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  1. Article ; Online: Frederic E. Mohs, M.D. (1910-2002): physician and innovator.

    Ross, Nicholas A / Saedi, Nazanin / Yeo, Charles J / Cowan, Scott

    The American surgeon

    2015  Volume 81, Issue 5, Page(s) 433–437

    MeSH term(s) General Surgery/history ; History, 20th Century ; History, 21st Century ; Inventions/history ; Physicians/history ; Wisconsin
    Language English
    Publishing date 2015-05-12
    Publishing country United States
    Document type Biography ; Historical Article ; Journal Article ; Portrait
    ZDB-ID 202465-2
    ISSN 1555-9823 ; 0003-1348
    ISSN (online) 1555-9823
    ISSN 0003-1348
    DOI 10.1177/000313481508100520
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  2. Article ; Online: Effects of hemoglobin C, D, E and S traits on measurements of hemoglobin A1c by twelve methods.

    Rohlfing, Curt / Hanson, Steven / Weykamp, Cas / Siebelder, Carla / Higgins, Trefor / Molinaro, Ross / Yip, Paul M / Little, Randie R

    Clinica chimica acta; international journal of clinical chemistry

    2016  Volume 455, Page(s) 80–83

    Abstract: Background: Hemoglobin C, D Punjab, E or S trait can interfere with hemoglobin A1c (HbA1c) results ... Bio-Rad D-100, Variant II NU and Variant II Turbo 2.0, Menarini HA-8180T thalassemia mode and HA-8180V ...

    Abstract Background: Hemoglobin C, D Punjab, E or S trait can interfere with hemoglobin A1c (HbA1c) results. We assessed whether they affect results obtained with 12 current assay methods.
    Methods: Hemoglobin AA (HbAA), HbAC, HbAD Punjab, HbAE and HbAS samples were analyzed on one enzymatic, nine ion-exchange HPLC and two Capillary Electrophoresis methods. Trinity ultra(2) boronate affinity HPLC was the comparative method. An overall test of coincidence of least-squared linear regression lines was performed to determine if HbA1c results were statistically significantly different from those of HbAA samples. Clinically significant interference was defined as >7% difference from HbAA at 6 or 9% HbA1c compared to ultra(2) using Deming regression.
    Results: All methods showed statistically significant effects for one or more variants. Clinically significant effects were observed for the Tosoh G8 variant mode and GX (all variants), GX V1.22 (all but HbAE) and G11 variant mode (HbAC). All other methods (Abbott Architect c Enzymatic, Bio-Rad D-100, Variant II NU and Variant II Turbo 2.0, Menarini HA-8180T thalassemia mode and HA-8180V variant mode, Sebia Capillarys 2 and Capillarys 3) showed no clinically significant differences.
    Conclusions: Several methods showed clinically significant interference with HbA1c results from one or more variants which could adversely affect patient care.
    MeSH term(s) Chromatography, Affinity/methods ; Chromatography, High Pressure Liquid/methods ; Chromatography, Ion Exchange/methods ; Electrophoresis, Capillary/methods ; Glycated Hemoglobin/analysis ; Hemoglobins/chemistry ; Humans
    Chemical Substances Glycated Hemoglobin A ; Hemoglobins ; hemoglobin A1c protein, human
    Language English
    Publishing date 2016-01-29
    Publishing country Netherlands
    Document type Journal Article ; Research Support, N.I.H., Extramural
    ZDB-ID 80228-1
    ISSN 1873-3492 ; 0009-8981
    ISSN (online) 1873-3492
    ISSN 0009-8981
    DOI 10.1016/j.cca.2016.01.031
    Database MEDical Literature Analysis and Retrieval System OnLINE

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  3. Article ; Online: The L.E.A.D. framework: using tools from evidence-based public health to address evidence needs for obesity prevention.

