, Superior/abnormalities ; Vena Cava, Superior/surgery
Language	English
Publishing date	2007-11
Publishing country	Netherlands
Document type	Journal Article
ZDB-ID	211007-6
ISSN	1552-6259 ; 0003-4975
ISSN (online)	1552-6259
ISSN	0003-4975
DOI	10.1016/j.athoracsur.2007.04.130
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Zs.A 252: Show issues

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Article ; Online: Complete atrioventricular canal: comparison of modified single-patch technique with two-patch technique.

Backer, Carl L / Stewart, Robert D / Bailliard, Frédérique / Kelle, Angela M / Webb, Catherine L / Mavroudis, Constantine

The Annals of thoracic surgery

2007 Volume 84, Issue 6, Page(s) 2038–46; discussion 2038–46

Abstract: Background: The purpose of this study was to compare the modified single-patch technique to the two-patch technique for infants with complete atrioventricular canal (CAVC) defects.: Methods: Between January 2000 and June 2006, 55 infants underwent ... ...

Abstract	Background: The purpose of this study was to compare the modified single-patch technique to the two-patch technique for infants with complete atrioventricular canal (CAVC) defects. Methods: Between January 2000 and June 2006, 55 infants underwent CAVC repair. Twenty-six patients had a modified single-patch technique; 29 patients had a two-patch technique. Trisomy 21 was present in 23 of 26 and 26 of 29 patients (p = not significant [ns]). Mean age was 4.4 +/- 1.3 months (single-patch) versus 5.5 +/- 1.9 months (two-patch, p < 0.02). Mean weight was 4.74 +/- 0.92 versus 5.28 +/- 1.67 kilograms (p = ns). Results: There was one death in the modified single-patch group (postoperative day 130, liver failure) and no deaths in the two-patch group. Cross-clamp times and cardiopulmonary bypass times were shorter in the modified single-patch group (97.3 +/- 19.9 vs 123.3 +/- 28.2 minutes, p < 0.0003; 128 +/- 25 vs 157 +/- 37, p < 0.03). Rastelli classification was type A (18 vs 14), B (1 vs 0), and C (7 vs 15). Mean size of the ventricular septal defect as assessed by transesophageal echocardiogram was 9 +/- 2 mm, (single-patch) versus 10 +/- 3 mm (two-patch) (p = ns). Median postoperative length of stay did not differ (10 vs 8 days). There was no difference in the degree of postoperative left or right AV valve insufficiency as assessed by serial echocardiography. One patient (4%) required reoperation for mitral insufficiency in the modified single-patch versus three patients in the two-patch group (10%, p = ns). There were no patients with third degree atrioventricular block or that required reoperation for residual VSD in the modified single-patch group. There was one patient with third-degree AV block that required a pacemaker and one patient who had reoperation for a residual ventricular septal defect in the two-patch group (p = ns). No patient in either group required reoperation for left ventricular outflow tract obstruction. Conclusions: The modified single-patch technique produced results comparable with the two-patch technique in younger patients with similarly sized ventricular septal defects. Furthermore, the modified single-patch technique was performed with significantly shorter cross-clamp and cardiopulmonary bypass times.
MeSH term(s)	Cardiac Surgical Procedures/methods ; Echocardiography, Transesophageal ; Endocardial Cushion Defects/surgery ; Heart Septal Defects, Ventricular/surgery ; Humans ; Infant ; Length of Stay ; Reoperation ; Ventricular Outflow Obstruction/surgery
Language	English
Publishing date	2007-12
Publishing country	Netherlands
Document type	Comparative Study ; Journal Article
ZDB-ID	211007-6
ISSN	1552-6259 ; 0003-4975
ISSN (online)	1552-6259
ISSN	0003-4975
DOI	10.1016/j.athoracsur.2007.04.129
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