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Article: Age-related changes in human thyroarytenoid muscles: a histological and histochemical study.

Kersing, W / Jennekens, F G I

European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery

2004 Volume 261, Issue 7, Page(s) 386–392

Abstract: We examined the thyroarytenoid muscles of 23 larynges in order to assess function-related characteristics and to discover age-related changes. The neonatal thyroarytenoid muscle differed from limb muscles in the slow maturation of fibre types. In adults, ...

Abstract	We examined the thyroarytenoid muscles of 23 larynges in order to assess function-related characteristics and to discover age-related changes. The neonatal thyroarytenoid muscle differed from limb muscles in the slow maturation of fibre types. In adults, we examined the medial part of the thyroarytenoid muscle. It showed a larger variation in fibre size and more endomysial connective tissue than is common for limb muscles. Structural and histochemical evidence of ageing developed from approximately the 6th decade. It comprised a marked increase of endomysial connective tissue and striking myopathic changes of muscle fibres. Up to 20% of the muscle fibres showed at some places of some sections evidence of mitochondrial accumulations and increased mitochondrial enzyme activity (ragged red fibres). The rise of such ragged red fibres is commonly related to the development of mutations in mitochondrial DNA. Significant myopathic changes including mitochondrial abnormalities develop in the thyrovocalis muscle with age and may play a role in the functional deficit of the larynx in old age.
MeSH term(s)	Adolescent ; Adult ; Aged ; Aged, 80 and over ; Aging/pathology ; Child, Preschool ; Female ; Histocytochemistry ; Humans ; Infant ; Infant, Newborn ; Laryngeal Muscles/anatomy & histology ; Laryngeal Muscles/growth & development ; Laryngeal Muscles/metabolism ; Laryngeal Muscles/pathology ; Male ; Middle Aged ; Muscle Development ; Myosins/metabolism
Chemical Substances	Myosins (EC 3.6.4.1)
Language	English
Publishing date	2004-08
Publishing country	Germany
Document type	Journal Article
ZDB-ID	1017359-6
ISSN	0937-4477
ISSN	0937-4477
DOI	10.1007/s00405-003-0702-z
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: Vinken and Bruyn's Handbook of Clinical Neurology. A witness of late-twentieth century neurological progress.

Koehler, P J / Jennekens, F G I

Journal of the history of the neurosciences

2008 Volume 17, Issue 1, Page(s) 46–55

Abstract: Vinken and Bruyn's Handbook of Clinical Neurology (HCN) is best characterized as an encyclopedia. In this paper we describe the origin, production, and reception of HCN. Data were gathered from a literature search, by screening of HCN-volumes, ... ...

Abstract	Vinken and Bruyn's Handbook of Clinical Neurology (HCN) is best characterized as an encyclopedia. In this paper we describe the origin, production, and reception of HCN. Data were gathered from a literature search, by screening of HCN-volumes, interviewing key-role persons and a study of an HCN-archive. The initiative for HCN was taken by two Excerpta Medica staff members, the one a strategist with expertise in information systems, the other a gifted neurologist with an expert knowledge of who is who in the world of neurological literature. Within a period of 38 years, 2799 authors, 28 volume editors, the two initiators, and a third chief editor for the American continent described the whole of neurology in 1909 chapters on all together 46,025 pages (excluding index volumes). HCN was sold mainly to medical institutes in affluent countries. A digital version of the revised edition was proposed by the editors but refused by the publisher for commercial reasons. HCN was in general well received by book reviewers. The main criticisms concerned the price of the volumes, lack of editorial control, inadequacy of indexes, and lack of cross references. HCN offers unrivalled information on the state of the art of the clinical neurosciences in the second half of the twentieth century. In addition, it contains extensive reviews of the history of neurological diseases in the volumes of the original edition.
MeSH term(s)	Encyclopedias as Topic ; History, 20th Century ; Humans ; Neurology/history ; Practice Patterns, Physicians'/history ; Publishing/history ; Reference Books
Language	English
Publishing date	2008
Publishing country	England
Document type	Historical Article ; Journal Article
ZDB-ID	1233549-6
ISSN	1744-5213 ; 0964-704X
ISSN (online)	1744-5213
ISSN	0964-704X
DOI	10.1080/09647040600820050
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Article: The central nervous system in systemic lupus erythematosus. Part 2. Pathogenetic mechanisms of clinical syndromes: a literature investigation.

