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  1. Book: Neuropsychiatry

    Koliatsos, Vassilis E.

    (Psychiatric clinics of North America ; volume 43, number 2 (June 2020))

    2020  

    Author's details editor Vassilis E. Koliatsos
    Series title Psychiatric clinics of North America ; volume 43, number 2 (June 2020)
    Collection
    Language English
    Size xiv Seiten, Seite 214-413, Illustrationen
    Publisher Elsevier
    Publishing place Philadelphia, Pennsylvania
    Publishing country United States
    Document type Book
    HBZ-ID HT020524752
    ISBN 978-0-323-78951-6 ; 0-323-78951-X
    Database Catalogue ZB MED Medicine, Health

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  2. Article ; Online: NAD

    Alexandris, Athanasios S / Koliatsos, Vassilis E

    Antioxidants & redox signaling

    2023  Volume 39, Issue 16-18, Page(s) 1167–1184

    Abstract: Significance: ...

    Abstract Significance:
    MeSH term(s) Humans ; Wallerian Degeneration/metabolism ; Wallerian Degeneration/pathology ; NAD/metabolism ; Peripheral Nervous System Diseases/metabolism ; Axons/metabolism ; Neurodegenerative Diseases/metabolism
    Chemical Substances NAD (0U46U6E8UK)
    Language English
    Publishing date 2023-09-07
    Publishing country United States
    Document type Journal Article ; Review ; Research Support, Non-U.S. Gov't ; Research Support, N.I.H., Extramural
    ZDB-ID 1483836-9
    ISSN 1557-7716 ; 1523-0864
    ISSN (online) 1557-7716
    ISSN 1523-0864
    DOI 10.1089/ars.2023.0350
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article ; Online: Neuropsychiatry Coming of Age.

    Koliatsos, Vassilis E

    The Psychiatric clinics of North America

    2020  Volume 43, Issue 2, Page(s) xiii–xiv

    MeSH term(s) Humans ; Neuropsychiatry
    Keywords covid19
    Language English
    Publishing date 2020-04-08
    Publishing country United States
    Document type Editorial ; Introductory Journal Article
    ZDB-ID 431518-2
    ISSN 1558-3147 ; 0193-953X
    ISSN (online) 1558-3147
    ISSN 0193-953X
    DOI 10.1016/j.psc.2020.03.001
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  4. Article ; Online: Update on the Neuropsychiatry of Substance Use Disorders.

    Kolodner, George / Koliatsos, Vassilis E

    The Psychiatric clinics of North America

    2020  Volume 43, Issue 2, Page(s) 291–304

    Abstract: This article reviews some of the recent discoveries about how neurobiological processes contribute to the understanding and treatment of substance use disorders. Particular focus is given to cannabis, opioids, and designer drugs. Important areas ... ...

    Abstract This article reviews some of the recent discoveries about how neurobiological processes contribute to the understanding and treatment of substance use disorders. Particular focus is given to cannabis, opioids, and designer drugs. Important areas addressed include triggers and cravings, the central roles of dopamine and stress, and the endocannabinoid system. Clinical relevance of these findings for withdrawal management and relapse prevention is discussed. Also highlighted are issues related to the opioid epidemic and consequences both of continuing federal prohibition of cannabis as well as its state-by-state relaxation.
    MeSH term(s) Analgesics, Opioid ; Craving ; Humans ; Opioid-Related Disorders/therapy ; Substance Withdrawal Syndrome/drug therapy ; Substance-Related Disorders/therapy
    Chemical Substances Analgesics, Opioid
    Language English
    Publishing date 2020-04-08
    Publishing country United States
    Document type Journal Article ; Review
    ZDB-ID 431518-2
    ISSN 1558-3147 ; 0193-953X
    ISSN (online) 1558-3147
    ISSN 0193-953X
    DOI 10.1016/j.psc.2020.02.011
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  5. Article ; Online: The Behavioral Neuroscience of Traumatic Brain Injury.

    Koliatsos, Vassilis E / Rao, Vani

    The Psychiatric clinics of North America

    2020  Volume 43, Issue 2, Page(s) 305–330

    Abstract: Traumatic brain injury is a calamity of various causes, pathologies, and extremely varied and often complex clinical presentations. Because of its predilection for brain systems underlying cognitive and complex behavioral operations, it may cause chronic ...

    Abstract Traumatic brain injury is a calamity of various causes, pathologies, and extremely varied and often complex clinical presentations. Because of its predilection for brain systems underlying cognitive and complex behavioral operations, it may cause chronic and severe psychiatric illness that requires expert management. This is more so for the modern epidemic of athletic and military brain injuries which are dominated by psychiatric symptoms. Past medical, including psychiatric, history, and comorbidities are important and relevant for formulation and management. Traumatic brain injury is a model for other neuropsychiatric disorders and may serve as an incubator of new ideas for neurodegenerative disease.
    MeSH term(s) Blast Injuries/psychology ; Brain Contusion/psychology ; Brain Injuries, Traumatic/psychology ; Diffuse Axonal Injury/psychology ; Humans ; Military Personnel ; Stress Disorders, Post-Traumatic/psychology
    Language English
    Publishing date 2020-05-20
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 431518-2
    ISSN 1558-3147 ; 0193-953X
    ISSN (online) 1558-3147
    ISSN 0193-953X
    DOI 10.1016/j.psc.2020.02.009
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  6. Book: Cell death and diseases of the nervous system

    Koliatsos, Vassilis E.