    Kumanyika, Shiriki / Brownson, Ross C / Cheadle, Allen

    Preventing chronic disease

    2012  Volume 9, Page(s) E125

    MeSH term(s) Evidence-Based Practice ; Female ; Health Policy ; Humans ; Information Dissemination/methods ; Male ; Obesity/prevention & control ; Organizational Culture ; Organizational Innovation ; Politics ; Population Surveillance ; Public Health Practice
    Language English
    Publishing date 2012-07-12
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, P.H.S.
    ZDB-ID 2135684-1
    ISSN 1545-1151 ; 1545-1151
    ISSN (online) 1545-1151
    ISSN 1545-1151
    DOI 10.5888/pcd9:120157
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  4. Article: Modulation of apolipoprotein D and apolipoprotein E mRNA expression by growth arrest and identification of key elements in the promoter.

    Do Carmo, Sonia / Séguin, Diane / Milne, Ross / Rassart, Eric

    The Journal of biological chemistry

    2001  Volume 277, Issue 7, Page(s) 5514–5523

    Abstract: Apolipoprotein D (apoD) and apolipoprotein E (apoE) are co-expressed in many tissues, and ...

    Abstract Apolipoprotein D (apoD) and apolipoprotein E (apoE) are co-expressed in many tissues, and, in certain neuropathological situations, their expression appears to be under coordinate regulation. We have previously shown that apoD gene expression in cultured human fibroblasts is up-regulated when the cells undergo growth arrest. Here, we demonstrate that, starting around day 2 of growth arrest, both apoD and apoE mRNA levels increase between 1.5- and 27-fold in other cell types, including mouse primary fibroblasts and fibroblast-like and human astrocytoma cell lines. To understand the regulatory mechanisms of apoD expression, we have used apoD promoter-luciferase reporter constructs to compare gene expression in growing cells and in cells that have undergone growth arrest. Analysis of gene expression in cells transfected with constructs with deletions and mutations in the apoD promoter and constructs with artificial promoters demonstrated that the region between nucleotides -174 and -4 is fully responsible for the basal gene expression, whereas the region from -558 to -179 is implicated in the induction of apoD expression following growth arrest. Within this region, an alternating purine-pyrimidine stretch and a pair of serum-responsive elements (SRE) were found to be major determinants of growth arrest-induced apoD gene expression. Evidence is also presented that SREs in the apoE promoter may contribute to the up-regulation of apoE gene expression following growth arrest.
    MeSH term(s) 3T3 Cells ; Animals ; Apolipoproteins/metabolism ; Apolipoproteins D ; Apolipoproteins E/metabolism ; Base Sequence ; Blotting, Northern ; COS Cells ; Cell Division ; Cell Nucleus/metabolism ; Cells, Cultured ; DNA/metabolism ; Exons ; Fibroblasts/metabolism ; Genes, Reporter ; Humans ; Introns ; Luciferases/metabolism ; Mice ; Mice, Inbred BALB C ; Mice, Inbred C57BL ; Molecular Sequence Data ; Mutagenesis, Site-Directed ; Promoter Regions, Genetic ; RNA/metabolism ; RNA, Messenger/metabolism ; Serum Response Element ; Time Factors ; Transfection ; Tumor Cells, Cultured ; Up-Regulation ; beta-Galactosidase/metabolism
    Chemical Substances Apolipoproteins ; Apolipoproteins D ; Apolipoproteins E ; RNA, Messenger ; RNA (63231-63-0) ; DNA (9007-49-2) ; Luciferases (EC 1.13.12.-) ; beta-Galactosidase (EC 3.2.1.23)
    Language English
    Publishing date 2001-11-15
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't
    ZDB-ID 2997-x
    ISSN 1083-351X ; 0021-9258
    ISSN (online) 1083-351X
    ISSN 0021-9258
    DOI 10.1074/jbc.M105057200
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  5. Article ; Online: Autoantibodies to survival of motor neuron complex in patients with polymyositis: immunoprecipitation of D, E, F, and G proteins without other components of small nuclear ribonucleoproteins.