Jennekens, F G I / Kater, L

Rheumatology (Oxford, England)

2002 Volume 41, Issue 6, Page(s) 619–630

Abstract: Objectives: To identify the pathogenetic mechanisms of central nervous system (CNS) syndromes of systemic lupus erythematosus (SLE) as described in the literature.: Methods: Using PUBMED, we performed a systematic search of publications from 1980 ... ...

Abstract	Objectives: To identify the pathogenetic mechanisms of central nervous system (CNS) syndromes of systemic lupus erythematosus (SLE) as described in the literature. Methods: Using PUBMED, we performed a systematic search of publications from 1980 onwards. Studies were eligible if they had been performed on patients or material from patients with CNS manifestations and definite SLE and when the CNS manifestations were not secondary. Criteria were formulated for the identification of pathogenetic mechanisms. Results: The single most important cause of the CNS syndromes of SLE is ischaemia due to narrowing or occlusion of small vessels, arteries and veins. Antiphospholipid antibodies and premature atherosclerosis play roles in these processes, but they have not been delineated definitely. Intracranial and intraspinal haemorrhages are much less frequent than ischaemia and are presumably in part due directly to SLE. Vasculitis may cause ischaemia or haemorrhage in the CNS and is involved occasionally, as shown by imaging and histological findings. White matter damage is heterogeneous and ill-understood. It includes white matter degeneration and myelin vacuolation of the spinal cord, and reversible leucoencephalopathy due to oedema. Antibody-induced neuronal dysfunction in the CNS is a realistic hypothesis and may involve anti-ribosomal P antibodies and several other antibodies. Deficiency of psychological reactions forms a separate and entirely different category of mechanisms. Conclusions: Causes have been identified or possible causes have been suggested for most of the CNS syndromes of SLE, thus offering rationales for different forms of prevention and therapy.
MeSH term(s)	Brain Ischemia/pathology ; Humans ; Lupus Vasculitis, Central Nervous System/etiology ; Lupus Vasculitis, Central Nervous System/pathology ; Terminology as Topic ; Vasculitis, Central Nervous System/pathology
Language	English
Publishing date	2002-06
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	1464822-2
ISSN	1462-0332 ; 1462-0324
ISSN (online)	1462-0332
ISSN	1462-0324
DOI	10.1093/rheumatology/41.6.619
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Article: The central nervous system in systemic lupus erythematosus. Part 1. Clinical syndromes: a literature investigation.

Jennekens, F G I / Kater, L

Rheumatology (Oxford, England)

2002 Volume 41, Issue 6, Page(s) 605–618

Abstract: Objectives: To establish the central nervous system (CNS) manifestations of systemic lupus erythematosus (SLE) as described in the literature and to compare the results with two previously published classifications.: Methods: Using PUBMED, a ... ...