    1999  

    Author's details ed. by Vassilis E. Koliatsos
    Keywords Nervous System Diseases / physiopathology ; Cell Death / physiology ; Disease Models, Animal ; Hirnschädigung ; Nervenzelle ; Apoptosis ; Nervendegeneration
    Subject Neurodegenerative Krankheit ; Neurodegenerative Erkrankung ; Cerebralschädigung ; Gehirn ; Hirnschaden ; Hirnläsion ; Zerebralschädigung ; Apoptose ; Programmierter Zelltod ; Ganglienzelle ; Neurozyt ; Neuron
    Language English
    Size XXI, 683 S. : Ill., graph. Darst.
    Publisher Humana Press
    Publishing place Totowa, NJ
    Publishing country United States
    Document type Book
    HBZ-ID HT009852251
    ISBN 0-89603-413-5 ; 978-0-89603-413-6
    Database Catalogue ZB MED Medicine, Health

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    1999 A 2434 order for loan Magazin

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  7. Article: A Clinical Approach to Cognitive Impairment.

    Koliatsos, Vassilis E

    Focus (American Psychiatric Publishing)

    2016  Volume 14, Issue 4, Page(s) 437–447

    Abstract: Cognitive impairment is ubiquitous in medicine and a common experience in everyday life. When severe, persistent, or progressive, it becomes a matter of clinical concern. Top causes of clinically significant cognitive impairment are neurodegenerative ... ...

    Abstract Cognitive impairment is ubiquitous in medicine and a common experience in everyday life. When severe, persistent, or progressive, it becomes a matter of clinical concern. Top causes of clinically significant cognitive impairment are neurodegenerative disease, stroke or atherosclerotic vascular disease of the brain, medication toxicity, and alcoholism. Several of these conditions are associated with aging of the population in industrialized nations, and they are comorbid with multiple other illnesses and polypharmacy. In evaluating such problems, history is paramount, including a detailed time course of symptoms and input from caregivers. Examination should include neurologic and mental status components and should be as thorough as possible. Ancillary testing should be strategic and address specific problems in the differential diagnosis. Appropriate referrals to other medical disciplines should be promptly arranged, with engagement of the primary care provider. The caregiver is often a hidden patient. The psychiatrist who works with cognitively impaired patients should stay abreast of developments in a rapidly evolving field and adopt a team approach to care with other clinical specialties and caregivers.
    Language English
    Publishing date 2016-10-07
    Publishing country United States
    Document type Journal Article
    ISSN 1541-4094
    ISSN 1541-4094
    DOI 10.1176/appi.focus.20160026
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  8. Article ; Online: Wallerian degeneration as a therapeutic target in traumatic brain injury.

    Koliatsos, Vassilis E / Alexandris, Athanasios S

    Current opinion in neurology

    2019  Volume 32, Issue 6, Page(s) 786–795

    Abstract: Purpose of review: Diffuse or traumatic axonal injury is one of the principal pathologies encountered in traumatic brain injury (TBI) and the resulting axonal loss, disconnection, and brain atrophy contribute significantly to clinical morbidity and ... ...

    Abstract Purpose of review: Diffuse or traumatic axonal injury is one of the principal pathologies encountered in traumatic brain injury (TBI) and the resulting axonal loss, disconnection, and brain atrophy contribute significantly to clinical morbidity and disability. The seminal discovery of the slow Wallerian degeneration mice (Wld) in which transected axons do not degenerate but survive and function independently for weeks has transformed concepts on axonal biology and raised hopes that axonopathies may be amenable to specific therapeutic interventions. Here we review mechanisms of axonal degeneration and also describe how these mechanisms may inform biological therapies of traumatic axonopathy in the context of TBI.
    Recent findings: In the last decade, SARM1 [sterile a and Toll/interleukin-1 receptor (TIR) motif containing 1] and the DLK (dual leucine zipper bearing kinase) and LZK (leucine zipper kinase) MAPK (mitogen-activated protein kinases) cascade have been established as the key drivers of Wallerian degeneration, a complex program of axonal self-destruction which is activated by a wide range of injurious insults, including insults that may otherwise leave axons structurally robust and potentially salvageable. Detailed studies on animal models and postmortem human brains indicate that this type of partial disruption is the main initial pathology in traumatic axonopathy. At the same time, the molecular dissection of Wallerian degeneration has revealed that the decision that commits axons to degeneration is temporally separated from the time of injury, a window that allows potentially effective pharmacological interventions.
    Summary: Molecular signals initiating and triggering Wallerian degeneration appear to be playing an important role in traumatic axonopathy and recent advances in understanding their nature and significance is opening up new therapeutic opportunities for TBI.
    MeSH term(s) Animals ; Axons/metabolism ; Axons/pathology ; Brain Injuries, Diffuse/metabolism ; Brain Injuries, Diffuse/pathology ; Brain Injuries, Traumatic/drug therapy ; Brain Injuries, Traumatic/metabolism ; Brain Injuries, Traumatic/pathology ; Humans ; Wallerian Degeneration/drug therapy ; Wallerian Degeneration/metabolism ; Wallerian Degeneration/pathology
    Language English
    Publishing date 2019-10-09
    Publishing country England
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, Non-P.H.S. ; Review
    ZDB-ID 1182686-1
    ISSN 1473-6551 ; 1350-7540
    ISSN (online) 1473-6551
    ISSN 1350-7540
    DOI 10.1097/WCO.0000000000000763
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  9. Article ; Online: Neuropsychiatry: Definitions, Concepts, and Patient Types.

