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Article ; Online: The path forward: Highlighting social justice pearls in public health campaigns and initiatives to deliver equitable healthy living medicine.

Huizar, Martha I / Alman, Rocio / Arena, Ross / Laddu, Deepika R

2022 Volume 71, Page(s) 51–57

Abstract: The prevalence of unhealthy living behaviors is largely driven by environments that support them and has become a key concern at global, national, and individual (patient) levels. Healthy Living Medicine offers a compelling path forward to move people ... ...

Abstract	The prevalence of unhealthy living behaviors is largely driven by environments that support them and has become a key concern at global, national, and individual (patient) levels. Healthy Living Medicine offers a compelling path forward to move people towards healthy living behaviors and better health outcomes when complemented by socially just and equitable public campaigns and initiatives. Some of the concepts that are critical for these campaigns and initiatives that will be discussed in this manuscript include the social determinants of health, the communication loop, health literacy, and implicit bias and discrimination. Considering what is practical and achievable, examples of actionable, socially-just strategies will be described to inform and encourage health professionals and other stakeholders to prioritize healthy living and reverse the poor health trajectory among our most vulnerable populations.
MeSH term(s)	Health Literacy ; Health Promotion ; Healthy Lifestyle ; Humans ; Social Justice ; Vulnerable Populations
Language	English
Publishing date	2022-04-29
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	209312-1
ISSN	1873-1740 ; 1532-8643 ; 0033-0620
ISSN (online)	1873-1740 ; 1532-8643
ISSN	0033-0620
DOI	10.1016/j.pcad.2022.04.003
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Article ; Online: The global food syndemic: The impact of food insecurity, Malnutrition and obesity on the healthspan amid the COVID-19 pandemic.

Huizar, Martha I / Arena, Ross / Laddu, Deepika R

Progress in cardiovascular diseases

2020 Volume 64, Page(s) 105–107

MeSH term(s)	COVID-19/epidemiology ; COVID-19/mortality ; Diet, Healthy ; Feeding Behavior ; Food Assistance ; Food Insecurity ; Global Health ; Humans ; Life Expectancy ; Malnutrition/epidemiology ; Malnutrition/mortality ; Malnutrition/physiopathology ; Nutritional Status ; Obesity/epidemiology ; Obesity/mortality ; Obesity/physiopathology ; Risk Assessment ; Risk Factors ; Syndemic
Keywords	covid19
Language	English
Publishing date	2020-07-10
Publishing country	United States
Document type	Journal Article
ZDB-ID	209312-1
ISSN	1873-1740 ; 1532-8643 ; 0033-0620
ISSN (online)	1873-1740 ; 1532-8643
ISSN	0033-0620
DOI	10.1016/j.pcad.2020.07.002
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Article: The global food syndemic: The impact of food insecurity, Malnutrition and obesity on the healthspan amid the COVID-19 pandemic

Huizar, Martha I / Arena, Ross / Laddu, Deepika R

Prog. cardiovasc. dis

Keywords	covid19
Publisher	WHO
Document type	Article
Note	WHO #Covidence: #639247
Database	COVID19

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Article ; Online: The global food syndemic

Huizar, Martha I. / Arena, Ross / Laddu, Deepika R.

Progress in Cardiovascular Diseases ; ISSN 0033-0620

The impact of food insecurity, Malnutrition and obesity on the healthspan amid the COVID-19 pandemic

2020

Keywords	Cardiology and Cardiovascular Medicine ; covid19
Language	English
Publisher	Elsevier BV
Publishing country	us
Document type	Article ; Online
DOI	10.1016/j.pcad.2020.07.002
Database	BASE - Bielefeld Academic Search Engine (life sciences selection)

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Article ; Online: Moving more and sitting less in schools: What's the next step?

Strieter, Lindsey / Arena, Ross / Huizar, Martha

Progress in cardiovascular diseases

2020 Volume 64, Page(s) 22–26

Abstract: Schools serve as the ideal location for implementing interventions to increase physical activity (PA) as children spend most of their day in the school setting. As adolescents become more and more sedentary and obesity statistics become more dire, ... ...