Abstract	Objectives: To establish the central nervous system (CNS) manifestations of systemic lupus erythematosus (SLE) as described in the literature and to compare the results with two previously published classifications. Methods: Using PUBMED, a systematic search was performed for publications from 1980 onwards on CNS syndromes of patients with SLE. A distinction was made between CNS syndromes induced by SLE and the CNS autoimmune diseases associated with SLE. Criteria were defined for inclusion of CNS syndromes or diseases as SLE-induced or SLE-associated. Results: The literature search yielded names of 30 syndromes and two diseases, but only 16 syndromes and one disease fulfilled the set of predefined criteria. Two syndromes-depression and anxiety-were predominantly psychological in origin in most patients; other syndromes were biological. Discussion: Strengths and weaknesses of two classifications of CNS syndromes are evaluated. The older of the two is long and has not been accepted fully. Brevity is an advantage of the American College of Rheumatology (ACR) nomenclature system. A disadvantage of this system is the concealment of differences in health risks by the pooling of items. Furthermore, the items of the system do not all belong to the same dimension: one is pathological and the others are clinical. To remedy these drawbacks, we suggest the rephrasing and subdivision of items and that the predominantly psychopathological syndromes should be dealt with separately in epidemiological studies. Conclusions: SLE may induce 16 different clinical syndromes of the CNS and is occasionally associated with one other CNS autoimmune disease. A modification of the ACR nomenclature system is proposed.
MeSH term(s)	Brain Diseases/etiology ; Cognition Disorders/etiology ; Humans ; Lupus Vasculitis, Central Nervous System/complications ; Lupus Vasculitis, Central Nervous System/diagnosis ; Lupus Vasculitis, Central Nervous System/psychology ; Syndrome ; Terminology as Topic
Language	English
Publishing date	2002-06
Publishing country	England
Document type	Journal Article ; Review
ZDB-ID	1464822-2
ISSN	1462-0332 ; 1462-0324
ISSN (online)	1462-0332
ISSN	1462-0324
DOI	10.1093/rheumatology/41.6.605
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Book: Myotone dystrofie

Jennekens, F. G. I

begeleiding en behandeling

2000

Author's details	redactie, F.G.I. Jennekens ... [et al.]
MeSH term(s)	Myotonic Dystrophy
Language	Dutch
Size	95 p. :, ill.
Publisher	Elsevier Gezondheidszorg
Publishing place	Maarssen
Document type	Book
ISBN	9789035222762 ; 9035222768
Database	Catalogue of the US National Library of Medicine (NLM)

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Book: Behandeling en begeleiding van patiënten met amyotrofe laterale sclerose

Jennekens, F. G. I

1997

Author's details	onder redactie van F.G.I. Jennekens en J.M.B.V. de Jong
MeSH term(s)	Amyotrophic Lateral Sclerosis
Language	Dutch
Size	x, 89 p. :, ill.
Publisher	Wetenschappelijke uitgeverij Bunge
Publishing place	Utrecht
Document type	Book
ISBN	9789063481308 ; 9063481306
Database	Catalogue of the US National Library of Medicine (NLM)

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Article ; Online: Rembrandt's 'Beggar with a wooden leg' and other comparable prints.

ten Kate, J J / Jennekens, F G I / Vos-Niël, J M E

The Journal of bone and joint surgery. British volume

2009 Volume 91, Issue 2, Page(s) 278–282

Abstract: Rembrandt's etching of a beggar with a wooden leg is notable because the two lower limbs of the presumed beggar are present and not deformed. Using the facilities of four specialised Dutch art institutes, we carried out a systematic investigation to find ...

Abstract	Rembrandt's etching of a beggar with a wooden leg is notable because the two lower limbs of the presumed beggar are present and not deformed. Using the facilities of four specialised Dutch art institutes, we carried out a systematic investigation to find other etchings and engravings of subjects with artificial legs supporting non-amputated limbs, from the period 1500 to 1700 AD. We discovered 28 prints produced by at least 18 artists. Several offered clues to a disorder of a knee, the lower leg or the foot. All individuals were adult males, suggesting the probability of traumatic lesions. We conclude that in this period artificial legs were not only used in the case of absence of part of a lower limb, but also for other reasons, notably disorders of the knee, lower leg or foot. They may also have been used to attract compassion.
MeSH term(s)	Amputees/history ; Artificial Limbs/history ; Empathy ; Engraving and Engravings/history ; History, 16th Century ; History, 17th Century ; History, 18th Century ; Humans ; Medicine in the Arts
Language	English
Publishing date	2009-02
Publishing country	England
Document type	Historical Article ; Journal Article
ZDB-ID	220626-2
ISSN	2044-5377 ; 0301-620X ; 0447-9076
ISSN (online)	2044-5377
ISSN	0301-620X ; 0447-9076
DOI	10.1302/0301-620X.91B2.21747
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Article: Medical therapy in spinal muscular atrophy: a realistic expectation?

