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Book ; Online ; E-Book: Urban ecology and global climate change

Bhadouria, Rahul / Upadhyay, Shweta / Tripathi, Sachchidanand / Singh, Pardeep

2022

Author's details	edited by Rahul Bhadouria, University of Delhi, Shweta Upadhyay, Banaras Hindu University, Sachchidanand Tripathi, University of Delhi, Pardeep Singh, PGDAV College, University of Delhi
Keywords	Urban ecology (Biology) / Environmental aspects ; Urban ecology (Sociology) / Environmental aspects ; Urbanization ; Overpopulation ; Climatic changes
Language	English
Size	1 Online-Ressource (xvi, 352 Seiten), Illustrationen
Publisher	Wiley
Publishing place	Hoboken, NJ
Publishing country	United States
Document type	Book ; Online ; E-Book
Note	Includes bibliographical references and index
Remark	Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
HBZ-ID	HT021314426
ISBN	978-1-11-980719-3 ; 9781119807186 ; 1-11-980719-0 ; 1119807182
Database	ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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Article ; Online: Corrigendum to "Biofunctionalized chrysin-conjugated gold nanoparticles neutralize Leishmania parasites with high efficacy" [Int. J. Biol. Macromol. Volume 205, 30 April 2022, Pages 211-219].

Raj, Shweta / Sasidharan, Santanu / Tripathi, Timir / Saudagar, Prakash

International journal of biological macromolecules

2024 Volume 264, Issue Pt 2, Page(s) 130924

Language	English
Publishing date	2024-03-19
Publishing country	Netherlands
Document type	Published Erratum
ZDB-ID	282732-3
ISSN	1879-0003 ; 0141-8130
ISSN (online)	1879-0003
ISSN	0141-8130
DOI	10.1016/j.ijbiomac.2024.130924
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Article ; Online: Antioxidant and antiarthritic potential of berberine:

Jain, Shweta / Tripathi, Shalini / Tripathi, Pushpendra Kumar

Chinese herbal medicines

2023 Volume 15, Issue 4, Page(s) 549–555

Abstract: Objective: To extract and isolate berberine from : Methods: The berberine was isolated from : Results: The antioxidant activity of berberine revealed potent antioxidant activity in DPPH, nitric oxide, and superoxide scavenging assays. The : ... ...

Abstract	Objective: To extract and isolate berberine from Methods: The berberine was isolated from Results: The antioxidant activity of berberine revealed potent antioxidant activity in DPPH, nitric oxide, and superoxide scavenging assays. The Conclusion: The ameliorating action on haematological parameters and proinflammatory biomarkers of berberine makes them a suitable remedy for the treatment of arthritis.
Language	English
Publishing date	2023-09-12
Publishing country	Singapore
Document type	Journal Article
ISSN	2589-3610
ISSN (online)	2589-3610
DOI	10.1016/j.chmed.2023.02.007
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Article ; Online: Adropin may enhance life span and function of corpus luteum by improving antioxidant potential in adult mouse.

Maurya, Shweta / Tripathi, Shashank / Arora, Taruna / Singh, Ajit

The Journal of steroid biochemistry and molecular biology

2024 , Page(s) 106524

Abstract: The corpus luteum (CL) is a temporary endocrine gland that synthesizes progesterone. The luteal progesterone plays a central role in the regulation of the estrous cycle as well as the implantation and maintenance of pregnancy. Our previous study showed ... ...