Abstract	Schools serve as the ideal location for implementing interventions to increase physical activity (PA) as children spend most of their day in the school setting. As adolescents become more and more sedentary and obesity statistics become more dire, efforts to increase physical literacy and PA should be heightened. Physical literacy is the ability for a child to understand the movement of their body and how it can be manipulated to increase activity for recreation or sports movement. When physical literacy is paired with school-based multi-component programs, children are more likely to make a behavior change. As educators know, children are more likely to make a behavior change when mastery of content is achieved, and the lesson is tailored to their needs. Even small changes, like moving a little more or adding an additional serving of vegetables to the diet, can make a profound impact. In the current review we discuss: 1) the state of PA within school systems; 2) provide a rationale for why school systems fail to meet said guidelines; and 3) suggest how guidelines can eventually be achieved through the promotion of physical literacy and effective school-based multi component programs.
MeSH term(s)	Exercise/physiology ; Health Promotion ; Humans ; Motor Activity/physiology ; School Health Services/organization & administration ; Schools ; Sedentary Behavior ; Sitting Position
Language	English
Publishing date	2020-12-25
Publishing country	United States
Document type	Journal Article ; Review
ZDB-ID	209312-1
ISSN	1873-1740 ; 1532-8643 ; 0033-0620
ISSN (online)	1873-1740 ; 1532-8643
ISSN	0033-0620
DOI	10.1016/j.pcad.2020.12.002
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Article ; Online: Diagnostic accuracy of the BioFire® FilmArray® pneumonia panel in COVID-19 patients with ventilator-associated pneumonia.

Cojuc-Konigsberg, Gabriel / Moscona-Nissan, Alberto / Guijosa, Alberto / Mireles Dávalos, Christian D / Martínez, María E Jiménez / Mújica Sánchez, Mario A / Hernández Huizar, Víctor F / Durán Barrón, Martha A / Gómez, Karen Villarreal / Andrade-Galindo, Regina / Ordóñez-Oviedo, Montserrat / Brito, Grecia Deloya / Vargas, Eduardo Becerril

BMC infectious diseases

2023 Volume 23, Issue 1, Page(s) 524

Abstract: Background: Ventilator-Associated pneumonia (VAP) is one of the leading causes of morbidity and mortality in critically ill COVID-19 patients in lower-and-middle-income settings, where timely access to emergency care and accurate diagnostic testing is ... ...

Abstract	Background: Ventilator-Associated pneumonia (VAP) is one of the leading causes of morbidity and mortality in critically ill COVID-19 patients in lower-and-middle-income settings, where timely access to emergency care and accurate diagnostic testing is not widely available. Therefore, rapid microbiological diagnosis is essential to improve effective therapy delivery to affected individuals, preventing adverse outcomes and reducing antimicrobial resistance. Methods: We conducted a cross-sectional study of patients with suspected VAP and COVID-19, evaluating the diagnostic performance of the BioFire® FilmArray® Pneumonia Panel (FA-PP). Respiratory secretion samples underwent standard microbiological culture and FA-PP assays, and the results were compared. Results: We included 252 samples. The traditional culture method detected 141 microorganisms, and FA-PP detected 277, resulting in a sensitivity of 95% and specificity of 60%, with a positive predictive value of 68% and negative predictive value of 93%. In samples with high levels of genetic material (> 10^5 copies/mL), the panel had a sensitivity of 94% and specificity of 86%. In addition, 40% of the culture-negative samples had positive FA-PP® results, of which 35% had > 10^5 copies/mL of genetic material. The most prevalent bacteria were Gram-negative bacilli, followed by Gram-positive cocci. The panel identified 98 genes associated with antimicrobial resistance, predominantly extended-spectrum beta-lactamases (28%). Conclusion: The FA-PP is a sensitive assay for identifying bacteria causing VAP in patients with COVID-19, with a greater capacity to detect bacteria than the conventional method. The timely microbiological recognition offered by this panel could lead to optimized decision-making processes, earlier tailored treatment initiation, and improved antibiotic stewardship practices.
MeSH term(s)	Humans ; Pneumonia, Ventilator-Associated/diagnosis ; Pneumonia, Ventilator-Associated/microbiology ; COVID-19/diagnosis ; Cross-Sectional Studies ; Bacteria/genetics ; Anti-Infective Agents ; Pneumonia ; COVID-19 Testing
Chemical Substances	Anti-Infective Agents
Language	English
Publishing date	2023-08-09
Publishing country	England
Document type	Journal Article
ZDB-ID	2041550-3
ISSN	1471-2334 ; 1471-2334
ISSN (online)	1471-2334
ISSN	1471-2334
DOI	10.1186/s12879-023-08486-4
Database	MEDical Literature Analysis and Retrieval System OnLINE

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Article ; Online: Post-extrasystolic potentiation as a predictor of premature ventricular contraction-cardiomyopathy in an animal model.