    Satoh, Minoru / Chan, Jason Y F / Ross, Steven J / Ceribelli, Angela / Cavazzana, Ilaria / Franceschini, Franco / Li, Yi / Reeves, Westley H / Sobel, Eric S / Chan, Edward K L

    Arthritis and rheumatism

    2011  Volume 63, Issue 7, Page(s) 1972–1978

    Abstract: ... of (35) S-methionine-labeled cell extracts. Sera with which D, E, F, and G proteins of snRNP was ... immunofluorescence.: Results: Three sera that immunoprecipitated D, E, F, and G proteins without other components ... of the diagnosis or of certain clinical characteristics. An unusual pattern of immunoprecipitation, in which the D ...

    Abstract Objective: Autoantibodies in the systemic rheumatic diseases are clinically useful biomarkers of the diagnosis or of certain clinical characteristics. An unusual pattern of immunoprecipitation, in which the D, E, F, and G proteins of small nuclear RNPs (snRNP) but without other components of the snRNP, was noticed at the autoantibody screening. The purpose of this study was to examine the target antigens and clinical manifestations associated with this specificity.
    Methods: Autoantibodies in sera from 1,966 American patients (including 434 with systemic lupus erythematosus, 121 with scleroderma, 86 with polymyositis/dermatomyositis [PM/DM]) and 248 Italian patients with autoimmune diseases were screened by immunoprecipitation of (35) S-methionine-labeled cell extracts. Sera with which D, E, F, and G proteins of snRNP was immunoprecipitated, but without the other snRNP proteins, were further examined by analysis of RNA components by immunoprecipitation (silver staining), Western blotting using survival of motor neuron (SMN) complex, and immunofluorescence.
    Results: Three sera that immunoprecipitated D, E, F, and G proteins without other components (U1-70K, A, B'/B, C) of the snRNP were found. Four additional proteins (130 kd, 120 kd, 38 kd, and 33 kd) were also commonly immunoprecipitated. The target antigen was identified as SMN complex (Gemin 3, Gemin 4, SMN, and Gemin 2, respectively), which plays a critical role in the assembly of snRNP. In immunofluorescence analyses, all 3 sera showed nuclear dots (Cajal bodies) and cytoplasmic staining. Only 1 serum was weakly positive on Western blotting of SMN, suggesting that these sera mainly recognize native molecule or quaternary structure. All 3 patients were white women with PM, an interesting finding, since deletion or mutation of SMN is known to cause spinal muscular atrophy.
    Conclusion: SMN complex was identified as a new Cajal body autoantigen recognized by sera from white patients with PM. The biologic and clinical significance of anti-SMN autoantibodies will need to be clarified.
    MeSH term(s) Adult ; Autoantibodies/immunology ; Autoimmune Diseases/immunology ; Blotting, Western ; Female ; Fluorescent Antibody Technique ; Humans ; Immunoprecipitation ; Lupus Erythematosus, Systemic/immunology ; Male ; Middle Aged ; Polymyositis/immunology ; Ribonucleoproteins, Small Nuclear/immunology ; SMN Complex Proteins/immunology ; Scleroderma, Systemic/immunology
    Chemical Substances Autoantibodies ; Ribonucleoproteins, Small Nuclear ; SMN Complex Proteins
    Language English
    Publishing date 2011-03-21
    Publishing country United States
    Document type Journal Article ; Research Support, Non-U.S. Gov't ; Research Support, U.S. Gov't, P.H.S.
    ZDB-ID 127294-9
    ISSN 1529-0131 ; 0004-3591 ; 2326-5191
    ISSN (online) 1529-0131
    ISSN 0004-3591 ; 2326-5191
    DOI 10.1002/art.30349
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  6. Article: RE: Rose, D. E. (1994). Viewpoint. AJA, 3(1), 6.