Abstract	The corpus luteum (CL) is a temporary endocrine gland that synthesizes progesterone. The luteal progesterone plays a central role in the regulation of the estrous cycle as well as the implantation and maintenance of pregnancy. Our previous study showed the expression of adropin and its receptor, GPR19, in the luteal cells and its significant role in luteinization. The aim of the present study was to investigate the in vitro effect of adropin on hCG-induced ovarian functions in adult mice. We also evaluated the effect of exogenous treatment with adropin on ovarian steroidogenesis and anti-oxidant parameters, with special emphasis on CL function. Our results demonstrated that adropin acts synergistically with hCG to promote ovarian steroidogenesis and survival by increasing the expression of StAR, 3β-HSD, and aromatase proteins and decreasing the BAX/BCL2 ratio. Exogenous adropin treatment increased progesterone production by increasing the expression of StAR and 3β-HSD enzymes in the mouse ovary. Also, adropin, via GPR19, inhibited the luteal oxidative stress by increasing nuclear translocation of NRF-2 in CL, which resulted in increased HO-1 expression and SOD, catalase activity. Decreased oxidative stress might inhibit the translocation of NF-κB into the nucleus of luteal cells, resulting into increased survival and decreased apoptosis, as evident by decreased lipid peroxidation, BAX/BCL2 ratio, caspase 3, active caspase 3 expression, and TUNEL-positive cells in adropin treated mice. Our findings suggest that adropin can be a promising candidate that could increase the lifespan of CL and might serve as a potential therapeutic option for treating luteal insufficiency.
Language	English
Publishing date	2024-04-24
Publishing country	England
Document type	Journal Article
ZDB-ID	1049188-0
ISSN	1879-1220 ; 0960-0760
ISSN (online)	1879-1220
ISSN	0960-0760
DOI	10.1016/j.jsbmb.2024.106524
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Article ; Online: REM Sleep Deprivation Alters Learning-Induced Cell Proliferation and Generation of Newborn Young Neurons in the Dentate Gyrus of the Dorsal Hippocampus.

Tripathi, Shweta / Jha, Sushil K

ACS chemical neuroscience

2022 Volume 13, Issue 2, Page(s) 194–206

Abstract: The hippocampus-dependent "trace-appetitive conditioning task" increases cell proliferation and the generation of newborn young neurons. Evidence suggests that adult hippocampal neurogenesis and rapid eye movement (REM) sleep play an essential role in ... ...

Abstract	The hippocampus-dependent "trace-appetitive conditioning task" increases cell proliferation and the generation of newborn young neurons. Evidence suggests that adult hippocampal neurogenesis and rapid eye movement (REM) sleep play an essential role in memory consolidation. On the other hand, REM sleep deprivation (REM-SD) induces detrimental effects on training-induced cell proliferation in the hippocampus's dentate gyrus (DG). Nonetheless, the role of REM sleep in the trace-appetitive memory and fate determination of the newly proliferated cells is not known. Here, we have studied the following: (I) the effects of 24 h of REM-SD (soon after training) on trace- and delay-appetitive memory and cell proliferation in the adult DG and (II) the effects of chronic (96 h) REM-SD (3 days after the training, the period in which newly generated cells progressed toward the neuronal lineage) on trace-appetitive memory and the generation of newborn young neurons. We used a modified multiple platform method for the selective REM-SD without altering non-REM (NREM) sleep. We found that 24 h of REM-SD, soon after trace-conditioning, impaired the trace-appetitive memory and the training-induced cell proliferation. Nevertheless, 96 h of REM-SD (3 days after the training) did not impair trace memory. Interestingly, 96 h of REM-SD altered the generation of newborn young neurons. These results suggest that REM sleep plays an essential role in training-induced cell proliferation and the fate determination of the newly generated cells toward the neuronal lineage.
MeSH term(s)	Cell Proliferation ; Dentate Gyrus ; Hippocampus ; Humans ; Neurons ; Sleep Deprivation
Language	English
Publishing date	2022-01-06
Publishing country	United States
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ISSN	1948-7193
ISSN (online)	1948-7193
DOI	10.1021/acschemneuro.1c00465
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Article ; Online: Molecular pathway of pancreatic cancer-associated neuropathic pain.

Giri, Shweta Subhash / Tripathi, Alok Shiomurti / Erkekoğlu, Pınar / Zaki, Magdi E A

Journal of biochemical and molecular toxicology

2024 Volume 38, Issue 4, Page(s) e23638

Abstract: The pancreas is a heterocrine gland that has both exocrine and endocrine parts. Most pancreatic cancer begins in the cells that line the ducts of the pancreas and is called pancreatic ductal adenocarcinoma (PDAC). PDAC is the most encountered pancreatic ... ...