    Koliatsos, Vassilis E / Wisner-Carlson, Robert / Watkins, Crystal

    The Psychiatric clinics of North America

    2020  Volume 43, Issue 2, Page(s) 213–227

    Abstract: Neuropsychiatry is an integrative discipline defined by its history, its preferred patients, and its theoretic framework. Dealing with human behavior needs to consider the brain, but such consideration should avoid oversimplification: neurologic ... ...

    Abstract Neuropsychiatry is an integrative discipline defined by its history, its preferred patients, and its theoretic framework. Dealing with human behavior needs to consider the brain, but such consideration should avoid oversimplification: neurologic understanding is not essential, necessary, or desirable in all conditions encountered in clinical psychiatry. Neuropsychiatric theory is founded on discoveries in the areas of synaptic plasticity and cortical/limbic anatomy (bottom-up), but also evolutionary biology and anthropology (top-down). Going forward, we need to synthesize vital information, distinguish the essential from the trivial or tenuous, and remain open to dialogue with allied disciplines, our patients, and our students.
    MeSH term(s) History, 19th Century ; History, 20th Century ; Humans ; Mental Disorders/pathology ; Neuropsychiatry
    Language English
    Publishing date 2020-05-21
    Publishing country United States
    Document type Historical Article ; Journal Article ; Research Support, Non-U.S. Gov't ; Review
    ZDB-ID 431518-2
    ISSN 1558-3147 ; 0193-953X
    ISSN (online) 1558-3147
    ISSN 0193-953X
    DOI 10.1016/j.psc.2020.02.007
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  10. Article ; Online: Primary Traumatic Axonopathy in Mice Subjected to Impact Acceleration: A Reappraisal of Pathology and Mechanisms with High-Resolution Anatomical Methods.

    Ziogas, Nikolaos K / Koliatsos, Vassilis E

    The Journal of neuroscience : the official journal of the Society for Neuroscience

    2018  Volume 38, Issue 16, Page(s) 4031–4047

    Abstract: Traumatic axonal injury (TAI) is a common neuropathology in traumatic brain injury and is featured by primary injury to axons. Here, we generated TAI with impact acceleration of the head in ... ...

    Abstract Traumatic axonal injury (TAI) is a common neuropathology in traumatic brain injury and is featured by primary injury to axons. Here, we generated TAI with impact acceleration of the head in male
    MeSH term(s) Animals ; Armadillo Domain Proteins/metabolism ; Axons/metabolism ; Axons/pathology ; Brain Injuries, Traumatic/diagnostic imaging ; Brain Injuries, Traumatic/pathology ; Cytoskeletal Proteins/metabolism ; Hydrogel, Polyethylene Glycol Dimethacrylate/chemistry ; Male ; Mice ; Mice, Inbred C57BL ; Microscopy, Fluorescence, Multiphoton/methods ; Microscopy, Fluorescence, Multiphoton/standards ; Nicotinamide-Nucleotide Adenylyltransferase/metabolism ; Pyramidal Tracts/pathology ; Sensitivity and Specificity ; Single-Cell Analysis/methods ; Single-Cell Analysis/standards
    Chemical Substances Armadillo Domain Proteins ; Cytoskeletal Proteins ; SARM1 protein, mouse ; Hydrogel, Polyethylene Glycol Dimethacrylate (25852-47-5) ; Nicotinamide-Nucleotide Adenylyltransferase (EC 2.7.7.1) ; Nmnat2 protein, mouse (EC 2.7.7.1)
    Language English
    Publishing date 2018-03-22
    Publishing country United States
    Document type Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, Non-P.H.S.
    ZDB-ID 604637-x
    ISSN 1529-2401 ; 0270-6474
    ISSN (online) 1529-2401
    ISSN 0270-6474
    DOI 10.1523/JNEUROSCI.2343-17.2018
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