Jennekens, F G

Clinical neurology and neurosurgery

1992 Volume 94 Suppl, Page(s) S89–92

Abstract: The hereditary spinal muscular atrophies (SMA) type I-III belong to those diseases for which even the thought of medical therapy seems forbidden. Two neurotrophic factors are, however, now known to exert a markedly stimulating effect on survival of motor ...

Abstract	The hereditary spinal muscular atrophies (SMA) type I-III belong to those diseases for which even the thought of medical therapy seems forbidden. Two neurotrophic factors are, however, now known to exert a markedly stimulating effect on survival of motor neurons in vivo! In principle such factors may become available by recombinant DNA techniques for experiments in animal models of SMA and if these experiments are successful for clinical trials in man. Medical therapy in SMA should aim primarily at patients early in the rapidly progressive phase of their disease, before massive loss of motoneuron has taken place.
MeSH term(s)	Adrenocorticotropic Hormone/therapeutic use ; Adult ; Humans ; Infant ; Infant, Newborn ; Male ; Motor Neurons/drug effects ; Muscular Atrophy, Spinal/drug therapy ; Nerve Degeneration/drug effects ; Nerve Growth Factors/therapeutic use ; Peptide Fragments/therapeutic use ; Spinal Muscular Atrophies of Childhood/drug therapy ; alpha-MSH/therapeutic use
Chemical Substances	Nerve Growth Factors ; Peptide Fragments ; ACTH (4-9) (56236-83-0) ; alpha-MSH (581-05-5) ; Adrenocorticotropic Hormone (9002-60-2)
Language	English
Publishing date	1992
Publishing country	Netherlands
Document type	Journal Article
ZDB-ID	193107-6
ISSN	1872-6968 ; 0303-8467
ISSN (online)	1872-6968
ISSN	0303-8467
DOI	10.1016/0303-8467(92)90034-z
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Book: Neurology of the inflammatory connective tissue diseases

Jennekens, F. G. I / Kater, Louis

(Major problems in neurology ; v. 35)

1999

Author's details	Frans G.I. Jennekens, Louis Kater
Series title	Major problems in neurology ; v. 35
MeSH term(s)	Connective Tissue Diseases/complications ; Inflammation/complications ; Neurologic Manifestations
Language	English
Size	364 p. :, ill. ;, 24 cm.
Publisher	W.B. Saunders
Publishing place	London ; Philadelphia
Document type	Book
ISBN	9780702022319 ; 0702022314
Database	Catalogue of the US National Library of Medicine (NLM)

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Article ; Online: Validation of a preterm infant cardiovascular system model under baroreflex control with heart rate and blood pressure data.

Jennekens, Ward / Dat, Marco / Bovendeerd, Peter H M / Wijn, Pieter F F / Andriessen, Peter

Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual International Conference

2011 Volume 2011, Page(s) 896–899

Abstract: ... of gestation with a birth weight of 1000 g and a closed ductus arteriosus by the end of the first week, that is ...

Abstract	In this paper we present an autonomic cardiovascular model of a preterm infant of 28 weeks of gestation with a birth weight of 1000 g and a closed ductus arteriosus by the end of the first week, that is capable of describing the complex interactions between heart rate, blood pressure and respiration. The hemodynamic model consists of a pulsatile heart and several vascular compartments, and is regulated by a baroreflex control system. The model is relatively simple to allow for a mathematical analysis of the dynamics but sufficiently complex to provide a realistic representation of the underlying physiology. The model provides (beat-to-beat) values of R-R interval and blood pressure that resemble realistic signals of preterm infants. The model is validated with experimental data obtained in preterm infants.
MeSH term(s)	Baroreflex/physiology ; Blood Pressure/physiology ; Computer Simulation ; Heart Rate/physiology ; Humans ; Infant, Newborn ; Infant, Premature/physiology ; Male ; Models, Cardiovascular ; Reproducibility of Results ; Sensitivity and Specificity
Language	English
Publishing date	2011-12-15
Publishing country	United States
Document type	Evaluation Study ; Journal Article ; Validation Study
ISSN	2694-0604
ISSN (online)	2694-0604
DOI	10.1109/IEMBS.2011.6090200
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