Abstract	The pancreas is a heterocrine gland that has both exocrine and endocrine parts. Most pancreatic cancer begins in the cells that line the ducts of the pancreas and is called pancreatic ductal adenocarcinoma (PDAC). PDAC is the most encountered pancreatic cancer type. One of the most important characteristic features of PDAC is neuropathy which is primarily due to perineural invasion (PNI). PNI develops tumor microenvironment which includes overexpression of fibroblasts cells, macrophages, as well as angiogenesis which can be responsible for neuropathy pain. In tumor microenvironment inactive fibroblasts are converted into an active form that is cancer-associated fibroblasts (CAFs). Neurotrophins they also increase the level of Substance P, calcitonin gene-related peptide which is also involved in pain. Matrix metalloproteases are the zinc-associated proteases enzymes which activates proinflammatory interleukin-1β into its activated form and are responsible for release and activation of Substance P which is responsible for neuropathic pain by transmitting pain signal via dorsal root ganglion. All the molecules and their role in being responsible for neuropathic pain are described below.
MeSH term(s)	Humans ; Substance P ; Neuralgia/etiology ; Pancreas ; Pancreatic Neoplasms/complications ; Fibroblasts ; Tumor Microenvironment
Chemical Substances	Substance P (33507-63-0)
Language	English
Publishing date	2024-04-13
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	1410020-4
ISSN	1099-0461 ; 1095-6670
ISSN (online)	1099-0461
ISSN	1095-6670
DOI	10.1002/jbt.23638
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Article ; Online: Adropin may promote insulin stimulated steroidogenesis and spermatogenesis in adult mice testes.

Tripathi, Shashank / Maurya, Shweta / Singh, Ajit

Journal of experimental zoology. Part A, Ecological and integrative physiology

2023 Volume 341, Issue 1, Page(s) 86–98

Abstract: Adropin is a versatile peptide which was discovered as a novel metabolic hormone that is involved in the regulation of lipid and glucose homeostasis. However, its possible role in the testicular function is not yet understood. The aim of our study was to ...

Abstract	Adropin is a versatile peptide which was discovered as a novel metabolic hormone that is involved in the regulation of lipid and glucose homeostasis. However, its possible role in the testicular function is not yet understood. The aim of our study was to explore the distribution pattern of adropin and GPR19 in various cell types and its possible role in testicular functions of adult mice. Immunohistochemical study revealed the intense immunoreactivity of adropin in the Leydig cells, while GPR19 showed intense immunoreactivity in the pachytene spermatocytes and mild immunoreactivity in Leydig cells and primary as well as secondary spermatocytes in mouse testis. Enho mRNA was also found to be expressed in the mouse testis. These findings suggested that adropin-GPR19 signaling may act in autocrine/paracrine manner to modulate testicular functions. Furthermore, to find out the direct role of adropin in the testicular function, in vitro study was performed in which testicular slices were cultured with adropin alone (10 and 100 ng/mL) and in combination with insulin (5 μg/mL). Adropin alone inhibited testicular testosterone synthesis by inhibiting the expression of P450-SCC, 3β-HSD, and 17β-HSD while along with insulin stimulated the testicular testosterone synthesis by increasing the expression of GPR19, IR, StAR, P450-SCC, 3β-HSD, and 17β-HSD. Adropin alone or in combination with insulin promoted germ cell survival and proliferation by upregulating the expression of PCNA, Bcl2, and pERK1/2. Thus, it can be concluded that adropin-GPR19 signaling promotes insulin stimulated steroidogenesis and germ cell survival as well as proliferation in the mice testes in an autocrine/paracrine manner.
MeSH term(s)	Animals ; Male ; Mice ; Insulin/metabolism ; Leydig Cells/metabolism ; Spermatogenesis/physiology ; Testis/metabolism ; Testosterone
Chemical Substances	Insulin ; Testosterone (3XMK78S47O) ; Enho protein, mouse
Language	English
Publishing date	2023-10-30
Publishing country	United States
Document type	Journal Article
ZDB-ID	1474896-4
ISSN	2471-5646 ; 1932-5223 ; 2471-5646 ; 1932-5231 ; 1552-499X
ISSN (online)	2471-5646 ; 1932-5223
ISSN	2471-5646 ; 1932-5231 ; 1552-499X
DOI	10.1002/jez.2763
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Article ; Online: Adropin may regulate corpus luteum formation and its function in adult mouse ovary.