Kowlgi, Gurukripa N / Ramirez, Rafael J / Kaszala, Karoly / Joslyn, Martha / Koneru, Jayanthi N / Ellenbogen, Kenneth A / Tan, Alex Y / Huizar, Jose F

Europace : European pacing, arrhythmias, and cardiac electrophysiology : journal of the working groups on cardiac pacing, arrhythmias, and cardiac cellular electrophysiology of the European Society of Cardiology

2020 Volume 22, Issue 5, Page(s) 813–820

Abstract: Aims: High premature ventricular contractions (PVCs) burden does not always predict the development of PVC-cardiomyopathy (CM). We sought to evaluate post-extrasystolic potentiation (PESP) of left ventricular ejection fraction (LVEF) to predict the ... ...

Abstract	Aims: High premature ventricular contractions (PVCs) burden does not always predict the development of PVC-cardiomyopathy (CM). We sought to evaluate post-extrasystolic potentiation (PESP) of left ventricular ejection fraction (LVEF) to predict the severity of PVC-CM in an animal model. Methods and results: Right ventricular apical bigeminal PVCs were introduced for 12 weeks in 11 canines to induce PVC-CM. Echocardiograms were performed to obtain LVEF without ectopy (Echo-1) and during PVCs (200 and 350 ms coupling intervals, Echo-2, and Echo-3, respectively), and premature atrial contractions (PACs) (Echo-4) at baseline and after 12 weeks of bigeminal PVCs. PESP was calculated as delta-LVEF between the sinus beat post-ectopy LVEF (Echo-2, -3, and -4, respectively) and LVEF without PVC (Echo-1) at baseline and 12 weeks of high PVC burden. A hyperdynamic LV function (LVEF > 70%) was noted in all animals only with early-coupled PVCs (LVEF at 200 ms: 74.4 ± 6%) at baseline. While PVC PESP at 200 ms had a strong significant correlation with the final 12-week LVEF (R = 0.8, P = 0.003), PVC PESP at 350 ms and PAC PESP had a positive but non-significant correlation (R = 0.53, P = 0.09, and R = 0.29, P = 0.34, respectively). Premature ventricular contraction PESP at 350 ms was significantly higher after PVC-CM had developed (delta-LVEF baseline 2.7 ± 2.9% vs. 12 weeks 18.6 ± 12.3% P < 0.001). Conclusion: Bigeminal early-coupled PVCs cause hyperdynamic left ventricular function in the structurally normal canine heart due to PESP. The degree of PESP at baseline is inversely proportional to the PVC-CM severity at 12 weeks and maybe a predictor of PVC-CM as it may assess the myocardial adaptation reserve to PVCs.
MeSH term(s)	Animals ; Cardiomyopathies ; Dogs ; Echocardiography ; Stroke Volume ; Ventricular Function, Left ; Ventricular Premature Complexes/diagnosis
Language	English
Publishing date	2020-03-17
Publishing country	England
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	1449879-0
ISSN	1532-2092 ; 1099-5129
ISSN (online)	1532-2092
ISSN	1099-5129
DOI	10.1093/europace/euaa025
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Article ; Online: Chronic stress decreases ornithine decarboxylase expression and protects against 1,2-dimethylhydrazine-induced colon carcinogenesis.

Zamora-González, Edgar Oswaldo / Castro-Félix, Patricia / Huizar-López, María Del Rosario / Casas-Solís, Josefina / Marques-González, María de la Luz Blanca Isabel / Martin Del Campo-Solís, Martha Fabiola / Santerre, Anne

Molecular biology reports

2020 Volume 47, Issue 12, Page(s) 9429–9439

Abstract: Biological response to stress depends on the type, timing, and severity of the stressor. Acute stressful environments may positively activate molecular and cellular mechanisms to favor adaptation; however, chronic stress is often associated with ... ...