    Ross, M

    American journal of audiology

    1994  Volume 3, Issue 2, Page(s) 78

    Language English
    Publishing date 1994-07-01
    Publishing country United States
    Document type Journal Article
    ZDB-ID 1162315-9
    ISSN 1558-9137 ; 1059-0889
    ISSN (online) 1558-9137
    ISSN 1059-0889
    DOI 10.1044/1059-0889.0302.78a
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  7. Article: Study of the decays D-->K pi pi e nu and D-->K* pi e nu.

    Anjos / Appel / Bean / Bracker / Browder / Cremaldi / Duboscq / Elliott / Escobar / Gibney / Hartner / Huber / Karchin / Kumar / Losty / Luste / Mantsch / Martin / McHugh /
    Menary / Morrison / Nash / Pinfold / Punkar / Purohit / Ross / Santoro / Schmidt / Shoup / Sliwa / Sokoloff / Souza / Sperka / Streetman / Stundia / Witherell

    Physical review. D, Particles and fields

    1992  Volume 45, Issue 7, Page(s) R2177–R2180

    Language English
    Publishing date 1992-04-01
    Publishing country United States
    Document type Journal Article
    ZDB-ID 209772-2
    ISSN 0556-2821 ; 2470-0010 ; 1550-7998
    ISSN 0556-2821 ; 2470-0010 ; 1550-7998
    DOI 10.1103/physrevd.45.r2177
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  8. Book ; Online ; E-Book: Textbook of pediatric rheumatology

    Petty, Ross E. / Laxer, Ronald M. / Lindsley, Carol B. / Wedderburn, Lucy R. / Mellins, Elizabeth D. / Fuhlbrigge, Robert C.

    (Expert consult)

    2022  

    Title variant Pediatric rheumatology
    Author's details Ross E. Petty, Ronald M. Laxer, Carol B. Lindsley, Lucy R. Wedderburn, Elizabeth D. Mellins, Robert C. Fuhlbrigge
    Series title Expert consult
    Keywords Electronic books
    Language English
    Size 1 Online-Ressource (748 Seiten), Illustrationen, Diagramme
    Edition Eight edition
    Publisher Elsevier
    Publishing place Philadelphia, PA
    Publishing country United States
    Document type Book ; Online ; E-Book
    Remark Zugriff für angemeldete ZB MED-Nutzerinnen und -Nutzer
    HBZ-ID HT020937720
    ISBN 978-0-323-63653-7 ; 9780323636520 ; 0-323-63653-5 ; 0323636527
    Database ZB MED Catalogue: Medicine, Health, Nutrition, Environment, Agriculture

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  9. Article ; Online: Study of the decay D+-->K-bar0-bare+ nu e.

    Anjos / Appel / Bean / Bracker / Browder / Cremaldi / Duboscq / Elliott / Escobar / Gibney / Hartner / Karchin / Kumar / Losty / Luste / Mantsch / Martin / McHugh / Menary /
    Morrison / Nash / Pinfold / Punkar / Purohit / Ross / Santoro / Schmidt / Shoup / Sliwa / Sokoloff / Souza / Sperka / Spalding / Streetman / Stundia / Witherell

    Physical review letters

    1991  Volume 67, Issue 12, Page(s) 1507–1510

    Language English
    Publishing date 1991-09-16
    Publishing country United States
    Document type Journal Article
    ZDB-ID 208853-8
    ISSN 1079-7114 ; 0031-9007
    ISSN (online) 1079-7114
    ISSN 0031-9007
    DOI 10.1103/PhysRevLett.67.1507
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  10. Book: An introduction to global health ethics

    Pinto, Andrew D. / Upshur, Ross E.G.

    2013  

    Title variant Global health ethics
    Author's details ed. by Andrew D. Pinto ; Ross E.G. Upshur
    Language English
    Size IX, 166 S.
    Publisher Routledge
    Publishing place London u.a.
    Publishing country Great Britain
    Document type Book
    HBZ-ID HT017189768
    ISBN 978-0-415-67352-5 ; 978-0-415-68183-4 ; 9780203082225 ; 0-415-67352-6 ; 0-415-68183-9 ; 0203082222
    Database Catalogue ZB MED Medicine, Health

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