Maurya, Shweta / Tripathi, Shashank / Arora, Taruna / Singh, Ajit

Hormones (Athens, Greece)

2023 Volume 22, Issue 4, Page(s) 725–739

Abstract: Background: Adropin, a unique peptide hormone, has been associated with the regulation of several physiological processes, including glucose homeostasis, fatty acid metabolism, and neovascularization. However, its possible role in ovarian function is ... ...

Abstract	Background: Adropin, a unique peptide hormone, has been associated with the regulation of several physiological processes, including glucose homeostasis, fatty acid metabolism, and neovascularization. However, its possible role in ovarian function is not understood. Our objective was to examine the expression of adropin and its putative receptor, GPR19, in the ovaries of mice at various phases of the estrous cycle. Methods: Immunohistochemistry and western blot analysis were performed to explore the localization and changes in expression of adropin and GPR19 in the ovaries during different phases of the estrous cycle in mice. Hormonal assays were performed with ELISA. An in vitro study was performed to examine the direct effect of adropin (10, 100 ng/ml) on ovarian function. Results: A western blot study showed that adropin and GPR19 proteins were maximum during the estrus phase of the estrous cycle. Interestingly, adropin and GPR19 displayed intense immunoreactivity in granulosa cells of large antral follicles and corpus luteum. This suggested the possible involvement of adropin in corpus luteum formation. Adropin treatment stimulated progesterone synthesis by increasing GPR19, StAR, CYP11A1, and 3β-HSD expressions, while it decreased estrogen synthesis by inhibiting 17β-HSD and aromatase protein expressions. Moreover, adropin treatment upregulated the cell cycle arrest-CDK inhibitor 1B (p27 Conclusions: Adropin GPR19 signaling promotes the synthesis of progesterone and upregulates the expression of p27
MeSH term(s)	Female ; Mice ; Animals ; Ovary/metabolism ; Progesterone/pharmacology ; Vascular Endothelial Growth Factor, Endocrine-Gland-Derived/metabolism ; Vascular Endothelial Growth Factor, Endocrine-Gland-Derived/pharmacology ; Corpus Luteum/metabolism ; Estrous Cycle
Chemical Substances	Progesterone (4G7DS2Q64Y) ; Vascular Endothelial Growth Factor, Endocrine-Gland-Derived
Language	English
Publishing date	2023-08-19
Publishing country	Switzerland
Document type	Journal Article
ZDB-ID	2075912-5
ISSN	2520-8721 ; 1109-3099
ISSN (online)	2520-8721
ISSN	1109-3099
DOI	10.1007/s42000-023-00476-0
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Article ; Online: Adropin, a novel hepatokine: localization and expression during postnatal development and its impact on testicular functions of pre-pubertal mice.

Tripathi, Shashank / Maurya, Shweta / Singh, Ajit

Cell and tissue research

2023 Volume 395, Issue 2, Page(s) 171–187

Abstract: Adropin, a multifaceted peptide, was identified as a new metabolic hormone responsible for regulating gluco-lipid homeostasis. However, its role in the testicular function is not yet understood. We aimed to investigate the localization and expression of ... ...