Abstract	Biological response to stress depends on the type, timing, and severity of the stressor. Acute stressful environments may positively activate molecular and cellular mechanisms to favor adaptation; however, chronic stress is often associated with detrimental health effects. Colon cancer (CC) is one of the leading causes of death associated with cancer and has been mentioned as a stress-related disease. In the present work, the effect of chronic stress on the initial phase of CC was evaluated, and special emphasis was placed on ornithine decarboxylase (ODC) expression and polyamines for their role in hyperproliferative diseases. BALB/c mice (n = 5/group) were administered the pro-carcinogen 1,2-dimethylhydrazine (DMH) for 8 weeks (20 mg/kg body weight/week) to induce colon carcinogenesis, and then exposed for 4 weeks to two physical stressors: restraint and forced-swimming. Distal colon inflammatory lesions and histomorphological changes were evaluated by hematoxylin-eosin staining; plasma corticosterone levels, colon ODC expression, and urinary polyamines were determined by competitive ELISA, RT-qPCR, Western Blot, and HPLC, respectively. The short-term exposure to DMH triggered colon inflammation, initiated colon carcinogenesis and increased ODC expression; meanwhile, the exposure to chronic stress activated the hypothalamic-pituitary-adrenal (HPA) axis, elicited the production of plasmatic corticosterone, and decreased ODC expression. The exposure of DMH-treated mice to chronic stress counteracted the inflammatory effect of DMH and maintained ODC homeostasis. In early phase of carcinogenesis, the exposure of DMH-treated mice to chronic stress had a positive effect against colon inflammation and maintained ODC homeostasis. The cross-talk between corticosterone, ODC expression, and inflammation in a tumor environment is discussed.
MeSH term(s)	1,2-Dimethylhydrazine/administration & dosage ; 1,2-Dimethylhydrazine/adverse effects ; Animals ; Carcinogenesis/drug effects ; Carcinogenesis/metabolism ; Carcinogens/administration & dosage ; Colon/metabolism ; Colonic Neoplasms/blood ; Colonic Neoplasms/chemically induced ; Colonic Neoplasms/urine ; Corticosterone/blood ; Female ; Hypothalamo-Hypophyseal System/metabolism ; Mice ; Mice, Inbred BALB C ; Ornithine Decarboxylase/metabolism ; Polyamines/urine ; Signal Transduction/drug effects ; Stress, Physiological
Chemical Substances	Carcinogens ; Polyamines ; Ornithine Decarboxylase (EC 4.1.1.17) ; 1,2-Dimethylhydrazine (IX068S9745) ; Corticosterone (W980KJ009P)
Language	English
Publishing date	2020-12-01
Publishing country	Netherlands
Document type	Journal Article
ZDB-ID	186544-4
ISSN	1573-4978 ; 0301-4851
ISSN (online)	1573-4978
ISSN	0301-4851
DOI	10.1007/s11033-020-06022-0
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Article ; Online: Persistent Proarrhythmic Neural Remodeling Despite Recovery From Premature Ventricular Contraction-Induced Cardiomyopathy.

Tan, Alex Y / Elharrif, Khalid / Cardona-Guarache, Ricardo / Mankad, Pranav / Ayers, Owen / Joslyn, Martha / Das, Anindita / Kaszala, Karoly / Lin, Shien-Fong / Ellenbogen, Kenneth A / Minisi, Anthony J / Huizar, Jose F

Journal of the American College of Cardiology

2019 Volume 75, Issue 1, Page(s) 1–13

Abstract: Background: The presence and significance of neural remodeling in premature ventricular contraction-induced cardiomyopathy (PVC-CM) remain unknown.: Objectives: This study aimed to characterize cardiac sympathovagal balance and proarrhythmia in a ... ...