Abstract	Adropin, a multifaceted peptide, was identified as a new metabolic hormone responsible for regulating gluco-lipid homeostasis. However, its role in the testicular function is not yet understood. We aimed to investigate the localization and expression of adropin and GPR19 during different phases of postnatal development. Immunohistochemical study revealed the intense reactivity of adropin in the Leydig cells during all phases of postnatal development, while GPR19 showed intense immunoreactivity in the pachytene spermatocytes and mild immunoreactivity in Leydig cells as well as primary and secondary spermatocytes. Western blot study revealed maximum expression of GPR19 in pre-pubertal mouse testis that clearly indicates maximum responsiveness of adropin during that period. So, we hypothesized that adropin may act as an autocrine/paracrine factor that regulates pubertal changes in mouse testis. To examine the effect of adropin on pubertal onset, we gave bilateral intra-testicular doses (0.5 and 1.5 µg/testis) to pre-pubertal mice. Adropin treatment promoted testicular testosterone synthesis by increasing the expression of StAR, 3β-HSD, and 17β-HSD. Adropin also promoted germ cell survival and proliferation by upregulating the expression of PCNA and downregulating the Bax/Bcl2 ratio and Caspase 3 expression resulting in fewer TUNEL-positive cells in adropin-treated groups. FACS analysis demonstrated that adropin treatment not only increases 1C to 4C ratio but also significantly increases the 1C (spermatid) and 1C to 2C ratio which demarcates accelerated germ cell differentiation and turnover of testicular cells. In conclusion, adropin promotes steroidogenesis, germ cell survival, as well as the proliferation in the pre-pubertal mouse testis that may hasten the pubertal transition in an autocrine/paracrine manner.
MeSH term(s)	Male ; Mice ; Animals ; Testis ; Leydig Cells/metabolism ; Spermatids/metabolism ; Cell Differentiation ; Testosterone/metabolism
Chemical Substances	Testosterone (3XMK78S47O)
Language	English
Publishing date	2023-12-13
Publishing country	Germany
Document type	Journal Article
ZDB-ID	125067-x
ISSN	1432-0878 ; 0302-766X
ISSN (online)	1432-0878
ISSN	0302-766X
DOI	10.1007/s00441-023-03852-9
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Article ; Online: Ontogeny of adropin and its receptor expression during postnatal development and its pro-gonadal role in the ovary of pre-pubertal mouse.

Maurya, Shweta / Tripathi, Shashank / Singh, Ajit

The Journal of steroid biochemistry and molecular biology

2023 Volume 234, Page(s) 106404

Abstract: Adropin, a highly conserved multifunctional peptide hormone, has a beneficial effect on the maintenance of gluco-lipid homeostasis, endothelial and cardiovascular functions. However, the expression and potential role of adropin in ovarian function are ... ...

Abstract	Adropin, a highly conserved multifunctional peptide hormone, has a beneficial effect on the maintenance of gluco-lipid homeostasis, endothelial and cardiovascular functions. However, the expression and potential role of adropin in ovarian function are not fully elucidated. The present study aimed to investigate the expression of adropin and GPR19 in the mice ovary during various stages of postnatal development. This study also explored whether the treatment of adropin can modulate the timing of puberty, for which pre-pubertal mice were treated with adropin. The result showed the intense immunoreactivity of adropin in TICs, while GPR19 immunoreactivity was noted in GCs in infantile, pre-pubertal, and pubertal mice ovary. Also, adropin and GPR19 are highly expressed in the CL of the ovary of reproductively active mice. The fact that adropin expression in the ovary at different stages of postnatal development positively correlated with circulating progesterone and estradiol indicated that it has a role in the production of steroid hormones. Furthermore, the results of in vivo studies in pre-pubertal mice showed that adropin promotes early folliculogenesis by enhancing the proliferation (PCNA) of GCs of cortical ovarian follicles and promotes estradiol production by enhancing the expression of GPR19, StAR, CYP11A1 and aromatase proteins. Also, adropin treatment increases the Bax/Bcl2 ratio and expression of cleaved caspase-3 and ERα proteins, which may result in increased apoptosis of medullary follicles leading to the formation of a well-developed interstitium with interstitial glandular cells. Collectively, these findings indicate that adropin may be a factor that accelerates pubertal development in the ovary and could be utilized as a therapeutic approach for treating pubertal delay.
MeSH term(s)	Animals ; Female ; Mice ; Cholesterol Side-Chain Cleavage Enzyme/metabolism ; Estradiol/metabolism ; Ovarian Follicle/physiology ; Ovary/metabolism ; Sexual Maturation
Chemical Substances	Cholesterol Side-Chain Cleavage Enzyme (EC 1.14.15.6) ; Estradiol (4TI98Z838E) ; Enho protein, mouse
Language	English
Publishing date	2023-09-23
Publishing country	England
Document type	Journal Article ; Research Support, Non-U.S. Gov't
ZDB-ID	1049188-0
ISSN	1879-1220 ; 0960-0760
ISSN (online)	1879-1220
ISSN	0960-0760
DOI	10.1016/j.jsbmb.2023.106404
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