Abstract	Background: The presence and significance of neural remodeling in premature ventricular contraction-induced cardiomyopathy (PVC-CM) remain unknown. Objectives: This study aimed to characterize cardiac sympathovagal balance and proarrhythmia in a canine model of PVC-CM. Methods: In 12 canines, the investigators implanted epicardial pacemakers and radiotelemetry units to record cardiac rhythm and nerve activity (NA) from the left stellate ganglion (SNA), left cardiac vagus (VNA), and arterial blood pressure. Bigeminal PVCs (200 ms coupling) were applied for 12 weeks to induce PVC-CM in 7 animals then disabled for 4 weeks to allow complete recovery of left ventricular ejection fraction (LVEF), versus 5 sham controls. Results: After 12 weeks of PVCs, LVEF (p = 0.006) and dP/dT (p = 0.007) decreased. Resting SNA (p = 0.002) and VNA (p = 0.04), exercise SNA (p = 0.01), SNA response to evoked PVCs (p = 0.005), heart rate (HR) at rest (p = 0.003), and exercise (p < 0.04) increased, whereas HR variability (HRV) decreased (p = 0.009). There was increased spontaneous atrial (p = 0.02) and ventricular arrhythmias (p = 0.03) in PVC-CM. Increased SNA preceded both atrial (p = 0.0003) and ventricular (p = 0.009) arrhythmia onset. Clonidine suppressed SNA and abolished all arrhythmias. After disabling PVC for 4 weeks, LVEF (p = 0.01), dP/dT (p = 0.047), and resting VNA (p = 0.03) recovered to baseline levels. However, SNA, resting HR, HRV, and atrial (p = 0.03) and ventricular (p = 0.03) proarrhythmia persisted. There was sympathetic hyperinnervation in stellate ganglia (p = 0.02) but not ventricles (p = 0.2) of PVC-CM and recovered animals versus sham controls. Conclusions: Neural remodeling in PVC-CM is characterized by extracardiac sympathetic hyperinnervation and sympathetic neural hyperactivity that persists despite normalization of LVEF. The altered cardiac sympathovagal balance is an important trigger and substrate for atrial and ventricular proarrhythmia.
MeSH term(s)	Animals ; Cardiomyopathies/diagnostic imaging ; Cardiomyopathies/physiopathology ; Dogs ; Echocardiography/methods ; Electrocardiography/methods ; Heart Rate/physiology ; Myocardial Contraction/physiology ; Recovery of Function/physiology ; Ventricular Premature Complexes/diagnostic imaging ; Ventricular Premature Complexes/physiopathology ; Ventricular Remodeling/physiology
Language	English
Publishing date	2019-12-27
Publishing country	United States
Document type	Journal Article ; Research Support, N.I.H., Extramural ; Research Support, Non-U.S. Gov't
ZDB-ID	605507-2
ISSN	1558-3597 ; 0735-1097
ISSN (online)	1558-3597
ISSN	0735-1097
DOI	10.1016/j.jacc.2019.10.046
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Article: Chronic stress decreases ornithine decarboxylase expression and protects against 1,2-dimethylhydrazine-induced colon carcinogenesis

Zamora-González, Edgar Oswaldo / Castro-Félix, Patricia / Huizar-López, María del Rosario / Casas-Solís, Josefina / Marques-González, María de la Luz Blanca Isabel / Martin del Campo-Solís, Martha Fabiola / Santerre, Anne

Molecular biology reports. 2020 Dec., v. 47, no. 12

2020

Abstract	Biological response to stress depends on the type, timing, and severity of the stressor. Acute stressful environments may positively activate molecular and cellular mechanisms to favor adaptation; however, chronic stress is often associated with detrimental health effects. Colon cancer (CC) is one of the leading causes of death associated with cancer and has been mentioned as a stress-related disease. In the present work, the effect of chronic stress on the initial phase of CC was evaluated, and special emphasis was placed on ornithine decarboxylase (ODC) expression and polyamines for their role in hyperproliferative diseases. BALB/c mice (n = 5/group) were administered the pro-carcinogen 1,2-dimethylhydrazine (DMH) for 8 weeks (20 mg/kg body weight/week) to induce colon carcinogenesis, and then exposed for 4 weeks to two physical stressors: restraint and forced-swimming. Distal colon inflammatory lesions and histomorphological changes were evaluated by hematoxylin–eosin staining; plasma corticosterone levels, colon ODC expression, and urinary polyamines were determined by competitive ELISA, RT-qPCR, Western Blot, and HPLC, respectively. The short-term exposure to DMH triggered colon inflammation, initiated colon carcinogenesis and increased ODC expression; meanwhile, the exposure to chronic stress activated the hypothalamic–pituitary–adrenal (HPA) axis, elicited the production of plasmatic corticosterone, and decreased ODC expression. The exposure of DMH-treated mice to chronic stress counteracted the inflammatory effect of DMH and maintained ODC homeostasis. In early phase of carcinogenesis, the exposure of DMH-treated mice to chronic stress had a positive effect against colon inflammation and maintained ODC homeostasis. The cross-talk between corticosterone, ODC expression, and inflammation in a tumor environment is discussed.
Keywords	1,2-dimethylhydrazine ; Western blotting ; acute exposure ; body weight ; carcinogenesis ; colon ; colorectal neoplasms ; corticosterone ; death ; homeostasis ; inflammation ; molecular biology ; ornithine decarboxylase ; polyamines ; stress response
Language	English
Dates of publication	2020-12
Size	p. 9429-9439.
Publishing place	Springer Netherlands
Document type	Article
Note	NAL-AP-2-clean
ZDB-ID	186544-4
ISSN	1573-4978 ; 0301-4851
ISSN (online)	1573-4978
ISSN	0301-4851
DOI	10.1007/s11033-020-06022-0
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